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Relevant bibliographies by topics / Beige mouse

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Academic literature on the topic 'Beige mouse'

Author: Grafiati

Published: 4 June 2021

Last updated: 18 February 2022

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Journal articles on the topic "Beige mouse"

1

Patiño, Maria Mercedes, Dwight Williams, Joan Ahrens, and John R. Graybill. "Experimental Histoplasmosis in the Beige Mouse." Journal of Leukocyte Biology 41, no. 3 (1987): 228–35. http://dx.doi.org/10.1002/jlb.41.3.228.

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Lounis, N., B. Ji, C. Truffot-Pernot, and J. Grosset. "Comparative activities of amikacin against Mycobacterium avium complex in nude and beige mice." Antimicrobial Agents and Chemotherapy 41, no. 5 (1997): 1168–69. http://dx.doi.org/10.1128/aac.41.5.1168.

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After 4 weeks of treatment, clarithromycin (CLAR) and amikacin showed similar antimicrobial activities against the Mycobacterium avium complex in mice. There was a difference, however, in the effectiveness of the drugs in different types of mice: both drugs displayed bactericidal effects in beige mice but only bacteriostatic effects in nude mice. Because the effectiveness of CLAR is less in nude mice than in beige mice, the predictive value of the nude mouse model for the efficacy of chemotherapy is less than that of the beige mouse model.

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Paulo, Esther, and Biao Wang. "Towards a Better Understanding of Beige Adipocyte Plasticity." Cells 8, no. 12 (2019): 1552. http://dx.doi.org/10.3390/cells8121552.

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Beige adipocytes are defined as Ucp1+, multilocular adipocytes within white adipose tissue (WAT) that are capable of thermogenesis, the process of heat generation. In both mouse models and humans, the increase of beige adipocyte population, also called WAT browning, is associated with certain metabolic benefits, such as reduced obesity and increased insulin sensitivity. In this review, we summarize the current knowledge regarding WAT browning, with a special focus on the beige adipocyte plasticity, collectively referring to a bidirectional transition between thermogenic active and latent states in response to environmental changes. We further exploit the utility of a unique beige adipocyte ablation system to interrogate anti-obesity effect of beige adipocytes in vivo.

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Hioki, K., K. Maruo, S. Suzuki, et al. "Studies on beige–nude mice with low natural killer cell activity 1. Introduction of the bg gene into nude mice and the characteristics of beige–nude mice." Laboratory Animals 21, no. 1 (1987): 72–77. http://dx.doi.org/10.1258/002367787780740761.

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To improve the take-rate of human tumours in nude mice, a nude mouse strain with the bg gene was established. The introduction of the bg gene was confirmed by examination of giant granules of blood neutrophils. The genetic profile of beige–nude mice was the same as that of the C57BL/6 strain according to genetic screening. The natural killer cell activity in the beige–nude mice was much lower than that in ordinary nude mice but slightly higher than that in beige mice. The reproductivity of beige–nude mice was as high as that of ordinary nude mice.

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Gangadharam, P. R. "Beige mouse model for Mycobacterium avium complex disease." Antimicrobial Agents and Chemotherapy 39, no. 8 (1995): 1647–54. http://dx.doi.org/10.1128/aac.39.8.1647.

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6

Ghayur, Tariq, Thomas A. Seemayer, Patricia A. L. Kongshavn, John G. Gartner, and Wayne S. Lapp. "GRAFT-VERSUS-HOST REACTIONS IN THE BEIGE MOUSE." Transplantation 44, no. 2 (1987): 261–66. http://dx.doi.org/10.1097/00007890-198708000-00017.

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Stephenson, Dennis A., Peter H. Glenister, and Janet E. Hornby. "Site of beige (bg) and leaden (ln) pigment gene expression determined by recombinant embryonic skin grafts and aggregation mouse chimaeras employing sash (Wsh) homozygotes." Genetical Research 46, no. 2 (1985): 193–205. http://dx.doi.org/10.1017/s0016672300022655.

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SUMMARYAggregation chimaeras were constructed by fusing embryos homozygous for sash (Wsh) with fuzzy, leaden beige (fz, ln, bg) homozygotes to investigate the site of action of the beige and leaden loci. The genotype of the hair follicle was identified by fuzzy alleles (+ +fz or fzfz). All melanocytes were derived from the fuzzy leaden beige population, as sash homozygotes do not produce functional melanocytes. Reciprocal recombinant epidermal/ /dermal skin grafts were constructed from 14-day embryonic skin of homozygous fzln bg and either albino (aa cc) or pink-eyed dilution (pp) embryos to test for any dermal expression of leaden or beige, since the epidermal and dermal genotype of the chimaeric hair follicles could differ.Patches of fuzzy and non-fuzzy hairs were distributed throughout the coats of two of the three chimaeras obtained. The pigmented regions were blue grey, typical of the leaden beige interactive phenotype. Large abnormal beige granules were found in fuzzy and non-fuzzy hairs. Melanocytes in both classes of growing follicles were nucleopetal, typical of leaden. Similarly, the results of the 14-day skin grafts showed that the beige and leaden loci are melanocyte-autonomous.The chimaeras showed a pigment distribution resembling the heterozygous sash phenotype. Thus the 1:1 gene dosage of sash: wild type in heterozygotes and chimaeras has an overall effect on pigment pattern that overrides the predicted random distribution of the melanocyte precursors.

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Schrijver, G., J. Schalkwijk, J. C. Robben, K. J. Assmann, and R. A. Koene. "Antiglomerular basement membrane nephritis in beige mice. Deficiency of leukocytic neutral proteinases prevents the induction of albuminuria in the heterologous phase." Journal of Experimental Medicine 169, no. 4 (1989): 1435–48. http://dx.doi.org/10.1084/jem.169.4.1435.

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Antiglomerular basement membrane (GBM) nephritis with massive albuminuria can be induced in mice by injection of heterologous antibodies against mouse GBM. The albuminuria and the glomerular lesions in this model are not mediated by complement, but are dependent on the presence of polymorphonuclear granulocytes (PMN) in the glomeruli. Neutral serine proteinases and reactive oxygen metabolites produced by activated PMN have been implicated as agents contributing to tissue damage. We examined the role of leukocytic neutral proteinases by comparing the glomerular damage and albuminuria after injection of rabbit anti-mouse GBM antibodies in normal control mice (C57BL/6J, +/+) and in beige mice (C57BL/6J,bg/bg) in which PMN are deficient of the neutral proteinases elastase and cathepsin G. The dose-dependent albuminuria that occurred in control mice after injection of 1.4-22 mg of anti-GBM antibodies was not observed in beige mice, despite a comparable influx of PMNs in the glomeruli. By electron microscopy both strains showed a similar attachment of PMN to the denuded GBM together with swelling and necrosis of endothelial cells. Elastase activity of extracts from PMN of beige mice was only 10-15% of the activity of control mice. In vitro, GBM degradation by PMN extracts of beige mice was 70% lower than that seen in control experiments. PMNs of beige and control mice showed no differences in superoxide production. In addition, administration of scavengers of reactive oxygen metabolites, such as catalase and desferrioxamine, did not prevent the albuminuria in this model. These findings support the important contribution of leukocytic neutral proteinases to the induction of albuminuria in the acute phase of anti-GBM nephritis in the mouse.

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Strausbauch, Paul, and Nanda Sehgal. "Three-Dimensional Reconstruction of Anomalous Beige Mouse Macrophage Lysosomes." Journal of Leukocyte Biology 46, no. 5 (1989): 441–49. http://dx.doi.org/10.1002/jlb.46.5.441.

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10

Borges, Ricardo, Ruth Jaén, Francisco Freire, José F. Gómez, Cristina Villafruela, and Eulalia Yanes. "Morphological and functional characterization of beige mouse adrenomedullary secretory vesicles." Cell and Tissue Research 304, no. 1 (2001): 159–64. http://dx.doi.org/10.1007/s004410000320.

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Dissertations / Theses on the topic "Beige mouse"

1

Richard, Virgile B. "Regulation of parathroid hormone-related protein in adult T-cell leukemia/lymphoma in a severe combined immuno-deficient/beige mouse model of humoral hypercalcemia of malignancy." The Ohio State University, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=osu1070464988.

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Zhang, Chengsheng. "Functional studies of HIV-1 Vpr protein and development of hu-PBL-SCID/beige mouse model for the studies of HIV-1 infection in vivo." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0001/NQ29133.pdf.

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Richard, Virgile B. "Regulation of parath[y]roid hormone-related protein in adult T-cell leukemia/lymphoma in a severe combined immuno-deficient/beige mouse model of humoral hypercalcemia of malignancy." Connect to this title online, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1070464988.

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Thesis (Ph. D.)--Ohio State University, 2003.<br>Document formatted into pages; contains xiv, 244 p. Includes bibliographical references. Abstract available online via OhioLINK's ETD Center; full text release delayed at author's request until 2005 Dec. 3.

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Wang, Hui [Verfasser], Martin [Akademischer Betreuer] Klingenspor, Thomas [Gutachter] Skurk, and Martin [Gutachter] Klingenspor. "Phenotypic characterization of a novel Ucp1-LUC-iRFP713 reporter mouse model for visualization and quantification of brown and beige fat recruitment / Hui Wang ; Gutachter: Thomas Skurk, Martin Klingenspor ; Betreuer: Martin Klingenspor." München : Universitätsbibliothek der TU München, 2019. http://d-nb.info/1211725154/34.

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Hsi, Dickie Belinda. "Advancing the Alb-uPA/SCID/Bg Chimeric Mouse." Phd thesis, 2009. http://hdl.handle.net/10048/765.

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The feasibility of the Alb-uPA/SCID/Bg chimeric mouse as a model for Hepatitis C Virus (HCV) infection was assessed experimentally by (1) the infection and treatment with another hepatotropic virus, Hepatitis B Virus (HBV) and (2) the infection of the model with HCV and the subsequent treatment of that infection with a pro-apoptotic factor (BID) targeted to infected hepatocytes. In the former, the infected mouse responded favorably, and in the manner of human patients, to a standard imunoglobulin therapy. In the latter, HCV-infected hepatocytes were successfully targeted for cell death, with repeated doses of Adenovirus-delivered BID being the most effective at inhibiting virus spread. Efficacy and toxic side-effects of BID treatment could be reconciled by modulating the timing between doses, the most effective tested being three doses of BID at 7-day intervals. Analyses of chimeric model production were undertaken to improve the quality of human hepatocyte engraftment (typically only 25-35% of mice receiving grafts are currently used experimentally). Minor variations in success rates were experienced with respect to donor age or health status, or the age of recipient mice within an operational window of 5 to 13 days from birth. The greatest obstacle to useful engraftment (aside from technical challenges) was deemed to be the genetic/cellular integrity of the recipient mouse. This conclusion was based on variable engraftment success with ‘healthy’ donor cell preparations and a consideration of variability in immune deficiency arising in mice within a SCID/Bg mouse colony.<br>Experimental Surgery

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Book chapters on the topic "Beige mouse"

1

"Beige Mouse." In Encyclopedia of Immunotoxicology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54596-2_200166.

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2

Sundberg, John P. "The Beige (bg) Mutation, Chromosome 13." In Handbook of Mouse Mutations with Skin and Hair Abnormalities. CRC Press, 2020. http://dx.doi.org/10.1201/9781003068952-25.

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3

Cynamon, M. H., and M. S. DeStefano. "Beige Mouse Model of Disseminated Mycobacterium avium Complex Infection." In Handbook of Animal Models of Infection. Elsevier, 1999. http://dx.doi.org/10.1016/b978-012775390-4/50175-5.

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