Relevant bibliographies by topics / Breast Cancer 25(OH)D Vitamin D

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Academic literature on the topic 'Breast Cancer 25(OH)D Vitamin D'

Author: Grafiati
Published: 7 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Breast Cancer 25(OH)D Vitamin D"

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Anak, Agung Ayu Ratih Dharmaiswari, and Kadek Sumantara Artha I. "Effectivity of Vitamin D in Preventing Breast Cancer and Evaluation of 25(OH)D Examination As A Laboratory Screening Tool." International Journal of Medical Science and Clinical Research Studies 5, no. 05 (2025): 696–701. https://doi.org/10.5281/zenodo.15347969.

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Vitamin D is currently associated with many diseases, not only bone health but also in autoimmune diseases, cancer, and neurological disorders. This study focuses on the mechanism relation vitamin D on breast cancer, especially with regard to estrogen receptor-positive (ER+) and human epidermal growth factor receptor 2-positive (HER 2+) cancers. Therapeutic supplementation is a commonly used approach to increase vitamin D levels. The effects of vitamin D have been investigated in various aspects, including regulation of cell growth and differentiation, apoptosis, intercellular contact, angioge
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Damayanti, Amilia Yuni, Dono Indarto, Brian Wasita, and Tonang Dwi Ardyanto. "Indeks massa tubuh, asupan vitamin D, dan serum 25-hydroxyvitamin D pada pasien kanker payudara." Jurnal Gizi Klinik Indonesia 14, no. 2 (2017): 56. http://dx.doi.org/10.22146/ijcn.17243.

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Background: Breast cancer is the most common malignancy in women in the world. Vitamin D helps control the majority of gene expression in female reproductive tissues. Body mass index (BMI) influences the bioavailability of vitamin D. Diet is one of the main sources of vitamin D and it is directly converted into 25-hydroxyvitamin D [25 (OH) D] in the liver.Objective: This study aimed to analyze the relationship between BMI and vitamin D intake and 25 (OH)D serum in patients with breast cancer.Method: Analytic observational with cross sectional design was used in this study. A total of 37 breast
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Jauhari, A. H. Asmali, N. Bhoo-Pathy, T. Islam, M. Y. Jalaludin, F. M. Moy, and N. A. Taib. "Serum 25(OH) Vitamin D Deficiency and Risk of Breast Cancer in Malaysia: A Case-Control Study." Journal of Global Oncology 4, Supplement 2 (2018): 23s. http://dx.doi.org/10.1200/jgo.18.64100.

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Background: Despite the emerging literature supporting the beneficial role of sufficient vitamin D level and its association with various cancers, current evidence on the vitamin D and the risk of getting breast cancer is still inconsistent. Furthermore, the study between vitamin D status and breast cancer risk among south east Asian women is limited. Aim: We aimed to investigate the association between vitamin D deficiency and the risk of getting breast cancer among Malaysian women. Methods: We conducted a retrospective nested case control study which consisted of Malaysian Breast Cancer Coho
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Zemlin, Cosima, Laura Altmayer, Caroline Stuhlert, et al. "Prevalence and Relevance of Vitamin D Deficiency in Newly Diagnosed Breast Cancer Patients: A Pilot Study." Nutrients 15, no. 6 (2023): 1450. http://dx.doi.org/10.3390/nu15061450.

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(1) Background: Vitamin D plays an important role in many types of cancer. It was the aim of this study to analyze serum 25-hydroxyvitamin D (25(OH)D) levels in newly diagnosed breast cancer patients, and the association with prognostic and lifestyle factors. (2) Methods: 110 non-metastatic breast cancer patients were included in the prospective observational “BEGYN” study at Saarland University Medical Center between September 2019 and January 2021. At the initiation visit, serum 25(OH)D levels were measured. Clinicopathological data on prognosis, nutrition, and lifestyle were extracted from
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Crew, Katherine D. "Vitamin D: Are We Ready to Supplement for Breast Cancer Prevention and Treatment?" ISRN Oncology 2013 (February 26, 2013): 1–22. http://dx.doi.org/10.1155/2013/483687.

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Vitamin D deficiency is a potentially modifiable risk factor that may be targeted for breast cancer prevention and treatment. Preclinical studies support various antitumor effects of vitamin D in breast cancer. Numerous observational studies have reported an inverse association between vitamin D status, including circulating 25-hydroxyvitamin D (25(OH)D) levels, and breast cancer risk. The relationship between vitamin D and mammographic density, a strong predictor of breast cancer risk, remains unclear. Studies analyzing the link between genetic polymorphisms in vitamin D pathway genes and bre
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Ilyan, Theresia, Dwi Retnoningrum, Meita Hendrianingtyas, Dian Widyaningrum, and Banundari Rachmawati. "Association of 25-hydroxyvitamin D, Cyclooxygenase-2 and Prostaglandin E2 Serum Levels in Breast Cancer Patients." Indonesian Biomedical Journal 13, no. 4 (2021): 426–32. http://dx.doi.org/10.18585/inabj.v13i4.1684.

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BACKGROUND: Serum levels of 25-hydroxyvitamin D (25(OH)D), prostaglandin E2 (PGE2), and cyclooxygenase 2 (COX2) expression differ between breast cancer stages. Since, previous studies showed mixed results, in this study, we aimed to analyze vitamin D levels related to breast cancer stages and serum levels of COX2 and PGE2 in Indonesia.METHODS: This was a cross sectional study involving 75 breast cancer patients. Subjects were divided into 3 groups, namely operable early stage (K1), locally advanced stage (K2), and advanced stage (K3). Venous blood samples were taken from each subject, then wer
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Radom, Agnieszka, Andrzej Wędrychowicz, Stanisław Pieczarkowski, Szymon Skoczeń, and Przemysław Tomasik. "Effect of Selected Factors on the Serum 25(OH)D Concentration in Women Treated for Breast Cancer." Nutrients 13, no. 2 (2021): 564. http://dx.doi.org/10.3390/nu13020564.

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Maintaining an optimal vitamin D concentration reduces the risk of recurrence and extends survival time in patients after breast cancer treatment. Data on vitamin D deficiency among Polish women after breast cancer therapy are limited. Thus, the aim of the study was the analysis of vitamin D status in post-mastectomy patients, considering such factors as seasons, social habits, vitamin D supplementation and its measurements. The study involved 94 women after breast cancer treatment. Serum vitamin D concentration was measured, and a questionnaire, gathering demographic and clinical data regardi
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Laney, Nathan, Jane Meza, Elizabeth Lyden, Judi Erickson, Kelly Treude, and Whitney Goldner. "The Prevalence of Vitamin D Deficiency Is Similar between Thyroid Nodule and Thyroid Cancer Patients." International Journal of Endocrinology 2010 (2010): 1–7. http://dx.doi.org/10.1155/2010/805716.

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Introduction. There are reported associations between vitamin D deficiency and breast, prostate, and colon cancer, but the relationship in thyroid cancer has not been evaluated.Methods. We evaluated serum calcium, creatinine, albumin, and 25-hydroxy vitamin D (25-OH-D) in 42 thyroid nodule, 45 thyroid cancer in remission, and 24 active thyroid cancer patients.Results. 25-OH-D was not different between groups. The percent with 25-OH-D levels<75 nmol/L was not significantly different between groups and was not affected by season of measurement, age, or cancer stage. Multivariate regression sh
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Butt, Maleeha Tahir. "Relation of Vitamin D Deficiency with Breast Cancer Staging in Patients from INMOL Cancer Hospital." Proceedings of Shaikh Zayed Medical Complex Lahore 35, no. 1 (2021): 27–32. http://dx.doi.org/10.47489/p000s351z7801-6mc.

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Introduction: Vitamin D deficiency can be critical and is reported to be as one of the factors for development and progression of breast carcinoma. Aims & Objectives: To measure and compare 25(OH)D (calcidiol) levels in healthy females and in breast carcinoma patients in four different stages. Place and duration of study: This cross sectional study and research was conducted in Shaikh Zayed Medical Complex in the Department of Biochemistry and Chemical Pathology from Jan 2018 to Jan 2019. The patients were selected from INMOL Cancer Hospital, Lahore. Material & Methods: The study was c
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Al-Rubiae, Sara Younis Mahmoud, and Salwa Hazim Almukhtar. "Association between Vitamin D levels and Breast Cancer at Mosul Nuclear Oncology Hospital: Case - Control study." Pakistan Journal of Medical and Health Sciences 15, no. 9 (2021): 2741–44. http://dx.doi.org/10.53350/pjmhs211592741.

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Breast tumor is the most common type of cancer found in women. The issue of whether levels of vitamin D are associated with an increased risk of breast cancer has been brought up. The study's goals are to learn whether low levels of vitamin D are linked to a greater risk of breast cancer. A case-control study was conducted in Mosul Nuclear Oncology Hospital to see how blood vitamin D concentrations, vitamin D supplementation, and sunlight exposure relate to breast cancer among women in Mosul. There were a total of 66 newly diagnosed histologically confirmed primary breast cancer cases among th
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Dissertations / Theses on the topic "Breast Cancer 25(OH)D Vitamin D"

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Verma, Anjali. "ERalpha isoforms modulate the tumorigenicity of 24R,25(OH)2D3 in estrogen-responsive cancer." VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/6016.

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Over 200,000 cases of breast cancer are diagnosed every year. Nearly 20% of these patients supplement their diets with some form of vitamin D. This high frequency of vitamin D supplement use may be due in part to research suggesting that cancer patients with higher serum vitamin D3 levels have better prognoses than patients with low serum vitamin D3. However, double-blind clinical trials on the efficacy of vitamin D3 supplementation in breast cancer have been inconclusive. A recent meta-analysis showed evidence of reduced cancer recurrence in patients taking vitamin D3 supplements who had ‘est
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Tommie, Jessica. "Serum Vitamin D Status and Breast Cancer Risk by Receptor Status: A Systematic Review." University of Cincinnati / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin150480332518945.

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Yousef, Fatimah Mohammadali. "Vitamin D Status and Breast Cancer in Saudi Arabian Women: Case Control Study." Diss., The University of Arizona, 2011. http://hdl.handle.net/10150/202986.

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Vitamin D is an essential nutrient in the human diet. A unique property of vitamin D is that it can be produced by endogenous synthesis in the skin following sufficient Ultraviolet B (UVB) radiation. In fact, our understanding of this compound has changed, such that it is no longer consider a true vitamin, but rather a steroid hormone. De-identified data for this analysis were derived from women residing in Jeddah, Saudi Arabia who completed routine medical visits in the summer of 2009 at King Fahad Hospital (KFH). In Chapter 1,“THE ASSOCIATION BETWEEN VITAMIN D STATUS IN NORMAL WEIGHT VERSUS
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Stahel, Anette. "24,25(OH)2D3 and Regulation of Catalase Activity in LNCaP Prostate Cancer." Thesis, University of Skövde, School of Life Sciences, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-475.

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<p>The vitamin D metabolite 1,25(OH)2D3 has long been known to inhibit growth of prostate cancer cells and this has been attributed to a VDR-mediated pathway controlling target gene expression, resulting in cell cycle arrest, apoptosis and differentiation. New research has shown that another vitamin D metabolite, 24,25(OH)2D3, inhibits proliferation of prostate cancer cells as well, more specifically, cells of the line LNCaP. It is not clear exactly how 24,25(OH)2D3 exerts this cancer growth inhibition but it has been shown that it is to some extent regulated via G protein coupled signalling p
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Stahel, Anette. "Effects of 1,25(OH)2D3 on Smad2 Activity in PC-3 Prostate Cancer Cells." Thesis, University of Skövde, School of Life Sciences, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-3807.

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<p>The vitamin D metabolite 1,25(OH)2D3 has long been known to inhibit growth of prostate cancer cells and this mainly through a VDR-mediated pathway controlling target gene expression, resulting in cell cycle arrest, apoptosis and differentiation. Another major way inwhich 1,25(OH)2D3 inhibits cell growth in prostate cancer is via membrane-initiated steroid signalling, which triggers activation of signal cascades upon steroid binding to a receptor complex, leading to induction of genes regulating cell growth, proliferation and apoptosis. The main prostate cancer inhibiting membrane-initiated
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Stahel, Anette. "1,25(OH)2D3 Initially Reduces TGFβ Activity in PC-3 Prostate Cancer Cells". Thesis, University of Skövde, School of Life Sciences, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-1336.

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<p>The vitamin D metabolite 1,25(OH)2D3 has long been known to inhibit growth of prostate cancer cells and this mainly through a VDR-mediated pathway controlling target gene expression, resulting in cell cycle arrest, apoptosis and differentiation. Another major way in which 1,25(OH)2D3 inhibits cell growth in prostate cancer is via membrane-initiated steroid signalling, which triggers activation of signal cascades upon steroid binding to a receptor complex, leading to induction of genes regulating cell growth, proliferation and apoptosis. The main prostate cancer inhibiting membrane-initiated
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Stahel, Anette. "1,25(OH)2D3 and Initial Regulation of Smad2/3 Activity in PC-3 Prostate Cancer Cells." Thesis, University of Skövde, School of Life Sciences, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-3806.

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<p>The vitamin D metabolite 1,25(OH)2D3 has long been known to inhibit growth of prostate cancer cells and this mainly through a VDR-mediated pathway controlling target gene expression, resulting in cell cycle arrest, apoptosis and differentiation. Another major way in which 1,25(OH)2D3 inhibits cell growth in prostate cancer is via membrane-initiated steroid signalling, which triggers activation of signal cascades upon steroid binding to a receptor complex, leading to induction of genes regulating cell growth, proliferation and apoptosis. The main prostate cancer inhibiting membrane-initiated
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Stahel, Anette. "24,25(OH)2D3 and Regulation of Catalase Activity in LNCaP Prostate Cancer Cells : A Study of Long-term Effects." Thesis, University of Skövde, School of Life Sciences, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-970.

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<p>The vitamin D metabolite 1,25(OH)2D3 has long been known to inhibit growth of prostate cancer cells and this has been attributed to a VDR-mediated pathway controlling target gene expression, resulting in cell cycle arrest, apoptosis and differentiation. New research has shown that another vitamin D metabolite, 24,25(OH)2D3, inhibits proliferation of prostate cancer cells as well, more specifically, cells of the line LNCaP. It is not clear exactly how 24,25(OH)2D3 exerts this cancer growth inhibition but it has been shown that it is to some extent regulated via G protein coupled signalling p
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Broadfield, Lindsay. "Combined Effects of N-3 Polyunsaturated Fatty Acids and 1alpha, 25-dihydroxyvitamin D on Breast Cancer Cell Growth." Thesis, 2013. http://hdl.handle.net/10214/7413.

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Omega-3 polyunsaturated fatty acids (PUFA) and vitamin D both have anti-cancer effects through common and unique pathways. The hypothesis of this thesis is that the combination of n-3 PUFA with 1,25(OH)2D3 will inhibit breast cancer cell growth in an additive or synergistic manner. A 3X3 factorial design was used to test the combinations of five PUFA treatments (α-linoleic acid (ALA, 18:3n3), eicosapentaenoic acid (EPA, 20:5n3) and docosahexaenoic acid (DHA, 22:6n3), γ-linolenic acid (GLA, 18:3n6) and arachidonic acid (AA, 20:4n6)) with 1,25(OH)2D3 on MCF-7, MDA-MB-231, and MCF-10A cell growth
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Book chapters on the topic "Breast Cancer 25(OH)D Vitamin D"

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"A NOVEL VITAMIN D3 ANALOGUE, 22-0XA-1 a, 25-DIHYDROXYVITAMIN D3, INHIBITS THE GROWTH OF HUMAN BREAST CANCER WITHOUT CAUSING HYPERCALCEMIA." In Vitamin D. De Gruyter, 1991. http://dx.doi.org/10.1515/9783110850345-143.

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Conference papers on the topic "Breast Cancer 25(OH)D Vitamin D"

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Sukik, Layan, Bushra Hoque, Linda Boutefnouchet, and Vijay Ganji. "Serum vitamin D concentrations are non-linearly related to breast cancer risk in postmenopausal women." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0138.

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Background: Post-menopausal women are at high risk for breast cancer. The association between serum 25-hydroxyvitamin D [25(OH)D] concentration and breast cancer in post-menopausal women is not well understood. Objectives: The aim of this study was to investigate the association between serum 25(OH)D and breast cancer using nationally representative sample surveys. Methodology: In this cross-sectional study, we used seven cycles of National Health and Nutrition Examination Surveys from 2001 through 2014. Participants were non-institutionalized post-menopausal women (n=8100). Logistic regressio
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Almeida Filho, Benedito de Sousa, Michelle Sako Omodei, Eduardo Carvalho Pessoa, Heloisa de Luca Vespoli, and Eliana Aguiar Petri Nahas. "NEGATIVE IMPACT OF SERUM VITAMIN D DEFICIENCY ON BREAST CANCER SURVIVAL." In XXIV Congresso Brasileiro de Mastologia. Mastology, 2022. http://dx.doi.org/10.29289/259453942022v32s1058.

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Introduction: It is known that breast cancer is the type of cancer that mostly affects women in the world, both in the developing and developed countries, with about 2.3 million new cases in 2020, comprising 25% of all cancers diagnosed in women. Vitamin D concentration has been studied as a risk and prognostic factor in women with breast cancer; its deficiency is common in women with postmenopausal breast cancer, and some evidence suggests that low vitamin D status increases the risk for disease development. The impact of vitamin D at the time of diagnosis on the outcome of patients with brea
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Omodei, Michelle Sako, Eduardo Carvalho Pessoa, Daniel Buttros, et al. "Effect of vitamin D supplementation on the pathological complete response to neoadjuvant chemotherapy in women with breast cancer: a randomized clinical trial." In Brazilian Breast Cancer Symposium 2024. Mastology, 2024. http://dx.doi.org/10.29289/259453942024v34s1002.

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Objective: The objective of this study was to evaluate the effect of vitamin D (VD) supplementation on the pathological complete response (pCR) rate in women with breast cancer (BC) undergoing neoadjuvant chemotherapy (NCT). Methodology: A randomized clinical trial was conducted on 80 women aged ≥45 years with a histological diagnosis of BC, who were eligible for NCT. Women were randomized to one of two groups that received either daily supplementation with 2,000 IU of cholecalciferol (VD, n=40) or placebo (n=40) during the chemotherapy period (6 months). The primary outcome measure was pCR. S
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Serrano, Davide, Harriet Johansson, Debora Macis, et al. "Abstract 167: A comparison of 25-OH-vitamin D levels and VDR polymorphisms in healthy and patients with breast intraepithelial neoplasia or cancer ." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-167.

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Ulrich, Cornelia M., Erin M. Siegel, Hermann Brenner, et al. "Abstract A82: 25(OH) vitamin D, folate, and vitamin B12biomarkers among international colorectal cancer patients." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Nov 7-10, 2010; Philadelphia, PA. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-10-a82.

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Cooney, Robert V., Weiwen Chai, Adrian A. Franke, et al. "Abstract 4821: Plasma retinol, but not 25-OH vitamin D or vitamin E, is associated with increased survival in colorectal cancer patients." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4821.

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Fabian, CJ, BF Kimler, T. Phillips, and CM Zalles. "Levels of 25-hydroxy-vitamin D in pre-menopausal women at high risk for development of breast cancer." In CTRC-AACR San Antonio Breast Cancer Symposium: 2008 Abstracts. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-5079.

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Abnet, Christian C., Yu Chen, Wong-Ho Chow, et al. "Abstract 2803: A pooled analysis of circulating 25(OH)D and upper gastrointestinal cancer risk in the Cohort Consortium Vitamin D Pooling Project of Rarer Cancers." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2803.

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Jacot, W., N. Firmin, L. Roca, et al. "Abstract P6-09-10: Impact of a tailored oral vitamin D supplementation regimen on serum 25-hydroxy vitamin D levels in early breast cancer patients: A randomized phase III study." In Abstracts: Thirty-Sixth Annual CTRC-AACR San Antonio Breast Cancer Symposium - Dec 10-14, 2013; San Antonio, TX. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/0008-5472.sabcs13-p6-09-10.

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Reimers, Laura L., Dawn L. Hershman, Matthew Maurer, et al. "Abstract A48: Pitfalls of using breast magnetic resonance imaging (MRI) in an early-phase breast cancer chemoprevention trial of vitamin D among high-risk postmenopausal women." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-a48.

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