Relevant bibliographies by topics / Bradycardia

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Bradycardia'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Bradycardia"

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Gautam, Binod, Ashmita Maharjan, and Suson Ghimire. "Bradycardia during laparoscopic surgeries: A cross-sectional study." Journal of Kathmandu Medical College 9, no. 1 (2020): 5–12. http://dx.doi.org/10.3126/jkmc.v9i1.33515.

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Background: Bradycardia occurring during laparoscopic surgery potentially leads to cardiac arrest and adverse outcomes. Apart from the vagal reflex for its genesis, the knowledge on frequency and risk factors is limited. 
 Objectives: To identify the bradycardia frequency and time points for its occurrence during laparoscopic surgeries.
 Methodology: In this hospital-based cross-sectional study, anaesthesia-related incident reports on bradycardia were collected from January to December 2019. Bradycardias (heart rate less than 60/minute) that occurred during laparoscopic surgeries wer
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Rozloznik, Miroslav, Julian F. R. Paton, and Mathias Dutschmann. "Repetitive paired stimulation of nasotrigeminal and peripheral chemoreceptor afferents cause progressive potentiation of the diving bradycardia." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 296, no. 1 (2009): R80—R87. http://dx.doi.org/10.1152/ajpregu.00806.2007.

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Hallmarks of the mammalian diving response are protective apnea and bradycardia. These cardiorespiratory adaptations can be mimicked by stimulation of the trigeminal ethmoidal nerve (EN5) and reflect oxygen-conserving mechanisms during breath-hold dives. Increasing drive from peripheral chemoreceptors during sustained dives was reported to enhance the diving bradycardia. The underlying neuronal mechanisms, however, are unknown. In the present study, expression and plasticity of EN5-bradycardias after paired stimulation of the EN5 and peripheral chemoreceptors was investigated in the in situ wo
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Cummings, Kevin J., Aihua Li, Evan S. Deneris, and Eugene E. Nattie. "Bradycardia in serotonin-deficient Pet-1−/− mice: influence of respiratory dysfunction and hyperthermia over the first 2 postnatal weeks." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 298, no. 5 (2010): R1333—R1342. http://dx.doi.org/10.1152/ajpregu.00110.2010.

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Neonatal rodents deficient in medullary serotonin neurons have respiratory instability and enhanced spontaneous bradycardias. This study asks if, in Pet-1−/− mice over development: 1) the respiratory instability leads to hypoxia; 2) greater bradycardia is related to the degree of hypoxia or concomitant hypopnea; and 3) hyperthermia exacerbates bradycardias. Pet-1+/+, Pet-1+/−, and Pet-1−/− mice [postnatal days (P) 4–5, P11–12, P14–15] were held at normal body temperature (Tb) and were then made 2°C hypo- and hyperthermic. Using a pneumotach-mask system with ECG, we measured heart rate, metabol
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García-Domingo, Mónica, José Ángel García-Pedraza, Juan Francisco Fernández-González, Cristina López, María Luisa Martín, and Asunción Morán. "Fluoxetine Treatment Decreases Cardiac Vagal Input and Alters the Serotonergic Modulation of the Parasympathetic Outflow in Diabetic Rats." International Journal of Molecular Sciences 23, no. 10 (2022): 5736. http://dx.doi.org/10.3390/ijms23105736.

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Comorbid diabetes and depression constitutes a major health problem, worsening associated cardiovascular diseases. Fluoxetine’s (antidepressant) role on cardiac diabetic complications remains unknown. We determined whether fluoxetine modifies cardiac vagal input and its serotonergic modulation in male Wistar diabetic rats. Diabetes was induced by alloxan and maintained for 28 days. Fluoxetine was administered the last 14 days (10 mg/kg/day; p.o). Bradycardia was obtained by vagal stimulation (3, 6 and 9 Hz) or i.v. acetylcholine administrations (1, 5 and 10 μg/kg). Fluoxetine treatment diminis
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Maltais-Bilodeau, Camille, Maryse Frenette, Geneviève Morissette, et al. "2 Systemic glucocorticoids and bradycardia in critically ill children: a retrospective study." Paediatrics & Child Health 25, Supplement_2 (2020): e1-e1. http://dx.doi.org/10.1093/pch/pxaa068.001.

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Abstract Background Glucocorticoids are widely used in the pediatric population. They are associated with numerous side effects including repercussions on the cardiovascular system. The impact on heart rate is not well known, but bradycardia has been reported, mostly with high doses. Objectives We described the occurrence of bradycardias and the variation of heart rate in critically ill children receiving glucocorticoids. Design/Methods We conducted a retrospective study including 1 month old to 18 year old children admitted to the Pediatric Intensive Care Unit between 2014 and 2017, who recei
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Fortrat, Jacques-Olivier. "Zipf’s Law of Vasovagal Heart Rate Variability Sequences." Entropy 22, no. 4 (2020): 413. http://dx.doi.org/10.3390/e22040413.

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Cardiovascular self-organized criticality (SOC) has recently been demonstrated by studying vasovagal sequences. These sequences combine bradycardia and a decrease in blood pressure. Observing enough of these sparse events is a barrier that prevents a better understanding of cardiovascular SOC. Our primary aim was to verify whether SOC could be studied by solely observing bradycardias and by showing their distribution according to Zipf’s law. We studied patients with vasovagal syncope. Twenty-four of them had a positive outcome to the head-up tilt table test, while matched patients had a negati
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Colombari, E., L. G. Bonagamba, and B. H. Machado. "Mechanisms of pressor and bradycardic responses to L-glutamate microinjected into the NTS of conscious rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 266, no. 3 (1994): R730—R738. http://dx.doi.org/10.1152/ajpregu.1994.266.3.r730.

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Microinjection of increasing doses of L-glutamate (L-Glu, 0.03-5.0 nmol/100 nl) into the nucleus tractus solitarii (NTS) produced a dose-related pressor and bradycardic response. Prazosin virtually abolished the pressor response but produced no changes in the bradycardic response to L-Glu, indicating that bradycardia is not reflex in origin. The bradycardic response was blocked by atropine. In three different groups of rats, excitatory amino acid receptors in the NTS were blocked by increasing doses of kynurenic acid (0.5, 2.0, and 10.0 nmol/100 nl) and the pressor and bradycardic responses to
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Porta-García, Miguel Ángel, Alberto Quiroz-Salazar, Eric Alonso Abarca-Castro, and José Javier Reyes-Lagos. "Bradycardia May Decrease Cardiorespiratory Coupling in Preterm Infants." Entropy 25, no. 12 (2023): 1616. http://dx.doi.org/10.3390/e25121616.

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Bradycardia, frequently observed in preterm infants, presents significant risks due to the immaturity of their autonomic nervous system (ANS) and respiratory systems. These infants may face cardiorespiratory events, leading to severe complications like hypoxemia and neurodevelopmental disorders. Although neonatal care has advanced, the influence of bradycardia on cardiorespiratory coupling (CRC) remains elusive. This exploratory study delves into CRC in preterm infants, emphasizing disparities between events with and without bradycardia. Using the Preterm Infant Cardio-Respiratory Signals (PIC
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Ahmad, Munir, Muhammad Yasir, and Sehar Fatima. "DRUG INDUCED BRADYCARDIA." Professional Medical Journal 25, no. 06 (2018): 908–13. http://dx.doi.org/10.29309/tpmj/2018.25.06.280.

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Background: Bradycardia in patients on rate slowing drugs i.e. beta blockers,digoxin and non dihydropyridine calcium channel blockers is common after discontinuation ofrate slowing drugs. Bradycardia persists in majority of patients, so bradycardia is not truly druginduced but due to underlying conduction system disease. Objectives: To determine the outcomein patients with bradycardia after discontinuation of rate slowing drugs in terms of frequencyof persistent bradycardia. Study Design: Descriptive cross-sectional. Place and Duration ofStudy: Cardiology Department, Faisalabad Institute of Ca
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Cardinal, René, Pierre Pagé, Michel Vermeulen, et al. "Spinal cord stimulation suppresses bradycardias and atrial tachyarrhythmias induced by mediastinal nerve stimulation in dogs." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 291, no. 5 (2006): R1369—R1375. http://dx.doi.org/10.1152/ajpregu.00056.2006.

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Spinal cord stimulation (SCS) applied to the dorsal aspect of the cranial thoracic cord imparts cardioprotection under conditions of neuronally dependent cardiac stress. This study investigated whether neuronally induced atrial arrhythmias can be modulated by SCS. In 16 anesthetized dogs with intact stellate ganglia and in five with bilateral stellectomy, trains of five electrical stimuli were delivered during the atrial refractory period to right- or left-sided mediastinal nerves for up to 20 s before and after SCS (20 min). Recordings were obtained from 191 biatrial epicardial sites. Before
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Dissertations / Theses on the topic "Bradycardia"

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Smith-White, Margaret A. Medical Sciences Faculty of Medicine UNSW. "A functional study of neuropeptide Y mediated attenuation of vagal-evoked bradycardia." Awarded by:University of New South Wales. School of Medical Sciences, 2003. http://handle.unsw.edu.au/1959.4/19090.

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In the heart, neuropeptide Y (NPY) released during stimulation of the sympathetic nerve attenuates vagal-evoked bradycardia for a prolonged period. The inhibitory action of NPY is proposed as being Y2 receptor mediated. In rat and mouse, anaesthetised with sodium pentobarbitone, the selective Y2 receptor antagonist BIIE0246 reduced the inhibition of cardiac vagal activity evoked by a Y2 agonist, N-acetyl [Leu28, 31] NPY 24-36. BIIE0246 also reduced the inhibitory effect on vagal action evoked by stimulation of the sympathetic nerve. Deletion of the receptor in Y2 receptor-knockout mice ab
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Ruck, Sylvia A. "Induced Bradycardia Effects on Angiogenesis, Growth and Development in Early Development in Chicken Embryos, Gallus Domesticus." Thesis, University of North Texas, 2010. https://digital.library.unt.edu/ark:/67531/metadc33199/.

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Cardiac performance, angiogenesis and growth was investigated during early chicken development. Heart rate, and thus arterial pulse pressure and cardiac output, were altered with the bradycardic drug ZD7288. Heart rates at 72 h of development of control embryos and those dosed with chicken Ringer were not different at 171 bpm. Acute and chronic application of ZD7288 caused significant bradycardia. Chronic dosing of Ringer and ZD7288 changed neither eye diameter nor development rate. Chronic dosing of ZD7288 did not significantly alter CAM vessel density close to the embryo (2, 3 and 4 mm) but
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Manley, Elizabeth. "Apneic Bradycardia : terrestrial and aquatic responses in man under working conditions." Thesis, Rhodes University, 1989. http://hdl.handle.net/10962/d1001838.

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The focus of this research was the reappraisal in physiological and psychophysical terms of current equivocal theories regarding the onset, course and termination of apneic bradycardia. Sixteen healthy male subjects participated in four separate testing sessions . Maximal oxygen consumption (VO₂ max) was measured on land and underwater using an identical direct, continuous progressive cycle ergometer test. On each of two other occasions subjects exercised in either environment at 50, 70 and 90% of the appropriate VO₂ max, during which time heart rate was continuously recorded. An initial apnei
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Subramanian, Melanie. "Relative Bradycardia With Hypertension in Traumatic Brain Injury: A Marker for Mortality?" Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:17295909.

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Traumatic brain injury (TBI) is a leading cause of death in patients under 45 years of age. Hypertension has been shown to be associated with increased mortality in patients with moderate to severe traumatic brain injury (TBI). Furthermore, it is known that relative bradycardia is associated with increased mortality in hypotensive patients. We conducted a study to evaluate the relationship of initial heart rate (HR) with outcome in hypertensive TBI patients. This was a retrospective study of adult patients with moderate to severe blunt TBI (GCS <13) that presented to a Level I trauma center (2
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St-Hilaire, Marie. "Chémoréflexes laryngés induits par l'acide, l'eau vs le salin chez les agneaux nouveau-nés durant le sommeil calme." Mémoire, Université de Sherbrooke, 2004. http://savoirs.usherbrooke.ca/handle/11143/3789.

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Mise en contexte : Les chémoréflexes laryngés (CRI) sont déclenchés suite au contact entre un liquide et la muqueuse laryngée. Chez un organisme mature, ces CRL sont responsables de mécanismes de protection des voies aériennes inférieures (VAI) tels que déglutitions, toux et réaction d'éveil afin d'éviter l'aspiration. Par contre, chez un organisme immature comme c'est le cas chez les nouveau-nés, ces CRL associent apnée, bradycardie, laryngospasme, hypertension et redistribution du débit sanguin. En période néonatale, ces CRL, déclenchés en réponse à un reflux gastro-oesophagien acide, sont t
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Mateos, Juan Carlos Pachon. "Estudo comparativo dos parâmetros eletrofisiológicos da estimulação endocárdica septal com a estimulação cardíaca endocárdica convencional." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/98/98131/tde-12062012-074413/.

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Fundamento: A estimulação endocárdica convencional do ventrículo direito em ápice ou na região subtricuspídea ocasiona grande alargamento do QRS e importante dessincronização do miocárdio comprometendo a função ventricular. Com o surgimento da estimulação bifocal do VD e com a necessidade de estimulação cardíaca menos deletéria, a estimulação septal do VD tem sido cada vez mais utilizada. Eventualmente têm sido relatados limiares de estimulação mais altos e ondas R menores na estimulação septal. Objetivo: Comparar os parâmetros eletrofisiológicos das estimulações apical e septal, no mesmo paci
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Lindholm, Peter. "Severe hypoxemia during apnea in humans : influence of cardiovascular responses /." Stockholm, 2002. http://diss.kib.ki.se/2002/91-7349-314-7/.

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Mosca, Nancy Walsh. "Holding premature infants during gavage feeding: Effect on apnea, bradycardia, oxygenation, gastric residual, gastrin, and behavioral state." Case Western Reserve University School of Graduate Studies / OhioLINK, 1995. http://rave.ohiolink.edu/etdc/view?acc_num=case1057766341.

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Montazeri, Ghahjaverestan Nasim. "Early detection of cardiac arrhythmia based on Bayesian methods from ECG data." Thesis, Rennes 1, 2015. http://www.theses.fr/2015REN1S061/document.

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L'apnée est une complication fréquente chez les nouveaux-nés prématurés. L'un des problèmes les plus fréquents est l'épisode d'apnée bradycardie dont la répétition influence de manière négative le développement de l'enfant. C'est pourquoi les enfants prématurés sont surveillés en continu par un système de monitoring. Depuis la mise en place de ce système, l'espérance de vie et le pronostic de vie des prématurés ont été considérablement améliorés et ainsi la mortalité réduite. En effet, les avancées technologiques en électronique, informatique et télécommunications ont conduit à l'élaboration d
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Wung, Shu-Fen. "Bradyarrhythmias: Clinical Presentation, Diagnosis, and Management." W B SAUNDERS CO-ELSEVIER INC, 2016. http://hdl.handle.net/10150/621215.

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Bradyarrhythmias are common clinical findings consisting of physiologic and pathologic conditions (sinus node dysfunction and atrioventricular [AV] conduction disturbances). Bradyarrhythmias can be benign, requiring no treatment; however, acute unstable bradycardia can lead to cardiac arrest. In patients with confirmed or suspected bradycardia, a thorough history and physical examination should include possible causes of sinoatrial node dysfunction or AV block. Management of bradycardia is based on the severity of symptoms, the underlying causes, presence of potentially reversible causes, pres
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Books on the topic "Bradycardia"

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Lennon, Krystal. Euphoric Bradycardia. PublishAmerica, 2006.

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PILBERY. Standby Cpd: Symptomatic Bradycardia. Class Publishing, 2015.

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Jr, Joseph C. Kunz, and Michele G. Kunz. Zombie Notes Bradycardia/Heartblocks. Dickson Keanaghan, LLC, 2011.

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Swain, Una Gabriele. Heart rates and diving bradycardia in beaver. 1985.

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Burnett, Catherine. Effects of choline and exercise on resting bradycardia in rats. 1988.

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Burnett, Catherine. Effects of choline and exercise on resting bradycardia in rats. 1988.

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Publications, ICON Health. Bradycardia - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Sinus nodal disease. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199685288.003.1459_update_003.

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Gill, Harminder S., and Jaswinder S. Gill. Causes, diagnosis, and therapeutic strategy in bradyarrhythmias. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0157.

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Bradyarrhythmias (defined as a heart rate &lt;60 beat/min) occur frequently in the critical care setting. Most are related to underlying disease processes and the multidrug therapies administered. Because of the intense monitoring of these patients, recognition is generally easy. Examination of the ECG will allow diagnosis of the type of bradycardia based on the sinus node, atrioventricular node and the infra-Hissian conducting system. The extent of conduction system disease can be estimated and this has an influence on the prognosis. Bradycardias causing haemodynamic collapse require treatmen
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Ramrakha, Punit, and Jonathan Hill, eds. Arrhythmias. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199643219.003.0010.

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The cardiac conduction system 478Bradycardia: general approach 480Sinus bradycardia 482Sinus pause 482Sick sinus syndrome 482Atrioventricular block 483Bundle branch block 484Tachycardia: general approach 486Tachycardia: emergency management 488Tachyarrhythmias: classification 490ECG diagnosis of tachyarrhythmias 492Supraventricular tachycardia 494...
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Book chapters on the topic "Bradycardia"

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Hudson, Korin B., J. Aidan Boswick, and William J. Brady. "Bradycardia." In The ECG in Prehospital Emergency Care. Blackwell Publishing Ltd., 2012. http://dx.doi.org/10.1002/9781118473740.ch8.

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Hsing, Jeffrey, and Paul Wang. "Bradycardia." In A Practical Approach to Cardiovascular Medicine. Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444393897.ch16.

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Good, Eric D., and Krit Jongnarangsin. "Bradycardia." In Practical Cardiology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-28328-5_17.

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Kusumoto, Fred M. "Bradycardia." In ECG Interpretation: From Pathophysiology to Clinical Application. Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-88880-4_10.

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Kibler, Hannah, and Sharon Vannoy. "Bradycardia." In Cardiovascular Manual for the Advanced Practice Provider. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-35819-7_8.

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Kusumoto, Fred. "Bradycardia." In ECG Interpretation. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-40341-6_10.

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McCombe, Kate, and Lara Wijayasiri. "Bradycardia." In The Primary FRCA Structured Oral Examination Study Guide 2, 3rd ed. CRC Press, 2025. https://doi.org/10.1201/9781003276470-60.

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Fok, Henry, Kerry Layne, and Adam Nabeebaccus. "Bradycardia and Malaise." In 100 Cases in Acute Medicine, 2nd ed. CRC Press, 2022. http://dx.doi.org/10.1201/9781003241171-57.

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Aktas, Mehmet K., and Ryan Mandell. "Cardiac Conduction and Bradycardia." In Clinical Cardiac Electrophysiology in Clinical Practice. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-5433-4_1.

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Upadhyay, Gaurav A., and Jagmeet P. Singh. "Bradycardia and Pacemakers/CRT." In MGH Cardiology Board Review. Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4483-0_25.

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Conference papers on the topic "Bradycardia"

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Ijaz, Sitara, Salaar Khan, Zulqarnain Asghar, and Haseeb Iqbal. "Detection of Sinus Bradycardia with Electrocardiogram using Machine Learning Techniques." In 2024 26th International Multitopic Conference (INMIC). IEEE, 2024. https://doi.org/10.1109/inmic64792.2024.11004401.

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Ayman, Nourhan, Aya Hesham, Razan Ehab, Mariam Emad, Ismail M. El-Badawy, and Zaid Omar. "ECG Baseline Wander Reduction Using Empirical Mode Decomposition: A Case Study on Preterm Infants With Bradycardia." In 2024 IEEE 8th International Conference on Signal and Image Processing Applications (ICSIPA). IEEE, 2024. http://dx.doi.org/10.1109/icsipa62061.2024.10686609.

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Sahoo, Nibedita, P. Unnikrishnan, Smita Vimala, and Ajay P. Hrishi. "Ictal Bradycardia: A Missed Etiology for Intraoperative Bradycardia." In 19th Annual Conference of the Indian Society of Neuroanaesthesiology and Critical Care (ISNACC). Thieme Medical and Scientific Publishers Private Limited, 2018. http://dx.doi.org/10.1055/s-0038-1636386.

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Wang, Qingjie, Sanjay Kharche, Gareth Jones, Cunjin Luo, Chengchun Tang, and Henggui Zhang. "Calcium leak induced sinus bradycardia." In 2015 Computing in Cardiology Conference (CinC). IEEE, 2015. http://dx.doi.org/10.1109/cic.2015.7411109.

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Schuitema, Kelly. "BRASH Syndrome Case Report and Brief Review." In 27th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2023. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.108_2023.

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BRASH syndrome is an acronym that stands for bradycardia, renal failure, AV node blocker, shock and hyperkalemia. The syndrome is precipitated by synergism from AV nodal blocking agents and hyperkalemia. The combination of the AV nodal blockade and renal failure leads to a cycle of severe bradycardia and hyperkalemia. We will discuss a case of BRASH syndrome with a 68 year old female who presented with generalized weakness and was found to be hypotensive, bradycardia, in acute renal failure with hyperkalemia likely related to metoprolol and amlodipine use.
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Killian, Peter, and Adam Kandil. "BRASH Syndrome: A Deadly Pentad of Symptoms." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.48_2024.

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Here we present the case of a male in his 70’s who presented with diarrhea and a syncopal episode and was ultimately diagnosed with BRASH syndrome. The acronym BRASH is a novel way to describe the constellation of symptoms that result in unfortunate synergistic affects that can result in profound bradycardia. In the setting of hyperkalemia and concurrent use of atrioventricular nodal blockers, there is potential for a worsening bradycardia and inevitable cardiovascular collapse if not identified.
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Patil, Kaustubh, and Rajesh Radhakrishnan. "Leadless pacing: Addressing barriers for bradycardia therapy in India." In 2013 IEEE Point-of-Care Healthcare Technologies (PHT). IEEE, 2013. http://dx.doi.org/10.1109/pht.2013.6461343.

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Yutsis, P. I., and M. J. Bucksbaum. "Computerized polygraphic audiovisual monitoring in infants with apnea-bradycardia." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.95041.

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Loganathan, Prakash Kannan, Prashant Mallya, and Vrinda Nair. "682 Hypoxia and Bradycardia episodes in neonatal elective intubations." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 15 June 2021–17 June 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-rcpch.141.

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Pravisani, G., A. Beuchee, L. Mainardi, and G. Carrault. "Short term prediction of severe bradycardia in premature newborns." In Computers in Cardiology, 2003. IEEE, 2003. http://dx.doi.org/10.1109/cic.2003.1291258.

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Reports on the topic "Bradycardia"

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Ai, Ming-Ying, and CJ Yang. Remdesivir-induced Bradycardia and Mortality in SARS-CoV-2 Infection: A Systematic Review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.6.0066.

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Claus, Ana, Borzooye Jafarizadeh, Azmal Huda Chowdhury, Neziah Pala, and Chunlei Wang. Testbed for Pressure Sensors. Florida International University, 2021. http://dx.doi.org/10.25148/mmeurs.009771.

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Currently, several studies and experiments are being done to create a new generation of ultra-low-power wearable sensors. For instance, our group is currently working towards the development of a high-performance flexible pressure sensor. However, with the creation of new sensors, a need for a standard test method is necessary. Therefore, we opted to create a standardized testbed to evaluate the pressure applied to sensors. A pulse wave is generated when the heart pumps blood causing a change in the volume of the blood vessel. In order to eliminate the need of human subjects when testing press
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Harris, Gregory, Brooke Hatchell, Davelin Woodard, and Dwayne Accardo. Intraoperative Dexmedetomidine for Reduction of Postoperative Delirium in the Elderly: A Scoping Review. University of Tennessee Health Science Center, 2021. http://dx.doi.org/10.21007/con.dnp.2021.0010.

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Background/Purpose: Post-operative delirium leads to significant morbidity in elderly patients, yet there is no regimen to prevent POD. Opioid use in the elderly surgical population is of the most significant risk factors for developing POD. The purpose of this scoping review is to recognize that Dexmedetomidine mitigates cognitive dysfunction secondary to acute pain and the use of narcotic analgesia by decreasing the amount of norepinephrine (an excitatory neurotransmitter) released during times of stress. This mechanism of action also provides analgesia through decreased perception and modul
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