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Relevant bibliographies by topics / Bile electrolytes

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Academic literature on the topic 'Bile electrolytes'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Bile electrolytes"

1

Tavoloni, Nicola. "Biliary excretion of inorganic electrolytes: its role in hepatic bile formation." Canadian Journal of Physiology and Pharmacology 63, no. 10 (1985): 1245–51. http://dx.doi.org/10.1139/y85-206.

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To define the role of inorganic electrolyte secretion in hepatic bile formation, the effects of secretin, glucagon, and differently structured bile acids on bile flow and composition were studied in the dog, guinea pig, and rat. In the dog and guinea pig, secretin (2.5–10 clinical units∙kg−1∙30 min−1) increased bile flow and bicarbonate concentration in bile, a finding consistent with the hypothesis that the hormone stimulates a bicarbonate-dependent secretion possibly at the level of the bile ductule–duct. In the rat, secretin (5–15 CU∙kg−1∙30 min−1) failed to increase bile secretion. Glucago

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2

Fasano, Alessio, Maria Carmela Verga, Francesco Raimondi, and Stefano Guandalini. "Effects of Deconjugated Bile Acids on Electrolyte and Nutrient Transport in the Rabbit Small Intestine In Vitro." Journal of Pediatric Gastroenterology and Nutrition 18, no. 3 (1994): 327–33. http://dx.doi.org/10.1002/j.1536-4801.1994.tb11183.x.

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To obtain new information on the poorly investigated mechanisms of the diarrheogenic effects of bile acids, we investigated the effects of the unconjugated bile acids chenodeoxycholate and ursodeoxycholate on the small intestine transport of electrolytes and nutrients in the rabbit jejunal and ileal mucosa mounted in vitro in Ussing or influx chambers. When added to the ileal mucosa at a concentration of 1 m M, both bile acids induced a secretory shift in ion transport; absorption of Na and Cl was abolished and secretion of bicarbonate was enhanced. No changes in short circuit current or elect

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3

Zsembery, Akos, Theresia Thalhammer, and Jürg Graf. "Bile Formation: a Concerted Action of Membrane Transporters in Hepatocytes and Cholangiocytes." Physiology 15, no. 1 (2000): 6–11. http://dx.doi.org/10.1152/physiologyonline.2000.15.1.6.

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A large number of membrane transport mechanisms in hepatocytes and cholangiocytes serves for the secretion of bile acids, various other organic anions, organic cations, lipids, and electrolytes. After their functional characterization, some of these mechanisms' individual transport molecules are now identified, allowing better understanding of inherited and acquired disorders of bile formation.

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Huang, Rang-Lang, Wen-Kai Huang, Xiang-Yi Xiao, Lin-Feng Ma, He-Zi-Rui Gu, and Guo-Ping Yang. "Diagnosis and treatment of post-cholecystectomy diarrhoea." World Journal of Gastrointestinal Surgery 15, no. 11 (2023): 2398–405. http://dx.doi.org/10.4240/wjgs.v15.i11.2398.

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The incidence of cholecystitis is relatively high in developed countries and may usually be attributed to gallstones, the treatment for which involves complete surgical removal of the gallbladder (cholecystectomy). Bile acids produced following cholecystectomy continue to flow into the duodenum but are poorly absorbed by the colon. Excessive bile acids in the colon stimulate mucosal secretion of water and electrolytes leading, in severe cases, to diarrhoea. Bile acid diarrhoea (BAD) is difficult to diagnose, requiring a comprehensive medical history and physical examination in combination with

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5

Khan, Fatir Dad, Motasim Billah, Sultan Zeb, Mohammad Usman, and Syed Aamer Hussain. "Bile duct injury during laparoscopic cholecystectomy." International journal of health sciences 7, S1 (2023): 460–67. http://dx.doi.org/10.53730/ijhs.v7ns1.14225.

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Background and Aim: Bile duct injuries (BDIs) are complex complications developed during laparoscopic cholecystectomy leading to the postoperative morbidity and mortality. Auxiliary bile duct injury, vascular structures injuries, portal vein, cyst duct leakage and damage to right hepatic artery are caused by bile duct injuries. The purpose of the present study was to investigate the incidence, clinical presentation, and treatment modality of bile duct injuries during laparoscopic cholecystectomy. Patients and Methods: This descriptive case series was conducted on 96 extra hepatic biliary injur

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Ammon, H. V., D. S. Cho, R. L. Loeffler, and K. L. Reetz. "Effects of taurodeoxycholate on in vivo water and solute transport in rat jejunum in absence and presence of calcium." American Journal of Physiology-Gastrointestinal and Liver Physiology 250, no. 2 (1986): G248—G251. http://dx.doi.org/10.1152/ajpgi.1986.250.2.g248.

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Bile acids and fatty acids enhance the permeability of brush-border membrane vesicles for calcium. It has been postulated that increased influx of calcium into the enterocyte might be responsible for the fluid secretion induced by dihydroxy bile acids and fatty acids. During in vivo perfusion studies of the rat jejunum, 15 mM taurodeoxycholate induced secretion of electrolytes and water (P less than 0.001), reduced glucose absorption (P less than 0.001), and enhanced the absorption of mannitol (P less than 0.0125) and calcium (P less than 0.001). Calcium absorption continued to be enhanced dur

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7

Rodriguez, Myrian R., Leandro R. Soria, María S. Ventimiglia, et al. "Endothelin-1 and -3 induce choleresis in the rat through ETB receptors coupled to nitric oxide and vagovagal reflexes." Clinical Science 125, no. 11 (2013): 521–32. http://dx.doi.org/10.1042/cs20120633.

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We have reported previously that centrally applied ET (endothelin)-1 and ET-3 induce either choleresis or cholestasis depending on the dose. In the present study, we sought to establish the role of these endothelins in the short-term peripheral regulation of bile secretion in the rat. Intravenously infused endothelins induced significant choleresis in a dose-dependent fashion, ET-1 being more potent than ET-3. Endothelins (with the exception of a higher dose of ET-1) did not affect BP (blood pressure), portal venous pressure or portal blood flow. ET-1 and ET-3 augmented the biliary excretion o

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Okolo, Charles, Thomas Wong, Mark W. Moody, and Toan D. Nguyen. "Effects of bile acids on dog pancreatic duct epithelial cell secretion and monolayer resistance." American Journal of Physiology-Gastrointestinal and Liver Physiology 283, no. 5 (2002): G1042—G1050. http://dx.doi.org/10.1152/ajpgi.00436.2001.

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Pancreatic duct epithelial cells (PDEC) mediate the secretion of fluid and electrolytes and are exposed to refluxed bile. In nontransformed cultured dog PDEC, which express many ion transport pathways of PDEC, 1 mM taurodeoxycholic acid (TDCA) stimulated an125I−efflux inhibited by DIDS and 5-nitro-2-(3-phenylpropylamino)benzoic acid (NPPB) and a86Rb+efflux inhibited by charybdotoxin. Inhibition by 1,2-bis(2-aminophenoxy)ethane- N,N,N′,N′-tetraacetic acid (BAPTA)-AM suggests mediation via increased intracellular Ca2+concentration, whereas the absence of lactate dehydrogenase release excludes ce

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Hegyi, Peter, Jozsef Maléth, Julian R. Walters, Alan F. Hofmann, and Stephen J. Keely. "Guts and Gall: Bile Acids in Regulation of Intestinal Epithelial Function in Health and Disease." Physiological Reviews 98, no. 4 (2018): 1983–2023. http://dx.doi.org/10.1152/physrev.00054.2017.

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Epithelial cells line the entire surface of the gastrointestinal tract and its accessory organs where they primarily function in transporting digestive enzymes, nutrients, electrolytes, and fluid to and from the luminal contents. At the same time, epithelial cells are responsible for forming a physical and biochemical barrier that prevents the entry into the body of harmful agents, such as bacteria and their toxins. Dysregulation of epithelial transport and barrier function is associated with the pathogenesis of a number of conditions throughout the intestine, such as inflammatory bowel diseas

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Smeets-Peeters, Marianne, Tim Watson, Mans Minekus, and Robert Havenaar. "A review of the physiology of the canine digestive tract related to the development ofin vitrosystems." Nutrition Research Reviews 11, no. 1 (1998): 45–69. http://dx.doi.org/10.1079/nrr19980005.

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AbstractFood and nutrition studies in animals and human beings often meet with technical difficulties and sometimes with ethical questions. An alternative to research in living animals is the dynamic multicompartmentalin vitromodel for the gastrointestinal tract described by Minekuset al.(1995) and Havenaar & Minekus (1996). The dynamic conditions that are simulated in this model are peristaltic movements, transit times, pH responses, secretion of enzymes and electrolytes and absorption of nutrients and water. To obtain data for anin vitromodel of the dog gastrointestinal tract, the litera

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Dissertations / Theses on the topic "Bile electrolytes"

1

GUO, JI-HONG, and 郭基宏. "Effects of bile salts on electrolyte balance in rats." Thesis, 1988. http://ndltd.ncl.edu.tw/handle/00572561572443257402.

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LIN, MING-ZHU, and 林明珠. "Effects of grass carp bile on electrolyte balance in rats." Thesis, 1987. http://ndltd.ncl.edu.tw/handle/99218088775915995657.

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Klouda, Jan. "Elektrochemická oxidace žlučových kyselin na elektrodách na bázi uhlíku. Možnosti využití v elektroanalýze." Master's thesis, 2015. http://www.nusl.cz/ntk/nusl-342917.

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The goal of this master's thesis was to examine the possibility of oxidation of seven selected bile acids and evaluate whether such processes are suitable for analytical purposes. The secondary goal was to describe the oxidation products of bile acid electrolysis. The experiments were carried out in a non-aqueous medium of acetonitrile and in a mixed medium of acetonitrile:water using linear sweep and cyclic voltammetry. The working electrode materials employed for voltammetric experiments were: highly oriented pyrolytic graphite, -cyclodextrin modified glassy carbon and boron doped diamond.

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Books on the topic "Bile electrolytes"

1

North Carolina. General Assembly. Legislative Committee on New Licensing Boards. Assessment report for amend electrolysis practice act/fees: House bill 1330, Senate bill 1009. Legislative Committee on New Licensing Boards, 2005.

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2

VanLancker, J. L. Molecular and Cellular Mechanisms in Disease : 1: Bioenergetics · Cell Specificity · Inborn Errors of Metabolism · Malnutrition · Calcium and Phosphorus Iron and Bile Pigments · Coagulopathies · Hormones Body Fluids and Electrolytes. Springer, 2011.

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VanLancker, J. L. Molecular and Cellular Mechanisms in Disease : 1: Bioenergetics · Cell Specificity · Inborn Errors of Metabolism · Malnutrition · Calcium and Phosphorus Iron and Bile Pigments · Coagulopathies · Hormones Body Fluids and Electrolytes. Springer London, Limited, 2012.

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4

Carle, Sarah. Cholecystitis and Cholangitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0034.

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Cholangitis is a bacterial infection of the biliary system that is commonly associated with mechanical obstruction of the cystic or common bile duct (CBD). Obstruction is usually caused by choledocholithiasis (which results from gallstone obstruction of the CBD) but may also be seen with biliary stricture, malignancy, or cyst. Bacterial proliferation may lead to gangrenous cholecystitis, gallbladder perforation (with potential for cholecystoenteric fistula creation), and/or sepsis. Patients should be managed based on clinical severity and symptoms with attention paid to volume and electrolyte

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Blaser, Annika Reintam, and Adam M. Deane. Normal physiology of the gastrointestinal system. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0172.

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The gastrointestinal (GI) system is responsible for digestion and absorption, but also has important endocrine, immune and barrier functions. Additionally, the GI system plays a major role in fluid, electrolyte and acid-base balance. The GI system is regulated by complex myogenic, neural and humoral mechanisms, and, in health, these are affected by the presence of luminal nutrient, thereby modulating function of the GI system. Accordingly, GI function varies depending on whether a person is fasted or in the postprandial state. Adequate fasting and postprandial perfusion, motility and exocrine

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Book chapters on the topic "Bile electrolytes"

1

Graf, J. "Hepatic Electrolyte Transport and Bile Formation." In Epithelial Secretion of Water and Electrolytes. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75033-5_22.

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2

Flanagan, Timothy F., and Florian Hackl. "Anatomy of the Gastrointestinal and Hepatic Systems." In Basic Anesthesia Review, edited by Alaa Abd-Elsayed. Oxford University PressNew York, 2024. http://dx.doi.org/10.1093/med/9780197584569.003.0292.

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Abstract The GI tract transports and absorbs water, electrolytes, and nutrients. Passage of materials is facilitated by peristaltic contractions of a smooth muscle layer and regulated by muscle sphincters. Liver anatomy, physiology, and pathophysiology are closely linked to the GI tract. Besides metabolic and synthetic functions, the liver is responsible for the production and secretion of bile into the duodenum via the common bile duct, facilitating fat absorption. The normal physiology, motility, and secretory activity of the GI tract can be altered by anesthetic drugs, and sympathetic and p

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3

"Digestive system." In Oxford Assess and Progress: Medical Sciences, edited by Jade Chow, John Patterson, Kathy Boursicot, and David Sales. Oxford University Press, 2012. http://dx.doi.org/10.1093/oso/9780199605071.003.0023.

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In essence, the digestive system is a four-layered tube that extends from mouth to anus. Its main purpose is the enzymatic digestion of food to produce smaller molecules that can then be absorbed into the body as nutrients. To achieve this the gut is regionally specialized to enable the serial processing of food and the absorption of food, water, and electrolytes as materials pass along the bowel. The four layers of the bowel are: 1) A mucosa surrounding the lumen, made up of a specialized epithelium, a lamina propria of connective tissue, and a layer of smooth muscle—the muscularis mucosae. 2

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4

"Digestive system." In Oxford Handbook of Medical Sciences, edited by Robert Wilkins, Ian Megson, and David Meredith. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198789895.003.0008.

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The chapter entitled ‘Digestive system’ opens with an overview of the anatomy of the abdomen, including the abdominal wall and peritoneal cavity, and the structure and histology of the major structures found therein, namely the intestinal tract (oesophagus, stomach, duodenum, jejunum, ileum, large intestine, rectum), spleen, liver, and pancreas. The functions of the gastrointestinal tract are described, covering motility, secretion (saliva, gastric acid, pancreatic fluid, bile, fluid, and electrolytes) and digestion/absorption of nutrients. Pathologies such as gastric and duodenal ulcers, mala

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