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Journal articles on the topic 'Bone formation'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Stützle, H., K. Hallfeldt, H. Mandelkow, S. Keßler, and L. Schweiberer. "Bone substitutes and bone formation." Der Orthopäde 27, no. 2 (1998): 118–25. http://dx.doi.org/10.1007/pl00003477.

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2

Bellingham, F. Richard. "Endometrial Bone Formation." Australian and New Zealand Journal of Obstetrics and Gynaecology 36, no. 1 (1996): 109–10. http://dx.doi.org/10.1111/j.1479-828x.1996.tb02943.x.

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3

PUZAS, J. EDWARD, MICHAEL D. MILLER, and RANDY N. ROSIER. "Pathologic Bone Formation." Clinical Orthopaedics and Related Research &NA;, no. 245 (1989): 269???281. http://dx.doi.org/10.1097/00003086-198908000-00042.

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4

Schiergens, Tobias S., Angela Reichelt, Wolfgang E. Thasler, and Markus Rentsch. "Abdominal Bone Formation." Journal of Gastrointestinal Surgery 19, no. 3 (2015): 579–80. http://dx.doi.org/10.1007/s11605-014-2737-4.

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5

Masayoshi, Yamaguchi, and Hua Gao-Balch Ying. "Role of Dietary Soybean Genistein in Osteoporosis Prevention." International Journal of Food Science, Nutrition and Dietetics 2, no. 2 (2013): 27–34. https://doi.org/10.19070/2326-3350-130006.

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Bone homeostasis is regulated through osteoclasts and osteoblasts. Osteoporosis, which is induced with its accompanying decrease in bone mass with increasing age, is widely recognized as a major public heath problem. Bone loss may be due to decreased osteoblastic bone formation and increased osteoclastic bone resorption. There is growing evidence that nutritional and food factors may play a part in the prevention of bone loss with aging and have been to be worthy of notice in the prevention of osteoporsis. Genistein, which is contained in soybeans, has been shown to have a stimu

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6

Tsartsalis, Athanasios, Charalambos Dokos, Georgia Kaiafa, et al. "Statins, bone formation and osteoporosis: hope or hype?" HORMONES 11, no. 2 (2012): 126–39. http://dx.doi.org/10.14310/horm.2002.1339.

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7

Navid, Aghadavudi, Kousha Kiana, Ariana Niyosha, Baghaei Kimia, and Sobhan Khademi Sayed. "Bone formation in oral surgery, the concept and limitation: A review of literature." World Journal of Biology Pharmacy and Health Sciences 14, no. 1 (2023): 247–51. https://doi.org/10.5281/zenodo.8037860.

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The process of bone formation is a crucial component of oral surgery, particularly in instances where bone deficiencies or defects exist. The success of bone formation in oral surgery depends on different factors, such as the quality and quantity of the bone graft, the patient’s local and systemic conditions, and surgical techniques. The major aim of this review is to provide a brief overview of the basics of bone formation and describe the concepts related to the regeneration of bone in oral surgery. This article also highlighted different factors leading to bone loss and the potential

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8

Habal, Mutaz B. "Bone Engineering, Bone Formation, or just Refined Bone Regeneration." Journal of Craniofacial Surgery 14, no. 3 (2003): 265. http://dx.doi.org/10.1097/00001665-200305000-00001.

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9

Isaac, Juliane, S. Loty, A. Hamdan, et al. "In Vitro Bone Formation on Bioactive Titanium." Key Engineering Materials 361-363 (November 2007): 939–42. http://dx.doi.org/10.4028/www.scientific.net/kem.361-363.939.

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Titanium has limitations in its clinical performance in dental and orthopaedic applications. Over the last decade, numerous implant surface modifications have been developed and are currently used with the aim of enhancing bone integration. In the present study, we have experimented a bioactive titanium prepared by a simple chemical and moderate heat treatment that leads to the formation of a bone-like apatite layer on its surface in simulated body fluids. We haved used foetal rat calvaria cell cultures to investigate bone nodule formation on bioactive titanium. Scanning electron microscopy (S

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10

Luriya, E. A., M. Owen, A. Ya Fridenshtein, S. A. Kuznetsov, E. N. Genkina, and V. V. Gosteva. "Bone formation in bone marrow organ cultures." Bulletin of Experimental Biology and Medicine 101, no. 4 (1986): 520–24. http://dx.doi.org/10.1007/bf00834432.

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11

Draenert, M. E., C. Martini, D. C. Watts, K. Draenert, and A. Wittig-Draenert. "Bone augmentation by replica-based bone formation." Dental Materials 36, no. 11 (2020): 1388–96. http://dx.doi.org/10.1016/j.dental.2020.08.005.

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12

Garrett, I., G. Gutierrez, and G. Mundy. "Statins and Bone Formation." Current Pharmaceutical Design 7, no. 8 (2001): 715–36. http://dx.doi.org/10.2174/1381612013397762.

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13

Karsenty, Gerard. "Re-tuning bone formation." Journal of Experimental Medicine 212, no. 1 (2015): 3. http://dx.doi.org/10.1084/jem.2121insight2.

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14

Urist, Marshall R., and Leonard F. Peltier. "Bone: Formation by Autoinduction." Clinical Orthopaedics and Related Research 395 (February 2002): 4–10. http://dx.doi.org/10.1097/00003086-200202000-00002.

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15

Louis, L. S., C. E. C. Kingman, and G. W. Cochrane. "Heterotopic intrauterine bone formation." Journal of Obstetrics and Gynaecology 27, no. 2 (2007): 208–9. http://dx.doi.org/10.1080/01443610601157331.

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16

Powell, Kendall. "Dishing up bone formation." Journal of Cell Biology 171, no. 3 (2005): 409. http://dx.doi.org/10.1083/jcb1713fta3.

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17

Jones, S. "Site-directed bone formation." Biofutur 1997, no. 167 (1997): 48. http://dx.doi.org/10.1016/s0294-3506(99)80361-5.

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18

Aoki, Jun, Itsuo Yamamoto, Megumu Hino, et al. "Reactive endosteal bone formation." Skeletal Radiology 16, no. 7 (1987): 545–51. http://dx.doi.org/10.1007/bf00351269.

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19

Yadav, Vijay K., and Patricia Ducy. "Lrp5 and bone formation." Annals of the New York Academy of Sciences 1192, no. 1 (2010): 103–9. http://dx.doi.org/10.1111/j.1749-6632.2009.05312.x.

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20

BARAN, R., and L. JUHLIN. "Bone dependent nail formation." British Journal of Dermatology 114, no. 3 (1986): 371–75. http://dx.doi.org/10.1111/j.1365-2133.1986.tb02830.x.

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21

Lind, Martin, and Cody B�nger. "Factors stimulating bone formation." European Spine Journal 10 (October 1, 2001): S102—S109. http://dx.doi.org/10.1007/s005860100269.

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22

Wallach, Stanley, Louis V. Avioli, and John H. Carstens. "Factors in bone formation." Calcified Tissue International 45, no. 1 (1989): 4–6. http://dx.doi.org/10.1007/bf02556652.

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23

Jin, Yong Jun, and Won Man Park. "Comparison of Extragraft Bone Formation after Anterior Cervical Discectomy and Fusion Using Simultaneous and Sequential Algorithms." Applied Sciences 11, no. 4 (2021): 1487. http://dx.doi.org/10.3390/app11041487.

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Extragraft bone formation is crucial for obtaining a successful outcome after spinal fusion surgery. However, the cause of bone formation is not well investigated. In this study, it was hypothesised that extragraft bone formation is generated by mechanical stimuli. A preoperative plan for anterior cervical discectomy and fusion was applied to the finite element model of the C5–C6 motion segment. Extragraft bone formations posterior to the interbody cage were simulated using simultaneous and sequential algorithms. While the simultaneous algorithm predicted the formation of extragraft bone bridg

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24

Wang, E. A., V. Rosen, J. S. D'Alessandro, et al. "Recombinant human bone morphogenetic protein induces bone formation." Proceedings of the National Academy of Sciences 87, no. 6 (1990): 2220–24. http://dx.doi.org/10.1073/pnas.87.6.2220.

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25

van Straalen, Jan P., Edward Sanders, Mark F. Prummel, and Gerard T. B. Sanders. "Bone-alkaline phosphatase as indicator of bone formation." Clinica Chimica Acta 201, no. 1-2 (1991): 27–33. http://dx.doi.org/10.1016/0009-8981(91)90021-4.

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26

Yavropoulou, Maria P., Helen P. Vafiadou, Olympia E. Anastasiou, Vasiliki Tsavdaridou, Georgia H. Kokaraki, and John G. Yovos. "Pioglitazone affects bone resorption but not bone formation." Bone 42 (March 2008): S91. http://dx.doi.org/10.1016/j.bone.2007.12.173.

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27

Zizelmann, Christoph, Ralf Schoen, Marc Christian Metzger, et al. "Bone formation after sinus augmentation with engineered bone." Clinical Oral Implants Research 18, no. 1 (2007): 69–73. http://dx.doi.org/10.1111/j.1600-0501.2006.01295.x.

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28

Ho, Miriel, Hatem Salem, Stephen Livesey, and Kathy Traianedes. "Bone formation and the development of bone marrow." Experimental Hematology 41, no. 8 (2013): S65. http://dx.doi.org/10.1016/j.exphem.2013.05.255.

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29

Yehudina, Ye.D., and I.Yu. Golovach. "LABORATORY ASPECTS AND CLINICAL SIGNIFICANCE OF BONE TURNOVER MARKERS." Annals of Mechnikov Institute, no. 3 (October 2, 2019): 7–18. https://doi.org/10.5281/zenodo.3469393.

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<strong>Introduction.</strong> With an aging population, there is a marked increase in prevalence of metabolic bone diseases, especially osteoporosis. A serious complication of osteoporosis is non-traumatic bone fractures, which significantly impair quality of life and are associated with comorbid conditions and high mortality. Diseases associated with impaired bone remodeling require timely diagnosis, treatment and monitoring. The consequent public health and socioeconomic burden warrant timely diagnosis, treatment and follow-up of these disorders. Knowing the limitations of radiological tech

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30

Shieh, Albert, Weijuan Han, Shinya Ishii, Gail A. Greendale, Carolyn J. Crandall, and Arun S. Karlamangla. "Quantifying the Balance Between Total Bone Formation and Total Bone Resorption: An Index of Net Bone Formation." Journal of Clinical Endocrinology & Metabolism 101, no. 7 (2016): 2802–9. http://dx.doi.org/10.1210/jc.2015-4262.

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31

Ganss, B., R. H. Kim, and J. Sodek. "Bone Sialoprotein." Critical Reviews in Oral Biology & Medicine 10, no. 1 (1999): 79–98. http://dx.doi.org/10.1177/10454411990100010401.

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The search for a protein nucleator of hydroxyapatite crystal formation has been a focus for the isolation and characterization of the major non-collagenous proteins in bone. Of the proteins characterized to date, bone sialoprotein (BSP) has emerged as the only bona fide candidate for nucleation. BSP is a highly glycosylated and sulphated phosphoprotein that is found almost exclusively in mineralized connective tissues. Characteristically, polyglutamic acid and arginine-glycine-aspartate (RGD) motifs with the ability to bind hydroxyapatite and cell-surface integrins, respectively, have been con

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32

Utari, Kresnoadi, Subiakto Rahmani Bima, Nur Rahmania Primanda, and Anissa Pramesti Rheyna. "Induction effect combination of Nigella sativa extract and bovine bone graft to the area of woven bone on the preservation socket post-tooth extraction." World Journal of Advanced Research and Reviews 17, no. 2 (2023): 564–69. https://doi.org/10.5281/zenodo.8108902.

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<strong>Background</strong>: Tooth extraction is a common dentistry procedure followed by alveolar bone resorption. Trauma that occurs in tooth extraction will induce exaggerating inflammatory process, leads to increased alveolar bone resorption. Bone resorption can be minimized by administering a combination of <em>Nigella sativa</em> extract and bovine bone graft. The combination material is expected to increase the woven bone area formation and speed up the alveolar bone remodeling process. <strong>Purpose</strong>: To determine the effect of induction of a combination of <em

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33

Ogawa, Y., D. K. Schmidt, R. M. Nathan, et al. "Bovine bone activin enhances bone morphogenetic protein-induced ectopic bone formation." Journal of Biological Chemistry 267, no. 20 (1992): 14233–37. http://dx.doi.org/10.1016/s0021-9258(19)49702-0.

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34

Yamaguchi, M. "Marine Alga Sargassum Horneri Component And Bone Homeostasis: Role In Osteoporosis Prevention." International Journal of Food Science, Nutrition and Dietetics 2, no. 1 (2013): 9–14. https://doi.org/10.19070/2326-3350- 130003.

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Bone homeostasis is maintained through a balance between osteoblastic bone formation and osteoclastic bone resorption. Aging induces bone loss due to decreased osteoblastic bone formation and increased osteoclastic bone resorption. Osteoporosis with its accompanying decrease in bone mass is widely recognized as a major public heath problem. Nutritional factors may play a role in the prevention of bone loss with aging. Among marine algae of Undaria pinnatifida, Sargassum horneri, Eisenia bicyclis, Cryptonemia scmitziana, Gelidium amansii, and Ulva pertusa Kjellman which were

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35

Boscher, Julie, Ines Guinard, Anita Eckly, François Lanza, and Catherine Léon. "Blood platelet formation at a glance." Journal of Cell Science 133, no. 20 (2020): jcs244731. http://dx.doi.org/10.1242/jcs.244731.

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ABSTRACTThe main function of blood platelets is to ensure hemostasis and prevent hemorrhages. The 1011 platelets needed daily are produced in a well-orchestrated process. However, this process is not yet fully understood and in vitro platelet production is still inefficient. Platelets are produced in the bone marrow by megakaryocytes, highly specialized precursor cells that extend cytoplasmic projections called proplatelets (PPTs) through the endothelial barrier of sinusoid vessels. In this Cell Science at a Glance article and the accompanying poster we discuss the mechanisms and pathways invo

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36

Madsen⁎, S. H., A. S. Goettrup, K. Henriksen, M. A. Karsdal, and A. C. Bay-Jensen. "Prednisolone increases cartilage formation, but decreases bone formation." Bone 47 (June 2010): S148. http://dx.doi.org/10.1016/j.bone.2010.04.338.

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37

Kim, Jung-Eun. "Transcriptional regulation of bone formation." Frontiers in Bioscience S3, no. 1 (2011): 126–35. http://dx.doi.org/10.2741/s138.

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38

Chen, Zhihao, Yan Zhang, Chao Liang, Lei Chen, Ge Zhang, and Airong Qian. "Mechanosensitive miRNAs and Bone Formation." International Journal of Molecular Sciences 18, no. 8 (2017): 1684. http://dx.doi.org/10.3390/ijms18081684.

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39

Karsenty, Gerard, Henry M. Kronenberg, and Carmine Settembre. "Genetic Control of Bone Formation." Annual Review of Cell and Developmental Biology 25, no. 1 (2009): 629–48. http://dx.doi.org/10.1146/annurev.cellbio.042308.113308.

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40

Collins, M., and C. Stratakis. "Bone Formation, Growth, and Repair." Hormone and Metabolic Research 48, no. 11 (2016): 687–88. http://dx.doi.org/10.1055/s-0042-119907.

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41

Patel, Vikas V., and Karin Payne. "Cellular Grafts for Bone Formation." SPINE 41 (April 2016): S13. http://dx.doi.org/10.1097/brs.0000000000001425.

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42

Iqbal, Jameel, Li Sun, and Mone Zaidi. "Coupling bone degradation to formation." Nature Medicine 15, no. 7 (2009): 729–31. http://dx.doi.org/10.1038/nm0709-729.

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43

Hunt, Jennifer L., Ronald Fairman, Marc E. Mitchell, et al. "Bone Formation in Carotid Plaques." Stroke 33, no. 5 (2002): 1214–19. http://dx.doi.org/10.1161/01.str.0000013741.41309.67.

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44

Schroeder, Gregory D., Christopher K. Kepler, Sibylle Grad, et al. "Does Riluzole Influence Bone Formation?" SPINE 44, no. 16 (2019): 1107–17. http://dx.doi.org/10.1097/brs.0000000000003022.

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45

Turner, Charles H., and Alexander G. Robling. "Mechanical loading and bone formation." BoneKEy-Osteovision 1, no. 9 (2004): 15–23. http://dx.doi.org/10.1138/20040135.

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46

Clarke, Joanna. "JAK inhibitors boost bone formation." Nature Reviews Rheumatology 16, no. 5 (2020): 249. http://dx.doi.org/10.1038/s41584-020-0406-4.

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47

WOZNEY, JOHN M., VICKI ROSEN, ANTHONY J. CELESTE, et al. "Novel Regulators of Bone Formation." Obstetrical & Gynecological Survey 44, no. 5 (1989): 387. http://dx.doi.org/10.1097/00006254-198905000-00028.

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48

KISHIDA, Akio. "Bone Formation on Polymer Surfaces." Kobunshi 48, no. 4 (1999): 266. http://dx.doi.org/10.1295/kobunshi.48.266.

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49

Ray, Katrina. "Silencing inhibitors of bone formation." Nature Reviews Rheumatology 8, no. 3 (2012): 122. http://dx.doi.org/10.1038/nrrheum.2012.17.

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50

Fukushima, Nobuhiro, Reiko Hanada, Hitoshi Teranishi, et al. "Ghrelin Directly Regulates Bone Formation." Journal of Bone and Mineral Research 20, no. 5 (2004): 790–98. http://dx.doi.org/10.1359/jbmr.041237.

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