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Relevant bibliographies by topics / Brain Myocardium Cell physiology

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Academic literature on the topic 'Brain Myocardium Cell physiology'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

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Journal articles on the topic "Brain Myocardium Cell physiology"

1

Ding, XiaoHui, Jeffrey L. Ardell, Fang Hua, et al. "Modulation of cardiac ischemia-sensitive afferent neuron signaling by preemptive C2 spinal cord stimulation: effect on substance P release from rat spinal cord." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 294, no. 1 (2008): R93—R101. http://dx.doi.org/10.1152/ajpregu.00544.2007.

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The upper cervical spinal region functions as an intraspinal controller of thoracic spinal reflexes and contributes to neuronal regulation of the ischemic myocardium. Our objective was to determine whether stimulation of the C2 cervical spinal cord (SCS) of rats modified the input signal at the thoracic spinal cord when cardiac ischemia-sensitive (sympathetic) afferents were activated by transient occlusion of the left anterior descending coronary artery (CoAO). Changes in c-Fos expression were used as an index of neuronal activation within the spinal cord and brain stem. The pattern of substa

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Oka, Naoki, Kuniya Asai, Raymond K. Kudej та ін. "Downregulation of caveolin by chronic β-adrenergic receptor stimulation in mice". American Journal of Physiology-Cell Physiology 273, № 6 (1997): C1957—C1962. http://dx.doi.org/10.1152/ajpcell.1997.273.6.c1957.

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Caveolae, flask-shaped invaginations of cell membranes, are believed to play pivotal roles in transmembrane transportation of molecules and cellular signaling. Caveolin, a structural component of caveolae, interacts directly with G proteins and regulates their function. We investigated the effect of chronic β-adrenergic receptor stimulation on the expression of caveolin subtypes in mouse hearts by immunoblotting and Northern blotting. Caveolin-1 and -3 were abundantly expressed in the heart and skeletal muscles, but not in the brain. Continuous (−)-isoproterenol, but not (+)-isoproterenol, inf

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Tedoldi, Angelo, Liam Argent, and Johanna M. Montgomery. "The role of the tripartite synapse in the heart: how glial cells may contribute to the physiology and pathophysiology of the intracardiac nervous system." American Journal of Physiology-Cell Physiology 320, no. 1 (2021): C1—C14. http://dx.doi.org/10.1152/ajpcell.00363.2020.

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One of the major roles of the intracardiac nervous system (ICNS) is to act as the final site of signal integration for efferent information destined for the myocardium to enable local control of heart rate and rhythm. Multiple subtypes of neurons exist in the ICNS where they are organized into clusters termed ganglionated plexi (GP). The majority of cells in the ICNS are actually glial cells; however, despite this, ICNS glial cells have received little attention to date. In the central nervous system, where glial cell function has been widely studied, glia are no longer viewed simply as suppor

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Osman, Abdel‐Hamid K., Naoto Minamino, and Hany E. Marei. "Identification and mapping of brain natriuretic peptide in the normal ventricular myocardium of a desert‐dwelling mammalian model, the camel ( Camelus dromedarius ): Immunohistochemical and ultrastructural study." Journal of Cellular Physiology 234, no. 3 (2018): 3067–77. http://dx.doi.org/10.1002/jcp.27126.

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Lin, Kuan-Ho, Wei-Wen Kuo, Ai-Zhi Jiang та ін. "Tetramethylpyrazine Ameliorated Hypoxia-Induced Myocardial Cell Apoptosis via HIF-1α/JNK/p38 and IGFBP3/BNIP3 Inhibition to Upregulate PI3K/Akt Survival Signaling". Cellular Physiology and Biochemistry 36, № 1 (2015): 334–44. http://dx.doi.org/10.1159/000374076.

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Background: Hemorrhagic shock (HS) is the major cause of death from trauma. Hemorrhagic shock may lead to cellular hypoxia and organ damage. Our previous findings showed that HS induced a cardiac apoptosis pathway and synergistically caused myocardial cell damage in diabetic rats under trauma-induced HS. Tetramethylpyrazine (TMP) is a major biologically active ingredient purified from the rhizome of Ligusticum wallichii (called Chuang Xiong in Chinese). Chuan Xiong rescued cells from synergistic cardiomyoblast cell injury under high-glucose (HG) conditions plus hypoxia. TMP is one of the most

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Oliveira, Fernando A., Silvia Guatimosim, Carlos H. Castro, et al. "Abolition of reperfusion-induced arrhythmias in hearts from thiamine-deficient rats." American Journal of Physiology-Heart and Circulatory Physiology 293, no. 1 (2007): H394—H401. http://dx.doi.org/10.1152/ajpheart.00833.2006.

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Extensive work has been done regarding the impact of thiamine deprivation on the nervous system. In cardiac tissue, chronic thiamine deficiency is described to cause changes in the myocardium that can be associated with arrhythmias. However, compared with the brain, very little is known about the effects of thiamine deficiency on the heart. Thus this study was undertaken to explore whether thiamine deprivation has a role in cardiac arrhythmogenesis. We examined hearts isolated from thiamine-deprived and control rats. We measured heart rate, diastolic and systolic tension, and contraction and r

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Niss, Omar, Michael D. Taylor, Robert Fleck, et al. "Diffuse Myocardial Fibrosis Is a Common Feature of Sickle Cell Anemia That Is Associated with Diastolic Dysfunction and Restrictive Cardiac Physiology." Blood 128, no. 22 (2016): 8. http://dx.doi.org/10.1182/blood.v128.22.8.8.

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Abstract Background: We have recently shown that the cardiomyopathy of sickle cell anemia (SCA) is characterized by restrictive physiology (diastolic dysfunction, left atrial [LA] enlargement and normal systolic function) superimposed on hyperdynamic features (left ventricular [LV] enlargement and eccentric hypertrophy) (JACC Cardiovasc Imaging 9:244-253;2016; PNAS 2016 in press). Similar to other restrictive cardiomyopathies, SCA-related cardiomyopathy may lead to mild, secondary pulmonary hypertension (PH) with elevated tricuspid regurgitant jet velocity (TRV), and can be complicated by arrh

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Cheng, Mian, Gang Wu, Yue Song, et al. "Celastrol-Induced Suppression of the MiR-21/ERK Signalling Pathway Attenuates Cardiac Fibrosis and Dysfunction." Cellular Physiology and Biochemistry 38, no. 5 (2016): 1928–38. http://dx.doi.org/10.1159/000445554.

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Backgroud: Myocardial fibrosis results in myocardial remodelling and dysfunction. Celastrol, a traditional oriental medicine, has been suggested to have cardioprotective effects. However, its underlying mechanism is unknown. This study investigated the ability of celastrol to prevent cardiac fibrosis and dysfunction and explored the underlying mechanisms. Methods: Animal and cell models of cardiac fibrosis were used in this study. Myocardial fibrosis was induced by transverse aortic constriction (TAC) in mice. Cardiac hypertrophy and fibrosis were evaluated based on histological and biochemica

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Asahara, Takayuki, and Atsuhiko Kawamoto. "Endothelial progenitor cells for postnatal vasculogenesis." American Journal of Physiology-Cell Physiology 287, no. 3 (2004): C572—C579. http://dx.doi.org/10.1152/ajpcell.00330.2003.

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In the past decade, researchers have defined committed stem or progenitor cells from various tissues, including bone marrow, peripheral blood, brain, liver, and reproductive organs, in both adult animals and humans. Whereas most cells in adult organs are composed of differentiated cells, which express a variety of specific phenotypic genes adapted to each organ's environment, quiescent stem or progenitor cells are maintained locally or in the systemic circulation and are activated by environmental stimuli for physiological and pathological tissue regeneration. Recently, endothelial progenitor

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Mizuguchi, Yasunori, Jie Chen, Surya V. Seshan, Dix P. Poppas, Hazel H. Szeto, and Diane Felsen. "A novel cell-permeable antioxidant peptide decreases renal tubular apoptosis and damage in unilateral ureteral obstruction." American Journal of Physiology-Renal Physiology 295, no. 5 (2008): F1545—F1553. http://dx.doi.org/10.1152/ajprenal.00395.2007.

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Unilateral ureteral obstruction (UUO) is characterized by decreases in renal function, increased interstitial fibrosis, tubular apoptosis, and cellular infiltration. It has been suggested that inhibition of tubular apoptosis may protect against renal damage in obstruction. We have recently developed a series of peptides which are concentrated in the inner mitochondrial membrane and prevent cell death. These peptides are also active in vivo, in myocardial infraction, ischemic brain injury, and amyotrophic lateral sclerosis models. We therefore used SS-31, a prototype of these peptides, and asse

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Dissertations / Theses on the topic "Brain Myocardium Cell physiology"

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Bhardwaj, Ratan D. "Cytoarcheology: understanding cellular turnover in the human brain and heart /." Stockholm, 2007. http://diss.kib.ki.se/2007/978-91-7357-130-2/.

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Lu, Chen. "Novel Therapeutic Approaches for Ischemic Heart and Brain Injury: Modulation of Toll-Like Receptor-Mediated Signaling Pathways and PI3K/Akt Signaling." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etd/2343.

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Innate immune and inflammatory responses contribute to myocardial and cerebral ischemia/reperfusion (I/R) injury. Toll-like receptors (TLRs) play a critical role in the induction of innate immune and inflammatory responses via activation of nuclear factor kappa B (NF-κB). We have shown that activation of NF-κB contributes to myocardial and cerebral I/R injury. Indeed, inhibition of TLR4-mediated NF-κB activation significantly decreased myocardial and cerebral I/R injury via activation of PI3K/Akt signaling. PI3K/Akt signaling is an important pathway in regulating cellular survival and inflamma

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Hasanally, Devin. "Bioactive oxidized phosphatidylcholines cause apoptotic cell death in cardiomyocytes during ischemia reperfusion." Springer-Verlag New York, 2014. http://hdl.handle.net/1993/30363.

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The main treatment for myocardial infarction is early reperfusion of ischemic tissue. Ischemia and reperfusion (IR) produces reactive oxygen species that oxidize membrane phospholipids. The production of oxidized lipids and their role on cell death in cardiac IR injury is unknown. Using in vitro model of IR, our goal was to identify oxidized phosphatidylcholines (OxPC) from cardiomyocytes, to determine their bioactivity on cardiomyocyte viability and mitochondrial permeability, and using an OxPC specific EO6 antibody inhibit OxPC activity on cardiomyocytes. Rat cardiomyocytes were exposed to

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Davis, Brandon James. "VEGF signaling mechanisms in increased blood brain barrier permeability following hypoxia." Diss., Search in ProQuest Dissertations & Theses. UC Only, 2007. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3261273.

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Houalla, Tarek. "Isolation of microglia from goldfish brain." Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31238.

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This study aims at providing a new technique for the isolation and culture of goldfish microglial cells. So far no protocol has been designed for the growth of these cells in vitro, despite the growing interest in the remarkable capacity of goldfish central nervous system (CNS) for regenerating severed axons. This newly developed technique has little or no similarity to those used in the isolation of mammalian microglia, and is distinguished by its simple setup and its fast yield for microglial cells. In addition, a virtually pure population of microglia was generated when plated on untreated

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Cohen, Ricky Israel. "Rat brain oligodendrocytes express muscarinic and adrenergic receptors." Thesis, McGill University, 1996. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=42006.

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The aim of the studies underlying this thesis was to characterize the muscarinic and adrenergic receptors expressed in rat brain oligodendrocytes' (OLs); determine if ligand binding alters second messenger levels classically associated with these families of receptors, such as inositol phosphates (InsP), intracellular calcium ( (Ca$ rm sp{2+} rbrack sb{i}),$ and cyclic AMP (cAMP); and the role of neurotransmitters, acetylcholine (Ach) or norepinephrine (NE) on oligodendrocyte growth.<br>CCH (carbachol), a stable Ach analog, caused a concentration and time dependent increase in the accumulation

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Folsom, Ryan J. "Rapid Adaptation of Dopamine D2 Receptor Responses in the Brain and Blood Following Acute Ethanol." BYU ScholarsArchive, 2014. https://scholarsarchive.byu.edu/etd/5248.

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Dopamine (DA) D2 receptor expression parallels DA levels in the brain and these autoreceptors have been shown to be modulated by long-term ethanol exposure. We have previously demonstrated that ventral tegmental area (VTA) GABA neurons also express D2 autoreceptors (D2R), and that DA and D2R agonists markedly enhance the excitability of VTA GABA neurons, opposite to their well-known auto-receptor inhibition of DA neurons. Most importantly, D2R antagonists block ethanol inhibition of VTA GABA neurons and D2R expression in VTA GABA neurons down-regulates with chronic ethanol, as others have sho

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Yau, Suk-yu, and 邱淑瑜. "Neuroprotective effects of physical exercise on stressed brain : its relationship to hippocampal neurogenesis and dendritic remodeling." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hdl.handle.net/10722/210329.

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Yau, Suk-yu. "Neuroprotective effects of physical exercise on stressed brain : its relationship to hippocampal neurogenesis and dendritic remodeling /." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B4322376X.

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Finuf, Christopher Scott. "Third Ventricle Width as a Metric for Fast and Efficient Detection of Atrophy in Traumatic Brain Injury." BYU ScholarsArchive, 2015. https://scholarsarchive.byu.edu/etd/5681.

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In an average year more than 1.7 million people will experience a traumatic brain injury (TBI) in the United States. It is known that atrophy occurs across a spectrum for TBI patients, ranging from mild to severe. Current conventional magnetic resonance imaging (MRI) methods are inconsistent in detecting this atrophy on the milder end of the spectrum. Also more contemporary imaging tools, although efficient, are too time consuming for clinical applicability. It is for these reasons that a quick and efficient measurement for detecting this atrophy is needed by clinicians. The measuring of third

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Books on the topic "Brain Myocardium Cell physiology"

1

Kao, Race L. Cellular cardiomyoplasty: Myocardial repair with cell implantation. Chapman & Hall, 1997.

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Media, Springer Science+Business, ed. Cellular cardiomyoplasty: Methods and protocols. Humana Press, 2013.

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How brain-like is the spinal cord?: Interacting cell assemblies in the nervous system. Springer-Verlag, 1988.

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Stress, the aging brain, and the mechanisms of neuron death. MIT Press, 1992.

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Visual population codes: Toward a common multivariate framework for cell recording and functional imaging. MIT Press, 2011.

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Thounthong, Phatiphat. A PEM fuel cell power source for electric vehicle applications. Nova Science, 2008.

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International Symposium on Cell Function and Disease (1988 Monterrey, Mexico). Cell function and disease. Plenum Press, 1988.

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Regenerating the heart: Stem cells and the cardiovascular system. Humana Press/Springer, 2011.

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Tolias, Christos M. Extracellular superoxide and nitric oxide: A real-time investigation in the role of free radicals in brain cell physiology and pathophysiology in vitro. typescript, 1999.

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Kempermann, Gerd. Adult neurogenesis: Stem cells and neuronal development in the adult brain. Oxford University Press, 2006.

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Book chapters on the topic "Brain Myocardium Cell physiology"

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Walaas, S. I., and A. C. Nairn. "Calcium-Regulated Protein Phosphorylation in Mammalian Brain." In Calcium and Cell Physiology. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70070-5_12.

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Munro, P. M. G., L. A. McLaughlin-Borlace, G. Pryce, N. L. Occleston, and J. Greenwood. "T-Cell Line Interactions with Brain Endothelial Monolayers." In Biology and Physiology of the Blood-Brain Barrier. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_45.

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Joó, Ferenc. "Significance of the Second Messenger Molecules in the Regulation of Cerebral Endothelial Cell Functions." In Biology and Physiology of the Blood-Brain Barrier. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_35.

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Dore-Duffy, Paula, Roumen Balabanov, and Ruth Washington. "Recovery from Acute Experimental Autoimmune Encephalomyelitis (EAE) Characterized by Endothelial Cell Unresponsiveness to Cytokines and Pericyte Activation." In Biology and Physiology of the Blood-Brain Barrier. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_57.

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Stanimirovic, Danica B., Rita Ball, Josée Wong, and Jon P. Durkin. "The Role of Protein Kinase C and Marcks Protein Phosphorylation in Rat Cerebromicrovascular Endothelial Cell Proliferation Induced by Astrocyte-Derived Factors." In Biology and Physiology of the Blood-Brain Barrier. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_36.

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Ahlemeyer, Barbara, Sabine Matys, and Peter Brust. "The Effect of Conditioned Medium of Porcine Astrocytes and Different Cell Lines on the Activity of the Marker Enzymes Alp and γ-GT in Endothelial Cells of Porcine Brain Microvessels." In Biology and Physiology of the Blood-Brain Barrier. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_38.

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Thornton, Claire, and Henrik Hagberg. "Mechanisms of Cell Death in the Developing Brain." In Fetal and Neonatal Physiology. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-323-35214-7.00007-x.

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Faber, D. S., and A. E. Pereda. "BRAIN AND NERVOUS SYSTEM | Physiology of the Mauthner Cell: Function." In Encyclopedia of Fish Physiology. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-374553-8.00280-x.

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Pereda, A. E., and D. S. Faber. "BRAIN AND NERVOUS SYSTEM | Physiology of the Mauthner Cell: Discovery and Properties." In Encyclopedia of Fish Physiology. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-374553-8.00007-1.

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Shpakov, Alexander Olegovich. "The Contribution of Changes in Adenylyl Cyclase Signaling System of the Brain and Myocardium to Etiology and Pathogenesis of Diabetes Mellitus." In Evolutionary Physiology and Biochemistry - Advances and Perspectives. InTech, 2018. http://dx.doi.org/10.5772/intechopen.73661.

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Conference papers on the topic "Brain Myocardium Cell physiology"

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Lindhout, Ivan, and Andis Klegeris. "Neurotrophins as intercellular signaling molecules of the brain regulate select immune functions of microglia." In Cell-to-Cell Metabolic Cross-Talk in Physiology and Pathology. MDPI, 2020. http://dx.doi.org/10.3390/cells2020-08927.

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Prudnikov, Igor, Anton Smirnov, and Volodymyr Tsyvkin. "The actin cytoskeleton is a key element of the apoptosome assembly in the developing brain." In Cell-to-Cell Metabolic Cross-Talk in Physiology and Pathology. MDPI, 2020. http://dx.doi.org/10.3390/cells2020-08923.

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"Course | School of Brain Cells & Circuits “Camillo Golgi”: From cell physiology to integrated signals and emerging brain functions." In Course | School of Brain Cells & Circuits “Camillo Golgi”: From cell physiology to integrated signals and emerging brain functions. Frontiers Media SA, 2019. http://dx.doi.org/10.3389/978-2-88945-584-3.

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