Relevant bibliographies by topics / Blood-Cerebrospinal Fluid Barrier (BCSFB)

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Blood-Cerebrospinal Fluid Barrier (BCSFB)'

Author: Grafiati
Published: 10 September 2021
Last updated: 29 July 2025

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Journal articles on the topic "Blood-Cerebrospinal Fluid Barrier (BCSFB)"

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Szmydynger-Chodobska, Joanna, Nathalie Strazielle, Jessica R. Gandy, et al. "Posttraumatic Invasion of Monocytes across the Blood—Cerebrospinal Fluid Barrier." Journal of Cerebral Blood Flow & Metabolism 32, no. 1 (2011): 93–104. http://dx.doi.org/10.1038/jcbfm.2011.111.

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The invasion of inflammatory cells occurring after ischemic or traumatic brain injury (TBI) has a detrimental effect on neuronal survival and functional recovery after injury. We have recently demonstrated that not only the blood-brain barrier, but also the blood-cerebrospinal fluid (CSF) barrier (BCSFB), has a role in posttraumatic recruitment of neutrophils. Here, we show that TBI results in a rapid increase in synthesis and release into the CSF of a major chemoattractant for monocytes, CCL2, by the choroid plexus epithelium, a site of the BCSFB. Using an in vitro model of the BCSFB, we also
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Tietz, Silvia, and Britta Engelhardt. "Brain barriers: Crosstalk between complex tight junctions and adherens junctions." Journal of Cell Biology 209, no. 4 (2015): 493–506. http://dx.doi.org/10.1083/jcb.201412147.

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Unique intercellular junctional complexes between the central nervous system (CNS) microvascular endothelial cells and the choroid plexus epithelial cells form the endothelial blood–brain barrier (BBB) and the epithelial blood–cerebrospinal fluid barrier (BCSFB), respectively. These barriers inhibit paracellular diffusion, thereby protecting the CNS from fluctuations in the blood. Studies of brain barrier integrity during development, normal physiology, and disease have focused on BBB and BCSFB tight junctions but not the corresponding endothelial and epithelial adherens junctions. The crossta
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Dunn, Jeff F., and Albert M. Isaacs. "The impact of hypoxia on blood-brain, blood-CSF, and CSF-brain barriers." Journal of Applied Physiology 131, no. 3 (2021): 977–85. http://dx.doi.org/10.1152/japplphysiol.00108.2020.

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The blood-brain barrier (BBB), blood-cerebrospinal fluid (CSF) barrier (BCSFB), and CSF-brain barriers (CSFBB) are highly regulated barriers in the central nervous system comprising complex multicellular structures that separate nerves and glia from blood and CSF, respectively. Barrier damage has been implicated in the pathophysiology of diverse hypoxia-related neurological conditions, including stroke, multiple sclerosis, hydrocephalus, and high-altitude cerebral edema. Much is known about the damage to the BBB in response to hypoxia, but much less is known about the BCSFB and CSFBB. Yet, it
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Ikeda-Murakami, Kei, Naoto Tani, Tomoya Ikeda, Yayoi Aoki, and Takaki Ishikawa. "Central Nervous System Stimulants Limit Caffeine Transport at the Blood–Cerebrospinal Fluid Barrier." International Journal of Molecular Sciences 23, no. 3 (2022): 1862. http://dx.doi.org/10.3390/ijms23031862.

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Caffeine, a common ingredient in energy drinks, crosses the blood–brain barrier easily, but the kinetics of caffeine across the blood–cerebrospinal fluid barrier (BCSFB) has not been investigated. Therefore, 127 autopsy cases (Group A, 30 patients, stimulant-detected group; and Group B, 97 patients, no stimulant detected group) were examined. In addition, a BCSFB model was constructed using human vascular endothelial cells and human choroid plexus epithelial cells separated by a filter, and the kinetics of caffeine in the BCSFB and the effects of 4-aminopyridine (4-AP), a neuroexcitatory agent
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Martens, Alexander, Nicole de Buhr, Hiroshi Ishikawa, Horst Schroten, and Maren von Köckritz-Blickwede. "Characterization of Oxygen Levels in an Uninfected and Infected Human Blood-Cerebrospinal-Fluid-Barrier Model." Cells 11, no. 1 (2022): 151. http://dx.doi.org/10.3390/cells11010151.

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The host–pathogen interaction during meningitis can be investigated with blood-cerebrospinal-fluid-barrier (BCSFB) cell culture models. They are commonly handled under atmospheric oxygen conditions (19–21% O2), although the physiological oxygen conditions are significantly lower in cerebrospinal fluid (CSF) (7–8% O2). We aimed to characterize oxygen levels in a Streptococcus (S.) suis-infected BCSFB model with transmigrating neutrophils. A BCSFB model with human choroid plexus epithelial cells growing on transwell-filters was used. The upper “blood”-compartment was infected and blood-derived n
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Furtado, André, Ana Catarina Duarte, Ana R. Costa, et al. "Circadian ABCG2 Expression Influences the Brain Uptake of Donepezil across the Blood–Cerebrospinal Fluid Barrier." International Journal of Molecular Sciences 25, no. 9 (2024): 5014. http://dx.doi.org/10.3390/ijms25095014.

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Donepezil (DNPZ) is a cholinesterase inhibitor used for the management of Alzheimer’s disease (AD) and is dependent on membrane transporters such as ABCG2 to actively cross brain barriers and reach its target site of action in the brain. Located in the brain ventricles, the choroid plexus (CP) forms an interface between the cerebrospinal fluid (CSF) and the bloodstream, known as the blood–CSF barrier (BCSFB). Historically, the BCSFB has received little attention as a potential pathway for drug delivery to the central nervous system (CNS). Nonetheless, this barrier is presently viewed as a dyna
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Madadi, Ahmad Khalid, and Moon-Jun Sohn. "Advances in Intrathecal Nanoparticle Delivery: Targeting the Blood–Cerebrospinal Fluid Barrier for Enhanced CNS Drug Delivery." Pharmaceuticals 17, no. 8 (2024): 1070. http://dx.doi.org/10.3390/ph17081070.

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The blood–cerebrospinal fluid barrier (BCSFB) tightly regulates molecular exchanges between the bloodstream and cerebrospinal fluid (CSF), creating challenges for effective central nervous system (CNS) drug delivery. This review assesses intrathecal (IT) nanoparticle (NP) delivery systems that aim to enhance drug delivery by circumventing the BCSFB, complementing approaches that target the blood–brain barrier (BBB). Active pharmaceutical ingredients (APIs) face hurdles like restricted CNS distribution and rapid clearance, which diminish the efficacy of IT therapies. NPs can be engineered to ex
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Gião, Tiago, Tiago Teixeira, Maria Rosário Almeida, and Isabel Cardoso. "Choroid Plexus in Alzheimer’s Disease—The Current State of Knowledge." Biomedicines 10, no. 2 (2022): 224. http://dx.doi.org/10.3390/biomedicines10020224.

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The choroid plexus (CP), located in each of the four ventricles of the brain, is formed by a monolayer of epithelial cells that surrounds a highly vascularized connective tissue with permeable capillaries. These cells are joined by tight junctions forming the blood–cerebrospinal fluid barrier (BCSFB), which strictly regulates the exchange of substances between the blood and cerebrospinal fluid (CSF). The primary purpose of the CP is to secrete CSF, but it also plays a role in the immune surveillance of the central nervous system (CNS) and in the removal of neurotoxic compounds from the CSF. Ac
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Wiatr, Marie, Simon Staubach, Ricardo Figueiredo, et al. "Echovirus-30 Infection Alters Host Proteins in Lipid Rafts at the Cerebrospinal Fluid Barrier In Vitro." Microorganisms 8, no. 12 (2020): 1958. http://dx.doi.org/10.3390/microorganisms8121958.

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Echovirus-30 (E-30) is a non-polio enterovirus responsible for meningitis outbreaks in children worldwide. To gain access to the central nervous system (CNS), E-30 first has to cross the blood-brain barrier (BBB) or the blood-cerebrospinal fluid barrier (BCSFB). E-30 may use lipid rafts of the host cells to interact with and to invade the BCSFB. To study enteroviral infection of the BCSFB, an established in vitro model based on human immortalized brain choroid plexus papilloma (HIBCPP) cells has been used. Here, we investigated the impact of E-30 infection on the protein content of the lipid r
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Banovic, Franjo, Sandrin Schulze, Mobarak Abu Mraheil, et al. "Different Involvement of Vimentin during Invasion by Listeria monocytogenes at the Blood–Brain and the Blood–Cerebrospinal Fluid Barriers In Vitro." International Journal of Molecular Sciences 23, no. 21 (2022): 12908. http://dx.doi.org/10.3390/ijms232112908.

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The human central nervous system (CNS) is separated from the blood by distinct cellular barriers, including the blood–brain barrier (BBB) and the blood–cerebrospinal fluid (CFS) barrier (BCSFB). Whereas at the center of the BBB are the endothelial cells of the brain capillaries, the BCSFB is formed by the epithelium of the choroid plexus. Invasion of cells of either the BBB or the BCSFB is a potential first step during CNS entry by the Gram-positive bacterium Listeria monocytogenes (Lm). Lm possesses several virulence factors mediating host cell entry, such as the internalin protein family—inc
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Dissertations / Theses on the topic "Blood-Cerebrospinal Fluid Barrier (BCSFB)"

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Chandorkar, Gurudatt Ajay Melethil Srikumaran K. "Mechanisms of blood-brain and blood-cerebrospinal fluid transport of aluminum in rats." Diss., UMK access, 2006.

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Thesis (Ph. D.)--School of Pharmacy. University of Missouri--Kansas City, 2006.<br>"A dissertation in pharmaceutical sciences and pharmacology." Advisor: Srikumaran Melethil. Typescript. Vita. Title from "catalog record" of the print edition Description based on contents viewed Dec. 20, 2007. Includes bibliographical references (leaves 159-192). Online version of the print edition.
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Kaur, Manjit. "Phytochemical mediated modulation of breast cancer resistance protein at the blood brain barrier and blood cerebrospinal fluid barrier." Thesis, Aston University, 2016. http://publications.aston.ac.uk/30065/.

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Drug delivery to the central nervous system (CNS) is significantly hindered by thepresence of the blood brain barrier (BBB) and blood cerebrospinal fluid barrier(BCSFB) and associated drug efflux transporter proteins. The aim of this work was to modulate the expression of breast cancer resistance protein (BCRP) at each barrier site using phytochemical modulators. In-vitro cellular models of both the BBB (PBMEC/C1-2) and BCSFB (Z310) were utilised and 18 phytochemical modulators screened for their cellular toxicity with IC50 values for the majority of phytochemicals being in excess of 100 μM. I
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Anckarsäter, Rolf. "Neurochemical and neuroendocrine reactions during non-neurological surgery /." Göteborg : Institute of Clinical Sciences, Department of Anesthesiology and Intensive Care, The Sahlgrenska Academy at the University of Gothenburg, 2010. http://hdl.handle.net/2077/2173.

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Pancotto, Theresa E. "Evaluation of cerebrospinal fluid biomarkers of endothelial damage and basement membrane degradation as indirect indicators of blood-brain barrier dysfunction in chronic canine hypothyroidism." Thesis, Virginia Tech, 2011. http://hdl.handle.net/10919/76955.

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A variety of neurologic illnesses including peripheral and cranial neuropathies, central vestibular disease, seizures and coma have been associated with hypothyroidism in dogs. Repeated studies have shown that there is loss of blood brain barrier (BBB) integrity in these animals. Current research has also shown the development cerebrospinal fluid abnormalities in neurologically normal hypothyroid dogs; a finding that is related to BBB degradation. This derangement may be secondary to atherosclerosis and vascular accidents. One possible mediator of vasospasm and ischemic brain injury is endothe
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Dahm, Tobias [Verfasser], and Hans-Georg [Akademischer Betreuer] Kräusslich. "Sequential transmigration of polymorphonuclear cells and naïve CD3+ T lymphocytes across the blood-cerebrospinal-fluid barrier in vitro following infection with Echovirus 30 / Tobias Dahm ; Betreuer: Hans-Georg Kräusslich." Heidelberg : Universitätsbibliothek Heidelberg, 2020. http://d-nb.info/1220081191/34.

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Frasca, Denis. "Étude de la distribution cérébrale de deux antibiotiques chez des patients de réanimation." Thesis, Poitiers, 2013. http://www.theses.fr/2013POIT1403/document.

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Pour exercer leur effet princeps, les médicaments doivent atteindre des concentrations nécessaires et suffisantes à leur site d'action tout en évitant la survenue d'effets indésirables. Les antibiotiques sont utilisés pour le traitement d'infection cérébroméningées dont la cible bactériologique se situe dans le liquide céphalorachidien (LCR) ou le liquide extracellulaire cérébral (LEC) un site d'action cérébral qui constitue aussi une cible pour des effets secondaires. La distribution de médicament dans le cerveau et le LCR est limitée par la présence des barrières hémato-encéphalique (BHE) et
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Volle, Romain. "Pathogénie des entérovirus : étude de la charge virale au cours de méningites et de la permissivité des cellules endothéliales microvasculaires cérébrales humaines." Thesis, Clermont-Ferrand 1, 2014. http://www.theses.fr/2014CLF1MM05/document.

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Les entérovirus humains (EV) constituent un groupe de virus à ARN d'une grande diversité génétique. Ils sont responsables d'infections cérébrales graves mais rares et de méningites bénignes mais fréquentes. Les évènements conduisant à l'entrée des EVs dans le système nerveux central (SNC), l'importance de la charge virale dans le liquide céphalo rachidien (LCR) et sa corrélation éventuelle avec l'intensité du processus inflammatoire réactionnel restent peu explorés. Comme de nombreux génotypes d'EV sont associés à des manifestations neurologiques semblables, des processus pathologiques communs
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Lobo, Diana Filipa Duarte. "Cerebrovascular and Blood-Brain Barrier Impairments in Machado-Joseph Disease." Master's thesis, 2017. http://hdl.handle.net/10362/25829.

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Central Nervous System (CNS)-barriers are essential to maintain brain homeostasis, protection and nutrition. Blood-brain barrier (BBB) is mainly constituted by brain endothelial cells, pericytes and astrocytes that restrict the communication between blood and the brain parenchyma. Blood-cerebrospinal fluid barrier (BCSFB) controls molecular exchange between and the cerebrospinal fluid in the epithelial cells of choroid plexus. Both barriers express tight junction (TJ) proteins that limit the paracellular permeability between adjacent cells. In several neurodegenerative diseases, BBB dysfunct
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Duarte, Ana Catarina Abreu. "Characterization of bitter taste receptors expression and function in the human blood-cerebrospinal fluid barrier." Doctoral thesis, 2021. http://hdl.handle.net/10400.6/11133.

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Bitter taste receptors (TR2) expression and functionality was recently reported in the rat choroid plexus (CP). CP epithelial cells establish a major brain barrier, the blood-cerebrospinal fluid barrier (BCSFB). Given their capacity to bind a large array of chemical compounds, we hypothesised that TR2 might be involved in monitoring the composition of blood and cerebrospinal fluid. Brain barriers play a critical role in the protection of the central nervous system (CNS) by hindering the access of toxic substances to the brain. Consequently, many drugs targeting neurological disorders are impa
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Gomes, Sara Ferreira Martins. "Induced Pluripotent Stem Cell-derived Brain Endothelial Cells as a Cellular Model to Study Neisseria meningitidis Infection." Doctoral thesis, 2019. https://nbn-resolving.org/urn:nbn:de:bvb:20-opus-188550.

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Bacterial meningitis occurs when blood-borne bacteria are able to penetrate highly specialized brain endothelial cells (BECs) and gain access to the meninges. Neisseria meningitidis (Nm) is a human-exclusive pathogen for which suitable in vitro models are severely lacking. Until recently, modeling BEC-Nm interactions has been almost exclusively limited to immortalized human cells that lack proper BEC phenotypes. Specifically, these in vitro models lack barrier properties, and continuous tight junctions. Alternatively, humanized mice have been used, but these must rely on known interactions and
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Books on the topic "Blood-Cerebrospinal Fluid Barrier (BCSFB)"

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D, Zheng Wei Ph, and Chodobski Adam, eds. The blood-cerebrospinal fluid barrier. Taylor & Francis, 2005.

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R, Helio Tomas, ed. The blood brain barrier. Nova Science Publishers, 2008.

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Keasley, Welch, Segal Malcolm B, and Davson Hugh 1909-, eds. Physiology and pathophysiology of the cerebrospinal fluid. Churchill Livingstone, 1987.

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Davson, Hugh. Physiology of the CSF and blood-brain barriers. CRC Press, 1996.

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Klaus, Felgenhauer, Holzgraefe Manfred, Prange Hilmar W, and International Quincke Symposium "Barrier Concepts and Cerebrospinal Fluid Analysis" (1991 : Göttingen, Germany), eds. CNS barriers and modern CSF diagnostics. VCH, 1993.

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J, Rovinsky Joseph, Guttmacher Alan Frank 1898-, Cherry Sheldon H, Berkowitz Richard L, and Kase Nathan G. 1930-, eds. Rovinsky and Guttmacher's Medical, surgical, and gynecologic complications of pregnancy. 3rd ed. Williams & Wilkins, 1985.

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B, Segal Malcolm, ed. Barriers and fluids of the eye and brain. CRC Press, 1992.

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Zheng, Wei, and Adam Chodobski. Blood-Cerebrospinal Fluid Barrier. Taylor & Francis Group, 2019.

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Zheng, Wei, and Adam Chodobski. Blood-Cerebrospinal Fluid Barrier. Taylor & Francis Group, 2005.

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Zheng, Wei. Blood-Cerebrospinal Fluid Barrier. Taylor & Francis Group, 2005.

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Book chapters on the topic "Blood-Cerebrospinal Fluid Barrier (BCSFB)"

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Tyrer, Peter J., Mark Slifstein, Joris C. Verster, et al. "Blood–Cerebrospinal Fluid Barrier." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_1097.

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Akanuma, Shin-ichi, Yoshiyuki Kubo, and Ken-ichi Hosoya. "Techniques for Evaluating Efflux Transport of Radiolabeled Drugs and Compounds from the Cerebrospinal Fluid Across the Blood-Cerebrospinal Fluid Barrier." In Blood-Brain Barrier. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8946-1_14.

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Furr, Martin. "Cerebrospinal Fluid and the Blood-Brain Barrier." In Equine Neurology. John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781118993712.ch2.

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Kam, Peter, Ian Power, Michael J. Cousins, and Philip J. Siddal. "Blood–Brain Barrier and Cerebrospinal Fluid (CSF)." In Principles of Physiology for the Anaesthetist. CRC Press, 2020. http://dx.doi.org/10.1201/9780429288210-5.

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Löwhagen, Pia, Barbro B. Johansson, and Claes Nordborg. "Nasal Cerebrospinal Fluid Drainage in Man." In New Concepts of a Blood—Brain Barrier. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-1054-7_19.

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Wildemann, Brigitte, and Jürgen Haas. "Multiple Sclerosis and the Blood–Cerebrospinal Fluid Barrier." In Current Human Cell Research and Applications. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-97-9495-9_8.

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Tumani, Hayrettin, and Harald Hegen. "CSF Albumin: Albumin CSF/Serum Ratio (Marker for Blood-CSF Barrier Function)." In Cerebrospinal Fluid in Clinical Neurology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-01225-4_9.

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Johanson, Conrad E., Edward G. Stopa, and Paul N. McMillan. "The Blood–Cerebrospinal Fluid Barrier: Structure and Functional Significance." In Methods in Molecular Biology. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-938-3_4.

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Kuschinsky, W. "Blood-Brain Barrier and the Production of Cerebrospinal Fluid." In Comprehensive Human Physiology. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-60946-6_28.

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McCabe, Shannon, Omar Al-Ali, Kirsten Limesand, Michael C. Kruer, and Ningning Zhao. "The Blood–Cerebrospinal Fluid Barrier and Brain Manganese Accumulation." In Current Human Cell Research and Applications. Springer Nature Singapore, 2025. https://doi.org/10.1007/978-981-97-9495-9_6.

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Conference papers on the topic "Blood-Cerebrospinal Fluid Barrier (BCSFB)"

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Costa, Leonardo, Jürgen Haas, Henriette Rudolph, et al. "The Choroid Plexus Is Permissive for a Preactivated Antigen-Experienced Memory B Cell Subset in Multiple Sclerosis." In Building Bridges in Medical Science 2021. Cambridge Medicine Journal, 2021. http://dx.doi.org/10.7244/cmj.2021.03.001.2.

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Background: The role of B cells in multiple sclerosis (MS) is increasingly recognized. B cells undergo compartmentalized redistribution in blood and cerebrospinal fluid (CSF) during active MS, whereby memory B cells accumulate in the CSF. While B-cell trafficking across the blood– brain barrier has been intensely investigated, cellular diapedesis through the blood–CSF barrier (BCSFB) is incompletely understood. Objectives: To investigate how B cells interact with the choroid plexus to transmigrate into the CSF, we isolated circulating B cells from healthy donors (HC) and MS patients, utilized
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Ficiara, E., F. D'Agata, S. Ansari, et al. "A mathematical model for the evaluation of iron transport across the blood-cerebrospinal fluid barrier in neurodegenerative diseases." In 2020 42nd Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC) in conjunction with the 43rd Annual Conference of the Canadian Medical and Biological Engineering Society. IEEE, 2020. http://dx.doi.org/10.1109/embc44109.2020.9175988.

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Borges, Isabella Sabião, João Victor Aguiar Moreira, Eustaquio Costa Damasceno Junior, et al. "Chronic inflammatory demyelinating polyradiculoneuropathy induced by paclitaxel." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.413.

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Background: Peripheral neuropathies in cancer are most often due to neurotoxic chemotherapeutic agents. Approximately 30% of patients receiving neurotoxic chemotherapy (CTX) will suffer from chemotherapy-induced peripheral neuropathy (CIPN). Paclitaxel is an extremely effective chemotherapeutic agent for the treatment of breast, ovarian, and lung cancer. However, paclitaxel-induced peripheral neuropathy occurs in 59-87% of patients who receive this drug. Paclitaxel is an anti-tubulin drug that causes microtubule stabilization, resulting in distal axonal degeneration, secondary demyelination an
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