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Journal articles on the topic 'Blood – Diseases'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

SHIBATA, AKIRA. "The blood.1.Interferon and blood diseases." Nihon Naika Gakkai Zasshi 82, no. 3 (1993): 450–54. http://dx.doi.org/10.2169/naika.82.450.

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2

Carey, Francis A., and Mary N. Sheppard. "Diseases of blood vessels." Surgery (Oxford) 39, no. 5 (2021): 241–47. http://dx.doi.org/10.1016/j.mpsur.2021.03.005.

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3

Rossiyskiy, D. M. "Organotherapy for blood diseases." Kazan medical journal 29, no. 8-9 (2022): 730–36. http://dx.doi.org/10.17816/kazmj89827.

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Hormones of various endocrine glands, being powerful regulators of growth, tissue nutrition and metabolism in the body and affecting both groups of the autonomic nervous system, the autonomic and sympathetic nervous system, at the same time have either an intensifying or inhibitory effect on the activity of the hematopoietic apparatus.

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4

Cluzeau, Thomas, Guillaume Robert, Alexandre Puissant, Arnaud Jacquel, Frédéric Luciano, and Patrick Auberger. "Autophagy and blood diseases." Hématologie 21, no. 2 (2015): 107–16. http://dx.doi.org/10.1684/hma.2015.0982.

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5

Lindop, George B. M. "Diseases of blood vessels." Surgery (Oxford) 27, no. 8 (2009): 313–19. http://dx.doi.org/10.1016/j.mpsur.2009.06.009.

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6

Sheppard, Mary N. "Diseases of blood vessels." Surgery (Oxford) 30, no. 8 (2012): 370–76. http://dx.doi.org/10.1016/j.mpsur.2012.05.017.

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7

Sheppard, Mary N. "Diseases of blood vessels." Surgery (Oxford) 33, no. 7 (2015): 295–301. http://dx.doi.org/10.1016/j.mpsur.2015.04.006.

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8

Carey, Francis A., and Mary N. Sheppard. "Diseases of blood vessels." Surgery (Oxford) 36, no. 6 (2018): 259–64. http://dx.doi.org/10.1016/j.mpsur.2018.03.011.

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9

Queral, Luis A. "Diseases of blood vessels." Journal of Vascular Surgery 3, no. 6 (1986): A1. http://dx.doi.org/10.1016/s0741-5214(86)70013-x.

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10

Queral, Luis A. "Diseases of blood vessels." Journal of Vascular Surgery 3, no. 6 (1986): 940. http://dx.doi.org/10.1016/0741-5214(86)90438-6.

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11

Abramson, David I. "Diseases of Blood Vessels." JAMA: The Journal of the American Medical Association 254, no. 14 (1985): 2000. http://dx.doi.org/10.1001/jama.1985.03360140162051.

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12

G, Kapatia. "Role of Peripheral Blood Film in Renal Diseases." Journal of Human Anatomy 7, no. 1 (2023): 1–5. http://dx.doi.org/10.23880/jhua-16000173.

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Peripheral blood smear examination is a diagnostic tool used in the assessment of various clinical conditions, including kidney diseases. While it is not a primary test for diagnosing kidney diseases, it can provide valuable insights into the presence of anemia, hemolysis, platelet abnormalities, infections, and other systemic changes that can be associated with kidney disorders. It is important to note that peripheral blood smear examination is just one aspect of a comprehensive diagnostic evaluation for kidney diseases and is not a direct method for diagnosing kidney diseases. Other tests, s

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13

Aditi, Sarda* Pravin Bhoyar Somesh Bawane Pratiksha Gawande Nilima Bhoskar Nilima Bhoskar Nilima M. Bhoskar Vaishnavi Bhedurkar. "Cardiovascular Diseases: A Review." International Journal of Pharmaceutical Sciences 3, no. 2 (2025): 1048–58. https://doi.org/10.5281/zenodo.14870725.

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Your heart is one of your body’s most important organs. Essentially a pump, the heart is a muscle made up of four chambers separated by valves and divided into two halves. Each half contains one chamber called an atrium and one called a ventricle. The atria (plural for atrium) collect blood, and the ventricles contract to push blood out of the heart. The right half of the heart pumps oxygen-poor blood (blood that has a low amount of oxygen) to the lungs where blood cells can obtain more oxygen. Then, the newly oxygenated blood travels from the lungs into the left atrium and the left vent

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14

Kalandarov, R. S., and L. L. Golovkina. "Blood groups and oncological diseases." Russian journal of hematology and transfusiology 66, no. 3 (2021): 417–23. http://dx.doi.org/10.35754/0234-5730-2021-66-3-417-423.

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Introduction. The study of the possible impact of the blood group system ABO and other antigenic systems of red blood cells for cancer patients is currently directed at a number of distinct paths, including the study of changes in the expression of a group of antigens in tumors; the appearance of new antigens on tumor cells; the risk of developing tumors depending on the blood group; the possible infl uence of blood group on the prognosis of the disease, etc.Aim — to examine the current state of the problem of the relationship between blood groups and cancer.Main findings. There is evidence th

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15

Mills, John A. "Inflammatory Diseases of Blood Vessels." Mayo Clinic Proceedings 77, no. 12 (2002): 1399. http://dx.doi.org/10.4065/77.12.1398.

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16

Hu, Mengyu, Yuxia Yang, Zhi Ji, and Jianyuan Luo. "RBM15 Functions in Blood Diseases." Current Cancer Drug Targets 16, no. 7 (2016): 579–85. http://dx.doi.org/10.2174/1568009616666160112105706.

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17

Hossfeld, D. K. "Neoplastic Diseases of the Blood." Annals of Oncology 16, no. 1 (2005): 175. http://dx.doi.org/10.1093/annonc/mdi005.

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18

Hashimoto, Ryota. "Blood biomarkers for neuropsychiatric diseases." Psychiatry and Clinical Neurosciences 72, no. 3 (2018): 139. http://dx.doi.org/10.1111/pcn.12624.

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19

Hokland, M., and P. Hokland. "Interferons for malignant blood diseases?" European Journal of Haematology 38, no. 3 (2009): 209–12. http://dx.doi.org/10.1111/j.1600-0609.1987.tb01165.x.

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20

Anderson, Kenneth C. "Neoplastic Diseases of the Blood." Mayo Clinic Proceedings 71, no. 8 (1996): 822. http://dx.doi.org/10.1016/s0025-6196(11)64851-9.

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21

McIntyre, O. Ross. "Neoplastic Diseases of the Blood." Mayo Clinic Proceedings 60, no. 6 (1985): 425. http://dx.doi.org/10.1016/s0025-6196(12)60855-6.

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22

Jokelainen, Kalle, Katja S. Salmela, Kari Humaloja, et al. "Blood dolichol in lysosomal diseases." Biochemistry and Cell Biology 70, no. 6 (1992): 481–85. http://dx.doi.org/10.1139/o92-074.

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Highly elevated serum total dolichol (free dolichol + dolichyl ester) concentrations have recently been found in two lysosomal storage diseases, aspartylglucosaminuria (AGU) and mannosidosis. The present study demonstrates that the increase of serum dolichol in AGU patients is caused by an increase of serum free dolichol. In 15 patients the mean serum level of free dolichol (227 ± 16 ng/mL) was 1.9 times higher (p < 0.001) than that in healthy controls (120 ± 6 ng/mL), while the amounts of dolichol fatty acid esters were similar in the patients and controls (110 ± 9 vs. 118 ± 6 ng/mL). In c

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23

Cheng, Tsung O. "Diseases That Lower Blood Pressure." Southern Medical Journal 82, no. 10 (1989): 1320. http://dx.doi.org/10.1097/00007611-198910000-00039.

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24

Geary, C. G. "Neoplastic Diseases of the Blood." British Journal of Cancer 53, no. 4 (1986): 581–82. http://dx.doi.org/10.1038/bjc.1986.95.

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25

BIEGUS, JAN, JAN SKÓRA, and ZBIGNIEW RYBAK. "Angiogenesis in blood vessels diseases." PRZEGLĄD FLEBOLOGICZNY 13, no. 5 (2005): 1–8. http://dx.doi.org/10.1066/s10003050036.

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26

KNUUTILA, SAKARI, MARTTI SIIMES, and PEKKA VUOPIO. "Chromosome pulverization in blood diseases." Hereditas 95, no. 1 (2009): 15–24. http://dx.doi.org/10.1111/j.1601-5223.1981.tb01323.x.

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27

Kohn, Donald B. "Gene therapy for blood diseases." Current Opinion in Biotechnology 60 (December 2019): 39–45. http://dx.doi.org/10.1016/j.copbio.2018.11.016.

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28

Hocking, William G. "Neoplastic Diseases of the Blood." JAMA: The Journal of the American Medical Association 267, no. 4 (1992): 580. http://dx.doi.org/10.1001/jama.1992.03480040132050.

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29

Hocking, William G. "Neoplastic Diseases of the Blood." JAMA: The Journal of the American Medical Association 254, no. 2 (1985): 280. http://dx.doi.org/10.1001/jama.1985.03360020114039.

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30

Nomura, Shosaku. "Extracellular vesicles and blood diseases." International Journal of Hematology 105, no. 4 (2017): 392–405. http://dx.doi.org/10.1007/s12185-017-2180-x.

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31

KARGALTSEVA, N. M., S. V. MALZEV, T. A. CHEBOTAREVA, and O. I. DANISHUK. "Blood microbiome with various diseases." Practical medicine 23, no. 1 (2025): 8–14. https://doi.org/10.32000/2072-1757-2025-1-8-14.

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Blood is considered sterile, but the 16S rRNA sequencing method has shown that the blood of healthy people contains bacterial DNA. Changes in the blood microbiome of newborns are detected when there is a suspicion of type 1 diabetes, autism, IgA nephropathy, obesity, celiac disease, and bloodstream infection. In these diseases, the blood microbiota is characterized by increased bacterial saturation and the presence of pathogenic microorganisms. The microbiota of the oral cavity and intestine is the cause of translocation and causes changes in the blood microbiota in young children. In type 1 d

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32

TOOYAMA, KAORU. "Apoptosis and internal medicine diseases. Blood diseases and apoptosis." Nihon Naika Gakkai Zasshi 86, no. 9 (1997): 1597–601. http://dx.doi.org/10.2169/naika.86.1597.

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33

UEDA, TAKANORI. "Blood transfusion therapy of internal medical field. Blood diseases and blood transfusion." Nihon Naika Gakkai Zasshi 85, no. 6 (1996): 857–61. http://dx.doi.org/10.2169/naika.85.857.

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34

Gregori, Luisa. "A diagnostic blood test for prion diseases." Blood 144, no. 18 (2024): 1853–54. http://dx.doi.org/10.1182/blood.2024026431.

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35

Dr., Hanan Javed Goreja Dr. Nouman Azam Dr. Efra Sikandar. "ANALYSIS OF RELATIONSHIP OF HIGH BLOOD PRESSURE AND HEART STROKE AMONG LOCAL POPULATION OF PAKISTAN: AN ENVIRONMENT BASED STUDY." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 06 (2018): 5129–32. https://doi.org/10.5281/zenodo.1288744.

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<strong><em>Introduction: </em></strong><em>High blood pressure</em><em> (HBP) is a leading major risk factor for </em><em>chronic diseases</em><em> and deaths. The prevalence of </em><em>patients with high blood pressure</em><em> (HT) had reached from 600 million in 1980 to one billion in 2008. <strong>Objectives of the study: </strong>The main objective of the study is to find the relationship of high blood pressure and heart stroke among local population of Pakistan. <strong>Methodology of the study: </strong>This study was conducted at health center of Rawalpindi,

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36

Dr., Waqar Khalid Dr. Amna Yaqoob Kanza Arooj. "ANALYSIS OF BURDEN OF HIGH BLOOD PRESSURE AND ITS IMPACT ON HEART STROKE AMONG LOCAL POPULATION OF PAKISTAN." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 08 (2018): 7984–88. https://doi.org/10.5281/zenodo.1404207.

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<strong><em>Introduction: </em></strong><em>High blood pressure (HBP) is a leading major risk factor for chronic diseases and deaths. The prevalence of patients with high blood pressure (HT) had reached from 600 million in 1980 to one billion in 2008. <strong>Objectives of the study: </strong>The main objective of the study is to find the relationship of high blood pressure and heart stroke among local population of Pakistan. <strong>Methodology of the study: </strong>This study was conducted at hospitals of Sargodha and Siddiqui Sadiq Memorial Trust Hospital</em> <em>

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37

Itzykson, Raphaël. "NPM1-mutated AML: how many diseases?" Blood 144, no. 7 (2024): 681–83. http://dx.doi.org/10.1182/blood.2024025032.

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38

Cildag, Songul, Yasemin Kara, and Taskin Senturk. "ABO blood groups and rheumatic diseases." European Journal of Rheumatology 4, no. 4 (2017): 250–53. http://dx.doi.org/10.5152/eurjrheum.2017.17044.

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39

Sato, N., S. Kawano, S. Tsuji, T. Ogihara, and S. Yamada. "Gastric Blood Flow in Ulcer Diseases." Scandinavian Journal of Gastroenterology 30, sup208 (1995): 14–20. http://dx.doi.org/10.3109/00365529509107756.

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40

Tomasik, Jaromir, and Grzegorz Władysław Basak. "Inflammasomes—New Contributors to Blood Diseases." International Journal of Molecular Sciences 23, no. 15 (2022): 8129. http://dx.doi.org/10.3390/ijms23158129.

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Inflammasomes are intracellular multimeric complexes that cleave the precursors of the IL-1 family of cytokines and various proteins, found predominantly in cells of hematopoietic origin. They consist of pattern-recognition receptors, adaptor domains, and the enzymatic caspase-1 domain. Inflammasomes become activated upon stimulation by various exogenous and endogenous agents, subsequently promoting and enhancing inflammatory responses. To date, their function has been associated with numerous pathologies. Most recently, many studies have focused on inflammasomes’ contribution to hematological

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41

Mustjoki, Satu. "Somatic mutations in “benign” blood diseases." Seminars in Hematology 59, no. 3 (2022): 121–22. http://dx.doi.org/10.1053/j.seminhematol.2022.08.003.

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42

Urabe, Akio. "Treatment of blood diseases and DDS." Drug Delivery System 19, no. 2 (2004): 90–94. http://dx.doi.org/10.2745/dds.19.90.

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43

Weatherall, D. J. "Prenatal Diagnosis of Inherited Blood Diseases." Clinics in Haematology 14, no. 3 (1985): 747–74. http://dx.doi.org/10.1016/s0308-2261(21)00503-8.

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44

Lang, Calvin A., Betty J. Mills, Walter Mastropaolo, and Marcia C. Liu. "Blood glutathione decreases in chronic diseases." Journal of Laboratory and Clinical Medicine 135, no. 5 (2000): 402–5. http://dx.doi.org/10.1067/mlc.2000.105977.

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45

Finch, Stuart C. "Book ReviewNeoplastic Diseases of the Blood." New England Journal of Medicine 312, no. 21 (1985): 1395. http://dx.doi.org/10.1056/nejm198505233122121.

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46

Fowlkes, Laura, and Jay M. Sullivan. "Estrogens, Blood Pressure, and Cardiovascular Diseases." Cardiology in Review 3, no. 2 (1995): 106–14. http://dx.doi.org/10.1097/00045415-199503000-00006.

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47

Zhang, Hanrui, Ciarán J. Mooney, and Muredach P. Reilly. "ABO Blood Groups and Cardiovascular Diseases." International Journal of Vascular Medicine 2012 (2012): 1–11. http://dx.doi.org/10.1155/2012/641917.

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ABO blood groups have been associated with various disease phenotypes, particularly cardiovascular diseases. Cardiovascular diseases are the most common causes of death in developed countries and their prevalence rate is rapidly growing in developing countries. There have been substantial historical associations between non-O blood group status and an increase in some cardiovascular disorders. Recent GWASs have identified ABO as a locus for thrombosis, myocardial infarction, and multiple cardiovascular risk biomarkers, refocusing attention on mechanisms and potential for clinical advances. As

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48

LI, HuiYuan, and RenChi YANG. "Research progress of hereditary blood diseases." SCIENTIA SINICA Vitae 47, no. 12 (2017): 1306–12. http://dx.doi.org/10.1360/n052017-00271.

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49

Peake, I. "The Molecular Basis of Blood Diseases." Journal of Medical Genetics 25, no. 11 (1988): 789–90. http://dx.doi.org/10.1136/jmg.25.11.789-a.

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50

Takeshita, Yukio, and Richard M. Ransohoff. "Blood-brain barrier and neurological diseases." Clinical and Experimental Neuroimmunology 6, no. 4 (2015): 351–61. http://dx.doi.org/10.1111/cen3.12229.

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