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Relevant bibliographies by topics / Bilirubin toxicity

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Academic literature on the topic 'Bilirubin toxicity'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Bilirubin toxicity"

1

Walker, Paul C. "Neonatal Bilirubin Toxicity." Clinical Pharmacokinetics 13, no. 1 (1987): 26–50. http://dx.doi.org/10.2165/00003088-198713010-00002.

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Hansen, Thor Willy Ruud. "Bilirubin Brain Toxicity." Journal of Perinatology 21, S1 (2001): S48—S51. http://dx.doi.org/10.1038/sj.jp.7210634.

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Cashore, William J., and Nai K. Leung. "Discussion: Toxicity of Bilirubin." Journal of Perinatology 21, S1 (2001): S59—S62. http://dx.doi.org/10.1038/sj.jp.7210637.

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HANSEN, T. W. R., and D. BRATLID. "Bilirubin and Brain Toxicity." Acta Paediatrica 75, no. 4 (1986): 513–22. http://dx.doi.org/10.1111/j.1651-2227.1986.tb10242.x.

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&NA;. "BILIRUBIN TOXICITY RESEARCH FUNDED." Advances in Neonatal Care 4, no. 1 (2004): 9. http://dx.doi.org/10.1016/j.adnc.2003.11.017.

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Amato, Maurizio. "Mechanisms of bilirubin toxicity." European Journal of Pediatrics 154, S4 (1995): S54—S59. http://dx.doi.org/10.1007/bf02191507.

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Oygür, Nihal, N. Gygür, D. Nuzumlali, A. Ersay, S. Velipasaojuvylu, and O. Yejuvyin. "Bilirubin toxicity: Outcome in infants with high bilirubin levels." European Journal of Pediatrics 155, no. 2 (1996): 145–46. http://dx.doi.org/10.1007/bf02075773.

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POLAND, RONALD L. "In Search of a `Gold Standard' for Bilirubin Toxicity." Pediatrics 89, no. 5 (1992): 823–24. http://dx.doi.org/10.1542/peds.89.5.823.

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In this issue of Pediatrics, Newman and Maisels1 have given us a thorough and thoughtful review of current knowledge about the toxicity of bilirubin in term, otherwise healthy, newborn infants and have concluded that there is little evidence that bilirubin toxicity can be predicted from the concentration of bilirubin in the serum. They propose that we worry about bilirubin less in this population and treat hyperbilirubinemia only if it reaches much higher concentrations than the traditional 20 mg/dL. Their conclusions and recommendations are very reasonable given that the serum bilirubin conce

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Hansen, Thor Willy Ruud. "Mechanisms of bilirubin toxicity: clinical implications." Clinics in Perinatology 29, no. 4 (2002): 765–78. http://dx.doi.org/10.1016/s0095-5108(02)00053-2.

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Rose, Jessica, and Rachel Vassar. "Movement disorders due to bilirubin toxicity." Seminars in Fetal and Neonatal Medicine 20, no. 1 (2015): 20–25. http://dx.doi.org/10.1016/j.siny.2014.11.002.

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Dissertations / Theses on the topic "Bilirubin toxicity"

1

Ives, N. K. "Bilirubin transport and toxicity in the central nervous system." Thesis, University of Cambridge, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604974.

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VIANELLO, ELEONORA. "In vivo evaluation of biomolecular mechanisms of bilirubin toxicity and pre-clinical therapies." Doctoral thesis, Università degli Studi di Trieste, 2017. http://hdl.handle.net/11368/2908155.

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Introduction. Neonatal jaundice is a common and benign condition usually resolved during the first week of life. Neonates with very high level of unconjugated bilirubin (UCB), with an increase of the free bilirubin (Bf), may develop encephalopathy, with different grade of severity. Typical symptoms include motor disorder, auditory dysfunction, memory and learning deficits, reflecting selective damage respectively for cerebellum and basal ganglia, inferior colliculus, and hippocampus. Several studies reported that bilirubin may activate signal cascades that culminate to cell survival or death,

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Haustein, Martin D. "Acute bilirubin toxicity causes hearing loss through presynaptic degeneration at a central glutamatergic synapse." Thesis, University of Leicester, 2011. http://hdl.handle.net/2381/10277.

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Severe neonatal jaundice causes bilirubin encephalopathy with complications such as ataxia, delayed neurodevelopment and deafness. The hair cells in the cochlea are spared suggesting central damage but the underlying mechanisms for this hearing loss are unknown. Utilising the Gunn rat model of hyperbilirubinemia I investigated the detrimental effect of high levels of bilirubin on the hearing system, using electrophysiological and imaging techniques combined with in vivo and in vitro studies of the auditory brainstem. My studies have focussed on synaptic transmission between the well characteri

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JAYANTI, SRI. "THE JANUS FACE OF BILIRUBIN: FROM NEONATAL TOXICITY TO THE POTENTIAL PROTECTION IN NEURODEGENERATIVE DISEASES." Doctoral thesis, Università degli Studi di Trieste, 2022. http://hdl.handle.net/11368/3030939.

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TASK 1: Bilirubin-induced neonatal neurotoxicity Unconjugated bilirubin is a metabolite of heme catabolism, and it exhibits Janus face for owning neurotoxicity and neuroprotective effects. It can induce neurotoxicity in neonates (kernicterus). It has been already used in animal models as well as in clinics in other diseases with relevant results. As a model of kernicterus, we used the Gunn rat. We observed the full protection given by Curc by preventing cerebellar hypoplasia by counteracting inflammation, redox imbalance, and glutamate neurotoxicity. The results suggest the potential of Curc i

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Weeramange, Chamitha Janani. "Inhibition of UDP-glucose dehydrogenase by 6-Thiopurine and its oxidative metabolites: possible mechanism for its interaction within the bilirubin excretion pathway and 6-Thiopurine associated toxicity." Diss., 2018. http://hdl.handle.net/2097/38874.

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Doctor of Philosophy<br>Department of Chemistry<br>Ryan Rafferty<br>6-Thiopurine (6TP), or 6-mercaptopurine, is an actively prescribed drug in the treatment of acute lymphocytic leukemia since 1952. Although 6TP has beneficial and promising therapeutic uses, severe toxicities are associated with its use such as jaundice and hepatotoxicity. These toxicities are due to the higher level of accumulation of bilirubin within the body. The bilirubin pathway involves the conjugation of water-insoluble bilirubin to two equivalents of UDPglucuronic acid (UDPGA) forming the water-soluble and excretable b

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Books on the topic "Bilirubin toxicity"

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Padmakumar, Anand D., and Mark C. Bellamy. Pathophysiology and causes of jaundice in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0192.

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Critically-ill patients develop jaundice for a variety of reasons. A good understanding of bilirubin metabolism can help the clinician to diagnose and treat jaundice. Intensive care unit (ICU) physicians commonly encounter elevated serum bilirubin in severely-ill patients, which can be associated with increased morbidity and mortality. A complex interaction of enzymatic pathways leads to safe excretion of bilirubin. This fine homeostasis is often disturbed and leads jaundice, which can be broadly classified into three main categories—prehepatic, hepatic, and post-hepatic. Common examples inclu

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Book chapters on the topic "Bilirubin toxicity"

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Maeda, Hiroshi, Masami Kimura, Ikuharu Sasaki, Yoshihiko Hirose, and Toshimitsu Konno. "Toxicity of Bilirubin and Detoxification by PEG-Bilirubin Oxidase Conjugate." In Poly(Ethylene Glycol) Chemistry. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4899-0703-5_11.

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Cashore, William J. "Bilirubin Metabolism and Toxicity in the Newborn." In Fetal and Neonatal Physiology. Elsevier, 2004. http://dx.doi.org/10.1016/b978-0-7216-9654-6.50124-7.

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"Pharmacology and therapeutics." In Oxford Handbook of Paediatrics 3e, edited by Robert C. Tasker, Carlo L. Acerini, Edward Holloway, Asma Shah, and Peter Lillitos. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198789888.003.0027.

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Medicines are an essential part of the treatment of children. They help to reduce morbidity and mortality. It is important to recognize, however, that all medicines have potential side effects and additionally medication errors can occur at any stage (prescription, dispensing, or administration). Due to physiological differences in relation to drug metabolism and the potential protein-displacing effect on bilirubin, neonates are at greater risk of drug toxicity. Drug therapy is an integral part of the management of both pain and sedation in children.

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Conference papers on the topic "Bilirubin toxicity"

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Piyaratne, Alisha, and Upeka Bandaranayake. "In silico Toxicity Analysis of Potential Wound Healing Drug Candidates to Assess Drug Suitability." In SLIIT INTERNATIONAL CONFERENCE ON ADVANCEMENTS IN SCIENCES AND HUMANITIES. Faculty of Humanities & Sciences, SLIIT, 2024. https://doi.org/10.54389/sseg9731.

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Drug development typically takes several years, but in-silico techniques, computational biology and chemical solutions can expedite toxicity analysis and other sections of the drug development process. Therefore, screening of potential drug candidates has become efficient. These techniques provide results quickly, cost-effectively, and without harming animals or humans. This research leverages bioinformatics to analyse the toxicity of chemical compounds in a potential wound-healing drug, specifically Bilirubin, Pinocembrin, and Resveratrol. Software such as Discovery Studios, Chem3D Pro, OpenB

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VELIKANOV, Vitaliy. "Effectiveness of the feed additive "AspiSorb" in the pathology of the digestive system in pigs." In Multifunctional adaptive fodder production 29 (77). Federal Williams Research Center of Forage Production and Agroecology, 2022. http://dx.doi.org/10.33814/mak-2022-29-77-188-195.

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The article presents the results of the scientific experiment on the study of a new sorbent feed additive "AspiSorb", the use of which will increase the effectiveness of veterinary measures in the prevention and treatment of the pigs’ digestive system diseases. It is shown that "AspiSorb" does not have chronic toxicity. It does not adversely affect the condition of healthy piglets, and the quality and safety of animal slaughter products. It helps to reduce the level of endogenous intoxication of the body and the rapid disappearance of signs of exsicosis, which is confirmed by the normalization

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Ali Mohammed, Imtithal. "Protective Role of Cranberry Extract Against Zovirax-Induced Spleen Dysfunction in Adult Female Wistar Rats." In XI. International Scientific Congress of Pure, Applied and Technological Sciences (MINAR Congress). Rimar Academy, 2024. https://doi.org/10.47832/minarcongress11-8.

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This research explored a possible protective function of antioxidants, such as cranberries, against zoviraxinduced spleen dysfunction in adult female Wistar rats. A collection of 24(adult female Wistar rats) was haphazardly appointed to four equal sets, each including six animals. They received the following treatment for (0–22–44) days per day. The first group, known as group (C), was obtained in (tap water), and served as a control. The second set (A1) obtained orally 150 mg/kg B.W. of cranberry alone. The third group (A2) received zovirax (450mg/kg B.W.) during the trial to elicit spleen to

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Reports on the topic "Bilirubin toxicity"

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Lawanprasert, Somsong, Chaiyo Chaichantipyuth, Supatra Srichairat, Nuansri Niwattisaiwong, and Laddawal Phivthong-ngam. Subchronic exposure of Pueraria mirifica in normal - and high cholesterol diet fed rats : influence on hepatic cytochrome P450, lipid profile and toxicity. Chulalongkorn University, 2004. https://doi.org/10.58837/chula.res.2004.28.

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Pueraria mirifica airy shaw and suvatabandhu, know locally as Kwao Keur, is a plant in family Leguminosae. In this study, effects of P.mirifica on hepatic cytochrome P450 (CYP), serum lipid profile and subchronic toxicity were investigated in male Wistar rats. Rats were randomly divided into four treatment groups as following: normal diet-fed group; normal diet-fed supplemented with P.mirifica group; high cholesterol diet-fed group; high cholesterol diet-fed supplemented with P.mirifica group. Each group comprised 10 rats. P.mirifica was administered orally at a dosage of 100 mg/kg/day for 90

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