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Journal articles on the topic 'Brain paralysis'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 February 2022

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1

MCSHANE, M. A., S. BOYD, B. HARDING, E. M. BRETT, and J. WILSON. "PROGRESSIVE BULBAR PARALYSIS OF CHILDHOOD." Brain 115, no. 6 (1992): 1889–900. http://dx.doi.org/10.1093/brain/115.6.1889.

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2

Yunita, Niken, Ocie Harum Wulan, Hastari Wuryastuty, and Raden Wasito. "Immunopathological Approach for Avian Influenza Virus Detection in Brain of Laying Bird with Clinical Signs of Torticollis and Curled Toe Paralysis." KnE Life Sciences 3, no. 6 (2017): 476. http://dx.doi.org/10.18502/kls.v3i6.1157.

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Infection with avian influenza virus (AIV) in laying birds field cases often lead to clinical signs of torticollis and curled toe paralysis. Clinical signs of torticollis and curled toe paralysis is similar to the clinical signs of Newcastle disease virus infection in poultry, making it difficult for the confirmation of diagnosis of infection with both viruses. Immunopathological immunohistochemistry streptavidin-biotin (IHC SB) is an antibody-based test to detect the presence of pathogens, especially AIV in poultry with the principle of antigen detection in tissue specimens. This research aim

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3

Birbaumer, Niels, Ander Ramos Murguialday, and Leonardo Cohen. "Brain–computer interface in paralysis." Current Opinion in Neurology 21, no. 6 (2008): 634–38. http://dx.doi.org/10.1097/wco.0b013e328315ee2d.

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4

Crotty, Shane, Laura Hix, Luis J. Sigal, and Raul Andino. "Poliovirus pathogenesis in a new poliovirus receptor transgenic mouse model: age-dependent paralysis and a mucosal route of infection." Journal of General Virology 83, no. 7 (2002): 1707–20. http://dx.doi.org/10.1099/0022-1317-83-7-1707.

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We constructed a poliovirus receptor (PVR) transgenic mouse line carrying a PVRδ cDNA driven by a β-actin promoter. We refer to this model as the cPVR mouse. The cPVR mice express Pvr in a variety of tissues (including small intestines, brain, spinal cord, muscle, blood and liver) and are susceptible to infection after intraperitoneal, intracerebral or intramuscular inoculation of poliovirus. After intraperitoneal inoculation, poliovirus replication is observed in cPVR muscle, brain, spinal cord and, notably, small intestine. The cPVR mice exhibit a striking age-dependent paralysis after intra

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5

Vafaee-Shahi, Mohammad, Roghayeh Saeedi, Neda Pak, Aina Riahi, and Saeide Ghasemi. "Snake Eye Appearance; A Rare Radiology Presentation in Acute Flaccid Paralysis: A Case Report." Open Neurology Journal 15, no. 1 (2021): 43–47. http://dx.doi.org/10.2174/1874205x02115010043.

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Background: Acute flaccid paralysis (AFP) is defined by the acute onset of weakness or paralysis with reduced muscle tone in children. There are many non-infectious and infectious causes. Snake eye appearance (SEA) is a rare radiologic appearance and helps narrow down differential diagnoses in flaccid paralysis. Case Presentation: Here, we reported a 6 months-old girl who was admitted with sudden onset flaccid paralysis. She was lethargic and ill without any detectable deep tendon reflexes. She had a high fever that had started 3 days earlier with malaise, poor feeding and coryza. The first ch

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6

Grattan-Smith, P. "Clinical and neurophysiological features of tick paralysis." Brain 120, no. 11 (1997): 1975–87. http://dx.doi.org/10.1093/brain/120.11.1975.

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7

Chaudhary, U., N. Birbaumer, and M. R. Curado. "Brain-Machine Interface (BMI) in paralysis." Annals of Physical and Rehabilitation Medicine 58, no. 1 (2015): 9–13. http://dx.doi.org/10.1016/j.rehab.2014.11.002.

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8

YAZAWA, Yoshiro, Kaoru UCHIDA, Hiroshi TANAKA, Shuzo INOUE, and Takahiro ISOZUMI. "Facial Paralysis due to Brain Tumor." Practica Oto-Rhino-Laryngologica 87, no. 4 (1994): 455–61. http://dx.doi.org/10.5631/jibirin.87.455.

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9

Hochberg, Leigh R. "4 Brain computer interface for paralysis." Journal of Neurology, Neurosurgery & Psychiatry 92, no. 8 (2021): A1.4—A2. http://dx.doi.org/10.1136/jnnp-2021-bnpa.4.

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Intracortically-based Brain-Computer Interfaces (iBCIs) are poised to revolutionize our ability to restore lost neurologic functions. By recording high resolution neural activity from the brain, the intention to move ones hand can be detected and decoded in real- time, potentially providing people with motor neuron disease (ALS), stroke, or spinal cord injury with restored or maintained ability to control communication devices, assistive technologies, and their own limbs. iBCIs also are central to the development of closed-loop neuromodulation systems, with great potential to serve people with

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10

LINKS, THERA P., MACHIEL J. ZWARTS, JAN T. WILMINK, WILLEMINA M. MOLENAAR, and HANS J. G. H. OOSTERHUIS. "PERMANENT MUSCLE WEAKNESS IN FAMILIAL HYPOKALAEMIC PERIODIC PARALYSIS." Brain 113, no. 6 (1990): 1873–89. http://dx.doi.org/10.1093/brain/113.6.1873.

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11

Birbaumer, Niels, Guillermo Gallegos-Ayala, Moritz Wildgruber, Stefano Silvoni, and Surjo R. Soekadar. "Direct Brain Control and Communication in Paralysis." Brain Topography 27, no. 1 (2013): 4–11. http://dx.doi.org/10.1007/s10548-013-0282-1.

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12

Coghlan, Andy. "Brain-controlled arm could help overcome paralysis." New Scientist 214, no. 2865 (2012): 10. http://dx.doi.org/10.1016/s0262-4079(12)61265-2.

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13

Servick, Kelly. "Brain signals ‘speak’ for person with paralysis." Science 373, no. 6552 (2021): 263. http://dx.doi.org/10.1126/science.373.6552.263.

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14

Kheirkhah, Mina, Stefan Brodoehl, Lutz Leistritz, et al. "Abnormal Emotional Processing and Emotional Experience in Patients with Peripheral Facial Nerve Paralysis: An MEG Study." Brain Sciences 10, no. 3 (2020): 147. http://dx.doi.org/10.3390/brainsci10030147.

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Abnormal emotional reactions of the brain in patients with facial nerve paralysis have not yet been reported. This study aims to investigate this issue by applying a machine-learning algorithm that discriminates brain emotional activities that belong either to patients with facial nerve paralysis or to healthy controls. Beyond this, we assess an emotion rating task to determine whether there are differences in their experience of emotions. MEG signals of 17 healthy controls and 16 patients with facial nerve paralysis were recorded in response to picture stimuli in three different emotional cat

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15

Murakami, Shingo, Naohito Hato, Takashi Doi, Mutsuhiko Mizobuchi, and Naoaki Yanagihara. "Role of Herpes Simplex Virus Infection in the Pathogenesis of Facial Paralysis in Mice." Annals of Otology, Rhinology & Laryngology 105, no. 1 (1996): 49–53. http://dx.doi.org/10.1177/000348949610500108.

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To clarify the role and site of herpes simplex virus (HSV) infection in the pathogenesis of facial paralysis, we examined the viral genome by the polymerase chain reaction and the neutralization antibody titer using microplates in an animal model. Following inoculation with HSV type 1 of the KOS strain into mouse auricles, HSV DNA appeared in the ipsilateral facial nerve on the 3rd day, and in bilateral facial nerves and the brain stem on the 10th day only in animals with facial paralysis. In animals without facial paralysis, no HSV DNA was detected in these tissues. The neutralization antibod

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16

Abbasi, Jennifer. "Advanced Brain-Computer Interface for People With Paralysis." JAMA 321, no. 6 (2019): 537. http://dx.doi.org/10.1001/jama.2019.0294.

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17

Mantegazza, Massimo, Sandrine Cestèle, and William A. Catterall. "Sodium channelopathies of skeletal muscle and brain." Physiological Reviews 101, no. 4 (2021): 1633–89. http://dx.doi.org/10.1152/physrev.00025.2020.

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Voltage-gated sodium channels initiate action potentials in nerve, skeletal muscle, and other electrically excitable cells. Mutations in them cause a wide range of diseases. These channelopathy mutations affect every aspect of sodium channel function, including voltage sensing, voltage-dependent activation, ion conductance, fast and slow inactivation, and both biosynthesis and assembly. Mutations that cause different forms of periodic paralysis in skeletal muscle were discovered first and have provided a template for understanding structure, function, and pathophysiology at the molecular level

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18

de Almeida Ribeiro, Paulo Rogério, Fabricio Lima Brasil, Matthias Witkowski, et al. "Controlling Assistive Machines in Paralysis Using Brain Waves and Other Biosignals." Advances in Human-Computer Interaction 2013 (2013): 1–9. http://dx.doi.org/10.1155/2013/369425.

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The extent to which humans can interact with machines significantly enhanced through inclusion of speech, gestures, and eye movements. However, these communication channels depend on a functional motor system. As many people suffer from severe damage of the motor system resulting in paralysis and inability to communicate, the development of brain-machine interfaces (BMI) that translate electric or metabolic brain activity into control signals of external devices promises to overcome this dependence. People with complete paralysis can learn to use their brain waves to control prosthetic devices

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19

Groome, James R., Frank Lehmann-Horn, Chunxiang Fan, et al. "NaV1.4 mutations cause hypokalaemic periodic paralysis by disrupting IIIS4 movement during recovery." Brain 137, no. 4 (2014): 998–1008. http://dx.doi.org/10.1093/brain/awu015.

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20

Sampedro Castañeda, Marisol, Edmar Zanoteli, Renata S. Scalco, et al. "A novelATP1A2mutation in a patient with hypokalaemic periodic paralysis and CNS symptoms." Brain 141, no. 12 (2018): 3308–18. http://dx.doi.org/10.1093/brain/awy283.

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21

Prasarnpun, S., J. Walsh, S. S. Awad, and J. B. Harris. "Envenoming bites by kraits: the biological basis of treatment-resistant neuromuscular paralysis." Brain 128, no. 12 (2005): 2987–96. http://dx.doi.org/10.1093/brain/awh642.

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22

Et.al, Chang-yong Joo. "Collaborative Maze Game Using EEG for Paralyzed Patients." Turkish Journal of Computer and Mathematics Education (TURCOMAT) 12, no. 6 (2021): 849–55. http://dx.doi.org/10.17762/turcomat.v12i6.2108.

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The brain movement of a general paralysis patient is normal even if the nerve cells that connect the brain and muscles are damaged and the body movement is hindered. When these patients live alone and cannot perform daily activities, they lose the motivation to live, their brains become dull, and their self-esteem degrades. Therefore, a method should be developed such that these patients can perform brain exercises on their own. The development of a maze-finding game based on mutual cooperation using electroencephalography (EEG) for patients with general paralysis is described herein. Paralyze

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23

Fan, Chunxiang, Frank Lehmann-Horn, Marc-André Weber, et al. "Transient compartment-like syndrome and normokalaemic periodic paralysis due to a Cav1.1 mutation." Brain 136, no. 12 (2013): 3775–86. http://dx.doi.org/10.1093/brain/awt300.

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24

Angeli, Claudia A., V. Reggie Edgerton, Yury P. Gerasimenko, and Susan J. Harkema. "Altering spinal cord excitability enables voluntary movements after chronic complete paralysis in humans." Brain 137, no. 5 (2014): 1394–409. http://dx.doi.org/10.1093/brain/awu038.

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25

Schottmann, Gudrun, Christiane Wagner, Franziska Seifert, Werner Stenzel, and Markus Schuelke. "MORC2 mutation causes severe spinal muscular atrophy-phenotype, cerebellar atrophy, and diaphragmatic paralysis." Brain 139, no. 12 (2016): e70-e70. http://dx.doi.org/10.1093/brain/aww252.

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26

Thakur, JS, Naina Verma, Shobha Mohindroo, Ramesh K. Azad, and NK Mohindroo. "Isolated tubercular hypoglossal nerve paralysis." Tropical Doctor 47, no. 3 (2016): 255–60. http://dx.doi.org/10.1177/0049475516649894.

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Hypoglossal nerve palsy is not an uncommon neurological finding but primary nasopharyngeal tuberculosis (TB) presenting as hypoglossal nerve palsy is very rare. A 31-year-old woman presented with headache and progressive tongue deviation towards the right side. Diagnostic nasal endoscopy revealed soft tissue mass lesion on the posterior wall of nasopharynx while MRI revealed isointense tumour in nasopharynx with normal hypoglossal nerve and brain. Histopathological examination found TB. We discuss the clinical challenges and possible pathogenesis of this rare clinical entity.

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27

Manivannan, Sivabalan, Victoria K. Baxter, Kimberly L. W. Schultz, Barbara S. Slusher, and Diane E. Griffin. "Protective Effects of Glutamine Antagonist 6-Diazo-5-Oxo-l-Norleucine in Mice with Alphavirus Encephalomyelitis." Journal of Virology 90, no. 20 (2016): 9251–62. http://dx.doi.org/10.1128/jvi.01045-16.

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ABSTRACTInflammation is a necessary part of the response to infection but can also cause neuronal injury in both infectious and autoimmune diseases of the central nervous system (CNS). A neurovirulent strain of Sindbis virus (NSV) causes fatal paralysis in adult C57BL/6 mice during clearance of infectious virus from the CNS, and the virus-specific immune response is implicated as a mediator of neuronal damage. Previous studies have shown that survival is improved in T-cell-deficient mice and in mice with pharmacological inhibition of the inflammatory response and glutamate excitotoxicity. Beca

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28

Galiautdinov, Rinat. "Brain Machine Interface." International Journal of Applied Research in Bioinformatics 10, no. 1 (2020): 26–36. http://dx.doi.org/10.4018/ijarb.2020010102.

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The main purpose of the article is to provide the solution which allows the muscles to work in a situation when neural connection is corrupted either due to illness or injury, which usually causes paralysis. The research is on the interpretation of the brain signals based on the analysis of neurotransmitters and the transformation of this analysis into the electric signals effecting on the muscle in the situation when neural circuit between a sensor/inter neuron and a motor neuron is broken. This method would allow paralyzed people to move their limbs and potentially to walk.

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29

Blazer, S., J. A. Hemli, P. O. Sujov, and J. Braun. "Neonatal bilateral diaphragmatic paralysis caused by brain stem haemorrhage." Archives of Disease in Childhood 64, no. 1 Spec No (1989): 50–52. http://dx.doi.org/10.1136/adc.64.1_spec_no.50.

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30

Artikova, Mavlyuda, and Nozima Nabiyeva. "NEUROVISUAL CHARACTERISTICS OF STRUCTURAL ALTERATIONS OF BRAIN AT CHILDREN CEREBRAL PARALUSIS AND EPILEPSY." JOURNAL OF NEUROLOGY AND NEUROSURGICAL RESEARCH 1, no. 1 (2020): 42–46. http://dx.doi.org/10.26739/2181-0982-2020-1-11.

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Cerebral paralysis is the leading cause and serious matter of children's neurological invalidity throughout the world. Nowadays the forms of children's cerebral paralysis (CCP), complicated by symptomatic epilepsy (SE)which intencifies the disease are often being registered. Frequency of SE occurence come to 18-80 percent. Magnetic resonance imaging (MRI) is the first method of the investigation of patients with CCP with SE or without it. The target of the research is the improvement of SE diagnostics at CCP on the basis ofMRI investigation. Our results have relatively displayed low sensitivit

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31

Wu, Fenfen, Wentao Mi, and Stephen C. Cannon. "Beneficial effects of bumetanide in a CaV1.1-R528H mouse model of hypokalaemic periodic paralysis." Brain 136, no. 12 (2013): 3766–74. http://dx.doi.org/10.1093/brain/awt280.

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32

Corrochano, Silvia, Roope Männikkö, Peter I. Joyce, et al. "Novel mutations in human and mouse SCN4A implicate AMPK in myotonia and periodic paralysis." Brain 137, no. 12 (2014): 3171–85. http://dx.doi.org/10.1093/brain/awu292.

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33

Kim, Jiyoung, and Kyoung Jin Hwang. "Isolated unilateral lingual paralysis in a supranuclear infarction." Neurology Asia 26, no. 3 (2021): 621–22. http://dx.doi.org/10.54029/2021urm.

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Lingual paralysis can result from damage to both upper and lower motor neurons. However, since the hypoglossal nucleus is innervated from both hemispheres simultaneously, unilateral lingual paralysis caused by upper motor neuron has rarely been reported. We report a case which a patient isolated unilateral lingual paralysis due to supranuclear infarction. A 50-year-old right-handed woman was admitted to our hospital due to suddenly developed dysarthria. Her tongue deviated to the right on protrusion without fasciculation or atrophy. A brain MRI showed focal lesion in the left corona radiate an

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34

Cabrera-Gómez, J. A., N. Echazabal-Santana, Y. Real-González, et al. "Hereditary Melkersson-Rosenthal syndrome and multiple sclerosis." Multiple Sclerosis Journal 11, no. 3 (2005): 364–66. http://dx.doi.org/10.1191/1352458505ms1164cr.

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The revision of MEDLINE from 1966 to 2003 did not report any association between multiple sclerosis (MS) and Melkersson—Rosenthal syndrome (MRS). This is a case report of a 51-year-old woman, with history of four recurrent Bell’s palsies. In 1999 she developed a right facial paralysis due to a supranuclear pyramidal lesion with right monoparesis. The family history showed five relatives with recurrent Bell’s paralysis and plicata tongue. Physical examination: right Bell’s paralysis, left supranuclear facial paralysis, furrowed tongue, right hemiparesis with pallor of the optic disks. Brain mag

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35

Kuzmenkin, Alexey, Vanesa Muncan, Karin Jurkat‐Rott, et al. "Enhanced inactivation and pH sensitivity of Na+ channel mutations causing hypokalaemic periodic paralysis type II." Brain 125, no. 4 (2002): 835–43. http://dx.doi.org/10.1093/brain/awf071.

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36

Kübler, A., and N. Birbaumer. "Brain–computer interfaces and communication in paralysis: Extinction of goal directed thinking in completely paralysed patients?" Clinical Neurophysiology 119, no. 11 (2008): 2658–66. http://dx.doi.org/10.1016/j.clinph.2008.06.019.

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37

Nakajima, Riho, Masashi Kinoshita, Tetsutaro Yahata, and Mitsutoshi Nakada. "Recovery time from supplementary motor area syndrome: relationship to postoperative day 7 paralysis and damage of the cingulum." Journal of Neurosurgery 132, no. 3 (2020): 865–74. http://dx.doi.org/10.3171/2018.10.jns182391.

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OBJECTIVESupplementary motor area (SMA) syndrome is defined as temporary paralysis after the resection of brain tumor localized in the SMA. Although in most cases paralysis induced by SMA resection resolves within a short period, the time until complete recovery varies and has not been precisely analyzed to date. In this study, the authors investigated factors for predicting the time required for recovery from paralysis after SMA resection.METHODSData from 20 cases were analyzed. All 20 patients (mean age 54.9 ± 12.6 years) had undergone resection of frontal lobe glioma involving the SMA. The

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38

Lacaux, Célia, Charlotte Izabelle, Giulio Santantonio, et al. "Increased creative thinking in narcolepsy." Brain 142, no. 7 (2019): 1988–99. http://dx.doi.org/10.1093/brain/awz137.

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Abstract Some studies suggest a link between creativity and rapid eye movement sleep. Narcolepsy is characterized by falling asleep directly into rapid eye movement sleep, states of dissociated wakefulness and rapid eye movement sleep (cataplexy, hypnagogic hallucinations, sleep paralysis, rapid eye movement sleep behaviour disorder and lucid dreaming) and a high dream recall frequency. Lucid dreaming (the awareness of dreaming while dreaming) has been correlated with creativity. Given their life-long privileged access to rapid eye movement sleep and dreams, we hypothesized that subjects with

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39

Wu, Fenfen, Wentao Mi, Yu Fu, Arie Struyk, and Stephen C. Cannon. "Mice with an NaV1.4 sodium channel null allele have latent myasthenia, without susceptibility to periodic paralysis." Brain 139, no. 6 (2016): 1688–99. http://dx.doi.org/10.1093/brain/aww070.

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40

Pyka, M., M. Burgmer, T. Lenzen, et al. "Brain correlates of hypnotic paralysis—a resting-state fMRI study." NeuroImage 56, no. 4 (2011): 2173–82. http://dx.doi.org/10.1016/j.neuroimage.2011.03.078.

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41

Ward, N. S., D. A. Oakley, R. S. J. Frackowiak, and P. W. Halligan. "Differential brain activations during intentionally simulated and subjectively experienced paralysis." Cognitive Neuropsychiatry 8, no. 4 (2003): 295–312. http://dx.doi.org/10.1080/13546800344000200.

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42

Birbaumer, Niels, and Leonardo G. Cohen. "Brain-computer interfaces: communication and restoration of movement in paralysis." Journal of Physiology 579, no. 3 (2007): 621–36. http://dx.doi.org/10.1113/jphysiol.2006.125633.

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43

Henderson, Andrew, Renee Fisher, Michael Epperly, et al. "Increased Longevity of Continuous Bone Marrow Cultures and Radioresistance of Bone Marrow Stromal Cells from SOD193A ALS (Amyotrophic Lateral Sclerosis) Mice." Blood 132, Supplement 1 (2018): 5679. http://dx.doi.org/10.1182/blood-2018-99-113580.

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Abstract Introduction: The SOD1G93A mouse model of ALS, demonstrates hind limb paralysis beginning at 90 - 100 days of age with stage 4 paralysis at 125 days of age and progressive neuromuscular loss. Materials & Methods: To determine whether deficiency of functional SOD1 influenced parameters of hematopoiesis, long-term bone marrow cultures were established from ALS and control mice. Bone marrow stromal cell lines derived from LTBMCs were tested for clonogenic radiation survival. We tested the effect of bone marrow transplant after total body irradiation on delay of paralysis. Results: SO

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44

Sawalha, Khalid, and Krishna Kakkera. "Acute Respiratory Failure From Hypermagnesemia Requiring Prolonged Mechanical Ventilation." Journal of Investigative Medicine High Impact Case Reports 8 (January 2020): 232470962098489. http://dx.doi.org/10.1177/2324709620984898.

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Electrolyte abnormalities are an underrecognized cause of respiratory failure in the intensive care unit. One such abnormality is a relatively rare phenomenon of hypermagnesemia resulting in paralysis. A 73-year-old Caucasian male patient presented to the emergency department with diffuse abdominal pain of 2-day duration. He received magnesium citrate and gastrointestinal cocktail for his constipation after initial imaging showed constipation. In view of acute worsening, follow-up computed tomography of the abdomen was done, which showed free air in upper abdomen along with free fluid. Hence,

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45

Wu, Tara J., Sean M. Lewis, and Peak Woo. "Neurosarcoidosis Presenting Initially as Idiopathic Vocal Cord Paralysis." Annals of Otology, Rhinology & Laryngology 128, no. 2 (2018): 157–61. http://dx.doi.org/10.1177/0003489418808814.

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Introduction: The presentation, course, and management of a rare laryngeal manifestation of neurosarcoidosis due to central nervous system (as opposed to peripheral nervous system) injury are described. Methods: The authors present 3 cases of vocal cord paralysis as the initial symptom of isolated neurosarcoidosis at a tertiary care laryngology clinic. Results: Laryngoscopy diagnosed unilateral vocal cord paralysis. Laryngeal electromyography revealed high vagal injury, prompting workup on brain magnetic resonance imaging. On magnetic resonance imaging, 2 cases showed basilar leptomeningeal in

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46

Uri, Nechama, and Gerald Schuchman. "Vestibular abnormalities in patients with Bell's palsy." Journal of Laryngology & Otology 100, no. 10 (1986): 1125–28. http://dx.doi.org/10.1017/s0022215100100696.

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AbstractIn order to assess the extent of the underlying pathology in Bell's palsy, the vestibular function of 24 patients was evaluated within the first week from the onset of facial paralysis. Seven patients had pathological findings by ENG and an additional three had subjective vestibular signs; four of these ten patients also showed abnormal auditory brain stem evoked potentials. There was no correlation between the severity of the paralysis or the prognosis for recovery and the presence of vestibular abnormalities.

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47

Tolentino, Camille, and Emmanuel Tadeus Cruz. "Bilateral Facial Nerve (Bell’s) Palsy in a 24-Year-Old Woman: A Case Report." Philippine Journal of Otolaryngology Head and Neck Surgery 35, no. 1 (2020): 60–62. http://dx.doi.org/10.32412/pjohns.v35i1.1285.

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ABSTRACT Objective: To report a case of acute bilateral facial nerve palsy in a 24-year-old woman and to present the differential diagnoses, pathophysiology, management and prognosis Methods:  Design: Case study Setting: Tertiary Private Hospital Patient: One (1) Result: A 24-year-old woman with fever, joint pains, cough, chest pain, difficulty ambulating and progressive facial muscle weakness was diagnosed with rheumatic fever. Bilateral facial nerve paralysis was noted, and Electromyography-Nerve Conduction Velocity (EMG-NCV) testing with special facial ne

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48

Dyachenko, Pavel A., and Anatoly G. Dyachenko. "A CASE OF MRI-NEGATIVE HERPES VIRUS ENCEPHALITIS PRESENTED BY RAMSAY HUNT SYNDROME." Wiadomości Lekarskie 73, no. 11 (2020): 2555–56. http://dx.doi.org/10.36740/wlek202011139.

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Ramsay Hunt syndrome (RHS) occurs due to reactivation of latent Varicella Zoster Virus (VZV) infection in the geniculate ganglion of the facial nerve. Major clinical symptoms include ipsilateral facial paralysis, otic pain, and herpetic vesicles (rashes) along the nerve with accompanying ear pain. Rarely clinical findings include retrograde transaxonal spread of the virus from the ganglion into the brain parenchyma with developing the encephalitis or multiple cranial nerve involvement. We describe here a patient with both RHS along with complicating brains

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49

Zinser, Elisabeth, Matthias Lechmann, Antje Golka, Manfred B. Lutz, and Alexander Steinkasserer. "Prevention and Treatment of Experimental Autoimmune Encephalomyelitis by Soluble CD83." Journal of Experimental Medicine 200, no. 3 (2004): 345–51. http://dx.doi.org/10.1084/jem.20030973.

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CD83 is up-regulated on the surface of dendritic cells (DCs) during maturation and has been widely used as a marker for mature DCs. Recently, we reported the recombinant expression of the extracellular immunoglobulin domain of human CD83 (hCD83ext). Using this soluble form of CD83, allogeneic as well as specific cytotoxic T lymphocyte proliferation could be blocked in vitro. Here we report the functional analysis of soluble CD83 in vivo, using murine experimental autoimmune encephalomyelitis (EAE) as a model. Strikingly, only three injections of soluble CD83 prevented the paralysis associated

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Aizawa, Kenneth. "Consciousness: Don't Give Up on the Brain." Royal Institute of Philosophy Supplement 67 (July 7, 2010): 263–84. http://dx.doi.org/10.1017/s1358246110000032.

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AbstractIn the extended mind literature, one sometimes finds the claim that there is no neural correlate of consciousness. Instead, there is a biological or ecological correlate of consciousness. Consciousness, it is claimed, supervenes on an entire organism in action. Alva Noë is one of the leading proponents of such a view. This paper resists Noë's view. First, it challenges the evidence he offers from neuroplasticity. Second, it presses a problem with paralysis. Third, it draws attention to a challenge from the existence of metamers and visual illusions.

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