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Journal articles on the topic 'Brain Positron Emission Tomography'

Author: Grafiati

Published: 4 June 2021

Last updated: 5 February 2022

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1

Macapinlac, Homer A. "Positron Emission Tomography of the Brain." Neuroimaging Clinics of North America 16, no. 4 (2006): 591–603. http://dx.doi.org/10.1016/j.nic.2006.08.001.

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2

Mishina, Masahiro. "Positron Emission Tomography for Brain Research." Journal of Nippon Medical School 75, no. 2 (2008): 68–76. http://dx.doi.org/10.1272/jnms.75.68.

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3

Macapinlac, Homer A. "Positron Emission Tomography of the Brain." PET Clinics 2, no. 1 (2007): 1–13. http://dx.doi.org/10.1016/j.cpet.2007.09.001.

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4

Pak, Kyoungjune, Seong-Jang Kim, and In Joo Kim. "Obesity and Brain Positron Emission Tomography." Nuclear Medicine and Molecular Imaging 52, no. 1 (2017): 16–23. http://dx.doi.org/10.1007/s13139-017-0483-8.

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5

Jamieson, Dara G., and Joel H. Greenberg. "Positron emission tomography of the brain." Computerized Medical Imaging and Graphics 13, no. 1 (1989): 61–79. http://dx.doi.org/10.1016/0895-6111(89)90079-7.

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6

Hooker, Jacob M., and Richard E. Carson. "Human Positron Emission Tomography Neuroimaging." Annual Review of Biomedical Engineering 21, no. 1 (2019): 551–81. http://dx.doi.org/10.1146/annurev-bioeng-062117-121056.

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Neuroimaging with positron emission tomography (PET) is the most powerful tool for understanding pharmacology, neurochemistry, and pathology in the living human brain. This technology combines high-resolution scanners to measure radioactivity throughout the human body with specific, targeted radioactive molecules, which allow measurements of a myriad of biological processes in vivo . While PET brain imaging has been active for almost 40 years, the pace of development for neuroimaging tools, known as radiotracers, and for quantitative analytical techniques has increased dramatically over the pa

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7

Volkow, Nora D., and Laurence R. Tancredi. "Positron Emission Tomography: A Technology Assessment." International Journal of Technology Assessment in Health Care 2, no. 4 (1986): 577–94. http://dx.doi.org/10.1017/s0266462300003421.

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Positron emission tomography (PET) is a new nuclear medicine technique that has recently entered the clinical realm of medicine. Although it is a technique that can be utilized for assessment of biochemical and physiological parameters of any organ in the body, it has particular utility in the investigation of the brain. PET poses unique advantages over previous imaging devices. For the first time, it is feasible to investigate directly various biological parameters of the brain in a noninvasive way. PET allows for investigating the functional, biochemical, physiological, and pharmacological c

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8

Wong, Terence Z., Gert J. van der Westhuizen, and R. Edward Coleman. "Positron emission tomography imaging of brain tumors." Neuroimaging Clinics of North America 12, no. 4 (2002): 615–26. http://dx.doi.org/10.1016/s1052-5149(02)00033-3.

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9

Spence, Alexander M., David A. Mankoff, and Mark Muzi. "Positron emission tomography imaging of brain tumors." Neuroimaging Clinics of North America 13, no. 4 (2003): 717–39. http://dx.doi.org/10.1016/s1052-5149(03)00097-2.

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10

DAGANI, RON. "F Positron Emission Tomography Advances Brain Imaging." Chemical & Engineering News 66, no. 33 (1988): 26–29. http://dx.doi.org/10.1021/cen-v066n033.p026.

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11

Levivier, Marc, Serge Goldman, Luc M. Bidaut, et al. "Positron Emission Tomography-Guided Stereotactic Brain Biopsy." Neurosurgery 31, no. 4 (1992): 792–97. http://dx.doi.org/10.1097/00006123-199210000-00029.

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12

Levivier, Marc, Serge Goldman, Luc M. Bidaut, et al. "Positron Emission Tomography-Guided Stereotactic Brain Biopsy." Neurosurgery 31, no. 4 (1992): 792–97. http://dx.doi.org/10.1227/00006123-199210000-00029.

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13

Fox, Peter. "Functional Brain Mapping with Positron Emission Tomography." Seminars in Neurology 9, no. 04 (1989): 323–29. http://dx.doi.org/10.1055/s-2008-1041341.

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14

Raichle, Marcus E. "Positron emission tomography: Progress in brain imaging." Nature 317, no. 6038 (1985): 574–75. http://dx.doi.org/10.1038/317574a0.

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15

Mazière, B., and M. Mazière. "Positron emission tomography studies of brain receptors." Fundamental & Clinical Pharmacology 5, no. 1 (1991): 61–91. http://dx.doi.org/10.1111/j.1472-8206.1991.tb00702.x.

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16

Heurling, Kerstin, Antoine Leuzy, My Jonasson, et al. "Quantitative positron emission tomography in brain research." Brain Research 1670 (September 2017): 220–34. http://dx.doi.org/10.1016/j.brainres.2017.06.022.

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17

Mintun, Mark A. "FDG and Amyloid Positron Emission Tomography." CNS Spectrums 13, S16 (2008): 21–24. http://dx.doi.org/10.1017/s1092852900027000.

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For over 20 years, researchers have used the tracer [18F]fluorodeoxyglucose (FDG) in positron emission tomography (PET) imaging. FDG PET imaging has been utilized to study the characteristic metabolic changes in Alzheimer’s disease (AD), and as more molecular imaging tracers become available for human research, PET will likely assume many new roles for investigating more specific abnormalities, such as amyloid deposition, in the future.FDG is a glucose analog that images glucose metabolism and also illustrates neural firing. Different synapse activity, particularly excitatory activity from glu

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18

Momose, K. Jack. "Book ReviewPositron Emission Tomography Atlas of Positron Emission Tomography of the Brain." New England Journal of Medicine 315, no. 7 (1986): 464–65. http://dx.doi.org/10.1056/nejm198608143150724.

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19

Ozdemir, Semra, Yusuf Ziya Tan, Fulya Koc Ozturk, and Fatih Battal. "Confirmation of Brain Death with Positron Emission Tomography." Journal of Pediatric Intensive Care 09, no. 01 (2019): 051–53. http://dx.doi.org/10.1055/s-0039-1696652.

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AbstractAfter recent advances regarding organ transplantation, accurate and timely diagnosis of brain death has gained importance. In the diagnosis of brain death, in addition to clinical findings, various ancillary tests are very crucial. In this study, the scintigraphic imaging of the brain death of an 8-year-old girl with both Tc-99m diethylenetriaminepentaacetic and 18F-fluorodeoxyglucose (FDG) has been presented. This case study shows that 18F-FDG positron emission tomography-computed tomography imaging can be a useful technique in evaluating brain death in patients.

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20

HOLCOMB, HENRY H. "Positron Emission Tomography; Positron Emission Tomography and Autoradiography: Principles and Applications for the Brain and Heart." American Journal of Psychiatry 146, no. 1 (1989): 116. http://dx.doi.org/10.1176/ajp.146.1.116.

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21

Martin, W. R. Wayne. "Positron Emission Tomography in Movement Disorders." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 12, no. 1 (1985): 6–10. http://dx.doi.org/10.1017/s0317167100046515.

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ABSTRACT:Positron emission tomography provides a method for the quantitation of regional function within the living human brain. Studies of cerebral metabolism and blood flow in patients with Huntington’s disease, Parkinson’s disease and focal dystonia have revealed functional abnormalities within substructures of the basal ganglia. Recent developments permit assessment of both pre-synaptic and post-synaptic function in dopaminergic pathways. These techniques are now being applied to studies of movement disorders in human subjects.

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22

Chiaravalloti, Agostino, and Alessandro Micarelli. "Brain Imaging with Positron Emission Tomography: Novel Radiopharmaceuticals." Current Medicinal Chemistry 25, no. 26 (2018): 3060. http://dx.doi.org/10.2174/092986732526180806162949.

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23

Juhász, Csaba, and Harry T. Chugani. "Imaging the epileptic brain with positron emission tomography." Neuroimaging Clinics of North America 13, no. 4 (2003): 705–16. http://dx.doi.org/10.1016/s1052-5149(03)00090-x.

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24

Meltzer, Carolyn Cidis, James T. Becker, Julie C. Price, and Eydie Moses-Kolko. "Positron emission tomography imaging of the aging brain." Neuroimaging Clinics of North America 13, no. 4 (2003): 759–67. http://dx.doi.org/10.1016/s1052-5149(03)00108-4.

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25

Sossi, Vesna. "Cutting-Edge Brain Imaging with Positron Emission Tomography." Neuroimaging Clinics of North America 17, no. 4 (2007): 427–40. http://dx.doi.org/10.1016/j.nic.2007.07.006.

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26

Fukuda, Hiroshi. "Visualization of Brain Function Using Positron Emission Tomography." Journal of the Visualization Society of Japan 14, Supplement2 (1994): 9–14. http://dx.doi.org/10.3154/jvs.14.supplement2_9.

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27

Garnett, E. Stephen. "Atlas of Positron Emission Tomography of the Brain." Radiology 159, no. 3 (1986): 598. http://dx.doi.org/10.1148/radiology.159.3.598.

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28

Phelps, M., and J. Mazziotta. "Positron emission tomography: human brain function and biochemistry." Science 228, no. 4701 (1985): 799–809. http://dx.doi.org/10.1126/science.2860723.

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29

Oldendorf, William H. "Atlas of Positron Emission Tomography of the Brain." Journal of Computer Assisted Tomography 10, no. 5 (1986): 822. http://dx.doi.org/10.1097/00004728-198609000-00021.

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30

Sossi, Vesna. "Cutting-Edge Brain Imaging with Positron Emission Tomography." PET Clinics 2, no. 1 (2007): 91–104. http://dx.doi.org/10.1016/j.cpet.2007.09.007.

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31

May, A. "Hypothalamic Deep Brain Stimulation in Positron Emission Tomography." Journal of Neuroscience 26, no. 13 (2006): 3589–93. http://dx.doi.org/10.1523/jneurosci.4609-05.2006.

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32

DOMINO, E., and H. TSUKADA. "Positron emission tomography to quantify brain nicotine abstinence." Clinical Pharmacology & Therapeutics 77, no. 2 (2005): P98. http://dx.doi.org/10.1016/j.clpt.2005.01.003.

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33

Herscovitch, Peter. "EVALUATION OF THE BRAIN BY POSITRON EMISSION TOMOGRAPHY." Rheumatic Disease Clinics of North America 19, no. 4 (1993): 765–94. http://dx.doi.org/10.1016/s0889-857x(21)00206-4.

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34

Breuer, Heike, Martin Meier, Sophie Schneefeld, et al. "Multimodality imaging of blood–brain barrier impairment during epileptogenesis." Journal of Cerebral Blood Flow & Metabolism 37, no. 6 (2016): 2049–61. http://dx.doi.org/10.1177/0271678x16659672.

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Insult-associated blood–brain barrier leakage is strongly suggested to be a key step during epileptogenesis. In this study, we used three non-invasive translational imaging modalities, i.e. positron emission tomography, single photon emission computed tomography, and magnetic resonance imaging, to evaluate BBB leakage after an epileptogenic brain insult. Sprague-Dawley rats were scanned during early epileptogenesis initiated by status epilepticus. Positron emission tomography and single photon emission computed tomography scans were performed using the novel tracer [68Ga]DTPA or [99mTc]DTPA, r

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35

Shlamkovich, Nathan, Haim Gavriel, Ephraim Eviatar, Mordechay Lorberboym, and Eliad Aviram. "Brain Positron Emission Tomography-Computed Tomography Gender Differences in Tinnitus Patients." Journal of the American Academy of Audiology 27, no. 09 (2016): 714–19. http://dx.doi.org/10.3766/jaaa.15067.

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Background: Increased metabolism in the left auditory cortex has been reported in tinnitus patients. However, gender difference has not been addressed. Purpose: To assess the differences in Positron emission tomography–computed tomography (PET-CT) results between the genders in tinnitus patients. Research Design: Retrospective cohort. Study Sample: Included were patients referred to our clinic between January 2011 and August 2013 who complained of tinnitus and underwent fluorodeoxyglucose (FDG)-PET to assess brain metabolism. Data Analysis: Univariate and multivariate nominal logistic regressi

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36

Herscovitch, Peter, Alexander P. Auchus, Mokhtar Gado, David Chi, and Marcus E. Raichle. "Correction of Positron Emission Tomography Data for Cerebral Atrophy." Journal of Cerebral Blood Flow & Metabolism 6, no. 1 (1986): 120–24. http://dx.doi.org/10.1038/jcbfm.1986.14.

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Because positron emission tomography (PET) provides measurements per unit volume of intracranial contents, these measurements may be affected by the inclusion of metabolically inactive CSF spaces in the volume in which they are made. Thus, PET measurements of CBF and metabolism may be artifactually lowered in normal aging and dementia, which are both associated with significant brain atrophy. We describe a method to correct global PET data, averaged over several tomographic slices, for cerebral atrophy by using measurements of CSF space volume obtained with quantitative x-ray computed tomograp

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37

McCarthy, M., JB Yuan, A. Campbell, NP Lenzo, and K. Butler-Henderson. "18F-fluorodeoxyglucose positron emission tomography imaging in brain tumours: The Western Australia positron emission tomography/cyclotron service experience." Journal of Medical Imaging and Radiation Oncology 52, no. 6 (2008): 564–69. http://dx.doi.org/10.1111/j.1440-1673.2008.02019.x.

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38

Rozental, Jack M. "Positron Emission Tomography (PET) and Single-Photon Emission Computed Tomography (SPECT) of Brain Tumors." Neurologic Clinics 9, no. 2 (1991): 287–305. http://dx.doi.org/10.1016/s0733-8619(18)30285-8.

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39

Kadir, Ahmadul, Amelia Marutle, Daniel Gonzalez, et al. "Positron emission tomography imaging and clinical progression in relation to molecular pathology in the first Pittsburgh Compound B positron emission tomography patient with Alzheimer’s disease." Brain 134, no. 1 (2010): 301–17. http://dx.doi.org/10.1093/brain/awq349.

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40

Young, Trevor, and Peter Williamson. "Brain Imaging in Functional Mental Disorders." Canadian Journal of Psychiatry 31, no. 7 (1986): 675–80. http://dx.doi.org/10.1177/070674378603100716.

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The application of brain imaging techniques to psychiatry is reviewed with respect to computerized tomography (CT), EEG topography, positron emission tomography (PET), and magnetic resonance imaging (MRI). While early computerized tomography studies have suggested structural abnormalities in schizophrenia, more recent studies have shown that most schizophrenics and patients with other disorders have normal CT scans. EEG topography and positron emission tomography have not been evaluated as fully as computerized tomography. However, preliminary studies indicate some functional abnormalities in

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41

OGAWA, TOSHIHIDE, KAZUO UEMURA, IWAO KANNO, et al. "Delayed radiation necrosis of brain evaluated positron emission tomography." Tohoku Journal of Experimental Medicine 155, no. 3 (1988): 247–60. http://dx.doi.org/10.1620/tjem.155.247.

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42

D′Souza, MariaMathew, Madhavi Tripathi, Abhinav Jaimini, Rajnish Sharma, Puja Panwar, and Anupam Mondal. "Novel positron emission tomography radiotracers in brain tumor imaging." Indian Journal of Radiology and Imaging 21, no. 3 (2011): 202. http://dx.doi.org/10.4103/0971-3026.85369.

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43

Raine, Adrian, Monte Buchsbaum, and Lori Lacasse. "Brain abnormalities in murderers indicated by positron emission tomography." Biological Psychiatry 42, no. 6 (1997): 495–508. http://dx.doi.org/10.1016/s0006-3223(96)00362-9.

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44

Vallabhajosula, Shankar. "Positron Emission Tomography Radiopharmaceuticals for Imaging Brain Beta-Amyloid." Seminars in Nuclear Medicine 41, no. 4 (2011): 283–99. http://dx.doi.org/10.1053/j.semnuclmed.2011.02.005.

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45

Ericson, K., H. von Holst, M. Mosskin, et al. "Positron Emission Tomography of Cavernous Haemangiomas of the Brain." Acta Radiologica. Diagnosis 27, no. 4 (1986): 379–83. http://dx.doi.org/10.1177/028418518602700402.

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Four cases with lesions suspected to be low-grade intracerebral tumours but later proved to be cavernous haemangiomas are described. The patients were examined with contrast enhanced CT and with positron emission tomography (PET). The lesions were partly calcified with a mild or no mass effect and a slight contrast enhancement at CT. There were signs of disrupture of the blood-lesion barrier also on radionuclide studies. PET with 11C-methionine and 11C-glucose showed a normal or decreased accumulation of the tracers. This combination of findings has not been encountered in intracranial tumours

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46

Jung, Ji-hoon, and Byeong-Cheol Ahn. "Current Radiopharmaceuticals for Positron Emission Tomography of Brain Tumors." Brain Tumor Research and Treatment 6, no. 2 (2018): 47. http://dx.doi.org/10.14791/btrt.2018.6.e13.

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47

Baron, J. C., D. Comar, E. Zarifian, et al. "Dopaminergic receptor sites in human brain: Positron emission tomography." Neurology 35, no. 1 (1985): 16. http://dx.doi.org/10.1212/wnl.35.1.16.

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48

Chugani, Harry T., Michael E. Phelps, and John C. Mazziotta. "Positron emission tomography study of human brain functional development." Annals of Neurology 22, no. 4 (1987): 487–97. http://dx.doi.org/10.1002/ana.410220408.

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49

Hino, Akihiko, Yoshio Imahori, Hiroshi Tenjin, et al. "Metabolic and Hemodynamic Aspects of Peritumoral Low-Density Areas in Human Brain Tumor." Neurosurgery 26, no. 4 (1990): 615–21. http://dx.doi.org/10.1227/00006123-199004000-00009.

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Abstract With the use of positron emission tomography, regional cerebral blood flow, oxygen utilization, and glucose utilization were measured in the peritumoral low-density areas on x-ray computed tomographic images in 23 patients with supratentorial brain tumors: 7 meningiomas, 11 malignant gliomas, and 5 metastatic brain tumors. Findings on positron emission tomography in these areas revealed characteristic patterns associated with the types of tumor and the degree of mass effect. It is likely that two different types of pathophysiological states exist in “peritumoral edema”: 1) primary isc

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50

Bai, Ping, Sha Bai, Michael S. Placzek, et al. "A New Positron Emission Tomography Probe for Orexin Receptors Neuroimaging." Molecules 25, no. 5 (2020): 1018. http://dx.doi.org/10.3390/molecules25051018.

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The orexin receptor (OX) is critically involved in motivation and sleep−wake regulation and holds promising therapeutic potential in various mood disorders. To further investigate the role of orexin receptors (OXRs) in the living human brain and to evaluate the treatment potential of orexin-targeting therapeutics, we herein report a novel PET probe ([11C]CW24) for OXRs in the brain. CW24 has moderate binding affinity for OXRs (IC50 = 0.253 μM and 1.406 μM for OX1R and OX2R, respectively) and shows good selectivity to OXRs over 40 other central nervous system (CNS) targets. [11C]CW24 has high b

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