Relevant bibliographies by topics / Basal ganglia-thalamo-cortical circuit

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Academic literature on the topic 'Basal ganglia-thalamo-cortical circuit'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Basal ganglia-thalamo-cortical circuit"

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Dirkx, Michiel F., Heidemarie Zach, Annelies J. van Nuland, Bastiaan R. Bloem, Ivan Toni, and Rick C. Helmich. "Cognitive load amplifies Parkinson’s tremor through excitatory network influences onto the thalamus." Brain 143, no. 5 (2020): 1498–511. http://dx.doi.org/10.1093/brain/awaa083.

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Abstract Parkinson’s tremor is related to cerebral activity in both the basal ganglia and a cerebello-thalamo-cortical circuit. It is a common clinical observation that tremor markedly increases during cognitive load (such as mental arithmetic), leading to serious disability. Previous research has shown that this tremor amplification is associated with reduced efficacy of dopaminergic treatment. Understanding the mechanisms of tremor amplification and its relation to catecholamines might help to better control this symptom with a targeted therapy. We reasoned that, during cognitive load, tremo
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Pasquini, M., G. Fabbrini, G. Moretti, et al. "Bradykinesia and Mental Slowness in Patients with Obsessive-compulsive Disorder." European Psychiatry 24, S1 (2009): 1. http://dx.doi.org/10.1016/s0924-9338(09)71268-4.

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Background:Clinical and experimental findings suggest that Obsessive-Compulsive Disorder (OCD) is due to an abnormality of the cortico-striato-thalamo-cortical circuit. Bradykinesia and mental slowness can be present in patients with basal ganglia disorders affecting the cortico-striato-thalamo-cortical circuit. Aim of this study is to investigate whether bradykinesia and mental slowness are present in patients with OCD.Methods:Participants comprised 19 non-depressed anti-psychotic free patients with OCD.Bradykinesia was assessed with the motor section of the Unified Parkinson's Disease Rating
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Spraker, Matthew B., Hong Yu, Daniel M. Corcos, and David E. Vaillancourt. "Role of Individual Basal Ganglia Nuclei in Force Amplitude Generation." Journal of Neurophysiology 98, no. 2 (2007): 821–34. http://dx.doi.org/10.1152/jn.00239.2007.

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The basal ganglia-thalamo-cortical loop is an important neural circuit that regulates motor control. A key parameter that the nervous system regulates is the level of force to exert against an object during tasks such as grasping. Previous studies indicate that the basal ganglia do not exhibit increased activity with increasing amplitude of force, although these conclusions are based mainly on the putamen. The present study used functional magnetic resonance imaging to investigate which regions in the basal ganglia, thalamus, and motor cortex display increased activity when producing pinch-gri
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Igarashi, Jun, Osamu Shouno, Jan Moren, Junichiro Yoshimoto, and Kenji Doya. "A Spiking Neural Network Model of the Basal Ganglia-thalamo-cortical Circuit toward Understanding of Motor Symptoms of Parkinson Disease." Brain & Neural Networks 22, no. 3 (2015): 103–11. http://dx.doi.org/10.3902/jnns.22.103.

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Civier, Oren, Daniel Bullock, Ludo Max, and Frank H. Guenther. "Computational modeling of stuttering caused by impairments in a basal ganglia thalamo-cortical circuit involved in syllable selection and initiation." Brain and Language 126, no. 3 (2013): 263–78. http://dx.doi.org/10.1016/j.bandl.2013.05.016.

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Singer, Harvey S., and Farhan Augustine. "The Pathophysiology of Tics; An Evolving Story." Current Drug Therapy 15, no. 2 (2020): 92–123. http://dx.doi.org/10.2174/1574885514666191121143930.

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Background: Tics, defined as quick, rapid, sudden, recurrent, non-rhythmic motor movements or vocalizations are required components of Tourette Syndrome (TS) - a complex disorder characterized by the presence of fluctuating, chronic motor and vocal tics, and the presence of co-existing neuropsychological problems. Despite many advances, the underlying pathophysiology of tics/TS remains unknown. Objective: To address a variety of controversies surrounding the pathophysiology of TS. More specifically: 1) the configuration of circuits likely involved; 2) the role of inhibitory influences on motor
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Merchant, Hugo, Jessica Grahn, Laurel Trainor, Martin Rohrmeier, and W. Tecumseh Fitch. "Finding the beat: a neural perspective across humans and non-human primates." Philosophical Transactions of the Royal Society B: Biological Sciences 370, no. 1664 (2015): 20140093. http://dx.doi.org/10.1098/rstb.2014.0093.

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Humans possess an ability to perceive and synchronize movements to the beat in music (‘beat perception and synchronization’), and recent neuroscientific data have offered new insights into this beat-finding capacity at multiple neural levels. Here, we review and compare behavioural and neural data on temporal and sequential processing during beat perception and entrainment tasks in macaques (including direct neural recording and local field potential (LFP)) and humans (including fMRI, EEG and MEG). These abilities rest upon a distributed set of circuits that include the motor cortico-basal-gan
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Jiang, S., C. Luo, J. Gong, et al. "Aberrant Thalamocortical Connectivity in Juvenile Myoclonic Epilepsy." International Journal of Neural Systems 28, no. 01 (2017): 1750034. http://dx.doi.org/10.1142/s0129065717500344.

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The purpose of this study was to investigate the functional connectivity (FC) of thalamic subdivisions in patients with juvenile myoclonic epilepsy (JME). Resting state functional magnetic resonance imaging (fMRI) and diffusion tensor imaging (DTI) data were acquired from 22 JME and 25 healthy controls. We first divided the thalamus into eight subdivisions by performing independent component analysis on tracking fibers and clustering thalamus-related FC maps. We then analyzed abnormal FC in each subdivision in JME compared with healthy controls, and we investigated their associations with clin
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Bokor, Gyula, and Peter D. Anderson. "Obsessive–Compulsive Disorder." Journal of Pharmacy Practice 27, no. 2 (2014): 116–30. http://dx.doi.org/10.1177/0897190014521996.

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Obsessive–compulsive disorder (OCD) is a common heterogeneous psychiatric disorder manifesting with obsessions and compulsions. Obsessions are intrusive, recurrent, and persistent unwanted thoughts. Compulsions are repetitive behaviors or mental acts that an individual feels driven to perform in response to the obsessions. The heterogeneity of OCD includes themes of obsessions, types of rituals, presence or absence of tics, etiology, genetics, and response to pharmacotherapy. Complications of OCD include interpersonal difficulties, unemployment, substance abuse, criminal justice issues, and ph
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Ullsperger, Markus, and D. Yves von Cramon. "The Role of Intact Frontostriatal Circuits in Error Processing." Journal of Cognitive Neuroscience 18, no. 4 (2006): 651–64. http://dx.doi.org/10.1162/jocn.2006.18.4.651.

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The basal ganglia have been suggested to play a key role in performance monitoring and resulting behavioral adjustments. It is assumed that the integration of prefrontal and motor cortico—striato—thalamo—cortical circuits provides contextual information to the motor anterior cingulate cortex regions to enable their function in performance monitoring. So far, direct evidence is missing, however. We addressed the involvement of frontostriatal circuits in performance monitoring by collecting event-related brain potentials (ERPs) and behavioral data in nine patients with focal basal ganglia lesion
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Dissertations / Theses on the topic "Basal ganglia-thalamo-cortical circuit"

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Reinel, Claudia. "Multidisziplinäre Untersuchung dopaminerger Mechanismen der repetitiven Störungen anhand von zwei Rattenmodellen dopaminerger Dysregulation." Doctoral thesis, Humboldt-Universität zu Berlin, Lebenswissenschaftliche Fakultät, 2015. http://dx.doi.org/10.18452/17390.

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Repetitive Störungen manifestieren sich als Leitsymptom in der Zwangsstörung und dem Tourette-Syndrom. Die Symptome werden als enthemmte Stereotypien eines desinhibierten Basalganglien-thalamo-kortikalen (BGTC) Regelkreises verstanden. Überdies wird als neurochemisches Korrelat ein dysregulatives Dopamin (DA)-System innerhalb dieser Kerngebiete nahegelegt, welches über ein überaktives Dopamintransporter (DAT)-System erklärt werden könnte. In der Induktion repetitiver Erkrankungen ist die Interaktion des BGTC Regelkreises und des DA-Systems dennoch unklar. In der vorliegenden Arbeit wurden da
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Book chapters on the topic "Basal ganglia-thalamo-cortical circuit"

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Benarroch, Eduardo E. "Thalamus and Thalamocortical Interactions." In Neuroscience for Clinicians, edited by Eduardo E. Benarroch. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190948894.003.0026.

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The thalamus is critical for the routing of information and coordinating the forebrain activity responsible for arousal, attention, sensory processing, motor control, cognition, and behavior. It contains first-order relay nuclei that selectively project to modality-specific primary cortical sensory areas, high-order relay nuclei that participate in cortico-thalamo-cortical interactions for attention and high-level cognitive processing, motor nuclei that mediate influences of the cerebellum and basal ganglia, and intralaminar and midline nuclei that participate global cortical activation and control the function of the striatum. All these nuclei provide excitatory inputs to the cortex via thalamocortical neurons. Thalamocortical activity is controlled by GABAergic neurons of the reticular nucleus of the thalamus, which regulate the firing pattern of thalamocortical neurons during the sleep-wake cycle and in the setting of selective attention. Extrathalamic GABAergic, cholinergic, and monoaminergic inputs also strongly regulate the thalamic circuits in a behavioral state-dependent manner. Disruption in thalamocortical circuits is a major mechanism in disorders of impaired awareness such as absence and temporal lobe seizures and in the pathophysiology of cognitive disorders.
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