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Relevant bibliographies by topics / Base excision

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Academic literature on the topic 'Base excision'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Base excision"

1

Krokan, H. E., and M. Bjoras. "Base Excision Repair." Cold Spring Harbor Perspectives in Biology 5, no. 4 (2013): a012583. http://dx.doi.org/10.1101/cshperspect.a012583.

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Intano, Gabriel W., C. Alex McMahan, John R. McCarrey, et al. "Base Excision Repair Is Limited by Different Proteins in Male Germ Cell Nuclear Extracts Prepared from Young and Old Mice." Molecular and Cellular Biology 22, no. 7 (2002): 2410–18. http://dx.doi.org/10.1128/mcb.22.7.2410-2418.2002.

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ABSTRACT The combined observations of elevated DNA repair gene expression, high uracil-DNA glycosylase-initiated base excision repair, and a low spontaneous mutant frequency for a lacI transgene in spermatogenic cells from young mice suggest that base excision repair activity is high in spermatogenic cell types. Notably, the spontaneous mutant frequency of the lacI transgene is greater in spermatogenic cells obtained from old mice, suggesting that germ line DNA repair activity may decline with age. A paternal age effect in spermatogenic cells is recognized for the human population as well. To

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Hawkins, Gregory A., and Les M. Hoffman. "Base excision sequence scanning." Nature Biotechnology 15, no. 8 (1997): 803–4. http://dx.doi.org/10.1038/nbt0897-803.

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Zharkov, D. O. "Base excision DNA repair." Cellular and Molecular Life Sciences 65, no. 10 (2008): 1544–65. http://dx.doi.org/10.1007/s00018-008-7543-2.

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Dianov, Grigory. "Base excision DNA repair." European Journal of Cancer 29 (January 1993): S32. http://dx.doi.org/10.1016/0959-8049(93)90770-g.

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Sidhu, Ashwin. "Base Excision Repair: A Review." Journal of BioMed Research and Reports 2, no. 4 (2023): 1–3. http://dx.doi.org/10.59657/2837-4681.brs.23.032.

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Base excision repair (BER) is a fundamental DNA repair pathway essential for maintaining the genomic integrity of all living organisms. This paper provides an overview of the mechanisms, key players, and regulatory aspects of the base excision repair pathway. BER serves as the primary defense against spontaneous DNA damage arising from endogenous and exogenous sources, including oxidative stress, deamination, and alkylation. This repair process involves a sequential series of enzymatic reactions orchestrated by various proteins, including DNA glycosylases, AP endonucleases, DNA polymerases, an

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Kurthkoti, Krishna, and Umesh Varshney. "Base excision and nucleotide excision repair pathways in mycobacteria." Tuberculosis 91, no. 6 (2011): 533–43. http://dx.doi.org/10.1016/j.tube.2011.06.005.

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Agnez-Lima, Lucymara F., Sílvia R. Batistuzzo de Medeiros, Bruno S. Maggi, and Giovanna A. S. Quaresma. "Base excision repair in sugarcane." Genetics and Molecular Biology 24, no. 1-4 (2001): 123–29. http://dx.doi.org/10.1590/s1415-47572001000100017.

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DNA damage can be induced by a large number of physical and chemical agents from the environment as well as compounds produced by cellular metabolism. This type of damage can interfere with cellular processes such as replication and transcription, resulting in cell death and/or mutations. The low frequency of mutagenesis in cells is due to the presence of enzymatic pathways which repair damaged DNA. Several DNA repair genes (mainly from bacteria, yeasts and mammals) have been cloned and their products characterized. The high conservation, especially in eukaryotes, of the majority of genes rela

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Maynard, Scott, Nadja C. de Souza-Pinto, Morten Scheibye-Knudsen, and Vilhelm A. Bohr. "Mitochondrial base excision repair assays." Methods 51, no. 4 (2010): 416–25. http://dx.doi.org/10.1016/j.ymeth.2010.02.020.

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Seeberg, Erling, Lars Eide, and Magnar Bjørås. "The base excision repair pathway." Trends in Biochemical Sciences 20, no. 10 (1995): 391–97. http://dx.doi.org/10.1016/s0968-0004(00)89086-6.

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Dissertations / Theses on the topic "Base excision"

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Prasad, Amalthiya. "Base Excision Repair in Chromatin." ScholarWorks @ UVM, 2008. http://library.uvm.edu/dspace/bitstream/123456789/180/1/amalthiyprasadfinal.pdf.

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Tait, Phillip Stuart. "Regulation of base excision repair." Thesis, University of Oxford, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.504608.

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Sleeth, Kate Michelle. "DNA ligases in base excision repair." Thesis, University of Reading, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.428317.

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Fletcher, Sally C. "Regulation of the base excision repair pathway." Thesis, University of Oxford, 2017. http://ora.ox.ac.uk/objects/uuid:e3db2385-f9aa-4289-9547-3e37cbeddec9.

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Maintenance of genomic stability is paramount for survival of an organism; failure to repair DNA damage ultimately leads to the accumulation of genetically unstable cells and the onset of different human diseases including cancer. DNA single strand breaks and base oxidation/alkylation are among the most frequent types of DNA damage occurring spontaneously in cells. Base excision repair (BER), which copes with the majority of these lesions, is therefore a fundamental DNA repair system. Accordingly, it is important to understand how BER is regulated, and particularly, how and if BER is affected

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Odell, Ian. "Modulation of Base Excision Repair by Nucleosomes." ScholarWorks @ UVM, 2010. http://scholarworks.uvm.edu/graddis/170.

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DNA in eukaryotes is packaged into nucleosomes, which present steric impediments to many of the factors and enzymes that act on DNA, including DNA repair enzymes. Within the nucleosome, DNA remains vulnerable to oxidative damage that can result from normal cellular metabolism, ionizing radiation, and various chemical agents. Oxidatively damaged DNA is repaired in a stepwise fashion via the base excision repair (BER) pathway. Other DNA repair pathways, including Nucleotide Excision Repair (NER), Mismatch Repair (MMR), Homologous Recombination (HR), and Non-homologous End-Joining (NHEJ) are all

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Sokhansanj, Bahrad Ali. "Mathematical models of human DNA base excision repair /." For electronic version search Digital dissertations database. Restricted to UC campuses. Access is free to UC campus dissertations, 2002. http://uclibs.org/PID/11984.

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Jiang, Zhongliang. "Epigenetic Instability Induced by DNA Base Lesion via DNA Base Excision Repair." FIU Digital Commons, 2017. https://digitalcommons.fiu.edu/etd/3566.

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DNA damage can cause genome instability, which may lead to human cancer. The most common form of DNA damage is DNA base damage, which is efficiently repaired by DNA base excision repair (BER). Thus BER is the major DNA repair pathway that maintains the stability of the genome. On the other hand, BER mediates DNA demethylation that can occur on the promoter region of important tumor suppressor genes such as Breast Cancer 1 (BRCA1) gene that is also involved in prevention and development of cancer. In this study, employing cell-based and in vitro biochemical approaches along with bisulfite DNA s

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Akbari, Mansour. "Human Base Excision Repair for Preservation of Genomic Stability." Doctoral thesis, Norwegian University of Science and Technology, Department of Cancer Research and Molecular Medicine, 2006. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-1807.

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Scott, A. D. "The excision repair of base damage in Saccharomyces cerevisiae." Thesis, Swansea University, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.638781.

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In <I>Saccharomyces cerevisiae RAD7 </I>and <I>RAD16</I> are required for nucleotide excision repair (NER) of CPDs in nontranscribed regions of the genome. An inducible component to NER in yeast is examined in this thesis. Excision of CPDs and a minor UV induced lesion characterised by sensitivity to endonuclease III (EIIISS), is enhanced following a prior UV irradiation. No enhancement of repair is detected in either the <I>rad7Δ</I> or the <I>rad16Δ</I> mutant. Since repair of EIIISS in transcriptionally silent DNA is independent of <I>RAD7 </I>and <I>RAD16</I>, these two genes may have two

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Thomson, Hellen Frances. "Biochemical characterisation of base excision repair enzymes in Neisseria meningitidis." Thesis, Imperial College London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.435770.

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Books on the topic "Base excision"

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Bhakat, Kishor K., and Tapas K. Hazra, eds. Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1.

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Bhakat, Kishor K., and Tapas K. Hazra, eds. Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1.

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1957-, Hickson Ian D., ed. Base excision repair of DNA damage. Landes Bioscience, 1997.

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Fang, Fang. Significance of DNA base excision repair in the maintenance of mitochondrial function in Saccharomyces cerevisiae. Brock University, Centre for Biotechnology, 2008.

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Moldave, Kivie, Samuel H. Wilson, Sankar Mitra, Amanda K. McCullough, and R. Steven Lloyd. Base Excision Repair. Elsevier Science & Technology Books, 2001.

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Wilson III, David M. The Base Excision Repair Pathway. WORLD SCIENTIFIC, 2015. http://dx.doi.org/10.1142/9776.

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Bhakat, Kishor K., and Tapas K. Hazra. Base Excision Repair Pathway: Methods and Protocols. Springer, 2023.

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Wilson, David M. Base Excision Repair Pathway: Molecular Mechanisms and Role in Disease Development and Therapeutic Strategies. World Scientific Publishing Co Pte Ltd, 2016.

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(Editor), Sankar Mitra, Amanda K. McCullough (Editor), R. Steven Lloyd (Editor), Samuel H. Wilson (Editor), and Kivie Moldave (Series Editor), eds. Base Excision Repair (Progress in Nucleic Acid Research and Molecular Biology, Volume 68) (Progress in Nucleic Acid Research and Molecular Biology). Academic Press, 2001.

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(Editor), Sankar Mitra, Amanda K. McCullough (Editor), R. Steven Lloyd (Editor), Samuel H. Wilson (Editor), and Kivie Moldave (Series Editor), eds. Base Excision Repair (Progress in Nucleic Acid Research and Molecular Biology, Volume 68) (Progress in Nucleic Acid Research and Molecular Biology). Academic Press, 2001.

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Book chapters on the topic "Base excision"

1

Sayed, Ibrahim M., Anirban Chakraborty, and Soumita Das. "Assays with Patient-Derived Organoids to Evaluate the Impact of Microbial Infection on Base Excision Repair (BER) Enzymes." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_10.

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Izumi, Tadahide. "Analysis of Copy Number Variation of DNA Repair/Damage Response Genes in Tumor Tissues." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_15.

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Tarpley, Mason, Yingling Chen, and Kishor K. Bhakat. "Genome-Wide Binding Analysis of DNA Repair Protein APE1 in Tumor Cells by ChIP-Seq." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_16.

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Ryan, Benjamin J., Tyler M. Weaver, Jonah J. Spencer, and Bret D. Freudenthal. "Generation of Recombinant Nucleosomes Containing Site-Specific DNA Damage." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_4.

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Law, Henry C. H., Dragana Noe, and Nicholas T. Woods. "Interactome Profiling of DNA Damage Response (DDR) Mediators with Immunoprecipitation-Mass Spectrometry." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_12.

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Perry, Megan, and Gargi Ghosal. "Isolation and Immunodetection of Enzymatic DNA–Protein Crosslinks by RADAR Assay." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_8.

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Kaja, Amala, Priyanka Barman, Shalini Guha, and Sukesh R. Bhaumik. "Tandem Affinity Purification and Mass-Spectrometric Analysis of FACT and Associated Proteins." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_14.

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McDonald, Drew T., Pam S. Wang, Jennifer Moitoza Johnson, and Miaw-Sheue Tsai. "Using Affinity Pulldown Assays to Study Protein–Protein Interactions of Human NEIL1 Glycosylase and the Checkpoint Protein RAD9–RAD1–HUS1 (9-1-1) Complex." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_13.

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Jaiswal, Aruna S., Elizabeth A. Williamson, Arunima S. Jaiswal, Kimi Kong, and Robert A. Hromas. "In Vitro Reconstitutive Base Excision Repair (BER) Assay." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_6.

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Malfatti, Matilde Clarissa, Giulia Antoniali, and Gianluca Tell. "In Vitro Assay to Measure APE1 Enzymatic Activity on Ribose Monophosphate Abasic Site." In Base Excision Repair Pathway. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3373-1_2.

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Conference papers on the topic "Base excision"

1

Si, Jiangbo, Zeyu Wang, Hang Hu, and Naofal Al-Dhahir. "Nonstationary Jammer Excision Based on Sparse Reconstruction and Support Vector Regression." In 2024 International Conference on Ubiquitous Communication (Ucom). IEEE, 2024. http://dx.doi.org/10.1109/ucom62433.2024.10695873.

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Qazlan, Salman Al, Muath Alfallaj, Mody Almarshad, and Abdullah Alobaid. "The Role of Intraoperative MRI in Excision of Clival Chordoma." In 30th Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1702658.

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Krishnaprabhu, Raju, Ari G. Chacko, Wilson P. Desouza, et al. "Simultaneous Endoscopic Endonasal and Transcranial Excision of Giant Pituitary Adenomas." In 32nd Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2023. http://dx.doi.org/10.1055/s-0043-1762035.

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Shaftel, Kelly, John Craig, Amrita Ray, Jacob Eide, and Karam Asmaro. "Endoscopic Endonasal Transtuberculum Approach for Excision of Heavily Calcified Tuberoinfundibular Craniopharyngioma." In 33rd Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2024. http://dx.doi.org/10.1055/s-0044-1780418.

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Imam, J. S., J. Seashore, N. Tapryal, T. Hazra, and V. J. Cardenas. "DNA Base Excision Repair and Inflammation in COPD." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4736.

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Sweasy, Joann B. "Abstract IA-005: Base excision repair and cancer." In Abstracts: AACR Virtual Special Conference on Radiation Science and Medicine; March 2-3, 2021. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1557-3265.radsci21-ia-005.

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"Base excision repair in non-canonical DNA structures." In Bioinformatics of Genome Regulation and Structure/Systems Biology (BGRS/SB-2022) :. Institute of Cytology and Genetics, the Siberian Branch of the Russian Academy of Sciences, 2022. http://dx.doi.org/10.18699/sbb-2022-575.

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Sokhansanj, B. A. "DNA Base Excision Repair Nanosystem Engineering: Model Development." In 2005 IEEE Engineering in Medicine and Biology 27th Annual Conference. IEEE, 2005. http://dx.doi.org/10.1109/iembs.2005.1616268.

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"Order and disorder in base excision DNA repair." In Биоинформатика регуляции и структуры геномов / системная биология. ИЦиГ СО РАН, 2024. http://dx.doi.org/10.18699/bgrs2024-11.1-17.

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Goshtasbi, Khodayar, Arash Abiri, Mehdi Abouzari, Harrison W. Lin, Hamid R. Djalilian, and Edward C. Kuan. "Thirty-Day Complications following Intradural Versus Extradural Excision of the Skull Base." In 30th Annual Meeting North American Skull Base Society. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1702490.

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Reports on the topic "Base excision"

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Straatsma, TP, J. A. McCammon, John H. Miller, et al. Biomolecular Simulation of Base Excision Repair and Protein Signaling. Office of Scientific and Technical Information (OSTI), 2006. http://dx.doi.org/10.2172/877558.

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Boorstein, Robert. Loss of DNA Base Excision Repair Capacity in Initiation and Progression of Breast Cancer. Defense Technical Information Center, 1997. http://dx.doi.org/10.21236/ada329529.

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Yeung, Anthony T. Base Excision Repair Gene Mutations and Polymorphisms as a Potential Modifier of Breast Cancer Risk. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada398986.

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Krosky, Daniel J. Rational Inhibitors of DNA Base Excision Repair Enzymes: New Tools for Elucidating the Role of BER in Cancer Chemotherapy. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada456909.

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Krosky, Daniel J., and James T. Stivers. Rational Inhibitors of DNA Base Excision (BER) Enzymes: New Tools for Elucidating the Role of BER in Cancer Chemotherapy. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada424155.

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Krosky, Daniel J. Rational Inhibitors of DNA Base Excision Repair Enzymes: New Tools for Elucidating the Role of BER in Cancer Chemotherapy. Addendum. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada502370.

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Krosky, Daniel J. Rational Inhibitors of DNA Base Excision Repair (BER) Enzymes: New Tools for Elucidating the Role of the BER in Cancer Chemotherapy. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada437740.

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Hase, Travis. In Vivo Quantification of Reactive Oxygen Species Demonstrates High Levels of Oxidative Stress in Base Excision Repair-Deficient Caenorhabditis Elegans: Implications for Associative Metabolic Phenotypes. Portland State University Library, 2013. http://dx.doi.org/10.15760/honors.10.

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Rinne, Mikael L., and Mark R. Kelley. DNA Base Excision Repair (BER) and Cancer Gene Therapy: Use of the Human N-Methylpurine DNA Glycosylase (MPG) to Sensitive Breast Cancer Cells to Low Dose Chemotherapy. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada407383.

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Norelli, John L., Moshe Flaishman, Herb Aldwinckle, and David Gidoni. Regulated expression of site-specific DNA recombination for precision genetic engineering of apple. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7587214.bard.

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Objectives: The original objectives of this project were to: 1) evaluate inducible promoters for the expression of recombinase in apple (USDA-ARS); 2) develop alternative selectable markers for use in apple to facilitate the positive selection of gene excision by recombinase (Cornell University); 3) compare the activity of three different recombinase systems (Cre/lox, FLP/FRT, and R/RS)in apple using a rapid transient assay (ARO); and 4) evaluate the use of recombinase systems in apple using the best promoters, selectable markers and recombinase systems identified in 1, 2 and 3 above (Collabor

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