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Books on the topic 'Biological Malaria'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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1

The mosquito war. New York: Tor, 2004.

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2

The mosquito war. New York: Forge, 2001.

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3

Nordin, M. Kepelbagaian biologi dan pemuliharaannya. Bangi: Penerbit Universiti Kebangsaan Malaysia, 1991.

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4

Eaton, John W. Malaria & the Red Cell (Progress in Clinical & Biological Resear). John Wiley & Sons Inc, 2000.

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5

Gregory, Bock, Cardew Gail, Novartis Foundation, and Symposium on Transport and Trafficking in the Malaria-Infected Erythrocyte (1999 : London, England), eds. Transport and trafficking in the malaria-infected erythrocyte. Chichester: John Wiley, 1999.

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6

Transport and Trafficking in the Malaria-Infected Erythrocyte - No. 226. John Wiley & Sons, 2000.

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7

Pombi, Marco, David Modiano, and Gilberto Corbellini. Malaria eradication in Italy: the story of a first success. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789833.003.0013.

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The Italian experience represents a historical example that education and sociopolitical vision can effectively contribute to the conquest of malaria. Until the end of 19th century, the infection spread mostly in the Central and Southern parts of Italy, with about 7 percent of the population affected and 20,000 annual deaths. Malaria showed different facies, owing to a complex vectorial system with remarkable ecological and behavioral differences. With the involvement of a critical mass of scientists and physicians, the etiology of malaria and the biological and socioeconomics determinants of the transmission dynamics were identified. This provided the opportunity to break the “malaria transmission chain” by an integrated approach. Moreover, Italy was the first country to develop special legislation for the fight against malaria, representing an example of integration between scientific, political, social and economic knowledge. In 1970, after decades of fight, the World Health Organization officially declared Italy free from malaria.

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8

Malaria and the red cell 2: Proceedings of the Second Workshop on Malaria and the Red Cell, held in Ann Arbor, Michigan, October 24, 1988 (Progress in clinical and biological research). A.R. Liss, 1989.

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9

MacAlister, V. A. The Mosquito War. Tor Books, 2004.

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10

François, Boller, and Fondation IPSEN pour la recherche thérapeutíque., eds. Biological markers of Alzheimer's disease. Berlin: Springer-Verlag, 1989.

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11

Ebmeier, K. P. Spect in Dementia (Advances in Biological Psychiatry). S. Karger Publishers (USA), 2003.

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12

Jha, Vivekanand. Acute kidney injury in the tropics. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0241.

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The spectrum of acute kidney injury (AKI) encountered in the hospitals of the tropical zone countries is different from that seen in the non-tropical climate countries, most of which are high-income countries. The difference is explained in large part by the influence of environment on the epidemiology of human disease. The key features of geographic regions falling in the tropical zones are climatic, that is, high temperatures and absence of winter frost, and economic, that is, lower levels of income. The causes and presentation of tropical AKI reflect these prevailing cultural, socioeconomic, climatic, and eco-biological characteristics.Peculiarities of tropical climate support the propagation of several infectious organisms that can cause AKI and the disease-transmitting vectors. In contrast to the developed world, where AKI usually develops in already hospitalized patients with multiorgan problems and iatrogenic factors play a major role, tropical AKI is acquired in the community due to issues of public health importance such as safe water, sanitation, infection control, and good obstetric practices. Infections such as malaria, leptospirosis, typhus, HIV, and diarrhoeal diseases; envenomation by animals or insects; ingestion of toxic herbs or chemicals; intravascular haemolysis; poisoning; and obstetric complications form the bulk of AKI in the tropics. Poor access to modern medical facilities and practices such as seeking treatment from traditional faith-healers contribute to poor outcomes.AKI extracts macro- and microeconomic costs from the affected population and reduces productivity. Improvement in the outcomes of tropical AKI requires improvement in basic public health through effective interventions, and accessibility to effective medical care.

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13

International Conference on Rabies in Eu. Rabies in Europe: First International Conference Kiev, Ukraine, 15-18 June, 2005 (Developments in Biologicals). S. Karger AG (Switzerland), 2006.

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14

Rota, Paul A., and William J. Bellini. Zoonotic paramyxoviruses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0047.

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Hendra virus (HeV), Nipah virus (NiV), and Menangle virus (MenV) are recently emergent paramyxoviruses that are responsible for zoonotic infections and represent potential threats to agriculture and humans. In particular, HeV and NiV cause fatal disease in animals and man, and outbreaks of NiV continue to occur almost annually in Southeast Asia. Molecular biologic studies have made substantial contributions to the characterization of these new paramyxoviruses by providing an accurate picture of their relative taxonomic positions, and molecular techniques were used to provide rapid diagnostic capabilities. In the outbreaks of NiV in Malaysia, Bangladesh, and India, molecular biological data quickly identified the etiologic agent present, and RT-PCR and serologic assays were used to rapidly confirm NiV infections in humans and animals. There has only been one report of human illness due to MenV and one study has detected an antibody response to a related rubulavirus, Tioman virus (TiV), in humans. It is interesting that all of these viruses share a common reservoir in large fruit bats. Because of their clear potential to cause severe disease in humans and animals, NiV and HeV have been designated as Class C Select Agents and have been the focus of intense study since their emergence.

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15

Iqbal, Khalid, and Henryk M. Wisniewski. Alzheimer's Disease and Related Disorders: Proceedings of the First International Conference on Alzheimer's Disease and Related Disorders, Held in L (Progress in Clinical and Biological Research). Wiley-Liss, 1990.

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16

Kiple, Kenneth F. Biology and African Slavery. Edited by Mark M. Smith and Robert L. Paquette. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780199227990.013.0014.

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This article reviews scholarship on the biology of African slaves. Mother Africa ensured that her sons and daughters could tolerate a disease environment sufficiently harsh that it served as a barrier to European outsiders for many centuries, keeping them confined to the coast and, save for some notable exceptions, away from the interior. Falciparum malaria and yellow fever, however, the chief ramparts in this barrier, did not remain confined to Africa. Rather, they reached the Americas with the Atlantic slave trade to rage among non-immune white and red people alike. But they largely spared blacks who were relatively resistant to these African illnesses, as well as to the bulk of those Eurasian diseases whose ravages were mostly directed at indigenous peoples. The sum of these pathogenic susceptibilities and immunities added up to the elimination of the latter (and white indentured servants) as contenders for tropical plantation labourers, and placed that onus squarely on the shoulders of the Africans. Yet, such a nomination in an age of rationalism bore with it the notion that black people, because of their ability to resist fevers, were sufficiently different biologically from Europeans as to constitute a separate branch of humankind and a lower one at that.

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17

P, Dostert, Erbamont Inc, and Fondazione Carlo Erba, eds. Early markers in Parkinson's and Alzheimer's diseases. Wien: Springer-Verlag, 1990.

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18

(Editor), Reinhard Marre, Yousef Abu Kwaik (Editor), Christopher Bartlett (Editor), Nicholas P. Cianciotto (Editor), Barry S. Fields (Editor), Matthias Frosch (Editor), Jorg Hacker (Editor), and Paul Christian Luck (Editor), eds. Legionella. ASM Press, 2001.

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19

(Editor), Adrian Barbul, Michael D. Caldwell (Editor), and David Marshall (Editor), eds. Clinical and Experimental Approaches to Dermal and Epidermal Repair: Normal and Chronic Wounds: Proceedings of the Third International Symposium on Ti (Progress in Clinical & Biological Research). Wiley-Liss, 1991.

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20

F, Schwartz Myrna, ed. Modular deficits in Alzheimer-type dementia. Cambridge, Mass: MIT Press, 1990.

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21

(Editor), James P. Nataro, Martin J. Blaser (Editor), and Susanna Cunningham-Rundles (Editor), eds. Persistent Bacterial Infections. ASM Press, 2000.

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