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Journal articles on the topic 'Bulimia nervosa'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Bossert, S., R. Laessle, and M. Junker. "Anamnestic similarities in bulimic inpatients with and without a history of anorexia nervosa." Psychiatry and Psychobiology 4, no. 2 (1989): 107–10. http://dx.doi.org/10.1017/s0767399x00002947.

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SummaryThe significance of a history of anorexia nervosa as regards the diagnosis and treatment outcome for bulimia is unclear. In a retrospective analysis of medical records of 59 inpatients with bulimia (DSM-III), variables related to personal and psychiatric family history did not reveal any differences in bulimics subtyped according to previous anorexia nervosa as defined in the criteria of Russell (1979). These anamnestic data support the results of studies indicating that no specific clinical and outcome variables are correlated with a history of anorexia nervosa in bulimia. The lower bo

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2

Cooper, Peter J., Deborah J. Charnock, and Melanie J. Taylor. "The Prevalence of Bulimia Nervosa." British Journal of Psychiatry 151, no. 5 (1987): 684–86. http://dx.doi.org/10.1192/bjp.151.5.684.

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There have been reports of a high prevalence of bulimic episodes and the syndromes of bulimia nervosa and DSM-III bulimia in community samples. A group of American authors recently compared the findings of a contemporary survey with those of a survey they had conducted previously and reported a three-fold increase in the prevalence of DSM-III bulimia. The present study replicates a community survey conducted four years ago in Britain. The prevalence of bulimic episodes, self-induced vomiting and bulimia nervosa found in the present survey was very similar to that found in the earlier study.

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3

Lacey, J. Hubert, and G. Smith. "Bulimia Nervosa." British Journal of Psychiatry 150, no. 6 (1987): 777–81. http://dx.doi.org/10.1192/bjp.150.6.777.

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This study examines the impact of pregnancy on the reported eating behaviour of 20 untreated normal body weight bulimia nervosa women; it also reports foetal and obstetric abnormalities and indicates the initial eating habits of the infants. The prevalence of binge-eating and self-induced vomiting reduced sequentially during each trimester of pregnancy. By the third trimester 15 women (75%) had stopped all bulimic behaviour and in the remainder the disturbed eating was less severe. Symptoms tended to return in the Puerperium and in nearly half the sample abnormal eating was more disturbed afte

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4

Zeeck, Hartmann, Sandholz, and Joos. "Bulimia nervosa." Therapeutische Umschau 63, no. 8 (2006): 535–38. http://dx.doi.org/10.1024/0040-5930.63.8.535.

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Die Bulimia nervosa ist durch Essanfälle und Verhaltensweisen gekennzeichnet, welche einer Gewichtszunahme entgegensteuern sollen (Erbrechen, Laxantienabusus, Hungerphasen u.a.). Sie ist eine multifaktoriell bedingte psychische Erkrankung, welche vor allem junge Frauen betrifft. Die Bulimie kann zu gravierendem Folgen auf körperlicher, psychischer und sozialer Ebene führen und bedarf in der Regel einer spezialisierten, psychotherapeutischen Behandlung. Diese kann in den meisten Fällen ambulant erfolgen, es muss jedoch die häufige Komorbidität mit weiteren psychischen Erkrankungen berücksichtig

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5

Schumaker, John F., William G. Warren, Gwenda S. Schreiber, and Craig C. Jackson. "DISSOCIATION IN ANOREXIA NERVOSA AND BULIMIA NERVOSA." Social Behavior and Personality: an international journal 22, no. 4 (1994): 385–92. http://dx.doi.org/10.2224/sbp.1994.22.4.385.

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The present study employed the Riley Questionnaire of Experiences of Dissociation in order to assess degree of dissociation in females diagnosed with anorexia nervosa and bulimia. The subjects consisted of 26 anorexic and 18 bulimic females, and a non eating-disordered control group of 22 females. Results indicated that eating disordered subjects, considered together, had significantly higher dissociation scores than the non eating-disordered control group. Additionally, when considered separately, both the anorexic and bulimic groups had significantly higher dissociation scores than the contr

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6

Fichter, M. M., N. Quadflieg, and W. Rief. "Course of multi-impulsive bulimia." Psychological Medicine 24, no. 3 (1994): 591–604. http://dx.doi.org/10.1017/s0033291700027744.

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SynopsisThirty-two consecutively admitted females with bulimia nervosa (purging type) according to DSM-IV and additional impulsive behaviours (multi-impulsive bulimia (MIB)) and 32 age-matched female controls with DSM-IV bulimia nervosa (purging type) (uni-impulsive bulimia (UIB)) were assessed longitudinally on admission and at discharge following in-patient therapy and at a 2-year follow-up. Multi-impulsive bulimics were defined as presenting at least three of the six of the following impulsive behaviours in their life-time in addition to their bulimic symptoms at admission: (a) suicidal att

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7

Bossert-Zaudig, S., M. Zaudig, M. Junker, M. Wiegand, and J.-C. Krieg. "Psychiatric comorbidity of bulimia nervosa inpatients: relationship to clinical variables and treatment outcome." European Psychiatry 8, no. 1 (1993): 15–23. http://dx.doi.org/10.1017/s0924933800001504.

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SummaryExperimental evidence suggesting that psychiatric comorbidity has important clinical and prognostic implications in bulimia nervosa has mostly been based on outpatient studies investigating a selection of co-existing psychopathological features with rather unstructured and not standardized diagnostic instruments. Using structured instruments (SCID-P, MDCL) for the diagnoses of DSM III-R axis I disorders and clinical interviews for the diagnosis of DSM III-R axis II disorders in 24 hospitalized bulimics, the present study demonstrated that more than half of the patients had two or three

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8

Wade, Tracey D., Cynthia M. Bulik, and Kenneth S. Kendler. "Reliability of lifetime history of bulimia nervosa." British Journal of Psychiatry 177, no. 1 (2000): 72–76. http://dx.doi.org/10.1192/bjp.177.1.72.

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BackgroundPrevious studies have found that the reliability of the lifetime prevalence of bulimia nervosa is low to moderate. However, the reasons for poor reliability remain unknown.AimsWe investigated the ability of a range of variables to predict reliability, sensitivity, and specificity of reporting of both bulimia nervosa and major depression.MethodTwo interviews, approximately 5 years apart, were completed with 2163 women from the Virginia Twin Registry.ResultsAfter accounting for different base rates, bulimia nervosa was shown to be as reliably reported as major depression. Consistent wi

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9

Robinson, P. H., S. A. Checkley, and G. F. M. Russell. "Suppression of Eating by Fenfluramine in Patients with Bulimia Nervosa." British Journal of Psychiatry 146, no. 2 (1985): 169–76. http://dx.doi.org/10.1192/bjp.146.2.169.

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SummaryFifteen patients with bulimia nervosa received fenfluramine (60 mg po) or placebo under double-blind, randomly ordered conditions. Two hours later food was presented. Significantly less food was eaten after fenfluramine and the quantity eaten was inversely correlated with serum fenfluramine levels. Significantly fewer patients reported bulimic symptoms during the test after fenfluramine, but no significant effect was demonstrated after leaving the ward. Fenfluramine caused drowsiness but did not reduce hunger ratings. Similarly, eating failed to reduce hunger ratings normally in the pat

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10

LESTER, N. A., P. K. KEEL, and S. F. LIPSON. "Symptom fluctuation in bulimia nervosa: relation to menstrual-cycle phase and cortisol levels." Psychological Medicine 33, no. 1 (2002): 51–60. http://dx.doi.org/10.1017/s0033291702006815.

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Background. Individuals with bulimia nervosa report significant symptom fluctuation, and some studies have suggested a premenstrual exacerbation of binge frequency. The purpose of this study is to explore the hormonal correlates of symptom fluctuation in bulimia nervosa.Method. For five consecutive weeks (one full menstrual cycle), eight women with bulimia nervosa and eight non-eating-disordered control women collected morning saliva samples and recorded several mood characteristics; the bulimic women also recorded binge and purge episodes. Subsequently, salivary cortisol and androgen levels w

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11

Fahy, Thomas, and Ivan Eisler. "Impulsivity and Eating Disorders." British Journal of Psychiatry 162, no. 2 (1993): 193–97. http://dx.doi.org/10.1192/bjp.162.2.193.

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Sixty-seven patients with bulimia nervosa and 29 patients with anorexia nervosa completed the Impulsiveness Questionnaire and questionnaires detailing severity of eating disorder. Bulimic patients had higher impulsivity scores than anorexic patients. Bulimics with high impulsivity scores did not have more severe eating disorders than low scorers. When 39 bulimics and 25 anorexics were interviewed about other impulsive behaviour, 51 % of bulimics and 28% of anorexics reported at least one other impulsive behaviour. Patients with so-called ‘multiimpulsive’ bulimia reported more severe eating dis

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12

Morgan, John Farnill, J. Hubert Lacey, and Philip M. Sedgwick. "Impact of pregnancy on bulimia nervosa." British Journal of Psychiatry 174, no. 2 (1999): 135–40. http://dx.doi.org/10.1192/bjp.174.2.135.

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BackgroundBulimia nervosa affects women at a peak age of reproductive functioning, but few studies have examined the impact of pregnancy on bulimia.AimTo examine the impact of pregnancy on symptoms of bulimia nervosa and associated psychopathology.MethodWomen actively suffering from bulimia nervosa during pregnancy (n=94) were interviewed using the eating disorder examination (12th edn) and structured clinical interview for DSM–III–R, with additional structured questions. Behaviours were recorded at conception, each trimester and postnatally. Relative risks were calculated for prognostic facto

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13

Huemer, Julia, Maria Haidvogl, Fritz Mattejat, et al. "Perception of Autonomy and Connectedness Prior to the Onset of Anorexia Nervosa and Bulimia Nervosa." Zeitschrift für Kinder- und Jugendpsychiatrie und Psychotherapie 40, no. 1 (2012): 61–68. http://dx.doi.org/10.1024/1422-4917/a000150.

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Objective: This study examines retrospective correlates of nonshared family environment prior to onset of disease, by means of multiple familial informants, among anorexia and bulimia nervosa patients. Methods: A total of 332 participants was included (anorexia nervosa, restrictive type (AN-R): n = 41 plus families); bulimic patients (anorexia nervosa, binge-purging type; bulimia nervosa: n = 59 plus families). The EATAET Lifetime Diagnostic Interview was used to establish the diagnosis; the Subjective Family Image Test was used to derive emotional connectedness (EC) and individual autonomy (I

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14

FRANKO, D. L., P. K. KEEL, D. J. DORER, et al. "What predicts suicide attempts in women with eating disorders?" Psychological Medicine 34, no. 5 (2004): 843–53. http://dx.doi.org/10.1017/s0033291703001545.

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Background. Suicide is a common cause of death in anorexia nervosa and suicide attempts occur often in both anorexia nervosa and bulimia nervosa. No studies have examined predictors of suicide attempts in a longitudinal study of eating disorders with frequent follow-up intervals. The objective of this study was to determine predictors of serious suicide attempts in women with eating disorders.Method. In a prospective longitudinal study, women diagnosed with either DSM-IV anorexia nervosa (n=136) or bulimia nervosa (n=110) were interviewed and assessed for suicide attempts and suicidal intent e

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15

Đurović, Marija. "Bulimia: Modern man challenge." Galenika Medical Journal 1, no. 1 (2022): 78–86. http://dx.doi.org/10.5937/galmed2201078d.

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The fact, that man has always been focused on external beauty is never surprising. The base of our identity is our body. However, it is worrying to know that whether they are obese, thin, or normally fed, people of both sexes and all age groups have never been as dissatisfied with their bodies as they are today. Many do not perceive eating disorders as serious diseases, but as someone's whim, the desire to be thin, and some even go so far as to accept a disturbed diet as healthy. We notice that healthy people talk about eating disorders in an affirmative tone - they don't understand that it is

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16

Fitzgerald, Barbara A., Jesse H. Wright, and Katherine D. Atala. "Bulimia nervosa." Postgraduate Medicine 84, no. 2 (1988): 119–23. http://dx.doi.org/10.1080/00325481.1988.11700368.

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17

Rushing, Jona M., Laura E. Jones, and Caroline P. Carney. "Bulimia Nervosa." Primary Care Companion to The Journal of Clinical Psychiatry 05, no. 05 (2003): 217–24. http://dx.doi.org/10.4088/pcc.v05n0505.

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18

Mehler, Philip S. "Bulimia Nervosa." New England Journal of Medicine 349, no. 9 (2003): 875–81. http://dx.doi.org/10.1056/nejmcp022813.

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19

Fairburn, C. G. "Bulimia nervosa." BMJ 300, no. 6723 (1990): 485–87. http://dx.doi.org/10.1136/bmj.300.6723.485.

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20

Svaldi, Jennifer, and Brunna Tuschen-Caffier. "Bulimia nervosa." PSYCH up2date 12, no. 05 (2018): 415–31. http://dx.doi.org/10.1055/a-0498-3661.

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21

Russell, Gerald, and Janet Treasure. "Bulimia nervosa." British Journal of Psychiatry 201, no. 1 (2012): 19. http://dx.doi.org/10.1192/bjp.bp.112.109645.

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22

Lilly, R. Z. "Bulimia nervosa." BMJ 327, no. 7411 (2003): 380–81. http://dx.doi.org/10.1136/bmj.327.7411.380.

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23

POWERS, PAULINE S. "Bulimia Nervosa." American Journal of Psychiatry 148, no. 8 (1991): 1082—a—1083. http://dx.doi.org/10.1176/ajp.148.8.1082-a.

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24

Erpen, Heinrich. "Bulimia nervosa." Daseinsanalyse 4, no. 4 (1987): 285–313. http://dx.doi.org/10.1159/000456200.

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25

LACEY, J. HUBERT, and G. SMITH. "Bulimia Nervosa." Obstetrical & Gynecological Survey 44, no. 3 (1989): 191–92. http://dx.doi.org/10.1097/00006254-198903000-00006.

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26

Sedghizadeh, Parish P. "Bulimia Nervosa." New England Journal of Medicine 368, no. 13 (2013): 1238. http://dx.doi.org/10.1056/nejmicm1207495.

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27

Hall, Richard C. W., Robert E. Blakey, and Anne Klassen Hall. "Bulimia Nervosa." Psychosomatics 33, no. 4 (1992): 428–36. http://dx.doi.org/10.1016/s0033-3182(92)71947-1.

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28

Herpertz, Stephan. "Bulimia nervosa." PiD - Psychotherapie im Dialog 2, no. 2 (2001): 139–53. http://dx.doi.org/10.1055/s-2001-15586.

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29

Mendhekar, D. N., Rajiv Mehta, and P. K. Srivastav. "Bulimia nervosa." Indian Journal of Pediatrics 71, no. 9 (2004): 861–62. http://dx.doi.org/10.1007/bf02730730.

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30

Palmer, Teresa A. "Anorexia Nervosa, Bulimia Nervosa." Nurse Practitioner 15, no. 4 (1990): 12???21. http://dx.doi.org/10.1097/00006205-199004000-00007.

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31

Srivastav, Yash, Mohd Faijan Mansoori, Aditya Srivastav, and Aniket Kumar. "Schematic Brief Outline: Bulimia Nervosa and its Medical-Based Management." International Neuropsychiatric Disease Journal 21, no. 4 (2024): 61–65. http://dx.doi.org/10.9734/indj/2024/v21i4442.

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The disease known as bulimia nervosa, which is typified by purging and binge eating, usually starts in adolescence and peaks at the age of 18. The ratio of female to male patients varies from 10:1 to 20:1, with a lifetime frequency of 3 per cent. The majority of bulimic individuals also suffer from other mental illnesses, such as depression or anxiety. Additionally, there is a correlation between substance misuse and promiscuity. Bulimia nervosa was initially identified as a "chronic phase of anorexia nervosa" in 1979 by British psychiatrist Gerald Russell. During this stage, patients overeat

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32

Cooper, Peter J., and Melanie J. Taylor. "Body Image Disturbance in Bulimia Nervosa." British Journal of Psychiatry 153, S2 (1988): 32–36. http://dx.doi.org/10.1192/s0007125000298966.

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Bulimia nervosa is a disorder which has only recently been identified and described (Russell, 1979). It is a condition, found almost exclusively in young women, in which episodes of excessive uncontrolled eating are the central feature. Patients with bulimia nervosa usually present at normal weight because they compensate for the bulimic episodes in a variety of ways, such as by inducing vomiting, abusing purgatives and exercising extreme dietary restraint. In addition to these disturbed eating habits, patients with bulimia nervosa have attitudes to their shape and weight similar to those foun

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33

Naessén, Sabine, Kjell Carlström, Rolf Glant, Hans Jacobsson, and Angelica Lindén Hirschberg. "Bone mineral density in bulimic women – influence of endocrine factors and previous anorexia." European Journal of Endocrinology 155, no. 2 (2006): 245–51. http://dx.doi.org/10.1530/eje.1.02202.

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Objective: Data concerning bone mineral density (BMD) in bulimia nervosa are contradictory and include both low and normal values. The aim of the present study was to elucidate possible endocrine-and nutrition-related factors predicting BMD in bulimic women. Design: Cross-sectional study. Methods: Seventy-seven bulimic patients and 56 age- and body mass index (BMI)-matched healthy controls were examined with respect to BMD (dual energy X-ray absorptiometry) and to serum levels of hormones and metabolic factors. Results: Bulimics had significantly lower spinal BMD and higher frequency of osteop

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34

MONTELEONE, P., F. BRAMBILLA, F. BORTOLOTTI, and M. MAJ. "Serotonergic dysfunction across the eating disorders: relationship to eating behaviour, purging behaviour, nutritional status and general psychopathology." Psychological Medicine 30, no. 5 (2000): 1099–110. http://dx.doi.org/10.1017/s0033291799002330.

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Background. Several recent studies have pointed to a dysfunction of serotonin transmission in patients with eating disorders. Notwithstanding, it is not known whether serotonergic abnormalities are related primarily to eating and/or purging behaviour, nutritional status or general psychopathological dimensions. Therefore, by using a validated neuroendocrine strategy, we investigated central serotonergic function in patients with anorexia nervosa, bulimia nervosa or binge-eating disorder who differ on the above parameters.Methods. Plasma prolactin response to D-fenfluramine (30 mg p.o.) or plac

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35

Schmidt, Ulrike, Jane Tiller, and Janet Treasure. "Setting the scene for eating disorders: childhood care, classification and course of illness." Psychological Medicine 23, no. 3 (1993): 663–72. http://dx.doi.org/10.1017/s0033291700025447.

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SynopsisThe aim of this study was to determine whether the childhood experiences of patients with anorexia nervosa and bulimia nervosa differ and affect the course of the illness. A semistructured interview developed by Harris et al. (1986) was used to assess the childhood family environment of 64 patients with restricting anorexia nervosa (RAN), 23 patients with bulimic anorexia nervosa (BAN), 37 bulimic patients with a history of anorexia nervosa (BN/HistAN) and 79 patients with normal weight bulimia nervosa (BN).There were no significant differences between groups in terms of parental menta

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36

Rodin, Gary M., Liane E. Johnson, Paul E. Garfinkel, Denis Daneman, and Anne B. Kenshole. "Eating Disorders in Female Adolescents with Insulin Dependent Diabetes Mellitus." International Journal of Psychiatry in Medicine 16, no. 1 (1987): 49–57. http://dx.doi.org/10.2190/hulh-ctpr-4v17-383c.

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Recent case reports have suggested an association between anorexia nervosa and/or bulimia with insulin-dependent diabetes mellitus (IDDM). Fifty-eight females aged fifteen to twenty-two with IDDM for more than one year were assessed for the presence of eating disorders. Patients were screened for eating and weight pathology using the Eating Disorder Inventory (EDI) and Eating Attitudes Test-26 (EAT-26). Glycosylated hemoglobin (HbAl) was measured to assess metabolic control. Subjects who scored above the cut-off points associated with eating and weight pathology were interviewed. Clinically si

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37

Turnbull, Sue, Anne Ward, Janet Treasure, Hershel Jick, and Laura Derby. "The Demand for Eating Disorder Care." British Journal of Psychiatry 169, no. 6 (1996): 705–12. http://dx.doi.org/10.1192/bjp.169.6.705.

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BackgroundAn epidemiological study of anorexia nervosa and bulimia nervosa in primary care was performed using the General Practice Research Database (GPRD).MethodThe GPRD was screened between 1988 and 1994 for newly diagnosed cases of anorexia nervosa and bulimia nervosa. The validity of the computer diagnosis was established by obtaining clinical details from a random sample of the general practitioners (GPs).ResultsIncidence rates for detection of cases by GPs in 1993 was 4.2 per 100 000 population for anorexia nervosa and 12.2 per 100 000 for bulimia nervosa The relative risks of females t

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38

Lan, Yufei. "Cognitive Behavioral Therapy for Bulimia Nervosa: A Review." Theoretical and Natural Science 70, no. 1 (2024): 128–34. https://doi.org/10.54254/2753-8818/2024.18848.

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Bulimia nervosa is a complex eating disorder that severely affects patients mental health and quality of life. And the Cognitive Behavioral Therapy (CBT), as a cutting-edge intervention, has profoundly revealed the cognitive mechanisms behind bulimic behaviors, paving new pathways for treatment. The paper delves into the strategies of CBT in bulimia intervention, emphasizing the core elements of emotional regulation, cognitive restructuring and behavioral experimentation. At the same time, the challenges that may arise during the implementation of CBT should not be overlooked, such as patient

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39

Sullivan, Patrick F., Cynthia M. Bulik, Frances A. Carter, and Peter R. Joyce. "The Significance of a History of Childhood Sexual Abuse in Bulimia Nervosa." British Journal of Psychiatry 167, no. 5 (1995): 679–82. http://dx.doi.org/10.1192/bjp.167.5.679.

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BackgroundChildhood sexual abuse (CSA) is found to have occurred to a substantial minority of women with bulimia nervosa. Its clinical significance is unclear.MethodWe studied 87 bulimic women in a clinical trial. Structured interviews determined the presence of CSA, DSM–III–R disorders, global functioning, and depressive and bulimic symptoms.ResultsForty-four per cent reported a history of CSA. Bulimic women with CSA reported earlier onset of bulimia, greater depressive symptoms, worse global functioning and more suicide attempts, and were more likely to meet criteria for bipolar II disorder,

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40

Foteini, P., P. Kamposiora, and G. Papavasiliou. "A Multidisciplinary Approach to the Functional and Esthetic Rehabilitation of a Patient with Bulimia Nervosa: A Clinical Report." International Journal of Dentistry and Oral Science (IJDOS) 2, no. 3 (2015): 53–58. https://doi.org/10.19070/2377-8075-1500012.

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Treatment of a patient with bulimia nervosa is a challenge for the dental clinician. The oral manifestations of bulimic behavior, which include sore and inflamed throat, swollen salivary glands, tooth wear and decay, require a multidisciplinary approach. This clinical report illustrates the treatment of a 22-year-old female patient with history of bulimia nervosa and dentition with extended signs of erosion. It included thorough diagnosis with the use of screening tool BITE (Bulimic Investigatory Test, Edinburgh) and initial evaluation of tooth wear with the TWI (Tooth Wear Inde

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41

Krauß, Eva, and Martina de Zwaan. "Anorexia nervosa und Bulimia nervosa." Psychiatrie und Psychotherapie up2date 1, no. 4 (2007): 273–89. http://dx.doi.org/10.1055/s-2007-970824.

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42

Treasure, J. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 3, no. 2 (1990): 211–14. http://dx.doi.org/10.1097/00001504-199004000-00007.

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43

Woodside, D. Blake. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 3, no. 4 (1990): 453–56. http://dx.doi.org/10.1097/00001504-199008000-00006.

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44

Treasure, Janet. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 4, no. 2 (1991): 236–41. http://dx.doi.org/10.1097/00001504-199104000-00009.

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45

Treasure, Janet. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 5, no. 2 (1992): 228–33. http://dx.doi.org/10.1097/00001504-199204000-00009.

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46

Smith, Jane Ellen, and Denise E. Laframboise. "Anorexia nervosa and bulimia nervosa." Current Opinion in Psychiatry 8, no. 6 (1995): 419–23. http://dx.doi.org/10.1097/00001504-199511000-00016.

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47

Müller, Astrid, and Martina de Zwaan. "Anorexia nervosa und Bulimia nervosa." Geburtshilfe und Frauenheilkunde 65, no. 6 (2005): R105—R120. http://dx.doi.org/10.1055/s-2005-865792.

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48

Krauß, E., and M. de Zwaan. "Anorexia nervosa und Bulimia nervosa." Frauenheilkunde up2date 2, no. 04 (2008): 347–68. http://dx.doi.org/10.1055/s-2008-1076910.

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49

Støylen, Inge Jarl, and Jon Chr Laberg. "Anorexia Nervosa and Bulimia Nervosa." Acta Psychiatrica Scandinavica 82 (June 28, 2008): 52–58. http://dx.doi.org/10.1111/j.1600-0447.1990.tb11086.x.

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50

Huber, M. "Anorexia nervosa und Bulimia nervosa." Der Nephrologe 13, no. 1 (2017): 12–18. http://dx.doi.org/10.1007/s11560-017-0205-3.

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