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Relevant bibliographies by topics / Bilirubin induced cardioprotection

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Academic literature on the topic 'Bilirubin induced cardioprotection'

Author: Grafiati

Published: 19 February 2023

Last updated: 31 July 2025

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Journal articles on the topic "Bilirubin induced cardioprotection"

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Ashfaq, Khurram, Shahbaz Ul Haq, Aila Tehreem, et al. "Hepatoprotective outcome of Silymarin against CCl4 induced hepatotoxicity in rabbit model." World Journal of Biology and Biotechnology 8, no. 2 (2023): 33. http://dx.doi.org/10.33865/wjb.008.02.1121.

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For centuries, medicinal plants have been used to treat human and animal ailments. Recent research indicated Silymarin as commonly employed antioxidant, hepatoprotective, cardioprotective, antiviral and antifibrotic. Anti-oxidant properties are believed to be responsible for its hepatoprotective effect. In this research, 46 rabbits were utilized in total. Each rabbit was separated into 04 groups: Group A (n=13) served as control and received neither Silymarin nor CCl4 treatment. Group B (n=13) was given 100 mg/Kg CCl4 as treatment. CCl4 and Silymarin were administered to Groups C and D (n=10),

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2

Henrrietta Ogadimma Nnadi and Vincent Ugochukwu Igbokwe. "Nutraceutical from Tetracapedium conophorum (Walnut) Protect against cadmium chloride induced hypertension in Albino rats." GSC Advanced Research and Reviews 10, no. 1 (2022): 031–41. http://dx.doi.org/10.30574/gscarr.2022.10.1.0021.

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The use of natural plant as food, nutraceuticals consumption, phytotherapy and nutritional therapy has recently become widely used in the treatment of diseases and shown to improve health. The study evaluated the hepatoprotective and cardioprotective effect of some nutraceuticals from phytosterol diet such as Tetracarpidiun conophorom on some parameter as found in albino rats. Cadmium chloride was used for the inducement of hypertension on the albino rats. Each study group was a total number of 10 rats in five different groups made up of the Control Group, Hypertensive Group, Standard group an

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3

Henrrietta, Ogadimma Nnadi, and Ugochukwu Igbokwe Vincent. "Nutraceutical from Tetracapedium conophorum (Walnut) Protect against cadmium chloride induced hypertension in Albino rats." GSC Advanced Research and Reviews 10, no. 1 (2022): 031–41. https://doi.org/10.5281/zenodo.5953933.

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The use of natural plant as food, nutraceuticals consumption, phytotherapy and nutritional therapy has recently become widely used in the treatment of diseases and shown to improve health. The study evaluated the hepatoprotective and cardioprotective effect of some nutraceuticals from phytosterol diet such as <em>Tetracarpidiun conophorom</em> on some parameter as found in albino rats. Cadmium chloride was used for the inducement of hypertension on the albino rats. Each study group was a total number of 10 rats in five different groups made up of the Control Group, Hypertensive Group

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4

Vulapalli, Sreesatya Raju, Zhongyi Chen, Balvin H. L. Chua, Tingchung Wang, and Chang-Seng Liang. "Cardioselective overexpression of HO-1 prevents I/R-induced cardiac dysfunction and apoptosis." American Journal of Physiology-Heart and Circulatory Physiology 283, no. 2 (2002): H688—H694. http://dx.doi.org/10.1152/ajpheart.00133.2002.

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Heme oxygenase (HO)-1 converts heme to bilirubin, carbon monoxide, and iron. Our prior work has suggested a cardioprotective role for HO-1 in heart failure. To test whether HO-1 (heat shock protein 32) prevents cardiomyocyte apoptosis and cardiac dysfunction after ischemia-reperfusion (I/R), we generated transgenic mice overexpressing HO-1 in the heart under the control of the α-myosin heavy chain promoter. HO-1 transcript and protein increased markedly in the heart only. In an isolated heart preparation, we observed an enhanced functional recovery during reperfusion after ischemia in the tran

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Qayyum, Muhammad Imran, Sami Ullah, Umer Rashid, et al. "Synthesis, Molecular Docking, and Preclinical Evaluation of a New Succinimide Derivative for Cardioprotective, Hepatoprotective and Lipid-Lowering Effects." Molecules 27, no. 19 (2022): 6199. http://dx.doi.org/10.3390/molecules27196199.

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Cardiac and hepatotoxicities are major concerns in the development of new drugs. Better alternatives to other treatments are being sought to protect these vital organs from the toxicities of these pharmaceuticals. In this regard, a preclinical study is designed to investigate the histopathological effects of a new succinimide derivative (Comp-1) on myocardial and liver tissues, and the biochemical effects on selected cardiac biomarkers, hepatic enzymes, and lipid profiles. For this, an initially lethal/toxic dose was determined, followed by a grouping of selected albino rats into five groups (

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Коваленя, Т. А., С. Н. Кирко, Е. Б. Белоновская, et al. "Protective Effect of the Flavonoid Naringin in Alcoholic Cardiomyopathy in Rats." Лабораторная диагностика. Восточная Европа 12, no. 4 (2023): 606–16. http://dx.doi.org/10.34883/pi.2023.12.4.012.

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Введение. Алкогольная кардиомиопатия, развивающаяся в результате длительного потребления алкоголя, представляет серьезную медико-социальную проблему. Цель. Оценить роль митохондриальной дисфункции в развитии алкогольной кардиомиопатии у крыс и выяснить возможный кардиопротекторный эффект флавоноида нарингина при алкогольной кардиомиопатии in vivo. Материалы и методы. Алкогольную интоксикацию крыс-самцов линии Вистар вызывали путем длительного введения этанола (8 недель, 4 г/кг массы) в/ж, нарингин вводили ежедневно в дозе 40,0 мг/кг массы животного. Структуру митохондрий кардиомиоцитов анализи

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7

Urade, Yoshihiro. "Biochemical and Structural Characteristics, Gene Regulation, Physiological, Pathological and Clinical Features of Lipocalin-Type Prostaglandin D2 Synthase as a Multifunctional Lipocalin." Frontiers in Physiology 12 (October 22, 2021). http://dx.doi.org/10.3389/fphys.2021.718002.

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Lipocalin-type prostaglandin (PG) D2 synthase (L-PGDS) catalyzes the isomerization of PGH2, a common precursor of the two series of PGs, to produce PGD2. PGD2 stimulates three distinct types of G protein-coupled receptors: (1) D type of prostanoid (DP) receptors involved in the regulation of sleep, pain, food intake, and others; (2) chemoattractant receptor-homologous molecule expressed on T helper type 2 cells (CRTH2) receptors, in myelination of peripheral nervous system, adipocyte differentiation, inhibition of hair follicle neogenesis, and others; and (3) F type of prostanoid (FP) receptor

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BAREJIA, ASIF, ACHARYA CR, AASHAL SHAH, ANKIT PATEL, and VIPUL NAVADIYA. "EXPLORATION OF CARDIOPROTECTIVE AND ANTIOXIDANT ACTION OF BILIRUBIN IN ISOPRENALINE-INDUCED ISCHEMIA MODEL OF MALE WISTAR ALBINO RATS." Asian Journal of Pharmaceutical and Clinical Research, February 7, 2023, 73–76. http://dx.doi.org/10.22159/ajpcr.2023.v16i2.46420.

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Objectives: Ischemic heart disease is one of the leading causes of death worldwide. Bilirubin, an endogenous product of heme catabolism, has remarkable antioxidant properties. This study was planned with an aim to evaluate cardioprotective potential of bilirubin in isoprenaline (ISO)- induced rat myocardial ischemia model. Methods: In this study, a total of 24 adult male Wistar albino rats (200–250 g) were divided into four groups of six rats each. Groups I, III, and IV, respectively, received distilled water (10 mL/kg bw), bilirubin (40 mg/kg bw), and bilirubin (60 mg/kg bw) intraperitoneally

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Li, Michael, Walter W. Chan, and Stephen D. Zucker. "Association Between Atazanavir‐Induced Hyperbilirubinemia and Cardiovascular Disease in Patients Infected with HIV." Journal of the American Heart Association 9, no. 19 (2020). http://dx.doi.org/10.1161/jaha.120.016310.

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BACKGROUND: Serum bilirubin is inversely associated with cardiovascular risk. Atazanavir, an HIV protease inhibitor that competitively inhibits bilirubin conjugation, provides a unique opportunity to examine whether selectively increasing bilirubin is cardioprotective. We sought to determine whether patients receiving atazanavir manifest a reduced risk of cardiovascular disease compared with those receiving darunavir, an HIV protease inhibitor that does not increase serum bilirubin. METHODS AND RESULTS: This was a retrospective cohort study of 1020 patients with HIV. The main outcome was time

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Vogel, Megan E., Gila Idelman, and Stephen D. Zucker. "Abstract 661: Bilirubin Prevents Atherosclerotic Plaque Formation in LDLR -/- Mice by Inhibiting Monocyte Migration Through the Disruption of Endothelial Vascular Cell Adhesion Molecule 1 (VCAM-1) and Intercellular Adhesion Molecule 1 (ICAM-1) Signaling." Arteriosclerosis, Thrombosis, and Vascular Biology 36, suppl_1 (2016). http://dx.doi.org/10.1161/atvb.36.suppl_1.661.

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Background: Epidemiologic studies support an inverse association between serum bilirubin levels and the risk of cardiovascular disease. Monocyte infiltration into the vascular endothelium, a critical early step in atherosclerotic plaque formation, is mediated primarily by the adhesion molecules VCAM-1 and ICAM-1. We postulate that bilirubin, a potent antioxidant, inhibits plaque formation by preventing monocyte transendothelial migration through the scavenging of VCAM-1 and ICAM-1 reactive oxygen species (ROS) signaling intermediaries. Methods: The effect of physiological concentrations (≤ 1.2

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Dissertations / Theses on the topic "Bilirubin induced cardioprotection"

1

Bakrania, Bhavisha. "Bilirubin Induced Cardioprotection: From Endogenous Protection to Therapeutic Potential." Thesis, Griffith University, 2015. http://hdl.handle.net/10072/367882.

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Over the past 30 years, knowledge concerning the in vivo relevance of bilirubin has evolved from being an inert waste product of haem catabolism to a physiologically important antioxidant and biomarker of cardiovascular disease (CVD). Elevated serum bilirubin concentrations, as observed in human Gilbert’s syndrome (GS), are associated with reduced incidence of atherosclerosis, ischaemic heart disease (IHD), and a reduction in overall cardiovascular mortality. However, a comprehensive understanding of the mechanisms that might explain these associations remains to be delineated. Aside

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