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Books on the topic 'Blood pumps'

Author: Grafiati
Published: 4 June 2021
Last updated: 13 February 2022

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1

Matsuda, Hikaru, ed. Rotary Blood Pumps. Tokyo: Springer Japan, 2000. http://dx.doi.org/10.1007/978-4-431-67917-2.

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2

Kaufman, Francine Ratner. Insulin pumps and continuous glucose monitoring. Alexandria, Va: American Diabetes Assocaiation, 2012.

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3

Gardner, A. M. N. The return of blood to the heart: Venous pumps in health and disease. London: J. Libbey, 1989.

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4

Gardner, A. M. N. The return of blood to the heart: Venous pumps in health and disease. 2nd ed. London: Libbey, 1993.

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5

Gardner, A. M. N. The return of blood to the heart: Venous pumps in health and disease. London: Libbey, 1989.

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6

Tyrone, Fernando, ed. Closed-loop control of blood glucose. Berlin: Springer, 2007.

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7

Steve, Parker. Pump it up!: Respiration and circulation. Chicago, Ill: Raintree, 2007.

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8

Steve, Parker. Pump it up!: Respiration and circulation. Chicago, Ill: Raintree, 2006.

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9

Stewart, Melissa. Pump it up!: The secrets of the heart and blood. New York: Marshall Cavendish Benchmark, 2010.

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10

Clinical application of intra-aortic balloon pump. 3rd ed. Armonk, N.Y: Futura Pub. Co., 1998.

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11

Association, American Diabetes, ed. American Diabetes Association guide to medical nutrition therapy for diabetes. 2nd ed. Alexandria, Va: American Diabetes Association, 2012.

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12

Matsuda, Hikaru. Rotary Blood Pumps: New Developments and Current Applications. Springer, 2007.

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13

Rotary Blood Pumps: New Developments and Current Applications. Springer, 2012.

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14

Matsuda, Hikaru. Rotary Blood Pumps: New Developments and Current Applications. Springer, 2012.

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15

Hikaru, Matsuda, ed. Rotary blood pumps: New developments and current applications. Tokyo: Springer, 2000.

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16

Wiffen, Philip, Marc Mitchell, Melanie Snelling, and Nicola Stoner. Therapy-related issues: nutrition and blood. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199603640.003.0024.

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Administration sets 498Intravenous (IV) administration pumps and other devices 500Management of magnesium imbalance 504Management of phosphate imbalance 506Management of hypokalaemia 508Guidelines for the treatment of hypocalcaemia 512Prescribing IV fluids 514Nutritional support in adults 519Normal nutritional requirements 520...
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17

Hussain, Syed Sufyan. Insulin pumps and continuous glucose monitoring made easy. 2016.

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18

P, Veres Joseph, and United States. National Aeronautics and Space Administration., eds. Flow analysis of the Cleveland clinic centrifugal pump. [Washington, DC]: National Aeronautics and Space Administration, 1997.

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19

author, Westfall Emily, and American Diabetes Association, eds. Insulin pumps and continuous glucose monitoring: A user's guide to effective diabetes management. 2nd ed. American Diabetes Association, 2017.

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20

Meyns, Bart. Ventricular Support With Miniature Rotary Blood Pumps: An Experimental Study (Acta Biomedica Lovaniensia , No 145). Coronet Books Inc, 1997.

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21

Staff, American Diabetes Association. Practical Insulin: A Handbook for Prescribing Providers. American Diabetes Association, 2015.

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22

Waberski, Andrew T., and Nina Deutsch. Transposition of the Great Arteries. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0010.

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Transposition of the great arteries is a congenital cardiac abnormality that presents in the neonatal period, most commonly as cyanosis. While variations in anatomic features exist, dextro-transposition of the great arteries, the most common form, results in 2 separate circulatory systems in parallel, such that the right ventricle pumps deoxygenated blood to the systemic circulation, and the left ventricle sends oxygenated blood back to the pulmonary circulation. To ensure survival, early diagnosis and intervention to allow for adequate mixing of blood is necessary. The arterial switch operation is the definitive treatment, usually undertaken in the first few days of life. Known complications of surgery include ischemia, bleeding, hemodynamic compromise, and arrhythmias. Anesthetic management must take these factors into account.
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23

Randolph, Joanne. Pump It Up: The Heart and Blood. Enslow Publishing, 2017.

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24

Hulse, Ed. The Blood 'n' Thunder Guide to Collecting Pulps. Murania Press, 2009.

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25

McKenzie, Ian. Single Ventricle Physiology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199764495.003.0031.

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Congenital cardiac abnormalities in which there is functionally only a single ventricle are a heterogeneous group of conditions. These include patients with marked hypoplasia of one ventricle, usually with hypoplasia or atresia of the inflow of the ventricle, such as in hypoplastic left heart syndrome or conditions where surgical separation of the flow to each ventricle is not possible, such as double-inlet left ventricle. The most common pathway for palliating these conditions will be to use cavopulmonary connections to provide lung blood flow direct from systemic venous return (reliant on systemic venous pressure). The single ventricle pumps to the systemic arterial circulation. Many of these patients will be long-term survivors and present with acute surgical conditions unrelated to their cardiac condition. The safe anesthesia management of patients with single ventricle physiology and cavopulmonary connections involves assessing their cardiovascular reserve and understanding the effects of hypovolemia, anesthesia, positive-pressure ventilation, and the procedure itself on their circulation.
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26

Timperley, Jonathan, and Sandeep Hothi. Hypotension. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0011.

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Hypotension is defined as a systolic arterial blood pressure of less than 90 mm Hg, or a diastolic arterial pressure of less than 60 mm Hg, and may lead to shock, with clinical evidence of inadequate blood supply to critical organs. It can be due to hypovolaemia, cardiac pump failure, or vasodilatation. This chapter describes the clinical approach to patient with hypotension.
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27

Little, Jean. Weird Vampire Tales: 30 Blood-Chilling Stories from the Weird Fiction Pulps. Gramercy, 1992.

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28

E, Weinberg Robert, Dziemianowicz Stefan R, and Greenberg Martin Harry, eds. Weird vampire tales: 30 blood-chilling stories from the weird fiction pulps. New York: Gramercy Books, 1992.

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29

Magder, Sheldon. Central venous pressure monitoring in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0132.

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Central venous pressure (CVP) is at the crucial intersection of the force returning blood to the heart and the force produced by cardiac function, which drives the blood back to the systemic circulation. The normal range of CVP is small so that before using it one must ensure proper measurement, specifically the reference level. A useful approach to hypotension is to first determine if arterial pressure is low because of a decrease in vascular resistance or a decrease in cardiac output. This is done by either measuring cardiac output or making a clinical assessment blood flow. If the cardiac output is decreased, next determine whether this is because of a cardiac pump problem or a return problem. It is at this stage that the CVP is most helpful for these options can be separated by considering the actual CVP or even better, how it changed with the change in cardiac output. A high CVP is indicative of a primary pump problem, and a low CVP and return problem. Understanding the factors that determine CVP magnitude, mechanisms that produce the components of the CVP wave form and changes in CVP with respiratory efforts can also provide useful clinical information. In many patients, CVP can be estimated on physical exam.
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30

Dreval, Alexander. Professional and flash on the monitoring of blood glucose levels of insulin pump therapy and without it. Aegitas publishing house, 2021. http://dx.doi.org/10.47359/978-0-369-40455-8.

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A new method of self-control of diabetes based on the results of continuous monitoring of glycemia (HMG) is especially relevant in patients who are on pump insulin therapy, especially since the start of pump insulin therapy is carried out with the mandatory installation of the HMG system [1,3]. Due to the novelty of these two methods (treatment of diabetes and control of glycemia) for a wide clinical practice, there is an urgent need to publish concise practical guides on this topic for doctors, both for self-study of these methods, and for advanced training courses. Based on the above and our experience of teaching at the Department of Endocrinology of the Federal Medical University of MONICA, this guide has been prepared, which will be useful, first of all, for endocrinologists, therapists working with patients with diabetes, as well as for senior students of medical institutes who are interested in new directions in practical medicine.
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31

Zhang, Peng-Fei, Yun Zhang, and Siew Yen Ho. Left ventricle: morphology and geometry. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0018.

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The left ventricle is a cone-shaped muscular pump which receives the blood from the left atrium through the inflow tract and ejects it to the aorta through the outflow tract. The double helical myocardial fibre formation is the basis of efficient motion, function, and morphology of the left ventricle. Physiological or pathological changes of these characteristics of the left ventricle can be evaluated by echocardiography. This chapter describes the morphology and geometry of the left ventricle, including the inflow tract, the outflow tract, double helix formation of left ventricle myocardium, and the echocardiographic assessment of left ventricle morphology and geometry.
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32

Stevens, David C., and Sabah Butty. Tips and Tricks of the AngioVac Device. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0039.

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The AngioVac system, which consists of a coil-reinforced large-bore cannula, bypass circuit, bubble trap/filter, and reinfusion cannula, allows percutaneous removal of unwanted vascular debris, such as venous thrombus or cardiac vegetations, during veno-veno bypass. External suction is applied via a centrifugal bypass pump and debris is funneled into the cannula and trapped in the bubble trap/filter. The blood is then returned through an 18 Fr venous reinfusion cannula. The use of the device in the iliocaval venous segments and right heart is effective and safe. Due to the challenging anatomy, pulmonary artery thrombectomy carries an increased risk of complication and should be undertaken with great care. The AngioVac system is a versatile tool for removing thrombus and other unwanted debris from the central venous system and the right heart.
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33

Tourneau, Thierry Le, Luis Caballero, and Tsai Wei-Chuan. Right atrium. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0024.

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The right atrium (RA) is located on the upper right-hand side of the heart and has relatively thin walls. From an anatomical point of view, the RA comprises three basic parts, the appendage, the vestibule of the tricuspid valve, and the venous component (superior and inferior vena cava, and the coronary sinus) receiving the deoxygenated blood. The RA is a dynamic structure dedicated to receive blood and to assist right ventricular (RV) filling. The three components of atrial function are the reservoir function during ventricular systole, the conduit function which consists in passive blood transfer from veins to the RV in diastole, and the booster pump function in relation to atrial contraction in late diastole to complete ventricular filling. Right atrial function depends on cardiac rhythm (sinus or atrial fibrillation), pericardial integrity, RV load and function, and tricuspid function. Right atrial dimension assessment is limited in two-dimensional (2D) echocardiography. Right atrial planimetry in the apical four-chamber view is commonly used with an upper normal value of 18-20 cm2. Minor and major diameters can also be measured. Three-dimensional (3D) echocardiography could overcome the limitation of conventional echocardiography in assessing RA size. Right atrial function has been poorly explored by echocardiography both in physiological and pathological contexts. Although tricuspid inflow and tissue Doppler imaging of tricuspid annulus can be used in the exploration of RA function, 2D speckle tracking and 3D echocardiography appear promising tools to dissect RA function and to overcome the limitations of standard echocardiography.
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34

van Hooijdonk, Roosmarijn T. M., and Marcus J. Schultz. Insulin and oral anti-hyperglycaemic agents in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0050.

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Dysglycaemia is frequently seen in the intensive care unit (ICU). Hyperglycaemia, hypoglycaemia and glycaemic variability are all independently associated with mortality and morbidity in critically-ill patients. It is common practice to treat hypergycaemia in these patients, while at the same time preventing hypoglycaemia and glycaemic variability. Insulin infusion is preferred over oral anti–hyperglycaemic agents for glucose control in the ICU because of the highly unpredictable biological availability of oral anti-hyperglycaemic agents during critical illness. Many oral anti–hyperglycaemic agents are relatively contraindicated in critically-ill patients. Intravenously-administered insulin has a predictable effect on blood glucose levels, in particular because of its short half-life. Notably, effective and safe insulin titration requires frequent blood glucose measurements, a dedicated lumen of a central venous catheter for infusion of insulin, an accurate syringe pump, and trained nurses for delicate adoptions of the infusion rate. Insulin infusion increases the risk of hypoglycaemia, which should be prevented at all times. In addition, precautions should be taken against overcorrection of hypoglycaemia, using only small amounts of glucose. Whether glycaemic variability can be kept minimal is uncertain. Use of continuous glucose measuring devices has the potential to improve glycaemic control in critically-ill patients.
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35

Combes, Alain, and Nicolas Bréchot. Intra-aortic balloon counterpulsation in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0153.

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The intra-aortic balloon pump (IABP) is a mechanical device consisting of a cylindrical polyethylene balloon that sits in the aorta, approximately 2 cm from the left subclavian artery. A computer-controlled console linked to either an electrocardiogramor a pressure transducer inflates the balloon with helium during diastole (counterpulsation) and actively deflates in systole. This results in an increase in coronary artery blood flow and cardiac output, and reduced left ventricular afterload. These actions combine to decrease myocardial oxygen demand and increase supply. Major complications include bleeding at the insertion site and retroperitoneal haemorrhage, critical ischaemia of the catheterized leg, catheter infection, and stroke. IABP duration usually varies from 48 to 72 hours. Weaning from IABP is not well defined; the most common approach is to reduce cycling of inflation to 1:2 or 1:4 for 15 minutes to several hours before device removal.
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36

Oostra, Roelof-Jan, Bjarke Jensen, and Antoon F. M. Moorman. An evolutionary perspective on the origin of the cardiovascular system of vertebrates. Edited by José Maria Pérez-Pomares, Robert G. Kelly, Maurice van den Hoff, José Luis de la Pompa, David Sedmera, Cristina Basso, and Deborah Henderson. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757269.003.0002.

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The origin of the cardiovascular system of vertebrates is inferred from comparisons of basal chordates but must also encompass bewildering discrepancies. Basal chordates like lancelets (cephalochordates) have a vascular pattern similar to that of a vertebrate embryo, but without a recognizable heart or myocardium. Instead, the ‘venous’ part of their circulation contains contractile vessels, located upstream and downstream of the liver. Tunicates (urochordates) have a tubular heart containing cardiomyocytes and enclosed by a pericardium. Their circulation is open and the dominant pacemaker activity can be at either end of the heart tube, causing blood flow to reverse periodically. Recent molecular investigations have proved that urochordates rather than cephalochordates are the closest living relatives of vertebrates. This implies that the cardiovascular peculiarities of lancelets may be primitive ancestral qualities and that the original building plan of the vertebrate circulation featured a post-hepatic as well as a pre-hepatic cardiac pump.
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37

Pirani, Tasneem, and Tony Rahman. Diagnosis and management of upper gastrointestinal haemorrhage in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0177.

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Upper gastrointestinal haemorrhage is a medical emergency that may present with haematemesis and/or melena. An exhaustive history and careful examination aids in identifying the cause of bleeding and directing appropriate management. Validated scoring systems exist to guide the urgency of endoscopic therapy, although these should not be used in isolation, but in conjunction with complete patient assessment. The initial priority should be to resuscitate and stabilize the patient using the airway, breathing, circulation, and disability framework. Resuscitation should be guided by clinical and physiological parameters. Patients should be managed in an environment where vital signs such as heart rate, blood pressure, respiratory rate, conscious level, and urine output are monitored at least hourly. Attempts should be made to correct coagulopathy. Specialist advice should be sought from haematologists for guidance on the most appropriate use of packed red cells and blood products. Over-transfusion should be avoided. Initiation of pre-endoscopy proton pump inhibitor therapy, in particular to avoid definitive endoscopic therapy, is not recommended. Diagnostic endoscopy and therapy should be conducted within 24 hours of presentation. Numerous endoscopic therapies exist—when epinephrine is used for local tamponade and vasoconstriction, application of dual modality treatment is recommended. In cases where endoscopic therapy fails or is not possible, radiological diagnosis, and embolization may become necessary. Occasionally, surgery is required for definitive treatment—close liaison with surgeons is therefore necessary, especially where initial endoscopy is considered suboptimal or re-bleeding occurs.
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38

Cheong, Adrian, Gabriel Steg, and Stefan K. James. ST-segment elevation myocardial infarction. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0043.

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Acute myocardial infarction with ST-segment elevation is a common and dramatic manifestation of coronary artery disease. It is caused by the rupture of an atherosclerotic plaque in a coronary artery, leading to its total thrombotic occlusion and resultant ischaemia and necrosis of downstream myocardium. The diagnosis of ST-segment elevation myocardial infarction is based on a syndrome of ischaemic chest pain symptoms, associated with typical ST-segment elevation on the electrocardiogram and an eventual rise in biomarkers of myocardial necrosis. The treatment of ST-segment elevation myocardial infarction is focused on re-establishing blood flow in the coronary artery involved, preferably by percutaneous coronary intervention, or by pharmacological thrombolysis in the case of expected lengthy time delays or lack of availability of facilities. Early mortality from ST-segment elevation myocardial infarction can be attributed to the sequelae or complications of myocardial ischaemia, or complications related to therapy. The former include arrhythmias (such as ventricular tachycardia or fibrillation), mechanical complications (such as ventricular free wall, septal, and mitral chordal rupture), and pump failure leading to cardiogenic shock. The latter includes haemorrhagic complications and coronary stent thrombosis. Given that myocardial necrosis is a critically time-dependent process, the organization of an ST-segment elevation myocardial infarction care system and adherence to the latest clinical trial evidence and guidelines are crucial to ensure that patients are treated in an optimal manner.
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39

Cheong, Adrian P., Gabriel Steg, and Stefan K. James. ST-segment elevation MI. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0043_update_001.

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Acute myocardial infarction with ST-segment elevation is a common and dramatic manifestation of coronary artery disease. It is caused by the rupture of an atherosclerotic plaque in a coronary artery, leading to its total thrombotic occlusion and resultant ischaemia and necrosis of downstream myocardium. The diagnosis of ST-segment elevation myocardial infarction is based on a syndrome of ischaemic chest pain symptoms, associated with typical ST-segment elevation on the electrocardiogram and an eventual rise in biomarkers of myocardial necrosis. The treatment of ST-segment elevation myocardial infarction is focused on re-establishing blood flow in the coronary artery involved, preferably by percutaneous coronary intervention, or by pharmacological thrombolysis in the case of expected lengthy time delays or lack of availability of facilities. Early mortality from ST-segment elevation myocardial infarction can be attributed to the sequelae or complications of myocardial ischaemia, or complications related to therapy. The former include arrhythmias (such as ventricular tachycardia or fibrillation), mechanical complications (such as ventricular free wall, septal, and mitral chordal rupture), and pump failure leading to cardiogenic shock. The latter includes haemorrhagic complications and coronary stent thrombosis. Given that myocardial necrosis is a critically time-dependent process, the organization of an ST-segment elevation myocardial infarction care system and adherence to the latest clinical trial evidence and guidelines are crucial to ensure that patients are treated in an optimal manner.
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40

J, Franz Marion, Bantle John P. 1947-, and American Diabetes Association, eds. American Diabetes Association guide to medical nutrition therapy for diabetes. Alexandria, Va: American Diabetes Association, 1999.

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41

Association, American Diabetes. American Diabetes Association Guide to Medical Nutrition Therapy for Diabetes (Clinical Education Series). American Diabetes Association, 2003.

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