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1

Chalmers, James, Eva Polverino, and Stefano Aliberti, eds. Bronchiectasis. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-61452-6.

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2

Chalmers, James D., Eva Polverino, and Stefano Aliberti, eds. Bronchiectasis. European Respiratory Society, 2018. http://dx.doi.org/10.1183/2312508x.erm8118.

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3

Dragicevic, Peter. Bacteriology and beta lactamase activity in bronchiectasis and their relationship to chemotherapy. University of Birmingham, 1989.

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4

Clarke, Elisa Margaret. The role of T lymphocyte mediated immunity in the pathogenesis of bronchiectasis. University of Birmingham, 1999.

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5

Mitchell, Joanne Lindsay. The immune response to non-typeable haemophilus influenzae in patients with bronchiectasis. University of Birmingham, 1997.

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6

Newall, Clare. The effect of pulmonary rehabilitation and inspiratory muscle training in patients with chronic obstructive pulmonary disease and bronchiectasis. University of Birmingham, 2000.

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7

Tino, Gregory. Bronchiectasis. Informa Healthcare, 2006.

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8

Bronchiectasis. W.B. Saunders Company, 2012.

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9

Floto, R. A., and C. S. Haworth, eds. Bronchiectasis. European Respiratory Society, 2011. http://dx.doi.org/10.1183/1025448x.erm5210.

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10

Jones, Andrew M., and Rowland J. Bright-Thomas. Bronchiectasis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0137.

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Bronchiectasis may be defined clinically as the chronic daily production of copious mucopurulent sputum. Pathologically, the disease is characterized by inflamed, thick-walled, dilated bronchi. Bronchiectasis has many potential underlying causes and associations with other diseases but in individual cases the underlying cause is often unknown. The typical symptom is cough with sputum. Bronchiectasis is a chronic condition; systemic complications are common and include tiredness, malaise, and weight loss. The aim of therapy should be to control symptoms, prevent complications and disease progression, while minimizing treatment burden. This chapter discusses bronchiectasis, including its etiology, symptoms, demographics, natural history, complications, diagnosis, prognosis, and treatment.
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11

Chalmers, James, Eva Polverino, and Stefano Aliberti. Bronchiectasis: The EMBARC Manual. Springer, 2019.

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12

Bronchiectasis terminating in pneumonia, simulating pulmonary and laryngeal tuberculosis. s.n., 1985.

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13

Publications, ICON Health. Bronchiectasis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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14

Carton, James. Lung pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0005.

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This chapter discusses lung pathology and covers respiratory malformations, respiratory failure, acute respiratory distress syndrome (ARDS), bronchiectasis, cystic fibrosis, pulmonary thromboembolism, pulmonary hypertension, asthma, chronic obstructive pulmonary disease (COPD), bacterial pneumonia, idiopathic pulmonary fibrosis (IPF), hypersensitivity pneumonitis, lung carcinoma, pleural effusion, pneumothorax, and malignant mesothelioma.
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15

Török, M. Estée, Fiona J. Cooke, and Ed Moran. Respiratory, head, and neck infections. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199671328.003.0014.

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This chapter covers the common cold, pharyngitis, retropharyngeal abscess, quinsy (peritonsillar abscess), Lemierre’s disease, croup, epiglottitis, bacterial tracheitis, laryngitis, sinusitis, mastoiditis, otitis externa, otitis media, dental infections, lateral pharyngeal abscess, acute bronchitis, chronic bronchitis, bronchiolitis, community-acquired pneumonia, aspiration pneumonia, hospital-acquired pneumonia, ventilator-associated pneumonia, pulmonary infiltrates with eosinophilia, empyema, lung abscess, cystic fibrosis, bronchiectasis, and pulmonary tuberculosis.
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16

N, Caine, National Co-ordinating Centre for HTA (Great Britain), and Health Technology Assessment Programme, eds. A randomised controlled crossover trial of nurse practitioner versus doctor-led outpatient care in a bronchiectasis clinic. NCCHTA, 2002.

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17

Bright-Thomas, Rowland J., and Andrew M. Jones. Cystic fibrosis. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0132.

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Cystic fibrosis is the most common lethal autosomal recessive disorder in Caucasians. There is no known survival advantage of the heterozygote carrier state. Chronic progressive pulmonary infection and bronchiectasis are the major causes of morbidity and mortality. The disease affects all ductal systems where the basic defect is manifest, including the pancreas, gastrointestinal tract, sinuses, hepatobiliary system, and male reproductive system, and has significant effects on nutrition and growth.
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18

Millar, Professor Ann B., Dr Richard Leach, Dr Rebecca Preston, et al. Respiratory diseases and respiratory failure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.0005.

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Chapter 5 covers respiratory diseases and respiratory failure, including clinical presentations of respiratory disease, assessment of diffuse lung disease, hypoxaemia, respiratory failure, and oxygen therapy, pneumonia, mycobacterial infection, asthma, chronic obstructive pulmonary disease (COPD), lung cancer, mediastinal lesions, pneumothorax, pleural disease, asbestos-related lung disease, diffuse parenchymal (interstitial) lung disease, sarcoidosis, pulmonary hypertension, acute respiratory distress syndrome, bronchiectasis and cystic fibrosis, bronchiolitis, eosinophilic lung disease, airways obstruction, aspiration syndromes, and near-drowning, pulmonary vasculitis, the immunocompromised host, sleep apnoea, and rare pulmonary diseases.
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19

Lawrence, T. P., and R. S. C. Kerr. Intracranial abscesses. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199204854.003.0241103.

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The incidence of intracranial abscess is low in countries where antimicrobial treatment for infections is widespread (e.g. 2–3 per million in the United Kingdom), but they remain frequent causes of space-occupying masses in developing countries.Aetiology—abscesses may be classified by (1) Route of transmission, including (a) direct—from a local source of infection, e.g. otitis media; (b) haematogenous—from a distant source, e.g. endocarditis, bronchiectasis, other septic lung conditions; or (c) following cranial surgery or fracture. (2) Microbiology—the commonest organisms are aerobic, anaerobic and micro-aerophilic streptococci, ...
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20

Herbert, Lara, and Bruce McCormick. Respiratory disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198719410.003.0005.

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This chapter describes the anaesthetic management of the patient with respiratory disease. It describes the assessment of respiratory function and preoperative respiratory investigations, and ventilatory strategies to reduce pulmonary complications. Common respiratory conditions covered include respiratory tract infection, smoking, asthma, chronic obstructive pulmonary disease, bronchiectasis, cystic fibrosis, obstructive sleep apnoea, sarcoidosis, restrictive pulmonary disease, and the patient with a transplanted lung. For each topic, preoperative investigation and optimization, treatment, and anaesthetic management are described. Recommendations for the patient who may require post-operative respiratory support (e.g. non-invasive ventilation) are provided.
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21

Herbert, Lara, and Bruce McCormick. Respiratory disease. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198719410.003.0005_update_001.

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This chapter describes the anaesthetic management of the patient with respiratory disease. It describes the assessment of respiratory function and preoperative respiratory investigations, and ventilatory strategies to reduce pulmonary complications. Common respiratory conditions covered include respiratory tract infection, smoking, asthma, chronic obstructive pulmonary disease, bronchiectasis, cystic fibrosis, obstructive sleep apnoea, sarcoidosis, restrictive pulmonary disease, and the patient with a transplanted lung. For each topic, preoperative investigation and optimization, treatment, and anaesthetic management are described. Recommendations for the patient who may require post-operative respiratory support (e.g. non-invasive ventilation) are provided.
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22

Jolly, Elaine, Andrew Fry, and Afzal Chaudhry, eds. Respiratory medicine. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199230457.003.0018.

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Chapter 18 covers the basic science and clinical topics relating to respiratory medicine which trainees are required to learn as part of their basic training and demonstrate in the MRCP. The chapter starts with an introduction to the respiratory system, before covering respiratory defence and physiology, respiratory investigations, respiratory failure, pneumonia, tuberculosis, cystic fibrosis, bronchiectasis, pleural effusion, chronic obstructive pulmonary Disease, adult respiratory distress syndrome, asthma , fungal lung diseases, pulmonary embolism , lung cancer, pulmonary fibrosis, extrinsic allergic alveolitis, occupational lung diseases, sarcoidosis, Cor pulmonale and pulmonary hypertension, pneumothorax, cough and haemoptysis, pulmonary eosinophilia, and obstructive sleep apnoea.
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23

Kreit, John W. Severe Obstructive Lung Disease. Edited by John W. Kreit. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190670085.003.0013.

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Although chronic obstructive lung disease, asthma, bronchiectasis, and bronchiolitis have very different causes, clinical features, and therapies, they share the same underlying pathophysiology. They are referred to as obstructive lung diseases because airway narrowing causes increased resistance and slowing of expiratory gas flow. Mechanical ventilation of patients with severe obstructive lung disease often produces two problems that must be recognized and effectively managed: over-ventilation and dynamic hyperinflation. Severe Obstructive Lung Disease reviews these two major adverse consequences of mechanical ventilation in patients with severe air flow obstruction. The chapter explains how to detect and correct both of these problems and provides guidelines for managing patients with respiratory failure caused by severe obstructive lung disease.
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24

Snell, Jamey, and Thomas J. Mancuso. Cystic Fibrosis. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0023.

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Cystic fibrosis (CF) is an inherited, autosomal recessive, multisystem disease. Dysfunction of the cystic fibrosis transmembrane conductance regulator protein (CFTR) in epithelial cells is the primary defect in CF. Defects in CFTR are the cause for lung disease, exocrine pancreatic insufficiency and failure, male infertility, and liver disease. CF can present with a variety of respiratory and gastrointestinal signs, including meconium ileus in the newborn period, hypernatremic dehydration, pulmonary insufficiency, nasal polyps, and insulin-dependent diabetes mellitus. As affected children grow, dysfunction in CFTR leads to chronic and progressive lung disease, characterized by suppurative infection and the development of bronchiectasis. CFTR dysfunction also affects exocrine function, leading to pancreatic insufficiency, malabsorption, and growth failure. In the past, history and physical exam with sweat chloride testing were the cornerstones of diagnosis. Diagnosis is now made with the newborn screening test for immunoreactive trypsinogen.
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