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Relevant bibliographies by topics / Beta adrenergic receptor blockaders

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Academic literature on the topic 'Beta adrenergic receptor blockaders'

Author: Grafiati

Published: 4 June 2021

Last updated: 21 February 2023

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Journal articles on the topic "Beta adrenergic receptor blockaders"

1

DiCarlo, S. E., R. W. Blair, V. S. Bishop, and H. L. Stone. "Role of beta 2-adrenergic receptors on coronary resistance during exercise." Journal of Applied Physiology 64, no. 6 (June 1, 1988): 2287–93. http://dx.doi.org/10.1152/jappl.1988.64.6.2287.

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The effects of regional alpha- and specific beta 2-adrenergic receptor blockade on measurements of late diastolic coronary resistance (LDCR) and mean coronary blood flow velocity (CBFV) during exercise were examined in 14 conscious adult mongrel dogs. Specific beta 2-adrenergic receptor blockade (ICI 118.551) significantly decreased CBFV and increased LDCR by blockade of beta 2-vasodilator tone independent of alpha-adrenergic receptor-mediated tone and independent of altering myocardial metabolism. alpha-Adrenergic receptor blockade (phentolamine, 1 mg) significantly increased CBFV and decreas

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Awad, E. W., and M. Anctil. "IDENTIFICATION OF beta-LIKE ADRENOCEPTORS ASSOCIATED WITH BIOLUMINESCENCE IN THE SEA PANSY RENILLA KOELLIKERI." Journal of Experimental Biology 177, no. 1 (April 1, 1993): 181–200. http://dx.doi.org/10.1242/jeb.177.1.181.

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Previous studies have reported pharmacological and biochemical evidence for the involvement of adrenergic substances in the regulation of neuroeffector activities in the bioluminescent cnidarian Renilla koellikeri (Cnidaria, Anthozoa). Therefore, direct radiobinding assays were developed to identify and characterize beta-adrenergic binding in membrane preparations from this species, using the two beta-antagonists [3H]dihydroalprenolol and [3H]CGP12177 as tracers. In addition, the effect of various beta-adrenergic agents on luminescence was examined. Binding of the radioligands at 25°C was rapi

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Jasper, J. R., H. J. Motulsky, L. C. Mahan, and P. A. Insel. "Beta-adrenoceptor metabolism in wild-type, Gs, and protein kinase A-variant S49 cells." American Journal of Physiology-Cell Physiology 259, no. 1 (July 1, 1990): C41—C46. http://dx.doi.org/10.1152/ajpcell.1990.259.1.c41.

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To determine the role of the stimulatory guanine nucleotide-binding protein, Gs, and adenosine 3',5'-cyclic monophosphate (cAMP)-dependent protein kinase in the basal metabolism of beta-adrenergic receptors in S49 lymphoma cells, we measured the return of receptor number and function after irreversible blockade of receptors. After inactivation of receptors with the irreversible ligand N8-(bromoacetyl)-N'-[3-(4-indolyoxy)-2-hydroxypropyl]-(2)-1,8-diam ino-p- methane (BIM), beta-adrenergic receptors (defined as [125I]iodocyanopindolol binding sites) reappeared in a biphasic manner, the faster ph

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Vatner, S. F., D. R. Knight, and T. H. Hintze. "Norepinephrine-induced beta 1-adrenergic peripheral vasodilation in conscious dogs." American Journal of Physiology-Heart and Circulatory Physiology 249, no. 1 (July 1, 1985): H49—H56. http://dx.doi.org/10.1152/ajpheart.1985.249.1.h49.

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Norepinephrine (NE) elicits alpha-adrenergic vasoconstriction and beta 1-adrenergic increases in heart rate and myocardial contractility. To determine whether NE can also elicit peripheral beta 1-adrenergic vasodilation, conscious dogs were studied after recovery from instrumentation for the measurement of cardiac output, arterial pressure, and left ventricular (LV) pressure and calculations of LV dP/dt and total peripheral resistance (TPR). NE, after pretreatment with hexamethonium and phentolamine, reduced mean arterial pressure 40 +/- 5% from 117 +/- 9 mmHg and TPR 60 +/- 5% from 0.058 +/-

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Kagiya, T., M. Hori, K. Iwakura, K. Iwai, Y. Watanabe, S. Uchida, H. Yoshida, A. Kitabatake, M. Inoue, and T. Kamada. "Role of increased alpha 1-adrenergic activity in cardiomyopathic Syrian hamster." American Journal of Physiology-Heart and Circulatory Physiology 260, no. 1 (January 1, 1991): H80—H88. http://dx.doi.org/10.1152/ajpheart.1991.260.1.h80.

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We investigated serial changes in myocardial norepinephrine content and myocardial adrenergic receptors during the development of cardiomyopathy in Syrian hamsters (Bio 14.6) and their age-matched healthy controls. We also examined phosphatidylinositide hydrolysis after alpha 1-adrenergic stimulation and the effects of alpha 1-blockade. We found that in the prehypertrophic stage, myocardial norepinephrine content and densities of alpha 1- and beta-adrenergic receptors were significantly higher in the cardiomyopathic hamsters than in the controls. However, in the early heart failure stage, beta

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Borst, M. M., R. Marquetant, W. Kubler, and R. H. Strasser. "Beta-blockade reduces effects of adenosine and carbachol by transregulation of inhibitory receptors and Gi proteins." American Journal of Physiology-Heart and Circulatory Physiology 272, no. 4 (April 1, 1997): H1672—H1679. http://dx.doi.org/10.1152/ajpheart.1997.272.4.h1672.

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Chronic blockade of stimulatory beta-adrenergic receptors may decrease inhibitory receptors of the adrenergic signal transduction system. This transregulation process might reduce the negative inotropic response of the myocardium to inhibitory receptor stimulation. Rats were treated for 6 days with the beta-blocker atenolol (2 mg/day). beta-Adrenergic receptors in cardiac plasma membranes increased from 49 +/- 6 to 75 +/- 9 fmol/mg protein (means +/- SE; P = 0.053), whereas muscarinic M2 receptors decreased (155 +/- 15 vs. 105 +/- 10 fmol/mg protein; P < or = 0.05). Moreover, inhibitory G a

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Kaufman, Beth D., and Robert E. Shaddy. "Beta-adrenergic receptor blockade and pediatric dilated cardiomyopathy." Progress in Pediatric Cardiology 24, no. 1 (November 2007): 51–57. http://dx.doi.org/10.1016/j.ppedcard.2007.08.004.

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Laughlin, M. H., and R. B. Armstrong. "Adrenoreceptor effects on rat muscle blood flow during treadmill exercise." Journal of Applied Physiology 62, no. 4 (April 1, 1987): 1465–72. http://dx.doi.org/10.1152/jappl.1987.62.4.1465.

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The purpose of this study was to examine the effects of the adrenergic receptors on the distribution of blood flow within and among skeletal muscles in rats. Blood flow was measured with the radiolabeled microsphere technique before exercise and during treadmill exercise at 15 or 60 m/min. Alpha- (phentolamine) or beta- (propranolol) adrenergic blocking drugs were administered, and then blood flow was measured and results compared with those from saline-treated rats. Before exercise, alpha-blockade caused increases in total muscle blood flow and in all fast-twitch muscles, whereas muscles comp

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Kuleshova, E. V., N. V. Kuzmenko, M. G. Pliss, and V. A. Tsyrlin. "Mechanisms of beta-blockers antihypertensive action." "Arterial’naya Gipertenziya" ("Arterial Hypertension") 27, no. 3 (August 4, 2021): 291–99. http://dx.doi.org/10.18705/1607-419x-2021-27-3-291-299.

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This article presents an analysis of data on the mechanisms of antihypertensive effect of β-adrenergic receptor blockers. The article describes the effectiveness of cardiotropic action of drugs to reduce high blood pressure (BP) with short-term and long-term action of compounds, the effect of blockers on the activity of plasma renin. The inﬂuence of β-blockers on the central mechanisms of blood circulation regulation is considered. Information on the effect of β-blockers on myogenic mechanisms of vascular tone regulation is presented. The possibilities of blockade of β-adrenergic receptors of

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Ferrara, L. Aldo, Teodoro Marotta, Paolo Rubba, Biagio De Simone, Giovanni Leccia, Stefano Soro, and Mario Mancini. "Effects of alpha-adrenergic and beta-adrenergic receptor blockade on lipid metabolism." American Journal of Medicine 80, no. 2 (February 1986): 104–8. http://dx.doi.org/10.1016/0002-9343(86)90168-3.

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Dissertations / Theses on the topic "Beta adrenergic receptor blockaders"

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Plank, David Michael. "Calcium dynamics, β-adrenergic receptor blockade, and cardiac function in failing and non-failing hearts". University of Cincinnati / OhioLINK, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1053431441.

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Plank, David M. "Calcium dynamics, [beta]-adrenergic receptor blockade, and cardiac function in failing and non-failing hearts." Cincinnati, Ohio : University of Cincinnati, 2003. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=ucin1053431441.

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Grogan, Shawn Patrick, of Western Sydney Hawkesbury University, Faculty of Environmental Management and Agriculture, and School of Agriculture and Rural Development. "Endocrine alteration of meat quality and gene expression in rats and deer." THESIS_FEMA_ARD_Grogan_S.xml, 1998. http://handle.uws.edu.au:8081/1959.7/724.

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Stress activates a number of endocrine pathways that alter an animal's physiology in a manner which can result in undesirable meat quality. Animals frequently exhibit meat quality defects, including ecchymosis, at slaughter due to the stress of slaughter. This thesis explores how stress related hormones interact with adrenergic receptors to alter muscle and vascular physiology. Fallow deer were exposed to either a transciptional regulator (hydrocortisone), a beta adrenergic recptor agonist (clenbuterol) or a beta adrenergic receptor antagonist (propranolol). The administration of hydrocortison

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Grogan, Shawn Patrick. "Endocrine alteration of meat quality and gene expression in rats and deer." Thesis, [Richmond, N.S.W.] : CSIRO Animal Production : School of Agriculture, University of Western Sydney, Hawkesbury, 1998. http://handle.uws.edu.au:8081/1959.7/724.

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Stress activates a number of endocrine pathways that alter an animal's physiology in a manner which can result in undesirable meat quality. Animals frequently exhibit meat quality defects, including ecchymosis, at slaughter due to the stress of slaughter. This thesis explores how stress related hormones interact with adrenergic receptors to alter muscle and vascular physiology. Fallow deer were exposed to either a transciptional regulator (hydrocortisone), a beta adrenergic recptor agonist (clenbuterol) or a beta adrenergic receptor antagonist (propranolol). The administration of hydrocortison

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Bagley, John Sorrell. "The beta adrenergic receptor in septic shock." Thesis, University of Aberdeen, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.305466.

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The myocardial depression found during septic shock has been a subject of intense study for many years. Many theories exist as to the precise cause of this condition but in this thesis the role of the β-adrenergic receptor is examined. Some investigators have found reduced response to β-adrenergic stimulation in models of septic shock in animals. This is of interest to the clinician since adrenergic stimulation is a key manipulation in the management of septic shock. An animal model of septic shock was established using a comparatively small dose of <i>Escherichia coli</i> endotoxin infused in

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Higgins, Thomas John Diatchenko Luda. "Molecular characterization of [beta]2 adrenergic receptor haplotypes." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2008. http://dc.lib.unc.edu/u?/etd,1602.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2008.<br>Title from electronic title page (viewed Sep. 16, 2008). "... in partial fulfillment of the requirements for the degree of Master's of Science in the Curriculum in Neurobiology." Discipline: Neurobiology; Department/School: Medicine. On title page, [beta] appers as Greek character and 2 appears in subscript.

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Vincent, Karla Kristine. "Transactivation of Beta 2 Adrenergic Receptor by Bradykinin type 2 Receptor via heterodimerization." Diss., Georgia Institute of Technology, 2009. http://hdl.handle.net/1853/37117.

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Although a long standing convention maintained that G Protein Coupled Receptors (GPCRs) exist in the plasma membrane solely as monomers, substantial work over the last two decades has demonstrated that these ubiquitous receptors can and in many cases, preferentially, exist as homodimers, heterodimers, or higher order oligomers. Often, two GPCRs of the same class heterodimerize; it is less common for two GPCRs of different signaling pathways to interact. The work presented here studied the physical and functional interaction of two GPCRs from discrete classes, the Beta 2 Adrenergic Receptor

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8

Snyder, Ellen Amanda Flood Patrick M. "[beta]2-adrenergic receptor modulation of macrophage inflammatory mediator production." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,1344.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2007.<br>Title from electronic title page (viewed Apr. 25, 2008). "... in partial fulfillment of the requirements for the degree of Master of Science in the Curriculum of Oral Biology." Discipline: Oral Biology; Department/School: Dentistry. On title page, [beta] appears as Greek character and 2 appears as subscript.

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Robson, Alice. "In vitro reconstitution and stability of a beta-adrenergic receptor." Thesis, University of Bristol, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.424005.

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Zolty, Ronald. "Beta 1 and Alpha 2C adrenergic receptor polymorphisms and response to beta blockers in heart failure patients /." Connect to full text via ProQuest. Limited to UCD Anschutz Medical Campus, 2007.

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Thesis (Ph.D. in Clinical Science) -- University of Colorado Denver, 2007.<br>Typescript. Includes bibliographical references (leaves 130-142). Free to UCD affiliates. Online version available via ProQuest Digital Dissertations;

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Books on the topic "Beta adrenergic receptor blockaders"

1

Cruickshank, J. M. Beta-blockers in clinical practice. Edinburgh: Churchill Livingstone, 1988.

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Prichard, Brian N. C. 1932-, ed. Beta-blockers in clinical practice. Edinburgh: Churchill Livingstone, 1987.

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Drugs looking for diseases: Innovative drug research and the development of the beta blockers and the calcium antagonists. Dordrecht: Kluwer Academic Publishers, 1991.

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4

Advances in adrenergic receptor biology. Amsterdam: Elsevier, 2011.

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5

Owens, Helen. The effect of beta-adrenergic receptor antagonists on the temporal accommodative response. Birmingham: Aston University. Department of Vision Sciences, 1991.

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1944-, Costello J. F., and Mann Ronald D. 1928-, eds. Beta agonists in the treatment of asthma: The proceedings. Carnforth, Lancs, UK: Parthenon Pub. Group, 1992.

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Richard, Beasley, and Pearce Neil, eds. The Role of beta receptor agonist therapy in asthma mortality. Boca Raton: CRC Press, 1993.

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1946-, Frishman William H., ed. Beta₃-adrenergic agonism: A new concept in human pharmacotherapy. Armonk, N.Y: Futura, 1995.

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Cruickshank, J. M. Beta-Blockers in Clinical Practice. 2nd ed. W.B. Saunders Company, 1994.

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1948-, Deedwania Prakash C., ed. Beta-blockers and cardiac arrhythmias. New York: M. Dekker, 1992.

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Book chapters on the topic "Beta adrenergic receptor blockaders"

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Schomburg, Dietmar, and Dörte Stephan. "beta-Adrenergic-receptor kinase." In Enzyme Handbook, 113–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-59025-2_21.

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Ross, Elliott M. "Reconstitution of the Beta-Adrenergic Receptor and Its Biochemical Functions." In The Beta-Adrenergic Receptors, 125–79. Totowa, NJ: Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_4.

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3

Erhardt, Paul W., and Lajos Matos. "Selective Beta-Adrenergic Receptor-Blocking Agents." In Analogue-based Drug Discovery, 193–232. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527608001.ch11.

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Stiles, Gary L. "Drug and Hormonal Regulation of the Beta-Adrenergic Receptor—Adenylate Cyclase System." In The Beta-Adrenergic Receptors, 345–86. Totowa, NJ: Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0463-3_8.

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Özkaya, Esen, and Kurtuluş Didem Yazganoğlu. "Beta Adrenergic Receptor Blockers (Class II Antiarrhythmics)." In Adverse Cutaneous Drug Reactions to Cardiovascular Drugs, 111–21. London: Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6536-1_6.

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Hershberger, Ray E. "Beta-Adrenergic Receptor Agonists and Antagonists in Heart Failure." In Congestive Heart Failure, 454–92. New York, NY: Springer New York, 1994. http://dx.doi.org/10.1007/978-1-4613-8315-4_25.

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Hammond, J., and J. L. Balligand. "Signalling Microdomains: The Beta-3 Adrenergic Receptor/NOS Signalosome." In Microdomains in the Cardiovascular System, 215–44. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-54579-0_11.

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Collins, Sheila, and S. Wang. "Molecular regulation of the β2-adrenergic receptor by long- and short-acting β-agonists." In Treatment of Asthma: The long-acting beta-2-agonists, 1–14. Vienna: Springer Vienna, 1998. http://dx.doi.org/10.1007/978-3-7091-7513-2_1.

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Davideit, Hanna, Annekathrin Haberland, Sabine Bartel, Sarah Schulze-Rothe, Johannes Müller, and Katrin Wenzel. "Determination of Agonistically Acting Autoantibodies to the Adrenergic Beta-1 Receptor by Cellular Bioassay." In Autoantibodies, 95–102. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8949-2_8.

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Kather, H. "Beta Adrenergic Receptor Mediated Stimulation of Adenine Nucleotide Catabolism and Purine Release in Human Adipocytes." In Purines in Cellular Signaling, 120–25. New York, NY: Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3400-5_20.

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Conference papers on the topic "Beta adrenergic receptor blockaders"

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Cano, E. J., J. A. Gonzalez, J. L. Perez-Lara, F. Casado, A. Zhang, and C. Zeana. "Beta-Adrenergic Receptor Blockers in E. coli Bacteremia: Severity of Presentation and Outcomes." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6499.

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Carroll, Christopher L., Craig Schramm, Indira Sadhu, and Aaron R. Zucker. "Beta-Adrenergic Receptor Haplotype Associated With Recurrent Near Fatal Asthma Exacerbations In Children." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a2509.

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Khan, Y., SJ Fowler, and G. Tavernier. "S140 Beta-2-adrenergic receptor gene polymorphisms in severe asthma: a systematic review." In British Thoracic Society Winter Meeting, Wednesday 17 to Friday 19 February 2021, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2021. http://dx.doi.org/10.1136/thorax-2020-btsabstracts.145.

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Kim, Joon H., Lawrence D. Longo, and Sean M. Wilson. "Attenuated Beta Adrenergic Receptor Mediated Pulmonary Vasodilation In High Altitude Term-Fetal Sheep." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4831.

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Ou, Z., L. Wang, X. Hong, Z. Shen, and Z. Shao. "Overexpression or Lack of beta 2-Adrenergic Receptor Correlate with Progression of Breast Cancer." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-4165.

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Stolk, R., E. van der Pasch, F. Naumann, J. Schouwstra, T. van Herwaarden, J. Gerretsen, R. Schambergen, et al. "Anti-Inflammatory Effects of Phenylephrine in Mice and Humans Mediated via the Beta Adrenergic Receptor." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2590.

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Zucker, Aaron R., Christopher L. Carroll, Indira Sadhu, and Craig Schramm. "Racial Differences Of Beta-Adrenergic Receptor Genotypes In Children Hospitalized For Near Fatal Asthma Exacerbations." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a3907.

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Carroll, Christopher L., Aaron R. Zucker, Kathleen Sala, and Craig M. Schramm. "Racial Differences Of Beta-Adrenergic Receptor Genotype & Response To Therapy In Children With Bronchiolitis." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4164.

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Dong, Junhong, Ping Wang, Qingluan Xu, Kunpeng Lv, Yu Wang, and Junhong Dong. "Expression of Beta-Adrenergic Receptor in Glioma LN229 Cells and Its Effect on Cell Proliferation." In 2016 7th International Conference on Education, Management, Computer and Medicine (EMCM 2016). Paris, France: Atlantis Press, 2017. http://dx.doi.org/10.2991/emcm-16.2017.140.

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Owens, H., B. Winn, B. Gilmartin, and J. R. Pugh. "The Effect of a Topical Beta-Adrenergic Receptor Antagonist on the Dynamics of Steady-State Accommodation." In Noninvasive Assessment of the Visual System. Washington, D.C.: Optica Publishing Group, 1991. http://dx.doi.org/10.1364/navs.1991.md9.

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Temporal fluctuations in the steady-state human ocular accommodation response are characterised by two principal frequency components: a high frequency component (HFC: 1-2.2Hz) and a low frequency component (LFC:<0.5Hz) (Campbell, Robson and Westheimer, 1959; Kotulak and Schor, 1986; Winn et al, 1989). Recent work has demonstrated that the location of the HFC is significantly correlated with arterial pulse (Winn et al, 1990). These findings have led to the proposal that the HFCs are not governed by an accommodative control system, although they may potentially be utilised in conjunction wit

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