Relevant bibliographies by topics / Bell’s palsy

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  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
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Academic literature on the topic 'Bell’s palsy'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 April 2022

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Journal articles on the topic "Bell’s palsy"

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Setiarini, Rohmania. "BELL’S PALSY: SUATU TINJAUAN PUSTAKA." JURNAL KEDOKTERAN 6, no. 2 (2021): 143. http://dx.doi.org/10.36679/kedokteran.v6i2.363.

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Bell’s palsy merupakan kelainan saraf fasialis yang paling banyak dijumpai. Gejala klinis bell’s palsy yaitu adanya lesi saraf fasialis akut tipe lower motor neuron yang terjadi secara tiba-tiba dan cepat. Sekitar 80% pasien sembuh spontan. Etiologi dan patofisiologi masih diperdebatkan. Kehamilan memiliki resiko tiga kali lipat terjadi bell’s palsy. Penegakkan diagnosis berdasarkan klinis. Terapi yang direkomendasikan yaitu pemberian steroid oral. Artikel ini merupakan sebuah studi pustaka.Kata kunci: Bell’s palsy, etiologi, diagnosis, penatalaksanaan
 ABSTRACTBell's palsy is the most common facial nerve disorder. The clinical symptom is acute lower motor neuron type facial nerve lesion that occurs suddenly and rapidly. About 80% of patients recover spontaneously. The etiology and pathophysiology are still being debated. Pregnancy has a threefold risk of developing Bell's palsy. Diagnosis based on clinical. The recommended therapy is oral steroid administration. This article was a literature review.Keyword: Bell’s palsy, diagnosis, etiology, treatment
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Gunawan, Gunawan, Inas Hanan Farihah, Lisa Raihan Lutfia, Muhamad Mahfuzzahroni, and Rizka Febriya Moestafa. "Mengenali Facial Palcy pada Trauma Wajah dan Tatalaksana Pembedahan." Jurnal Syntax Fusion 1, no. 11 (2021): 720–34. http://dx.doi.org/10.54543/fusion.v1i11.100.

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Bell's palsy is the most common neurological disorder affecting the cranial nerves with an onset that is rapid and unilateral, and it is common cause of facial paralysis worldwide. Bell's palsy occurs due to compression or enlargement of the stylomastoid foramen and causes nerve obstruction or damage caused by trauma, infection, inflammation, autoimmune, ischemic. This article aims to review bell's palsy, specifically motor nerve alignment, surgical disorders and management of bell's palsy. Source searches were carried out on online portals for journal publications such as Google Scholar (scholar.google.com) and the National Centre for Biotechnology Information/NCBI (ncbi.nlm.nih.gov), with the keyword “Facial palcy, dan Bell’s palcy”. In the United States, the annual incidence of Bell's palsy is approximately 23 cases per 100,000 people. Permanent facial paralysis and non-transient functional deficits are the main indications for surgical reconstruction of facial nerve function. The indications for surgery depend on the severity of the nerve lesion, blunt trauma that causes nondegenerative neuropraxia does not require surgical reconstruction, whereas disorders leading to degenerative neurotmesis require surgery.
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Alherabi, Ameen, Talal Al-Khatib, Firas R. Abi Sheffah, et al. "Knowledge and Awareness Regarding Bell’s Palsy Among the General Population in the Western Region of Saudi Arabia." Journal of Healthcare Sciences 02, no. 01 (2021): 01–08. http://dx.doi.org/10.52533/johs.2021.2101.

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Background: Bell's palsy is a paralysis of the seventh cranial nerve that controls the muscles of the face, leading to sudden weakness on one side of the face. Of the total, 60–75% of Bell’s palsy cases are considered idiopathic. Thirty percent of the patients have poor recovery as they may experience facial muscle weakness, facial disfigurement, psychological trauma, and facial pain. Method: This study aimed to examine the awareness level regarding Bell’s palsy among the general population of the Makkah, Jeddah and Taif regions of Saudi Arabia. A cross-sectional study design was undertaken from June to September 2021, included 418 participants, and data were collected using an Arabic self-administered online survey. Institutional review board approval and informed consent were obtained. Results: There was a significant association between sex and the knowledge of Bell’s palsy (P=.022). In addition, a previous history of Bell’s palsy was significantly associated with a good knowledge level: 38.1% of participants with a previous history of facial palsy had a good awareness level compared with 17.6% of those who did not (P=.019). Conclusion: The overall awareness regarding Bell’s palsy was unsatisfactory. More efforts, including health education programs, should be made to improve public awareness regarding this disorder and to achieve favorable outcomes and avoid future complications of the condition.
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Bharti, Nasrin. "Role of Neuromuscular Electrical Stimulation in Bell’s Palsy: A Case Study of Three Patients." International Journal of Science and Healthcare Research 6, no. 4 (2021): 235–41. http://dx.doi.org/10.52403/ijshr.20211034.

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Bell's palsy is an idiopathic, unilateral facial paralysis, caused by a malfunction anywhere along the facial nerve's peripheral portion, from the pons distally. Bell's palsy is treated by removing the cause of nerve injury, strengthening the face muscles, and restoring facial function. Physical therapy in the form of neuromuscular electrical stimulation (NMES), massage and facial exercises is used as adjuvant to hasten recovery. The aim of this study is to access of role of neuromuscular electrical stimulation (NMES) treatment in Bell’s palsy patients. A detailed neurological assessment of three patients was done with emphasis on facial muscles and severity of paralysis was graded according to House Brackmann scale (HBS). Conventional physiotherapy was given in the form of electrical stimulation, facial massage, exercises and functional re-education on a daily basis. Patients were assessed at weekly and 1months after the treatment. They experienced complete recovery within 1month follow-up, no recurrence was observed and all patients have normal facial movement. Physiotherapy in the form of NMES and facial exercises has a effective role in the early management of Bell’s palsy. Keywords: Bell’s palsy; neuromuscular electrical stimulation; House Brackmann scale; physiotherapy.
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Maloney, W. J. "Bell’s Palsy." Journal of Dental Research 90, no. 5 (2010): 580–82. http://dx.doi.org/10.1177/0022034510384629.

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Davis, Abigail, and Michael James Gilhooley. "Bell’s palsy." InnovAiT: Education and inspiration for general practice 9, no. 2 (2016): 93–98. http://dx.doi.org/10.1177/1755738015620443.

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Reich, Stephen G. "Bell’s Palsy." CONTINUUM: Lifelong Learning in Neurology 23, no. 2 (2017): 447–66. http://dx.doi.org/10.1212/con.0000000000000447.

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Vakharia, Kavita, and Kalpesh Vakharia. "Bell’s Palsy." Facial Plastic Surgery Clinics of North America 24, no. 1 (2016): 1–10. http://dx.doi.org/10.1016/j.fsc.2015.08.001.

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Minnerop, Martina, Martin Herbst, Rolf Fimmers, Bertfried Matz, Thomas Klockgether, and Ullrich Wüllner. "Bell’s palsy." Journal of Neurology 255, no. 11 (2008): 1726–30. http://dx.doi.org/10.1007/s00415-008-0008-6.

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Kim, So Young, Jee Hye Wee, Chanyang Min, Dae-Myoung Yoo, and Hyo Geun Choi. "Relationship between Bell’s Palsy and Previous Statin Use: A Case/Non-Case Study." International Journal of Environmental Research and Public Health 17, no. 22 (2020): 8408. http://dx.doi.org/10.3390/ijerph17228408.

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We intended to determine the relationship between previous statin use and Bell’s palsy in a large study population receiving statins for the past 2 years. The Korean National Health Insurance Service—Health Screening Cohort data from 2002 to 2015 were collected. Participants with Bell’s palsy (n = 3203) were matched with participants without Bell’s palsy (n = 12,812). The number of days of previous statin use for 2 years before the onset of Bell’s palsy was analyzed using conditional logistic regression. Subgroups of age, sex, obesity, smoking, alcohol consumption, total cholesterol, and blood pressure were analyzed for any association between Bell’s palsy and prior statin use. The Bell’s palsy group reported greater statin use than the non-Bell’s palsy group (84.6 (standard deviation, SD = 201.7) vs. 74.4(SD = 189.4), p = 0.009). Previous statin use was associated with Bell’s palsy in the crude model (95% confidence intervals = 1.03–1.19, p = 0.006). However, this relationship disappeared when the possible covariates were adjusted for in model 2. All subgroups showed no increased odds for Bell’s palsy in previous statin users. We did not find an association between Bell’s palsy and previous statin use in this Korean population aged ≥40 years.
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Dissertations / Theses on the topic "Bell’s palsy"

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Ellsahli, Lutfia Ali. "The management of Bell’s palsy at selected community health centres in the Cape Metropolitan District of the Western Cape, South Africa." University of the Western Cape, 2015. http://hdl.handle.net/11394/4733.

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Magister Scientiae (Physiotherapy) - MSc(Physio)<br>Bell’s palsy (BP), a fairly common disorder predominantly prevalent in the adult age group, affects nerves and muscles in the face causing paralysis or dropping of one side of the face. Clients with Bell’s palsy face many challenges, including psychological, physical and emotional. A long recovery period and/or delayed complete healing could lead to a negative effect on many aspects of an individual's life. How society perceives the person could negatively influence the client’s self-confidence. The management of Bell’s palsy depends on the individual case and may include medication, physiotherapy and as a last option, surgery. The aim of the study was to investigate the management of Bell’s palsy at primary health care level in the Cape Metropolitan District of the Western Cape. The study specific objectives was to investigate the management or treatment protocol of clients with Bell’s palsy, to determine the tendency for referral for physiotherapy, to determine whether an association exists between the type of management or treatment received and the recovery of clients with Bell’s palsy and to explore the impact Bell's Palsy has on the clients. The over-arching design of the study was the sequential explanatory mixed methods design where qualitative data was used to assist in explaining and interpreting the findings of a primarily quantitative study. Stratified random sampling was done proportionately to ensure equal representation. A self-administered questionnaire, comprising of four sections, was used to collect quantitative data that was analysed using SPSS version 21. Descriptive statistics was employed to summarise the data on the socio-demographic information of the clients. Inferential statistics was used to determine the distributions of cases in the various groups. Significant differences tested for using the Chisquare test and effect size through Cramer’s V tests. A semi-structured interview guide was developed based on the results of the analysis of the quantitative data. Focus group discussions were employed to a sub-sample of the clients with Bell’s palsy. Permission an ethical clearance will be obtained from Senate Higher Degrees Committee at the University of the Western Cape (UWC), the Western Cape Department of Health and the facility managers of the participating CHCs.
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Jaishankar, Gayatri, April Lowery, and Roger Smalligan. "Bell's Palsy." Digital Commons @ East Tennessee State University, 2009. https://dc.etsu.edu/etsu-works/8877.

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Morgan, M. "Bell's palsy and infection." Thesis, University of Aberdeen, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.593159.

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Despite various pointers to an infectious aetiology, the cause of Bell's palsy remains obscure. We examined paired sera from 62 patients with facial palsy and 50 age and sex matched contemporaneous controls. Significantly more patients than controls had IgM antibodies by ELISA to varicella zoster virus (56.4% vs 20%, p=0.0001) and herpes simplex virus (41.9% vs 18%, p=0.006). Additionally, significantly more patients than controls were positive for CF antibody to varicella zoster virus (14.5% vs 0%, p=0.004) but not to herpes simplex or cytomegalovirus. Significantly more controls than patients (54% vs 25.8%, p=0.002) had no evidence of antigenic stimulation by any member of the herpesvirus group. No significant difference between patients and controls in seropositivity by IgM ELISA to cytomegalovirus, Epstein-Barr virus and IFA for human herpes virus 6 was found. Furthermore, there was no significant difference between the two groups as to evidence of recent infection by the following agents: rubella virus and <I>Borrelia burgdorferi</I> by IgM ELISA, influenza A, influenza B, adenovirus, respiratory syncytial virus, mumps and measles viruses, <I>Mycoplasma pneumoniae, Coxiella burnetii</I> and chlamydia spp. by complement fixation test. The first reported case of clinically and serologically proven <I>Mycoplasma pneumoniae</I> pneumonia associated with Bell's palsy is described. The rate of complete recovery at 6-8 weeks after onset was not significantly different in patients who were given steroids compared to those who were not. Ear related symptoms were the most common, occurring in 12 of 65 cases, but only three (4.6%) had clinical shingles (vesicles in ear).
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Berg, Thomas. "Medical treatment and grading of Bell's palsy." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-100947.

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Merabet, Mehdi. "Abordagem terapêutica em reabilitação da paralisia facial periférica: revisão da literatura." Bachelor's thesis, [s.n.], 2018. http://hdl.handle.net/10284/6729.

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Projeto de Graduação apresentado à Universidade Fernando Pessoa como parte dos requisitos para obtenção do grau de Licenciado em Fisioterapia<br>Introdução: A paralisia facial periférica (PFP) é uma patologia dos músculos da face mais frequentemente encontrada pelos fisioterapeutas. Para além da perda motora dos músculos faciais, a assimetria facial leva a uma alteração psicológica, com perda da autoestima, autoconfiança, alteração da personalidade e preconceito estético. Objetivo: Analisar as diferentes intervenções terapêuticas na reabilitação de pacientes com PFP. Metodologia: Foi realizada uma pesquisa nas bases de dados, Pubmed e PEDro, com as palavras de pesquisa paralisia facial, paralisia de Bell e fisioterapia cumprindo os critérios de seleção. Resultados: Foram incluídos nesta revisão 7 estudos randomizados controlados e ensaios clínicos com qualidade metodológica de 5,2/10 na escala de PEDro. Os estudos abordam a diferentes intervenções terapêuticas tais como agentes físicos, técnicas orientais e exercícios terapêuticos. Conclusão: Concluiu-se que os diferentes programas de intervenção terapêutica em pacientes com PFP foram eficazes, sendo que o laser de alta intensidade, a electroacupunctura/acupunctura e exercícios neuromuscular mostraram os resultados mais significativos.<br>Introduction: Peripheral facial paralysis (PFP) is a facial muscle pathology most frequently encountered by physiotherapists. In addition to the motor loss of the facial muscles, facial asymmetry leads to a psychological change, with loss of self-esteem, self-confidence, personality change and aesthetic prejudice. Objective: Analyse the different therapeutic interventions in the rehabilitation of patients with PFP. Methods: A research was carried out in the databases, Pubmed and PEDro, with the words search facial palsy, Bell's palsy and physiotherapy, fulfilling the selection criteria. Results: We included 7 randomized controlled trials and clinical trials with methodological quality of 5.2/10 on the PEDro scale. The studies address different therapeutic interventions such as physical agents, oriental techniques and therapeutic exercises. Conclusion: It was concluded that the different therapeutic intervention programs in patients with PFP were effective, and the high intensity laser, electropuncture/acupuncture and neuromuscular exercises showed the most significant results.<br>N/A
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Williamson, I. G. "The nature of Bell's palsy : its aetiology, and the role of Herpes simplex virus." Thesis, University of Edinburgh, 1992. http://hdl.handle.net/1842/27055.

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This thesis aims to investigate the nature and aetiology of Bell's palsy by studying its natural history and epidemiology in general practice, and by means of virological studies. This thesis is concerned principally with testing the first part (sentence) of this hypothesis. A new hypothesis is outlined of the aetiology of Bell's palsy which provides a framework for the investigations:- "Bell's palsy is due to a reactivation of HSV in the geniculate ganglion. During this process, neurotransmitters (opioid peptides) and interferon are produced. These cause local vaso-dilation and damage, particularly to the suprageniculate part of the facial nerve." This thesis is concerned principally with testing the first part (sentence) of this hypothesis. The virological studies set out to examine a possible role for HSV in Bell's palsy, which is contingent on the belief that HSV is normally resident or resident to some degree in the geniculate ganglia of the general population. The evidence of the DNA/DNA hybridization study suggests that HSV may be ubiquitously present in human cadaveric geni? culate ganglia. From these a substantial proportion might be expected to reactivate. In contrast the observed incidence of Bell's palsy in the descriptive study of 16.4 per 100,000 per year suggests that if HSV is a cause the mere occurrence of reactivation is an inadequate explanation of the disease mechanism. The epidemiological studies describe Bell's palsy in British general practice where cases are less strongly selected than in hospital studies. By means of a case-control study and match-pair analysis further inves? tigations are made as to the effect of different factors including various types of stress in the aetiology of Bell's palsy. The results of these studies suggest numerous aetiological agents,of particular relevance to the hypothesis are genetic factors, states of increased "stress" and opioid "sensivity", which are discussed. In conclusion the balance of evidence is compatible with the proposed hypothesis, which in the author's opinion justifies further research, especially since it carries treatment implications.
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Wenceslau, Lais Garcia Capel. "Eletromiografia de superfície e avaliação clínica da mímica facial em pacientes com paralisia facial periférica idiopática." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/5/5170/tde-06082015-144731/.

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INTRODUÇÃO: A paralisia facial periférica idiopática apresenta uma variabilidade clínica de sinais e sintomas que dificultam a realização de uma avaliação objetiva e precisa, com influência no estabelecimento de prognóstico. A eletromiografia de superfície é um exame não invasivo e indolor que permite o estudo da atividade elétrica muscular. Visando compreender quais os efeitos da paralisia facial na atividade elétrica muscular durante o tempo de instalação da doença, o objetivo deste estudo foi utilizar a eletromiografia de superfície para captar a resposta elétrica de músculos da face durante a avaliação clínica do sorriso e correlacionar as respostas musculares com o tempo de instalação da paralisia facial. MÉTODOS: Participaram 140 adultos divididos em três grupos: Grupo I - 35 participantes com paralisia facial periférica com instalação de até 3 meses; Grupo II - 35 participantes com paralisia facial periférica com instalação entre 3 a 6 meses; Grupo III - 70 controles saudáveis. Todos foram submetidos à avaliação que consistiu na aplicação de uma escala clínica para avaliação da mímica facial e da realização do exame de eletromiografia de superfície em região de músculos zigomático e risório. RESULTADOS: Há evidências que os grupos com paralisia facial, independentemente do tempo de início da doença, se diferenciaram significativamente do grupo de indivíduos saudáveis quanto a atividade muscular captada durante o repouso e no sorriso voluntário para ambas as regiões musculares testadas. Os grupos com paralisia facial não se diferenciaram significativamente quando considerada a ativação muscular para nenhuma das avaliações realizadas. O grupo com maior tempo de paralisia facial, apresentou ativação muscular mais assimétrica durante o sorriso voluntário se comparado aos demais grupos. A assimetria muscular foi mais evidente se considerado o funcionamento do músculo risório. CONCLUSÃO: A compatibilização da análise dos dados indica que a avaliação muscular da face por meio da eletromiografia de superfície é reprodutível e é capaz de diferenciar indivíduos com e sem comprometimento muscular<br>INTRODUCTION: Idiopathic facial palsy presents a clinical variability of signs and symptoms, wich turns difficult to perform an objective and accurate assessment, and influences the prognosis. Surface electromyography is a noninvasive and painless test that allows the study of muscle electrical activity. The purpose of this study was to assess, through surface electromyography, the activity of the risorius and zygomaticus muscles, during the production of voluntary smiles and to compare these data between two groups of individuals with different onset times of peripheral facial palsy. METHODS: 140 adults divided into three groups: Group 1 - 35 individuals with Peripheral Facial Palsy onset time between 0 and 3 months; Group 2 - 35 individuals with Peripheral Facial Palsy onset time between 3 and 6 months; Control Group - 70 healthy controls. All of the participants were submitted to the following assessments: clinical protocol for the assessment of facial mimic and surface electromyography of the risorius and zygomaticus muscles. RESULTS: the results suggest that the groups of individuals with Peripheral Facial Palsy differed from the control group considering muscle activity during rest and during the production of voluntary smiles, regardless of the onset time of the disease. The groups with Peripheral Facial Palsy did not differ between themselves in any of the tested situations. The group with Peripheral Facial Palsy with longer onset time presented greater muscle activation asymmetry during the production of the voluntary smiles when compared to the other two groups. Muscle asymmetry was more evident when considering the results for the risorius muscle. CONCLUSION: A compatibilização da análise dos dados indica que a avaliação muscular da face por meio da eletromiografia de superfície é reprodutível e é capaz de diferenciar indivíduos com e sem comprometimento muscular
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Ton, Gi-l., and 唐吉利. "Stroke risk in Bell’s palsy patient receiving Traditional Chinese Medicine treatment: A retrospective cohort study." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/ae2335.

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Lu, I.-Chen, and 盧奕丞. "Effects of Stellate Ganglion Block on Physical Change of Head, Neck and Upper Extremities and effectiveness of treatment in Bell’s Palsy Patients." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/96599820431694634333.

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碩士<br>高雄醫學大學<br>醫學研究所碩士班<br>93<br>Background and aim：Bell’s palsy is defined as peripheral idiopathic facial palsy. Those patients suffered from acute unilateral facial weakness to different extent from mild to severe. The etiology of Bell’s palsy may be due to ischemia, virus infection, congenital infection and unkown. No matter what the cause is, the dysregulation of vasa nervorum microcirculation played a major role in Bell’s palsy. Nerve block was considered as one of an effective method to interrupt dysregulation of circulation. About therapy for Bell’s palsy, merely medical treatment and surgical decompression were discussed in recent review article. However, stellate ganglion block(SGB) has been reported to of excellent efficiency for treatment of Bell’s palsy in our previous study. We evaluated recovery of 31 cases of Bell’s palsy who received SGB as a major part of therapy. Meanwhile, we also investigated in physiological changes after stellate ganglion block by using digital infrared thermography system and reference of laser doppler flowmetry. The goal of this study was to present thermographic assessment of the effectiveness of stellate ganglion block performed for Bell’s palsy patients. Methods：We collected 31 Bell’s palsy patients (17 females, 14 males ,average age 36.7 years old) in this study from April 2004 to November 2004 in pain clinic. We performed SGB at the base of 6th cervical transverse process with 6 ml of 1% plain lidocaine. Every patients received stellate ganglion block twice a week for one month, total block number of each patient was eight. The extent and degree of sympathetic blockade before and after SGB was evaluated by the digital infrared thermography system. Thermography was performed before and 5,10,15,20,25,30 minutes after SGB respectively. We documented temperature change at 5 areas of both lesion and non-lesion sides: (1)frontal , (2)cheek, (3)labial, (4)neck, (5)palm. We excluded one subject who did not show Horner''s syndrome. No severe side effect occurred in each patient. Paired t-test was applied to examined our result. Results：We noted that after SGB surface temperature of bilateral five detected areas were increasing as time went by. The highest temperature occurred at 10 minute after SGB, and average increase temperature was 1.40±0.54 ℃ which was of statistical significance(P<0.05). Among all 5 areas we investigated, labial region was of highest increased surface temperature at 10 minute post-SGB ; it was 2.50℃ higher than surface temperature pre-SGB. Average increased surface temperature of lesion side was 0.22±0.07℃ higher than non-lesion side, but it was not of statistical significance(p>0.05). Digital infrared thermography system could revealed surface temperature change of head , neck and forearm in color image; furthermore, it also provided evidence of physical change after SGB such as improvement of blood circulation of head and neck, increase in surface temperature and enhancement of heat distribution. In our study the rate of total recovery was 93.5 percent within one month period of treatment and following up. Conclusion：Thermography is a useful method for the assessment of stellate blockade effectiveness. Our result supported the affirmative value of stellate ganglion block in improving the blood circulation of Bell’s palsy patients.
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Abreu, Ana Francisca Lopes Ribeiro de Oliveira. "Paralisia Facial Periférica: Estado da arte." Master's thesis, 2020. http://hdl.handle.net/10316/97706.

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Trabalho Final do Mestrado Integrado em Medicina apresentado à Faculdade de Medicina<br>A paralisia facial periférica pode repercutir-se em vários aspetos pessoais e interpessoais da vida do doente, sejam estes funcionais, sociais, psicológicos ou económicos e, deste modo, adquirir dimensões desastrosas na vida do lesado. (1)É conhecido um largo espectro de causas que poderá estar na origem da paralisia facial periférica: infeciosas/inflamatórias, traumáticas, neoplásicas, imunológicas, congénitas, entre outras. Contudo, nenhuma delas provou estar presente na etiologia da maioria dos casos conhecidos, daí que estes sejam frequentemente considerados idiopáticos e designados como paralisia de Bell. (2,3) No que diz respeito à evolução da paralisia de Bell, o quadro clínico é variável, agravando-se nas primeiras 48 horas. A maior parte dos doentes recupera espontaneamente (4), com uma regeneração completa em 3 meses (5,6) e, em apenas 15 a 20% dos casos, surgem sequelas (7). Alguns estudos confirmam que não ocorre persistência de paralisia facial severa ou completa neste tipo de etiologia. (8) A anamnese e o exame físico devem auxiliar a caracterizar a lesão quanto à localização, grau, extensão, etiologia e duração desta. (1) O diagnóstico é normalmente clínico e obtido por exclusão de outras causas, pelo que deverá haver uma avaliação rigorosa de todos os sinais e sintomas relacionados com as regiões e estruturas inervadas pelo nervo facial. (3)O tratamento deve ser individualizado com base na esperança média de vida, preferências do doente, impacto na vida social e défices funcionais. (1) É, geralmente, médico e com recurso a corticosteróides de elevada dosagem (7,9,10), salvo algumas exceções em que o recurso a antivirais e antibioterapia deverão ser ponderados. (9,11,12) O tratamento cirúrgico ficará reservado para casos particulares, sendo os seus objetivos os mesmos de um tratamento médico, com particular prioridade para o encerramento ocular e a simetria no sorriso. (13)A idade e os sinais ou sintomas não são fatores significativos no prognóstico desta patologia (10), sendo este baseado em exames eletrofisiológicos que permitem, através de fórmulas, determinar a funcionalidade restante do nervo facial e dos músculos após a lesão. (14–16)<br>The peripheral facial paralysis is a pathology that can directly impact the patient’s functional, social, economic and psychological life. (1)A large spectrum of causes that can be in the origin of peripheral facial palsy is known as infectious/inflammatory, traumatic, neoplasia, immunological, congenital, between others. Although, none of them has been proved to be present in most of the known cases, being frequently considered idiopathic and designated as Bell’s palsy. (2,3) The clinical signs of Bell’s palsy are variable and usually intensify in the first 48 hours. Most of the patients recover spontaneously (4), with a full regeneration in 3 months (5,6) and sequels are proved to appear in 15 to 20% of the cases. (7) Some studies support that, in this type of aetiology, a persistency of severe or complete facial paralysis does not occur. (8)The anamnesis and the physical examination is essential to establish the location, grade, extension, aetiology and duration of the lesion. (1) The diagnosis is usually obtained by the exclusion of other causes, being crucial the strict evaluation of the clinical signs and symptoms associated with all regions and structures innervated by the facial nerve. (3)The treatment must be performed according to the individual’s average life expectancy, preferences, social life and functional deficits. (1) Most of the times the treatment is medical with high doses of corticosteroids (7,9,10), except in some specific situations where the use of antivirals must be considered. (9,11,12) The surgical treatment is indicated in some particular cases, with the same purposes as the medical treatment but with proper priority in ocular closing and smile symmetry. (13)The prognostic of this pathologic is not significantly affected by the age and beginning of the clinical signs or symptoms. (10) It should be based on electrophysiologic exams that acknowledge, through formulas, the functionality of the facial nerve and muscles before the lesion. (14–16)
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Books on the topic "Bell’s palsy"

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Parker, James N., and Philip M. Parker. The official patient's sourcebook on bell's palsy. Edited by Icon Group International Inc. Icon Health Publications, 2003.

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Polskie pasy kontuszowe. Wydawn. Literackie, 1985.

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Kałamajska-Saeed, Maria. Polskie pasy kontuszowe. Krajowa Agencja Wydawn., 1987.

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Parker, James N., and Philip M. Parker. Bell's palsy: A medical dictionary, bibliography, and annotated research guide to Internet references. ICON Health, 2003.

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Phillips, John. Medical management of Bell’s palsy. Edited by John Phillips and Sally Erskine. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834281.003.0014.

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This chapter discusses Sullivan, Swan, Donnan, Morrison, Smith, McKinstry, Davenport, Vale, Clarkson, Hammersley, Hayavi, McAteer, Stewart, and Daly’s 2007 paper on the medical management of Bell’s palsy including the design of the study (outcome measures, results, conclusions, and a critique).
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Kutz, Walter. Surgical management of Bell’s palsy. Edited by John Phillips and Sally Erskine. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834281.003.0015.

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This chapter discusses Gantz, Rubinstein, Gidley, and Woodworth’s 1999 paper on the surgical management of Bell’s palsy including the design of the study (outcome measures, results, conclusions, and a critique).
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Shaibani, Aziz. Facial Weakness. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0005.

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Unilateral or bilateral facial weakness is an important manifestation of many neuromuscular disorders; some of them are as simple as Bell’s palsy while others are as serious as Guillain-Barrésyndrome. Facial weakness can be easily mimicked, and therefore psychogenic etiology should always be borne in mind. Peripheral facial weakness affects all functions and parts, while central weakness may save the upper face and may affect emotional and voluntary functions differentially. There are several causes of bilateral facial palsy but statistically, Bell’s palsy is still the most common. Long term complications of facial palsy can be devastating and facial rejuvination surgery is usually only modestly effective.
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Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 44-Year-Old Man with Bilateral Facial Droop. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0017.

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Facial neuropathy is most commonly seen as an idiopathic unilateral palsy known as Bell’s palsy. Generally, acute onset of typical lower motor neuron facial weakness that is not associated with other atypical or suspicious features, remains unilateral, and recovers completely requires no further workup. A recurrent or bilateral peripheral facial palsy makes an idiopathic cause less likely and prompts a more in-depth workup. The appropriate work-up of unilateral or bilateral facial palsy guided by the presence or absence of associated clinical findings is discussed. The major differentials for bilateral facial paresis include brainstem (especially pontine and prepontine) tumors, Lyme disease (especially in endemic areas), basal menigitides, Guillain-Barre syndrome, and sarcoidosis.
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Nageshwaran, Sathiji, Heather C. Wilson, Anthony Dickenson, and David Ledingham. Disorders of peripheral nerves and motor neuron disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199664368.003.0007.

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This chapter discusses the clinical features and evidence-based drug treatment regimens of polyneuropathies (Guillain–Barré syndrome (GBS), chronic inflammatory demyelinating polyneuropathy (CIDP), multifocal motor neuropathy, paraproteinaemic neuropathies, and vasculitic neuropathies), mononeuropathies (Bell’s palsy), systemic conditions with peripheral nerve involvement (Sjögren’s and sarcoidosis), and motor neuron disease (MND).
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Shaibani, Aziz. Facial Weakness. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0005.

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Unilateral or bilateral facial weakness is an important manifestation of many neuromuscular disorders; some of them are as simple as Bell’s palsy, while others are as serious as Guillain-Barré syndrome (GBS). Facial weakness can be easily mimicked, and therefore, psychogenic etiology should always be kept in mind. Peripheral facial weakness affects all functions and parts, while central weakness may save the upper face and may affect emotional and voluntary functions differentially. Botulinum toxin injection has become a common cause of facial palsy, therefore detailed history is crucial. Examination of the sensory and motor systems is important to define the type and cause. Imaging and electrodiagnostic testing are often needed in the diagnostic process.
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Book chapters on the topic "Bell’s palsy"

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DePiero, Theslee Joy. "Bell’s Palsy." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_444.

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Hall, Whitney E., Sumayya J. Almarzouqi, Michael L. Morgan, and Andrew G. Lee. "Bell’s Palsy." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-35951-4_1278-1.

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Syms, Mark, and Mitchell Ramsey. "Bell’s Palsy." In Evidence-Based Otolaryngology. Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-49979-6_17.

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DePiero, Theslee Joy. "Bell’s Palsy." In Encyclopedia of Clinical Neuropsychology. Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_444.

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Leung, Alexander K. C., Marcus Schmitt, Christie P. Thomas, et al. "Bell’s Palsy." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8481.

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Hall, Whitney E., Sumayya J. Almarzouqi, Michael L. Morgan, and Andrew G. Lee. "Bell’s Palsy." In Encyclopedia of Ophthalmology. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-540-69000-9_1278.

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Owusu, James A., and Kofi Derek O. Boahene. "Bell’s Palsy." In Encyclopedia of Otolaryngology, Head and Neck Surgery. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-23499-6_365.

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DePiero, Theslee Joy. "Bell’s Palsy." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_444-2.

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Rana, Abdul Qayyum, and John Anthony Morren. "Facial Weakness (Bell’s Palsy)." In Neurological Emergencies in Clinical Practice. Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-5191-3_4.

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Bonkowsky, V., K. Deusch, E. Moschovakis, C. Wagner-Manslau, and R. Kau. "Immunological Findings in Bell’s Palsy." In The Facial Nerve. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-85090-5_180.

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Conference papers on the topic "Bell’s palsy"

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Karaganova, Irina, and Stefka Mindova. "Bell’s palsy." In The 4th International Virtual Conference on Advanced Scientific Results. Publishing Society, 2016. http://dx.doi.org/10.18638/scieconf.2016.4.1.338.

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Parastatidou, S., D. Karakaidos, G. Kafalidis, et al. "P124 Familial bell’s palsy with recurrency." In 8th Europaediatrics Congress jointly held with, The 13th National Congress of Romanian Pediatrics Society, 7–10 June 2017, Palace of Parliament, Romania, Paediatrics building bridges across Europe. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2017. http://dx.doi.org/10.1136/archdischild-2017-313273.212.

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Karolonek, J., A. Radeloff, and M. Loewenthal. "Peripheral facial palsy (Bell’s palsy) as the first symptom of advanced lung cancer." In 100 JAHRE DGHNO-KHC: WO KOMMEN WIR HER? WO STEHEN WIR? WO GEHEN WIR HIN? Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1728916.

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Yamashita, Emilly Sayuri, and Hilton Mariano da Silva Júnior. "Garcin syndrome by giant cell lung tumor." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.056.

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Context: Garcin syndrome is the presence of at least seven ipsilateral cranial nerves palsy, absence of long tract motor or sensory disturbance and intracranial hypertension, and skull base osteoclastic injury. Case report: A 46-year-old woman presented left hypoesthesia. One week later she developed left palsy peripheral facial, the Bell’s palsy. Then, she manifested left deafness and diplopia. The MRI revealed an extra axial lesion in left pre-pontine area. She reported weight loss of 11 kg in 4 months and denied smoking. Palpable lymph nodes were noted on left cervical chain. Neurological examination revealed left cranial nerves V, VI, VII, VIII, IX, X, XI and XII paralysis. Tonus, strength and sensitivity were normal in 4 limbs. After four months, another MRI exhibited an extra axial tumor along the meninges. Chest CT showed an ill-defined left lower lobe lesion. The biopsy revealed a giant cell lung carcinoma (GCC), with cervical bones and encephalus metastasis. She underwent a WBRT radiotherapy. After seven sessions, she presented a cardiorespiratory arrest with death. Conclusion: We can conclude that Garcin syndrome rare condition can have a GCC metastasis involvement. GCC is common um male and smokers. This is the first report of Bell’s palsy as a primary manifestation of GCC, and is also the first case that GCC have Garcin syndrome association. We can assume GCC is a etiology of Garcin syndrome and Bell’s palsy, including in non-smokers.
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Marques, A. M. C., L. G. P. Soares, R. C. Marques, A. L. B. Pinheiro, M. Dent, and Leonardo Longo. "Laser Phototherapy As Modality of Clinical Treatment in Bell’s Palsy." In ADVANCES IN LASEROLOGY - SELECTED PAPERS OF LASER FLORENCE 2010: The 50th Birthday of Laser Medicine World. AIP, 2011. http://dx.doi.org/10.1063/1.3626914.

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Penrose, S., H. Walsh, and Y. Kumar. "G322(P) Not bell’s palsy anymore? lyme disease (ld) until proven otherwise." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.312.

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Hines, Declan, and Manali Chitre. "1488 Management of Bell’s palsy in children – a review of current evidence." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 15 June 2021–17 June 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-rcpch.679.

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Cui, Han, Weizheng Zhong, Mingxing Zhu, et al. "Facial Electromyography Mapping in Healthy and Bell’s Palsy Subjects: A High-Density Surface EMG Study." In 2020 42nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC) in conjunction with the 43rd Annual Conference of the Canadian Medical and Biological Engineering Society. IEEE, 2020. http://dx.doi.org/10.1109/embc44109.2020.9175316.

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Frigo, Yasmim Nadime José, Hendrick Henrique Fernandes Gramasco, Ana Flavia Andrade, et al. "Acute inflammatory polyradiculoneuropathy of atypical presentation." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.576.

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Introduction: Guillain-Barré syndrome is an acute / subacute inflammatory polyradiculoneuropathy that classically results in flaccid areflex palsy. However, there are other possibilities of clinical presentation that must be remembered so that an adequate diagnosis and treatment is carried out. Case report: Female patient, 23 years old, without comorbidities, with complaint of paresthesia in extremities and right peripheral facial paralysis, having diagnosis until then of Bell’s Palsy. She denied previous or current infectious complaints. The neurological examination revealed facial diparesis, proximal weakness of the lower limbs that made walking difficult, tactile and painful hypoesthesia in the feet, with reflexes 1+/4+ in the lower limbs and 3+/4+ in the upper limbs. An investigation was started with CSF collection that showed albuminocytological dissociation (proteins 440 mg/dl and leukocytes 01 mm3). Neuroimaging exams showed contrast impregnation in facial and trigeminal nerves. A diagnosis of acute inflammatory polyradiculoneuropathy was made and treatment with human immunoglobulin was initiated for 5 days. Electroneuromyography showed peripheral, sensory-motor polyradiculoneuropathy and questioned the physiopathological possibility of juxtaparanodopathy. The patient presented a significant and early improvement after treatment. Conclusions: It is essential to consider that Guillain-Barré syndrome has symptom variability, especially according to its pathophysiology and clinical and electrophysiological variant, thus avoiding that conditions such as this one are underdiagnosed.
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Gauza, Mateus de Miranda, Andréia Canello, Henrique Muller Genero, Rafael Marques Mendes, Maria Francisca Moro Longo, and Jordana Dolores Villar Lino. "Ramsay Hunt syndrome - a case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.298.

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Context: Ramsay Hunt syndrome is uncommon, with an incidence of 5 / 100,000 people (1), characterized by the triad of ipsilateral peripheral facial paralysis, otalgia and vesicles in the ear canal and / or auricular pavilion (1,2,3). It results from the reactivation of the Varicella Zoster virus in the geniculate ganglion and may extend to other cranial pairs, causing inflammation, edema and nerve dysfunction (1,3). As the second leading cause of peripheral facial palsy (1), its importance lies in a lower likelihood of recovery with a worse prognosis compared to Bell’s palsy (4). Case report: M.R.S, female, 54 years old, went to the emergency room due to ophthalmological complaints (blurred vision, diplopia, floaters, photophobia), nausea and vomiting, associated with vertigo, headache and facial hypoesthesia on the left. With the worsening of the condition, she was referred to the neurology service of the municipal hospital São José, presenting dysarthria, vesicular lesions in the left external auditory canal, preserved tactile sensitivity, MRC grade V and facial paralysis on the left (House Brackmann grade IV). Lumbar puncture and cerebrospinal fluid analysis were performed to discard other pathologies of the central nervous system. The diagnosis of Ramsay Hunt syndrome was established and treatment with Acyclovir and prednisone was started. After worsening renal function, drug therapy was suspended and venous hydration increased. She continued with improved renal function and was discharged with valacyclovir and corticosteroids. Conclusions: The importance of early diagnosis and treatment is emphasized in order to avoid permanent sequelae of the disease (1,3).
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Reports on the topic "Bell’s palsy"

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Abas, Razif, Aspalilah Alias, Mohd Amir Kamaruzzaman, et al. Cranial Polyneuropathy in Bell’s Palsy: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.6.0111.

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Zhang, Yingrong, Sanchun Tan, Jieyu Wang, et al. A scoping review protocol of systematic reviews and meta-analyses to acupuncture for the treatment of peripheral facial paralysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.3.0084.

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Review question / Objective: To conduct a systematic comprehensive review for Acupuncture treatment of peripheral facial paralysis and to evaluate the efficacy and safety of acupuncture therapy for peripheral facial paralysis. Condition being studied: Peripheral facial paralysis, known as peripheral facial never palsy, includes Bell’s palsy and Ramsay Hunt syndrome.Any medical conditions such as infection, malignancy and autoimmune issues can result it. Idiopathic Bell's palsy is the most common disease causing peripheral facial nerve palsy, which clinical features include unilateral weakness of the facial nerve, hyperacusis, dysgeusia, dry eye or uncontrollable tears, but the etiology of it is unclear. Ramsay Hunt syndrome, less common than Bell’s palsy, is often caused by herpes zoster virus, which clinical features are unilateral weakness of face with ear herpes, tinnitus and dizziness. Facial paralysis patients with ear herpes can be diagnosed with Ramsay Hunt syndrome. Peripheral facial paralysis not only result the dyskinesia of facial muscles but also affect the quality of patient’s life.There are lot of evidence shows that Acupuncture can be used in any period and any kind of peripheral facial paralysis.However, we still lack systematic reviews to assess the efficacy and safety of acupuncture therapy. As a result, we conduct a scoping review of systematic reviews and meta-analyses to address this gap.
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Li, Bing. Effectiveness comparisons of acupuncture treatments for Bell palsy in adults: a Bayesian network meta-analysis protocol. INPLASY - International Platform of Registered Systematic Review Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.4.0019.

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Corticosteroids improve recovery rates after Bell’s palsy. National Institute for Health Research, 2016. http://dx.doi.org/10.3310/signal-000336.

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