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Relevant bibliographies by topics / Brain Alcoholism Alcohol / Dissertations / Theses

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Dissertations / Theses on the topic 'Brain Alcoholism Alcohol'

Author: Grafiati

Published: 4 June 2021

Last updated: 18 February 2022

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1

Arlinde, Christina. "Gene expression profiling in animal models of alcoholism /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7140-133-4/.

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2

van, der Vaart Andrew D. "Molecular Brain Adaptations to Ethanol: Role of Glycogen Synthase Kinase-3 Beta in the Transition to Excessive Consumption." VCU Scholars Compass, 2018. https://scholarscompass.vcu.edu/etd/5510.

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Alcoholism is a complex neuropsychiatric disease that is characterized by compulsive alcohol use and intensifying cravings and withdrawals, often culminating in physiologic dependency. Fundamental alterations in brain chemistry underlie the transition from initial ethanol exposure to repetitive excessive use. Key mediators of this adaptation include changes in gene expression and signal transduction. Here we investigated gene expression pathways in prefrontal cortex and nucleus accumbens following acute or chronic ethanol treatment, to identify genes with potentially conserved involvement in t

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3

Alexander-Kaufman, Kimberley Louise. "Proteomics of the human alcoholic brain: Implications for the pathophysiology of alcohol-related brain damage." The University of Sydney, 2008. http://hdl.handle.net/2123/2692.

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Doctor of Philosophy (PhD)<br>Proteomics is rapidly achieving recognition as a complimentary and perhaps superior approach to examine global changes in protein abundance in complex biological systems and the value of these techniques in neuropsychiatry is beginning to be acknowledged. Characterizing the brain’s regional proteomes provides a foundation for the detection of proteins that may be involved in disease-related processes. Firstly, optimal conditions were achieved for the application of two dimensional-gel electrophoresis (2D-GE)-based proteomics with postmortem human brain tissue. The

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4

Hård, Julia. "Långvarigt bruk av alkohol ger kramper och epilepsi : Ett arbete om alkohols effekter på hjärnan." Thesis, Linnéuniversitetet, Institutionen för kemi och biomedicin (KOB), 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-65066.

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Alkohol har funnits sedan urminnes tider och är något som de flesta ungdomar och vuxna är bekanta med. Flertalet vet också att för mycket alkohol på lång sikt kan orsaka skador, framförallt på lever (fettlever) och njurar. Men inte alla vet att alkohol skadar hjärnan och kan ge kramper samt epilepsi. Alkohol har olika effekter på kroppen. Akut kan det öka den inhiberande och minska den excitatoriska signaleringen i hjärnan. Långvarigt kan det öka den excitatoriska signaleringen, minska den inhibitoriska samt öka alkoholtoleransen. I hjärnan är balansen mellan den inhibitoriska och excitatorisk

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5

Buckley, Stella Tracey. "GabaA receptor-mediated neurotransmission in human alcoholic brain /." St. Lucia, Qld, 2003. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe17286.pdf.

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6

Bazov, Igor. "Epigenetic Dysregulations in the Brain of Human Alcoholics : Analysis of Opioid Genes." Doctoral thesis, Uppsala universitet, Institutionen för farmaceutisk biovetenskap, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-270321.

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Neuropeptides are special in their expression profiles restricted to neuronal subpopulations and low tissue mRNA levels. Genetic, epigenetic and transcriptional mechanisms that define spatiotemporal expression of the neuropeptide genes have utmost importance for the formation and functions of neural circuits in normal and pathological human brain. This thesis focuses on regulation of transcription of the opioid/nociceptin genes, the largest neuropeptide family, and on identification of adaptive changes in these mechanisms associated with alcoholism as model human pathology. Two epigenetic mech

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7

Roy, Arnab. "Evolving spike neural network based spatio-temporal pattern classifiers with an application to identifying the alcoholic brain." Thesis, State University of New York at Binghamton, 2014. http://pqdtopen.proquest.com/#viewpdf?dispub=3630956.

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<p> We introduce a novel approach to evolving spike neural network (SNN) based Spatio-temporal (ST) pattern classifiers that can detect occurrences of hidden structures in a ST data. We test this learning paradigm to find characteristic electrical patterns in visually evoked response potentials (VERPs) generated by an alcoholic brain. </p><p> We cast the alcoholic classification task as a multiple feature selection (FS) problem. The FS problems are grouped under 2 classes: the spatial task and the temporal task. The objective of the spatial FS task is to choose a correct subset of electroen

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8

Troni, Kelly Lendini 1980. "Estudo sobre o uso do resíduo da etapa de deceragem de óleo de farelo de arroz para a produção de ácidos graxos e álcoois graxos." [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/266628.

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Orientador: Roberta Ceriani<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Engenharia Química<br>Made available in DSpace on 2018-08-23T01:21:42Z (GMT). No. of bitstreams: 1 Troni_KellyLendini_M.pdf: 3567932 bytes, checksum: fb83c16b04e83a1bcc666f11c5237dec (MD5) Previous issue date: 2013<br>Resumo: A cera do óleo de farelo de arroz (OFA) é um resíduo da etapa de deceragem no processo de refino de óleos. Tradicionalmente, as ceras naturais, que em excesso geram turbidez no óleo vegetal embalado, são removidas por cristalização e filtração a baixa temperatura. Ess

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9

Berman, Ari Ethan. "Brain region gene expression responds discretely to chronic alcohol withdrawal with specific disruption of the hippocampus during intoxication." Thesis, 2005. http://hdl.handle.net/2152/2686.

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10

Liu, Jianwen. "Studies of the global gene expression changes in alcoholic human brain and blood." Thesis, 2005. http://hdl.handle.net/2152/2267.

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11

Toalston, Jamie E. "Peri-adolescent Alcohol Consumption Enhances the Reinforcing and Stimulatory Properties of Ethanol within the Adult Mesolimbic Dopamine System in Alcohol Preferring P Rats." Thesis, 2012. http://hdl.handle.net/1805/2893.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Research in the alcohol preferring (P) rat has indicated that peri-adolescent alcohol (EtOH) consumption enhances the acquisition of oral operant EtOH self-administration, inhibits the extinction of responding for EtOH, augments EtOH-seeking behaviors, and increases relative reward value of EtOH during adulthood. Experiment 1 was conducted to determine if these adult effects of peri-adolescent EtOH intake could be observed using an Intracranial Self-Administration (ICSA) model. It was hypothesized that an increased sensitivity to the

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12

Lenne, Frida. "Studies attempting to reverse brain damages induced by alcohol with the aim to find new strategies for treatment of patients with alcoholism." Thesis, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-205218.

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13

Hartshorne, Claire. "The effects of alcoholic hangover on human performance." Thesis, 2000. http://hdl.handle.net/10413/3812.

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This dissertation aims at determining the possible effects of alcoholic hangover on human behaviour by examining the effects of acute alcohol consumption (> 1g/kg) 14-16 hours following alcohol ingestion on simple and choice reaction times, divided attention tasks and driving skills. The hypotheses are that cognitive and behavioural functioning is impaired even after the blood alcohol concentration level has returned to zero The California Computerised Assessment Package (CALCAP) together with selected driving skills tasks, repeated breath analysis measures, a biographical questionnaire, a sub

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14

Oster, Scott M. "Chronic Ethanol Drinking by Alcohol-preferring Rats Increases the Sensitivity of the Mesolimbic Dopamine System to the Reinforcing and Stimulating Effects of Cocaine." Thesis, 2013. http://hdl.handle.net/1805/3440.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Alcohol and cocaine are commonly co-abused drugs, and those meeting criteria for both cocaine and alcohol use disorders experience more severe behavioral and health consequences than those with a single disorder. Chronic alcohol (ethanol) drinking increased the reinforcing and dopamine (DA) neuronal stimulating effects of ethanol within mesolimbic regions of the central nervous system (CNS) of alcohol-preferring (P) rats. The objectives of the current study were to determine if chronic continuous ethanol drinking produced: (1) altera

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15

Wagner, Jennifer Lynne. "Effects of coadministration of D-Napvsipq [NAP] and D-Sallrsipa [SAL] on spatial learning after developmental alcohol exposure." Thesis, 2013. http://hdl.handle.net/1805/5066.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Despite warnings about the dangers of drinking during pregnancy, little progress has been made in reducing alcohol drinking among women of childbearing age. Even after the recognition of pregnancy, 15% of women continue to drink, 3% of which admit to binge drinking. Because we cannot stop women from drinking during pregnancy, and many children with fetal alcohol spectrum disorders (FASD) are adopted, there is a significant need to develop postnatal interventions that can improve the long-term outcome of children adversely affected by

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16

Sugarman, Roy. "A neuropsychological investigation of the role of cortical arousal in the alcohol related brain syndrome." Thesis, 2014. http://hdl.handle.net/10210/10109.

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D.Litt. et Phil.<br>The present work set out to evaluate whether the division on a neuropsychological basis between Korsakoff's amnesia and Kevin Walsh's Adaptive Behavioural Syndrome (ABS) was justified (Walsh, 1989). The research took the approach that the supposed agents responsible for the ABS (neurotoxicity of alcohol) and Korsakoff's syndrome (thiamine avitaminosis) had not been proven to produce site-specific lesions. Using Bowden (1990) as a point of departure, Luria's (1973) classic discussion of the hierarchical nature of brain functioning was used to generate the hypothesis that the

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17

Lee, Chang Hoon. "Splicing of human GABAB receptor subunit 1 (GABAB1) in non-alcoholic and alcoholic brains." Thesis, 2011. http://hdl.handle.net/2152/ETD-UT-2011-12-4576.

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Gamma-aminobutyric acid type B (GABAB) receptor is a G protein coupled receptor (GPCR) that mediates decreased neural activity. It has two subunits, GABAB1 and GABAB2. Previous complementary DNA (cDNA) microarray data showed strong GABAB1 signals from human prefrontal cortex using an intron 4 region probe, and these studies indicated that novel intron 4 containing GABAB1 splicing variants exist. We cloned GABAB1k, l, m, and n including mouse GABAB1j. Expression of these variants are much lower than other major known splicing variants, but GABAB1k, l, m, and n levels are similar across brain ti

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18

Panyod, Suraphan, and 裴尤德. "Investigation of Allicin on Gut Microbiota-Liver-Brain Interaction in Mice with Alcoholic Fatty Liver Disease." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/jsdt76.

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博士<br>國立臺灣大學<br>食品科技研究所<br>105<br>Excessive alcohol consumption can cause alcoholic fatty liver disease (AFLD). Recently, the scientists have found that gut microbiota may play a role in the pathogenesis or progression of certain liver diseases, including alcoholic liver disease, either through the direct effects of bacteria or their by-products (lipopolysaccharides, LPS) by triggering inflammatory mediators such as tumor necrosis factor α (TNF-α). These inflammatory mediators can lead to systemic inflammation and organ damage, particularly to the liver and brain. Allicin is a pharmacologicall

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19

Huang, Ming-Chyi, and 黃名琪. "Differential Patterns of Serum Brain-Derived Neurotrophic Factor Levels and Circadian Rhythm Gene Expression in Alcoholic Patients with and without Delirium Tremens During Acute Withdrawal." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/85732885295130964202.

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博士<br>臺北醫學大學<br>醫學科學研究所<br>98<br>Background: Alcohol withdrawal-enhanced neuroadaptation contributes to the addictive process. Delirium tremens (DTs) is the most serious complication of alcohol withdrawal syndrome (AWS) and postulated to be a clinically distinct phenotype among AWS. Brain-derived neurotrophic factor (BDNF) plays an important role in neuronal plasticity and learning related to addiction. Meanwhile, current evidence indicated a clear interaction between chronic alcohol consumption and disrupted circadian rhythmicity which is regulated by several circadian clock genes including h

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