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Journal articles on the topic 'Bone physiology and vitamin A'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

White, C. P., N. A. Morrison, E. M. Gardiner, and J. A. Eisman. "Vitamin D receptor alleles and bone physiology." Journal of Cellular Biochemistry 56, no. 3 (1994): 307–14. http://dx.doi.org/10.1002/jcb.240560306.

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2

Larmonier, C. B., R. M. T. McFadden, F. M. Hill, et al. "High vitamin D3 diet administered during active colitis negatively affects bone metabolism in an adoptive T cell transfer model." American Journal of Physiology-Gastrointestinal and Liver Physiology 305, no. 1 (2013): G35—G46. http://dx.doi.org/10.1152/ajpgi.00065.2013.

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Decreased bone mineral density (BMD) represents an extraintestinal complication of inflammatory bowel disease (IBD). Vitamin D3 has been considered a viable adjunctive therapy in IBD. However, vitamin D3 plays a pleiotropic role in bone modeling and regulates the bone formation-resorption balance, depending on the physiological environment, and supplementation during active IBD may have unintended consequences. We evaluated the effects of vitamin D3 supplementation during the active phase of disease on colonic inflammation, BMD, and bone metabolism in an adoptive IL-10−/− CD4+ T cell transfer

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3

Mazurais, D., M. J. Darias, M. F. Gouillou-Coustans, et al. "Dietary vitamin mix levels influence the ossification process in European sea bass (Dicentrarchus labrax) larvae." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 294, no. 2 (2008): R520—R527. http://dx.doi.org/10.1152/ajpregu.00659.2007.

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The influence of dietary vitamins on growth, survival, and morphogenesis was evaluated until day 38 of posthatching life in European sea bass larvae ( Dicentrarchus labrax). A standard vitamin mix (VM), at double the concentration of the U.S. National Research Council's recommendations, was incorporated into larval feeds at 0.5%, 1.5%, 2.5%, 4.0%, and 8.0% to give treatments VM 0.5, VM 1.5, VM 2.5, VM 4.0, and VM 8.0, respectively. The group fed the VM 0.5 diet all died before day 30. At day 38, the larvae group fed VM 1.5 had 33% survival, while the other groups, with higher vitamin levels, s

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Abegg, Kathrin, Nicole Gehring, Carsten A. Wagner, et al. "Roux-en-Y gastric bypass surgery reduces bone mineral density and induces metabolic acidosis in rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 305, no. 9 (2013): R999—R1009. http://dx.doi.org/10.1152/ajpregu.00038.2013.

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Roux-en-Y gastric bypass (RYGB) surgery leads to bone loss in humans, which may be caused by vitamin D and calcium malabsorption and subsequent secondary hyperparathyroidism. However, because these conditions occur frequently in obese people, it is unclear whether they are the primary causes of bone loss after RYGB. To determine the contribution of calcium and vitamin D malabsorption to bone loss in a rat RYGB model, adult male Wistar rats were randomized for RYGB surgery, sham-operation–ad libitum fed, or sham-operation–body weight-matched. Bone mineral density, calcium and phosphorus balance

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Massé, P. G., E. E. Delvin, P. V. Hauschka, et al. "Perturbations in factors that modulate osteoblast functions in vitamin B6 deficiency." Canadian Journal of Physiology and Pharmacology 78, no. 11 (2000): 904–11. http://dx.doi.org/10.1139/y00-072.

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It was hypothesized that the widespread structural defect of collagen in connective tissue of vitamin B6deficient-animals and the consequent alteration in bone biomechanical properties cause an additional stress to their inflammed swollen tibiotarsometatarsal joints. The present study showed a 32% elevation (P &lt 0.02) in mean plasma free cortisol concentration. Vitamin D metabolism was impaired but without changing plasma calcium homeostasis and bone mineral content. Mean plasma calcitriol [1,25(OH)2D] concentration was significantly reduced (P < 0.001). Because plasma calcidiol conce

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6

Lane, Ginny, Christine Nisbet, Susan J. Whiting, and Hassan Vatanparast. "Canadian newcomer children’s bone health and vitamin D status." Applied Physiology, Nutrition, and Metabolism 44, no. 7 (2019): 796–803. http://dx.doi.org/10.1139/apnm-2018-0705.

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Adequate calcium intake and supply of vitamin D during childhood play important roles in ensuring adequate bone mass gain to achieve optimal peak bone mass. The Healthy Immigrant Children study employed a mixed-method cross-sectional study design to characterize the health and nutritional status of 300 immigrant and refugee children aged 3–13 years who had been in Canada for less than 5 years. This paper presents bone mineral content and vitamin D status data along with qualitative data that deepen the understanding of newcomer bone health status. A significantly higher percentage of refugee c

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7

Rangel, Letícia Batista Azevedo, Daniel de Siqueira, Olívia do Rosário Soares, et al. "Vitamin K Supplementation Modulates Bone Metabolism and Ultra-Structure of Ovariectomized Mice." Cellular Physiology and Biochemistry 51, no. 1 (2018): 356–74. http://dx.doi.org/10.1159/000495234.

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Background/Aims: Osteoporosis is a bone metabolic disease that affects mostly post-menopausal women. There has been shown that vitamin K (VK) supplementation during menopause may decrease bone loss as well as risk of bone breaking. Aiming to clarify the beneficial role of VK in bone metabolism during menopause, we investigated mineral metabolism and bone ultrastructure of ovariectomized (OVX) mice. Methods: To determine the effects chronic use of VK in bone structure and mineral metabolism in OVX mice, we used several methods, such as DXA, µCTScan, and SEM as well as biomolecular techniques, s

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8

Kelly, James, Audrey Lin, Chiachien J. Wang, Sil Park, and Ichiro Nishimura. "Vitamin D and Bone Physiology: Demonstration of Vitamin D Deficiency in an Implant Osseointegration Rat Model." Journal of Prosthodontics 18, no. 6 (2009): 473–78. http://dx.doi.org/10.1111/j.1532-849x.2009.00446.x.

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9

Tajima, Orie, Noriko Ashizawa, Tomoo Ishii, et al. "Interaction of the effects between vitamin D receptor polymorphism and exercise training on bone metabolism." Journal of Applied Physiology 88, no. 4 (2000): 1271–76. http://dx.doi.org/10.1152/jappl.2000.88.4.1271.

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Bone metabolism is strongly influenced by heredity and environmental factors. To investigate interaction of the effects between vitamin D receptor polymorphism by Fok I and resistance exercise training on bone metabolism, young male subjects with FF genotype (F, n = 10) and Ff or ff genotypes (f, n = 10) followed 1 mo of weight training, and changes in bone metabolism were compared. An additional 14 subjects served as a sedentary control. Biomarkers of bone formation, bone-specific alkaline phosphatase, and osteocalcin were significantly increased by training in both F and f groups. 1,25-Dihyd

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10

Bouillon, Roger. "Vitamin D: Basic and clinical research in vitamin D, vitamin D analogs and bone health." Bone 47 (October 2010): S355. http://dx.doi.org/10.1016/j.bone.2010.09.049.

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11

Hagino, Hiroshi. "Vitamin D3 analogs for the treatment of osteoporosis." Canadian Journal of Physiology and Pharmacology 93, no. 5 (2015): 327–32. http://dx.doi.org/10.1139/cjpp-2014-0419.

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Vitamin D supplementation is recommended whenever patients are given therapeutic drugs for osteoporosis, to make their calcium (Ca) balance positive. Vitamin D is converted to 25-hydroxyvitamin D in the liver, and then activated to become 1α,25-dihydroxyvitamin D in the kidneys. The active vitamin D acts in the intestine to stimulate Ca absorption and maintain the Ca balance. 2β-(3-Hydroxypropyloxy)-1α,25-dihydroxyvitamin D3 (eldecalcitol) and 2-methylene-19-nor-(20S)-1α,25-dihydroxyvitamin D3 (2MD) are newly developed vitamin D analogs, with a substitution at the 2 position of 1α,25-dihydroxy

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12

Gross, M., and R. Kumar. "Physiology and biochemistry of vitamin D-dependent calcium binding proteins." American Journal of Physiology-Renal Physiology 259, no. 2 (1990): F195—F209. http://dx.doi.org/10.1152/ajprenal.1990.259.2.f195.

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The vitamin D-dependent calcium binding proteins (calbindins) are members of the troponin-C superfamily of proteins that occur in a number of calcium-transporting tissues such as the intestine, the distal tubule of the kidney, and the placenta. They are also present in other tissues such as the brain, peripheral nervous system, pancreas, parathyroid gland, and bone. In some tissues, such as the adult brain, the proteins occur in the absence of the vitamin. The proteins bind calcium in "EF" hand structures and are "calcium-sensitive" in that they undergo a conformational change on binding calci

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13

Kaisermann, Morrys C. "Vitamin K family effects on bone growth." Bone 48, no. 6 (2011): 1427. http://dx.doi.org/10.1016/j.bone.2011.03.757.

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14

Taylor, Sarah N. "Vitamin D in Toddlers, Preschool Children, and Adolescents." Annals of Nutrition and Metabolism 76, no. 2 (2020): 30–41. http://dx.doi.org/10.1159/000505635.

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<b><i>Background:</i></b> Vitamin D supplementation is known to both prevent and treat rickets, a disease of hypomineralized bone. Childhood is a period of great bone development and, therefore, attention to the vitamin D needed to optimize bone health in childhood is imperative. <b><i>Summary:</i></b> Observational studies have pointed to a vitamin D status, as indicated by a 25-hydroxyvitamin D concentration, of 50 nmol/L to ensure avoidance of rickets and of 75 nmol/L to optimize health. However, the benefits of achieving these levels of vitam

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15

Popa, Daniela-Saveta, Galya Bigman, and Marius Emil Rusu. "The Role of Vitamin K in Humans: Implication in Aging and Age-Associated Diseases." Antioxidants 10, no. 4 (2021): 566. http://dx.doi.org/10.3390/antiox10040566.

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As human life expectancy is rising, the incidence of age-associated diseases will also increase. Scientific evidence has revealed that healthy diets, including good fats, vitamins, minerals, or polyphenolics, could have antioxidant and anti-inflammatory activities, with antiaging effects. Recent studies demonstrated that vitamin K is a vital cofactor in activating several proteins, which act against age-related syndromes. Thus, vitamin K can carboxylate osteocalcin (a protein capable of transporting and fixing calcium in bone), activate matrix Gla protein (an inhibitor of vascular calcificatio

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16

Dusso, Adriana S., Alex J. Brown, and Eduardo Slatopolsky. "Vitamin D." American Journal of Physiology-Renal Physiology 289, no. 1 (2005): F8—F28. http://dx.doi.org/10.1152/ajprenal.00336.2004.

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The vitamin D endocrine system plays an essential role in calcium homeostasis and bone metabolism, but research during the past two decades has revealed a diverse range of biological actions that include induction of cell differentiation, inhibition of cell growth, immunomodulation, and control of other hormonal systems. Vitamin D itself is a prohormone that is metabolically converted to the active metabolite, 1,25-dihydroxyvitamin D [1,25(OH)2D]. This vitamin D hormone activates its cellular receptor (vitamin D receptor or VDR), which alters the transcription rates of target genes responsible

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17

Vogelsang, H., P. Ferenci, W. Woloszczuk, et al. "Bone disease in vitamin D-deficient patients with Crohn's disease." Digestive Diseases and Sciences 34, no. 7 (1989): 1094–99. http://dx.doi.org/10.1007/bf01536381.

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18

Norman, Anthony W. "Bone biochemistry and physiology from the perspectives of the vitamin D endocrine system." Current Opinion in Rheumatology 4, no. 3 (1992): 375–82. http://dx.doi.org/10.1097/00002281-199206000-00016.

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19

Minola, E., G. Colussi, M. E. De Ferrari, and R. Brusamolino. "Characterization by bone histology of hypophosphatemic vs vitamin-D deficiency bone disease." Bone 19, no. 3 (1996): 153. http://dx.doi.org/10.1016/s8756-3282(96)90822-4.

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20

Marcinkowska, Ewa. "The Vitamin D System in Humans and Mice: Similar but Not the Same." Reports — Medical Cases, Images, and Videos 3, no. 1 (2020): 1. http://dx.doi.org/10.3390/reports3010001.

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Vitamin D is synthesized in the skin from 7-dehydrocholesterol subsequently to exposure to UVB radiation or is absorbed from the diet. Vitamin D undergoes enzymatic conversion to its active form, 1,25-dihydroxyvitamin D (1,25D), a ligand to the nuclear vitamin D receptor (VDR), which activates target gene expression. The best-known role of 1,25D is to maintain healthy bones by increasing the intestinal absorption and renal reuptake of calcium. Besides bone maintenance, 1,25D has many other functions, such as the inhibition of cell proliferation, induction of cell differentiation, augmentation

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21

Jorde, Rolf, Astrid Kamilla Stunes, Julia Kubiak, et al. "Effects of vitamin D supplementation on bone turnover markers and other bone-related substances in subjects with vitamin D deficiency." Bone 124 (July 2019): 7–13. http://dx.doi.org/10.1016/j.bone.2019.04.002.

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22

Brown, Ronald B., Afrozul Haq, Charles F. Stanford, and Mohammed S. Razzaque. "Vitamin D, phosphate, and vasculotoxicity." Canadian Journal of Physiology and Pharmacology 93, no. 12 (2015): 1077–82. http://dx.doi.org/10.1139/cjpp-2015-0083.

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Vascular calcification is a complex process that results in the ectopic deposition of calcium-phosphate hydroxyapatite. Medial and intimal vascular calcification is frequently present in patients with diabetes mellitus and chronic kidney disease (CKD), and markedly increases the morbidity and mortality of these patients. Increased serum levels of calcium and phosphate, along with the use of active vitamin D metabolites, are commonly implicated in the evolvement of vascular wall mineralization in CKD patients. Because CKD patients have lower serum levels of vitamin D, they are routinely prescri

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23

Jiang, Xiaorui, Botao Huang, Huiying Yang та ін. "TGF-β1 is Involved in Vitamin D-Induced Chondrogenic Differentiation of Bone Marrow-Derived Mesenchymal Stem Cells by Regulating the ERK/JNK Pathway". Cellular Physiology and Biochemistry 42, № 6 (2017): 2230–41. http://dx.doi.org/10.1159/000479997.

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Background/Aims: Osteoarthritis (OA) is characterized by degradation of cartilage, sole cell type of which is chondrocytes. Bone marrow-derived mesenchymal stem cells (BMSCs) possess multipotency and can be directionally differentiated into chondrocytes under stimulation. This study was aimed to explore the possible roles of vitamin D and transforming growth factor-β1 (TGF-β1) in the chondrogenic differentiation of BMSCs. Methods: BMSCs were isolated from femurs and tibias of rats and characterized by flow cytometry. After stimulation with vitamin D, BMSC proliferation and migration were measu

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24

Granjon, David, Olivier Bonny, and Aurélie Edwards. "A model of calcium homeostasis in the rat." American Journal of Physiology-Renal Physiology 311, no. 5 (2016): F1047—F1062. http://dx.doi.org/10.1152/ajprenal.00230.2016.

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We developed a model of calcium homeostasis in the rat to better understand the impact of dysfunctions such as primary hyperparathyroidism and vitamin D deficiency on calcium balance. The model accounts for the regulation of calcium intestinal uptake, bone resorption, and renal reabsorption by parathyroid hormone (PTH), vitamin D3, and Ca2+itself. It is the first such model to incorporate recent findings regarding the role of the calcium-sensing receptor (CaSR) in the kidney, the presence of a rapidly exchangeable pool in bone, and the delayed response of vitamin D3synthesis. Accounting for tw

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Levy, David S., Rickinder Grewal, and Thu H. Le. "Vitamin K deficiency: an emerging player in the pathogenesis of vascular calcification and an iatrogenic consequence of therapies in advanced renal disease." American Journal of Physiology-Renal Physiology 319, no. 4 (2020): F618—F623. http://dx.doi.org/10.1152/ajprenal.00278.2020.

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Vascular calcification is a known complication of chronic kidney disease (CKD). The prevalence of vascular calcification in patients with non-dialysis-dependent CKD stages 3–5 has been shown to be as high as 79% ( 20 ). Vascular calcification has been associated with increased risk for mortality, hospital admissions, and cardiovascular disease ( 6 , 20 , 50 , 55 ). Alterations in mineral and bone metabolism play a pivotal role in the pathogenesis of vascular calcification in CKD. As CKD progresses, levels of fibroblast growth factor-23, parathyroid hormone, and serum phosphorus increase and le

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Kim, G. K., and J. Q. Del Rosso. "Does isotretinoin have effect on vitamin D physiology and bone metabolism in acne patients?" Yearbook of Dermatology and Dermatologic Surgery 2012 (January 2012): 161–62. http://dx.doi.org/10.1016/j.yder.2012.02.001.

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27

Mathews, C. H., R. Brommage, and H. F. DeLuca. "Role of vitamin D in neonatal skeletal development in rats." American Journal of Physiology-Endocrinology and Metabolism 250, no. 6 (1986): E725—E730. http://dx.doi.org/10.1152/ajpendo.1986.250.6.e725.

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The role of vitamin D in rat pup growth and skeletal development without the influence of nutritional factors was investigated. Pups from vitamin D-replete and vitamin D-deficient dams receiving identical amounts of milk for 20 days were compared. Body weight gain, femur ash content and histomorphometric analyses of diaphysial and distal femur were determined. Up to 20 days of age, growth and skeletal development of the pups were completely normal in the absence of vitamin D. Skeletal changes found in vitamin D deficiency were not observed, i.e., there was no increased volume of osteoid or lac

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28

Hauschka, P. V., J. B. Lian, D. E. Cole, and C. M. Gundberg. "Osteocalcin and matrix Gla protein: vitamin K-dependent proteins in bone." Physiological Reviews 69, no. 3 (1989): 990–1047. http://dx.doi.org/10.1152/physrev.1989.69.3.990.

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29

Pigarova, Ekaterina A., and Alexandra A. Petrushkina. "Non-classical effects of vitamin D." Osteoporosis and Bone Diseases 20, no. 3 (2018): 90–101. http://dx.doi.org/10.14341/osteo2017390-101.

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Since the discovery of vitamin D, interest in role of vitamin D in human body is consistently growing, and there is increasing evidence that vitamin D is not only essential to bone health but may also be involved in physiology of many other tissues. Thus understanding of its aspects in particular tissues appears to be important because of possible contribution to pathophysiologic processes. Intracrine regulatory systems associated with widely expressed vitamin D metabolizing enzymes, ways of cellular intake and signal pathways involved are of particular interest. Association of local effects w

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30

Ardawi, M. S., M. H. Qari, and A. A. Rouzi. "Vitamin-D receptor gene polymorphisms and bone mineral density." Bone 47 (June 2010): S150—S151. http://dx.doi.org/10.1016/j.bone.2010.04.347.

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31

Meir, Tomer, Ronen Levi, Liesbet Lieben, et al. "Deletion of the vitamin D receptor specifically in the parathyroid demonstrates a limited role for the receptor in parathyroid physiology." American Journal of Physiology-Renal Physiology 297, no. 5 (2009): F1192—F1198. http://dx.doi.org/10.1152/ajprenal.00360.2009.

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1,25(OH)2D3 decreases parathyroid hormone (PTH) gene transcription through the vitamin D receptor (VDR). Total body VDR−/− mice have high PTH levels, hypocalcemia, hypophosphatemia, and bone malformations. To investigate PTH regulation by the VDR specifically in the parathyroid, we generated parathyroid-specific VDR knockout mice ( PT-VDR−/−). In both strains, there was a decrease in parathyroid calcium receptor (CaR) levels. The number of proliferating parathyroid cells was increased in the VDR−/− mice but not in the PT-VDR−/− mice. Serum PTH levels were moderately but significantly increased

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32

Kito, Hiroaki, Haruka Morihiro, Yuka Sakakibara, et al. "Downregulation of the Ca2+-activated K+ channel KCa3.1 in mouse preosteoblast cells treated with vitamin D receptor agonist." American Journal of Physiology-Cell Physiology 319, no. 2 (2020): C345—C358. http://dx.doi.org/10.1152/ajpcell.00587.2019.

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The maturity of osteoblasts by proliferation and differentiation in preosteoblasts is essential for maintaining bone homeostasis. The beneficial effects of vitamin D on bone homeostasis in mammals have been demonstrated experimentally and clinically. However, the direct actions of vitamin D on preosteoblasts remain to be fully elucidated. In this study, we found that the functional activity of intermediate-conductance Ca2+-activated K+ channels (KCa3.1) positively regulated cell proliferation in MC3T3-E1 cells derived from mouse preosteoblasts by enhancing intracellular Ca2+ signaling. We exam

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33

Zhang, Yan, Wan-Ping Lai, Chun-Fu Wu, Murray J. Favus, Ping-Chung Leung, and Man-Sau Wong. "Ovariectomy worsens secondary hyperparathyroidism in mature rats during low-Ca diet." American Journal of Physiology-Endocrinology and Metabolism 292, no. 3 (2007): E723—E731. http://dx.doi.org/10.1152/ajpendo.00445.2006.

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Estrogen deficiency impairs intestinal Ca absorption and induces bone loss, but its effects on the vitamin D-endocrine system are unclear. In the present study, calciotropic hormones levels, renal vitamin D metabolism, 1,25-dihydroxyvitamin D3 [1,25(OH)2D3]-dependent intestinal calcium absorption, and bone properties in 3-mo-old sham-operated (sham) or ovariectomized (OVX) rats fed either a normal-Ca (NCD; 0.6% Ca, 0.65% P) or a low-Ca (LCD; 0.1% Ca, 0.65% P) diet for 2 wk were determined. LCD increased serum 1,25(OH)2D3 levels in both sham and OVX rats. Serum parathyroid hormone [PTH(1–84)] l

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34

Yanagi, Hisako, Shigeo Tomura, Satoshi Kurihara, Kei Yamana, Hideo Hamaguchi, and Shigeru Tsuchiya. "Vitamin D Receptor Gene Polymorphisms and Bone Mineral Density in Hemodialysis Patients." Nephron 77, no. 1 (1997): 114–15. http://dx.doi.org/10.1159/000190258.

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35

Denis, I., G. Cournot, H. Lacroix, C. Colin, E. Zerath, and A. Pointillart. "In vivo bone metabolism and ex vivo bone marrow osteoprogenitors in vitamin D-deprived pigs." Bone 26, no. 5 (2000): 491–98. http://dx.doi.org/10.1016/s8756-3282(00)00257-x.

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Van Pottelbergh, I., S. Goemaere, D. De Bacquer, A. De Paepe, and M. Kaufman. "Vitamin D receptor gene allelic variants, bone density, and bone turnover in community-dwelling men." Bone 31, no. 5 (2002): 631–37. http://dx.doi.org/10.1016/s8756-3282(02)00867-0.

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37

Mølgaard, C., A. Larnkjær, K. D. Cashman, C. Lamberg-Allardt, J. Jakobsen, and K. F. Michaelsen. "Does vitamin D supplementation of healthy Danish Caucasian girls affect bone turnover and bone mineralization?" Bone 46, no. 2 (2010): 432–39. http://dx.doi.org/10.1016/j.bone.2009.08.056.

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38

Verma, Divij, Rahul Kumar, Raquel S. Pereira, et al. "Vitamin K antagonism impairs the bone marrow microenvironment and hematopoiesis." Blood 134, no. 3 (2019): 227–38. http://dx.doi.org/10.1182/blood.2018874214.

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Abstract Vitamin K antagonists (VKAs) have been used in 1% of the world’s population for prophylaxis or treatment of thromboembolic events for 64 years. Impairment of osteoblast function and osteoporosis has been described in patients receiving VKAs. Given the involvement of cells of the bone marrow microenvironment (BMM), such as mesenchymal stem cells (MSCs) and macrophages, as well as other factors such as the extracellular matrix for the maintenance of normal hematopoietic stem cells (HSCs), we investigated a possible effect of VKAs on hematopoiesis via the BMM. Using various transplantati

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39

Shimada, Takashi, Yuji Yamazaki, Motoo Takahashi, et al. "Vitamin D receptor-independent FGF23 actions in regulating phosphate and vitamin D metabolism." American Journal of Physiology-Renal Physiology 289, no. 5 (2005): F1088—F1095. http://dx.doi.org/10.1152/ajprenal.00474.2004.

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FGF23 suppresses both serum phosphate and 1,25-dihydroxyvitamin D [1,25D] levels in vivo. Because 1,25D itself is a potent regulator of phosphate metabolism, it has remained unclear whether FGF23-induced changes in phosphate metabolism were caused by a 1,25D-independent mechanism. To address this issue, we intravenously administered recombinant FGF23 to vitamin D receptor (VDR) null (KO) mice as a rapid bolus injection and evaluated the early effects of FGF23. Administration of recombinant FGF23 further decreased the serum phosphate level in VDR KO mice, accompanied by a reduction in renal sod

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40

Abdellaoui, Selma, Bilal Bengana, Abdenour Boukabous, and Salima Lefkir-Tafiani. "Vitamin D deficiency pandemic and extra bone effects." Batna Journal of Medical Sciences (BJMS) 7, no. 2 (2020): 142–47. http://dx.doi.org/10.48087/bjmsra.2020.7217.

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Le phénomène de déficit en vitamine D a pris une grande ampleur aujourd’hui, ce déficit connaît un effet de « pandémie » mondiale, il n’épargne aucune frontière ni catégorie d’âge. Les connaissances de la physiologie de cette vitamine ont progressé de manière considérable, faisant passer sa conception d’une hormone purement osseuse à une hormone ayant un rôle sur la santé globale. En effet, son double rôle d’immunomodulateur et de contrôle de la prolifération cellulaire est possible grâce à son métabolite actif secrété de façon autocrine par certains tissus, et la répartition ubiquitaire de se

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Granjon, David, Olivier Bonny, and Aurélie Edwards. "Coupling between phosphate and calcium homeostasis: a mathematical model." American Journal of Physiology-Renal Physiology 313, no. 6 (2017): F1181—F1199. http://dx.doi.org/10.1152/ajprenal.00271.2017.

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We developed a mathematical model of calcium (Ca) and phosphate (PO4) homeostasis in the rat to elucidate the hormonal mechanisms that underlie the regulation of Ca and PO4balance. The model represents the exchanges of Ca and PO4between the intestine, plasma, kidneys, bone, and the intracellular compartment, and the formation of Ca-PO4-fetuin-A complexes. It accounts for the regulation of these fluxes by parathyroid hormone (PTH), vitamin D3, fibroblast growth factor 23, and Ca2+-sensing receptors. Our results suggest that the Ca and PO4homeostatic systems are robust enough to handle small per

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42

Heaney, Robert P. "Vitamin D and Bone Health—Discussion Points Following the Recent Institute of Medicine Recommendations." US Endocrinology 07, no. 02 (2011): 137. http://dx.doi.org/10.17925/use.2011.07.02.137.

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The 2011 Institute of Medicine recommendations for vitamin D—both the recommended daily amount (RDA) and the vitamin D status judged adequate for bone health—are too low. Calcium absorption, osteoporotic fracture risk reduction, and healing of histological osteomalacia all require values above 30 ng/ml, and probably even 40 ng/ml. Furthermore, the proposed RDA (600 international units per day up to the age of 70) is not compatible with the blood level of 25-hydroxyvitamin D (i.e., 20 ng/ml) recommended in the same report. Concerns regarding adverse consequences of higher intakes or status leve

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Genever, P. G., and I. R. Dickson. "Influence of vitamin D status on hyaluronan localization in bone." Bone 18, no. 5 (1996): 429–35. http://dx.doi.org/10.1016/8756-3282(96)00046-4.

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Dickson, I., and J. Walls. "Influence of vitamin a on bone collagen synthesis in vitro." Bone 6, no. 4 (1985): 277. http://dx.doi.org/10.1016/8756-3282(85)90018-3.

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Dickson, I. R., and J. Walls. "Influence of vitamin A on bone matrix formation in vitro." Bone 6, no. 6 (1985): 480. http://dx.doi.org/10.1016/8756-3282(85)90276-5.

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Hodges, SJ, S. Petursson, D. Harrington, C. Hopper, B. Henderson, and M. Harris. "P28. Phylloquinone (vitamin K1) content of bone in young adults." Bone 15, no. 4 (1994): 458. http://dx.doi.org/10.1016/8756-3282(94)90862-1.

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Wong, Dickson, Dana N. Broberg, Jagroop Doad, et al. "Effect of Memantine Treatment and Combination with Vitamin D Supplementation on Body Composition in the APP/PS1 Mouse Model of Alzheimer’s Disease Following Chronic Vitamin D Deficiency." Journal of Alzheimer's Disease 81, no. 1 (2021): 375–88. http://dx.doi.org/10.3233/jad-201137.

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Background: Vitamin D deficiency and altered body composition are common in Alzheimer’s disease (AD). Memantine with vitamin D supplementation can protect cortical axons against amyloid-β exposure and glutamate toxicity. Objective: To study the effects of vitamin D deprivation and subsequent treatment with memantine and vitamin D enrichment on whole-body composition using a mouse model of AD. Methods: Male APPswe/PS1dE9 mice were divided into four groups at 2.5 months of age: the control group (n = 14) was fed a standard diet throughout; the remaining mice were started on a vitamin D-deficient

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Krahenbühl, Tathyane, Juliano Henrique Borges, Antonio de Azevedo Barros-Filho, Gil Guerra-Junior, and Ezequiel Moreira Gonçalves. "Assessment of bone mineral density in young female handball players." Brazilian Journal of Kinanthropometry and Human Performance 20, no. 1 (2018): 102–13. http://dx.doi.org/10.5007/1980-0037.2018v20n1p102.

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Optimizing bone mass gain during childhood and adolescence may help prevent bone diseases in advanced ages. The aim of this study was to verify the bone mineral density (BMD) and bone mineral content (BMC) in female adolescent’s handball players. This is a cross-sectional study where 68 female adolescents (12–17 years) were allocated into two groups: handball players (n = 29) (HG) and control group (n = 39) (CG). BMC and BMD from total body (TB), total body less head (TBLH), lumbar spine (L1–L4), femoral neck (FN), Ward’s triangle (WT) and respectively Z-scores were measured using dual-energy

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Dikker, Okan, Seldag Bekpinar, Gul Ozdemirler, et al. "Evaluation of the Relation Between Omentin-1 and Vitamin D in Postmenopausal Women With or Without Osteoporosis." Experimental and Clinical Endocrinology & Diabetes 126, no. 05 (2017): 316–20. http://dx.doi.org/10.1055/s-0043-120110.

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Abstract Introduction Crosstalk between bone and adipose tissues is implicated in several pathologic conditions related to bone metabolism. Omentin-1, a 34-kD protein, is released from omental adipose tissue. A few studies indicated the effect of omentin-1 on bone health and bone mineral density (BMD) and the interaction of omentin-1 with vitamin D. Therefore, this study aimed to investigate the relationship between omentin-1, vitamin D, and BMD in postmenopausal women with osteoporosis compared with non-osteoporotic counterparts. Materials and methods Forty postmenopausal women with osteoporo

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Okuda, N., S. Takeda, K. Shinomiya, et al. "ED-71, a novel vitamin D analog, promotes bone formation and angiogenesis and inhibits bone resorption after bone marrow ablation." Bone 40, no. 2 (2007): 281–92. http://dx.doi.org/10.1016/j.bone.2006.08.017.

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