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Dissertations / Theses on the topic 'Bone formation'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Samizadeh, S. "Bone formation on calcium phosphate bone substitute materials." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/19891/.

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A large number of bone substitute materials are available; for which some authors claim osteoconductivity and some osteoinductivity. In order to rank these materials an in vivo analysis was carried out. These materials were chosen based on their availability and claimed mode of action. Silicon substituted Hydroxyapatite (SiHA), Hydroxyapatite (HA), Resorbable Calcium Phosphate Silicon, Skelite [siliconstabilized tricalcium phosphate-based bone substitute], Pro Osteon 500R [coralline HA], BiIonic [Yttrium stabilized SiHA] and two non-calcium phosphate, Dimeneralised Bone Matrix (DBM) based biom

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2

Chen, Jinbiao Prince of Wales Clinical School UNSW. "In vitro and in vivo bone formation - assessment and application." Awarded by:University of New South Wales. Prince of Wales Clinical School, 2006. http://handle.unsw.edu.au/1959.4/24922.

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Background: Bone-grafting materials are required in orthopaedic surgery to treat bone defects. Bone formation assessment is required for the development of new strategies and approaches and for quality assurance and quality control of currently available materials. Approaches to the assessment of bone formation are yet to be systematically established, quantified and standardized. Aims: the overall aim of this study was to establish a set of comprehensive quantitative approaches for the assessment of bone formation and to evaluate the role of osteoblastic cells, growth factors, and scaffolds

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3

Hrit, Manuela. "Acceleration of bone formation in distraction osteogenesis by bone morphogenetic protein-7." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=101142.

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The manipulation of the molecular mechanisms that govern distraction osteogenesis (DO) in order to increase the biomechanical strength of new bone and to accelerate its synthesis has been the topic of intense research during the past decades.<br>Bone morphogenetic proteins (BMPs) play an important role in bone formation. In this study, using a rabbit model of DO, the expressions of BMP's major intracellular signalling molecules, Smad proteins, was analyzed and correlated with the expression of BMP ligands and receptors. Based on these results, which confirmed post-receptor activity for the BMP

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4

Hu, Kai. "VEGF-Dependent Mechanisms Controlling Osteoblast Differentiation and Bone Formation During Bone Repair." Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:17467316.

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Osteoblast-derived vascular endothelial growth factor (VEGF) is important for bone development and postnatal bone homeostasis. Several studies have demonstrated that VEGF affects bone repair and regeneration; however, the cellular mechanisms by which it works are not fully understood. In this study, we investigated the functions of osteoblast-derived VEGF in healing of a cortical bone defect. In addition, how VEGF signaling modulates BMP2 functions during bone healing was also examined. To define the roles of osteoblast-derived VEGF in bone repair, a mouse tibial monocortical defect model was

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5

Williams, Bristol Marie. "Effects of tricalcium phosphate coated titanium on adjacent early bone formation." View the abstract Download the full-text PDF version, 2007. http://etd.utmem.edu/ABSTRACTS/2007-005-Williams-index.html.

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Thesis (M.S. )--University of Tennessee Health Science Center, 2007<br>Title from title page screen (viewed on July 28, 2008). Research advisor: Joo L. Ong, Ph.D. Document formatted into pages (iv, 36 p. : ill.). Vita. Abstract. Includes bibliographical references (p. 33-36).

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6

Lean, Jennifer Maree. "Mechanical stimulation of bone formation in the rat." Thesis, St George's, University of London, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263682.

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7

Moroz, Adam. "Reduced order modelling of bone resorption and formation." Thesis, De Montfort University, 2011. http://hdl.handle.net/2086/5409.

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The bone remodelling process, performed by the Bone Multicellular Unit (BMU) is a key multi-hierarchically regulated process, which provides and supports various functionality of bone tissue. It is also plays a critical role in bone disorders, as well as bone tissue healing following damage. Improved modelling of bone turnover processes could play a significant role in helping to understand the underlying cause of bone disorders and thus develop more effective treatment methods. Moreover, despite extensive research in the field of bone tissue engineering, bonescaffold development is still very

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8

Gundle, Roger. "Microscopical and biochemical studies of mineralised matrix production by bone-derived cells." Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.282203.

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9

Wang, Jason Lee. "Effects of aging and remodeling on bone microdamage formation." Thesis, Georgia Institute of Technology, 2010. http://hdl.handle.net/1853/37114.

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Skeletal fragility is characterized by low bone mass, negative changes in bone microarchitecture, and compromised tissue matrix properties, including accumulation of microdamage. Microdamage accumulates in vivo from daily physiological loading and is targeted for repair through a normal remodeling process, thus preventing microcrack growth and potential fracture. However, impaired remodeling is associated with aging and osteoporosis, resulting in an increased accumulation of microdamage which contributes to reduced bone mechanical properties. The current clinical method for assessing increased

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10

Lozano-Carrascal, Naroa. "Topical Application of Bisphosphonates to Enhance Alveolar New Bone Formation." Doctoral thesis, Universitat Internacional de Catalunya, 2017. http://hdl.handle.net/10803/456485.

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This PhD thesis is a compendium of three publications, which sets out to broaden our knowledge and understanding of the topical application of bisphosphonates alone or mixed with a bone graft in alveolar bone defects, to evaluate the potential capacity of them to preserve/enhance alveolar new bone formation. In recent years, research has focused on improving bone substitutes to achieve faster and better regeneration by morphologic and biochemical modification. Bisphosphonates are a group of drugs that reduce bone resorption by inhibiting the formation, recruitment activity of mature osteoc

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11

Rivera, Jaime Rodrigo. "Bone formation around implants in adult transgenic mice with selective Runx2-II deficiency." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2008. https://www.mhsl.uab.edu/dt/2008m/rivera.pdf.

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12

Yoshida, Keiji. "Stimulation of bone formation and prevention of bone loss by prostaglandin E EP4 receptor activation." Kyoto University, 2002. http://hdl.handle.net/2433/149332.

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13

Alison, Susan Jean School of Medicine UNSW. "The control of bone formation by neuropeptide Y receptors." Awarded by:University of New South Wales. School of Medicine, 2006. http://handle.unsw.edu.au/1959.4/26188.

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Osteoporosis is a growing health concern, characterised by deterioration of bone and increased fracture incidence. Anabolic treatments for reversing bone loss are presently limited. A bone anabolic response was recently reported following deletion of hypothalamic neuropeptide Y2 receptors in mice. In contrast, no discernable bone phenotype was observed in Y4 receptor knockout (Y4-/-) mice, revealing specificity between the Y receptors in their control of bone formation. Studies in this thesis revealed a second anabolic response in the absence of another Y receptor subtype; the Y1 receptor. The

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14

Allsopp, Richard Patrick. "The role of the vascular endothelium in bone formation." Thesis, University of Oxford, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.386827.

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15

Pungchanchaikul, Patimaporn. "Palatal bone formation is regulated by palatal shelf fusion." Thesis, University College London (University of London), 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.500037.

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16

Li, Jun. "Indian hedgehog stimulates chondrocyte hypertrophic differentiation inendochondral bone formation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39558009.

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17

Dobson, Katharine Rebecca. "Studies into the effects of androgens on bone formation." Thesis, University of Sheffield, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.301007.

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18

Taylor, Amanda Faith. "The role of glutamate in bone formation in vitro." Thesis, University of York, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341824.

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19

Baba, Ismail Yanny Marliana. "Three-dimensional structured hybrid scaffolds for enhanced bone formation." Thesis, Keele University, 2016. http://eprints.keele.ac.uk/3215/.

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The most common clinical treatments for large bone deficiencies resulting from trauma, disease or infection are autograft, allograft or bone graft substitutes (BGS). However, these treatments still have limitations for clinical applications. Thus, this project aims to fabricate an optimal scaffold design for enhanced bone formation. Human bone is not solely hydroxyapatite (HA) but consists of multi-ionic substitutions in the HA lattice. Here, we have developed multi-substituted HA (SiCHA) nanopowders as bone substitute materials. SiCHA-2 was found to closely mirror the composition of the bone

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20

Deegan, Anthony John. "Novel tissue engineering approaches to enhance natural bone formation." Thesis, Keele University, 2016. http://eprints.keele.ac.uk/3224/.

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The bone tissue engineering community has been striving to develop novel approaches that mimic natural bone formation. The rapid generation of mineralised bone tissue with a capacity for vascularisation and the selection of highly osteogenic cell sources are still the focus of research today. This study addresses three novel approaches in these key areas. Mineralisation in bone tissue involves stepwise cell – cell and cell – extracellular matrix (ECM) interactions. Regulation of the osteoblast culture microenvironment can manipulate osteoblast proliferation and mineralisation rates and consequ

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21

Kimura, Hiroaki. "Cthrc1 is a positive regulator of osteoblastic bone formation." Kyoto University, 2009. http://hdl.handle.net/2433/124307.

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22

Retting, Kelsey Nicole. "Smad proteins and the regulation of endochondral bone formation." Diss., Restricted to subscribing institutions, 2008. http://proquest.umi.com/pqdweb?did=1666396551&sid=5&Fmt=2&clientId=1564&RQT=309&VName=PQD.

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23

Minaur, Nicola Jane. "Methotrexate and bone formation and turnover in rheumatoid arthritis." Thesis, University of Bath, 1998. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285341.

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24

Manitzky, Louise J. "Mathematical modelling of intramembranous bone formation during fracture healing." Thesis, Queensland University of Technology, 2014. https://eprints.qut.edu.au/78983/1/Louise_Manitzky_Thesis.pdf.

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During fracture healing, many complex and cryptic interactions occur between cells and bio-chemical molecules to bring about repair of damaged bone. In this thesis two mathematical models were developed, concerning the cellular differentiation of osteoblasts (bone forming cells) and the mineralisation of new bone tissue, allowing new insights into these processes. These models were mathematically analysed and simulated numerically, yielding results consistent with experimental data and highlighting the underlying pattern formation structure in these aspects of fracture healing.

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25

Liu, Jin. "Increased CKIP-1 suppresses Smad-dependent BMP signaling to inhibit bone formation during aging." HKBU Institutional Repository, 2016. https://repository.hkbu.edu.hk/etd_oa/327.

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Emerging evidence indicates that the dysregulation of protein ubiquitination plays a crucial role in aging-associated diseases. Smad-dependent canonical BMP signaling pathway is indispensable for osteoblastic bone formation, which could be disrupted by the ubiquitination and subsequent proteasomal degradation of Smad1/5, the key molecules for BMP signaling transduction. However, whether the dysregulation of Smad1/5 ubiquitination and disrupted BMP signaling pathway are responsible for the age-related bone formation reduction is still underexplored. Casein kinase-2 interacting protein-1 (CKIP-1

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26

O'Connor, Rose Deeter. "MeCP2 deficiency decreases bone formation and reduces bone volume in a rodent model of Rett syndrome." Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file, 161 p, 2009. http://proquest.umi.com/pqdweb?did=1891570941&sid=6&Fmt=2&clientId=8331&RQT=309&VName=PQD.

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27

Wei, Fei. "The osteoimmune effect of bone morphogenetic protein-2 in bone regeneration and biomaterial modification." Thesis, Queensland University of Technology, 2020. https://eprints.qut.edu.au/198148/1/Fei_Wei_Thesis.pdf.

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Bone morphogenetic protein-2 (BMP2), one of most well-known osteoinductive molecules, has been extensively used in the orthopedics and dentistry. However, inflammatory reactions are regularly reported for its side effect. This project investigated the immune environment induced by BMP2 and unveiled the regulation of BMP2 in the cross talk between immune cells and bone forming cells. This project provides better understanding of BMP2 application in the treatment of fracture healing and bone-related diseases.

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28

Li, Jun. "Indian hedgehog stimulates chondrocyte hypertrophic differentiation in endochondral bone formation." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/HKUTO/record/B39558009.

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29

Hamade, Fares. "Enhanced bone formation during distraction osteogenesis in FGFR3 deficient mice." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112630.

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Distraction Osteogenesis (DO) is a technique for bone lengthening and filling of bone defects following trauma, infection or resection of tumors. DO consists of an osteotomy of the bone to be lengthened, followed by controlled distraction of the bone segments with an external fixator until the desired lengthening is obtained (distraction phase). This is followed by the consolidation phase, during which the external fixator is kept in place until the newly formed bone in the distracted zone consolidates. This phase is long and may cause numerous problems. Ongoing research aims at finding a meth

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30

Gartland, Alison. "The role of the P2X7 receptor in bone cell formation." Thesis, University of Liverpool, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.343968.

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31

García, Gareta E. "Development of a bone tissue-engineered construct to enhance new bone formation in revision total hip replacement." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1338147/.

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The main issue associated with revision total hip replacements (rTHRs) is how to generate new bone and restore bone stock for fixation of the revision stem. Bone tissue engineering (BTE) seeks the generation of constructs ex vivo in order to replace damaged or lost bone. The aim of this thesis was to develop a bone tissue-engineered construct with a calcium-phosphate (CaP) coated porous metal scaffold seeded throughout its structure with mesenchymal stem cells (MSCs) in order to enhance new bone formation at rTHRs. The study had in vitro and in vivo phases. For the in vitro phase, CaP coatings

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32

Kawai, Mariko. "Ectopic bone formation by human bone morphogenetic protein-2 gene transfer to skeletal muscle using transcutaneous electroporation." Kyoto University, 2004. http://hdl.handle.net/2433/147446.

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33

Shekaran, Asha. "Beta 1 integrins in bone formation during development and engineering integrin-specific hydrogels for enhanced bone healing." Diss., Georgia Institute of Technology, 2013. http://hdl.handle.net/1853/51720.

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Healing large bone defects remains a clinical challenge. While autografts are the gold standard treatment for large bone defects, they are limited by availability and donor site pain. Growth factor treatments such as BMP therapy provide a promising alternative but are expensive and present clinical safety concerns, primarily due to delivery of BMPs at supraphysiological doses. Integrins are ECM receptors which mediate crucial cell functions such as adhesion and differentiation. Therefore, understanding the role of integrins in bone formation and directing desired interactions may enable modula

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34

Miao, Dengshun. "Studies on the actions of bone anabolic drugs in vivo and in vitro." Thesis, University of Sheffield, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.300362.

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35

Weston, Darlene Adele. "Approaches to the investigation of periosteal new bone formation in palaeopathology." Thesis, University College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.405263.

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36

Sanghani-Kerai, Anita. "Do stem cells transfected with CXCR4 enhance bone formation in osteoporosis?" Thesis, University College London (University of London), 2018. http://discovery.ucl.ac.uk/10045319/.

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Osteoporosis affects bone mass and bone micro-architecture, reducing mechanical strength. SDF-1 and its ligand CXCR4 play significant roles in the migration and engraftment of mesenchymal stem cells (MSCs). The aim of this study was to investigate the effects of CXCR4 transfected MSCs on bone formation in osteopenic rats. The hypothesis was that MSCs genetically modified to over-express CXCR4, would enhance migration of stem cells from osteopenic rats and when injected intravenously in ovariectomised (OVX) rats, would improve bone formation. MSCs were harvested from femora of young, OVX and ad

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37

Ferguson, James. "THE SPATIAL AND TEMPORAL ROLE OF EZH2 IN SKULL BONE FORMATION." Case Western Reserve University School of Graduate Studies / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=case1530898825341447.

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38

Horne, Jacqueline Avril. "Mathematical modelling of soft callus formation in early murine bone repair." Thesis, Queensland University of Technology, 2013. https://eprints.qut.edu.au/63823/1/Jacqueline_Avril_Horne_Thesis.pdf.

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Fracture healing is a complicated coupling of many processes. Yet despite the apparent complexity, fracture repair is usually effective. There is, however, no comprehensive mathematical model addressing the multiple interactions of cells, cytokines and oxygen that includes extra-cellular matrix production and that results in the formation of the early stage soft callus. This thesis develops a one dimensional continuum transport model in the context of early fracture healing. Although fracture healing is a complex interplay of many local factors, critical components are identified and used t

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39

Lu, Luhui, and 陆璐慧. "Molecular control of osteo-chondroprogenitors formation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B44673966.

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40

Kim, Michael S. "Gene Expression in Bone Cells." Thesis, Griffith University, 2006. http://hdl.handle.net/10072/366180.

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Osteoclast formation is a complex process that is yet to be clearly defined. Osteoclasts differentiate from monocytic precursors to large multinuclear cells via the actions of two crucial cytokines: macrophage colony stimulating factor (M-CSF) and receptor activator of NFKB ligand (RANKL). These two cytokines bind to the osteoclast precursor cells, activating various down stream signalling pathways, inducing genes required for differentiation and for activation of osteoclasts. Exposure of monocytic precursors to M-CSF alone leads to differentiation into macrophages. Osteoclast differentiation

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41

Clayton, Angela Ann. "Analysis of an Eocene Bone-bed, Contained within the Lower Lisbon Formation, Covington County, Alabama." Wright State University / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=wright1310391028.

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42

Walker, Katherine Elizabeth. "Studies on the effects of estrogen in human osteogenic cells." Thesis, University of Sheffield, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.245692.

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43

Morrison, Matthew Sam. "Osteoclast function : role of extracellular pH and ATP." Thesis, University College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.369218.

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44

Roberts, Ellen. "Investigations into a novel osteoclastic antigen." Thesis, University of Liverpool, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263794.

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45

Öberg, Sven. "Bone Healing after implantation of bone substitute materials. Experimental studies in estrogen deficiency." Doctoral thesis, Umeå University, Odontology, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-138.

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<p>Bone formation and bone healing were studied in the mandible, tibia and skull bones in adult, healthy and estrogen deficient rabbits implanted with different bone substitutes. </p><p>In the first study an evaluation of the differences in bone regeneration in and around solid (Alveograf *) and porous hydroxyapatite (Interpore 200*) was undertaken. The implant material was placed into experimentally made bone defects and in half of the defects hydroxyapatite was mixed with a fibrin sealant (Tisseel *). The material alone or mixed with Tisseel was also placed subperiostally in the mandible. Th

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46

Davey, Tamara. "Functional characterisation of a novel osteoclast-derived factor." University of Western Australia. School of Surgery and Pathology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0219.

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[Truncated abstract] Intracellular communication between osteoclasts and osteoblasts is imperative to maintain bone integrity. A myriad of molecules are responsible for regulating osteoblast and osteoclast activity. In particular, it is well documented that osteoblast-derived factors are crucial in directly controlling osteoclast formation and function. Since bone formation is coupled to bone resorption, it would be expected that osteoclasts also have some role in regulating the growth and function of osteoblast cells. However, despite extensive research upon osteoclast and osteoblast biology,

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47

Simmons, Craig Alexander. "Modelling and characterization of mechanically regulated tissue formation around bone-interfacing implants." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape3/PQDD_0022/NQ49943.pdf.

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48

Eijken, Hermanus Johannes Marco. "Human osteoblast differentiation and bone formation: growth factors, hormones & regulatory networks." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2007. http://hdl.handle.net/1765/10597.

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49

Husseini, Abdallah. "Bone formation during fracture repair in mice deficient for the cyp24a1 gene." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=106532.

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Vitamin D is a key regulator of mineral and bone homeostasis. The enzyme CYP24A1 is responsible for transforming vitamin D into 24,25(OH)2D. The putative biological activity of 24,25(OH)2D remains unclear. Previous studies showed an increase in the circulating levels of this metabolite following a fracture in chicks. Our laboratory has engineered a mouse model deficient for the Cyp24a1 gene for studying the role of 24,25(OH)2D. We set out to study the role of 24,25(OH)2D in endochondral and intramembranous bone formation in mammalian fracture repair in this mouse model. Methods: Wild-type and

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50

Rapp, Anna Elise [Verfasser]. "Effect of MSC-administration on bone formation and repair / Anna Elise Rapp." Ulm : Universität Ulm. Medizinische Fakultät, 2015. http://d-nb.info/1067924396/34.

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