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Journal articles on the topic 'Brain motion'

Author: Grafiati
Published: 4 June 2021
Last updated: 15 February 2022

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1

BRANDT, THOMAS. "MAN IN MOTION." Brain 114, no. 5 (1991): 2159–74. http://dx.doi.org/10.1093/brain/114.5.2159.

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2

Schmidt-Kassow, Maren, and Stefan Debener. "Editorial: Brain in Motion." Brain Research 1716 (August 2019): 1–2. http://dx.doi.org/10.1016/j.brainres.2019.01.027.

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3

Cornette, L. "Human cerebral activity evoked by motion reversal and motion onset. A PET study." Brain 121, no. 1 (January 1, 1998): 143–57. http://dx.doi.org/10.1093/brain/121.1.143.

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4

ZEKI, S. "CEREBRAL AKINETOPSIA (VISUAL MOTION BLINDNESS)." Brain 114, no. 2 (1991): 811–24. http://dx.doi.org/10.1093/brain/114.2.811.

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5

Schnider, Armin, Klemens Gutbrod, and Christian W. Hess. "Motion imagery in Parkinson's disease." Brain 118, no. 2 (1995): 485–93. http://dx.doi.org/10.1093/brain/118.2.485.

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6

David, Anthony S., and Carl Senior. "Implicit motion and the brain." Trends in Cognitive Sciences 4, no. 8 (August 2000): 293–95. http://dx.doi.org/10.1016/s1364-6613(00)01511-4.

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7

Azzopardi, Paul, and Alan Cowey. "Motion discrimination in cortically blind patients." Brain 124, no. 1 (January 2001): 30–46. http://dx.doi.org/10.1093/brain/124.1.30.

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8

Kyme, Andre Z., Stephen Se, Steven R. Meikle, and Roger R. Fulton. "Markerless motion estimation for motion-compensated clinical brain imaging." Physics in Medicine & Biology 63, no. 10 (May 17, 2018): 105018. http://dx.doi.org/10.1088/1361-6560/aabd48.

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9

Ajina, Sara, Christopher Kennard, Geraint Rees, and Holly Bridge. "Motion area V5/MT+ response to global motion in the absence of V1 resembles early visual cortex." Brain 138, no. 1 (November 27, 2014): 164–78. http://dx.doi.org/10.1093/brain/awu328.

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10

Rizzo, Matthew, Mark Nawrot, and Josef Zihl. "Motion and shape perception in cerebral akinetopsia." Brain 118, no. 5 (1995): 1105–27. http://dx.doi.org/10.1093/brain/118.5.1105.

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11

ffytche, D. H., C. N. Guy, and S. Zeki. "Motion specific responses from a blind hemifield." Brain 119, no. 6 (1996): 1971–82. http://dx.doi.org/10.1093/brain/119.6.1971.

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12

Vaina, Lucia M., Alan Cowey, Rhea T. Eskew, Marjorie LeMay, and Thomas Kemper. "Regional cerebral correlates of global motion perception." Brain 124, no. 2 (February 2001): 310–21. http://dx.doi.org/10.1093/brain/124.2.310.

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13

Wild, Benedict. "How Does the Brain Tell Self-Motion from Object Motion?" Journal of Neuroscience 38, no. 16 (April 18, 2018): 3875–77. http://dx.doi.org/10.1523/jneurosci.0039-18.2018.

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14

Deutschländer, Angela, Katharina Hüfner, Roger Kalla, Thomas Stephan, Thomas Dera, Stefan Glasauer, Martin Wiesmann, Michael Strupp, and Thomas Brandt. "Unilateral vestibular failure suppresses cortical visual motion processing." Brain 131, no. 4 (March 5, 2008): 1025–34. http://dx.doi.org/10.1093/brain/awn035.

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15

Yoshiyama, Kenji, Akinori Nakamura, Kersten Diers, Masahiko Bundo, Kentaro Ono, Yoshiya Mori, Hideyuki Hattori, and Kengo Ito. "Brain magnetic responses to biological motion." Neuroscience Research 58 (January 2007): S229. http://dx.doi.org/10.1016/j.neures.2007.06.519.

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16

Okamoto, Ruth J., Anthony J. Romano, Curtis L. Johnson, and Philip V. Bayly. "Insights Into Traumatic Brain Injury From MRI of Harmonic Brain Motion." Journal of Experimental Neuroscience 13 (January 2019): 117906951984044. http://dx.doi.org/10.1177/1179069519840444.

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Measurements of dynamic deformation of the human brain, induced by external harmonic vibration of the skull, were analyzed to illuminate the mechanics of mild traumatic brain injury (TBI). Shear wave propagation velocity vector fields were obtained to illustrate the role of the skull and stiff internal membranes in transmitting motion to the brain. Relative motion between the cerebrum and cerebellum was quantified to assess the vulnerability of connecting structures. Mechanical deformation was quantified throughout the brain to investigate spatial patterns of strain and axonal stretch. Strain magnitude was generally attenuated as shear waves propagated into interior structures of the brain; this attenuation was greater at higher frequencies. Analysis of shear wave propagation direction indicates that the stiff membranes (falx and tentorium) greatly affect brain deformation during imposed skull motion as they serve as sites for both initiation and reflection of shear waves. Relative motion between the cerebellum and cerebrum was small in comparison with the overall motion of both structures, which suggests that such relative motion might play only a minor role in TBI mechanics. Strain magnitudes and the amount of axonal stretch near the bases of sulci were similar to those in other areas of the cortex, and local strain concentrations at the gray-white matter boundary were not observed. We tentatively conclude that observed differences in neuropathological response in these areas might be due to heterogeneity in the response to mechanical deformation rather than heterogeneity of the deformation itself.
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17

Apthorp, Deborah, D. Samuel Schwarzkopf, Christian Kaul, Bahador Bahrami, David Alais, and Geraint Rees. "Direct evidence for encoding of motion streaks in human visual cortex." Proceedings of the Royal Society B: Biological Sciences 280, no. 1752 (February 7, 2013): 20122339. http://dx.doi.org/10.1098/rspb.2012.2339.

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Temporal integration in the visual system causes fast-moving objects to generate static, oriented traces (‘motion streaks’), which could be used to help judge direction of motion. While human psychophysics and single-unit studies in non-human primates are consistent with this hypothesis, direct neural evidence from the human cortex is still lacking. First, we provide psychophysical evidence that faster and slower motions are processed by distinct neural mechanisms: faster motion raised human perceptual thresholds for static orientations parallel to the direction of motion, whereas slower motion raised thresholds for orthogonal orientations. We then used functional magnetic resonance imaging to measure brain activity while human observers viewed either fast (‘streaky’) or slow random dot stimuli moving in different directions, or corresponding static-oriented stimuli. We found that local spatial patterns of brain activity in early retinotopic visual cortex reliably distinguished between static orientations. Critically, a multivariate pattern classifier trained on brain activity evoked by these static stimuli could then successfully distinguish the direction of fast (‘streaky’) but not slow motion. Thus, signals encoding static-oriented streak information are present in human early visual cortex when viewing fast motion. These experiments show that motion streaks are present in the human visual system for faster motion.
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18

Gilaie-Dotan, Sharon, Ayse P. Saygin, Lauren J. Lorenzi, Ryan Egan, Geraint Rees, and Marlene Behrmann. "The role of human ventral visual cortex in motion perception." Brain 136, no. 9 (August 26, 2013): 2784–98. http://dx.doi.org/10.1093/brain/awt214.

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19

Culham, Jody. "Attention-Grabbing Motion in the Human Brain." Neuron 40, no. 3 (October 2003): 451–52. http://dx.doi.org/10.1016/s0896-6273(03)00689-5.

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20

Godenschweger, F., U. Kägebein, D. Stucht, U. Yarach, A. Sciarra, R. Yakupov, F. Lüsebrink, P. Schulze, and O. Speck. "Motion correction in MRI of the brain." Physics in Medicine and Biology 61, no. 5 (February 11, 2016): R32—R56. http://dx.doi.org/10.1088/0031-9155/61/5/r32.

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21

Braddick, Oliver J., Justin M. D. O'Brien, John Wattam-Bell, Janette Atkinson, Tom Hartley, and Robert Turner. "Brain Areas Sensitive to Coherent Visual Motion." Perception 30, no. 1 (January 2001): 61–72. http://dx.doi.org/10.1068/p3048.

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22

Soellinger, Michaela, Salome Ryf, Peter Boesiger, and Sebastian Kozerke. "Assessment of human brain motion using CSPAMM." Journal of Magnetic Resonance Imaging 25, no. 4 (2007): 709–14. http://dx.doi.org/10.1002/jmri.20882.

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23

Puce, Aina, and David Perrett. "Electrophysiology and brain imaging of biological motion." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 358, no. 1431 (February 19, 2003): 435–45. http://dx.doi.org/10.1098/rstb.2002.1221.

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The movements of the faces and bodies of other conspecifics provide stimuli of considerable interest to the social primate. Studies of single cells, field potential recordings and functional neuroimaging data indicate that specialized visual mechanisms exist in the superior temporal sulcus (STS) of both human and non–human primates that produce selective neural responses to moving natural images of faces and bodies. STS mechanisms also process simplified displays of biological motion involving point lights marking the limb articulations of animate bodies and geometrical shapes whose motion simulates purposeful behaviour. Facial movements such as deviations in eye gaze, important for gauging an individual's social attention, and mouth movements, indicative of potential utterances, generate particularly robust neural responses that differentiate between movement types. Collectively such visual processing can enable the decoding of complex social signals and through its outputs to limbic, frontal and parietal systems the STS may play a part in enabling appropriate affective responses and social behaviour.
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24

Sunaert, Stefan, Paul Van Hecke, G. Marchal, and G. A. Orban. "Motion-responsive regions of the human brain." Experimental Brain Research 127, no. 4 (August 4, 1999): 355–70. http://dx.doi.org/10.1007/s002210050804.

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25

Schenk, T. "Visual motion perception after brain damage: I. Deficits in global motion perception." Neuropsychologia 35, no. 9 (September 1997): 1289–97. http://dx.doi.org/10.1016/s0028-3932(97)00004-3.

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26

Thaler, Lore, Jennifer Milne, Stephen R. Arnott, and Melvyn A. Goodale. "Brain areas involved in echolocation motion processing in blind echolocation experts." Seeing and Perceiving 25 (2012): 140. http://dx.doi.org/10.1163/187847612x647720.

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People can echolocate their distal environment by making mouth-clicks and listening to the click-echoes. In previous work that used functional magnetic resonance imaging (fMRI) we have shown that the processing of echolocation motion increases activity in posterior/inferior temporal cortex (Thaler et al., 2011). In the current study we investigated, if brain areas that are sensitive to echolocation motion in blind echolocation experts correspond to visual motion area MT+. To this end we used fMRI to measure brain activity of two early blind echolocation experts while they listened to recordings of echolocation and auditory source sounds that could be either moving or stationary, and that could be located either to the left or to the right of the listener. A whole brain analysis revealed that echo motion and source motion activated different brain areas in posterior/inferior temporal cortex. Furthermore, the relative spatial arrangement of echo and source motion areas appeared to match the relative spatial arrangement of area MT+ and source motion areas that has been reported for sighted people (Saenz et al., 2008). Furthermore, we found that brain areas that were sensitive to echolocation motion showed a larger response to echo motion presented in contra-lateral space, a response pattern typical for visual motion processing in area MT+. In their entirety the data are consistent with the idea that brain areas that process echolocation motion in blind echolocation experts correspond to area MT+.
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27

Morland, A. B. "Visual perception of motion, luminance and colour in a human hemianope." Brain 122, no. 6 (June 1, 1999): 1183–98. http://dx.doi.org/10.1093/brain/122.6.1183.

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28

Saygin, A. P. "Superior temporal and premotor brain areas necessary for biological motion perception." Brain 130, no. 9 (September 1, 2007): 2452–61. http://dx.doi.org/10.1093/brain/awm162.

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29

Koldewyn, K., D. Whitney, and S. M. Rivera. "The psychophysics of visual motion and global form processing in autism." Brain 133, no. 2 (November 3, 2009): 599–610. http://dx.doi.org/10.1093/brain/awp272.

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30

Andrews, Timothy J., and Allison N. McCoy. "Can Illusory Motion Disrupt Tracking Real Motion?" Perception 26, no. 3 (March 1997): 269–75. http://dx.doi.org/10.1068/p260269.

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When rotating stripes or other periodic stimuli cross the retina at a critical rate, a reversal in the direction of motion of the stimuli is often seen. This illusion of motion perception was used to explore the roles of retinal and perceived motion in the generation of optokinetic nystagmus. Here we show that optokinetic nystagmus is disrupted during the perception of this illusion. Thus, when perceived and actual motion are in conflict, subjects fail to track the veridical movement. This observation suggests that the perception of motion can directly influence optokinetic nystagmus, even in the presence of a moving retinal image. A conflict in the neural representation of motion in different brain areas may explain these findings.
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31

Lloyd-Fox, Sarah, Anna Blasi, Nick Everdell, Clare E. Elwell, and Mark H. Johnson. "Selective Cortical Mapping of Biological Motion Processing in Young Infants." Journal of Cognitive Neuroscience 23, no. 9 (September 2011): 2521–32. http://dx.doi.org/10.1162/jocn.2010.21598.

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How specialized is the infant brain for perceiving the facial and manual movements displayed by others? Although there is evidence for a network of regions that process biological motion in adults—including individuated responses to the perception of differing facial and manual movements—how this cortical specialization develops remains unknown. We used functional near-infrared spectroscopy [Lloyd-Fox, S., Blasi, A., & Elwell, C. Illuminating the developing brain: The past, present and future of functional near-infrared spectroscopy. Neuroscience and Biobehavioral Reviews, 34, 269–284, 2010] to investigate the ability of 5-month-old infants to process differing biological movements. Infants watched videos of adult actors moving their hands, their mouth, or their eyes, all in contrast to nonbiological mechanical movements, while hemodynamic responses were recorded over the their frontal and temporal cortices. We observed different regions of the frontal and temporal cortex that responded to these biological movements and different patterns of cortical activation according to the type of movement watched. From an early age, our brains selectively respond to biologically relevant movements, and further, selective patterns of regional specification to different cues occur within what may correspond to a developing “social brain” network. These findings illuminate hitherto undocumented maps of selective cortical activation to biological motion processing in the early postnatal development of the human brain.
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32

Drummond, P. D. "Facial pain increases nausea and headache during motion sickness in migraine sufferers." Brain 127, no. 3 (November 7, 2003): 526–34. http://dx.doi.org/10.1093/brain/awh061.

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33

Beckers, G., and S. Zeki. "The consequences of inactivating areas V1 and V5 on visual motion perception." Brain 118, no. 1 (February 1995): 49–60. http://dx.doi.org/10.1093/brain/118.1.49.

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34

Buchel, C. "The functional anatomy of attention to visual motion. A functional MRI study." Brain 121, no. 7 (July 1, 1998): 1281–94. http://dx.doi.org/10.1093/brain/121.7.1281.

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35

Vaina, Lucia M., Alan Cowey, Marianna Jakab, and Ron Kikinis. "Deficits of motion integration and segregation in patients with unilateral extrastriate lesions." Brain 128, no. 9 (June 23, 2005): 2134–45. http://dx.doi.org/10.1093/brain/awh573.

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36

Pavlova, Marina, Christel Bidet-Ildei, Alexander N. Sokolov, Christoph Braun, and Ingeborg Krägeloh-Mann. "Neuromagnetic Response to Body Motion and Brain Connectivity." Journal of Cognitive Neuroscience 21, no. 5 (May 2009): 837–46. http://dx.doi.org/10.1162/jocn.2009.21050.

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Visual detection of body motion is of immense importance for daily-life activities and social nonverbal interaction. Although neurobiological mechanisms underlying visual processing of human locomotion are being explored extensively by brain imaging, the role of structural brain connectivity is not well understood. Here we investigate cortical evoked neuromagnetic response to point-light body motion in healthy adolescents and in patients with early periventricular lesions, periventricular leukomalacia (PVL), that disrupt brain connectivity. In a simultaneous masking paradigm, participants detected the presence of a point-light walker embedded in a few sets of spatially scrambled dots on the joints of a walker. The visual sensitivity to camouflaged human locomotion was lower in PVL patients. In accord with behavioral data, root-mean-square (RMS) amplitude of neuromagnetic trace in response to human locomotion was lower in PVL patients at latencies of 180–244 msec over the right temporal cortex. In this time window, the visual sensitivity to body motion in controls, but not in PVL patients, was inversely linked to the right temporal activation. At later latencies of 276–340 msec, we found reduction in RMS amplitude in PVL patients for body motion stimuli over the right frontal cortex. The findings indicate that disturbances in brain connectivity with the right temporal cortex, a key node of the social brain, and with the right frontal cortex lead to disintegration of the neural network engaged in visual processing of body motion. We suspect that reduced cortical response to body motion over the right temporal and frontal cortices might underlie deficits in visual social cognition.
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37

Luks, Tracy L., and Gregory V. Simpson. "Preparatory deployment of attention to motion activates higher-order motion-processing brain regions." NeuroImage 22, no. 4 (August 2004): 1515–22. http://dx.doi.org/10.1016/j.neuroimage.2004.04.008.

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38

Niedeggen, Michael, and Eugene R. Wist. "Motion evoked brain potentials parallel the consistency of coherent motion perception in humans." Neuroscience Letters 246, no. 2 (April 1998): 61–64. http://dx.doi.org/10.1016/s0304-3940(98)00222-5.

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39

Schenk, T. "Visual motion perception after brain damage: II. Deficits in form-from-motion perception." Neuropsychologia 35, no. 9 (September 1997): 1299–310. http://dx.doi.org/10.1016/s0028-3932(97)00005-5.

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40

Toschi, Nicola, Jieun Kim, Roberta Sclocco, Andrea Duggento, Riccardo Barbieri, Braden Kuo, and Vitaly Napadow. "Motion sickness increases functional connectivity between visual motion and nausea-associated brain regions." Autonomic Neuroscience 202 (January 2017): 108–13. http://dx.doi.org/10.1016/j.autneu.2016.10.003.

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41

Grossman, E., M. Donnelly, R. Price, D. Pickens, V. Morgan, G. Neighbor, and R. Blake. "Brain Areas Involved in Perception of Biological Motion." Journal of Cognitive Neuroscience 12, no. 5 (September 2000): 711–20. http://dx.doi.org/10.1162/089892900562417.

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These experiments use functional magnetic resonance imaging (fMRI) to reveal neural activity uniquely associated with perception of biological motion. We isolated brain areas activated during the viewing of point-light figures, then compared those areas to regions known to be involved in coherent-motion perception and kinetic-boundary perception. Coherent motion activated a region matching previous reports of human MT/MST complex located on the temporo-parieto-occipital junction. Kinetic boundaries activated a region posterior and adjacent to human MT previously identified as the kinetic-occipital (KO) region or the lateral-occipital (LO) complex. The pattern of activation during viewing of biological motion was located within a small region on the ventral bank of the occipital extent of the superior-temporal sulcus (STS). This region is located lateral and anterior to human MT/MST, and anterior to KO. Among our observers, we localized this region more frequently in the right hemisphere than in the left. This was true regardless of whether the point-light figures were presented in the right or left hemifield. A small region in the medial cerebellum was also active when observers viewed biological-motion sequences. Consistent with earlier neuroimaging and single-unit studies, this pattern of results points to the existence of neural mechanisms specialized for analysis of the kinematics defining biological motion.
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42

Enzmann, D. R., and N. J. Pelc. "Brain motion: measurement with phase-contrast MR imaging." Radiology 185, no. 3 (December 1992): 653–60. http://dx.doi.org/10.1148/radiology.185.3.1438741.

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43

Kim, C. Y., and R. Blake. "Brain activity reflects implied motion in abstract paintings." Journal of Vision 7, no. 9 (March 23, 2010): 781. http://dx.doi.org/10.1167/7.9.781.

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44

Kourtzi, Zoe, Bart Krekelberg, and Richard J. A. van Wezel. "Linking form and motion in the primate brain." Trends in Cognitive Sciences 12, no. 6 (June 2008): 230–36. http://dx.doi.org/10.1016/j.tics.2008.02.013.

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45

Alonso, Jose Manuel. "Motion processing picks up speed in the brain." Nature 558, no. 7708 (May 23, 2018): 38–39. http://dx.doi.org/10.1038/d41586-018-04289-9.

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46

Schultz, J., M. Brockhaus, H. H. Bulthoff, and K. S. Pilz. "What the Human Brain Likes About Facial Motion." Cerebral Cortex 23, no. 5 (April 24, 2012): 1167–78. http://dx.doi.org/10.1093/cercor/bhs106.

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47

Zeng, L. L., D. Wang, M. D. Fox, M. Sabuncu, D. Hu, M. Ge, R. L. Buckner, and H. Liu. "Neurobiological basis of head motion in brain imaging." Proceedings of the National Academy of Sciences 111, no. 16 (April 7, 2014): 6058–62. http://dx.doi.org/10.1073/pnas.1317424111.

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48

Maclaren, Julian, Michael Herbst, Oliver Speck, and Maxim Zaitsev. "Prospective motion correction in brain imaging: A review." Magnetic Resonance in Medicine 69, no. 3 (May 8, 2012): 621–36. http://dx.doi.org/10.1002/mrm.24314.

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49

Poirier, Colline, Simon Baumann, Pradeep Dheerendra, Olivier Joly, David Hunter, Fabien Balezeau, Li Sun, et al. "Auditory motion-specific mechanisms in the primate brain." PLOS Biology 15, no. 5 (May 4, 2017): e2001379. http://dx.doi.org/10.1371/journal.pbio.2001379.

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50

Gilbert, Cole. "Brain Connectivity: Revealing the Fly Visual Motion Circuit." Current Biology 23, no. 18 (September 2013): R851—R853. http://dx.doi.org/10.1016/j.cub.2013.08.018.

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