Relevant bibliographies by topics / Bone formation/resorption

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Bone formation/resorption'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Bone formation/resorption"

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Li, Binbin, and Shifeng Yu. "Genistein Prevents Bone Resorption Diseases by Inhibiting Bone Resorption and Stimulating Bone Formation." Biological & Pharmaceutical Bulletin 26, no. 6 (2003): 780–86. http://dx.doi.org/10.1248/bpb.26.780.

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Slootweg, M. C., W. W. Most, E. van Beek, L. P. C. Schot, S. E. Papapoulos, and C. W. G. M. Löwik. "Osteoclast formation together with interleukin-6 production in mouse long bones is increased by insulin-like growth factor-I." Journal of Endocrinology 132, no. 3 (March 1992): 433–38. http://dx.doi.org/10.1677/joe.0.1320433.

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ABSTRACT Insulin-like growth factor-I (IGF-I) is a potent stimulator of bone formation. Whether this growth factor also induces bone resorption has not been studied in detail. We used two organ culture systems to examine the direct effect of IGF-I on bone resorption. Fetal mouse radii/ulnae, containing mature osteoclasts, showed no response to IGF-I, indicating that osteoclastic activity is not influenced by IGF-I. Fetal mouse metacarpals/metatarsals, containing just osteoclast precursors and progenitors, showed an increase in resorption in response to IGF-I, indicating that IGF-I stimulates t
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Yavropoulou, Maria P., Helen P. Vafiadou, Olympia E. Anastasiou, Vasiliki Tsavdaridou, Georgia H. Kokaraki, and John G. Yovos. "Pioglitazone affects bone resorption but not bone formation." Bone 42 (March 2008): S91. http://dx.doi.org/10.1016/j.bone.2007.12.173.

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Kitaura, Hideki, Aseel Marahleh, Fumitoshi Ohori, Takahiro Noguchi, Wei-Ren Shen, Jiawei Qi, Yasuhiko Nara, Adya Pramusita, Ria Kinjo, and Itaru Mizoguchi. "Osteocyte-Related Cytokines Regulate Osteoclast Formation and Bone Resorption." International Journal of Molecular Sciences 21, no. 14 (July 21, 2020): 5169. http://dx.doi.org/10.3390/ijms21145169.

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The process of bone remodeling is the result of the regulated balance between bone cell populations, namely bone-forming osteoblasts, bone-resorbing osteoclasts, and the osteocyte, the mechanosensory cell type. Osteoclasts derived from the hematopoietic stem cell lineage are the principal cells involved in bone resorption. In osteolytic diseases such as rheumatoid arthritis, periodontitis, and osteoporosis, the balance is lost and changes in favor of bone resorption. Therefore, it is vital to elucidate the mechanisms of osteoclast formation and bone resorption. It has been reported that osteoc
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Franco, Leonardo, and Mario Alejandro Ortíz Salazar. "Biochemical markers of bone metabolism." Revista Estomatología 18, no. 1 (September 28, 2017): 30–34. http://dx.doi.org/10.25100/re.v18i1.5707.

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The quantity and quality of bone tissue renewal are dependent on the generation of new bone (deposition) mediated by osteoblasts and the loss (resorption) mediated by osteoclasts. For each of these processes there are important markers that can be measured in serum or urine.
 Resorption markers are products of metabolic degradation of bone matrix in particu-lar of the type I collagen (hydroxyproline, pyridinoline and deoxypyridinoline). In addition, the resorptive activity can also be evaluated through the tartrate resistant acid phosphatase (TRAP) and calcium-creatinine ratio in urine. B
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Erjavec, Igor, Jelena Brkljacic, Slobodan Vukicevic, Boris Jakopovic, and Ivan Jakopovich. "Mushroom Extracts Decrease Bone Resorption and Improve Bone Formation." International Journal of Medicinal Mushrooms 18, no. 7 (2016): 559–69. http://dx.doi.org/10.1615/intjmedmushrooms.v18.i7.10.

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Yamaguchi, Masayoshi. "Role of zinc in bone formation and bone resorption." Journal of Trace Elements in Experimental Medicine 11, no. 2-3 (1998): 119–35. http://dx.doi.org/10.1002/(sici)1520-670x(1998)11:2/3<119::aid-jtra5>3.0.co;2-3.

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Buckland, Jenny. "Dual role for Wnt4: bone formation and bone resorption." Nature Reviews Rheumatology 10, no. 10 (August 26, 2014): 575. http://dx.doi.org/10.1038/nrrheum.2014.146.

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Hirayama, T., A. Sabokbar, and NA Athanasou. "Effect of corticosteroids on human osteoclast formation and activity." Journal of Endocrinology 175, no. 1 (October 1, 2002): 155–63. http://dx.doi.org/10.1677/joe.0.1750155.

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Chronic corticosteroid treatment is known to induce bone loss and osteoporosis. Osteoclasts are specialised bone-resorbing cells that are formed from mononuclear phagocyte precursors that circulate in the monocyte fraction. In this study we have examined the effect of the synthetic glucocorticoid, dexamethasone, on human osteoclast formation and bone-resorbing activity. Human monocytes were cultured for up to 21 days on glass coverslips and dentine slices, with soluble receptor activator for nuclear factor kappaB ligand (RANKL; 30 ng/ml) and human macrophage-colony stimulating factor (M-CSF; 2
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Lean, J. M., J. W. M. Chow, and T. J. Chambers. "The rate of cancellous bone formation falls immediately after ovariectomy in the rat." Journal of Endocrinology 142, no. 1 (July 1994): 119–25. http://dx.doi.org/10.1677/joe.0.1420119.

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Abstract We have recently found that administration of oestradiol-17β (OE2) to rats stimulates trabecular bone formation. It is not known, however, whether oestrogen has a similar action on bone formation rate under physiological circumstances. Oestrogen is known to suppress bone resorption, and oestrogen-deficient states in the rat, as in humans, are associated with an increase in bone resorption that entrains an increase in bone formation. To see if the latter masks a relative reduction in bone formation, due to oestrogen deficiency, we measured bone formation very early after ovariectomy, b
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Dissertations / Theses on the topic "Bone formation/resorption"

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Moroz, Adam. "Reduced order modelling of bone resorption and formation." Thesis, De Montfort University, 2011. http://hdl.handle.net/2086/5409.

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The bone remodelling process, performed by the Bone Multicellular Unit (BMU) is a key multi-hierarchically regulated process, which provides and supports various functionality of bone tissue. It is also plays a critical role in bone disorders, as well as bone tissue healing following damage. Improved modelling of bone turnover processes could play a significant role in helping to understand the underlying cause of bone disorders and thus develop more effective treatment methods. Moreover, despite extensive research in the field of bone tissue engineering, bonescaffold development is still very
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Lean, Jennifer Maree. "Mechanical stimulation of bone formation in the rat." Thesis, St George's, University of London, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263682.

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Miao, Dengshun. "Studies on the actions of bone anabolic drugs in vivo and in vitro." Thesis, University of Sheffield, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.300362.

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Althnaian, Thnaian Ali. "Factors that regulate osteoclast formation and bone resorption in regenerating deer antlers." Thesis, Royal Veterinary College (University of London), 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.439832.

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Davey, Tamara. "Functional characterisation of a novel osteoclast-derived factor." University of Western Australia. School of Surgery and Pathology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0219.

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[Truncated abstract] Intracellular communication between osteoclasts and osteoblasts is imperative to maintain bone integrity. A myriad of molecules are responsible for regulating osteoblast and osteoclast activity. In particular, it is well documented that osteoblast-derived factors are crucial in directly controlling osteoclast formation and function. Since bone formation is coupled to bone resorption, it would be expected that osteoclasts also have some role in regulating the growth and function of osteoblast cells. However, despite extensive research upon osteoclast and osteoblast biology,
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Loomer, Peter Michael. "The direct effects of Porphyromonas gingivalis 2561 on bone formation and mineral resorption in vitro." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp02/NQ27685.pdf.

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Owens, Beatrice H. "Dose-dependent effects of salmon calcitonin on bone turnover in ovariectomized rats." [Johnson City, Tenn. : East Tennessee State University], 2004. http://etd-submit.etsu.edu/etd/theses/available/etd-1120104-223525/unrestricted/OwensB120204f.pdf.

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Thesis (Ph.D.)--East Tennessee State University, 2004.<br>Title from electronic submission form. ETSU ETD database URN: etd-1120104-223525 Includes bibliographical references. Also available via Internet at the UMI web site.
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Kassem, Ali. "Toll-like receptors (TLRs) and inflammatory bone modeling." Doctoral thesis, Umeå universitet, Institutionen för odontologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-110296.

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Patients with inflammatory or infectious conditions such as periodontitis, peri-implantitis, osteomyelitis, rheumatoid arthritis, septic arthritis and loosened joint prosthesis display varying severity of destruction in the adjacent bone tissue. Bone loss in inflammatory diseases is considered a consequence of cytokine induced RANKL and subsequent enhanced osteoclast formation. Hence, osteotropic cytokines and their receptors have been suggested to be important for the pathogenesis of inflammation-induced osteolysis. It is, here, suggested that bacterial components, so called “pathogen associa
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Lotz, Ethan M. "Designing Biomimetic Implant Surfaces to Promote Osseointegration under Osteoporotic Conditions by Revitalizing Mechanisms Coupling Bone Resorption to Formation." VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/5908.

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In cases of compromised bone remodeling like osteoporosis, insufficient osseointegration occurs and results in implant failure. Implant retention relies on proper secondary fixation, which is developed during bone remodeling. This process is disrupted in metastatic bone diseases like osteoporosis. Osteoporosis is characterized low bone mass and bone strength resulting from either accelerated osteoclast-mediated bone resorption or impaired osteoblast-mediated bone formation. These two processes are not independent phenomena. In fact, osteoporosis can be viewed as a breakdown of the cellular com
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Mbimba, Thomas S. Jr. "TRAFICKING PROTEIN PARTICLE COMPLEX (TRAPPC) -9:A NOVEL PROTEIN REGULATOR OF NF-kB MEDIATED BONE FORMATION AND RESORPTION." Kent State University / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=kent1448841594.

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Books on the topic "Bone formation/resorption"

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Loomer, Peter Michael. The direct effects of Porphyromonas Gingivalis 2561 on bone formation and mineral resorption in vitro. [Toronto: University of Toronto, Faculty of Dentistry], 1997.

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Tai, Victoria. The effects of leukotriene Bb4s on osteoclast formation and osteoclastic bone resorption and the role of osteoblastic cells in these processes. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1999.

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Pharmacological Approach to the Study of the Formation and the Resorption Mechanism of Hard Tissues. MEDICO DENTAL MEDIA INTERNATIONAL, 1994.

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Effects of prostaglandin Eb2s on chick bone rudiments in vitro: Studies on bone resorption and formation. Ottawa: National Library of Canada, 1990.

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Wordsworth, B. P. Skeletal dysplasias. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0150.

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Bone is metabolically active throughout life and metabolic disturbances may have wide-ranging consequences that are not restricted to altering its mechanics. The study of some genetic bone diseases has already provided remarkable insights into the normal regulation of bone metabolism. Skeletal dysplasias are developmental disorders of the chondro-osseous tissues commonly resulting in short stature, which is often disproportionate. The underlying mutations are often in the structural genes encoding components of the matrix but may also involve growth factors or cell signalling. In contrast, the
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Javaid, Kassim. Paget’s disease of bone. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0274.

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Paget’s disease of bone is an uncommon bone disorder with increased bone resorption and disorganized bone formation of woven bone. Its cause is unclear; there is a clear genetic component but additional environmental factors are important, given the reduction in severity and prevalence in the UK. Paget’s disease is usually asymptomatic and detected by an unexplained raised alkaline phosphatase on routine biochemistry. Symptoms include focal bone pain, including headache. Other symptoms include bone deformity and complications such as fracture and nerve conduction. Paget’s disease can sometimes
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Sprague, Stuart M., and James M. Pullman. Spectrum of bone pathologies in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0122.

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Histologic bone abnormalities begin very early in the course of chronic kidney disease. The KDIGO guidelines recommend that bone disease in patients with chronic kidney disease should be diagnosed on the basis of bone biopsy examination, with bone histomorphometry. They have also proposed a new classification system (TMV), using three key features of bone histology—turnover, mineralization, and volume—to describe bone disease in these patients. However, bone biopsy is still rarely performed today, as it involves an invasive procedure and highly specialized laboratory techniques. High-turnover
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Javaid, Kassim. Osteoporosis and fragility fracture. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0275.

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Osteoporosis is defined as a systemic bone disease with reduction in both bone density and microarchitectural integrity, resulting in an increase in fragility fracture risk. It is a multifactorial disease which, through effects on bone formation and resorption, reduces the peak bone mass achieved during early adulthood and increases the rate of bone loss in later adulthood. Osteoporosis is clinically silent until a fragility fracture occurs. There are 3 million patients with osteoporosis in the UK, with over 200 000 fractures per year and 80 000 hip fractures. This chapter addresses the causes
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Skiba, Grzegorz. Fizjologiczne, żywieniowe i genetyczne uwarunkowania właściwości kości rosnących świń. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_gs_2020.

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Bones are multifunctional passive organs of movement that supports soft tissue and directly attached muscles. They also protect internal organs and are a reserve of calcium, phosphorus and magnesium. Each bone is covered with periosteum, and the adjacent bone surfaces are covered by articular cartilage. Histologically, the bone is an organ composed of many different tissues. The main component is bone tissue (cortical and spongy) composed of a set of bone cells and intercellular substance (mineral and organic), it also contains fat, hematopoietic (bone marrow) and cartilaginous tissue. Bones a
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Book chapters on the topic "Bone formation/resorption"

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Caniggia, Angelo. "Pathophysiology of Bone Formation and Resorption." In Bone Regulatory Factors, 235–52. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-1508-8_14.

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Schett, Georg. "Bone Formation Versus Bone Resorption in Ankylosing Spondylitis." In Advances in Experimental Medicine and Biology, 114–21. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-0298-6_8.

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Smith, Roger. "Role of Parathyroid Hormone in Bone Formation and Resorption." In Bone Regulatory Factors, 111–20. Boston, MA: Springer US, 1990. http://dx.doi.org/10.1007/978-1-4757-1508-8_8.

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Braidman, I. P. "Cellular Mechanisms of Bone Resorption and Formation." In Osteoporosis, 27–49. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-010-9580-8_2.

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Birkhold, Annette I., and Bettina M. Willie. "Registered Micro-Computed Tomography Data as a Four-Dimensional Imaging Biomarker of Bone Formation and Resorption." In Biomarkers in Bone Disease, 557–86. Dordrecht: Springer Netherlands, 2017. http://dx.doi.org/10.1007/978-94-007-7693-7_7.

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Birkhold, Annette I., and Bettina M. Willie. "Registered Micro-Computed Tomography Data as a Four-Dimensional Imaging Biomarker of Bone Formation and Resorption." In Biomarkers in Bone Disease, 1–30. Dordrecht: Springer Netherlands, 2015. http://dx.doi.org/10.1007/978-94-007-7745-3_7-1.

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Detsch, Rainer, Helmar Mayr, Daniel Seitz, and Günter Ziegler. "Is Hydroxyapatite Ceramic Included in the Bone Remodelling Proccess? An In Vitro Study of Resorption and Formation Processes." In Bioceramics 20, 1123–26. Stafa: Trans Tech Publications Ltd., 2007. http://dx.doi.org/10.4028/0-87849-457-x.1123.

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Bennett, Alan. "The Formation of Prostaglandins and Related Substances." In Prostaglandins in Bone Resorption, 1–10. CRC Press, 2020. http://dx.doi.org/10.1201/9780429279195-1.

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Harvey, Wilson. "Source of Prostaglandins and their Influence on Bone Resorption and Formation." In Prostaglandins in Bone Resorption, 27–42. CRC Press, 2020. http://dx.doi.org/10.1201/9780429279195-3.

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Mundy, G. R., G. D. Roodman, L. F. Bonewald, R. O. C. Oreffo, and B. F. Boyce. "Assays for bone resorption and bone formation." In Peptide Growth Factors Part C, 502–10. Elsevier, 1991. http://dx.doi.org/10.1016/0076-6879(91)98049-c.

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Conference papers on the topic "Bone formation/resorption"

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Altman, Allison R., Beom Kang Huh, Abhishek Chandra, Wei-Ju Tseng, Ling Qin, and X. Sherry Liu. "3D In Vivo Bone Dynamic Imaging of PTH’s Anabolic Action." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14671.

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Aging shifts bone remodeling toward a negative balance between bone formation and resorption, causing bone loss and increased fracture risk. Anti-resorptive agents are commonly used to inhibit bone resorption and stabilize bone mass. While they are effective to prevent further bone loss, there is also a great need for anabolic agents which can reverse bone deterioration and regain lost skeletal integrity. Intermittent parathyroid hormone (PTH) treatment is the only FDA-approved anabolic treatment for osteoporosis, which greatly stimulates bone formation. Combined therapy of anti-resorptive dru
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Bon, JM, Y. Zhang, J. Pilewski, D. Zaldonis, A. Zeevi, K. McCurry, S. Greenspan, and FC Sciurba. "Plasma Inflammatory Markers Associated with Bone Resorption and Formation in COPD." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4040.

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Hutchinson, AF, MA Thompson, DM Smallwood, S. Bozinovski, GP Anderson, LB Irving, and PR Ebeling. "Acute Chronic Obstructive Pulmonary Disease (COPD) Exacerbations Are Associated with Uncoupling of Bone Resorption from Bone Formation." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4037.

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Penninger, Charles L., Ryan K. Roeder, Glen L. Niebur, and John E. Renaud. "Investigation of Osteoclast Resorption Mechanisms in a Hybrid Cellular Automaton Model of Bone Remodeling." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176171.

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Bone is a living tissue which is continually adapting to its biological environment via continuous formation and resorption. It is generally accepted that bone remodeling occurs in response to daily mechanical loading. The remodeling process enables various functions, such as damage repair, adaptation to mechanical loads, and mineral homeostasis [1]. The cells that are responsible for the bone remodeling process are the bone resorbing osteoclasts and the bone forming osteoblasts. These cells closely coordinate their actions in a basic multicellular unit to renew “packets” of bone.
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Grisar, J., M. Aringer, K. Redlich, PM Bernecker, W. Wolozcszuk, JS Smolen, and P. Pietschmann. "SAT0035 Ankylosing spondylitis, psoriatic arthritis and reactive arthritis show increased bone resorption and differ with regard to bone formation." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.387.

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Chen, Haiming, Eric Sanchez, Cathy S. Wang, Mingjie Li, Jennifer Li, Kevin D. Delijani, Zhiwei Li, Benjamin Bonavida, Daniel Levitt, and James R. Berenson. "Abstract LB-305: The tyrosine kinase inhibitor bafetinib (INNO-406) inhibits osteoclast formation and bone resorption." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-lb-305.

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Shaul, J., D. Hall, T. Turner, R. Urban, and R. Hill. "Impact of Alendronate on Biomaterial Resorption, Bone Formation, and Strength in Humoral Critical-Sized Defects in Dogs." In OSTEOLOGIE 2019. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1679999.

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Joiner, Danese M., Ethan L. H. Daley, and Steven A. Goldstein. "The Effects of the Inhibition of Connexin 43 on Pre-Osteoblasts and Their Response to Mechanical Stimulation." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192700.

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It is well established that bone can adapt to the demands of daily mechanical usage. Mechanical loading can result in bone formation depending on the magnitude, duration, and frequency. Unloading, which can occur during bed rest, micro-gravity exposure and a variety of clinical conditions, can result in bone resorption. In vitro studies have demonstrated that osteoblasts and osteocytes respond to mechanical stimulation, especially oscillatory fluid shear stress. Mechano-responses have included increases in inter- and intra-cellular communication through gap junctions and soluble factors such a
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Slyfield, Craig R., Ryan E. Tomlinson, Evgeniy V. Tkachenko, Kyle E. Neimeyer, Grant J. Steyer, David L. Wilson, and Christopher J. Hernandez. "Sub-Micron 3D Fluorescent Imaging and Visualization of Remodeling Cavities in Cancellous Bone." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193099.

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The mechanical properties of cancellous bone are determined from a combination of bone quantity (volume), the material properties of the mineralized tissue, and microarchitecture. Bone remodeling is the primary process through which bone mass and structure are altered in the adult skeleton. Bone remodeling involves the coordinated activity of osteoclast and osteoblast cells, which resorb and then form bone at an isolated location on the cancellous bone surface. Because bone resorption precedes formation, each bone remodeling event in cancellous bone is associated with a temporary void on the b
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Caouette, Christiane, Martin N. Bureau, and L’Hocine Yahia. "Secondary Stability of a Composite Biomimetic Cementless Hip Stem." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192176.

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Total hip replacement is one of the most successful and frequent surgery in the world; over a million of these procedures are performed every year, and the numbers are growing with the ageing of the general population. The patients who receive these implants also are younger nowadays. Major problems however still subsist with traditional hip stems: aseptic loosening is a common cause of revision surgery. The main causes of aseptic loosening are both mechanical and biological in origin. Mechanical causes include stress shielding and micromotions at bone-implant interface, and biological causes
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