Academic literature on the topic 'Barry Commoner'

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Journal articles on the topic "Barry Commoner"

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McGowan, Alan H. "Remembering Barry Commoner." Environment: Science and Policy for Sustainable Development 55, no. 2 (March 2013): 17. http://dx.doi.org/10.1080/00139157.2013.765312.

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Egan, Michael. "Why Barry Commoner Matters." Organization & Environment 22, no. 1 (March 2009): 6–18. http://dx.doi.org/10.1177/1086026609333421.

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Brett-Crowther, Michael. "Barry Commoner (1917–2012)." International Journal of Environmental Studies 69, no. 6 (December 2012): 867–68. http://dx.doi.org/10.1080/00207233.2012.739723.

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Markowitz, Steven. "Barry Commoner and the Current Environmental Crisis." American Journal of Public Health 108, S2 (April 2018): S53. http://dx.doi.org/10.2105/ajph.2018.304411.

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Testa, Chicco. "Barry Commoner's Day." NEW SOLUTIONS: A Journal of Environmental and Occupational Health Policy 8, no. 1 (May 1998): 69–73. http://dx.doi.org/10.1177/104829119800800110.

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“Barry Commoner's Day” represents for me a great opportunity to settle up my debt of gratitude to Barry Commoner. I thank Barry for the personal friendship he has honored me with for many years, allowing me to take advantage of his experience, his good advice, and his scientific and political teaching. He used to be an incorrigible optimist. And I hope he has not changed with the passing of the years because God knows how much environmentalists need people like him since they have a tendency to complain and foretell misfortunes. But Barry's contribution is not just optimism. His great contribution lies in his ability in matching economic and social rationality, technological progress, and the minimization of environmental impacts. That is to say, the finding that at the basis of the processes involving the destruction of natural resources, there is often an irrational behavior, which is technologically and economically disadvantageous.
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Labotz, Dan. "Barry Commoner, a Great Presidential Candidate (1917–2012)." NEW SOLUTIONS: A Journal of Environmental and Occupational Health Policy 22, no. 4 (February 2013): 419–21. http://dx.doi.org/10.2190/ns.22.4.b.

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Montague, Peter. "Barry Commoner: The Father of Grass-Roots Environmentalism." NEW SOLUTIONS: A Journal of Environmental and Occupational Health Policy 8, no. 1 (May 1, 1998): 1. http://dx.doi.org/10.2190/p51g-hm6k-u42u-jag5.

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Montague, Peter. "Barry Commoner: the Father of Grass-Roots Environmentalem." NEW SOLUTIONS: A Journal of Environmental and Occupational Health Policy 8, no. 1 (May 1998): 7–16. http://dx.doi.org/10.1177/104829119800800103.

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In the late 1950s and early 1960s, Barry Commoner and his colleagues in the Committee for Nuclear Information and at Washington University in St. Louis developed many of the fundamental arguments and ideas that, today, underpin and propel the grassroots movement for environmental justice, such as: moral wisdom resides in the citizenry; scientists have no special wisdom in moral matters; scientists must make alliances with citizens; pollution must be prevented because it cannot be managed; the burden of proof rests on the polluter; citizens have a right to know; the principle of precautionary action should guide our decisions; environmental impact assessments are essential; and risk assessment is political.
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Berlinguer, Giovanni. "The Contribution of Barry Commoner to the Renewal of the Italian Left." NEW SOLUTIONS: A Journal of Environmental and Occupational Health Policy 8, no. 1 (May 1998): 65–68. http://dx.doi.org/10.1177/104829119800800109.

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Barry Commoner had a strong positive influence on the ideas and policies of the Italian left since the 1970s. His books were translated, and he was frequently present in the environmental movement His criticism of the “Soviet model” found a favorable echo in the Italian Communist Party, whose autonomy had grown in the post-war period, and helped to include new ideas in its policy. Nowadays, left or center-left parties and alliances, often including “green” forces, lead the governments in thirteen out of fifteen countries belonging to the European Union, as a result of democratic elections. This is a result of both a strong tradition and of a renewal of strategies and programs to which Barry Commoner contributed with his ideas and his political courage.
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Mazzocchi, Tony. "Crossing Paths: Science and the Working Class." NEW SOLUTIONS: A Journal of Environmental and Occupational Health Policy 8, no. 1 (May 1998): 27–32. http://dx.doi.org/10.1177/104829119800800105.

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My first meeting with Barry Commoner developed out of my interest in nuclear weapons testing, an issue that had engrossed Commoner and the Committee for Nuclear Information (CNI) for some time. Then, our union local worked collaboratively with the scientists on the Baby Tooth Survey which showed an association between nuclear fallout and the level of strontium-90 in children's teeth. Linking the CNI scientists and the working class in this way produced results and Commoner and his colleagues made themselves available to respond to corporate scientists on issues such as toxic workplaces exposures. We were successful in raising occupational health and safety questions because we had credible scientists on our side. Through the association with Barry, we changed the face of the workplace and thousands of people are alive today because we helped pass the federal Occupational Safety and Health Act. A whole cadre of health and safety specialists came out of those efforts.
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Dissertations / Theses on the topic "Barry Commoner"

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Egan, Michael. "Barry Commoner and the science of survival." Online access for everyone, 2004. http://www.dissertations.wsu.edu/Dissertations/Fall2004/m%5Fegan%5F120104.pdf.

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Wight, Philip A. "From Citizens to Consumers: The Countercultural Roots of Green Consumerism." Ohio University / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1368030088.

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Nilsson, Peter. "NGO involvement in the UN Conference on the Human Environment in Stockholm 1972. Interrelations Between Intergovernmental Discourse Framing and Activist Influence." Thesis, Linköping University, Department of Management and Economics, 2004. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-2201.

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The UN Conference on the Human Environment in Stockholm 1972 has been recognized as bringing political attention to environmental problems. Researchers have acknowledged the importance of NGO activities during the conference, initiating a trend of engagement of NGOs in official global meetings. But NGOs were not permitted to speak at the plenary or participate in working groups in the official Conference. The influence of NGOs could still be substantial but in another arenas delivering perceptions, knowledge and information to the general public and officials, directly or through the intense media coverage of the conference. NGOs engaged in these parallel activities and individuals in the official initiating process are central to this research.

The purpose of this study is to analyze how Swedish NGOs and their related networks influenced environmental discourses during and following the UN conference on the human environment in Stockholm 1972. The purpose is also to analyze how they in turn were effected by the conference process and the context in which NGOs function.

This study is concerned with how social movements became engaged in official global meetings and the effects of this process. It is a study of the interrelations between intergovernmental discourse framing and activist influence. To understand this we take in consideration what motivated the actions of relevant actors, how actors selected strategies to obtain there purposes and how diverse frames of understanding emerged.

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Cox, Charles John. "Persistent EBV infection in the common marmoset : the effect of immunization with envelope glyco protein gp 340." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266318.

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Pésinho, Inês Vaz. "Multiple Sclerosis vs. Guillain-Barré syndrome: differences in two autoimmune disorders with a common target in two different regions." Master's thesis, 2019. http://hdl.handle.net/10451/43421.

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Trabalho Final de Mestrado Integrado, Ciências Farmacêuticas, Universidade de Lisboa, Faculdade de Farmácia, 2019
A Esclerose Múltipla (EM) e o Síndrome Guillain-Barré (SGB) são ambos doenças autoimunes e desmielinizantes que afetam, respetivamente, o Sistema Nervoso Central (SNC) e o Sistema Nervoso Periférico (SNP), pertencendo ainda a um grupo de doenças neurodegenerativas que envolvem lesões inflamatórias associadas a desmielinização, induzindo dano no axónio e consequente neurodegeneração, o que leva a uma perda de função progressiva. A EM é uma doença inflamatória crónica do SNC sendo a causa mais frequente de distúrbios neurológicos em jovens adultos. É uma doença que consiste na inflamação, desmielinização e uma variável perda axonal. A sua etiologia ainda não é completamente conhecida, mas presume-se que envolva a interação entre fatores genéticos e ambientais, estimulando um ataque autoimune e consequentes danos na mielina e nos axónios. Clinicamente, a maior parte dos doentes tem uma fase recidivante-remitente, caracterizada pela presença de surtos seguida de recuperação. Destes doentes, a maioria progride para uma doença secundária progressiva e os restantes doentes desenvolvem uma Esclerose Múltipla primária progressiva. Alguns doentes têm ainda um síndrome clinicamente isolado que corresponde a um primeiro episódio de sintomas neurológicos no SNC, sendo que estes podem ou não evoluir para Esclerose Múltipla. Em termos de tratamento, estão aprovados os medicamentos modificadores de doença, especialmente no caso de doença recidivante-remitente. A SGB é uma doença inflamatória, mas do SNP, sendo a causa mais frequente de paralisia flácida aguda. Esta doença autoimune é antecedida por uma infeção viral ou bacteriana, como vírus Influenza ou Campilobacter jejuni, que são capazes de desencadear uma resposta imune anormal direcionada contra os componentes dos nervos periféricos, por mimetismo molecular. As formas mais frequentes são polineuropatia desmielinizante inflamatória aguda e neuropatia motora axonal aguda, existindo ainda a neuropatia motora sensorial axonal aguda e a síndrome de Miller Fisher. Os doentes com SGB têm insuficiência respiratória e disfunção autónoma como complicações associadas. O tratamento é composto por uma abordagem multidisciplinar que inclui cuidados médicos gerais e imunoterapia. As prioridades na investigação da EM e da SGB incluem o desenvolvimento de biomarcadores e um melhor conhecimento da imunopatogénese, para que haja medicina personalizada.
Multiple Sclerosis (MS) and Guillain-Barré Syndrome (GBS) are both demyelinating and autoimmune disorders affecting, respectively, the central nervous system (CNS) and the peripheral nervous system (PNS), which means they belong to a group of neurodegenerative diseases that involve inflammatory lesions associated with demyelination, inducing axonal damage and consequent neurodegeneration, leading to progressive loss of function. MS is a chronic inflammatory disorder of the CNS and is assumed to be the most frequent cause of neurological disability in young adults. This disorder consists in inflammation, demyelination and variable levels of axonal loss. The etiology is still unknown but it is presumed to involve interaction between genetic and environmental factors that triggers an autoimmune attack, resulting in damaged myelin and axons. Clinically, most of the patients experience a relapsing-remitting phase, characterized by relapses followed by recovery. The majority of them, late on enter in a progressive phase called secondary progressive MS. The remaining patients pursue a progressive course that is called primary progressive MS. There is also clinically isolated syndrome corresponding to a first episode of neurologic symptoms in the CNS, and people who experience it may or may not develop MS. In terms of therapeutic options, disease-modifying treatments are approved specially to treat relapsing remitting form of the disease. GBS is also an inflammatory demyelinating disease of the PNS and it is the most frequent cause of acute flaccid paralysis. This autoimmune disorder is, in most cases, preceded by viral or bacterial infections, such as Campylobacter jejuni or Influenza virus, that are capable of triggering an abnormal immune responses directed against components of the peripheral nerves by molecular mimicry. Clinically, the most frequent forms of GBS is acute inflammatory demyelinating polyradiculoneuropathy and acute motor axonal neuropathy, but there is also acute motor-sensory axonal neuropathy and Miller-Fischer syndrome. Patients with GBS commonly have respiratory insufficiency and autonomic dysfunction as associated complications. The treatment of this syndrome is composed by a multidisciplinary approach that includes general medical care and immunotherapies. The priorities for MS and GBS investigation include establishment of biomarkers and an improved knowledge of the immunopathogenesis, to go towards personalized medicine.
Farmácia Lisboa; Hospital de Santo António dos Capuchos
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"The study of Chinese medicinal herbs and Chinese food items commonly consumed in Hong Kong for the induction of Epstein-barr virus-specific early antigen in the Raji cell line." Chinese University of Hong Kong, 1989. http://library.cuhk.edu.hk/record=b5886210.

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Books on the topic "Barry Commoner"

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L, Eichler Barry, ed. A common cultural heritage: Studies on Mesopotamia and the biblical world in honor of Barry l. Eichler. Bethesda, Md: CDL Press, 2011.

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Egan, Michael. Barry Commoner and the Science of Survival. The MIT Press, 2007. http://dx.doi.org/10.7551/mitpress/1522.001.0001.

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Barry Commoner And The Science Of Survival The Remaking Of American Environmentalism. MIT Press (MA), 2009.

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Barry Commoner and the Science of Survival: The Remaking of American Environmentalism (Urban and Industrial Environments). The MIT Press, 2007.

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L, Kriebel David, and Dunn Mary Lee, eds. Barry Commoner's contribution to the environmental movement: Science and social action. Amityville, N.Y: Baywood, 2002.

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Kriebel, David L., and Mary Lee Dunn. Barry Commoner's Contribution to the Environmental Movement: Science and Social Action. Taylor & Francis Group, 2019.

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Lee Dunn, Mary. Barry Commoner’s Contribution to the Environmental Movement: Science and Social Action. Edited by David Kriebel. Routledge, 2019. http://dx.doi.org/10.4324/9781315225050.

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Kriebel, David, ed. Barry Commoner's Contribution to the Environmental Movement: Science and Social Action. Baywood Publishing Co., Inc., 2000. http://dx.doi.org/10.2190/bcc.

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Brealey, David, and Nicholas Hirsch. Diagnosis, assessment, and management of Guillain–Barré syndrome. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0246.

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The Guillain–Barré Syndrome describes a spectrum of acute inflammatory polyneuropathies and is the commonest cause of acute flaccid paralysis within the western world. The pathophysiology is complex and poorly understood, but appears to be an immune-mediated destruction of either the myelin sheath and/or the axons, predominantly of motor nerves. The clinical presentation is classically a rapid, ascending, flaccid paralysis, with minimal sensory deficit. This may ascend to involve respiratory or bulbar muscle function. These patients need careful monitoring and, if deteriorating, should be electively intubated and ventilated. Autonomic instability and sensory disturbance, including pain, is common. Treatment of the underlying condition relies upon immunomodulation with either intravenous immunoglobulin or plasma exchange. Supportive care is aimed at maintaining a safe airway, ventilatory support, and managing the complications of autonomic dysfunction and prolonged immobility. Mortality rates range up to 20%, but are significantly better in specialist neuromedical units. Survivors are often left with significant disability.
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Kriebel, David L. Barry Commoner's Contribution to the Environmental Movement: Science and Social Action (Work, Health and Environment Series). Baywood Publishing Company, 2000.

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Book chapters on the topic "Barry Commoner"

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Tosato, G., S. E. Pike, and R. M. Blaese. "Reversal of Common Variable Hypogammaglobulinemia-Associated Suppressor Cell Activity by Specific Carbohydrates." In Epstein-Barr Virus and Associated Diseases, 43–52. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2625-0_4.

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Dreier, Peter. "Barry Commoner 1917–2012." In Key Thinkers on The Environment, 243–49. Routledge, 2017. http://dx.doi.org/10.4324/9781315543659-44.

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Montague, Peter. "Barry Commoner: The Father of Grass-Roots Environmentalism." In Barry Commoner’s Contribution to the Environmental Movement: Science and Social Action, 5–14. Routledge, 2019. http://dx.doi.org/10.4324/9781315225050-2.

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Montague, Peter. "Barry Commoner: The Father of Grass-Roots Environmentalism." In Barry Commoner's Contribution to the Environmental Movement: Science and Social Action. Baywood Publishing Co., Inc., 2000. http://dx.doi.org/10.2190/bccc1.

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"In the Thunderclap’s Wake." In Barry Commoner and the Science of Survival. The MIT Press, 2007. http://dx.doi.org/10.7551/mitpress/1522.003.0003.

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"Guarding the Public." In Barry Commoner and the Science of Survival. The MIT Press, 2007. http://dx.doi.org/10.7551/mitpress/1522.003.0004.

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"The New Jeremiad." In Barry Commoner and the Science of Survival. The MIT Press, 2007. http://dx.doi.org/10.7551/mitpress/1522.003.0005.

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"When Scientists Disagree." In Barry Commoner and the Science of Survival. The MIT Press, 2007. http://dx.doi.org/10.7551/mitpress/1522.003.0006.

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"Biological Capital." In Barry Commoner and the Science of Survival. The MIT Press, 2007. http://dx.doi.org/10.7551/mitpress/1522.003.0007.

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"The “Other” Environmentalism." In Barry Commoner and the Science of Survival. The MIT Press, 2007. http://dx.doi.org/10.7551/mitpress/1522.003.0008.

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Conference papers on the topic "Barry Commoner"

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Gupta, Ishita, Ayesha Jabeen, Maria K. Smatti, Hamda A. Al-Thawadi, Gheyath K. Nasrallah, Ali A. Sultan, Moussa Al-Khalaf, Semir Vranic, and Ala-Eddin Al-Moustafa. "Co-Prevalence of Human Papillomavirus and Epstein Barr Virus in Healthy Blood Donors in Qatar." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0120.

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Introduction: Infections with human oncoviruses such as high-risk human papillomaviruses (HPVs) and Epstein-Barr virus (EBV) are globally prevalent in the adult population. Both viruses are strongly associated with several types of human carcinomas such as cervical, head and neck, nasopharyngeal and gastric. In the present study, we explored the prevalence of these two oncoviruses in the healthy population of Qatar. Methods: The study included 385 healthy blood donors that reflect diverse nationalities in the Qatari community (Qatar, Egypt, Syria, Jordan, Pakistan, and India). DNA was extracted from the peripheral blood and genotyping was done using PCR and nested-PCR targeting E6 and E7 as well as LMP1 genes of HPVs and EBV, respectively. Results: The age of participants (378 males and 7 females) ranged between 19 and 68 years (mean 37.12 ± 9.3 years). Our data indicate that 55% and 61% of the tested samples were HPVs and EBV positive, respectively. Moreover, we found that there was (40%) co-presence of both HPVs and EBV in our samples. The most common high-risk HPV types in Qatar included HPV 59 (55%), 31 (54%), 52 (49%), 51 (49%), 58 (47%) and 35 (46%). While, HPV 16 and 18 were detected in 38% and 36% of the samples, respectively. Notably, all samples showed multiple HPVs infections. Conclusion: Our study reveals for the first time a high prevalence of both EBV and HPVs among healthy individuals in Qatar. More significantly, most cases had multiple HPV types infection in addition to the co-presence of both viruses in a substantial proportion of the samples. Given the important possible cooperative role of these viruses in human carcinogenesis, preventive measures using available and upcoming vaccines are of paramount importance.
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Folts, J. D. "A MODEL OF ACUTE PLATELET THROMBUS FORMATION IN STENOSED CORONARY AND CAROTID ARTERIES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643712.

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There is currently a great deal of interest in the diagnosis and treatment of unstable angina and silent ischemia.Many feel that these syndromes are due, in part, to periodic accumulation of platelet thrombi which subsequently embolize.In addition, anti-piatelet therapy is also considered necessary for patients after coronary artery bypass grafts (CABG'S), balloon angioplasty, and thrombolysis. Currently the two antiplatelet agents most commonly prescribed for the patient conditions mentioned above are aspirin (ASA), alone or in combination with dipyridamole (Dip). ASA reduces cardiac events in patients with unstable angina, and prolongs CABG graft patency. The addition of Dip to ASA therapy is very confusing since most studies done compared ASA + Dip to placebo. In several studies however,when an ASA group was compared to an ASA + Dip group there was no significant difference.We have developed and will describe ananimal model of coronary artery stenosis in the dog and the pig, or carotid arterystenosis in the monkey and the rabbit, with intimal damage, that simulates some ofthe conditions that exist in patients with coronary or carotid artery disease. The artery to be studied is dissected outand blood flow is continuously measured with an electromagnetic flowmeter probe. As acute platelet thrombus formation (APTF) developes in the stenosed lumen, the blood flow declines to low levels, producing ischemia until the thrombus emobolizesdistally resulting in abrupt restoration of blood flow. These cyclical flow reductions (CFR's), when they occur in the coronaries, produce ECG changes identical to those observed in patients with silent ischemia and unstable angina. They also produce significant transient regional dyskinesis of the ventricular wall, which resolves when blood flow is restored. Histologic examination of myocardial tissue in the bed distal to the stenosis shows focal areas of ischemic change presumably caused by the embolized platelet emboli.We have examined factors which exacerbate the size and frequency of these CFR"ssuch as; IV infusion of epinephrine (E) 0.4 μg/kg/min for 15 min, ventilating the animals with cigarette smoke, infusing nicotine IV, or placing chewing tobacco under the tongue.We have examined four groups of agentswhich prevent APTF in our model.1. Antiplatelet agents including ASA, indomethacin, ibuprofen and several other NSAI agentsas well as several experimental thromboxane synthetase inhibitors. These agents all block the production of TXA2and inhibit APTF in our model. Unfortunately the IV infusion of E reinstates APTtemporarily (by another biochemical pathway) until the E is metabolized. High (2-4 mg/kg) doses of Dip, alone or with sub threshold dose of ASA does nothing to I APTF.However,0.6mg/kg of chi orpromaz i ne abolishes APTF in all four species and protects agents renewal of APTF by E.2. Dietary Substances In our model, caffeine 10 mg/kg, or the extract from two garlic cloves, or enough ethanol to achieve a blood alcohol level of 0.07 mg% all significantly inhibit or abolish APTF in our model.3. Metabolic Inhibitors POCA, an oral hypoglycemic agent, which inhibits mitochondrial beta oxidation of fatty acids also inhibits APTF in our model possibly by reducing ATP production in the platelet.4. We have studied a monoclonal antibody(developed by Dr. Barry Coller) to the platelet I Ib�I I la glycoprotein receptor where fibrinogen binds platelets into aggregates and ultimately leads to APTF. This antibody 0.3 mg/kg/completely inhibits APTF, and also strongly inhibits in vitro platelet aggregation in response to either ADP or collagen given alone or each combined with E. This antibody is the most potent inhibitor of APTF that we have studied.
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