Relevant bibliographies by topics / BFU-E (Burst Forming Unit Erythroid)

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Academic literature on the topic 'BFU-E (Burst Forming Unit Erythroid)'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "BFU-E (Burst Forming Unit Erythroid)"

1

Royet, J., G. Mouchiroud, S. Arnaud, T. Oddos, S. Galland, and JP Blanchet. "Kidney cell lysates contain an activity that stimulates mature erythroid burst-forming-unit (mBFU-E) proliferation." Blood 76, no. 10 (1990): 1965–71. http://dx.doi.org/10.1182/blood.v76.10.1965.1965.

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Abstract This study reports the detection of an activity that stimulates the development of a subclass of burst-forming unit-erythroid (BFU-E) progenitors giving rise to small bursts in semi-solid cultures established in the presence of saturating concentrations of erythropoietin. These progenitors are considered to be mature BFU-E. The activity is found in extracts from kidney cells and appears to be physiologically regulated as it was respectively enhanced and decreased in kidneys from anemic and polycythemic mice. The disappearance of activity in kidney-cell extracts during long-term polycy
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2

Royet, J., G. Mouchiroud, S. Arnaud, T. Oddos, S. Galland, and JP Blanchet. "Kidney cell lysates contain an activity that stimulates mature erythroid burst-forming-unit (mBFU-E) proliferation." Blood 76, no. 10 (1990): 1965–71. http://dx.doi.org/10.1182/blood.v76.10.1965.bloodjournal76101965.

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This study reports the detection of an activity that stimulates the development of a subclass of burst-forming unit-erythroid (BFU-E) progenitors giving rise to small bursts in semi-solid cultures established in the presence of saturating concentrations of erythropoietin. These progenitors are considered to be mature BFU-E. The activity is found in extracts from kidney cells and appears to be physiologically regulated as it was respectively enhanced and decreased in kidneys from anemic and polycythemic mice. The disappearance of activity in kidney-cell extracts during long-term polycythemia co
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3

DeZern, Amy E., Michael A. McDevitt, Richard J. Jones, and Robert A. Brodsky. "Burst-Forming Unit-Erythroid Assays (BFU-E) Can Help Distinguish Cellular Bone Marrow Failure Disorders." Blood 120, no. 21 (2012): 3475. http://dx.doi.org/10.1182/blood.v120.21.3475.3475.

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Abstract Abstract 3475 Cytopenias in the context of cellular marrows cause significant morbidity for patients and are challenging for clinicians to diagnose and treat. Often the differential diagnosis includes pure red cell aplasia (PRCA), large granular lymphocyte leukemia (LGL), marrow suppression associated with systemic inflammation, and clonal bone marrow failure disorders such as myelodysplastic syndrome (MDS) or paroxysmal nocturnal hemoglobinuria (PNH). Unfortunately, it can be difficult to differentiate between these possibilities, and correct treatment depends on accurate diagnosis.
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4

Aucella, F. "Desferrioxamine improves burst-forming unit-erythroid (BFU-E) proliferation in haemodialysis patients." Nephrology Dialysis Transplantation 13, no. 5 (1998): 1194–99. http://dx.doi.org/10.1093/ndt/13.5.1194.

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5

Rich, IN. "The effect of 5-fluorouracil on erythropoiesis." Blood 77, no. 6 (1991): 1164–70. http://dx.doi.org/10.1182/blood.v77.6.1164.1164.

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Abstract The effects of a single dose (150 mg/kg) of 5-fluorouracil on mature erythroid and erythropoietic and multipotential in vitro precursor populations in the bone marrow and spleen and circulating biologically (erythroid colony forming unit [CFU-E] assay) and immunologically active (enzyme-linked immunosorbent assay) erythropoietin (Epo) are described. All mature erythroid (reticulocytes, erythrocytes) and in vitro erythropoietic precursors (CFU-E, erythroid burst-forming unit [BFU-E]) are severely reduced, if not eradicated. Transient repopulation of the pure BFU-E and CFU-E populations
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6

Rich, IN. "The effect of 5-fluorouracil on erythropoiesis." Blood 77, no. 6 (1991): 1164–70. http://dx.doi.org/10.1182/blood.v77.6.1164.bloodjournal7761164.

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The effects of a single dose (150 mg/kg) of 5-fluorouracil on mature erythroid and erythropoietic and multipotential in vitro precursor populations in the bone marrow and spleen and circulating biologically (erythroid colony forming unit [CFU-E] assay) and immunologically active (enzyme-linked immunosorbent assay) erythropoietin (Epo) are described. All mature erythroid (reticulocytes, erythrocytes) and in vitro erythropoietic precursors (CFU-E, erythroid burst-forming unit [BFU-E]) are severely reduced, if not eradicated. Transient repopulation of the pure BFU-E and CFU-E populations on days
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7

Dai, CH, SB Krantz, and KM Zsebo. "Human burst-forming units-erythroid need direct interaction with stem cell factor for further development." Blood 78, no. 10 (1991): 2493–97. http://dx.doi.org/10.1182/blood.v78.10.2493.2493.

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Abstract To understand the factors that regulate the early growth and development of immature erythroid progenitor cells, the burst-forming units-erythroid (BFU-E), it is necessary to have both highly purified target cells and a medium free of serum. When highly purified human blood BFU-E were cultured in a serum-free medium adequate for the growth of later erythroid progenitors, BFU-E would not grow even with the addition of recombinant human interleukin-3 (rIL-3), known to be essential for these cells. However, the addition of recombinant human stem cell factor (rSCF), which supports germ ce
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8

Dai, CH, SB Krantz, and KM Zsebo. "Human burst-forming units-erythroid need direct interaction with stem cell factor for further development." Blood 78, no. 10 (1991): 2493–97. http://dx.doi.org/10.1182/blood.v78.10.2493.bloodjournal78102493.

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To understand the factors that regulate the early growth and development of immature erythroid progenitor cells, the burst-forming units-erythroid (BFU-E), it is necessary to have both highly purified target cells and a medium free of serum. When highly purified human blood BFU-E were cultured in a serum-free medium adequate for the growth of later erythroid progenitors, BFU-E would not grow even with the addition of recombinant human interleukin-3 (rIL-3), known to be essential for these cells. However, the addition of recombinant human stem cell factor (rSCF), which supports germ cell and pl
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9

Flygare, Johan, Violeta Rayon Estrada, Chanseok Shin, Sumeet Gupta та Harvey F. Lodish. "HIF1α synergizes with glucocorticoids to promote BFU-E progenitor self-renewal". Blood 117, № 12 (2011): 3435–44. http://dx.doi.org/10.1182/blood-2010-07-295550.

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AbstractWith the aim of finding small molecules that stimulate erythropoiesis earlier than erythropoietin and that enhance erythroid colony-forming unit (CFU-E) production, we studied the mechanism by which glucocorticoids increase CFU-E formation. Using erythroid burst-forming unit (BFU-E) and CFU-E progenitors purified by a new technique, we demonstrate that glucocorticoids stimulate the earliest (BFU-E) progenitors to undergo limited self-renewal, which increases formation of CFU-E cells > 20-fold. Interestingly, glucocorticoids induce expression of genes in BFU-E cells that contain prom
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10

Suzuki, Norio, Naruyoshi Suwabe, Osamu Ohneda, et al. "Identification and characterization of 2 types of erythroid progenitors that express GATA-1 at distinct levels." Blood 102, no. 10 (2003): 3575–83. http://dx.doi.org/10.1182/blood-2003-04-1154.

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AbstractTranscription factor GATA-1 is essential for the development of the erythroid lineage. To ascertain whether strict control of GATA-1 expression level is necessary for achieving proper erythropoiesis, we established transgenic mouse lines expressing green fluorescent protein (GFP) under the control of the GATA-1 gene hematopoietic regulatory domain. We examined the GATA-1 expression level by exploiting the transgenic mice and found 2 GFP-positive hematopoietic progenitor fractions in the bone marrow. One is the GFPhigh fraction containing mainly CFU-E and proerythroblasts, which coexpre
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Dissertations / Theses on the topic "BFU-E (Burst Forming Unit Erythroid)"

1

Lauret, Evelyne. "Contribution à l'étude du contrôle humoral de la détermination de la différenciation des cellules souches hématopoiétiques pluripotentes (CFU-s) chez la souris." Paris 11, 1985. http://www.theses.fr/1985PA112310.

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Le système murin est utilisé pour étudier les mécanismes de régulation du choix de différenciation que fait la cellule souche pluripotente hématopoïétique (CFU-S) vers l’une ou l’autre des lignées sanguines. La méthode employée pour cette étude est basée sur l’analyse histologique des nodules spléniques formés par le développement des clones de CFU-S dans la rate de souris irradiée ayant reçu de la moelle 10 jours auparavant. Les proportions de chaque catégorie histologique étant définies et constantes pour une souche de souris donnée, il apparait que tout changement des proportions des différ
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