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Journal articles on the topic 'BIOLOGICAL STRESS'

Author: Grafiati
Published: 4 June 2021
Last updated: 27 April 2022

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1

Straumanis, John J. "Everyday Biological Stress Mechanisms." Journal of Clinical Psychiatry 64, no. 3 (March 15, 2003): 344–45. http://dx.doi.org/10.4088/jcp.v64n0318b.

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2

Simm, Andreas, and Lars-Oliver Klotz. "Stress and biological aging." Zeitschrift für Gerontologie und Geriatrie 48, no. 6 (July 24, 2015): 505–10. http://dx.doi.org/10.1007/s00391-015-0928-6.

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3

Gupta, Sunjai. "Stress—Conceptual and biological aspects." Behaviour Research and Therapy 35, no. 9 (September 1997): 887. http://dx.doi.org/10.1016/s0005-7967(97)84647-5.

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4

Dishman, Rod K. "Biological Psychology, Exercise, and Stress." Quest 46, no. 1 (February 1994): 28–59. http://dx.doi.org/10.1080/00336297.1994.10484109.

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5

Ackman, R. G. "Methods in Biological Oxidative Stress." Trends in Food Science & Technology 15, no. 1 (January 2004): 46. http://dx.doi.org/10.1016/j.tifs.2003.09.001.

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6

Pritchard, John B. "Comparative models and biological stress." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 283, no. 4 (October 1, 2002): R807—R809. http://dx.doi.org/10.1152/ajpregu.00415.2002.

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7

Eysenck, H. J. "Stress: Conceptual and biological aspects." Personality and Individual Differences 20, no. 6 (June 1996): 810–11. http://dx.doi.org/10.1016/0191-8869(96)83457-x.

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8

Mathur, R., J. Behari, and K. N. Sharma. "Biological responses of audiogenic stress." International Journal of Biometeorology 30, no. 4 (December 1986): 315–21. http://dx.doi.org/10.1007/bf02189368.

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9

Bryant, Richard A. "Acute Stress Reactions: Can Biological Responses Predict Posttraumatic Stress Disorder?" CNS Spectrums 8, no. 9 (September 2003): 668–74. http://dx.doi.org/10.1017/s1092852900008853.

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ABSTRACTWhat biological responses characterize those acute trauma reactions that develop into chronic psychiatric disorder? The need to understand the genesis of posttraumatic psychological disorders has resulted in much attention on biological reactions in the initial aftermath of trauma exposure. This review outlines the prevailing biological models of acute stress reaction and critiques the available evidence concerning biological responses to trauma that are associated with subsequent psychological disorder. The roles of peritraumatic dissociation and vulnerability factors for acute stress reaction are also reviewed. The major challenges for research on psychobiological responses to trauma are highlighted.
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10

Van Den Heuvel, Michael R. "Biological Indicators of Aquatic Ecosystem Stress." Transactions of the American Fisheries Society 133, no. 2 (March 2004): 492. http://dx.doi.org/10.1577/1548-8659(2004)133<0492a:bioaes>2.0.co;2.

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11

Reinert, Robert. "Biological Indicators of Stress in Fish." Transactions of the American Fisheries Society 121, no. 2 (March 1, 1992): 274–76. http://dx.doi.org/10.1577/1548-8659-121.2.274.

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12

Haddy, Richard I., and Richard D. Clover. "The biological processes in psychological stress." Families, Systems, & Health 19, no. 3 (2001): 291–302. http://dx.doi.org/10.1037/h0089453.

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13

Yettram, A. L. "Stress Analysis: Application to biological structures." Physics Bulletin 36, no. 7 (July 1985): 285. http://dx.doi.org/10.1088/0031-9112/36/7/013.

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14

Gregson, Olga, and Terry Looker. "The biological basis of stress management." British Journal of Guidance & Counselling 22, no. 1 (January 1994): 13–26. http://dx.doi.org/10.1080/03069889408253662.

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15

Henry, JP. "Biological Basis of the Stress Response." Physiology 8, no. 2 (April 1, 1993): 69–73. http://dx.doi.org/10.1152/physiologyonline.1993.8.2.69.

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Recent work shows that differing perceptions of stress result in different patterns of neuroendocrine activation. An easily handled challenge elicits norepinephrine and testosterone rises with success. With increasing anxiety, active coping shifts to a more passive mode. Epinephrine, prolactin, renin, and fatty acids increase. As the distress grows, cortisol augments.
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16

Henry, James P. "Biological basis of the stress response." Integrative Physiological and Behavioral Science 27, no. 1 (January 1992): 66–83. http://dx.doi.org/10.1007/bf02691093.

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17

Sies, H. "Biological Redox Systems and Oxidative Stress." Cellular and Molecular Life Sciences 64, no. 17 (June 14, 2007): 2181–88. http://dx.doi.org/10.1007/s00018-007-7230-8.

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18

McKeever, Victoria M., and Maureen E. Huff. "A Diathesis-Stress Model of Posttraumatic Stress Disorder: Ecological, Biological, and Residual Stress Pathways." Review of General Psychology 7, no. 3 (September 2003): 237–50. http://dx.doi.org/10.1037/1089-2680.7.3.237.

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The symptoms captured within the contemporary diagnostic definition of posttraumatic stress disorder (PTSD) have been studied for more than 100 years. Yet, even with increasingly advanced discoveries regarding the etiology of PTSD, a comprehensive and up-to-date etiological model that incorporates both medical and psychological research has not been described and systematically studied. The diathesis-stress model proposed here consolidates existing medical and psychological research data on etiological factors associated with PTSD into 3 causal pathways: residual stress, ecological, and biological. In combination, these pathways illuminate how PTSD might develop and who might be at higher risk for developing the disorder. Research and treatment implications related to the diathesis-stress model are discussed.
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19

FEDER, M. E., and J. C. WALSER. "The biological limitations of transcriptomics in elucidating stress and stress responses." Journal of Evolutionary Biology 18, no. 4 (July 2005): 901–10. http://dx.doi.org/10.1111/j.1420-9101.2005.00921.x.

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20

Kültz, Dietmar. "Defining biological stress and stress responses based on principles of physics." Journal of Experimental Zoology Part A: Ecological and Integrative Physiology 333, no. 6 (January 5, 2020): 350–58. http://dx.doi.org/10.1002/jez.2340.

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21

Robinson, Alexandra M. "Let's Talk about Stress: History of Stress Research." Review of General Psychology 22, no. 3 (September 2018): 334–42. http://dx.doi.org/10.1037/gpr0000137.

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The reference to stress is ubiquitous in modern society, yet it is a relatively new field of research. The following article provides an overview of the history of stress research and its iterations over the last century. In this article, I provide an overview of the earliest stress research and theories introduced through physiology and medicine and eventually as a concept in psychology. I begin with an exploration of the research of biological stressors 1st explored by experimental physiologist Claude Bernard and eventually adopted as a foundational concept in stress research when Walter Cannon expanded on Bernard's work and identified homeostasis. The contributions of Hans Selye, considered the father of stress research; Sir William Osler; Yerkes and Dodson; and Richard Lazarus are also discussed. Finally, I discuss how, in the new millennium, research on psychological stress has expanded across disciplines ranging from physiology to medicine, chemistry, endocrinology, neurosciences, epidemiology, psychiatry, epigenetics, and psychology, reflecting the complexity of the construct both theoretically and biologically.
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22

HIRAI, ITARU. "Work of stress protein in biological protection." RADIOISOTOPES 46, no. 5 (1997): 325–26. http://dx.doi.org/10.3769/radioisotopes.46.325.

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23

Gruenewald, Tara L., and Teresa E. Seeman. "Stress and Aging: A Biological Double Jeopardy?" Annual Review of Gerontology and Geriatrics 30, no. 1 (November 1, 2010): 155–77. http://dx.doi.org/10.1891/0198-8794.30.155.

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24

Danese, A. "Biological embedding of child stress through inflammation." Journal of Psychosomatic Research 74, no. 6 (June 2013): 543–44. http://dx.doi.org/10.1016/j.jpsychores.2013.03.030.

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25

BREMNER, J. DOUGLAS, and ERIC VERMETTEN. "Stress and development: Behavioral and biological consequences." Development and Psychopathology 13, no. 3 (September 2001): 473–89. http://dx.doi.org/10.1017/s0954579401003042.

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Childhood abuse is an important public health problem; however, little is known about the effects of abuse on the brain and neurobiological development. This article reviews the behavioral and biological consequences of childhood abuse and places them in a developmental context. Animal studies show that both positive and negative events early in life can influence neurobiological development in unique ways. Early stressors such as maternal separation result in lasting effects on stress-responsive neurobiological systems, including the hypothalamic–pituitary–adrenal (HPA) axis and noradrenergic systems. These studies also implicate a brain area involved in learning and memory, the hippocampus, in the long-term consequences of early stress. Clinical studies of patients with a history of abuse also implicate dysfunction in the HPA axis and the noradrenergic and hippocampal systems; however, there are multiple questions related to chronicity of stress, developmental epoch at the time of the stressor, presence of stress-related psychiatric disorders including posttraumatic stress disorder and depression, and psychological factors mediating the response to trauma that need to be addressed in this field of research. Understanding the effects of abuse on the development of the brain and neurobiology will nevertheless have important treatment and policy implications.
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26

Leonard, Brian. "A biological linkbetween stress, depression and cancer?" Cancer Nursing Practice 3, no. 5 (June 2004): 17–19. http://dx.doi.org/10.7748/cnp.3.5.17.s14.

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27

Matumoto, Masayasu, and Masatsugu Hori. "Stress Proteins: Biological Function and Clinical Significance." Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 35, no. 7 (1998): 521–29. http://dx.doi.org/10.3143/geriatrics.35.521.

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28

Martel, Jan, David M. Ojcius, Yun-Fei Ko, and John D. Young. "Phytochemicals as Prebiotics and Biological Stress Inducers." Trends in Biochemical Sciences 45, no. 6 (June 2020): 462–71. http://dx.doi.org/10.1016/j.tibs.2020.02.008.

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29

KAHN, J. P., C. MICHAUD, N. DE TALANCE, M. LAXENAIRE, M. MEJEAN, and C. BURLET. "EMOTIONAL AND BIOLOGICAL RESPONSES TO EXAMINATION STRESS." Clinical Neuropharmacology 15 (1992): 123B. http://dx.doi.org/10.1097/00002826-199202001-00237.

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30

Popper, Arthur N., Andrew S. Kane, and Michael S. Smith. "Biological responses to acoustical stress in fishes." Journal of the Acoustical Society of America 112, no. 5 (November 2002): 2432. http://dx.doi.org/10.1121/1.4779983.

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31

Bergant, Anton M., Harald Kirchler, Kurt Heim, Günther Daxenbichler, Manfred Herold, and Hans Schröcksnadel. "Childbirth as a Biological Model for Stress?" Gynecologic and Obstetric Investigation 45, no. 3 (1998): 181–85. http://dx.doi.org/10.1159/000009952.

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32

PARAMANYA, Additiya. "ROLE OF OXIDATIVE STRESS IN BIOLOGICAL SYSTEMS." Middle East Journal of Science 5, no. 2 (December 29, 2019): 155–62. http://dx.doi.org/10.23884/mejs.2019.5.2.07.

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33

Pitman, Roger K., Ann M. Rasmusson, Karestan C. Koenen, Lisa M. Shin, Scott P. Orr, Mark W. Gilbertson, Mohammed R. Milad, and Israel Liberzon. "Biological studies of post-traumatic stress disorder." Nature Reviews Neuroscience 13, no. 11 (October 10, 2012): 769–87. http://dx.doi.org/10.1038/nrn3339.

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34

Ursano, Robert J. "Trauma, Posttraumatic Stress Disorder, and Biological Processes." Psychosomatic Medicine 74, no. 2 (2012): 118–19. http://dx.doi.org/10.1097/psy.0b013e318249c50a.

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35

Theorell, T�res. "Biological stress markers and misconceptions about them." Stress and Health 19, no. 2 (2003): 59–60. http://dx.doi.org/10.1002/smi.960.

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36

Vattem, Dhiraj Anil, Sarah Neely, Nick Swift, Deana Townsend, Leanna McMillin, Brandon Jamison, Vatsala Maitin, C. Reed Richardson, Ignacio Cisneros, and Sandra Duesler. "Silicates: Novel Modulators of Biological Stress Response?" Free Radical Biology and Medicine 49 (January 2010): S131. http://dx.doi.org/10.1016/j.freeradbiomed.2010.10.359.

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37

Rybtsov, Stanislav, and Tatiana Berezina. "3128 – THE RETIREMENT STRESS INCREASES BIOLOGICAL AGE: SEARCHING STRESS-INDUCED INFLAMMATORY AND IMMUNOSENESCENCE FACTORS OF BIOLOGICAL AGING ACCELERATION." Experimental Hematology 88 (August 2020): S78. http://dx.doi.org/10.1016/j.exphem.2020.09.137.

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38

Daskalakis, Nikolaos P., Allison C. Provost, Richard G. Hunter, and Guia Guffanti. "Noncoding RNAs: Stress, Glucocorticoids, and Posttraumatic Stress Disorder." Biological Psychiatry 83, no. 10 (May 2018): 849–65. http://dx.doi.org/10.1016/j.biopsych.2018.01.009.

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39

Mondal, Tapan Kumar, Rebecca T. Emeny, Donghong Gao, Jeffrey G. Ault, Jane Kasten-Jolly, and David A. Lawrence. "A physical/psychological and biological stress combine to enhance endoplasmic reticulum stress." Toxicology and Applied Pharmacology 289, no. 2 (December 2015): 313–22. http://dx.doi.org/10.1016/j.taap.2015.09.013.

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40

Aston-Jones, G. "Brain noradrenergic neurons, stress and post-traumatic stress disorder." Biological Psychiatry 35, no. 9 (May 1994): 709. http://dx.doi.org/10.1016/0006-3223(94)90995-4.

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41

Andrews, Clare, Daniel Nettle, Maria Larriva, Robert Gillespie, Sophie Reichert, Ben O. Brilot, Thomas Bedford, Pat Monaghan, Karen A. Spencer, and Melissa Bateson. "A marker of biological age explains individual variation in the strength of the adult stress response." Royal Society Open Science 4, no. 9 (September 2017): 171208. http://dx.doi.org/10.1098/rsos.171208.

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The acute stress response functions to prioritize behavioural and physiological processes that maximize survival in the face of immediate threat. There is variation between individuals in the strength of the adult stress response that is of interest in both evolutionary biology and medicine. Age is an established source of this variation—stress responsiveness diminishes with increasing age in a range of species—but unexplained variation remains. Since individuals of the same chronological age may differ markedly in their pace of biological ageing, we asked whether biological age—measured here via erythrocyte telomere length—predicts variation in stress responsiveness in adult animals of the same chronological age. We studied two cohorts of European starlings in which we had previously manipulated the rate of biological ageing by experimentally altering the competition experienced by chicks in the fortnight following hatching. We predicted that individuals with greater developmental telomere attrition, and hence greater biological age, would show an attenuated corticosterone (CORT) response to an acute stressor when tested as adults. In both cohorts, we found that birds with greater developmental telomere attrition had lower peak CORT levels and a more negative change in CORT levels between 15 and 30 min following stress exposure. Our results, therefore, provide strong evidence that a measure of biological age explains individual variation in stress responsiveness: birds that were biologically older were less stress responsive. Our results provide a novel explanation for the phenomenon of developmental programming of the stress response: observed changes in stress physiology as a result of exposure to early-life adversity may reflect changes in ageing.
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42

Chrsitian, Sarbach, and Postaire Eric. "Biological markers of oxidative stress in exhaled air." Archives of Pharmacy and Pharmaceutical Sciences 4, no. 1 (January 31, 2020): 010–12. http://dx.doi.org/10.29328/journal.apps.1001021.

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43

GENG, Liuna, Xue WANG, Peng XIANG, and Jin YANG. "Hair Cortisol: A Biological Marker of Chronic Stress." Advances in Psychological Science 23, no. 10 (2015): 1799. http://dx.doi.org/10.3724/sp.j.1042.2015.01799.

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44

Motamedzad, Majid, and Mansour R. Azari . "Heat Stress Evaluation Using Environmental and Biological Monitoring." Pakistan Journal of Biological Sciences 9, no. 3 (January 15, 2006): 457–59. http://dx.doi.org/10.3923/pjbs.2006.457.459.

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45

KOHNO, Masahiro, and Akitane MORI. "Biological approach for the stress of magnetic fields." Okayama Igakkai Zasshi (Journal of Okayama Medical Association) 106, no. 9-10 (1994): 931–38. http://dx.doi.org/10.4044/joma1947.106.9-10_931.

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46

CICCHETTI, DANTE, and ELAINE F. WALKER. "Editorial: Stress and development: Biological and psychological consequences." Development and Psychopathology 13, no. 3 (September 2001): 413–18. http://dx.doi.org/10.1017/s0954579401003017.

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This Special Issue of Development and Psychopathology is devoted to the psychological and biological consequences of stress across the developmental course. Contributions in this Special Issue address topics that are central to elucidating the impact that stress exerts on developmental outcomes. These issues are investigated through examining a diverse array of populations, including rodent and nonhuman primate samples, as well as cohorts of maltreated children and adolescents with and without posttraumatic stress disorder (PTSD), children who were adopted from Romanian orphanages at differing points during infancy, aging Holocaust survivors and their offspring, children with depressive disorder, adolescents with schizotypal personality disorder, and adults with bipolar and unipolar mood disorders.
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47

Lecrubier, Y. "The biological, functional and personal consequences of stress." European Psychiatry 20, S3 (October 2005): S301. http://dx.doi.org/10.1016/s0924-9338(05)80179-8.

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48

Squier, Thomas C. "Oxidative stress and protein aggregation during biological aging." Experimental Gerontology 36, no. 9 (September 2001): 1539–50. http://dx.doi.org/10.1016/s0531-5565(01)00139-5.

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49

Sapkota, Niraj, Dev Shah, and Md Islam. "Biological interaction of stress and irritable bowel syndrome." International Journal of Medical Science and Public Health 3, no. 10 (2014): 1182. http://dx.doi.org/10.5455/ijmsph.2014.150920141.

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50

Aponte-Santamaría, Camilo, Jan Brunken, and Frauke Gräter. "Stress Propagation through Biological Lipid Bilayers in Silico." Journal of the American Chemical Society 139, no. 39 (September 25, 2017): 13588–91. http://dx.doi.org/10.1021/jacs.7b04724.

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