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Books on the topic 'Blood parasites'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 April 2022

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1

Kinnunen, Ronald E. Impact of sea lamprey parasitism on the blood features of hemopoietic tissues of rainbow trout. Ann Arbor, Mich: Great Lakes Fishery Commission, 1985.

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2

The Cure for all Diseases: With many case histories of diabetes, high blood pressure, seizures, chronic fatigue syndrome, migraines, Alzheimer's, Parkinson's, multiple sclerosis, and others showing that all of these can be simply investigated and cured. San Diego, CA: New Century Press, 1995.

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3

Clark, Hulda Regehr. The cure for all diseases: With many case histories of diabetes, high blood pressure, seizures, chronic fatigue syndrome, migraines, Alzheimer's, Parkinson's, multiple sclerosis, and others showing that all of these can be simply investigated and cured. San Diego, CA: ProMotion Pub., 1995.

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4

Yeovil, Jack. Orgy of the Blood Parasites. Pocket Books, 1994.

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5

Figures Of Medicine Blood Face Transplants Parasites. Fordham University Press, 2013.

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6

Figures Of Medicine Blood Face Transplants Parasites. Fordham University Press, 2013.

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7

Bilharzia hæmatobium: Blood fluke. [S.l: s.n., 1985.

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8

Miller, Louis. Immunity to Blood Parasites of Animals and Man. Springer, 2013.

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9

Miller, Louis. Immunity to Blood Parasites of Animals and Man. Springer, 2013.

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10

Siddall, Mark Edward. Fish blood parasites (apicomplexa: Adeleina) and their leech vectors: biology, philogeny and coevolution. 1994.

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11

Smith, James W. Blood and Tissue Parasites (Atlas of Diagnostic Medical Parasitology, Vol 1/Book and Slides). Amer Society of Clinical, 1985.

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12

C. M., Ph.D. Hawkey. A Colour Atlas of Comparative Veterinary Haematology: Normal and Abnormal Blood Cells in Mammals, Birds and Reptiles/Section on Blood Parasites. C.V. Mosby, 1989.

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13

Cox, F. E. G. Babesiosis and malaria. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0055.

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Babesiosis and malaria are rare zoonoses that, with new developments in diagnosis and the application of molecular techniques, are becoming increasingly frequently recognised. Babesia species infect millions of cattle and unknown numbers of sheep, dogs, horses, and wildlife throughout the world but human infections are very uncommon. There are two distinct forms of human babesiosis. In Europe the causative agent is Babesia divergens, a natural parasite of cattle transmitted by the tick Ixodes ricinis. B. divergens infections in humans are extremely rare and nearly all have been recorded from asplenic or otherwise immunocompromised patients. In the USA, human babesiosis is more common than in Europe, although still very rare, and is not restricted to immunocompromised individuals. The causative agents are Babesia microti and B. duncani, common parasites of rodents, transmitted by the tick Ixodes scapularis. In addition there have been sporadic reports of human babesiosis from other parts of the world but in most cases the species of Babesia involved has not been characterised. Malaria parasites and Babesia both inhabit red blood cells during part of their life cycles and these stages cause the diseases, malaria and babesiosis, which are similar in many respects. The facts that humans can occasionally acquire malaria and babesiosis from animals, that both parasites appear similar when seen in blood films and that both cause similar symptoms can cause problems in diagnosis and these rare infections are, therefore, of interest to clinicians and epidemiologists.
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14

Beattie, R. Mark, Anil Dhawan, and John W.L. Puntis. Bacterial, fungal, and parasitic infections of the liver. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569862.003.0059.

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Bacterial sepsis 428Spirochaetal infections 431Rickettsial infections 432Fungal infections 432Parasitic infections 434Granulomatous hepatitis 437Infectious agents can affect the liver either via direct invasion or by release of toxins. The liver's dual blood supply renders it uniquely susceptible to infection, receiving blood from the intestinal tract via the hepatic portal system, and from the systemic circulation via the hepatic artery. Because of this unique perfusion, the liver is frequently exposed to systemic or intestinal infections or the mediators of toxaemia. The biliary tree provides a further conduit for gut bacteria or parasites to access the liver parenchyma....
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15

Mistakes Were Made: Four Essays about a Young Writer Learning That the Road to Success Is Paved with Failure, Psychic Gurus and Blood-Sucking Parasites. Penguin Random House, 2015.

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16

E, Brecher Mark, and American Association of Blood Banks., eds. Bacterial and parasitic contamination of blood components. Bethesda, Md: AABB Press, 2003.

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17

Mueller, Dana. Malaria and Dengue Fever. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0052.

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Malaria is a vector-borne parasitic illness characterized by acute fever, headache, chills, and vomiting. Medications must target both the parasite’s active and inactive forms. During pregnancy, treatment regimens should consist of quinine and clindamycin. Person-to-person transmission can occur via sharing of blood products or during pregnancy. It is possible to contract malaria even while on prophylactic medications because resistance is widespread. Country-specific recommendations for prophylaxis can be found in the CDC’s annual Health Information for International Travel Protection against mosquito bites. Dengue Fever is a vector-borne viral infection that causes a flu-like illness with occasional lethal complications. It occurs primarily in tropical and subtropical regions. All treatment is supportive, ranging from oral rehydration to intravenous fluid administration and vasopressor support. Aspirin and NSAIDs are contraindicated in this population. Person-to-person transmission can occur via sharing of blood products or during pregnancy, although vertical transmission is rare.
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18

Barsoum, Rashad S. Schistosomiasis. Edited by Neil Sheerin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0181_update_001.

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AbstractSchistosomes are blood flukes that parasitize humans, apes, cattle, and other animals. In these definitive hosts they are bisexual, and lay eggs which are shed to fresh water where they complete an asexual cycle in different snails, ending in the release of cercariae which infect the definitive hosts to complete the life cycle.Seven of over 100 species of schistosomes are human pathogens, causing disease in different organs depending on the parasite species. Racial and genetic factors are involved in susceptibility, severity, and sequelae of infection.Morbidity is induced by the host’s immune response to schistosomal antigens. The latter include tegument, microsomal, gut, and oval antigens. The former are important in the process of invasion and establishment of infection, oval antigens in formation of granulomata which lead to fibrosis in different sites, and the gut antigens constitute the main circulating antigens in established infection, leading to immune-complex disease, particularly in the kidneys. The host immunological response includes innate and adaptive mechanisms, the former being the front line responsible for removing 90% of the infecting cercarial load. Adaptive immunity includes a Th1 phase, dominated by activation of an acute inflammatory response, followed by a prolonged Th2 phase which is responsible for immunity to re-infection as well as progression of tissue injury. Switching from Th1 to Th2 phases is controlled by functional and morphological change in the antigen-presenting cells, which is achieved by molecules of host as well as parasitic origin.Many cells participate in parasite killing, but also in the induction of tissue injury. The most potent of these is the eosinophil, which by binding antibodies to the parasite, particularly immunoglobulin E, facilitates parasite elimination. However, this process is complex, including agonist as well as antagonist pathways, which provide escape mechanisms for the parasite to survive, thereby achieving a delicate balance that permits schistosomes to live for decades in the infected host.
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19

Mavhunga, Clapperton Chakanetsa. The Mobile Workshop. The MIT Press, 2018. http://dx.doi.org/10.7551/mitpress/9780262535021.001.0001.

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The tsetse fly is a pan-African insect that bites an infective forest animal and ingests blood filled with invisible parasites, which it carries and transmits into cattle and people as it bites them, leading to n'gana (animal trypanosomiasis) and sleeping sickness. This book examines how the presence of the tsetse fly turned the forests of Zimbabwe and southern Africa into an open laboratory where African knowledge formed the basis of colonial tsetse control policies. The book traces the pestiferous work that an indefatigable, mobile insect does through its movements, and the work done by humans to control it. The book restores the central role not just of African labor but of African intellect in the production of knowledge about the tsetse fly. It describes how European colonizers built on and beyond this knowledge toward destructive and toxic methods, including cutting down entire forests, forced “prophylactic” resettlement, massive destruction of wild animals, and extensive spraying of organochlorine pesticides. Throughout, the book uses African terms to describe the African experience, taking vernacular concepts as starting points in writing a narrative of ruzivo (knowledge) rather than viewing Africa through foreign keywords.
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20

Garcia, Lynne S. Laboratory Diagnosis Of Blood-borne Parasitic Diseases, M15-a: Approved Guideline. NATIONAL COMMITTEE FOR CLINICAL LAB STANDARDS, 2000.

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21

Entropy in Bloom: Stories. Night Shade, 2017.

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22

Barsoum, Rashad S. Schistosomiasis. Edited by Neil Sheerin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0182_update_001.

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AbstractSchistosomiasis is a parasitic disease that affects millions of people in 78 countries, where it is held responsible for considerable morbidity and mortality. It is caused by a blood fluke, which provokes an immunological response to hundreds of its antigens. This induces multi-organ pathology through the formation of tissue granulomata or circulating immune complexes. In addition, it is amyloidogenic and carcinogenic, through the interaction of immunological perturbation with confounding metabolic and genetic factors. The primary targets of schistosomiasis are urinary and hepatointestinal.The lower urinary tract is mainly affected in S. haematobium infection, and may lead to chronic pyelonephritis and/or obstructive nephropathy. The colon and liver are the targets of S. mansoni and S. japonicum infection, leading to hepatic fibrosis, portal hypertension, and liver failure. S. mansoni may also lead to immune complex glomerulonephritis, which is discussed elsewhere. Both S. haematobium and S. mansoni ova may be carried with the venous circulation to the lungs, where they provoke granulomatous and immune-mediated endothelial injury leading to cor-pulmonale. Ova may be subsequently carried with the arterial circulation to form ‘metastatic’ granulomas in other tissues, notably the brain (S. japonicum), spinal cord (S. haematobium), skin, conjunctiva, and genital organs.Schistosomiasis is preventable. World Health Organization programmes have successfully eradicated or reduced the incidence of infection in many countries, particularly Egypt and China. Prevention strategies include health education, raising hygiene standards, and interruption of the parasite’s life cycle by snail control and mass treatment. The search for a vaccine continues. Effective antiparasitic treatment is now possible with high elimination rates. Available agents include praziquantel and artemether for all species, metrifonate for S. haematobium, and oxamniquine for S. mansoni. Successful outcome correlates with early intervention, before fibrosis has occurred.
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23

Kotton, Camille Nelson. Infection. Edited by Jeremy R. Chapman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0284_update_001.

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The immunosuppression which makes organ transplantation possible increases the risk of infections, both ordinary and opportunistic. The accurate diagnosis and management of infection after organ transplantation reduces morbidity and improves survival. Infections can be acquired in the hospital (i.e. nosocomial infections), from the transplant itself, from the blood product donor, from reactivation of latent infection in the host or from community exposure. Although viral infections are the most common, bacterial, fungal, and parasitic infections are also seen. While the intensity of immunosuppression is at its highest for a year after solid organ transplant, most opportunistic infections occur in the first 6 months.
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24

Schofield, C. J. American trypanosomosis (Chagas disease). Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0050.

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American trypanosomosis is due to infection with Trypanosoma cruzi (Protozoa, Kinetoplastidae). This is a widespread parasite of small mammals and marsupials throughout most of the Americas, roughly from the Great Lakes of North America (approx. 42 ° N) to southern Argentina (approx. 46 ° S). It is mainly transmitted by blood-sucking bugs of the subfamily Triatominae (Hemiptera, Reduviidae) which are widespread in the Americas, but rare in the Old World. Except in some research laboratories, and infected immigrants from Latin America, T.cruzi has not been reported from the Old World, although closely-related trypanosome species are commonly found in Old and New World bats.Human infection with T.cruzi is generally known as Chagas disease, taking the name of Brasilian clinician Carlos Justiniano das Chagas who first described it from patients in central Brasil (Chagas 1909). Chagas isolated and described the parasite, correctly deduced most of its life-cycle and clinical symptoms associated with the infection, identified the insect vectors and some of the reservoir hosts, and also trialed initial attempts to control it. He was nominated at least twice for the Nobel prize in medicine (Coutinho and Dias 2000; Lewinsohn 2003).Although difficult to treat, Chagas disease can be controlled by measures to halt transmission, primarily by eliminating domestic populations of the insect vectors, together with serological screening to avoid transmission by blood donation from infected donors. Since 1991, a series of multinational initiatives have used this approach to halt transmission over vast regions of the areas previously endemic for the human infection. Estimated prevalence of the human infection has declined from the 1990 estimate of 16–18 million people infected, to the current estimate of just over 7 million infected (OPS 2006; Schofield & Kabayo 2008). Prevalence is expected to decline further, and control strategies are now being adjusted to develop a sustainable system of disease surveillance, focal vector control, and specific treatment for any new cases (Schofield et al. 2006; WHO 2007). Guidance for diagnosis and treatment is also required for non-endemic countries, where recent years have seen increasing migration from Latin America such that cases of chronic Chagas disease have now been reported from amongst Latin American migrants in Europe, USA and Canada, and Japan, together with some congenital cases and transmission from infected blood donors and by organ transplant.
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25

Tobar, Ximena, and Shannon B. Putman. Viral Gastroenteritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0030.

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Viral gastroenteritis is a diarrheal disease associated with nausea, vomiting, headache, abdominal cramping, myalgias, and low-grade fever. Stools are often described as watery, with bouts of diarrhea and emesis that can occur on an hourly basis. Blood or mucus in the stool is suggestive of a bacterial or parasitic process. Additionally, the presence of fecal leukocytes excludes viral infection, as it is suggestive of colonic inflammation. Treatment is mainly supportive with appropriate hydration, including oral rehydration and/or intravenous fluids, being the key intervention. Specific antiviral agents are not available. Prevention and control of spread are important issues for viral gastroenteritis. Hand washing alone may reduce the spread of infection. The use of alcohol-based hand sanitizers and daily disinfection of surfaces with quaternary ammonium wipes has reduced the spread of Norovirus and was found superior to handwashing alone.
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26

Pediatric ICD-10-CM. American Academy of Pediatrics, 2015. http://dx.doi.org/10.1542/9781581109016.

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In this first edition, Pediatric ICD-10-CM: A Manual for Provider-Based Coding strives to bring to the pediatric provider, coder, and biller the most accurate and easy-to use manual on ICD-10-CM yet. Composed entirely with a pediatrics focus, this manual exclusively features codes and guidelines for physician- and provider-based coding, all in a simplified yet familiar format. The full draft of the ICD-10-CM code set comes in at well over 1,000 pages. This book condenses that large and potentially cumbersome book into 400 pages of the most relevant,pediatrics-related codes and guidelines. It also fully integrates into the tabular list specific chapter and code guidelines. Guideline are now included directly at the chapter and code level, ensuring that coders will always use the right codes in right circumstances Features include Integrated codes and guidelines Simplified yet familiar layout Tabular and indexed navigation Pediatric-focused and provider-based guidance And more... Contents Include: ICD-10-CM Official Guidelines for Coding and Reporting: FY 2015 Certain Infectious and Parasitic Diseases (A00-B99) Neoplasms (C00-D49) Diseases of the Blood and Blood-Forming Organs and Certain Disorders Involving the Immune Mechanism (D50-D89) Endocrine, Nutritional and Metabolic Diseases (E00-E89) Mental, Behavioral and Neurodevelopmental Disorders (F01-F99) Diseases of the Nervous System (G00-G99) Diseases of the Eye and Adnexa (H00-H59) Diseases of the Ear and Mastoid Process (H60-H95) Diseases of the Circulatory System (I00-I99) Diseases of the Respiratory System (J00-J99) Diseases of the Digestive System (K00-K95) Diseases of the Skin and Subcutaneous Tissue (L00-L99) Diseases of the Musculoskeletal System and Connective Tissue (M00-M99) Diseases of the Genitourinary System (N00-N99) Pregnancy, Childbirth, Certain Conditions Originating in the Perinatal Period (P00-P99) Congenital Malformations, Deformations and Chromosomal Abnormalities (Q00-Q99) Symptoms, Signs, and Abnormal Clinical and Laboratory Findings (R00-R99) Injury, Poisoning and Consequences of Certain Other External Causes (S00-T88) External Causes of Morbidity (V00-Y99) Factors Influencing Health Status and Contact With Health Services (Z00-Z99)
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27

Jenkins, Graham Curtis, and J. D. Williams. Infection and Hematology. Butterworth-Heinemann, 1994.

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28

C, Jenkins George, and Williams, J. D. 1931 Mar.-, eds. Infection and haematology. Oxford: Butterworth-Heinemann, 1994.

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