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Relevant bibliographies by topics / Blood Blood groups

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Academic literature on the topic 'Blood Blood groups'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Blood Blood groups"

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Sandler, S. Gerald. "Bloody Brilliant: A History of Blood Groups and Blood Groupers." Immunohematology 33, no. 2 (2019): 82–83. http://dx.doi.org/10.21307/immunohematology-2019-013.

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Sahani, Neelam, Chintamani Pathak, Somshankar Chowdhury, Preeti Sharma, Shweta Sushmita, and Indrani Dhawan. "The Prevalence of ABO and Rh Blood Groups in General Population and Comparing Male and Female Blood Group Distribution." Annals of Applied Bio-Sciences 5, no. 3 (September 18, 2018): A77–82. http://dx.doi.org/10.21276/aabs.2303.

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Vona, G., Margherita Salis, P. Bitti, and Valeria Succa. "Blood groups of the Sardinian population (ltaly)." Anthropologischer Anzeiger 52, no. 4 (December 13, 1994): 297–304. http://dx.doi.org/10.1127/anthranz/52/1994/297.

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Kirkman, Emrys. "Blood groups." Anaesthesia & Intensive Care Medicine 8, no. 5 (May 2007): 200–202. http://dx.doi.org/10.1016/j.mpaic.2007.02.007.

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Kirkman, Emrys. "Blood groups." Anaesthesia & Intensive Care Medicine 11, no. 6 (June 2010): 232–35. http://dx.doi.org/10.1016/j.mpaic.2010.02.016.

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Olefir, Irina Anatolievna. "Blood groups." Spravočnik vrača obŝej praktiki (Journal of Family Medicine), no. 6 (June 1, 2021): 71–75. http://dx.doi.org/10.33920/med-10-2106-09.

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On June 14, 1868, Karl Landsteiner, an outstanding scientist, known for his works in the field of immunohematology and immunochemistry, who received the Nobel Prize for the discovery of blood group systems in 1930, was born in a Viennese family. In 1900, Karl Landsteiner published a work in which he described in detail the process of agglutination that occurs when the blood plasma of one person is mixed with the red blood cells of another one. At that time, the scientist came to the conclusion that this phenomenon was of an immunological nature. In 1901, Landsteiner decided to divide human blood into three subgroups: A, B, and C; a little later, the AB group was added to them, while the C group was renamed as O. In addition, it was Landsteiner who invented a fairly simple scheme that allows developing and introducing the basic principles of blood transfusion into wide practice, and the world got a wonderful opportunity to save hundreds and thousands of human lives. Thanks to this discovery, made more than 100 years ago, more than 100 million donations are made every year around the world, more than half of which are in developed countries with high living standards and incomes. Here people come to blood donation deliberately, and not for the sake of receiving financial or any other benefit. Thanks to blood transfusion, it became possible to successfully carry out many surgical interventions accompanied by the loss of a large amount of blood, exchange blood transfusion for hemolytic disease of newborns, and substitution therapy for many pathological conditions. Karl Landsteiner’s work was highly appreciated: in 1930, due to the discovery of blood groups, he became the Nobel Prize laureate in the field of medicine.

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Seiguer, Alberto C. "Blood Relations: Blood Groups and Anthropology." American Journal of Tropical Medicine and Hygiene 34, no. 2 (March 1, 1985): 414. http://dx.doi.org/10.4269/ajtmh.1985.34.2.tm0340020414a.

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Sidhu, L. S., P. Malhotra, and S. P. Singh. "AB0 and Rh blood groups in diabetes mellitus." Anthropologischer Anzeiger 46, no. 3 (September 16, 1988): 269–75. http://dx.doi.org/10.1127/anthranz/46/1988/269.

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Marzban, M., M. S. Kamali, and T. Hosseinbasi. "Blood groups of the people of Ahwaz, Iran." Anthropologischer Anzeiger 46, no. 1 (April 21, 1988): 83–89. http://dx.doi.org/10.1127/anthranz/46/1988/83.

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Rath, GirijaPrasad, Ranadhir Mitra, and Nitasha Mishra. "Blood groups systems." Indian Journal of Anaesthesia 58, no. 5 (2014): 524. http://dx.doi.org/10.4103/0019-5049.144645.

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Dissertations / Theses on the topic "Blood Blood groups"

1

Olsson, Martin L. "Molecular genetic studies of the blood group ABO locus in man." Lund : Dept. of Transfusion Medicine, Institute of Laboratory Medicine, Lund University, 1997. http://catalog.hathitrust.org/api/volumes/oclc/38985966.html.

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Altayar, Malik Abdullah. "Next generation sequencing-based genotyping of human blood groups : FY, JK and ABO genes." Thesis, University of Plymouth, 2017. http://hdl.handle.net/10026.1/9325.

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Serological discrepancies in matching blood group antigens between donors and patients for blood transfusion may lead to alloimmunisation, especially in multiply transfused patients. Blood group genotyping (BGG) has contributed in reducing this issue. ABO, Fy and Jk antigens are among those to be causative for alloimmunisation through transfusion or pregnancy. The number of alleles of these clinically significant blood groups is ever increasing. Currently, all commercially available high-throughput BGG platforms are only based on pre-defined polymorphisms. Consequently, novel or rare alleles that might have clinical significance are not identified. Next generation sequencing (NGS) circumvents this issue by providing high-throughput comprehensive genotyping of blood group genes in discovery mode to find all existing and novel mutations. Accordingly, a large number of individuals can be genotyped in a single run. Here, we describe an NGS-based method coupled with long-range polymerase chain reaction (LR-PCR) for high-throughput, rapid and extensive genotyping of FY, JK and ABO blood group genes. The Ion Torrent Personal Genome Machine (PGMTM) was used for sequencing the entire FY, JK and ABO blood group genes including flanking regions. Accordingly, high resolution genotyping was obtained. 53 genomic DNA samples were sequenced and genotyped for FY, 67 for JK and 47 for ABO. Sequencing data were aligned to the gene reference sequence derived from the human genome (hg19) to analyse variants. Analysis was accomplished by software packages, such as Ion Torrent SuiteTM plugins. Sanger sequencing of cDNA and cDNA clones was used to confirm findings in the JK gene. The sequencing data had a coverage depth of more than 5000x for FY, 700x for JK and 600x for ABO. NGS data matched with the serological phenotypes of FY alleles FY*A, FY*B and FY*02 Null main polymorphisms, such as FY*A/FY*B (125G > A) in exon 2 and (-67 T > C) in the promotor region. JK variant analysis revealed that the JK*01W.01 allele (130G > A) is common (10/67 samples) with normal antigenicity. The previously described silencing polymorphism (810G > A), leading to a purported JK*B null allele, restores a splice site and does not correlate with loss of Jkb antigenicity (10/67 samples). JK intron analysis revealed several new JK alleles described in this thesis. All 7 exons, introns and the flanking regions of the ABO gene were covered by only four amplicons. Several rare O alleles were found, such as O73 and O75, while one suggested novel O allele was characterised by a missense SNP 482G > A (Arg161His) in exon 7. The ABO reference sequence from hg19 appeared to resemble (O01 and O02) alleles. The intronic SNPs might be used to distinguish between alleles more accurately as a correlation of the intronic SNPs with the alleles was noted for the homozygous O alleles. It is predicted that NGS-based genotyping will replace not only microarray-based genotyping but also serology in the blood group typing of individuals, with great advancements in technology and molecular knowledge being expected in the near future.

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Nilsson, Stinabritt. "Synthesis of blood-group and tumour-associated oligosaccaharides and a bacterial polysaccharide fragment." Lund : Organic Chemistry 2, Lund Institute of Technology, University of Lund, 1992. http://books.google.com/books?id=U-dqAAAAMAAJ.

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Du, Toit Masha. "Investigating the efficacy of XML and stylesheets to render electronic courseware for multiple learning styles." Master's thesis, University of Cape Town, 2007. http://hdl.handle.net/11427/6393.

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Includes bibliographical references (leaves 87-90)
The objective of this project was to test the efficacy of using Extensible Markup Language (XML) - in particular the DocBook 5.0b5 schema - and Extensible Stylesheet Language Transformation (XSLT) to render electronic courseware that can be dynamically re-formatted according to a student's individual learning style. The text of a typical lesson was marked up in XML according to the DocBook schema, and several XSLT stylesheets were created to transform the XML document into different versions, each according to particular learning needs. These learning needs were drawn from the Felder-Silverman learning style model. The notes had links to trigger JavaScript functions that allowed the student to reformat the notes to produce different views of the lesson.

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Varzi, Ali Mohammad. "Development and applications of molecular technologies for blood group genotyping." Thesis, University of Aberdeen, 2010. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=165837.

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Haemagglutination is the recognised serologic technique for common (ABO & Rh) blood group antigen phenotyping with limitations; typing multi-transfused patients and non-invasive foetal blood group determination. Molecular technological advances in characterising the 30 blood group systems have also generated PCR based direct genotyping techniques. Their utility in routine blood banking practice is a rapidly evolving field. The study aims were (1) to establish PCR-SSP assays for KEL, FY and JK blood group genotyping, (2) to evaluate HEA BeadChipTM technology for SNPs detection of RHCc, RHEe, CO, DI, DO, FY, JK, KEL, LU, LW, MNS and SC and haemoglobinopathy S, against serology considering reproducibility, reliability, sensitivity and labour saving potential (3) to evaluate the specificity and sensitivity of TaqMan Real-Time PCR for NIPD of foetal RHD7, RHC, RHc, RHE and SRY status and (4) to establish Real-Time PCR assays and MGB TaqMan probes for 8 sets of gender-independent “Bi-allelic” Short Insertion/Deletion Polymorphisms (SIDPs) as internal assay controls confirming the presence of cell-free foetal DNA (cffDNA). The PCR-SSP results for KEL, FY and JK typing results showed complete concordance with serology for all samples except 1×JKa and 7×Fyb; discrepancies resolved by subsequent DNA sequencing. The HEA BeadChipTM microarray validation on gDNA (n=224) and 22 saliva samples, giving overall allele detection (ADR) and concordance rates (CoR) of >99.8% for the 24 alleles. The Fyx allele (Fyb/Fyx: 265C>T) frequency in Scottish donors (5.4%) was much higher than expected. Saliva-derived gDNA was less sensitive than buffy coat-derived gDNA; ADR 89.9% and 100% respectively. NIPD foetal blood group genotyping by Real-Time PCR of 51 alloimmunised pregnancies (n=104 samples, 12 to ≥40 weeks) with was 100% accurate for RHD7, RHC and RHE assays; 95.7% for RHc and 99% for SRY. The utility of Real-Time TaqMan assays for 8 selected SIDPs as paternal (foetal) markers, were assessed using gDNA, cell-free DNA (cfDNA) from 61 donors and 6 extended families and finally with cffDNA from 13 pregnancies. Based on these research findings, many of the molecular assays are now established in Aberdeen.

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Bianco-Miotto, Tina. "Loss of ABO antigens in haematological malignancies." Adelaide, S.A, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09phb578.pdf.

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"May 2002" Includes bibliographical references (leaves 229-251) Describes the investigation of the alteration of ABH antigen expression on the surface of red blood cells in patients with haematological malignancies.

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Sommerville, W. "An evaluation of human erythrocyte sulphydryl groups." Thesis, University of Strathclyde, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.382552.

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Nardan, Denise. "Acid hydrolysis of neutral glycosphingolipids thesis submitted in fulfillment of the degree of Doctorate of Philosophy, Auckland University of Technology, June 2007 /." Click here to access this resource online, 2007. http://repositoryaut.lconz.ac.nz/theses/1389/.

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Orono, Lisa Lorraine. "Novel sensor for rapid detection of blood cell types magnetostrictive microcantilevers /." Auburn, Ala., 2005. http://repo.lib.auburn.edu/2005%20Summer/master's/ORONA_LISA_41.pdf.

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Sheehan, C. P. "The application of the enzyme-linked immunosorbent assay (ELISA) to ABH grouping in forensic science." Thesis, University of Surrey, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381661.

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Books on the topic "Blood Blood groups"

1

Human blood groups. Oxford: Blackwell Science, 1995.

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Daniels, Geoff. Human blood groups. 2nd ed. Malden, MA: Blackwell Science, 2002.

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Blood relations: Blood groups and anthropology. Oxford: Oxford University Press, 1985.

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Daniels, Geoff. Human Blood Groups. Oxford, UK: Wiley-Blackwell, 2013. http://dx.doi.org/10.1002/9781118493595.

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Schenkel-Brunner, Helmut. Human Blood Groups. Vienna: Springer Vienna, 2000. http://dx.doi.org/10.1007/978-3-7091-6294-1.

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Schenkel-Brunner, Helmut. Human Blood Groups. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7.

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Daniels, Geoff, ed. Human Blood Groups. Oxford, UK: Blackwell Science Ltd, 2002. http://dx.doi.org/10.1002/9780470987018.

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E, Dodd Barbara, and Lincoln Patrick J, eds. Blood group serology. 6th ed. Edinburgh: Churchill Livingstone, 1988.

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Imelda, Bromilow, and ebrary Inc, eds. Essential guide to blood groups. 2nd ed. Chichester, West Sussex, UK: Wiley-Blackwell, 2010.

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Imelda, Bromilow, ed. Essential guide to blood groups. Malden, Mass: Blackwell Pub., 2007.

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Book chapters on the topic "Blood Blood groups"

1

Bier, Otto G. "Blood Groups." In Fundamentals of Immunology, 227–42. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70393-5_8.

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Cooling, Laura. "Carbohydrate blood groups." In Rossi's Principles of Transfusion Medicine, 159–75. Chichester, WestSussex: John Wiley & Sons, Ltd., 2016. http://dx.doi.org/10.1002/9781119013020.ch13.

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Schenkel-Brunner, Helmut. "Introduction." In Human Blood Groups, 1–7. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7_1.

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Schenkel-Brunner, Helmut. "Polyagglutination." In Human Blood Groups, 283–98. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7_10.

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Schenkel-Brunner, Helmut. "Sid and Cad." In Human Blood Groups, 299–309. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7_11.

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Schenkel-Brunner, Helmut. "Sialic Acid-Containing Receptors for Cold Agglutinins." In Human Blood Groups, 310–18. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7_12.

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Schenkel-Brunner, Helmut. "Rh System." In Human Blood Groups, 319–43. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7_13.

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Schenkel-Brunner, Helmut. "Chido and Rodgers." In Human Blood Groups, 344–50. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7_14.

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Schenkel-Brunner, Helmut. "Duffy." In Human Blood Groups, 351–55. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7_15.

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Schenkel-Brunner, Helmut. "Kell." In Human Blood Groups, 356–64. Vienna: Springer Vienna, 1995. http://dx.doi.org/10.1007/978-3-7091-3686-7_16.

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Conference papers on the topic "Blood Blood groups"

1

Sills, T. H., and S. Heptinstall. "BLOOD TAURINE AFTER MYOCARDIAL INFARCTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643021.

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Taurine, an amino acid that is present in high concentrations in the heart, is released from the heart after myocardial damage. There is evidence that the concentration of taurine in whole blood is raised after myocardial infarction (MI), and it has been suggested that blood taurine may be a measure of the degree of infarction. We have obtained serial measurements of blood taurine in patients admitted to a coronary care unit and have compared the results with those obtained for two cardiac enzymes (AST and HBD) and other blood parameters.The patients were divided into two groups: those for whom there was a peak of AST activity (> 40 i.u./l) (Group 1, n = 24) and those for whom AST and HBD was not raised (Group 2, n = 15). For Group 1 patients, mean results were obtained for each of the parameters for the day on which AST peaked (designated Day 0) and for preceeding and subsequent days. For Group 2 a single mean was obtained. Results marked * in the table differ significantly (p < 0.05 or lower) from those for Group 2:It can be seen that blood taurine was significantly raised after MI and followed a pattern similar to the neutrophil count. Furthermore, several positive correlations (r = 0.63-0.79) were obtained between taurine and neutrophil count in both groups, but not between taurine and AST or HBD.In another investigation we measured the amounts of taurine in neutrophils, platelets and plasma from patients with MI (n = 5) and controls (n = 9). We found no differences in the amounts present per neutrophil, per platelet or per ml of plasma.Our data suggest that the increased level of taurine in blood after MI merely reflects the increased number of neutrophils present in blood following the event.

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Kamberi, Naser, and Hyzer Rizani. "Reciprocal Genetic Effects in Weight and Blood Groups." In University for Business and Technology International Conference. Pristina, Kosovo: University for Business and Technology, 2018. http://dx.doi.org/10.33107/ubt-ic.2018.373.

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Agustina, Susanti, Doddy Rusmono, and Riche Cynthia Johan. "Reading Behaviour Base on Biological Information of Blood Groups." In 1st International Conference on Educational Sciences. SCITEPRESS - Science and Technology Publications, 2017. http://dx.doi.org/10.5220/0007036000580066.

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Galvin, DAJ, A. C. Meek, P. Pate, and C. N. McCollum. "DO PLATELET INHIBITORS INCREASE OPERATIVE BLOOD LOSS?" In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644225.

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Although platelet inhibitory therapy improves arterial graft patency, surgeons are anxious that preoperative administration may increase operative bleeding. We investigated the effect of platelet inhibitors on blood loss during femoral artery replacement in dogs.Thirty greyhounds were randomised to receive placebo, a thromboxane antagonist GR32191 25mg (Glaxo Group Research) or aspirin 150mg (ASA) plus dipyridamole 50mg (DPM) twice daily starting 48 hours prior to implanting a 6cm length of 6mm PTFE in the femoral artery using standardised incision, mobilisation and anastomosis with 6.0 prolene. All bleeding was collected in swabs which were then thoroughly washed in 2L heparinised saline. The erythrocytes were haemolysed by adding potassium cyanide and the haemoglobin concentration measured in a Coulter Haemoglobinometer (Coulter Electronics). Blood loss was calculated by comparison to 1:500 dilution of the same animal’s venous blood. The bleeding time of the arterial anastomosis was also recorded.The mean (± sem) blood loss was similar in all three groups tending to be slightly less with GR32191 and ASA ± DPM at 135125 and 115±121 mis respectively, compared to 152±129 mis on placebo (NS). Anastomosis bleeding time appeared to be prolonged at 390±131 secs by the thromboxane antagonist compared to 291±40 with placebo and 224±136 with ASA and DPM, but this difference did not achieve statistical significance. There was a significant correlation (r=0.53) between blood loss and anastomotic bleeding time (p<0.001).This method of measuring blood loss is easily applicable to patients and does not demonstrate any important tendency to increased bleeding with preoperative platelet inhibitors.

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Skjønsberg, O. H., P. Kierulf, L. F. Engebretsen, G. Gjønnes, and H. C. Godal. "INCREASED THROMBIN GENERATION DURING COLLECTION OF BLOOD FROM DONORS TAKING ORAL CONTRACEPTIVES (OC)." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644275.

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450 ml blood was drawn into Fenwal PVC-bags from 26 OC-users and 28 non-users. The groups were comparable with regard to sex, age, smoking habits and blood collecting time. Thrombin generation was estimated as the fibrinopeptide A (FPA) concentrations in the bags immediately after ending the donations. The blood was also analyzed following storage at 4°C for 24 hours.Subsequent to donation, the median FPA level in the control group was 4.2 (range 1.8-18.9) and in the OC-group 7.5 (range 2.2-113.7) (p°0.05), reflecting a more pronounced thrombin generation during collection of blood from OC-users. The level of prekallikrein (PKK) was also higher in blood drawn from OC-users (97.0% (73-128) in the control group versus 121.5% (79166) in the OC-group (p°0.001)), as was the level of FVIII:C, the latter difference was, however, not significant (p=0.06). The A T-111 concentrations were similar in the two groups.No cold promoted activation could be observed following storage of the bags at 4°C for 24 hours, neither was any change observed in the levels of FPA, PKK or AT-III. There was no difference between the groups with regard to decay of FV111: C upon storage.We conclude that thrombin generation is more pronounced during collection of blood from donors taking OC. Only some of the OC-users seem to generate appreciable amounts of FPA (19% above 30 ng/ml), and it is important to notice that all bags containing alarmingly high levels of FPA were drawn from women taking OC.

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Abbate, R., M. Boddi, S. Favilla, G. Costanzo, R. Paniccia, and G. F. Paniccia. "WHOLE BLOOD AGGREGOMETER IN THE ASSESSMENT OF PLATELET HYPER-AGGREGABILITY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644554.

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The aim of this study has been to investigate the reliability of platelet aggregation in whole blood in some clinical conditions associated to thromboembolic complications.18 healthy subjects, 15 patients affected by ischemic heart disease (IHD) and 15 patients affected by insulin independent diabetes, free of vascular complications, were studied. Collagen induced (2.5 mg/L f.c.) platelet aggregation was evaluated both in whole blood (WB) by using impedance whole blood aggregometer (Chrono-Log) and in platelet rich plasma (PRP) by Born aggregometer. Aggregation was significantly higher in whole blood than in PRP in all the groups investigated (p < 0.01). No significant difference was found in PRP aggregation among the three groups, whereas WB aggregation was significantly higher in the two patient groups (IHD 79.5 + 14.2%, Diabetes 81.3 + 17.6%) than in controls (64.8 ± 14.1%) (p < 0.01 for both comparisons). No relationship was found between WB aggregation and Hct or platelet number in any of the groups studied. A slight relationship was found between megathrombocyte count and WE aggregation values (r=0.31, p < 0.05).Collagen platelet aggregation in WB seems to be provided with higher sensibility than PRP aggregation in detecting hyper-aggregability, probably because it does not imply the selection of platelet populations with loss of larger platelets and of other blood cells.

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Lan, H., A. M. Al-Jumaily, W. Hing, and A. Lowe. "Biomechanical Basis of Oscillometric Blood Pressure Measuring Technique." In ASME 2009 International Mechanical Engineering Congress and Exposition. ASMEDC, 2009. http://dx.doi.org/10.1115/imece2009-11857.

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Non-invasive blood pressure (BP) measurement has been used clinically for over a century to diagnose hypertension. Compared with the auscultatory technique, the oscillometric technique requires less professional training and is widely used in automatic BP measurement devices. Currently, most of these devices measure and record amplitude of cuff pressure oscillation, and then calculate diastolic and systolic pressure using characteristic ratios and designed algorithms. A finite element (FE) model is developed to study the biomechanical basis of this technique. The model identifies that errors were caused by mechanical factors of the soft tissue and the shape of the arm. By personalizing the parameters for each patient, the accuracy of the measurement will be improved for all age groups.

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Jalali, Ali, C. Nataraj, Margaret Butchy, and Ali Ghaffari. "Feature Extraction and Abnormality Detection in Autonomic Regulation of Cardiovascular System." In ASME 2011 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2011. http://dx.doi.org/10.1115/detc2011-48617.

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The objective of this study is to develop an efficient methodology for classifying patients suffering any type of blood pressure dysregulation from healthy subjects. Four features of malfunctions in blood pressure regulation are introduced, and a criterion is proposed for each feature to evaluate and distinguish patients from healthy subjects. The evaluated features are based on the analysis of difference between data related to healthy subjects and those collected from patients. The proposed criteria are implemented on a group of healthy and patient subjects by collecting their systolic blood pressure (SBP) and their heart rate (HR) time series. The proposed method is applied on three different groups of subjects each containing four healthy and eleven patients. It is shown that the algorithm properly detects the status of all fifteen subjects in one group and fourteen subjects in two groups. The results obtained indicate that the selected features have remarkable capability in detection of blood pressure dysregulation.

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Liu, Fengjuan, and Yuechen Liu. "Analysis on the Dependence of Students with Different Blood Groups on Mobile Devices." In the 2019 4th International Conference. New York, New York, USA: ACM Press, 2019. http://dx.doi.org/10.1145/3345094.3345102.

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oude Egbrink, Mirjam G. A., Geert Jan Tangelder, Dick W. Slaaf, and Robert S. Reneman. "IN VIVO CHANGES IN SYSTEMIC PCO2 AND PO2 INFLUENCE THE THROMBOEMBOLIC REACTION FOLLOWING WALL PUNCTURE IN VENULES BUT NOT IN ARTERIOLES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643180.

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Changes in pH and PCO2 influence the aggregation of blood platelets in response to various agents in vitro. In the present study intravital video-microscopy was used to investigate whether changes in systemic blood gas values influence the thromboembolic reaction in vivo as induced by vessel wall injury.The microtrauma was induced by puncturing the walls of microvessels in the rabbit mesentery (diameter range: 20-40 μm) with glass micropipets (tip diameters: 6-8 μm). The thromboembolic reactions were compared in two groups of anesthetized rabbits. The control group was ventilated to keep the blood gas values within normal ranges (means: pH=7.40, pCO2=32.9 mmHg, pO2=104.7 mmHg). The experimental group breathed spontaneously (mean blood gas values: pH=7.34, pCO2=50.5 mmHg, pO2=48.1 mmHg). The pCO2 and pO2 values were significantly different between both groups.In arterioles and venules of both groups bleeding and thrombus formation started immediately following wall puncture. Bleeding times were short (medians between 1.0 and 2.6 s). Parts of the thrombi started to embolize between 11.4 and 18.2 s following wall puncture (medians). In the control group embolization continued for 101 s in the arterioles and 17 s in the venules; during these periods 6 and 1 emboli were produced, respectively (all median values). In the experimental group the duration of embolization in the arterioles was 143 s in which period 7.5 emboli were produced, values not significantly different from control. In the venules of the experimental group embolization and hence platelet reaction went on uninhibited during the whole observation period of 600 s and 30 emboli were produced. Fluid dynamic factors cannot explain the differences in thromboembolic reaction between the control and experimental venules; vessel diameters and red blood cell velocities were not significantly different between both groups. Therefore, it is likely that the change in thromboembolic reaction in the venules results from the changes in systemic PCO2 and/or pO2. The different reactions in arterioles and venules in response to the altered systemic blood gas values might arise from different reactions in the vessel walls.

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Reports on the topic "Blood Blood groups"

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Mozgovaya, E. E., S. A. Bedina, I. A. Zborovskaya, A. S. Trofimenko, and S. S. Khortieva. XANTINOXIDASE, XANTINDEGHYROGENASE, SUPEROXIDEDISMUTASIS ACTIVITY OF BLOOD PLASMA IN THE GROUP OF PATIENTS WITH EXTRA ARTICULAR MANIFESTATIONS OF RHEUMATOID ARTHRITIS. Планета, 2018. http://dx.doi.org/10.18411/978-5-907109-24-7-2018-xxxv-212-218.

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Mankelow, Tosti J., Nicholas Burton, Fanney O. Stedansdottir, Frances A. Spring, Stephen F. Parsons, Jan S. Pesersen, Cristiano L. P. Oliveira, et al. The Laminin 511/521 Binding Site on the Lutheran Blood Group Glycoprotein is Located at theFlexible Junction of Ig Domains 2 and 3. Office of Scientific and Technical Information (OSTI), July 2007. http://dx.doi.org/10.2172/945355.

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Treadwell, Jonathan R., James T. Reston, Benjamin Rouse, Joann Fontanarosa, Neha Patel, and Nikhil K. Mull. Automated-Entry Patient-Generated Health Data for Chronic Conditions: The Evidence on Health Outcomes. Agency for Healthcare Research and Quality (AHRQ), March 2021. http://dx.doi.org/10.23970/ahrqepctb38.

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Background. Automated-entry consumer devices that collect and transmit patient-generated health data (PGHD) are being evaluated as potential tools to aid in the management of chronic diseases. The need exists to evaluate the evidence regarding consumer PGHD technologies, particularly for devices that have not gone through Food and Drug Administration evaluation. Purpose. To summarize the research related to automated-entry consumer health technologies that provide PGHD for the prevention or management of 11 chronic diseases. Methods. The project scope was determined through discussions with Key Informants. We searched MEDLINE and EMBASE (via EMBASE.com), In-Process MEDLINE and PubMed unique content (via PubMed.gov), and the Cochrane Database of Systematic Reviews for systematic reviews or controlled trials. We also searched ClinicalTrials.gov for ongoing studies. We assessed risk of bias and extracted data on health outcomes, surrogate outcomes, usability, sustainability, cost-effectiveness outcomes (quantifying the tradeoffs between health effects and cost), process outcomes, and other characteristics related to PGHD technologies. For isolated effects on health outcomes, we classified the results in one of four categories: (1) likely no effect, (2) unclear, (3) possible positive effect, or (4) likely positive effect. When we categorized the data as “unclear” based solely on health outcomes, we then examined and classified surrogate outcomes for that particular clinical condition. Findings. We identified 114 unique studies that met inclusion criteria. The largest number of studies addressed patients with hypertension (51 studies) and obesity (43 studies). Eighty-four trials used a single PGHD device, 23 used 2 PGHD devices, and the other 7 used 3 or more PGHD devices. Pedometers, blood pressure (BP) monitors, and scales were commonly used in the same studies. Overall, we found a “possible positive effect” of PGHD interventions on health outcomes for coronary artery disease, heart failure, and asthma. For obesity, we rated the health outcomes as unclear, and the surrogate outcomes (body mass index/weight) as likely no effect. For hypertension, we rated the health outcomes as unclear, and the surrogate outcomes (systolic BP/diastolic BP) as possible positive effect. For cardiac arrhythmias or conduction abnormalities we rated the health outcomes as unclear and the surrogate outcome (time to arrhythmia detection) as likely positive effect. The findings were “unclear” regarding PGHD interventions for diabetes prevention, sleep apnea, stroke, Parkinson’s disease, and chronic obstructive pulmonary disease. Most studies did not report harms related to PGHD interventions; the relatively few harms reported were minor and transient, with event rates usually comparable to harms in the control groups. Few studies reported cost-effectiveness analyses, and only for PGHD interventions for hypertension, coronary artery disease, and chronic obstructive pulmonary disease; the findings were variable across different chronic conditions and devices. Patient adherence to PGHD interventions was highly variable across studies, but patient acceptance/satisfaction and usability was generally fair to good. However, device engineers independently evaluated consumer wearable and handheld BP monitors and considered the user experience to be poor, while their assessment of smartphone-based electrocardiogram monitors found the user experience to be good. Student volunteers involved in device usability testing of the Weight Watchers Online app found it well-designed and relatively easy to use. Implications. Multiple randomized controlled trials (RCTs) have evaluated some PGHD technologies (e.g., pedometers, scales, BP monitors), particularly for obesity and hypertension, but health outcomes were generally underreported. We found evidence suggesting a possible positive effect of PGHD interventions on health outcomes for four chronic conditions. Lack of reporting of health outcomes and insufficient statistical power to assess these outcomes were the main reasons for “unclear” ratings. The majority of studies on PGHD technologies still focus on non-health-related outcomes. Future RCTs should focus on measurement of health outcomes. Furthermore, future RCTs should be designed to isolate the effect of the PGHD intervention from other components in a multicomponent intervention.

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