Relevant bibliographies by topics / Behavioral genetics

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Behavioral genetics'

Author: Grafiati
Published: 4 June 2021
Last updated: 29 July 2024

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Journal articles on the topic "Behavioral genetics"

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Morin-Chassé, Alexandre. "Behavioral Genetics, Population Genetics, and Genetic Essentialism." Science & Education 29, no. 6 (November 4, 2020): 1595–619. http://dx.doi.org/10.1007/s11191-020-00166-y.

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Plomin, Robert. "Behavioral Genetics." Journal of Nervous and Mental Disease 177, no. 10 (October 1989): 645. http://dx.doi.org/10.1097/00005053-198910000-00020.

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Hohenboken, William D. "Behavioral Genetics." Veterinary Clinics of North America: Food Animal Practice 3, no. 2 (July 1987): 217–29. http://dx.doi.org/10.1016/s0749-0720(15)31149-x.

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Kerbusch, J. M. L. "Behavioral genetics." Acta Psychologica 75, no. 2 (November 1990): 181–82. http://dx.doi.org/10.1016/0001-6918(90)90097-y.

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Flavell, Steven W., David M. Raizen, and Young-Jai You. "Behavioral States." Genetics 216, no. 2 (October 2020): 315–32. http://dx.doi.org/10.1534/genetics.120.303539.

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Caenorhabditis elegans’ behavioral states, like those of other animals, are shaped by its immediate environment, its past experiences, and by internal factors. We here review the literature on C. elegans behavioral states and their regulation. We discuss dwelling and roaming, local and global search, mate finding, sleep, and the interaction between internal metabolic states and behavior.
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Cesarini, David, Magnus Johannesson, Patrik K. E. Magnusson, and Björn Wallace. "The Behavioral Genetics of Behavioral Anomalies." Management Science 58, no. 1 (January 2012): 21–34. http://dx.doi.org/10.1287/mnsc.1110.1329.

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Plomin, Robert, and Richard Rende. "Human Behavioral Genetics." Annual Review of Psychology 42, no. 1 (January 1991): 161–90. http://dx.doi.org/10.1146/annurev.ps.42.020191.001113.

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Zietsch, Brendan P., Teresa R. de Candia, and Matthew C. Keller. "Evolutionary behavioral genetics." Current Opinion in Behavioral Sciences 2 (April 2015): 73–80. http://dx.doi.org/10.1016/j.cobeha.2014.09.005.

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BAYATI, Suhail, and Ishtar ALMATLOB. "Aquarium Behavioral Genetics." Zeugma Biological Science 4 (August 21, 2023): 7–17. http://dx.doi.org/10.55549/zbs.1340942.

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Eley, Thalia C. "From Behavioral Genetics to Molecular Genetics." Marriage & Family Review 33, no. 1 (January 6, 2003): 57–74. http://dx.doi.org/10.1300/j002v33n01_05.

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Dissertations / Theses on the topic "Behavioral genetics"

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Brown, Elizabeth. "The Behavioral Genetics of Olfaction in Drosophila melanogaster." University of Cincinnati / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1490351166817714.

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Fuller, Tyson David. "Insights into neurodevelopmental disorders: molecular and behavioral studies using the zebrafish." Diss., University of Iowa, 2019. https://ir.uiowa.edu/etd/6945.

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Neurodevelopmental disorders (ND) present a significant burden on society as over 5% of the US population is diagnosed with a ND. While environmental and biological factors have been associated with some cases of NDs, many still have unknown etiology. Strong comorbidities of NDs have been shown suggesting common biological processes of disease development. Sequencing technologies have allowed for the unprecedented identification of new candidate genes associated with NDs and many genes have been linked to multiple NDs. Developing robust methods to functionally validate these candidates is a critical next step for aiding patients with NDs. Using the zebrafish (Danio rerio), we characterized the developmental requirement of epilepsy candidate genes in the context of gene knockdown (KD). We demonstrated three different larval responses to pentylenetetrazol (PTZ) (hyperactive, hypoactive, or the same as control). We characterized the two genes resulting in a hyperactive response, Zinc Finger Homeobox 3 (ZFHX3) and Spectrin Repeat Domain Containing Nuclear Envelope Protein 1 (SYNE1), in greater detail. ZFHX3 is expressed in distinct brain regions during development and shows strong expression along nerve fiber tracts. SYNE1 shows broad expression during development that is enriched in the brain. Using CRISPR/Cas9 we generated a predicted null SYNE1 allele and recapitulated the seizure sensitivity phenotype in mutant larvae. Using a 60-hour behavioral assay we also demonstrate a generalized daytime hyperactivity in SYNE1 mutants. Our studies confirm ZFHX3 and SYNE1 as strong candidates for further study in epilepsy and suggest a role for SYNE1 in multiple NDs such as autism and attention-deficit/hyperactivity disorder.
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Wang, Zhe. "A moderated transactional link between child behavioral problems and parenting: A longitudinal- and behavioral- genetic study." Diss., Virginia Tech, 2013. http://hdl.handle.net/10919/50824.

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Parenting behaviors and a variety of behavioral problems in children covary. The current study first aimed to examine how and why parenting and child behavioral problems are linked in middle childhood. In particular, a longitudinal design (1364 children assessed from 54 months to 5th grade) was used to examine whether the developmental link between parenting and child behavioral problems were reciprocal. A twin design (131 pairs of monozygotic and 173 pairs of dizygotic twins assessed from 6 to 8 years of age on average) was used to examine the underlying genetic and nongenetic etiology of this link. In addition, using these two samples, the current study also aimed to examine whether parental attributes, including negative affect, executive function, and social cognitive factors, modulate the link between parenting and child behavioral problems. Results across these two studies suggested that parenting and child behavioral problems mutually influenced the development of each other over time, potentially through both evocative and passive gene-environment correlation processes and environmental transmissions. In addition, maternal dispositional anger modulated the effects of child behavioral problems on changes in maternal parenting quality over time. Finally, implications of the current study were also discussed.
Ph. D.
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Gardner, Kathryn Regan. "Examining the Genetic, Epigenetic and Behavioral Traits Associated with African American Childhood Obesity." Master's thesis, Temple University Libraries, 2012. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/159645.

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Molecular Biology and Genetics
M.S.
Obesity rates are rising rapidly in the United States, reaching epidemic proportions. Insights into which genes predispose individuals to develop obesity are a necessity. If people at risk for obesity can be identified, individualized treatment programs can be designed based on the individuals' genetic and epigenetic predisposition to help decrease the rate of obesity and obesity-related diseases and deaths. This study will be focusing on the genes FTO, MAOA, SH2B1, CCKAR, NEGR1, LEPR, DNMT3B, and BDNF that have been previously associated with obesity risk and obesity-related phenotypes. Transcript levels of FTO and MAOA were analyzed using quantitative real-time RTPCR, promoter methylation was examined utilizing methylation-sensitive restriction enzyme digestion assays designed for each of the eight gene promoters, and the genotype at eight SNPs, previously associated with obesity, were examined. These data were compared to data gathered on body composition, eating behavior, and temperament. The goals of this project were to replicate results from previous research suggesting associations between certain genetic variants to body composition measures, to identify novel associations between genetic and epigenetic variations and body composition, eating behavior, and temperament, and to provide evidence that the genes previously correlated to obesity in adults is also correlated to measures of obesity and obesity-related phenotypes in children. Decreased levels of methylation in the promoter of BDNF were associated with different eating behaviors including, decreased food fussiness and decreased satiety response. These results were statistically significant after Bonferroni correction for multiple testing. Genotype analysis at the SNP, rs4923461, in BDNF identified an association between the G allele and increased emotional under-eating in males. This association also remained significant after Bonferroni correction. These data gathered for BDNF may suggest a novel role for BDNF in the regulation of energy balance and obesity. The data analysis for all expression, methylation, and genotype data identified associations with 16 different obesity-related phenotypes. These phenotypes included; three measures of body composition, seven eating behaviors, two measures of food intake, one measure of self-regulation, and three measures of temperament. These associations were held to a lower statistical standard and are considered suggestive pending replication in a larger sample. This research was able to provide novel insight into genetic and epigenetic alterations that modify obesity-related phenotypes in African American children. A cumulative genetic and epigenetic "obesity risk factor" score was derived using all significant and suggestive associations to obesity-related phenotypes. The score was derived from the methylation analysis from all eight gene promoters, SNPs from LEPR, DNMT3B, and BDNF, and expression data for MAOA and FTO. The "obesity-risk factor" score was significantly higher in obese compared to non-obese individuals, suggesting the combined genetic and epigenetic approach has value in the prediction of childhood obesity in African Americans.
Temple University--Theses
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Zhang, Xiaodong. "Molecular and Behavioral Mechanisms of Aversive Olfactory Learning in C. elegans." Thesis, Harvard University, 2011. http://dissertations.umi.com/gsas.harvard:10030.

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The mechanisms of learning and memory are fundamental to our understanding of brain function. The aim of this thesis is to characterize the molecular, cellular and behavioral mechanisms underlying the aversive olfactory learning in the model organism C. elegans, to gain insight into animal learning in general. At the molecular level, I focused on the function of the transforming growth factor-\(\beta\) \((TGF-\beta)\) signaling pathway. The \(TGF-\beta\) pathway is conserved throughout the Metazoa and is critical for diverse physiological processes. It has also been implicated in neural plasticity, but the exact mechanisms remain elusive. Utilizing a behavioral assay that measures adult olfactory learning, I have found that DBL-1, a C. elegans \(TGF-\beta\) homolog, is required for learned olfactory avoidance of pathogenic bacteria. Mutations in DBL-1 signal transduction pathway, including those in the dbl-1 ligand, sma-6 and daf-4 receptors, and sma-3 SMAD, abolish the learning ability of adult animals. I have identified AVA neurons, a pair of command interneurons critical for olfactory sensorimotor response, as the essential release site of DBL-1 ligand in regulating learning. AVA neuronal activity is repressed by training, accompanied by an increase in the amount of DBL-1 secreted from AVA neurons after training. Remarkably, artificial inhibition of AVA activity in the absence of training is sufficient to increase AVA secretion of DBL-1, supporting a model in which experience dependent changes in neuronal activity lead to altered DBL-1 \(TGF-\beta\) signaling. Downstream of DBL-1 ligand, I found that the type I receptor SMA-6 primarily acts in the hypodermis to promote olfactory plasticity at the adult stage. At the behavioral level, I examined the taxis behavior of C. elegans toward different bacteria and the effect of training on its taxis strategy. Preliminary results suggest that C. elegans may be capable of modulating its olfactory preference by means of differentially adjusting its navigation strategy. In summary, this thesis uncovered the critical role of DBL-1 \(TGF-\beta\) signaling in C. elegans learning, and alterations in behavioral components underlying olfactory plasticity. These findings are expected to shed light on learning and memory in other animals as well.
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DuPree, Michael G. "A candidate gene study and a full genome screen for male homosexuality." Connect to this title online, 2002. http://etda.libraries.psu.edu/theses/approved/WorldWideIndex/ETD-209/index.html.

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Abbot, Douglas Kilpatrick. "Evolutionary genetics of gall-forming aphids: Population and behavioral processes." Diss., The University of Arizona, 2001. http://hdl.handle.net/10150/279854.

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I investigated patterns of genetic variation in the North American gall-forming aphid, Pemphigus obesinymphae. In Chapters 2a and 2b, I developed and then implemented clone-specific molecular markers to investigate clonal mixing in P. obesinymphae . During its gall-forming phase, P. obesinymphae clones produce aggressive larval "soldiers", which altruistically defend their colonies from natural enemies. I showed that movement occurs between galls, indicating that P. obesinymphae colonies are not pure clones. I also showed that intruders behave selfishly, by not defending unrelated clones, and by accelerating development into reproductive adults. These results reveal a greater degree of complexity and conflict in aphid social groups than previously known. In Chapter 2c, I surveyed molecular variation in P. obesinymphae and its bacterial endosymbiont, Buchnera aphidicola. I found levels of variation at two Buchnera loci to be similar to those estimated from a previous study on a distantly-related aphid, Uroleucon ambrosiae. In the western US, P. obesinymphae and B. aphidicola were nearly monomorphic, and in the eastern US, estimates of synonymous divergence ranged from 0.08 to 0.16%. Most polymorphisms in sub-populations at low frequencies, indicating a recent purge of ancestral polymorphism. These results emphasize the importance of aphid population biology in shaping evolutionary patterns in B. aphidicola. In Chapter 2d, I explored the role of life cycle variation in speciation between Pemphigus aphids. P. obesinymphae and P. populi-transversus are closely-related and sympatric on the cottonwood, Populus deltoides (Salicaceae), but they have distinctly different life cycles. P. populi-transversus has a sexual stage that occurs in the fall, while P. obesinymphae produces sexuales in late spring. Field evidence indicates that intermediate phenotypes rarely occur, and mitochondrial and bacterial endosymbiont DNA sequences show no maternal gene flow between the two species. I considered the possibility of an initial allopatric phase in the divergence, and discuss the sequence of evolutionary changes that likely led to the sympatric divergence of P. populi-transversus and P. obesinymphae. The most plausible interpretation of available data is that a shift in timing of the life cycle in an ancestral population spurred divergence between the species pair.
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Hayat, Roshanai Afsaneh. "Psychological and Behavioral Aspects of Receiving Genetic Counseling for Hereditary Cancer." Doctoral thesis, Uppsala universitet, Vårdvetenskap, 2010. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-128870.

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The overall aims of this thesis were to investigate psychological and behavioral effects of receiving cancer genetic counseling for breast, ovarian and colorectal cancer and/or with a family history of these cancer types and to determine whether counselees’ informational needs were met. Study I was performed 3-7 years post-counseling. Participants (n=214) reported a relatively high level of anxiety but a low level of depression compared to cancer patients in general. However, there was no indication that the distress experienced was due to the counseling. Moderate changes in life and family relations, high level of adherence to recommended controls and satisfaction was reported. Study II was a randomized control trial (RCT) intervention study which involved 147 counselees. An increase in the level of knowledge and correct estimation of personal risk was reported in both the intervention and control groups, although this increase declined at later follow-up. Enhanced information led to significantly greater satisfaction with the given information, and the way of informing relatives. Most counselees had shared information with their at-risk relatives. Study III focused on sharing information with at-risk relatives among participants in study II and their relatives (n=81). Counselees were interviewed and answered a questionnaire, whilst their relatives only answered the questionnaire. Counselees reported positive/neutral feelings about communicating genetic information and mostly interpreted their relatives’ reactions as positive/ neutral. Also, approximately 50% of relatives reported positive/neutral reactions and were generally satisfied with the received information. Study IV was conducted in Sweden and Norway based on 235 counselees. Counselees expected counselors to be skillful and thoughtful, take them seriously and provide risk estimations and medical information. Most important issues to counselees were satisfactorily addressed by the counselors. Analyzing importance rankings resulted in five categories of needs: a need for facts, caring communication and medical information, need for understanding and support in sharing genetic information, practical care and medical/practical information. In conclusion, no adverse psychological or behavioral effect on counselees was observed. Apparently, genetic counseling is managed properly and counselors successfully address counselees’ needs. Providing extended information does not seem necessary, however, tailoring information to individual counselees needs may create a more effective counseling.
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Ploense, Kyle Lawrence. "Self-Administration Results in Dynamic Changes in DNA Methylation of the Dorsal Medial Prefrontal Cortex throughout Forced Abstinence, and after Re-exposure to Cues." Thesis, University of California, Santa Barbara, 2018. http://pqdtopen.proquest.com/#viewpdf?dispub=10689933.

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Similar to the pattern observed in people with substance abuse disorders, laboratory animals will exhibit escalation of cocaine intake when the drug is readily available and will exhibit increased drug-seeking behaviors after long periods of abstinence. Additionally, there are long term changes in neuron structure, receptor function, and neurotransmission associated with abstinence from cocaine in humans and animals. DNA methylation is an epigenetic modification to the DNA structure that mediates mRNA expression to confer different cell types, but has recently been implicated in learning and memory mechanisms. The long-term control that DNA methylation has over gene expression in animals makes it a prime candidate for controlling gene expression over the course of abstinence in animals with previous drug experience. Therefore, here, I investigated the contribution of behavioral contingency of cocaine administration on escalation of cocaine intake and re-exposure to cocaine cues as well as DNA methylation and gene expression within the dorsal medial prefrontal cortex (dmPFC) in adult male Sprague-Dawley rats. I exposed rats to daily training for saline (1 h/ day) or cocaine (0.25 mg/kg/inf) in limited- (1 h access per day), prolonged- (6 h access per day), or limited + yoked-access (1 h contingent + 5 h non-contingent access per day) for 15 days. Rats were then put through forced abstinence for 1, 14, or 60 days, and then the dmPFC was dissected out. Saline- and prolonged-access rats were additionally separated into cue- and no cue- conditions after 60 days of abstinence, where cue rats were re-exposed to the operant chamber without cocaine delivery for 2 h. These studies led to 4 main findings. 1) cocaine contingency affects mRNA expression for glutamatergic genes, 2) DNA methylation changes dynamically throughout abstinence, 3) re-exposure to cocaine cues rapidly alters DNA methylation and mRNA expression, and 4) DNA methylation, hydroxymethylation, and transcription factor binding all contribute to altered mRNA expression.

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Eicher, John Dickinson. "Examining the Genetic Underpinnings of Commonly Comorbid Language Disorders." Thesis, Yale University, 2014. http://pqdtopen.proquest.com/#viewpdf?dispub=3580677.

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Impairments in various aspects of language, including the manipulation and comprehension of verbal and written language, are common in pediatric populations. Some disorders of language are secondary to other clinical presentations, while others, such as dyslexia (or reading disability [RD]), language impairment (LI), speech sound disorder (SSD), and autism spectrum disorders (ASD), have primary deficits in language skills. Each of these is a distinct disorder with unique clinical presentations and deficits. For instance, children with RD have deficits in reading and the use of written language, while those with LI have deficits in the manipulation and comprehension of verbal language. Additionally, children with SSD have difficulties in the production of speech sounds, while children with ASD may have delays or regressions in language and an inability to use complex, proper syntax and pragmatics. However, there is substantial comorbidity of these disorders, as children affected with one of these disorders are more likely to have or develop another disorder than their typically developing peers. These 'disorders—RD, LI, SSD, and ASD—are complex traits, with significant environmental and genetic components contributing to each. Similar to their phenotypic relationships, there is limited evidence that these disorders may share genetic contributors. In fact, these shared genetic components may explain the common phenotypic comorbidities of these disorders. Therefore, the overall goal of this project is to determine whether and to what extent RD, LI, SSD, and ASD share genetic associations with the hypothesis that these disorders have common genetic contributors. To accomplish this goal, I assess whether genetic associations were shared among these disorders or specific to individual disorders. First, I expand the association of the RD environmental risk factor, prenatal exposure to nicotine, to include LI and show the association of dopamine-related genes ANKK1 and DRD2 to LI. Second, two RD risk genes, DCDC2 and KIAA0319, located within the DYX2 locus on chromosome 6p22, show associations with both LI and SSD. Third, I identify ZNF385D as a novel risk gene for subjects affected with comorbid RD and LI. I also assess the neuroimaging implications of DYX2 genes and ZNF385D, specifically in regards to cortical thickness, fiber tract volume, and fractional anisotropy. Finally, two LI risk genes, ATP2C2 and CMIP located within the SLI1 locus on chromosome 16, are associated with language skills of subjects with ASD. Taken together, these results characterize the relationship of previously identified risk genes to other related language disorders and identify novel risk genes that specifically contribute to language comorbidity. Shared genetic associations among these language disorders appear to be commonplace as opposed to the exception. However, the question remains of how these genetic variants interact with each other and other genes/exposures to ultimately lead to one or more of these language deficits seen clinically.

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Books on the topic "Behavioral genetics"

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Plomin, Robert. Behavioral genetics. 5th ed. New York: Worth Publishers, 2008.

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1948-, Plomin Robert, ed. Behavioral genetics. 4th ed. New York: Worth Publishers, 2001.

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Plomin, Robert. Behavioral genetics. 5th ed. New York: Worth Publishers, 2008.

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F, DiLalla Lisabeth, ed. Behavioral genetics. Mahwah, NJ: Lawrence Erlbaum, 1998.

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1948-, Plomin Robert, ed. Behavioral genetics. 3rd ed. New York: W.H. Freeman, 1997.

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1934-, DeFries J. C., and McClearn G. E. 1927-, eds. Behavioral genetics: A primer. 2nd ed. New York: W.H. Freeman, 1990.

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Pietropaolo, Susanna, Frans Sluyter, and Wim E. Crusio, eds. Behavioral Genetics of the Mouse. Cambridge: Cambridge University Press, 2014. http://dx.doi.org/10.1017/cbo9781107360556.

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Crusio, Wim E., Frans Sluyter, Robert T. Gerlai, and Susanna Pietropaolo, eds. Behavioral Genetics of the Mouse. Cambridge: Cambridge University Press, 2009. http://dx.doi.org/10.1017/cbo9781139541022.

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Malykh, S. B. Psikhogenetika--teorii︠a︡, metodologii︠a︡, ėksperiment. Moskva: "Ėpidavr", 2004.

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Plomin, Robert. Genetyka zachowania. Warsaw: Wydawnictwo Naukowe, 2001.

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Book chapters on the topic "Behavioral genetics"

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Abel, Ernest L. "Behavior Genetics." In Behavioral Teratogenesis and Behavioral Mutagenesis, 33–56. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0735-8_2.

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Webster, A. Bruce. "Behavioral Genetics." In Commercial Chicken Meat and Egg Production, 87–99. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0811-3_7.

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Speicher, Michael R. "Behavioral Genetics." In Vogel and Motulsky's Human Genetics, 649. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-37654-5_25.

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Joseph, Jay. "Behavioral Genetics." In Encyclopedia of Critical Psychology, 151–56. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-5583-7_25.

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Gregory, Alice M., Harriet A. Ball, and Tanya M. M. Button. "Behavioral Genetics." In The Wiley-Blackwell Handbook of Childhood Social Development, 27–44. Oxford, UK: Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444390933.ch2.

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Loehlin, John C. "Behavioral Genetics." In Encyclopedia of Personality and Individual Differences, 411–21. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-319-24612-3_734.

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Loehlin, John C. "Behavioral Genetics." In Encyclopedia of Personality and Individual Differences, 1–12. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-28099-8_734-1.

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Loehlin, John C. "Behavioral Genetics." In Encyclopedia of Personality and Individual Differences, 1–11. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-28099-8_734-2.

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Friedrich, Juliane. "Behavioral Genetics." In Encyclopedia of Animal Cognition and Behavior, 1–11. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-47829-6_1401-1.

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Plomin, Robert, John C. DeFries, Ian W. Craig, and Peter McGuffin. "Behavioral genetics." In Behavioral genetics in the postgenomic era., 3–15. Washington: American Psychological Association, 2003. http://dx.doi.org/10.1037/10480-001.

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Conference papers on the topic "Behavioral genetics"

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Tsuruda, Jennifer M. "Using honey bee genetics to breed for behavioral resistance toVarroa mites." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93423.

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Ziebarth, Jesse D., Melloni N. Cook, Biao Li, Robert W. Williams, Lu Lu, and Yan Cui. "Systems genetics analysis of molecular pathways underlying ethanol-induced behavioral phenotypes." In 2010 Biomedical Sciences and Engineering Conference (BSEC). IEEE, 2010. http://dx.doi.org/10.1109/bsec.2010.5510821.

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Ziebarth, Jesse. "7.5: Presentation session: Poster session and reception: “Systems genetics analysis of molecular pathways underlying ethanol-induced behavioral phenotypes”." In 2010 Biomedical Sciences and Engineering Conference (BSEC). IEEE, 2010. http://dx.doi.org/10.1109/bsec.2010.5510822.

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Tracy, William Martin. "Genetic drift resolves Selten's Chain Store Paradox." In the Behavioral and Quantitative Game Theory. New York, New York, USA: ACM Press, 2010. http://dx.doi.org/10.1145/1807406.1807421.

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Krawiec, Krzysztof, and Una-May O'Reilly. "Behavioral programming." In GECCO '14: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2014. http://dx.doi.org/10.1145/2576768.2598288.

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Kakizako, Kosuke, and Yoshiko Hanada. "Genetic Programming for Optimizing Behavioral Rules of Agents Mimicking Human Behavior Patterns." In 2022 Joint 12th International Conference on Soft Computing and Intelligent Systems and 23rd International Symposium on Advanced Intelligent Systems (SCIS&ISIS). IEEE, 2022. http://dx.doi.org/10.1109/scisisis55246.2022.10002152.

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Wahyuningsih, Heni Puji, Bhisma Murti, Eny Lestari, and Reviono Reviono. "The Influence of Social Capital, Parenting, and Environment on Quality of Life among 2-4 Years Old Children." In The 7th International Conference on Public Health 2020. Masters Program in Public Health, Universitas Sebelas Maret, 2020. http://dx.doi.org/10.26911/the7thicph.01.15.

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Background: Quality of life is actually a broader construct that encompasses aspects of life that may not be amenable to healthcare service. The quality of life of children is a subjective perception of satisfaction or happiness on quality of life. The quality of life is influenced by various factors, namely health conditions, socio-economic status, parenting styles, and the environment. According to HL Bloom’s theory, health status is determined by 40 percent of environmental factors, 30 percent of behavioral factors, 20 percent of health services, and 10 percent of genetics or heredity. The purpose of this study was to determine the effect of social capital, parenting, and the environment on the quality of life among children. Subjects and Method: This was a retrospective cohort study. Total of 400 children aged 2-4 years old who reside in the desa layak anak villages and ordinary villages in the region of Sleman regency. The dependent variable was quality of life among children. The independent variables were social capital, parenting, and the environment. Data were obtained from in-depth interview and questionnaire. Data were analyzed using path analysis. Results: The good quality of life of children was affected directly by positive social capital (b = 0.084; SE = 0.049; p = 0.001), good parenting style (b = 0.123; SE = 0.050; p <0.001), and good environment (b = 0.128; SE = 0.048; p <0.001). Conclusion: Social capital, parenting and environment have a direct influence on the quality of life among children. Keywords: quality of life, children, social capital, parenting, environment Correspondence: Heni Puji Wahyuningsih. Doctoral Program of Development Counseling, Universitas Sebelas Maret/ School of Health Polytechnis, Yogyakarta, Indonesia. Email: heni.pujiw@-poltekkesjogja.ac.id DOI: https://doi.org/10.26911/the7thicph.01.15
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Cully, Antoine, and Yiannis Demiris. "Hierarchical behavioral repertoires with unsupervised descriptors." In GECCO '18: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3205455.3205571.

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Pindur, Adam, and Hitoshi Iba. "Behavioral Locality in Genetic Programming." In 12th International Conference on Evolutionary Computation Theory and Applications. SCITEPRESS - Science and Technology Publications, 2020. http://dx.doi.org/10.5220/0010113400810091.

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Ilyasov, R. A., A. G. Nikolenko, and H. W. Kwon. "GENETIC IMPROVEMENT OF HONEY BEES FOR KEEPING IN EXTREMAL CLIMATIC CONDITIONS." In V International Scientific Conference CONCEPTUAL AND APPLIED ASPECTS OF INVERTEBRATE SCIENTIFIC RESEARCH AND BIOLOGICAL EDUCATION. Tomsk State University Press, 2020. http://dx.doi.org/10.17223/978-5-94621-931-0-2020-55.

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Genetic improvement of honey bee populations based on molecular genetics features is faster and precision in comparison with morphometry and behavior-based methods. We developed the method based on nine nuclear microsatellite loci that allow a selection of most adaptive honey bee colonies by genetically defined features. Our study the heterozygosity of the dark European bee A. m. mellifera inhabiting the extremely cold region of the Ural Mountains to provide a marker-assisted selection for revealing the high adapted to extremely cold climate honey bee population can be applied for markerassisted selection of honey bees adapted to beekeeping in extremal climatic conditions.
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Reports on the topic "Behavioral genetics"

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Stanley, Craig, Charles Hadley King, Michelle Thornton, and Rob Kulathinal. Behavioral Genetics: Investigating the genes of a complex phenotype in fruit flies. Genetics Society of America Peer-Reviewed Education Portal (GSA PREP), January 2016. http://dx.doi.org/10.1534/gsaprep.2016.001.

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Bovbjerg, Dana H. Genetic Factors in Breast Cancer: Center for Interdisciplinary Behavioral Research. Fort Belvoir, VA: Defense Technical Information Center, October 2004. http://dx.doi.org/10.21236/ada429513.

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Paul, Satashree. The Criminal Behavior of Genes. Science Repository OÜ, November 2020. http://dx.doi.org/10.31487/sr.blog.14.

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Identifying the crucial role of genetics in criminal behavior implies there must be something known as a “Crime Gene”. Genes come out as the strongest predictor of whether a person has predisposition towards crime or any criminal behavior.
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Pryor, R. J. Developing robotic behavior using a genetic programming model. Office of Scientific and Technical Information (OSTI), January 1998. http://dx.doi.org/10.2172/569136.

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Singh, Anjali. What Is Optogenetics and How Does It Work? ConductScience, July 2022. http://dx.doi.org/10.55157/cs20220704.

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Optogenetics is a biotechnological method that combines optical systems and genetic engineering to control and monitor the functions of cells, tissues, and organisms. It involves using light-sensitive proteins called opsins to manipulate specific cells or regions with precision. This technique has revolutionized neuroscience, allowing researchers to study neural circuits and behavior by turning cells on and off. Opsins are categorized into microbial and animal types, each with specific functions. Optogenetic experiments require opsins, suitable plasmids or viral vectors, and a light source. This method has broad applications in neurology, animal behavior, and physiology, providing insights into various biological processes. It is used to map neural circuits, study diseases, and understand behaviors.
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Conley, Dalton, and Emily Rauscher. Genetic Interactions with Prenatal Social Environment: Effects on Academic and Behavioral Outcomes. Cambridge, MA: National Bureau of Economic Research, May 2010. http://dx.doi.org/10.3386/w16026.

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Pryor, Richard J., and Mark J. Schaller. Developing close combat behaviors for simulated soldiers using genetic programming techniques. Office of Scientific and Technical Information (OSTI), October 2003. http://dx.doi.org/10.2172/918361.

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PRYOR, RICHARD J., and DIANNE C. BARTON. Developing Maneuvering Behaviors for a Glider UAV Using a Genetic Programming Model. Office of Scientific and Technical Information (OSTI), September 2002. http://dx.doi.org/10.2172/803293.

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Jung, Carina, Karl Indest, Matthew Carr, Richard Lance, Lyndsay Carrigee, and Kayla Clark. Properties and detectability of rogue synthetic biology (SynBio) products in complex matrices. Engineer Research and Development Center (U.S.), September 2022. http://dx.doi.org/10.21079/11681/45345.

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Synthetic biology (SynBio) aims to rationally engineer or modify traits of an organism or integrate the behaviors of multiple organisms into a singular functional organism through advanced genetic engineering techniques. One objective of this research was to determine the environmental persistence of engineered DNA in the environment. To accomplish this goal, the environmental persistence of legacy engineered DNA building blocks were targeted that laid the foundation for SynBio product development and application giving rise to “post-use products.” These building blocks include genetic constructs such as cloning and expression vectors, promoter/terminator elements, selectable markers, reporter genes, and multi-cloning sites. Shotgun sequencing of total DNA from water samples of pristine sites was performed and resultant sequence data mined for frequency of legacy recombinant DNA signatures. Another objective was to understand the fate of a standardized contemporary synthetic genetic construct (SC) in the context of various chassis systems/genetic configurations representing different degrees of “genetic bioavailability” to the environmental landscape. These studies were carried out using microcosms representing different environmental matrices (soils, waters, wastewater treatment plant (WWTP) liquor) and employed a novel genetic reporter system based on volatile organic compounds (VOC) detection to assess proliferation and persistence of the SC in the matrix over time.
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Brannon, Ernest L. Columbia River White Sturgeon Genetics and Early Life History: Population Segregation and Juvenile Feeding Behavior, 1987 Final Report. Office of Scientific and Technical Information (OSTI), June 1988. http://dx.doi.org/10.2172/6783328.

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