Relevant bibliographies by topics / BCL7

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'BCL7'

Author: Grafiati
Published: 21 December 2024
Last updated: 31 July 2025

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Journal articles on the topic "BCL7"

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Dietrich, Nicholas, Kevin Trotter, James M. Ward, and Trevor K. Archer. "BRG1 HSA domain interactions with BCL7 proteins are critical for remodeling and gene expression." Life Science Alliance 6, no. 5 (2023): e202201770. http://dx.doi.org/10.26508/lsa.202201770.

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The SWI/SNF complex remodels chromatin in an ATP-dependent manner through the subunits BRG1 and BRM. Chromatin remodeling alters nucleosome structure to change gene expression; however, aberrant remodeling can result in cancer. We identified BCL7 proteins as critical SWI/SNF members that drive BRG1-dependent gene expression changes. BCL7s have been implicated in B-cell lymphoma, but characterization of their functional role within the SWI/SNF complex has been limited. This study implicates their function alongside BRG1 to drive large-scale changes in gene expression. Mechanistically, the BCL7
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Jadayel, Dalal M., Lucy R. Osborne, Lionel J. A. Coignet, et al. "The BCL7 gene family: deletion of BCL7B in Williams syndrome." Gene 224, no. 1-2 (1998): 35–44. http://dx.doi.org/10.1016/s0378-1119(98)00514-9.

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McBride, Amanda, Clare M. Adams, Ramkrishna Mitra, and Christine M. Eischen. "Bclw Overexpression Predicts Aggressive Disease in B-Cell Lymphomas." Blood 136, Supplement 1 (2020): 29. http://dx.doi.org/10.1182/blood-2020-142545.

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B-cell lymphomas encompass a phenotypically and genetically heterogenous subgroup within hematologic malignancies. Despite this heterogeneity, the ability to evade apoptosis is a unifying feature across B-cell lymphomas, and alterations within the Bcl-2 family of apoptosis regulatory proteins is a key mechanism for this evasion. While overexpression of the anti-apoptotic Bcl-2 family member BCL2 has been widely described in multiple B-cell lymphomas, altered expression of other anti-apoptotic proteins within this family, including BCLX, MCL1, A1 and, in particular, BCLW has been under-investig
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Kahraman, Dudu Solakoglu, Gulden Diniz, Cengiz Ceylan, et al. "Prognostic impact of BCL2, BCL6 and MYC status in de novo diffuse large B-cell lymphoma: a regional study of 43 patients." International Journal of Research in Medical Sciences 7, no. 5 (2019): 1720. http://dx.doi.org/10.18203/2320-6012.ijrms20191665.

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Background: Diffuse large B-cell lymphoma (DLBCL) is an aggressive non-Hodgkin lymphoma with marked biologic heterogeneity. We aimed to evaluate the status of MYC, BCL2, BCL6 in patients with DLBCL.Methods: Herein, we have investigated the prognostic relevance of MYC, BCL2 and BCL6 from 43 de novo DLBCL patients.Results: In this study, protein overexpression of BCL2 and BCL6 was encountered in 46.5% (n=20) and 27.9% (n=12) of the tumors, respectively. Rearrangements in MYC, BCL6, and BCL2 were detected in 9.3% (n=4), 25.6% (n=11), and 4.7% (n=2) of the cases, respectively. Any statistically si
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Sun, Guoxian, and Lya Montella. "Oncogene Amplification as an Incidental Finding in FISH Testing for Gene Rearrangements in Lymphoid Hematopoietic Neoplasms." Blood 118, no. 21 (2011): 2505. http://dx.doi.org/10.1182/blood.v118.21.2505.2505.

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Abstract Abstract 2505 Oncogene amplification resulting in overexpression, although common in solid tumors, is rare in hematopoietic neoplasms. This is particularly true in lymphoid neoplasms compared to AML where MYC, MLL or RUNX1 (AML1) amplification has been mostly seen, and to CML where BCR/ABL fusion gene amplification has been also reported. Typically, lymphoid neoplasms are tested at diagnosis by FISH for specific reciprocal chromosome translocations that lead to overexpression of deregulated oncogenes such as BCL1, BCL2, BCL6 and MYC in B-cell lymphoma and myeloma or BCR/ABL gene fusio
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González de Villambrosia, Sonia, Mercedes Colorado, Andres Insunza, et al. "B Cell Lymphoma Unclassifiable, with Features Intermediate Between Diffuse Large B Cell Lymphoma and Burkitt Lymphoma and Diffuse Large B Cell Lymphoma NOS with Doble/Triple Translocations: Immunophenotypic Analysis." Blood 126, no. 23 (2015): 5037. http://dx.doi.org/10.1182/blood.v126.23.5037.5037.

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Abstract Background: Diffuse large B-cell lymphoma (DLBCL) is a clinically and molecularly heterogeneous disease. We can identify two subgroups with aggressive clinical course and higher risk of treatment failure after standard therapy: B cell lymphoma unclassifiable (BCLU) with features intermediate between DLBCL and Burkitt lymphoma (BL) and B-cell lymphomas with double/triple translocation (DHL/THL). Previous reports suggest that these types of lymphomas may show a common immunophenotype, providing another tool in the challenge of their diagnosis. Objetives: To analyze the immnunophenotype
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Leski, Tomasz A., Clayton C. Caswell, Marcin Pawlowski, et al. "Identification and Classification of bcl Genes and Proteins of Bacillus cereus Group Organisms and Their Application in Bacillus anthracis Detection and Fingerprinting." Applied and Environmental Microbiology 75, no. 22 (2009): 7163–72. http://dx.doi.org/10.1128/aem.01069-09.

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ABSTRACT The Bacillus cereus group includes three closely related species, B. anthracis, B. cereus, and B. thuringiensis, which form a highly homogeneous subdivision of the genus Bacillus. One of these species, B. anthracis, has been identified as one of the most probable bacterial biowarfare agents. Here, we evaluate the sequence and length polymorphisms of the Bacillus collagen-like protein bcl genes as a basis for B. anthracis detection and fingerprinting. Five genes, designated bclA to bclE, are present in B. anthracis strains. Examination of bclABCDE sequences identified polymorphisms in
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Dupont, Thibault, Zhenghong Dong, ShaoNing Yang, Ari Melnick, and Leandro Cerchietti. "Combinatorial Targeting of BCL6 and Anti-Apoptotic Proteins in Diffuse Large B-Cell Lymphoma (DLBCL) and Follicular Lymphoma (FL)." Blood 120, no. 21 (2012): 64. http://dx.doi.org/10.1182/blood.v120.21.64.64.

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Abstract Abstract 64 BCL6 represents a survival factor in DLBCL and FL since specific BCL6 inhibitors (i.e: the peptidomimetic RI-BPI and the small molecule 79-6) kill DLBCL and transformed FL (tFL) cell lines. Our group showed that BCL2 and other anti-apoptotic genes are transcriptionally repressed by BCL6 and could be reactivated upon treatment with RI-BPI or 79-6. We also showed that BCL6 and BCL2 control distinct and non-overlapping survival pathways in these lymphomas. This suggests that blocking the function of anti-apoptotic proteins might overcome any resistance that these proteins mig
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Pophali, Priyanka, Lisa M. Marinelli, Rhett P. Ketterling, et al. "High Level MYC Amplification in Aggressive B-Cell Lymphomas: Is It a Marker of Aggressive Disease?" Blood 132, Supplement 1 (2018): 1693. http://dx.doi.org/10.1182/blood-2018-99-115484.

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Abstract MYC amplification (amp) is a marker of poor prognosis in many non-hematologic malignancies. While MYC translocations in B cell lymphoma (BCL) have been extensively studied, little is known about the significance of MYC amp. Recent studies describe increased MYC copy numbers (3-10 copies/cell) to be associated with more aggressive BCL. The WHO 2017 does not include MYC amp in the definition of high-grade BCL (HGBCL) with MYC and BCL2 and/or BCL6 rearrangement ("double-hit lymphoma", DHL). However, it also states that high-level MYC amp occurring together with a MYC rearrangement, and c
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Pedersen, Mette Ølgod, Anne Ortved Gang, Tim Svenstrup Poulsen, et al. "Concurrent BCL2 and MYC Translocations In a Prospective Cohort of Diffuse Large B-Cell Lymphomas." Blood 116, no. 21 (2010): 319. http://dx.doi.org/10.1182/blood.v116.21.319.319.

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Abstract Abstract 319 Background: Concurrent chromosomal translocations involving the BCL2 and MYC protooncogenes, so-called double-hit, is found both in diffuse large B-cell lymphoma (DLBCL) and in a newly defined B-cell lymphoma category with features overlapping between DLBCL and Burkitt lymphoma (BCLU, 2008 WHO classification). Few studies have been published on series of double-hit B-cell lymphomas, and to our knowledge only retrospective series, reporting an average frequency of around 5%. Therefore some authors suspected that the frequency of double-hit translocations was underestimated
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Dissertations / Theses on the topic "BCL7"

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Martin, Franck. "Structural and functional studies of chromatin remodeling complex mamalian SWI / SNF." Electronic Thesis or Diss., Strasbourg, 2024. http://www.theses.fr/2024STRAJ044.

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La chromatine est une structure dynamique régulée par différents mécanismes épigénétiques parmi lesquels le remodelage de la chromatine dépendant de l’ATP comme le SWI/SNF. Leur importance est telle que les mutations des protéines de remodelage de la chromatine sont fortement associées à plusieurs maladies dont le cancer. Par exemple les protéines BCL7, qui sont de nouvelles sous unité centrales récemment identifié du complexe SWI/SNF des mammifère, sont associées à différents types de cancers comme dans le Diffuse Large B-cell Lymphoma (DLBCL). Les informations sur les protéines BCL7 sont à c
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Barrans, Sharon Louise. "Immunophenotypic and molecular approaches to the classification of diffuse large B cell lymphoma." Thesis, Manchester Metropolitan University, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.366169.

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Li, Yue. "Investigating Selected Mechanisms of Modulation of BECN1-mediated Autophagy." Diss., North Dakota State University, 2019. https://hdl.handle.net/10365/29775.

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Autophagy is a lysosomal degradation pathway wherein cytoplasmic components not needed by or harmful to the cell are degraded and recycled. BECN homologs are key autophagy proteins consisting of an intrinsically disordered region (IDR), flexible helical domain (FHD), coiled-coil domain (CCD) and β-α repeated, autophagy-specific domain (BARAD). Diverse proteins modulate autophagy by binding BECN1. Understanding the mechanisms by which these proteins regulate BECN1-mediated autophagy is important for developing therapeutics targeting these proteins. Toward this goal, we have developed purificati
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Thompson, Brian M. "Amino-terminal sequences of the bacillus anthracis exosporium proteins BCLA and BCLB important for localization and attachment to the spore surface." Diss., Columbia, Mo. : University of Missouri-Columbia, 2008. http://hdl.handle.net/10355/5700.

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Thesis (M.S.)--University of Missouri-Columbia, 2008.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. "August 2008" Includes bibliographical references.
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Kunze, Doreen. "Small interfering RNA-vermittelte Hemmung der Apoptoseinhibitoren BCL2, BCL-XL, XIAP und Survivin in Zellkultur- und Mausmodellen des humanen Harnblasenkarzinoms." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2012. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-81888.

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Das Harnblasenkarzinom (BCa) stellt in Deutschland die vierthäufigste Tumorneuerkrankung und die zehnthäufigste krebsbedingte Todesursache bei Männern dar. Nichtmuskelinvasive BCa werden organerhaltend aus der Blasenwand entfernt und zur Rezidiv- und Progressionsprophylaxe mittels intravesikaler Chemo- oder Immuntherapien behandelt. Trotz dieser adjuvanten Therapien, die mit starken Nebenwirkungen verbunden sein können, ist nur eine bedingte Minimierung des Rezidivrisikos möglich. Besonders im fortgeschrittenen Stadium weisen Harnblasenkarzinome eine schlechte Prognose auf. Obwohl das BCa eine
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Mahn, Friederike Marie [Verfasser]. "Bruchereignisse in den Onkogenorten BCL2, BCL6 und MYC bei aggressiven B-Zell-Lymphomen im Kindesalter : molekularzytogenetische Analysen im Rahmen der NHL-BFM-Studie / Friederike Maria Mahn." Kiel : Universitätsbibliothek Kiel, 2010. http://d-nb.info/101998337X/34.

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Viant, Charlotte. "Régulation du développement et de la fonction des cellules innées lymphoïdes NKp46+." Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM4018/document.

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Il existe différents groupes de cellules lymphoïdes innées (ILC) qui ont été caractérisées en fonction des facteurs de transcriptions indispensables à leur différenciation et des cytokines qu’elles sécrètent. Les ILC1, dont font partie les cellules Natural Killer (NK), expriment T-bet et produisent de l’IFN-γ. Les ILC2 sont caractérisées par GATA-3 et sécrètent de l’IL-5 et de l’IL-13. Quant aux ILC3, elles ont été identifiées par leur sécrétion d’IL-17 et d’IL-22 ainsi que par l’expression de RORγt.Mon travail de thèse m’a amené à étudier différents aspects de la biologie des cellules NK et I
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Hakert, James Damian. "The crosstalk between notch1 and BCL6." Tallahassee, Fla. : Florida State University, 2010. http://purl.fcla.edu/fsu/lib/digcoll/undergraduate/honors-theses/2181936.

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Thesis (Honors paper)--Florida State University, 2010.<br>Advisor: Dr. Yoichi Kato, Florida State University, College of Arts and Sciences, Dept. of Chemistry and Biochemistry. Includes bibliographical references.
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Warner, Andrew. "Borylative cyclisation of alkynes using BCl3." Thesis, University of Manchester, 2017. https://www.research.manchester.ac.uk/portal/en/theses/borylative-cyclisation-of-alkynes-using-bcl3(7a4e56f3-e8c6-4c68-97ec-4596ef5e0ce2).html.

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Boron trichloride, a cheap and commercially available Lewis acid, has been demonstrated to activate alkynes possessing appropriate nucleophiles, facilitating borylative cyclisation. This reaction furnishes polycyclic compounds possessing a new C(sp2)-B bond externally to the newly formed ring (through concomitant C-C and C-B bond formation). The RBCl2 intermediates generated from cyclisation were esterified with pinacol to furnish air/moisture stable boronic esters. This methodology has been applied to the following classes of starting materials: 1,4-disubstituted but-1-ynes (including N- and
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Leal, Cristina Tavares. "Identificação da família BCL2 como alvo terapêutico no tratamento das neoplasias mieloproliferativas associadas à mutação da JAK2V617F." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17154/tde-06042018-114114/.

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As neoplasias mieloproliferativas (NMPs) negativas para o rearranjo t(9;22)/BCRABL1, incluindo Policitemia Vera (PV), Trombocitemia Essencial (TE) e Mielofibrose Primária (MFP), são doenças hematopoéticas clonais e estão frequentemente associadas à mutação JAK2V617F. Apesar dos avanços no conhecimento da fisiopatologia após a descoberta da mutação JAK2V617F e do desenvolvimento de inibidores da JAK2, o tratamento permanece não curativo. Sabe-se que as célulastronco mais primitivas nas NMPs são responsáveis pela iniciação da doença e que a expansão dos precursores mieloeritróides contribui para
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Books on the topic "BCL7"

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Gavathiotis, Evripidis, ed. BCL-2 Family Proteins. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-8861-7.

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Hetz, Claudio, ed. BCL-2 Protein Family. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6706-0.

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Guelachvili, G., ed. Linear Triatomic Molecules - BClH+ (HBCl+) - COSe (OCSe). Springer-Verlag, 1995. http://dx.doi.org/10.1007/b46104.

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Weil, Harry H. Structuring the smaller corporation under the BCL. Pennsylvania Bar Institute, 1989.

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Catholic Church. National Conference of Catholic Bishops. Committee on the Liturgy. Thirty-five years of the BCL newsletter. United States Conference of Catholic Bishops, 2004.

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Beaton, Jennifer. Amplification and cloning of Bcl-xL and Bcl-xS to obtain in-vitro production of protein products. Laurentian University, 1995.

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United States. National Aeronautics and Space Administration., ed. The apparent strain stability and repeatability of a BCL3 resistance strain gage. National Aeronautics and Space Administration, 1991.

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Tomlin, Jennifer Leigh. Novel biological approaches for detecting oncogenic cooperation with Bcl-2. National Library of Canada, 1999.

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Conference, British Comparative Literature Association. Literary representations of the self: Papers from the fifth triennial BCLA conference 1989. Oxford University Press for the University of St Andrews, 1990.

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Wachek, Volker. Einfluss der BCL-2 Expression auf die Chemoresistenz des malignen Melanoms. [s.n.], 1997.

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Book chapters on the topic "BCL7"

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Posypaiko, V. I., and E. A. Alekseeva. "BCl3." In Phase Equilibria in Binary Halides. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-9024-4_10.

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Ruggiero, Marco, and John W. Anderson. "Bcl2." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_562-4.

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Ruggiero, Marco, and John W. Anderson. "Bcl2." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-642-27841-9_562-5.

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Brocke-Heidrich, Katja. "BCL3." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_565-2.

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Ruggiero, Marco, and John W. Anderson. "Bcl2." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_562.

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Brocke-Heidrich, Katja. "BCL3." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-46875-3_565.

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Ruggiero, Marco. "Bcl2." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_562.

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Brocke-Heidrich, Katja. "BCL3." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-16483-5_565.

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Saidak, Zuzana, Zakaria Ezzoukhry, Jean-Claude Maziere, et al. "BAX (BCl2-Associated X Protein), BCL2L4 (BCL-2 Like 4)." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_100109.

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Vini, Ravindran, Sreeja Sreekumar, Juberiya M. Azeez, and Sreeja Sreeharshan. "Pomegranate Extract Protects Endothelial Cells from TNF-α Associated Damage." In Proceedings of the Conference BioSangam 2022: Emerging Trends in Biotechnology (BIOSANGAM 2022). Atlantis Press International BV, 2022. http://dx.doi.org/10.2991/978-94-6463-020-6_27.

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AbstractPomegranates are known for being rich in polyphenols and are considered to have immense therapeutic potential. The present study investigates the hypothesis that the Methanolic Extract of Pomegranate (PME), a rich source of antioxidants, may reverse the adverse effects of TNF-α in endothelial cells. This was done by pre-treating the endothelial cells EA.hy926 with PME (80 µg/ml) before subjecting them to apoptotic stimuli, which was TNF-α in combination with cyclohexamide. PME was found to rescue a population of cells from apoptosis induced by TNF-α modulating the levels of BCL2 and BA
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Conference papers on the topic "BCL7"

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Salame, Jéssica Paola, Lucas Loss Cantele, Gabriela Gavasso, Beliza Loss, and Karla Patricia Casemiro. "LINFOMA DIFUSO DE GRANDES CÉLULAS B CUTÂNEO SECUNDÁRIO À DOENÇA TESTICULAR PRÉVIA, COM 4 ANOS DE INTERVALO - RELATO DE CASO." In I Congresso Brasileiro de Estudos Patológicos On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/conbesp/18.

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Introdução: Linfomas cutâneos podem ser primários ou secundários. Os primários são os mais frequentes linfomas extra-nodais, com incidência em torno de 10 casos por milhão de habitantes por ano, sendo que destes, 20-30% são Linfomas cutâneos primários de células B. Embora idênticos morfologicamente, podem mostrar cursos clínicos bem distintos, sendo os primários geralmente mais indolentes em comparação com os secundários, que apresentam maiores taxas de doença disseminada e estadiamento avançado ao diagnóstico. Em um estudo, disseminação cutânea foi notada entre 0 e 46 meses após a doença prim
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Hoppe, Michal, Shuangyi Fan, Patrick Jaynes, et al. "Abstract PO-35: Prognostic significance of MYC, BCL2, and BCL6 colocalization at single-cell resolution in DLBCL." In Abstracts: AACR Virtual Meeting: Advances in Malignant Lymphoma; August 17-19, 2020. American Association for Cancer Research, 2020. http://dx.doi.org/10.1158/2643-3249.lymphoma20-po-35.

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Zoeller, JJ, RT Bronson, D. Sampath, J. Leverson, and JS Brugge. "Abstract P4-14-02: Neutralization of BCL2/BCL-XL enhances the cytotoxicity of T-DM1 in vivo." In Abstracts: Thirty-Eighth Annual CTRC-AACR San Antonio Breast Cancer Symposium; December 8-12, 2015; San Antonio, TX. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.sabcs15-p4-14-02.

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Vedernikova, V. O., P. V. Spirin, and V. S. Prasolov. "THE DETERMINATION OF THE RUNX3 TRANSCRIPTION FACTOR’S CONTRIBUTION TO THE SUSTENANCE OF THE MALIGNANT STATUS OF LEUKEMIA CELLS." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-224.

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We established the contribution of RUNX3 to the survival of AML cell line and to the expression of the number of genes. We demonstrated that RUNX3 is closely connected with the antiapoptotic protein Bcl2. It has been shown that RUNX3 is associated with the sensitivity to Venetoclax. The effect of a decrease in oxygen level on the survival of AML cells, expression of RUNX3 and Bcl2 and the sensitivity to different drugs.
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Onen, Onursal, Alper Sisman, Patricia Kruk, and Rasim O. Guldiken. "An Urinary Biosensor for Early Stage Ovarian Cancer Detection: Experimental Characterization." In ASME 2012 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/imece2012-87850.

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In this study, an experimental characterization of a piezoelectric ultrasonic MEMS biosensor for detection of anti-apoptotic protein Bcl-2 in sub ng/ml scale is presented. Bcl-2 is demonstrated to be elevated at different stages of ovarian cancer in urine ranging from 0.5 to 12 ng/ml. Here, shear horizontal (SH) polarized surface acoustic waves (SAWs) were utilized by interdigital transducers (IDTs), which were micro fabricated on piezoelectric ST cut Quartz wafers. SH SAWs were generated and sensed by a pair of IDTs, separated by judiciously designed a delay path in-between with for most effe
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Brock, Benjamin, Aydın Buluç, and Katherine Yelick. "BCL." In ICPP 2019: 48th International Conference on Parallel Processing. ACM, 2019. http://dx.doi.org/10.1145/3337821.3337912.

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Walters, Madeline A., Zhaoyan Fan, and Burak Sencer. "Data-Based Modeling for Reactive Ion Etching: Effectiveness of an Artificial Neural Network Model for Estimating Tungsten Silicon Nitride Etch Rate." In ASME 2020 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/imece2020-23992.

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Abstract This paper presents a data-based approach for modeling a plasma etch process by estimating etch rate based on controlled input parameters. This work seeks to use an Artificial Neural Network (ANN) model to correlate controlled tool parameters with etch rate and uniformity for a blanket 1100 Å WSiN thin film using Cl2 and BCl3 chemistry. Experimental data was collected using a Lam 9600 PTX plasma metal etch chamber in an industrial cleanroom. The WSiN film was deposited over 3000 Å TEOS to ensure adhesion, with an 8-inch bare silicon wafer as the base layer. Controlled tool parameters
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Onen, Onursal, Patricia Kruk, and Rasim Guldiken. "Design of Urinary Biomarker Sensor for Early Ovarian Cancer Detection." In ASME 2011 International Mechanical Engineering Congress and Exposition. ASMEDC, 2011. http://dx.doi.org/10.1115/imece2011-62818.

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In this paper, our efforts on the design, surface functionalization and characterization of ultrasonic MEMS sensor for early ovarian cancer is presented. The sensor detects urinary anti-apoptotic protein Bcl-2 level that has been presented as being elevated for different stages of ovarian cancer. Our novel biosensor approach employs a pair of MEMS ultrasound transducers for generating and sensing surface acoustic waves and a delay path in-between with oriented Bcl-2 antibodies (C8C) attached. Piezoelectric surface acoustic wave devices are employed for sensor for their high coupling efficiency
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Huang, Zi-Xian, and Chuan-Xian Ren. "Rethinking Correlation Learning via Label Prior for Open Set Domain Adaptation." In Thirty-Third International Joint Conference on Artificial Intelligence {IJCAI-24}. International Joint Conferences on Artificial Intelligence Organization, 2024. http://dx.doi.org/10.24963/ijcai.2024/98.

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Open Set Domain Adaptation (OSDA) aims to transfer knowledge from a labeled source domain to an unlabeled target domain, where known classes exist across domains while unknown classes are present only in the target domain. Existing methods rely on the clustering structure to identify the unknown classes, which empirically induces a large identification error if the unknown classes are a mixture of multiple components. To break through this barrier, we formulate OSDA from the view of correlation and propose a correlation metric-based framework called Balanced Correlation Learning (BCL). BCL emp
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Martinez Torre, S., C. Carreño, L. Sordo, et al. "Severity, symptomatology, and treatment duration for paediatric mental health disorders: A retrospective analysis from a conflict affected region of northern Nigeria." In MSF Paediatric Days 2022. MSF-USA, 2022. http://dx.doi.org/10.57740/88gr-bc57.

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Reports on the topic "BCL7"

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ตั้งกิจวานิชย์, พิสิฐ. บทบาทของตัวบ่งชี้ทางระบบภูมิคุ้มกันที่เกี่ยวข้องกับการรักษาด้วยยาต้านไวรัสและการเกิดมะเร็งตับในผู้ป่วยที่ติดเชื้อไวรัสตับอักเสบบีแบบเรื้อรัง : รายงานการวิจัย. จุฬาลงกรณ์มหาวิทยาลัย, 2017. https://doi.org/10.58837/chula.res.2017.14.

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B-cell activating factor (BAFF) เป็น cytokine ที่สำคัญในการกระตุ้นเซลล์เม็ดเลือดขาวชนิด B cell ที่เกี่ยวข้องกับการเกิดไวรัสตับอักเสบบี แต่อย่างไรก็ตามบทบาทของ BAFF ในผู้ป่วยมะเร็งตับที่เกิดจากการติดเชื้อไวรัสตับอักเสบบียังไม่ทราบแน่ชัด การศึกษานี้จึงมีวัตถุประสงค์ในการตรวจวัดระดับ BAFF ในพลาสมาและหาความสัมพันธ์กับความหลากหลายทางพันธุกรรมบนยีน BAFF rs9514828 และ rs12583006 และการทำนายและพยากรณ์ความรุนแรงของโรค โดยตรวจวัดระดับของ BAFF ในพลาสมาของผู้ที่มีสุขภาพดีกลุ่มควบคุม 100 คน กลุ่มผู้ป่วยที่มีการติดเชื้อไวรัสตับอักเสบบี 290 คน และกลุ่มผู้ป่วยมะเร็งตับจากการติดเชื้อไวรัสตับอักเสบบี 200 คน ผลก
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ตั้งกิจวานิชย์, พิสิฐ. บทบาทของตัวบ่งชี้ทางระบบภูมิคุ้มกันที่เกี่ยวข้องกับการรักษาด้วยยาต้านไวรัสและการเกิดมะเร็งตับในผู้ป่วยที่ติดเชื้อไวรัสตับอักเสบบีแบบเรื้อรัง : รายงานการวิจัย. จุฬาลงกรณ์มหาวิทยาลัย, 2018. https://doi.org/10.58837/chula.res.2018.29.

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B-cell activating factor (BAFF) เป็น cytokine ที่สำคัญในการกระตุ้นเซลล์เม็ดเลือดขาวชนิด B cell ที่เกี่ยวข้องกับการเกิดไวรัสตับอักเสบบี แต่อย่างไรก็ตามบทบาทของ BAFF ในผู้ป่วยมะเร็งตับที่เกิดจากการติดเชื้อไวรัสตับอักเสบบียังไม่ทราบแน่ชัด การศึกษานี้จึงมีวัตถุประสงค์ในการตรวจวัดระดับ BAFF ในพลาสมาและหาความสัมพันธ์กับความหลากหลายทางพันธุกรรมบนยีน BAFF rs9514828 และ rs12583006 และการทำนายและพยากรณ์ความรุนแรงของโรค โดยตรวจวัดระดับของ BAFF ในพลาสมาของผู้ที่มีสุขภาพดีกลุ่มควบคุม 100 คน กลุ่มผู้ป่วยที่มีการติดเชื้อไวรัสตับอักเสบบี 290 คน และกลุ่มผู้ป่วยมะเร็งตับจากการติดเชื้อไวรัสตับอักเสบบี 200 คน ผลก
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Machen, Terry. BCL-2, Ca, and Apoptosis in Breast Cancer. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada394121.

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Kim, Hyeong-Reh. Role of Bcl-2 in Breast Cancer Progression. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada383052.

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Xu, Liang. Tumor-Targeted Silencing of Bcl-2/Bcl-xl by Self-Assembled Sirna-Nanovectors as a Novel Molecular Therapy for Breast Cancer. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada475350.

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Marassi, Francesca M. Structural Basis for Bcl-2-Regulated Mitochondrion-Dependent Apoptosis. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada429719.

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Jamerson, Matthew. Cooperation of Bcl-XL and c-Myc in Mammary Tumorigenesis. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada396438.

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Shul, R. J., C. I. H. Ashby, C. G. Willison, et al. GaN etching in BCl{sub 3}Cl{sub 2} plasmas. Office of Scientific and Technical Information (OSTI), 1998. http://dx.doi.org/10.2172/658195.

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Schor, Nina F. Exploiting BCL-2 Overexpression in the Chemotherapy of Breast Cancer. Defense Technical Information Center, 1998. http://dx.doi.org/10.21236/ada350950.

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Jamerson, Matthew H. Cooperation of Bcl-xL and c-Myc in Mammary Tumorigenesis. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada391341.

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