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Relevant bibliographies by topics / Benign Dyskeratosis

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Academic literature on the topic 'Benign Dyskeratosis'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Benign Dyskeratosis"

1

Dithmar, S., R. D. Stulting, and H. E. Grossniklaus. "Hereditary benign intraepithelial dyskeratosis." Der Ophthalmologe 95, no. 10 (1998): 684–86. http://dx.doi.org/10.1007/s003470050335.

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2

Shields, C. L., J. A. Shields, and R. C. Eagle. "Hereditary Benign Intraepithelial Dyskeratosis." Archives of Ophthalmology 105, no. 3 (1987): 422–23. http://dx.doi.org/10.1001/archopht.1987.01060030142045.

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3

Nair, Nandakumar Gopinathan, and B. M. Athira. "Acantholytic dyskeratotic acanthoma: A rare clinicopathological entity – A case report and review of literature." Journal of Skin and Sexually Transmitted Diseases 2 (October 15, 2020): 115–18. http://dx.doi.org/10.25259/jsstd_20_2020.

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Acantholytic dyskeratotic acanthoma (ADA) is a rare benign condition characterized by circumscribed epidermal proliferation displaying both acantholysis and dyskeratosis. It is of unknown etiology and pathogenesis. We report a case of ADA of long duration in a 75-year-old female along with a review of literature with special reference to cases reported during the past 10 years.

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4

Al Mahmoud, B., N. Al Hayki, and H. Al Meslamani. "Hereditary Benign Intra-Epithelial Dyskeratosis (HBID)." Qatar Medical Journal 2011, no. 1 (2011): 16. http://dx.doi.org/10.5339/qmj.2011.1.16.

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5

Baroni, Adone, Marco Palla, Francesco Saverio Aiello, et al. "Hereditary benign intraepithelial dyskeratosis: case report." International Journal of Dermatology 48, no. 6 (2009): 627–29. http://dx.doi.org/10.1111/j.1365-4632.2009.03893.x.

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6

Jham, Bruno Correia, Ricardo Alves Mesquita, Maria Cássia Ferreira Aguiar, and Maria Auxiliadora Vieira Carmo. "Hereditary benign intraepithelial dyskeratosis: a new case?" Journal of Oral Pathology & Medicine 36, no. 1 (2006): 55–57. http://dx.doi.org/10.1111/j.1600-0714.2006.00456.x.

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7

Badam, Yashwitha, Vijaya Mohan Rao Avisa, A. Sumalatha, G. Pavan Kumar Reddy, and A. Bhavana. "Sporadic dyskeratosis congenita in a male – A case report." IP Indian Journal of Clinical and Experimental Dermatology 8, no. 1 (2022): 55–56. http://dx.doi.org/10.18231/j.ijced.2022.012.

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Dyskeratosis congenita was first described in 1960 as Zinsser-Engman-Cole syndrome. It is a rare inherited condition with a progressive nature and a tendency to involve multiple systems like pulmonary, gastrointestinal, genitourinary, cerebral, and dental. It has an X-linked recessive (most common) or Autosomal dominant or recessive inheritance with a high male preponderance. The genetic defect lies in the DKC1 gene which encodes for Dyskerin protein. Dyskeratosis congenita patients are at a higher risk of development of malignancies, pulmonary fibrosis and eventually aplastic anemia and bone

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8

Al-Mohammedi, Faisal, Gillian C. de Cannes, and Richard I. Crawford. "Friction-Induced Pagetoid Dyskeratosis." Journal of Cutaneous Medicine and Surgery 17, no. 4 (2013): 250–52. http://dx.doi.org/10.2310/7750.2012.12071.

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Background: Pagetoid dyskeratosis (PD) is characterized by pale cells within the epidermis resembling those of Paget disease. These cells have been seen as an incidental finding in a variety of benign papules most commonly located in intertriginous areas. The lesion is considered a reactive process in which a small proportion of the normal population of keratinocytes is altered. Among the triggers for this lesion, friction has been suggested; however, a direct cause-and-effect relationship has not yet been reported. Results: We confirmed the relationship between PD and friction in a biopsy tak

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9

Cummings, Thomas J., Leslie G. Dodd, Christopher R. Eedes, and Gordon K. Klintworth. "Hereditary Benign Intraepithelial Dyskeratosis: An Evaluation of Diagnostic Cytology." Archives of Pathology & Laboratory Medicine 132, no. 8 (2008): 1325–28. http://dx.doi.org/10.5858/2008-132-1325-hbidae.

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Abstract Context.—Hereditary benign intraepithelial dyskeratosis (HBID) is a rare autosomal dominant disorder characterized by elevated epibulbar and oral plaques and hyperemic conjunctival blood vessels. The condition is predominantly seen in Native Americans belonging to the Haliwa-Saponi tribe located in northeastern North Carolina. Objective.—To determine whether HBID can be diagnosed using cytologic preparations of the conjunctiva, and whether the cytologic findings correlated with the genetic linkage involving a duplication in chromosome 4 (4q35). Design.—Cytologic preparations from conj

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10

Eagle, R. C. "Hereditary Benign Intraepithelial Dyskeratosis: An Evaluation of Diagnostic Cytology." Yearbook of Ophthalmology 2009 (January 2009): 237–38. http://dx.doi.org/10.1016/s0084-392x(09)79019-7.

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Book chapters on the topic "Benign Dyskeratosis"

1

Biradar, Vijay. "Hereditary Benign Intraepithelial Dyskeratosis." In Mucocutaneous Lesions in Dentistry. Jaypee Brothers Medical Publishers (P) Ltd., 2016. http://dx.doi.org/10.5005/jp/books/12662_21.

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