Log in

Relevant bibliographies by topics / Biochemical toxicology

Contents

Journal articles
Dissertations / Theses
Books
Book chapters
Conference papers

Academic literature on the topic 'Biochemical toxicology'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Biochemical toxicology.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Biochemical toxicology"

1

Fry, Jeffrey R. "Book Review: Biochemical Toxicology." Alternatives to Laboratory Animals 14, no. 4 (June 1987): 397. http://dx.doi.org/10.1177/026119298701400418.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Vaidya, Vishal S., Kartik Shankar, Udayan M. Apte, Sharmilee P. Sawant, and Pallavi B. Limaye. "Introduction to Biochemical Toxicology." International Journal of Toxicology 20, no. 5 (September 2001): 331–33. http://dx.doi.org/10.1177/109158180102000511.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Bridges, James W. "Frontiers in biochemical toxicology." Trends in Pharmacological Sciences 6 (January 1985): S11—S15. http://dx.doi.org/10.1016/0165-6147(85)90235-4.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Wells, Peter G., and Louise M. Winn. "Biochemical Toxicology of Chemical Teratogenesis." Critical Reviews in Biochemistry and Molecular Biology 31, no. 1 (January 1996): 1–40. http://dx.doi.org/10.3109/10409239609110574.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Marzin, D. "Biochemical toxicology: A practical approach." Biochimie 70, no. 2 (February 1988): 299. http://dx.doi.org/10.1016/0300-9084(88)90081-8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Hodgson, Ernest. "Biochemical toxicology: A practical approach." Analytical Biochemistry 167, no. 1 (November 1987): 210–11. http://dx.doi.org/10.1016/0003-2697(87)90154-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Gibson, G. Gordon. "Biochemical toxicology: A practical approach." Trends in Pharmacological Sciences 8, no. 5 (May 1987): 195. http://dx.doi.org/10.1016/0165-6147(87)90169-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Chipman, J. K. "Biochemical toxicology — A practical approach." Clinica Chimica Acta 168, no. 1 (September 1987): 119–20. http://dx.doi.org/10.1016/0009-8981(87)90278-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Guengerich, F. Peter. "Life and Times in Biochemical Toxicology." International Journal of Toxicology 24, no. 1 (January 2005): 5–21. http://dx.doi.org/10.1080/10915810590918670.

Full text

Abstract:

The biochemical facets of toxicology have always had a major role in providing insight into mechanisms. Some of the history of the development of this area is summarized, including metabolism, enzymology, and the chemistry of reactive intermediates. Knowledge in these fields has had a major impact in the areas of drug metabolism and safety assessment, which are both critical steps in the development of pharmaceuticals and the rational use of commodity chemicals. The science of toxicology has developed considerably with input from other disciplines and today is poised to emerge as a predictive science with even more dramatic impact. The challenges ahead are considerable but there is renewed excitement in the potential of the field. As in the past, further advances in the field of toxicology will require the input of knowledge from many disciplines.

APA, Harvard, Vancouver, ISO, and other styles

10

Eisler, Ronald. "Aquatic Toxicology: Molecular, Biochemical, and Cellular Perspectives." Transactions of the American Fisheries Society 124, no. 5 (September 1, 1995): 786–87. http://dx.doi.org/10.1577/1548-8659-124.5.786.

Full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Biochemical toxicology"

1

Coleman, M. D. "The biochemical pharmacology and toxicology of anti-parasitic agents." Thesis, Aston University, 2004. http://publications.aston.ac.uk/21356/.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Liu, Yandi. "A study of the biochemical development and toxicology of the seed of Santalum spicatum." Thesis, Curtin University, 1997. http://hdl.handle.net/20.500.11937/2454.

Full text

Abstract:

The seed of Santalum spicatum is rich in a fixed oil (59% by weight), which is characterised by a high percentage of acetylenic, ethylenic ximenynic acid (35% of total fatty acids). A number of important aspects of the seed fixed oil, its composition in developing seeds, its triacylglycerols molecular species in the oil, the nutrition and toxicity of the oil feeding, and the possible bioactivity of ximenynic acid in mice were investigated.The identification of cis and trans isomers of ximenynic acid in the seed oil, and the metabolite of ximenynic acid in mouse liver lipid fractions were achieved using 2-amino-2-methyl-1-propanol to form 2-substituted 4,4-dimethyloxazoline derivatives, which were analysed by gas chromatography with mass spectrometric detection.Changes in proximate and fatty acid composition were investigated in developing seed collected weekly from about seven days after flowering to maturity. It was determined that moisture and carbohydrate contents decreased significantly during the development sequence, while fixed oil content increased from 0.3% to 50% (by weight) with seed development. A corresponding increase in the proportions of both oleic and ximenynic acids occurred suggesting a precursor/product relationship. Mature seed collected from different locations in Western Australia showed minor differences in characteristics and lipid composition, which may have been influenced by geographical origin and harvesting year of samples.The lipid components from the seed oil were separated using thin-layer chromatography and the individual triglyceride bands were characterised by high performance liquid chromatography and gas chromatography using flame ionisation and mass spectrometric detection after removal from the plate. The triximenynin (trisantalbin) band showed no other contaminating fatty acids and was obtained in a relatively pure state.A nutrition and toxicity study was performed by feeding a semi-synthetic diet containing sandalwood seed oil to a level of 15% of total energy content to a group of mice for one month and another group for two months. The most significant effect of sandalwood seed oil ingestion when compared with a standard lab diet (5% fat, by weight) and a canola oil-enriched diet (15% fat, by weight) was an apparent reduction in body weight gain, which may be the effect of ximenynic acid as a growth retardant. Serum aspartate aminotransferase levels were determined in the mice as an indicator of hepatotoxicity. These levels were higher in mice fed the sandalwood seed oil diet than those fed the standard lab diet, suggesting that ximenynic acid may affect liver-specific enzyme activity. Analysis of the total lipid fatty acids of various tissues and organs of mice showed only a low incorporation of ximenynic acid into the general tissues (0.3-3% by weight), and its absence in the brain.This study suggests a few health benefits from consumption of large quantities of sandalwood seed oil in the diet. These include a low lipid content in blood, heart, muscle, increase in the 16:1/16:0 and 18:1/18:0 ratios, production of increased levels of 18:1 (n-9) and docosahexaenoic acid, and decreased levels of arachidonic acid in certain tissues. There were no specific pathological, morphological or mortality changes observed in the mice.Sandalwood seed may be both a food and a medicine.

APA, Harvard, Vancouver, ISO, and other styles

3

Jackson, John B. "The biochemical toxicology of serum carboxylesterase in pigeons (Columba livia)." Thesis, University of Reading, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.240286.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Tormey, William Patrick. "The provision of biochemical investigations in forensic toxicology for coroners." Thesis, Ulster University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.646399.

Full text

Abstract:

Death certification plays a central role in health service planning therefore identification of specific causes of death is critical. The requirements of biochemical toxicology as set out in the Coroners Acts in Ireland, Northern Ireland and England and Wales will be parsed to facilitate the construction of a modern best practice template for coroners' toxicology. The Royal College of Pathologists (RCPath) provides published standard guidelines for pathologists reporting to coroners. Their adequacy will be critically evaluated to facilitate reform with the intention of maximising the accuracy of death certification. The roles of psychological factors, tobacco smoking, non steroidal anti-inflammatory drugs, and cannabis in cardiac death will be detailed. The potential for adverse drug reactions to prescription medication to cause death by misadventure will be explored. The role for the expert witness in the inquisitorial coroners system to improve the accuracy of the causes of death and thus the verdict will be explored. My experience has shown that misinterpretation of presence of cannabis in autopsy blood and urine samples is common and this underlines the need for true expert guidance for the coroner. The current practice in biochemical toxicology of screening blood, urine and vitreous humor will be critically evaluated and the necessity for a wide ranging screen of potential toxins as a contributor to the cause of death examined. The appropriate analytes on the screening menu will be determined by local cultural factors. Gas and liquid chromatography with mass spectrometry are the methods of choice. The interpretation of isopropanol, ethanol and ketones in post-mortem blood will be considered as will t~e role of alcohol in death. The apparent population exposure to poisons as reported by Poisons Information Services will be used to explore the dichotomy between the usually benign outcome of common poisons and the often lethal consequences of poisoning by prescription and illicit drugs. The aim of this research is to use the template of the current legal requirements and routine laboratory procedures to suggest reforms which will improve the analytical protocol and reporting of biochemical toxicology in the coronial system resulting in greater accuracy in delineating the causes of death The narrative of this thesis travels through the areas of the coroners acts especially in the Republic of Ireland and the United Kingdom where forensic biochemistry plays a role in the specification of the causes of deaths. There is a deconstruction of the place of doctors in the coronial system and in the new arrangements following the passage of the Coroners and Justice Act 2009 in England and Wales and the potential for change in the Coroners Bill in the Republic of Ireland which fell with the dissolution of the Dail in20ll. There is an examination of the autopsy guidelines issued by the RCPath and suggestions for change which have been published. There is an analysis of the place of cannabinoids in coroners ' cases and publications setting out the position are included. A series of recommendations are made regarding improvement in practice for the reporting of biochemical toxicology in the coronial system. Two cases where there appears to be potential misinterpretation of the toxicological evidence, which may result in the review of the causes of death, are detailed as relevant clinical examples. Some laboratory pitfalls in relation to alcohol analysis have been demonstrated and the consequences of a protocol free service have been detailed with a prescription for improvement and practical solutions to improve outcomes. The under-estimation of the impact of tobacco toxicity is also addressed as is the potential for error due to lack of appreciation of drug-drug interactions. The role of biochemistry in the post-mortem diagnosis of alcoholic and diabetic ketoacidosis is discussed. Multidisciplinary reviews of biochemical toxicology for the coroners' court are suggested as the best safeguard of accurate interpretation to assist coronial enquiry. Conclusions suggesting standard operating procedures for post-mortem scenarios are detailed where possible.

APA, Harvard, Vancouver, ISO, and other styles

5

Liu, Yandi. "A study of the biochemical development and toxicology of the seed of Santalum spicatum." Curtin University of Technology, School of Pharmacy, 1997. http://espace.library.curtin.edu.au:80/R/?func=dbin-jump-full&object_id=12031.

Full text

Abstract:

The seed of Santalum spicatum is rich in a fixed oil (59% by weight), which is characterised by a high percentage of acetylenic, ethylenic ximenynic acid (35% of total fatty acids). A number of important aspects of the seed fixed oil, its composition in developing seeds, its triacylglycerols molecular species in the oil, the nutrition and toxicity of the oil feeding, and the possible bioactivity of ximenynic acid in mice were investigated.The identification of cis and trans isomers of ximenynic acid in the seed oil, and the metabolite of ximenynic acid in mouse liver lipid fractions were achieved using 2-amino-2-methyl-1-propanol to form 2-substituted 4,4-dimethyloxazoline derivatives, which were analysed by gas chromatography with mass spectrometric detection.Changes in proximate and fatty acid composition were investigated in developing seed collected weekly from about seven days after flowering to maturity. It was determined that moisture and carbohydrate contents decreased significantly during the development sequence, while fixed oil content increased from 0.3% to 50% (by weight) with seed development. A corresponding increase in the proportions of both oleic and ximenynic acids occurred suggesting a precursor/product relationship. Mature seed collected from different locations in Western Australia showed minor differences in characteristics and lipid composition, which may have been influenced by geographical origin and harvesting year of samples.The lipid components from the seed oil were separated using thin-layer chromatography and the individual triglyceride bands were characterised by high performance liquid chromatography and gas chromatography using flame ionisation and mass spectrometric detection after removal from the plate. The triximenynin (trisantalbin) band showed no other contaminating fatty acids and was obtained in a relatively pure state.A ++
nutrition and toxicity study was performed by feeding a semi-synthetic diet containing sandalwood seed oil to a level of 15% of total energy content to a group of mice for one month and another group for two months. The most significant effect of sandalwood seed oil ingestion when compared with a standard lab diet (5% fat, by weight) and a canola oil-enriched diet (15% fat, by weight) was an apparent reduction in body weight gain, which may be the effect of ximenynic acid as a growth retardant. Serum aspartate aminotransferase levels were determined in the mice as an indicator of hepatotoxicity. These levels were higher in mice fed the sandalwood seed oil diet than those fed the standard lab diet, suggesting that ximenynic acid may affect liver-specific enzyme activity. Analysis of the total lipid fatty acids of various tissues and organs of mice showed only a low incorporation of ximenynic acid into the general tissues (0.3-3% by weight), and its absence in the brain.This study suggests a few health benefits from consumption of large quantities of sandalwood seed oil in the diet. These include a low lipid content in blood, heart, muscle, increase in the 16:1/16:0 and 18:1/18:0 ratios, production of increased levels of 18:1 (n-9) and docosahexaenoic acid, and decreased levels of arachidonic acid in certain tissues. There were no specific pathological, morphological or mortality changes observed in the mice.Sandalwood seed may be both a food and a medicine.

APA, Harvard, Vancouver, ISO, and other styles

6

Dahl, Ulrika. "Integrating biochemical and growth responses in ecotoxicological assays with copepods." Doctoral thesis, Stockholm University, Department of Applied Environmental Science (ITM), 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-8218.

Full text

Abstract:

The understanding of effects of chemical exposure in nature is lagging behind. Predictions of harmful effects of chemicals on aquatic organisms rely mainly on ecotoxicity tests. To improve the understanding of the biological linkage between the cellular and organismal responses to a chemical in an ecotoxicological test, the major aim of this doctoral thesis was to investigate the usefulness of two biochemical endpoints, contents of RNA and ecdysteroids, by incorporating them with life-history traits of copepods (Crustacea). To do so, the two methods needed to be established at our laboratory. Both biochemical methods are more commonly used in basic biological research, but I here present its usefulness in ecotoxicological testing. It was found that individual RNA content as a biochemical endpoint was significantly altered in the brackish water harpacticoid copepod Nitocra spinipes when exposed to the pesticide Lindane (paper IV) and low concentrations (0.16

APA, Harvard, Vancouver, ISO, and other styles

7

Bazara, Salem Mohammed. "Biochemical studies on the toxicity and carcinogenicity of PCBs and related compounds." Thesis, Brunel University, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.235935.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Shipp, Nancy Gillett. "Characterization of mitoxantrone cardiotoxicity in cultured heart cells." Diss., The University of Arizona, 1991. http://hdl.handle.net/10150/185453.

Full text

Abstract:

The use of the anthracenedione mitoxantrone as an antitumor agent is steadily increasing. While the toxicities associated with its use are significantly less than those observed following treatment with the widely used doxorubicin, mitoxantrone cardiotoxicity is clearly a substantial clinical problem. Current information on the mechanism by which mitoxantrone causes toxicity in heart tissue is limited. Thus, the goal of these studies was to describe a model system in which mitoxantrone cardiotoxicity can be studied, and begin to describe the mechanism by which mitoxantrone exerts its cardiotoxic effect. These experiments have shown that cultured neonatal rat heart cells are an effective model system for studying mitoxantrone-induced cytotoxicity and biochemical changes in heart tissue. Cultured heart cells develop dose- and time-dependent toxicity following a short exposure to near-pharmacologically achievable drug concentrations. Furthermore, histologic changes characteristic of this drug are also observed at the light and electron microscopic level. Initial experiments aimed at defining mitoxantrone mechanism of action showed that mitoxantrone likely does not stimulate a significant production of active oxygen species, or have a specific effect on mitochondrial function. However, there is evidence to support the possibility that mitoxantrone can form a reactive intermediate in vitro. These studies have shown that covalent binding of mitoxantrone to proteins can occur under certain conditions. Mitoxantrone toxicity is lowered with the addition of ICRF-187, a metal chelating agent. Protection is not due to inactivation of mitoxantrone, decreased mitoxantrone uptake, or a delayed increase in cytosolic calcium. Similar protection is observed against doxorubicin and the oxidized form of mitoxantrone, but not against the non-hydroxylated analog of mitoxantrone, ametantrone. Furthermore, in a cell-free system, mitoxantrone can form complexes with both copper (II) and iron (III). Mitoxantrone metal binding is reversible as ICRF-187 as well as other chelators can remove the metals from these complexes. These data suggests that metal chelation is involved in the enhancement of mitoxantrone toxicity in vitro.

APA, Harvard, Vancouver, ISO, and other styles

9

Subhahar, Michael. "Pharmacokinetics and pharmacodynamics of some NSAIDs in horses : a pharmacological, biochemical and forensic study." Thesis, University of Central Lancashire, 2013. http://clok.uclan.ac.uk/9244/.

Full text

Abstract:

Non-steroidal anti- inflammatory drugs (NSAIDs) have been in use for over 100 years to treat pain, exerting their analgesic effect by inhibiting prostaglandin (PG) synthesis via the COX pathway. Some of the NSAIDs have adverse side effects including ulceration of the stomach and cardiovascular events which are associated with bleeding. Search is still going on to find a safe NSAID. Two new coxib NSAIDs, namely celecoxib and etoricoxib have been developed and they exert marked beneficial effects in reducing pain in humans and other small animals with little or no side effects. No such study has been done on horses to see if they can tolerate the drug as an analgesic pain killer. This study was designed to investigate the effects of the two coxib NSAIDs, celecoxib and etoricoxib in six retired race horses to determine any adverse side effects of the drugs, the time course changes in their metabolism and elimination once administered orally in known physiological doses and the metabolites produced by each drug over time. The study employed well established clinical and biochemical techniques to measure blood-borne parameters and the metabolism of each drug. The results show that either etoricoxib or celecoxib had no adverse side effects on blood borne parameters and the stomach of the horses. Pharmacokinetic study following oral administration of 2 mg/kg b wt of either celecoxib or etoricoxib to the six race horses showed a Cmax of 1.15 ± 0.3 µg/ml, tmax, to be 4.09 ± 1.60 hr and a terminal half- life of 15.52 ± 1.99hr for celecoxib and a Cmax of 1.0± 0.09 µg/ml, tmax of 0.79 ± 0.1 hr and, terminal half- life of 11.51 ± 1.56 hr, respectively for etoricoxib. The results also show that each coxib is metabolized in the horse and both the parent drug and its metabolites are found in the urine, plasma and faeces. The results have also shown that even small traces of either drug or its metabolites can be measured in urine samples even 120 hours following oral administration. The main metabolites found in plasma, urine and faeces are hydroxyl celecoxib and carboxycelecoxib when celecoxib was administered orally to the 6 retired race horses. Similarly, hydroxymethyletoricoxib, carboxylic etoricoxib, hydroxymethyl-1-N-oxide metabolite of etoricoxib and hydroxymethyletoricoxib glucuronide were also found in plasma, urine and faeces following oral administration etoricoxib .to the animals. The results for either horse haeptocytes or camel liver show to some extend similar metabolites. In conclusion, the results show that both drugs have no adverse side effects in the horse and their metabolites are completely eliminated within 120 hours following oral administration.

APA, Harvard, Vancouver, ISO, and other styles

10

Linderoth, Maria. "Biochemical characterisation of landfill leachate toxicity in fish." Doctoral thesis, Stockholm : Department of Applied Environmental Science (ITM), Stockholm university, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:su:diva-951.

Full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Books on the topic "Biochemical toxicology"

1

Smart, Robert C., and Ernest Hodgson, eds. Molecular and Biochemical Toxicology. Hoboken, NJ, USA: John Wiley & Sons, Inc., 2008. http://dx.doi.org/10.1002/9780470285251.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Timbrell, John A. Principles of biochemical toxicology. 4th ed. New York: Informa Healthcare USA, 2008.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

1932-, Hodgson Ernest, and Levi Patricia E, eds. Introduction to biochemical toxicology. 2nd ed. Norwalk, Conn: Appleton & Lange, 1994.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

1932-, Hodgson Ernest, Bend John R, and Philpot Richard M, eds. Reviews in biochemical toxicology. New York: Elsevier, 1985.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

5

1932-, Hodgson Ernest, Bend John R, and Philpot Richard M, eds. Reviews in biochemical toxicology. New York: Elsevier, 1987.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

6

Timbrell, John A. Principles of biochemical toxicology. London: Taylor & Francis, 1985.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

7

1932-, Hodgson Ernest, Bend John R, and Philpot Richard M, eds. Reviews in biochemical toxicology. New York: Elsevier, 1988.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

8

1932-, Hodgson Ernest, and Smart Robert C. 1954-, eds. Introduction to biochemical toxicology. 3rd ed. New York: Wiley, 2001.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

9

David, Zakim, and Vessey Donald A, eds. Biochemical pharmacology and toxicology. New York: Wiley, 1985.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

10

1954-, Smart Robert C., and Hodgson Ernest 1932-, eds. Molecular and biochemical toxicology. 4th ed. Hoboken, N.J: John Wiley & Sons, 2008.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Book chapters on the topic "Biochemical toxicology"

1

Jeffery, E. H. "Biochemical Mechanisms of Aluminum Toxicity." In Toxicology of Metals, 139–61. Berlin, Heidelberg: Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79162-8_7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Smith, Susan Y., and Rana Samadfam. "Biochemical Markers of Bone Turnover." In Molecular and Integrative Toxicology, 175–201. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56192-9_5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Bondy, S. C. "Biochemical Contributions to Neurotoxicology." In Recent Advances in Nervous System Toxicology, 43–65. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-0887-4_3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Isom, Gary E., and Joseph L. Borowitz. "Biochemical mechanisms of cyanide toxicity." In Toxicology of Cyanides and Cyanogens, 70–81. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118628966.ch5.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Borràs, Miquel, Santiago Llacuna, Assumpta Górriz, and Jacint Nadal. "Hematological and Biochemical Parameters in Pollution-Exposed Mice." In Archives of Toxicology, 189–95. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-46856-8_17.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Galli, Corrado L., and Marina Marinovich. "In Vitro Biochemical Markers of Skin Toxicity." In Skin Pharmacology and Toxicology, 165–79. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4684-7902-7_9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Zhang, Zhiyong, and Baohong Zhang. "Biochemical and Physiological Toxicity of Nanoparticles in Plant." In Environmental Toxicology and Toxicogenomics, 225–39. New York, NY: Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1514-0_16.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Hedili, A., J. M. Warnet, M. Thevenin, C. Martin, M. Yacoub, and J. R. Claude. "Biochemical Investigation of Atractylis Gummifera L. Hepatotoxicyty in the Rat." In Archives of Toxicology, 312–15. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74117-3_58.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Lugassy, Daniel, and Lewis Nelson. "Postmortem pathological and biochemical diagnosis of cyanide poisoning." In Toxicology of Cyanides and Cyanogens, 268–75. Chichester, UK: John Wiley & Sons, Ltd, 2016. http://dx.doi.org/10.1002/9781118628966.ch19.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Wang, Dayong. "Endpoints for Assessing the Toxicity on Biochemical Processes." In Exposure Toxicology in Caenorhabditis elegans, 259–86. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-6129-0_9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Biochemical toxicology"

1

Sakhno, T. A., M. P. Semenenko, and V. N. Grin. "DYNAMICS OF BIOCHEMICAL INDICATORS OF BLOOD IN FRESH COWS ON THE BACKGROUND OF HEPATOPROTECTOR APPLICATION." In "International Scientific and Practical Conference" THEORY AND PRACTICE OF VETERINARY PHARMACY, ECOLOGY AND TOXICOLOGY IN AIC ", dedicated to the centenary of the Department of Pharmacology and Toxicology, SPbSUVM. FSBEI HE St. Petersburg SUVM, 2021. http://dx.doi.org/10.52419/3006-2021-2-211-212.

Full text

Abstract:

The influence of a new injectable hepatoprotective drug livasen in the prevention of hepatosis in highly productive fresh cows has been studied. The performed pharmacoprophylaxis has shown high efficiency in the correction of diseases of the hepatobiliary system and metabolic insufficiency.

APA, Harvard, Vancouver, ISO, and other styles

2

Soulsby, Michael, Emily Johnson, Nisreen Akel, Rakhi Agarwal, Dana Gaddy, Maxim Dobretsov, Parimal Chowdhury, and Olga Tarasenko. "PANCREATIC HISTOLOGY AND ASSOCIATED BIOCHEMICAL CHANGES IN RATS ON HIND-LIMB SUSPENSION." In BIOLOGY, NANOTECHNOLOGY, TOXICOLOGY, AND APPLICATIONS: Proceedings of the 5th BioNanoTox and Applications International Research Conference. AIP, 2011. http://dx.doi.org/10.1063/1.3587463.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!