Relevant bibliographies by topics / Branchial arch

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Branchial arch'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 June 2025

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Journal articles on the topic "Branchial arch"

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Cohen, M. Michael. "Branchial Arch Syndromes." Cleft Palate-Craniofacial Journal 32, no. 6 (1995): 524. http://dx.doi.org/10.1597/1545-1569(1995)032<0524:bas>2.3.co;2.

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Cohen, M. Michael. "Branchial Arch Syndromes." Cleft Palate-Craniofacial Journal 32, no. 6 (1995): 524. http://dx.doi.org/10.1597/1545-1569_1995_032_0524_bas_2.3.co_2.

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Alfi, David, Din Lam, and Jaime Gateno. "Branchial Arch Syndromes." Atlas of the Oral and Maxillofacial Surgery Clinics 22, no. 2 (2014): 167–73. http://dx.doi.org/10.1016/j.cxom.2014.04.003.

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Al-Mufarrej, Faisal M., David G. Stoddard, and Uldis Bite. "Branchial Arch Anomalies." Plastic and Reconstructive Surgery 130 (November 2012): 7. http://dx.doi.org/10.1097/01.prs.0000421704.82385.61.

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Thakur, Ashoka Nand, and Priyambada . "Branchial Sinus with Cyst - Two Case Reports with Review of Literature." International Journal of Research and Review 8, no. 11 (2021): 375–77. http://dx.doi.org/10.52403/ijrr.20211147.

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Branchial cleft anomalies are well described, with the second arch anomaly being the commonest. Remains of cervical sinus of His may persist as a branchial cyst. A branchial sinus is formed when 2nd branchial arch fails to meet the 5th pharyngeal arch. Peak age for presentation of branchial cysts is in the third decade and that of the congenital sinuses and fistulae is at birth. The association of a branchial cyst with branchial sinus is very rare. We are presenting two cases had branchial cyst along with branchial sinus. It was managed successfully with complete excision. Histopathological examination confirmed the association. Keywords: Branchial Sinus, Branchial cyst,
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Goswami, Jayanta Kumar, Abhijit Sarma, and Chandan Jyoti Saikia. "RARE BRANCHIAL ARCH ANOMALIES." Journal of Evidence Based Medicine and Healthcare 3, no. 19 (2016): 752–55. http://dx.doi.org/10.18410/jebmh/2016/171.

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Johnson, I. J. M., J. V. Soames, and J. P. Birchall. "Fourth branchial arch fistula." Journal of Laryngology & Otology 110, no. 4 (1996): 391–93. http://dx.doi.org/10.1017/s0022215100133730.

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AbstractA case is presented of a rare congenital anomaly of the fourth branchial arch, which presented as an abscess in the anterior triangle, related to a fistula communicating with the pyriform fossa. Histopathological examination showed the fistula to be associated with thyroid tissue supporting the hypothesis that the ventral wing of the fourth pouch contributes to the thyroid gland.
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Thomas, T., H. Kurihara, H. Yamagishi, et al. "A signaling cascade involving endothelin-1, dHAND and msx1 regulates development of neural-crest-derived branchial arch mesenchyme." Development 125, no. 16 (1998): 3005–14. http://dx.doi.org/10.1242/dev.125.16.3005.

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Numerous human syndromes are the result of abnormal cranial neural crest development. One group of such defects, referred to as CATCH-22 (cardiac defects, abnormal facies, thymic hypoplasia, cleft palate, hypocalcemia, associated with chromosome 22 microdeletion) syndrome, exhibit craniofacial and cardiac defects resulting from abnormal development of the third and fourth neural crest-derived branchial arches and branchial arch arteries. Mice harboring a null mutation of the endothelin-1 gene (Edn1), which is expressed in the epithelial layer of the branchial arches and encodes for the endothelin-1 (ET-1) signaling peptide, have a phenotype similar to CATCH-22 syndrome with aortic arch defects and craniofacial abnormalities. Here we show that the basic helix-loop-helix transcription factor, dHAND, is expressed in the mesenchyme underlying the branchial arch epithelium. Further, dHAND and the related gene, eHAND, are downregulated in the branchial and aortic arches of Edn1-null embryos. In mice homozygous null for the dHAND gene, the first and second arches are hypoplastic secondary to programmed cell death and the third and fourth arches fail to form. Molecular analysis revealed that most markers of the neural-crest-derived components of the branchial arch are expressed in dHAND-null embryos, suggesting normal migration of neural crest cells. However, expression of the homeobox gene, Msx1, was undetectable in the mesenchyme of dHAND-null branchial arches but unaffected in the limb bud, consistent with the separable regulatory elements of Msx1 previously described. Together, these data suggest a model in which epithelial secretion of ET-1 stimulates mesenchymal expression of dHAND, which regulates Msx1 expression in the growing, distal branchial arch. Complete disruption of this molecular pathway results in growth failure of the branchial arches from apoptosis, while partial disruption leads to defects of branchial arch derivatives, similar to those seen in CATCH-22 syndrome.
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Whitworth, I. H., S. K. Suvarna, R. G. Wight, and G. P. Walsh-Waring. "Fourth branchial arch anomaly: a rare incidental finding in an adult." Journal of Laryngology & Otology 107, no. 3 (1993): 238–39. http://dx.doi.org/10.1017/s002221510012273x.

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A rare finding of a fourth branchial arch anomaly discovered at laryngectomy is presented. Clinical and histological findings are documented. The incidence and presentation of fourth branchial arch anomalies is discussed.
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Hall, A. Brad, Shannon Hasara, and Phillip Coker. "Identification of a branchial cleft anomaly via handheld point-of-care ultrasound." Journal of Ultrasonography 22, no. 88 (2022): 67–69. http://dx.doi.org/10.15557/jou.2022.0012.

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Aim of the study: Branchial anomalies result from incomplete obliteration of the branchial arch structures during embryogenesis. Second branchial arch anomalies are commonly found on the lower third of the neck, with an opening at the anterior border of the sternocleidomastoid muscle, and may drain secretions or purulent material. This case demonstrates the use of handheld point-of-care ultrasound to aid in the diagnosis of a branchial anomaly. Case description: The patient presented with a “hole” in the neck with intermittent drainage from the site. A 2 mm defect in the skin was noted anterior to the sternocleidomastoid muscle. A handheld ultrasound system was used to identify a well-defined, hypoechoic, cyst-like structure. Given the history, physical findings, and point-of-care ultrasound imaging, the diagnosis of a second branchial cleft sinus was made. Conclusions: The use of point-of-care ultrasound and knowledge of the sonographic characteristics of these lesions can assist the physician in the diagnosis of branchial arch anomalies.
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Dissertations / Theses on the topic "Branchial arch"

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Wang, Yu Ling. "Mechanisms of triazole and retinoic acid-induced branchial arch malformation." Thesis, St George's, University of London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338915.

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Ryll, B. "The role of Hand2 in branchial arch and head-shoulder patterning." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/20470/.

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Comprehending gnathostome evolution requires insights into key cellular and molecular components of craniofacial and shoulder development. For the work of this PhD, I made use of genetically modified mouse models to study aspects of mammalian head and shoulder morphogenesis by triple fluorescent RNA in situ hybridisation, immunohistochemistry and high resolution imaging. First- I use a genetically defined sentinel cell population labelled by the Hand2-Cre transgene to establish the expansion of the distal-most branchial arch domain and correlate this by triple fluorescent RNA in situ hybridisation with the system controlling proximo-distal branchial arch patterning, the Dlx system. I find that the axis of the Dlx system does not correspond to the proximo-distal but an endodermal-ectodermal axis of the arch and rotates during development; the overall expansion of the arch is explicable by telescopic outgrowth along this new axis. Second- I study the cellular and molecular characteristics of head/ shoulder skeleto-muscular connectivity and the contribution of limb lateral plate mesoderm to the shoulder girdle, which allows me to identify part of the manubrium sterni as the ‘lost’ mammalian procoracoid and to demonstrate that the interaction between lateral plate mesodermal subpopulations is non-random. Third- I establish novel roles for Hand2 in lower incisor ameloblasts and in laminar dermal bone formation, suggesting a fundamental role for Hand2 in epithelial and mesenchymal cell layer arrangements. My detailed study of the murine frontal bone reveals that the establishment of an internal and an external layer initiates dermal bone formation; the latter shows intermediate molecular periosteal/ perichondrial characteristics and generates the intermediate layer by a Hand2-dependent invagination process. For a comparative amphibian data set, I begin to establish genetic lineage labelling as technique in Xenopus tropicalis. I generate and test a Xenopus Hand2-Cre transgene and establish a stable generic Xenopus tropicalis Cre-reporter line by I-SceI mediated transgenesis.
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Choudhury, Anuradha. "The regulation and function of DLX genes in the first branchial arch." Thesis, King's College London (University of London), 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.420629.

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Hung, Siu-chun. "Analysis of abnormal branchial arch structures of a Hoxb3 transgenic mouse mutant using a lacZ Reporter mouse line." Click to view the E-thesis via HKUTO, 2004. http://sunzi.lib.hku.hk/hkuto/record/B31971830.

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Hung, Siu-chun, and 洪少俊. "Analysis of abnormal branchial arch structures of a Hoxb3 transgenic mouse mutant using a lacZ Reporter mouse line." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B31971830.

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Losa, Llabata Marta. "Gene regulation in embryonic development." Thesis, University of Manchester, 2016. https://www.research.manchester.ac.uk/portal/en/theses/gene-regulation-in-embryonic-development(8a9efb79-1ca9-409e-89b9-9d66213e593f).html.

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Branchial arches (BAs) are a series of transient structures that develop on the ventro-lateral surface of the head in vertebrate embryos. BAs initially appear as a series of similar segments; as development proceeds each BA will contribute to different structures. Here, it was investigated the transcriptional mechanisms that instruct the different fates of the BAs in development. Initially, each BA contains a blood vessel, known as aortic arch (AA) artery, that connects the dorsal aorta with the heart. Remodelling of the AAs is crucial to form the adult heart circulation. This process leads to regression of the anterior AAs, running though the first and second BAs (BA1 and BA2), and persistence of the AAs contained in more posterior BAs (PBA). To identify the mechanisms that control remodelling of the AAs, we compared the transcriptomes and epigenomic landscapes of different BAs. Using RNA-seq and H3K27Ac ChIP-seq, we uncovered the activation of a vascular smooth muscle cell (VSMC) differentiation transcriptional program exclusively in the PBAs (and not in BA1/BA2). In support of this finding, we show that VSMC differentiation occurs specifically in the PBAs, but not BA1-2 in mouse embryonic development. Despite the absence of VSMC differentiation in developing BA1-2, cells harvested from these tissues reveal a spontaneous tendency to differentiate towards VSMC fate when grown in vitro, and activate several VSMC-specific genes (Myocd, Acta2, Tagln, Jag1). Together, our results suggest that forming VSMCs is a key process for the persistence of AAs. We also showed that cells derived from all BAs have the potential to differentiate to VSMCs in vitro. However, only cells in the PBAs differentiate to VSMCs in vivo, resulting in the maintenance of posterior AAs. In this study, we also uncovered a novel transcriptional principle that specifies the fate of BA2. Using ChIP-seq, we found that binding of Meis transcription factors establish a ground pattern in the BAs. Hoxa2, which specifies BA2 identity, selects a subset of Meis-bound sites. Meis binding is strongly increased at these sites, which coincide with active enhancers, linked to genes highly expressed in the BA2 and regulated by Hoxa2. Thus, Hoxa2 modifies a ground state binding of Meis to instruct segment-specific transcriptional programs.
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Luise, Fabiana. "A systems biology approach to model the development of the mouse second branchial arch in mouse embryonic stem cells." Thesis, University of Manchester, 2018. https://www.research.manchester.ac.uk/portal/en/theses/a-systems-biology-approach-to-model-the-development-of-the-mouse-second-branchial-arch-in-mouse-embryonic-stem-cells(95f1e66d-4260-413c-bc9a-2eb106df4c84).html.

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The generation of the face and neck is unique to vertebrate embryonic development, and requires a complex orchestration of dynamic molecular and tissue interactions. Elucidation of the molecular control of craniofacial development is important for the understanding of morphogenesis, evolution and disease. Branchial arches (BAs) are transient structures of early craniofacial development giving rise to different components of the oropharyngeal apparatus. BA development requires the contribution of endoderm, mesoderm, ectoderm and cranial neural crest cells (CNCCs). CNCCs form transiently during embryogenesis and are characterised by extensive multipotency and migratory abilities. CNCCs migration represents a key step in the evolution of the vertebrate head. This research aimed to uncover the molecular mechanisms regulating the mouse second branchial arch (IIBA), which contributes to the formation of the middle ear and neck. IIBA development depends on the expression of the transcription factor Hoxa2. Abnormal development of the IIBA is responsible for many craniofacial congenital malformations, therefore, dissecting the IIBA molecular control is important for gaining insights into development and disease mechanisms. Inference of regulatory networks from omic data has provided a valuable tool to study biological mechanisms. A network model from mouse IIBA transcriptomic data was generated by using an overlapping module algorithm. This model identified CBL, CTNNB1, EP300, EGFR and CDH1 (E-CADHERIN) as putative regulatory proteins associated with IIBA development and suggested the presence of an epithelial to mesenchymal transition (EMT) event. EMT is a crucial process occurring during embryonic development and adult tissue homeostasis, which is characterised by loss of cell-cell adhesion and increased cellular motility. A prerequisite for EMT is the loss of E-CADHERIN. By means of a network comparison approach, the study uncovered a statistically significant overlap between the IIBA network model and one generated from transcriptomic data of wild type (wtD3) versus E-cadherin knock-out (Ecad-/-) mouse embryonic stem cells (mESCs) (Ecad-/- vs wtD3), with candidate proteins shared between the two models. The function of the common putative regulator EP300 and its related histone epigenetic signature H3K27ac was interrogated in both wtD3 and Ecad-/- mESCs by chromatin immunoprecipitation and massive parallel sequencing (ChIP-seq) analysis. The results showed that EP300 and H3K27ac binding signatures were enriched in Ecad-/- mESCs, suggestive of a poised epigenetic EMT phenotype. EP300 and H3K27ac were increased at network candidate genomic loci. Conversely, ChIP-seq analysis on human induced pluripotent stem cells (hiPSCs) treated with an E-CADHERIN inhibiting peptide revealed absence of overlap with the network, suggesting they could map to a different stage of IIBA development. The function of the candidate molecules identified through the IIBA network model was tested by chemical inhibition assays in mESCs induced to differentiate with retinoic acid (RA) treatment and subsequent analysis of Hoxa2 transcript expression. CDH1 and CTNNB1 repress Hoxa2 transcript expression, whereas CBL and EGFR are shown to be required for Hoxa2 activation. This research is the the first demonstration of a network biology approach to the study of IIBA development in silico and confirmation of candidate protein function using an in vitro model of IIBA development.
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Bontemps, Claire. "Premier arc branchial et formation de la face." Paris 5, 2003. http://www.theses.fr/2003PA05CD02.

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Notre travail porte sur les mécanismes de développement de l'appareil masticateur à partir des dérivés mésenchymateux du premier arc branchial. Les résultats obtenus sur le cartilage de Meckel précisent son importance comme axial initial de la formation de la mandibule. Les muscles masticateurs se développent comme la plupart des muscles en deux générations de fibres. Le masséter est particulier car certaines de ses fibres expriment les isoformes ontogéniques de la myosine chez l'adulte. La deuxième partie de notre travail porte sur quelques observations de malformations faciales. Les malformations avec atteinte du trijumeau (syndrome de Franceschetti, fentes labio-nasales), montrent son importance dans l'induction des territoires correspondants. Dans des malformations faciales sans atteinte du nerf trijumeau, les dérivés du premier arc se développent normalement. Des observations d'holoprosencéphalie objectivent le rôle des bourgeons maxillaires dans la formation de la lèvre supérieure.
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PARTOUCHE, FRANCINE. "La fente n 7 de tessier : a propos d'une observation." Clermont-Ferrand 1, 1993. http://www.theses.fr/1993CLF1MS18.

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Hebibi, Nadra. "Donnees de morphometrie branchiale dans quelques cas d'adaptation chez la truite arc-en-ciel." Université Louis Pasteur (Strasbourg) (1971-2008), 1988. http://www.theses.fr/1988STR13086.

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Books on the topic "Branchial arch"

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Vester, Udo, and Stefanie Weber. Branchio-oto-renal syndrome. Edited by Adrian Woolf. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0358.

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Branchio-oto-renal (BOR) syndrome involves branchial arch fistulas or cysts, ear malformations with hearing loss, and anomalies of the kidney. BOR syndrome is inherited in an autosomal dominant trait and is caused in most cases by mutations in the EYA1 gene. A few families with gene mutations in SIX1 or SIX5 have also been described. The variability of clinical symptoms is wide. Renal involvement is observed in the majority of cases ranging from mild anomalies (e.g. dilation or duplication of the urinary tract) to severe hypodysplasia of the kidneys which eventually lead to renal failure. Branchio-otic syndrome (BOS) is characterized by branchial arch and ear anomalies without detectable renal pathology. BOR and BOS can be seen within the same family.
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Stavans, Ilan, ed. Telenovelas. ABC-CLIO, LLC, 2010. http://dx.doi.org/10.5040/9798216024019.

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Drama! Excess! Men in bee suits! Often erroneously compared to soap operas of the United States, outside of the necessary and sometimes fantastical dramatic story arc, however, the telenovela differs greatly from U.S. soap operas and have regional and cultural distinctions throughout Latin America. In Telenovelas, Ilan Stavans has gathered over two-dozen essays covering the telenovela for readers to better understand the phenomenon and its myriad layers. Branching off from radionovelas, the telenovela was exported from pre-Castro Cuba during the 1950s. The essays found in Telenovelas covers a broad view of the genre, television's impact in Latino culture, as well as more in-depth discussions of specific telenovelas throughout the Spanish-speaking television audience in the North America. Also explored is how telenovelas depict stereotypes, respond to gender and class roles, and examines the differences in topic and thematic choices as well as production values unique to each country.
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Book chapters on the topic "Branchial arch"

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Baertschiger, Reto M., and Lourenço Sbragia. "Branchial Clefts and Arch Anomalies." In Pearls and Tricks in Pediatric Surgery. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-51067-1_44.

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Abdulkader, Faisal, Francis J. Lannigan, and Mahmoud Taha. "Branchial Arch: Anatomy and Anomalies." In Textbook of Clinical Otolaryngology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-54088-3_57.

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Kerkfeld, Valentin, and Ulrich Meyer. "Treatment Principles of Branchial Arch Diseases." In Fundamentals of Craniofacial Malformations. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-28069-6_9.

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Meyer, Ulrich. "Biological Basis of Branchial Arch Diseases." In Fundamentals of Craniofacial Malformations. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-46024-2_10.

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Meyer, Ulrich, and Valentin Kerkfeld. "Diagnosis and Classification of Branchial Arch Diseases." In Fundamentals of Craniofacial Malformations. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-28069-6_8.

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Lommen, Julian, Valentin Kerkfeld, and Ulrich Meyer. "Facial Reconstruction in Adult Branchial Arch Disease Patients." In Fundamentals of Craniofacial Malformations. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-51773-0_17.

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Garrett, Katherine. "Evaluation and Treatment of the Horse with Fourth Branchial Arch Defects." In Advances in Equine Upper Respiratory Surgery. John Wiley & Sons, Inc., 2014. http://dx.doi.org/10.1002/9781118834183.ch13.

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Uchino, Akira. "Branching Variations from the Aortic Arch and Aortic Arch Anomaly." In Atlas of the Supraaortic Craniocervical Arterial Variations. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-6803-6_1.

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Guyot, Laurent, Pierre Seguin, and Hervé Benateau. "Kystes et fistules du 2e arc branchial." In Techniques en chirurgie maxillo-faciale et plastique de la face. Springer Paris, 2010. http://dx.doi.org/10.1007/978-2-8178-0073-8_60.

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Babovic, Neira, Vittoria Laghi, and Harald Kloft. "Branching Structure for Reinforcement Anchorage Produced with Wire and Arc Additive Manufacturing Technique." In Construction 3D Printing. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-64269-2_33.

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Conference papers on the topic "Branchial arch"

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Carvalho, Cláudio, Jonas Costa, Raul Lopes, Ana Karolina Maia, Nicolas Nisse, and Cláudia Linhares Sales. "Characterizing Networks Admitting k Arc-disjoint Branching Flows." In Encontro de Teoria da Computação. Sociedade Brasileira de Computação - SBC, 2020. http://dx.doi.org/10.5753/etc.2020.11089.

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An s-branching flow f in a network N = (D,c) (where c is the capacity function) is a flow that reaches every vertex in V(D) \ {s} from s while loosing exactly one unit of flow in each vertex other than s. In other words, the difference between the flow entering a vertex v and a flow leaving a vertex v is one whenever v is different from s. It is known that the hardness of the problem of finding k arc-disjoint s-branching flows in network N is linked to the capacity c of the arcs in N: the problem is solvable in polynomial time if every arc has capacity n - l, for fixed l, and NP-complete in most other cases, with very few cases open. We further investigate a conjecture by Costa et al. from 2019 that aims to characterize networks admitting k arc-disjoint s-branching flows, generalizing a classical result by Edmonds that provides such characterization when all arcs have capacity n-1. We show that, in general, the conjecture is false. However, on the positive side, it holds for digraphs formed by out-branchings together with parallel arcs.
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Nardi, Asaph, Idit Avrahami, Moshe Halak, Daniel Silverberg, and Moshe Brand. "Hemodynamical Aspects of Endovascular Repair for Aortic Arch Aneurisms." In ASME 2014 12th Biennial Conference on Engineering Systems Design and Analysis. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/esda2014-20234.

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The presented study is focused on the hemodynamics aspects of thoracic aortic aneurysm and approaches for restoring hemodynamics in the aortic arch. The study includes numerical investigation of the aortic arch hemodynamics of a healthy aorta, aorta with aneurysm, and of two endovascular repairing procedures. The first endovascular repair approach is the total aortic arch hybrid debranching. The second implantation uses chimney graft technique. The analysis includes the fluid dynamics in the aorta and branching arteries under time-dependent physiological conditions. The results show the effect of aneurysm on blood flow in the descending aorta and in aortic arch side branches. In the aneurysmatic case, the aneurysm provokes a highly disturbed flow and large recirculation regions, especially during diastole. Out of the two endovascular techniques, the hybrid procedure was found preferred from hemodynamics point of view, with less disturbed and recirculating regions. Although the chimney procedure requires less manufacturing times and cost, it is associated with higher risks rate, and therefore, it is recommended only for emergency cases. This study may shade light on the hemodynamic factors for these complications, and provide insights on ways to improve the procedure.
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de Lima, Edirlei Soares, Bruno Feijo, and Antonio L. Furtado. "Adaptive Branching Quests Based on Automated Planning and Story Arcs." In 2021 20th Brazilian Symposium on Computer Games and Digital Entertainment (SBGames). IEEE, 2021. http://dx.doi.org/10.1109/sbgames54170.2021.00012.

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Carr, Cornelia S., Nazar Mohammed, Lama Shuayb, Maryam Alkuwari, and Abdulaziz M. Alkhulaifi. "Comparison of Aortic Arch Branching Patterns between Bicuspid and Tricuspid Aortic Valve Patients in Qatar." In Qatar Foundation Annual Research Conference Proceedings. Hamad bin Khalifa University Press (HBKU Press), 2016. http://dx.doi.org/10.5339/qfarc.2016.hbpp1806.

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Thibodeaux, Jennifer, Ronnie W. Kisor, Jacob M. King, and Charles E. Taylor. "Flow Control Device for Branching Arteries of the Aortic Arch in a Mock Circulatory Loop." In 2016 32nd Southern Biomedical Engineering Conference (SBEC). IEEE, 2016. http://dx.doi.org/10.1109/sbec.2016.51.

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Kazakidi, Asimina, Marzena Wylezinska, Yvette Bohraus, et al. "Numerical Modelling of Blood Flow in the Mouse Aortic Arch Using Inflow Velocities Obtained by Phase-Contrast MRI." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19270.

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Atherosclerotic lesions have a highly non-uniform distribution in regions of arterial branching and curvature, consistent with hemodynamic factors, in particular wall shear stress (WSS), controlling their development. The widespread and increasing use of the mouse as a model for studying atherosclerosis has encouraged investigation of the hemodynamics of the mouse aortic arch [1–3], in which previous studies have revealed areas of high and low lesion prevalence and variation in the expression of pro-atherogenic molecules [4]. Our previous computational simulations [1–2] did not produce distributions of WSS that explain the pattern of lesions. We are currently investigating whether incorporation of more realistic aortic root velocity measurements, obtained using phase-contrast magnetic resonance imaging (PC-MRI), into these simulations can improve the correlation with disease. Here we present velocities obtained by PC-MRI and preliminary simulations employing the data.
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Chen, Chia-Yuan, Michael J. Patrick, Paola Corti, David Frakes, Beth L. Roman, and Kerem Pekkan. "In Vivo Hemodynamic Performance of Wild-Type vs. Mutant Zebrafish Embryos Using High-Speed Confocal Micro-PIV." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19317.

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In developing cardiovascular systems, definite performance comparison between disease and healthy hemodynamics requires quantitative tools to support advanced microscopy. Mutations in the activin receptor-like kinase 1 (ALK1) gene are responsible for the autosomal dominant vascular disease, hereditary hemorrhagic telangiectasia type 2 (HHT2), characterized by high flow arteriovenous malformations (AVMs) [1]. Recent studies show that the zebrafish mutant violet beauregrade (vbg), which harbors a mutation in alk1, develops an abnormal circulation with dilated cranial vessels and AVMs [2]. Quantitative understanding of mechanical influences on the alk1 mutant phenotype will aid treatment of HHT2 patients. Inspired by earlier studies that demonstrate the capability of using confocal micro-PIV technique to quantify biofluid dynamics in vivo [3], primarily in major vessels (dorsal aorta, vitelline veins), the present study focused on secondary branching great vessels of zebrafish embryos where microcirculation flow regimes are different. Furthermore, confocal microscopy, essentially being an imaging modality, requires rigorous validation efforts with respect to the gold standard measurement protocols (such as PIV) and synthetic scan data. Another objective of this work was to document the intra-species differences of wall shear stress (WSS) and flow physics during embryonic development in aortic arch systems of zebrafish [4].
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Bressan, Robert da Silva, and Danilo Artigas. "Task Scheduling for Subsea Flexible Pipes Decommissioning." In Offshore Technology Conference. OTC, 2021. http://dx.doi.org/10.4043/31066-ms.

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Abstract Subsea flexible pipelines removal is subject to order restrictions, mostly caused by crossings. It is proposed to create a computational algorithm to design an optimal order of vessel intervention over a field. A real field was studied, and, from it, the mathematical base model was created upon graph theory, with great correlation with the minimum feedback arc set problem. Vessel movements were discretized and reduced to removal, reposition, and cut, leading to a state search. A-star algorithm was implemented to guide the search for the solution. Then, the complete algorithm was built, tested in a minimal environment, and finally applied to the real instance. To improve performance, a beam search filtering was envisioned, using seven ranking functions. Constructed model is suspected to be NP-hard, by correlation to minimum feedback arc set problem, leading to a large space search. Instances containing under 100 crossings were solved optimally, without needing any assistance. After implementing the heuristics and beam search, solution time was lowered by about 20 times, demonstrating the effectiveness of the technique. Also, ranking functions for pipe repositioning based on crossing count led to better results than crossing density. For cutting, an approximation based on feedback arc set was used. GreedyFAS was employed and gave satisfactory results. Bigger instances containing around 3000 crossings could not be solved optimally in a reasonable time, even with the heuristics. Improvements in A-star estimation function and bound the solution branches might lead to an optimal solution for these larger instances. Model proposed simplifies the operational order decisions and helps build the scheduling of operations. As it is based on state search, other aspects in logistics, vessel capacities and steps in decommissioning processes may be added, adjusting the neighboring weights and branching, keeping the same core.
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Teshima, Hiromasa, Kohei Kojima, and Yang Ju. "Fabrication of Anodic Aluminum Oxide Template and Cu Nanowire Surface Fastener." In ASME 2013 International Technical Conference and Exhibition on Packaging and Integration of Electronic and Photonic Microsystems. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/ipack2013-73125.

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There is an urgent need in surface mount technology (SMT) for a nontoxic, reusable and low temperature bonding technique which can afford good mechanical support as well as electrical contact. Meanwhile in the nanotechnology, many excellent and unique structure-related properties such as the high mechanical strength, the high conductivity and the adhesion ability of gecko feet have been studied. Our lab proposes a new patterned structure of Au nanowire array named nanowire surface fastener (NSF), which cold bonding for surface mount technology can be realized at room temperature. Then various methods have been developed to fabricate nanowire, such as arc discharge, catalytic CVD growth and template synthesis, and so on. Among these methods, the template method has been widely used for preparing one-dimensional nanostructures such as metals, semiconductors, polymers, and other materials by electrochemical, electroless deposition or sol-gel technique. Especially anodic aluminum oxide template assisted way has attached considerable attention due to its unique structure properties, such as controllable pore diameter, extremely narrow pore size distribution with high densities, high aspect ratios, and ideally cylindrical pore shape. The well arranged porous anodic aluminum oxide membrane is fabricated from aluminum film by two steps zM oxalic acid electrolytes. The anodic aluminum oxide membrane was investigated for features such as pore size, interpore distance, and thickness by 40 V. It is important for fabrication of porous anodic aluminum oxide template to find out elimination of the barrier layer of oxide and the pore extending rate by 0.5 M phosphoric acid. Morphologies of surface of aluminum film between anodization process and the anodic aluminum oxide barrier layer was researched by using atomic force microscope and scanning electron microscope. Results showed that the anodic aluminum oxide having the same diameter of the pore and the well arranged pore array without branching channel was obtained. The diameter of the pore before the pore extending treatment is 42 nm and the diameter of the pore after the pore extending treatment for 30 minutes is 86 nm. It was found that the diameter of the pore increased per 15 nm by the pore extending treatment for 10 minutes. We fabricated the through-hole anodic aluminum oxide template and made Cu nanowire by the template of our own making. By using Cu nanowire, we try to produce nanowire surface fastener and evaluate its properties.
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Reports on the topic "Branchial arch"

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Bongsebandhu-phubhakdi, Saknan, and Anan Srikiatkhachorn. On-media axon branching and adhesion investigation of neurons as stimulated by modulated potentials on micro-patterned gold substrate. Faculty of Medicine, Chulalongkorn University, 2016. https://doi.org/10.58837/chula.res.2016.22.

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The main focus of this research paper is on-media axon branching and adhesion investigation of neurons as stimulated by modulated potentials on micro-patterned gold substrate. Due to the prolonged and inefficient procedures of nerve repair, it is essential that we effectively incorporate different parameters and techniques as well as investigate cell-cell and cell-substrate interactions to explore new boundaries. This could lead to more operational options for nerve regeneration. Initially, the behavior of cell growth is first observed. 3T3 and Neuro2A cells are grown according to specific protocols allowing the observations of appropriate parameters needed to optimize the cells' development and proliferation. After thorough examination, the two cell subjects will be grown on patterned and non-patterned gold-coated substrates. Previously, "Cathode Arc Sputtering" and "Magnetron Sputtering" techniques are used to coat gold particles on polystyrene substrates and distributions of the thin films are then analyzed. Different patterning techniques, such as "Stencil Patterning" and "Microcontact Printing" are then applied to create a number of patterns on the substrates. Furthermore, 3D patterns will be induced by electrical potentials to generate magnetic fields near neurons. Various structured patterns as well as the overall shapes of the magnetic fields are speculated to have different effects on neural behaviors. Thus, cell-substrate adhesion interactions, manipulation of neuronal growth and proliferation using electrical potentials will be explored on pure gold substrates in this research. Specifically, the ambition of this research is to contribute to the development of neuron circuits that will allow more efficient procedures for nerve repair. This research's greatest hope is not only to provide current developments with extensive data for further improvements, but also to comprehend better the constraints restraining the breakthroughs of novel technologies.
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