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Books on the topic 'Basal ganglia model'

Author: Grafiati

Published: 10 March 2023

Last updated: 31 July 2025

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1

Chakravarthy, V. Srinivasa, and Ahmed A. Moustafa. Computational Neuroscience Models of the Basal Ganglia. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8494-2.

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2

C, Houk James, Davis Joel L. 1942-, and Beiser David G, eds. Models of information processing in the basal ganglia. MIT Press, 1994.

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3

C, Houk James, Davis Joel L. 1942-, and Beiser David G, eds. Models of information processing in the basal ganglia. MIT Press, 1995.

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4

International Basal Ganglia Society. Symposium. The basal ganglia II: Structure and function : current concepts. Plenum Press, 1987.

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5

P, Riederer, and Wesemann W, eds. Parkinson's disease: Experimental models and therapy. Springer-Verlag, 1995.

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6

Ely, Budding Deborah, ed. Subcortical structures and cognition: Implications for neuropsychological assessment. Springer, 2009.

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7

Steele, Vaughn R., Vani Pariyadath, Rita Z. Goldstein, and Elliot A. Stein. Reward Circuitry and Drug Addiction. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0044.

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Addiction is a complex neuropsychiatric syndrome related to dysregulation of brain systems including the mesocorticolimbic dopamine reward circuit. Dysregulation of reward circuitry is related to each of the three cyclical stages in the disease model of addiction: maintenance, abstinence, and relapse. Parsing reward circuitry is confounded due to the anatomical complexity of cortico-basal ganglia-thalamocortical loops, forward and backward projections within the circuit, and interactions between neurotransmitter systems. We begin by introducing the neurobiology of the reward system, specifical

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8

Symposium, International Basal Ganglia Society. The basal ganglia II. Plenum, 1987.

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9

Chakravarthy, V. Srinivasa, and Ahmed A. Moustafa. Computational Neuroscience Models of the Basal Ganglia. Springer, 2018.

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10

Chakravarthy, V. Srinivasa. Computational Neuroscience Models of the Basal Ganglia. Springer, 2018.

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11

Houk, James C., Joel L. Davis, and David G. Beiser, eds. Models of Information Processing in the Basal Ganglia. The MIT Press, 1994. http://dx.doi.org/10.7551/mitpress/4708.001.0001.

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12

Davis, Joel L., James C. Houk, and David G. Beiser. Models of Information Processing in the Basal Ganglia. MIT Press, 2019.

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13

Parkinson's Disease: Experimental Models And Therapy (Journal of Neural Transmission. Supplement). Springer-Verlag Telos, 1996.

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14

Levine, Michael S., Elizabeth A. Wang, Jane Y. Chen, Carlos Cepeda, and Véronique M. André. Altered Neuronal Circuitry. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199929146.003.0010.

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In mouse models of Huntington’s disease (HD), synaptic alterations in the cerebral cortex and striatum are present before overt behavioral symptoms and cell death. Similarly, in HD patients, it is now widely accepted that early deficits can occur in the absence of neural atrophy or overt motor symptoms. In addition, hyperkinetic movements seen in early stages are followed by hypokinesis in the late stages, indicating that different processes may be affected. In mouse models, such behavioral alterations parallel complex biphasic changes in glutamate-mediated excitatory, γ‎-aminobutyric acid (GA

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15

Koziol, Leonard F., and Deborah Ely Budding. Subcortical Structures and Cognition: Implications for Neuropsychological Assessment. Springer New York, 2010.

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16

Benarroch, Eduardo E. Neuroscience for Clinicians. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190948894.001.0001.

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The aim of this book is to provide the clinician with a comprehensive and clinical relevant survey of emerging concepts on the organization and function of the nervous system and neurologic disease mechanisms, at the molecular, cellular, and system levels. The content of is based on the review of information obtained from recent advances in genetic, molecular, and cell biology techniques; electrophysiological recordings; brain mapping; and mouse models, emphasizing the clinical and possible therapeutic implications. Many chapters of this book contain information that will be relevant not only

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17

Menon, Vinod. Arithmetic in the Child and Adult Brain. Edited by Roi Cohen Kadosh and Ann Dowker. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199642342.013.041.

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This review examines brain and cognitive processes involved in arithmetic. I take a distinctly developmental perspective because neither the cognitive nor the brain processes involved in arithmetic can be adequately understood outside the framework of how developmental processes unfold. I review four basic neurocognitive processes involved in arithmetic, highlighting (1) the role of core dorsal parietal and ventral temporal-occipital cortex systems that form basic building blocks from which number form and quantity representations are constructed in the brain; (2) procedural and working memory

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18

Vallar, Giuseppe, and Nadia Bolognini. Unilateral Spatial Neglect. Edited by Anna C. (Kia) Nobre and Sabine Kastner. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199675111.013.012.

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Left unilateral spatial neglect is the most frequent and disabling neuropsychological syndrome caused by lesions to the right hemisphere. Over 50% of right-brain-damaged patients show neglect, while right neglect after left-hemispheric damage is less frequent. Neglect patients are unable to orient towards the side contralateral to the lesion, to detect and report sensory events in that portion of space, as well as to explore it by motor action. Neglect is a multicomponent disorder, which may involve the contralesional side of the body or of extra-personal physical or imagined space, different

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