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Journal articles on the topic 'Basophilia'

Author: Grafiati

Published: 5 September 2021

Last updated: 2 February 2022

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1

Bodger, MP, GL Mounsey, J. Nelson, and PH Fitzgerald. "A monoclonal antibody reacting with human basophils." Blood 69, no. 5 (1987): 1414–18. http://dx.doi.org/10.1182/blood.v69.5.1414.1414.

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Abstract Bsp-1 is an IgM murine monoclonal antibody raised against the human erythroblastic leukemia cell line (HEL) that reacts with basophils but not neutrophils or eosinophils. Western blotting techniques showed that Bsp-1 reacts with a 45-kilodalton surface antigen on HEL cells. The distribution of Bsp-1 antigen on leukemic cells is confined to a basophilic leukemia cell line, KU812, chronic myeloid leukemia with basophilia, and some cases of acute undifferentiated leukemia. Bsp-1 might therefore be a useful reagent for the study of basophil function and differentiation.

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2

Bodger, MP, GL Mounsey, J. Nelson, and PH Fitzgerald. "A monoclonal antibody reacting with human basophils." Blood 69, no. 5 (1987): 1414–18. http://dx.doi.org/10.1182/blood.v69.5.1414.bloodjournal6951414.

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Bsp-1 is an IgM murine monoclonal antibody raised against the human erythroblastic leukemia cell line (HEL) that reacts with basophils but not neutrophils or eosinophils. Western blotting techniques showed that Bsp-1 reacts with a 45-kilodalton surface antigen on HEL cells. The distribution of Bsp-1 antigen on leukemic cells is confined to a basophilic leukemia cell line, KU812, chronic myeloid leukemia with basophilia, and some cases of acute undifferentiated leukemia. Bsp-1 might therefore be a useful reagent for the study of basophil function and differentiation.

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3

Tanaka, Yasuhiro, Atsushi Tanaka, Akiko Hashimoto, Kumiko Hayashi, and Isaku Shinzato. "Acute Myeloid Leukemia with Basophilic Differentiation Transformed from Myelodysplastic Syndrome." Case Reports in Hematology 2017 (2017): 1–6. http://dx.doi.org/10.1155/2017/4695491.

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Myelodysplastic syndrome (MDS) terminally transforms to acute myeloid leukemia (AML) or bone marrow failure syndrome, but acute myeloid leukemia with basophilic differentiation has been rarely reported. An 81-year-old man was referred to our department for further examination of intermittent fever and normocytic anemia during immunosuppressive treatment. Chromosomal analysis showed additional abnormalities involving chromosome 7. He was diagnosed as having MDS. At the time of diagnosis, basophils had not proliferated in the bone marrow. However, his anemia and thrombocytopenia rapidly worsened

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4

Antohe, Ion, Angela Dăscălescu, Cătălin Dănăilă, et al. "FLT-3 ITD Positive Acute Basophilic Leukemia with Rare Complex Karyotype Presenting with Acute Respiratory Failure: Case Report." Revista Romana de Medicina de Laborator 26, no. 1 (2018): 87–94. http://dx.doi.org/10.1515/rrlm-2017-0036.

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Abstract Background: Acute basophilic leukemia is a rare subtype of acute myeloid leukemia, as categorized by the 2008 World Health Organization classification of myeloid neoplasms. Acute basophilic leukemia diagnosis requires thorough morphological, cytochemical, immunophenotypic, molecular, and cytogenetic studies and exclusion of other hematological neoplasms associating basophilia. The disease course is defined by histamine driven, occasionally life-threatening respiratory, cardiovascular, cutaneous or digestive complications, as well as primary refractoriness to standard therapy. Clinical

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5

Ohnmacht, Caspar, and David Voehringer. "Basophil effector function and homeostasis during helminth infection." Blood 113, no. 12 (2009): 2816–25. http://dx.doi.org/10.1182/blood-2008-05-154773.

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AbstractBasophils are effector cells of the innate immune system that are associated with allergic inflammation and infections with helminth parasites. However, their development and in vivo functions are largely unknown. Here, we characterize basophil development, turnover, tissue localization, and effector function during infection with the helminth Nippostrongylus brasiliensis. Our results demonstrate that under homeostatic conditions basophils have a lifespan of about 60 hours. N brasiliensis–induced basophilia is caused by increased de novo production of basophils in the bone marrow. Baso

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6

Mukai, Kaori, Maya J. BenBarak, Masashi Tachibana, et al. "Critical role of P1-Runx1 in mouse basophil development." Blood 120, no. 1 (2012): 76–85. http://dx.doi.org/10.1182/blood-2011-12-399113.

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Abstract Runx1 P1N/P1N mice are deficient in the transcription factor distal promoter-derived Runt-related transcription factor 1 (P1-Runx1) and have a > 90% reduction in the numbers of basophils in the BM, spleen, and blood. In contrast, Runx1P1N/P1N mice have normal numbers of the other granulocytes (neutrophils and eosinophils). Although basophils and mast cells share some common features, Runx1P1N/P1N mice have normal numbers of mast cells in multiple tissues. Runx1P1N/P1N mice fail to develop a basophil-dependent reaction, IgE-mediated chronic allergic inflammation of the skin, but res

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7

Agis, Hermine, Maria T. Krauth, Leonhard Muellauer, Lawrence B. Schwartz, Hans P. Horny, and Peter Valent. "Enumeration and Immunologic Characterization of Basophils in Normal Bone Marrow and Patients with Myeloproliferative Disorders." Blood 104, no. 11 (2004): 4754. http://dx.doi.org/10.1182/blood.v104.11.4754.4754.

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Abstract Basophils are highly specialized granulocytes that express a unique profile of antigens and increase in myeloproliferative disorders. In chronic myeloid leukemia (CML), basophilia is an independent prognostic variable. So far, however, no reliable immunohistochemical approach for routine-detection and enumeration of bone marrow (bm) basophils has become available. To overcome this disadvantage, we have applied the anti-basophil antibody 2D7 on formalin-fixed, paraffin-embedded sections of normal bm and bm from patients (pts) with chronic myeloid leukemia (CML; chronic phase, n=21; acc

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8

Kane, John P. "Infectious basophilia?" American Journal of Hematology 91, no. 2 (2016): E8. http://dx.doi.org/10.1002/ajh.24243.

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9

Boiten, Henk-Jan, and Eva de Jongh. "Atypical basophilia." Blood 132, no. 5 (2018): 551. http://dx.doi.org/10.1182/blood-2018-05-849901.

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10

Balan, Maria, Aimee Hope, Joseph Cassidy, Maureen McCullough, and Peter J. O’Brien. "Marked paraneoplastic basophilia accompanying eosinophilia in a cat with alimentary T-cell lymphoma." Journal of Feline Medicine and Surgery Open Reports 3, no. 2 (2017): 205511691773018. http://dx.doi.org/10.1177/2055116917730180.

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Case summary A 5-year-old male neutered domestic shorthair cat was referred with a history of persistent pyrexia, pica, soft faeces, inappetence, intermittent vomiting, mild-to-moderate granulocytosis and mild hypercalcaemia. No significant improvement was noted after antibiotic and corticosteroid treatment, except that the hypercalcaemia resolved. Physical examination, including thoracic auscultation, and abdominal and peripheral lymph node palpation, were unremarkable. On admission, haematology revealed moderate leukocytosis (36.8 × 109/l) with moderate-to-marked eosinophilia (21.3 × 109/l)

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11

Kantarjian, HM, TL Smith, KB McCredie, et al. "Chronic myelogenous leukemia: a multivariate analysis of the associations of patient characteristics and therapy with survival." Blood 66, no. 6 (1985): 1326–35. http://dx.doi.org/10.1182/blood.v66.6.1326.1326.

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Abstract The prognostic importance of patient pretreatment clinical and laboratory features was investigated in a group of 303 patients with Philadelphia chromosome-positive benign-phase chronic myelogenous leukemia. Intensive chemotherapy was given to 97 patients, and 78 underwent an early elective splenectomy. The overall median survival time, dated from hospital admission, was 39 months. Patient characteristics associated with shortened survival were age 60 years or older, black race, the presence of hepatomegaly, splenomegaly, symptoms, weight loss, and poor performance status. Adverse blo

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12

Kantarjian, HM, TL Smith, KB McCredie, et al. "Chronic myelogenous leukemia: a multivariate analysis of the associations of patient characteristics and therapy with survival." Blood 66, no. 6 (1985): 1326–35. http://dx.doi.org/10.1182/blood.v66.6.1326.bloodjournal6661326.

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The prognostic importance of patient pretreatment clinical and laboratory features was investigated in a group of 303 patients with Philadelphia chromosome-positive benign-phase chronic myelogenous leukemia. Intensive chemotherapy was given to 97 patients, and 78 underwent an early elective splenectomy. The overall median survival time, dated from hospital admission, was 39 months. Patient characteristics associated with shortened survival were age 60 years or older, black race, the presence of hepatomegaly, splenomegaly, symptoms, weight loss, and poor performance status. Adverse blood and bo

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13

Matsushima, Takafumi, Hiroshi Handa, Akihiko Yokohama, et al. "Prevalence and clinical characteristics of myelodysplastic syndrome with bone marrow eosinophilia or basophilia." Blood 101, no. 9 (2003): 3386–90. http://dx.doi.org/10.1182/blood-2002-03-0947.

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By retrospectively analyzing 288 patients with de novo myelodysplastic syndrome (MDS), we sought to determine the prevalence and clinical characteristics of bone marrow eosinophilia and basophilia that were detected at presentation. Bone marrow eosinophilia and basophilia were defined as a differential count of each cell type exceeding 5.0% and 1.0%, respectively. Of 288 patients with MDS, 36 (12.5%) fulfilled this criterion for bone marrow eosinophilia (MDS-Eos); 34 patients (11.8%) showed basophilia (MDS-Bas), and 11 (3.8%) satisfied both criteria (MDS-EosBas). The remaining 229 patients had

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14

Yamagata, Tetsuya, Noboru Miura, Kouichi Hirai, Kiyoshi Kurokawa, and Masaaki Higashihara. "NATURAL INTERFERON-ALFA FOR BASOPHILIA." British Journal of Haematology 82, no. 4 (1992): 780–81. http://dx.doi.org/10.1111/j.1365-2141.1992.tb06965.x.

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15

Suda, J., M. Eguchi, Y. Akiyama, et al. "Differentiation of blast cells from a Down's syndrome patient with transient myeloproliferative disorder." Blood 69, no. 2 (1987): 508–12. http://dx.doi.org/10.1182/blood.v69.2.508.508.

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Abstract A male neonate with Down's syndrome and congenital myeloproliferative disorder was studied. His blood picture showed the unique coexistence of leukocytosis with matured cells and a large number of blast cells. The in vitro proliferation and differentiation of blast cells into various lineages in the presence of phytohemagglutinin-stimulated leukocyte conditioned medium (PHA-LCM) was examined by using a liquid culture and a methylcellulose culture system. The differentiation of blast cells into myeloid cells was confirmed by specific cytochemical stainings, electron microscopy, and an

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16

Suda, J., M. Eguchi, Y. Akiyama, et al. "Differentiation of blast cells from a Down's syndrome patient with transient myeloproliferative disorder." Blood 69, no. 2 (1987): 508–12. http://dx.doi.org/10.1182/blood.v69.2.508.bloodjournal692508.

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A male neonate with Down's syndrome and congenital myeloproliferative disorder was studied. His blood picture showed the unique coexistence of leukocytosis with matured cells and a large number of blast cells. The in vitro proliferation and differentiation of blast cells into various lineages in the presence of phytohemagglutinin-stimulated leukocyte conditioned medium (PHA-LCM) was examined by using a liquid culture and a methylcellulose culture system. The differentiation of blast cells into myeloid cells was confirmed by specific cytochemical stainings, electron microscopy, and an immunolog

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17

Sperr, Wolfgang R., Thomas Pfeiffer, Michael Kundi, Christian Sillaber, Susanne Herndlhofer, and Peter Valent. "Serum Tryptase Is a Strong Predictive Biomarker That Improves Prognostication in Ph+ Chronic Myeloid Leukemia." Blood 120, no. 21 (2012): 2783. http://dx.doi.org/10.1182/blood.v120.21.2783.2783.

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Abstract Abstract 2783 Although the prognosis of patients with chronic myeloid leukemia (CML) has improved significantly with the advent and use of novel BCR/ABL tyrosine kinase inhibitors (TKI) in recent years, still a number of patients fail to achieve a durable molecular remission or they relapse. It is of particular importance to define the prognosis in individual patients as early as possible, as timely intervention with novel TKI may improve long term outcome. Basophilia is one of the most predictive and best documented biomarkers in CML at diagnosis. However, in high risk patients, the

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18

Codeluppi, Luca, Paolo Spagnolo, Manuela Tondelli, Maria Chiara Malaguti, and Jessica Mandrioli. "Recurrent cerebrospinal fluid basophilia in neurosarcoidosis." Acta Neurologica Belgica 115, no. 3 (2014): 497–99. http://dx.doi.org/10.1007/s13760-014-0406-8.

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19

Knotkova, Z., S. Mazanek, M. Hovorka, M. Sloboda, and Z. Knotek. "Haematology and plasma chemistry of Bornean river turtles suffering from shell necrosis and haemogregarine parasites ." Veterinární Medicína 50, No. 9 (2012): 421–26. http://dx.doi.org/10.17221/5643-vetmed.

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Nine Bornean river turtles (Orlitia borneensis, Gray, 1873) suffering from lethargy, ulcerations and caseous necrosis of the plastron were evaluated for haematology and plasma chemistry. Intra-erythrocytic haemogregarine parasites were associated with anaemia, low haemoglobin, basophilia, eosinophilia, heterophilia and azurophilia. After eight months of treatment consisting of antibiotics, debridement and scrubbing of lesions with enilconazole or povidone iodine, rehydration, deworming and tube feeding, lymphocytes, basophils, eosinophils, heterophils and azurophils returned to the normal rang

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20

Schulte-Hermann, R., B. Kraupp-Grasl, W. Bursch, U. Gerbracht, and I. Timmermann-Trosiener. "Effects of Non-Genotoxic Hepatocarcinogens Phenobarbital and Nafenopin on Phenotype and Growth of Different Populations of Altered Foci in Rat Liver." Toxicologic Pathology 17, no. 4_part_1 (1989): 642–50. http://dx.doi.org/10.1177/0192623389017004109.

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Non-genotoxic hepatocarcinogens share the ability to induce liver growth in rodents. Phenobarbital (PB), as one prototype compound, promotes the development of liver tumors; altered cell foci of the clear-eosinophilic phenotype, also identified by gamma-glutamyltransferase expression, appear to be precursor lesions. These foci seem to over-respond to the growth-inducing effect of PB. In contrast, the question as to whether peroxisome inducers are also tumor promoters is still unsettled. We will present evidence which strongly suggests that the peroxisome inducer, nafenopin (Naf), promotes tumo

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21

Aichberger, Karl J., Matthias Mayerhofer, Maria-Theresa Krauth, et al. "BCR/ABL Induces Expression of Histidine Decarboxylase and Synthesis of Histamine in CML Cells." Blood 106, no. 11 (2005): 4835. http://dx.doi.org/10.1182/blood.v106.11.4835.4835.

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Abstract Basophil numbers are typically elevated in patients with chronic myeloid leukemia (CML) and characteristically increase during disease progression. As a consequence, blood histamine levels are highly upregulated in CML. We examined the biochemical basis of production of histamine in CML cells and analyzed the effects of the CML-related oncoprotein BCR/ABL on the generation of this mediator. Expression of histamine and of histidine decarboxylase (HDC), the major enzyme involved in histamine synthesis, were examined in primary CML cells obtained from patients with chronic phase (CP) CML

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22

MITRE, EDWARD, and THOMAS B. NUTMAN. "LACK OF BASOPHILIA IN HUMAN PARASITIC INFECTIONS." American Journal of Tropical Medicine and Hygiene 69, no. 1 (2003): 87–91. http://dx.doi.org/10.4269/ajtmh.2003.69.87.

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23

Feriel, Joffrey, François Depasse, and Franck Geneviève. "How I investigate basophilia in daily practice." International Journal of Laboratory Hematology 42, no. 3 (2019): 237–45. http://dx.doi.org/10.1111/ijlh.13146.

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24

Ogunleye, Foluso, Mohammed Ibrahim, Emily Allen, et al. "BCR-ABL Testing by Polymerase Chain Reaction in Patients With Neutrophilia: The William Beaumont Hospital Experience and the Case for Rational Laboratory Test Requests." Journal of Oncology Practice 12, no. 12 (2016): e1001-e1005. http://dx.doi.org/10.1200/jop.2016.014449.

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Chronic myeloid leukemia (CML) is a myeloproliferative neoplasm resulting from the fusion of the BCR-ABL genes, forming the Philadelphia chromosome. The diagnosis is often suspected when there is leukocytosis with left shift and basophilia. Confirmation of the diagnosis requires a demonstration of BCR-ABL by polymerase chain reaction. Using data from the William Beaumont laboratory data registry, we conducted a retrospective review of all the orders for BCR-ABL tests sent to the clinical pathology laboratory between March 11, 2014 and September 12, 2014. We concluded that the presence of concu

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25

Tinegate, H. N., and M. N. Chetty. "Basophilia as a feature of the myelodysplastic syndrome." Clinical & Laboratory Haematology 8, no. 3 (1986): 269–71. http://dx.doi.org/10.1111/j.1365-2257.1986.tb00108.x.

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26

Ma, S. K., J. C. W. Chan, T. S. K. Wan, A. Y. Y. Chan, and L. C. Chan. "Myelodysplastic Syndrome with Myelofibrosis and Basophilia: Detection of Trisomy 8 in Basophils by Fluorescence in-situ Hybridization." Leukemia & Lymphoma 31, no. 3-4 (1998): 429–32. http://dx.doi.org/10.3109/10428199809059238.

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27

Jogdand, Prajakta, Premkumar Siddhuraj, Michiko Mori, et al. "Eosinophils, basophils and type 2 immune microenvironments in COPD-affected lung tissue." European Respiratory Journal 55, no. 5 (2020): 1900110. http://dx.doi.org/10.1183/13993003.00110-2019.

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Although elevated blood or sputum eosinophils are present in many patients with COPD, uncertainties remain regarding the anatomical distribution pattern of lung-infiltrating eosinophils. Basophils have remained virtually unexplored in COPD. This study mapped tissue-infiltrating eosinophils, basophils and eosinophil-promoting immune mechanisms in COPD-affected lungs.Surgical lung tissue and biopsies from major anatomical compartments were obtained from COPD patients with severity grades Global Initiative for Chronic Obstructive Lung Disease stages I–IV; never-smokers/smokers served as controls.

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28

Brown, Laura E., Da Zhang, Diane L. Persons, Abdulraheem Yacoub, Shivani Ponnala, and Wei Cui. "A 26-Year-Old Female with Systemic Mastocytosis with Associated Myeloid Neoplasm with Eosinophilia and Abnormalities ofPDGFRB, t(4;5)(q21;q33)." Case Reports in Hematology 2016 (2016): 1–4. http://dx.doi.org/10.1155/2016/4158567.

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Various translocations involving thePDGFRBgene are identified in myeloid neoplasms. However, thePRKG2/PDGFRBfusion gene associated with t(4;5)(q21;q33) has previously been reported in only 3 patients. We present the case of a 26-year-old woman with microcytic anemia, basophilia, thrombocytosis, and massive splenomegaly, who was found to have systemic mastocytosis and associated clonal hematological non-mast cell lineage disease (SM-AHNMD), with myeloid neoplasm withPRKG2/PDGFRBrearrangement. Initial findings included basophilia (37%, 4.1 k/μL), hypercellular marrow with eosinophilia, and incre

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29

Sompolinsky, D., T. Katzenstein, and L. Lundberg. "Circulatory basophilia in guinea pigs with delayed-type hypersensitivity." Allergy 47, no. 4 (1992): 303–8. http://dx.doi.org/10.1111/j.1398-9995.1992.tb02058.x.

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30

Servitzoglou, Marina, Maria Grenzelia, Margarita Baka, et al. "A Novel Karyotype in Acute Myeloid Leukemia with Basophilia." Pediatric Hematology and Oncology 31, no. 2 (2014): 149–56. http://dx.doi.org/10.3109/08880018.2014.883655.

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31

Jácomo, Rafael H., Viviane Furlan Lozano, Jose Gastao da Cunha Neto, and Sandra Soares Costa. "What's the Meaning of Basophilia in Sysmex XE-2100?" Archives of Pathology & Laboratory Medicine 135, no. 4 (2011): 415. http://dx.doi.org/10.5858/2010-0441-le.1.

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32

Valent, P., E. Spanblochl, HC Bankl, et al. "Kit ligand/mast cell growth factor-independent differentiation of mast cells in myelodysplasia and chronic myeloid leukemic blast crisis." Blood 84, no. 12 (1994): 4322–32. http://dx.doi.org/10.1182/blood.v84.12.4322.bloodjournal84124322.

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Autonomous, factor-independent growth and differentiation of malignant cells in preleukemic and leukemic disease states is a well-recognized phenomenon and is often associated with a poor prognosis. Mast cells are distinct hematopoietic cells and express a unique profile of antigens. Growth and differentiation of normal mast cells is dependent on mast cell growth factor (MGF), the ligand of the c-kit protooncogene product. In this study, we screened for mast cell-lineage involvement in 52 patients suffering from myeloid leukemias, myelodysplastic syndromes (MDS), systemic mastocytosis, or othe

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33

Thorn, Catherine E., and Isabelle Aubert DVM. "Abdominal Mass Aspirate from a Cat with Eosinophilia and Basophilia." Veterinary Clinical Pathology 28, no. 4 (1999): 139–41. http://dx.doi.org/10.1111/j.1939-165x.1999.tb01064.x.

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34

Amouzadeh-Ghadikolai, Omid, Gerhard Reicht, Franz Quehenberger, and Christoph Robier. "Basophilia of the peripheral blood in patients with ulcerative colitis." Scandinavian Journal of Gastroenterology 55, no. 2 (2020): 248–50. http://dx.doi.org/10.1080/00365521.2019.1710247.

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35

Stranieri, Angelica, Roberta Ferrari, Sergio Zanzani, and Gabriele Rossi. "Sysmex XT-2000iV scattergram analysis in a cat with basophilia." Veterinary Clinical Pathology 45, no. 2 (2016): 225–28. http://dx.doi.org/10.1111/vcp.12340.

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36

Braga, Gisele Wally, Maria de Lourdes Lopes Ferrari Chauffaille, José Eduardo Cajado Moncau, Elizabeth Xisto Souto, Maria Regina Regis Silva, and José Kerbauy. "Chronic myeloid leukemia (CML): prognostic factors and survival analysis." Sao Paulo Medical Journal 114, no. 1 (1996): 1083–90. http://dx.doi.org/10.1590/s1516-31801996000100005.

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The prognostic value of different factors upon diagnosis of CML was analysed in 45 Philadelphia (Ph1)-positive patients. The median survival was 48 months. Univariate analysis showed 5 poor prognostic factors (male sex, under 45 years-old, bone marrow blasts greater than or equal to 10 percent, blood basophils greater than or equal to 6 percent and blood eosinophils greater than or equal to 6 percent) which provided for the development of a clinical staging system: Stage I with none or one factor and a two-year survival rate of 100 percent; Stage II with two or three factors and two-year survi

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37

Cotter, Paul F. "Basophilia and Basophiliosis in Caged Hens at 18 and 77 Weeks." International Journal of Poultry Science 16, no. 2 (2017): 23–30. http://dx.doi.org/10.3923/ijps.2017.23.30.

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38

Chauhan, Kriti, Jatin Sarin, and Vinay Bhatia. "A case of BCR-ABL-Negative myeloproliferative neoplasm presenting with basophilia." Clinical Cancer Investigation Journal 9, no. 5 (2020): 212. http://dx.doi.org/10.4103/ccij.ccij_62_20.

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39

Vundinti, BabuRao, Manisha Madkaikar, Shantashri Vaidya, and Kanjaksha Ghosh. "Deletion of ABL/BCR on der(9) associated with severe basophilia." Indian Journal of Human Genetics 17, no. 2 (2011): 100. http://dx.doi.org/10.4103/0971-6866.86198.

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40

Shang, Huifeng, Ruimin Li, Xiaolei Chai, Xueyan Chen, and Keyu Liu. "AML-MRC with Basophilia Mimicking CLPD Due to Abnormally Clumped Chromatin." Indian Journal of Hematology and Blood Transfusion 36, no. 4 (2020): 781–83. http://dx.doi.org/10.1007/s12288-020-01304-w.

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41

Iwakiri, Rika, Koiti Inokuchi, Kazuo Dan, and Takeo Nomura. "Marked basophilia in acute promyelocytic leukaemia treated with all-trans retinoic acid: molecular analysis of the cell origin of the basophils." British Journal of Haematology 86, no. 4 (1994): 870–72. http://dx.doi.org/10.1111/j.1365-2141.1994.tb04845.x.

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42

Nahajevszky, Sarolta, Maria Magocsi, Gabor Mikala, Hajnalka Andrikovics, Emma Adam, and Tamas Masszi. "Effective Treatment of Basophilic Leukemia of the Central Nervous System in a CML Patient with the Combination of Imatinib Mesylate and Valproic Acid." Blood 106, no. 11 (2005): 4886. http://dx.doi.org/10.1182/blood.v106.11.4886.4886.

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Abstract Imatinib is effective in all stages of CML but the level of the drug in the cerebral spinal fluid is two logs lower than in the plasma. In November, 2000 a 35 years old man in CML chronic phase was treated with interferon (10MU/day) and achieved complete hematologic remission. Two years later he progressed to myeloid blastic crisis. The patient was treated with an acute leukemia protocol (60mg/m2 daunorubicin days 1–3, 200 mg/m2 ara-C days 1–7). After hematological recovery 600 mg/day imatinib was administered continuously. 9 months later headache and vomiting indicated CNS involvemen

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43

Yasuda, Hajime, Nanae Aritaka, Jun Ando, Michihiro Hirama, Norio Komatsu, and Takao Hirano. "Chronic Myelogenous Leukemia with Mild Basophilia as the Predominant Manifestation at Presentation." Internal Medicine 50, no. 5 (2011): 501–2. http://dx.doi.org/10.2169/internalmedicine.50.4695.

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44

Monteiro, Gaby E. R., and Gervásio H. Bechara. "Cutaneous Basophilia in the Resistance of Goats toAmblyomma cajennenseNymphs after Repeated Infestations." Annals of the New York Academy of Sciences 1149, no. 1 (2008): 221–25. http://dx.doi.org/10.1196/annals.1428.026.

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45

Mochizuki, Hiroyuki, Takahiro Seki, Yoshitaka Nakahara, et al. "Chronic myelogenous leukaemia with persistent neutrophilia, eosinophilia and basophilia in a cat." Journal of Feline Medicine and Surgery 16, no. 6 (2013): 517–21. http://dx.doi.org/10.1177/1098612x13505576.

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El-Rifai, Wa'el, Tom Pettersson, Marcelo L. Larramendy, and Sakari Knuutila. "Lineage involvement and karyotype in a patient with myelodysplasia and blood basophilia." European Journal of Haematology 53, no. 5 (2009): 288–92. http://dx.doi.org/10.1111/j.1600-0609.1994.tb01321.x.

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Koumas, S., C. Prokopiou, M. Lerni, O. Seimeni, and N. Neokleous. "Isochromosome 17q10 associated with basophilia in primary myelofibrosis while with JAK2 inhibitor." Annals of Hematology 94, no. 8 (2015): 1421–22. http://dx.doi.org/10.1007/s00277-015-2380-5.

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Katsura, Yukitaka, Kazumi Suzukawa, Toru Nanmoku, et al. "Myelodysplastic syndrome accompanied by basophilia and eosinophilia with t(5;12)(q31;p13)." Cancer Genetics and Cytogenetics 178, no. 1 (2007): 85–88. http://dx.doi.org/10.1016/j.cancergencyto.2007.05.020.

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Delmonte, John, Hagop M. Kantarjian, Elihu Estey, et al. "Single Center Experience with Philadelphia Chromosome-Positive Acute Myeloid Leukemia." Blood 110, no. 11 (2007): 3505. http://dx.doi.org/10.1182/blood.v110.11.3505.3505.

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Abstract Background: Rare instances of Philadelphia chromosome-positive (Ph+) acute myeloid leukemia (AML) have been reported, constituting <1% of de novo cases. However, differentiating these cases from chronic myeloid leukemia presenting in blast phase (CML-BP) has proven difficult. Several clinical and pathologic criteria have been proposed to distinguish Ph+ AML from CML-BP, including absence of an antecedent hematologic disorder, lack of evidence of a chronic or accelerated phase of CML after induction therapy, infrequent splenomegaly and peripheral eosinophilia or basophilia, and bone

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Quintás-Cardama, Alfonso, Hagop Kantarjian, and Jorge Cortes. "Basophilic Blast Phase (B-BP) of Chronic Myelogenous Leukemia (CML)." Blood 110, no. 11 (2007): 4562. http://dx.doi.org/10.1182/blood.v110.11.4562.4562.

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Abstract Approximately 65% of patients with CMP BP exhibit a myeloid phenotype, 30% a lymphoid, and 5% of cases have and undifferentiated or mixed phenotype. Although basophilia is a common feature in both the peripheral blood and the bone marrow during the chronic phase of the disease, transformation to B-BP is a rare occurrence with only anecdotal cases reported. To evaluate the incidence and outcome of B-BP, we reviewed 410 patients with CML who underwent transformation to BP at M.D. Anderson Cancer Center between January 1999 and April 2007. Of them, only 2 (0.5%) cases were diagnosed as h

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