Log in

Relevant bibliographies by topics / Bulloni / Journal articles

To see the other types of publications on this topic, follow the link: Bulloni.

Journal articles on the topic 'Bulloni'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 March 2023

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the top 50 journal articles for your research on the topic 'Bulloni.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Browse journal articles on a wide variety of disciplines and organise your bibliography correctly.

1

Gardner, Kerry M., and Richard I. Crawford. "Distinguishing Epidermolysis Bullosa Acquisita From Bullous Pemphigoid Without Direct Immunofluorescence." Journal of Cutaneous Medicine and Surgery 22, no. 1 (2017): 22–24. http://dx.doi.org/10.1177/1203475417722734.

Full text

Abstract:

Background: It has been postulated that periodic acid–Schiff staining of basement membrane can predict direct immunofluorescence patterns seen in epidermolysis bullosa acquisita and bullous pemphigoid. It has also been suggested that the type of inflammatory infiltrate or presence of fraying of basal keratinocytes may differentiate these two conditions. Objective: In this study, we aimed to confirm these observations. Methods: We reviewed 13 cases of direct immunofluorescence-confirmed epidermolysis bullosa acquisita and 19 cases of direct immunofluorescence-confirmed bullous pemphigoid, all w

APA, Harvard, Vancouver, ISO, and other styles

2

Dabelsteen, E. "Molecular Biological Aspects of Acquired Bullous Diseases." Critical Reviews in Oral Biology & Medicine 9, no. 2 (1998): 162–78. http://dx.doi.org/10.1177/10454411980090020201.

Full text

Abstract:

Bullous diseases of the oral mucosa and skin were originally classified on the basis of clinical and histological criteria. The discovery of autoantibodies in some of these patients and the introduction of molecular biology have resulted in a new understanding of the pathological mechanisms of many of the bullous lesions. In this article, updated topics of the immune-mediated bullous lesions which involve oral mucosa and skin are reviewed. Pemphigus antigens, which are desmosomal-associated proteins and belong to the cadherin superfamily of cell adhesion proteins, have been isolated, and their

APA, Harvard, Vancouver, ISO, and other styles

3

Bodman, M., S. Friedman, and LB Clifford. "Bullosis diabeticorum. A report of two cases with a review of the literature." Journal of the American Podiatric Medical Association 81, no. 10 (1991): 561–63. http://dx.doi.org/10.7547/87507315-81-10-561.

Full text

Abstract:

Bullosis diabeticorum is a rare complication of long-standing diabetes mellitus. Bullous lesions, which appear like burn-induced blisters, occur suddenly without trauma in the feet. These bullae heal spontaneously without scarring; however, recurrence is common. The etiology of bullosis diabeticorum remains unknown. Several theories exist as to causal and contributory factors related to this cutaneous manifestation of diabetes mellitus.

APA, Harvard, Vancouver, ISO, and other styles

4

Laamari, Kaoutar, Hanane Baybay, Samia Mrabat, Zakia Douhi, Sara Elloudi, and Fatima Zahra Mernissi. "Hereditary Epidermolysis Bullosa: New Description." Dermatology and Dermatitis 5, no. 1 (2020): 01–02. http://dx.doi.org/10.31579/2578-8949/066.

Full text

Abstract:

Epidermolysis bullosa (EB) is a heterogeneous group of genetically determined, mechano-bullous disorders characterized by blister formation in response to mechanical trauma. The blistering of the skin occurs in the varying degrees of severity and can severely incapacitate the life of the afflicted patient. Epidermolysis Bullosa Simplex (EBS), the most commonly occurring type, is dominantly inherited where treatment still remains a major challenge.

APA, Harvard, Vancouver, ISO, and other styles

5

Umar, Rana Muhammad, Azhar Ali Khan, and Waqar Ahmad. "Epidermolysis Bullosa Acquisita in a patient with End Stage Renal Disease: Case study." Journal of the Pakistan Medical Association 73, no. 2 (2023): 405–6. http://dx.doi.org/10.47391/jpma.4959.

Full text

Abstract:

Epidermolysis Bullosa Acquisita (EBA) is a rare disorder characterised by sub- epithelial mucocutaneous blistering. Its presentation may be in the form of inflammatory bullous eruption or non-inflammatory tense bullae, milia, and scarring. EBA is an immune disorder mediated with antibodies against type VII collagen, which is a major component of basement membrane zones of the skin and mucosa. Adults are more commonly affected; however, a few paediatric cases have also been reported. Risk factors for its development are still under study. Very few cases are seen of EBA in patients with End Stag

APA, Harvard, Vancouver, ISO, and other styles

6

Mahato, Shyam Kumar, Susana Lama, Nikhil Agarwal, and Nagendra Chaudhary. "Inherited Epidermolysis Bullosa: A case report." Journal of Universal College of Medical Sciences 3, no. 3 (2015): 39–42. http://dx.doi.org/10.3126/jucms.v3i3.24248.

Full text

Abstract:

Epidermolysis bullosa (EB) is a heterogeneous group of genetically determined, mechano-bullous disorders characterized by blister formation in response to mechanical trauma. The blistering of the skin occurs in the varying degrees of severity and can severely incapacitate the life of the afflicted patient. Epidermolysis Bullosa Simplex (EBS), the most commonly occurring type, is dominantly inherited where treatment still remains a major challenge. We report a newborn female with blistering of the skin during the immediate neonatal period.

APA, Harvard, Vancouver, ISO, and other styles

7

Ozturan, O., A. Yenigun, N. Degirmenci, and F. Yilmaz. "‘Conchae bullosis’: a rare case with bilateral triple turbinate pneumatisations." Journal of Laryngology & Otology 127, no. 1 (2012): 73–75. http://dx.doi.org/10.1017/s0022215112002514.

Full text

Abstract:

AbstractBackground:Concha bullosa is the pneumatisation of intranasal conchae (usually the middle turbinate, and rarely the inferior or superior turbinate); however, the term is generally used to describe aeration of the middle concha. Superior concha bullosa is a rare finding, and only a few cases of inferior concha bullosa have been reported in the medical literature. When symptomatic, concha bullosa may cause various problems including nasal congestion, headache, postnasal drip, anosmia and, sometimes, epiphora.Methodology:Computed tomography, following history-taking and physical examinati

APA, Harvard, Vancouver, ISO, and other styles

8

Budiyani, Laras, Adeputri Tanesha Idayu, Ika Prasetya Wijaya, and Alvina Widhani. "Sindrom Hiper IgE dengan Manifestasi Bullous pemphigoid dan Striktur Esofagus." Jurnal Penyakit Dalam Indonesia 3, no. 2 (2016): 100. http://dx.doi.org/10.7454/jpdi.v3i2.96.

Full text

Abstract:

Sindrom hiper IgE (HIES) adalah suatu defisiensi kompleks imun primer yang jarang terjadi, memiliki manifestasi klinis yang beragam. Mutasi STAT 3 merupakan dasar dari sindrom hiper IgE klasik yang berhubungan dengan abnormalitas wajah, gigi, rangka, dan jaringan ikat yang tidak terlihat pada tipe resesif. Abses kulit berulang adalah gambaran utama pada HIES. Pemphigoid bullosa adalah kelainan autoimun yang memiliki karakteristik manifestasi bula pada kulit terkait membrane mukosa. Adanya gangguan autoimun yang terjadi secara bersamaan merupakan kelainan yang jarang. Striktur esofagus adalah s

APA, Harvard, Vancouver, ISO, and other styles

9

Salas-Alanis, Julio Cesar, Cesar Eduardo Rosales-Mendoza, and Jorge Ocampo-Candiani. "Bullous Mastocytosis Mimicking Congenital Epidermolysis Bullosa." Case Reports in Dermatology 6, no. 2 (2014): 129–33. http://dx.doi.org/10.1159/000362755.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

De Jong, Marcelus C. J. M. "Bullous Pemphigoid and Epidermolysis Bullosa Acquisita." Archives of Dermatology 132, no. 2 (1996): 151. http://dx.doi.org/10.1001/archderm.1996.03890260053008.

Full text

APA, Harvard, Vancouver, ISO, and other styles

11

Tešanović Perković, Deša, Zrinka Bukvić Mokos, and Branka Marinović. "Epidermolysis Bullosa Acquisita—Current and Emerging Treatments." Journal of Clinical Medicine 12, no. 3 (2023): 1139. http://dx.doi.org/10.3390/jcm12031139.

Full text

Abstract:

Epidermolysis bullosa acquisita (EBA) is a rare chronic autoimmune subepidermal blistering disease of the skin and mucous membranes, usually beginning in adulthood. EBA is induced by autoantibodies to type VII collagen, a major component of anchoring fibrils in the dermal–epidermal junction (DEJ). The binding of autoantibodies to type-VII collagen subsequently leads to the detachment of the epidermis and the formation of mucocutaneous blisters. EBA has two major clinical subtypes: the mechanobullous and inflammatory variants. The classic mechanobullous variant presentation consists of skin fra

APA, Harvard, Vancouver, ISO, and other styles

12

Leonova, Maria A., and Nikolay N. Murashkin. "Pruriginosa Pattern of Dystrophic Epidermolisys Bullosa: Clinical Case." Pediatric pharmacology 19, no. 6 (2023): 479–83. http://dx.doi.org/10.15690/pf.v19i6.2476.

Full text

Abstract:

Background. Epidermolisys bullosa pruriginosa is a rare pattern of dystrophic epidermolisys bullosa and characterized by severe itching that accompanies the formation of papules, plaques and nodes primarily on the lower limbs skin and imitating prurigo nodularis. Nowadays, less than 100 cases of this disease are reported in the world, thus, the presentation of this clinical case is relevant. Clinical case description. The authors describe the clinical case of pruriginous pattern of dominant dystrophic bullous epidermolysis in 14-year-old female patient. Conclusion. Diagnosis of this disease pa

APA, Harvard, Vancouver, ISO, and other styles

13

Erpuleva, Yu V., L. V. Shurova, and L. I. Dmitrienko. "The experience of using a modern mixture for oral nutrition in a child with epidermolysis bullosa." Clinical nutrition and metabolism 1, no. 1 (2020): 47–51. http://dx.doi.org/10.17816/clinutr20703.

Full text

Abstract:

Bullous epidermolysis is a congenital disease characterized by skin vulnerability due to the formation of blisters and erosion on the skin and mucous membranes, which makes it difficult for a child to eat naturally. The problem of providing the necessary nutrients to children with this serious illness remains relevant. The article shows its own clinical experience in the use of modern enteral nutritional mixtures in a child with epidermolysis bullosa. Given the skin manifestations, the possibility of infection of the catheter for prolonged parenteral nutrition, children with bullous epidermoly

APA, Harvard, Vancouver, ISO, and other styles

14

Pratasava, Valeryia, Vikram N. Sahni, Aishwarya Suresh, et al. "Bullous Pemphigoid and Other Pemphigoid Dermatoses." Medicina 57, no. 10 (2021): 1061. http://dx.doi.org/10.3390/medicina57101061.

Full text

Abstract:

The pemphigoid family of dermatoses is characterized by autoimmune subepidermal blistering. The classic paradigm for pemphigoid, and the most common member, is bullous pemphigoid. Its variable clinical presentation, with or without frank bullae, is linked by significant pruritus afflicting the elderly. Mucous membrane pemphigoid is an umbrella term for a group of subepidermal blistering dermatoses that favor the mucosal membranes and can scar. Epidermolysis bullosa acquisita is a chronic blistering disorder characterized by skin fragility, sensitivity to trauma, and its treatment-refractory na

APA, Harvard, Vancouver, ISO, and other styles

15

Sayeed, Sk Jakaria Been, Md Mujibur Rahman, AKM Humayon Kabir, et al. "Bullous Systemic Lupus Erythematosus." Journal of Medicine 19, no. 2 (2018): 123–25. http://dx.doi.org/10.3329/jom.v19i2.37233.

Full text

Abstract:

Bullous systemic lupus erythematosus (BSLE) is extremely rare but distinct disease, characterized by vesicobullous skin eruptions in systemic lupus erythematosus (SLE). It can develop either before or after a diagnosis of SLE has been established. BSLE is characterized by a dermatitis herpetiformis-like histology and an autoimmunity to type VII collagen. It must be differentiated from other autoimmune vesicobullous diseases such as epidermolysis bullosa acquisita, dermatitis herpetiformis, linear IgA disease, and bullous pemphigoid. Its important to combine clinical, histological, and immunofl

APA, Harvard, Vancouver, ISO, and other styles

16

Korekawa, Ayumi, Takahide Kaneko, Koji Nakajima, et al. "Mycosis fungoides bullosa associated with bullous pemphigoid." International Journal of Dermatology 54, no. 9 (2015): e366-e368. http://dx.doi.org/10.1111/ijd.12821.

Full text

APA, Harvard, Vancouver, ISO, and other styles

17

Siddig, Omayma, Mayson B. Mustafa, Yousif Kordofani, John Gibson, and Ahmed M. Suleiman. "The epidemiology of autoimmune bullous diseases in Sudan between 2000 and 2016." PLOS ONE 16, no. 7 (2021): e0254634. http://dx.doi.org/10.1371/journal.pone.0254634.

Full text

Abstract:

Objectives Autoimmune bullous diseases vary in their clinico-epidemiological features and burden across populations. Data about these diseases was lacking in Sudan. We aimed to describe the epidemiological profile and to estimate the burden of autoimmune bullous diseases in Sudan. Methods This was a retrospective cross-sectional study conducted at Khartoum Dermatological and Venereal Diseases Teaching Hospital. We used routinely collected health care data, and included all patients with an autoimmune bullous disease who presented to the hospital between 2001 and 2016. Results Out of the 4736 p

APA, Harvard, Vancouver, ISO, and other styles

18

Di Pentima, Lorenza, and Sara Ramelli. "Attaccamento e bullismo: un confronto tra bulli, vittime, bulli-vittime e non-coinvolti in età scolare." MALTRATTAMENTO E ABUSO ALL'INFANZIA, no. 2 (August 2020): 119–45. http://dx.doi.org/10.3280/mal2020-002007.

Full text

Abstract:

Scopo del presente studio è stato analizzare il fenomeno del bullismo secondo la prospettiva dell'attaccamento, ponendo a confronto i bulli, i bulli-vittima, le vittime e i non-coinvolti nei modelli mentali e nei comportamenti socio-emozionali (in particolare aggressività, evitamento dell'interazione, manifestazioni di ansia e depressione). Hanno partecipato allo studio 365 bambini, 208 maschi (57%) e 157 femmine (43%), di 5 scuole di Roma, di età compresa tra 8 e 11 anni (M = 9.28, DS = 0.81). Gli strumenti impiegati sono stati: Nomina dei Pari (Menesini, 2003), per individuare i ruoli di bul

APA, Harvard, Vancouver, ISO, and other styles

19

Ali, Chowdhury Mohammad, SM Bakhtiar Kamal, and Md Mosaraf Hossain Khan. "Bullous systemic lupus erythematosus a case report." Journal of Dhaka Medical College 22, no. 1 (2013): 90–92. http://dx.doi.org/10.3329/jdmc.v22i1.15691.

Full text

Abstract:

Bullous systemic lupus erythematosus (BSLE) is a rare but distinct disease, characterized by vesicobullous skin eruptions in systemic lupus erythematosus (SLE). It can arise either before or after a diagnosis of SLE has been established. BSLE is characterized by a dermatitis herpetiformis-like histology and an autoimmunity to type VII collagen. It must be differentiated from other autoimmune vesicobullous diseases such as epidermolysis bullosa acquisita, dermatitis herpetiformis, linear IgA disease, and bullous pemphigoid. A combination of clinical, histological, and immunofluorescence finding

APA, Harvard, Vancouver, ISO, and other styles

20

Anastasia, Monica Cherlady, Anna Mailasari Kusuma Dewi, and Farah Hendara Ningrum. "The Correlation Between Bullous Concha Volume and Type of Nasal Septal Deviation with Lund-Mackay CT Score in Chronic Rhinosinusitis." Medica Hospitalia : Journal of Clinical Medicine 9, no. 3 (2022): 353–59. http://dx.doi.org/10.36408/mhjcm.v9i3.800.

Full text

Abstract:

BACKGROUND: Chronic rhinosinusitis is inflammation of the mucoperiosteum lining of the nose and paranasal sinuses for more than 12 weeks. Computed Tomography (CT scan) of the paranasal sinuses is the gold standard for diagnosing chronic rhinosinusitis. Nasal septal deviation and bullous concha are anatomical variation that often found in CT scan of chronic rhinosinusitis patients. The severity of chronic rhinosinusitis can be evaluated with Lund Mackay CT score. AIM: To analyse the correlation between bullous concha volume and type of nasal septum deviation with Lund-Mackay CT Score in chronic

APA, Harvard, Vancouver, ISO, and other styles

21

Mota, Amanda Nascimento Cavalleiro de Macedo, Natalia Solon Nery, and Carlos Baptista Barcaui. "Case for diagnosis." Anais Brasileiros de Dermatologia 88, no. 4 (2013): 652–54. http://dx.doi.org/10.1590/abd1806-4841.20132114.

Full text

Abstract:

We present a case of bullosis diabeticorum. It is a rare disorder, probably underdiagnosed, associated with long-term diabetes mellitus. Its etiology remains unclear. It is characterized by tense blisters, with serous content, recurrent and spontaneous on normal skin especially in the acral regions. Displays self-limiting course. No specific laboratory tests for diagnosis of this bullous disease exist. Clinical and conservative management to prevent secondary infection reduces morbidity in diabetic patients.

APA, Harvard, Vancouver, ISO, and other styles

22

Deen, Kristyn, and Jason Wu. "Porphyria Cutanea Tarda Masquerading as Epidermolysis Bullosa Acquisita: A Report of Two Cases." Case Reports in Dermatology 7, no. 2 (2015): 129–35. http://dx.doi.org/10.1159/000435830.

Full text

Abstract:

Porphyria cutanea tarda (PCT) is the most common type of porphyria worldwide and is often initially diagnosed when cutaneous manifestations arise. We present two patients where misdiagnosis of PCT occurred due to the condition masquerading as epidermolysis bullosa acquisita histologically. In patients with undifferentiated bullous/erosive skin conditions occurring in photo-distributed regions, PCT should be considered in the differential diagnosis irrespective of histopathological findings on biopsies and further investigated and treated appropriately.

APA, Harvard, Vancouver, ISO, and other styles

23

SI, Gargadi. "A Case of Epidermolysis Bullosa in Bhuth: A Case Report." Journal of Surgical Case Reports and Images 3, no. 4 (2020): 01–02. http://dx.doi.org/10.31579/2690-1897/028.

Full text

Abstract:

Epidermolysis Bullosa (EB) is a group of inherited Bullous disorders characterized by formation of blistering following minor trauma (Nikolksy Sign), healing with scarring. It is caused by one or more mutations in at least one of 20 different genes that synthesize structural proteins that are involve in adherence of epidermal to dermis. We observed and cared for a 2 day old female neonate with low birth weight, normal antenatal history who presented at birth with spontaneous bullous skin eruptions to parts of skin of the upper and lower limbs which ruptured with minor mechanical trauma. She ha

APA, Harvard, Vancouver, ISO, and other styles

24

Tikhonovskaya, I. V., and M. A. Katina. "Subepidermal bullous dermatoses. Part II. Bullous pemphigoid, mucous membrane pemphigoid, epidermolysis bullosa acquisita." Vestnik of Vitebsk State Medical University 18, no. 3 (2019): 7–15. http://dx.doi.org/10.22263/2312-4156.2019.3.7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

25

PAPADOGIANNAKIS (E. Ι. ΠΑΠΑΔΟΓΙΑΝΝΑΚΗΣ), E. I. "Contemporary aspects on the immunopathogenesis of autoimmune diseases of the epidermal basement membrane in the dog." Journal of the Hellenic Veterinary Medical Society 56, no. 1 (2017): 27. http://dx.doi.org/10.12681/jhvms.15066.

Full text

Abstract:

Autoimmune diseases of the epidermal basement membrane are the result of the immune system self-activation against specific antigens of its essential structural elements. This group of skin diseases is characterized by the destruction of connecting bonds between the membrane zone and dermis, which eventually leads to the dermoepidermal separation and the formation of subepidermal vesicles and bullae. In this article, the immunopathogenesis and the clinical, histopathological and immunohistochemical features of each of these skin diseases are briefly reviewed. The autoimmune diseases of the can

APA, Harvard, Vancouver, ISO, and other styles

26

Mittal, Lavina, Suparna Madivalara Yallappa, and Praveen Kumar Shanmugam Reddy. "A middle-aged male with bullous lesions and co-morbidities." International Journal of Research in Dermatology 6, no. 5 (2020): 683. http://dx.doi.org/10.18203/issn.2455-4529.intjresdermatol20203533.

Full text

Abstract:

<p>Epidermolysis bullosa acquisita (EBA) is a rare acquired type of mechano bullous disease affecting the dermal-epidermal junction (DEJ) of trauma prone acral surfaces. It manifests as tense vesicles, bullae, and milia and typically heals as atrophic hypo or hyperpigmented scars. Classic noninflammatory mechano bullous EBA typically presents at a mean age of 48 years. A 57 years old male patient, presented with itchy fluid filled lesions over the face since, 2 months. On cutaneous examination, discrete and grouped papulo-vesicles on an erythematous base and areas of erosions present ove

APA, Harvard, Vancouver, ISO, and other styles

27

Kubanov, А. A., A. EH Karamova, V. I. Al'banova, V. V. CHikin, and E. S. Monchakovskaya. "CONGENITAL EPIDERMOLYSIS BULLOSA: PECULIARITIES OF EPIDERMIS REGENERATION AND METHODS OF TREATMENT." Vestnik dermatologii i venerologii 93, no. 4 (2017): 28–37. http://dx.doi.org/10.25208/0042-4609-2017-93-4-28-37.

Full text

Abstract:

Congenital epidermolysis bullosa is a group of hereditary skin diseases caused by mutations in the genes of structural proteins of the dermoepidermal junction of the skin, characterized by formation of blisters and erosions at the smallest mechanical trauma. In patients with severe subtypes of borderline and dystrophic epidermolysis bullosa there are long-term erosive and ulcerative defects with disruption of the healing process. Factors that impede healing include: malnutrition, anemia, pain, inactivity, local factors (presence of infection, prolonged inflammation, extensive nature of the les

APA, Harvard, Vancouver, ISO, and other styles

28

Hashimoto, K., S. Takahashi, and I. Freilich. "Bullous congenital ichthyosiform erythroderma masquerading as dystrophic epidermolysis bullosa." Journal of Cutaneous Pathology 12, no. 2 (1985): 130–41. http://dx.doi.org/10.1111/j.1600-0560.1985.tb01614.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

29

PRUSSICK, RONALD, ADITYA K. GUPTA, DALAL M. ASSAAD, WEDAD M. HANNA, and DANIEL N. SAUDER. "EPIDERMOLYSIS BULLOSA ACQUISITA WITH EEATURES OF BULLOUS LUPUS ERYTHEMATOSUS." International Journal of Dermatology 33, no. 3 (1994): 192–95. http://dx.doi.org/10.1111/j.1365-4362.1994.tb04949.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

30

Hakikah, Tika, Dolly Irfandy, and Bestari Jaka Budiman. "Sakit Kepala pada Variasi Anatomi Hidung dan Sinus Paranasal." Jurnal Kesehatan Andalas 10, no. 3 (2022): 206. http://dx.doi.org/10.25077/jka.v10i3.1919.

Full text

Abstract:

The anatomical variations of the nasal and sinus paranasal can cause headaches. The most common variants have deviated septum, concha bullosa, and agger nasi cells. These variations can cause narrowing of the osteomeatal complex. Computed tomography was the primary modality for evaluating anatomical variations of the nasal and paranasal sinuses in diagnosis and management. Functional endoscopic sinus surgery was generally performed to restore the sinuses' drainage and ventilation in anatomical variations. It has been reported a 45-year-old woman with a congested left nostril and headache that

APA, Harvard, Vancouver, ISO, and other styles

31

Hughes, Amy P., and Jeffrey P. Callen. "Epidermolysis Bullosa Acquisita Responsive to Dapsone Therapy." Journal of Cutaneous Medicine and Surgery 5, no. 5 (2001): 397–99. http://dx.doi.org/10.1177/120347540100500505.

Full text

Abstract:

Background: Epidermolysis bullosa acquisita (EBA) is a chronic subepidermal blistering disease that is frequently resistant to therapy. Objective: A 58-year-old man who had a one-year history of a bullous eruption involving the hands, forearms, trunk, scalp, and oral mucosa. Histopathology revealed a subepidermal bulla, and direct and indirect immunofluorescence studies were consistent with EBA. The patient failed respond to niacinamide and tetracycline and oral prednisone 40 mg per day. Methods: Complete control of his blistering was achieved within two months of initiating oral dapsone, 150

APA, Harvard, Vancouver, ISO, and other styles

32

Lee, Chang Woo. "Subtle Differences in the Morphology of Bullous Lesions between Epidermolysis Bullosa Acquisita and Bullous Pemphigoid." Journal of Dermatology 19, no. 7 (1992): 443–44. http://dx.doi.org/10.1111/j.1346-8138.1992.tb03257.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

33

Osawa, Masumi, Toshio Demitsu, Sunao Toda, et al. "A Case of Mixed Bullous Disease of Epidermolysis bullosa acquisita and Linear IgA Bullous Dermatosis." Dermatology 211, no. 2 (2005): 146–48. http://dx.doi.org/10.1159/000086445.

Full text

APA, Harvard, Vancouver, ISO, and other styles

34

Murashkin, Nikolay N., Leonid A. Opryatin, Alexander I. Materikin, Eduard T. Ambarchyan, Roman V. Epishev, and Maria A. Nefedova. "Epidermolysis Bullosa Acquisita in Children: Case Series." Current Pediatrics 18, no. 1 (2019): 56–64. http://dx.doi.org/10.15690/vsp.v18i1.1992.

Full text

Abstract:

Background. Epidermolysis bullosa acquisita (EBA) is chronic disease accompanied with subepidermal blistering on skin and mucous membranes as a result of autoimmune aggression to type VII collagen. EBA diagnostics in children is complicated due to similarity of clinical presentation with other bullous dermatosis in children.Clinical Case Description. The description of three clinical cases of EBA in children is provided. It is shown that for establishing the diagnosis it is necessary to estimate clinical evidence and to define the depth of blisters according to the results of histological exam

APA, Harvard, Vancouver, ISO, and other styles

35

Kollipara, Ramya, Christopher Downing, Michael Lee, Jacqueline Guidry, Samantha Robare-Stout, and Stephen Tyring. "Blistering Distal Dactylitis in an Adult." Journal of Cutaneous Medicine and Surgery 19, no. 4 (2015): 397–99. http://dx.doi.org/10.1177/1203475415574971.

Full text

Abstract:

Background: Blistering distal dactylitis is a localised infection of the anterior pad of the distal phalanx of the digits. It most commonly affects children and uncommonly adults with a history of immunosuppression or trauma. Objective: A case of blistering distal dactylitis in a 32-year-old male is reported. The patient was not immunocompromised and did not report trauma. Conclusion: Blistering distal dactylitis may rarely present in adults (including those who are not immunocompromised or reporting trauma) and thus should be in the differential diagnosis for an adult presenting with bullae a

APA, Harvard, Vancouver, ISO, and other styles

36

Akin Belli, Asli, Nazmi Geyik, and Emine Dervis. "A case of bullous scabies misdiagnosed as acquired epidermolysis bullosa." International Journal of Dermatology 54, no. 11 (2015): e499-e501. http://dx.doi.org/10.1111/ijd.12920.

Full text

APA, Harvard, Vancouver, ISO, and other styles

37

Paduano, Francesco, Emma Colao, Teresa Grillone, et al. "A Familial Form of Epidermolysis Bullosa Simplex Associated with a Pathogenic Variant in KRT5." Genes 12, no. 10 (2021): 1503. http://dx.doi.org/10.3390/genes12101503.

Full text

Abstract:

Epidermolysis bullosa simplex is a disease that belongs to a group of genodermatoses characterised by the formation of superficial bullous lesions caused by minor mechanical trauma to the skin. The skin fragility observed in the EBS is mainly caused by pathogenic variants in the KRT5 and KRT14 genes that compromise the mechanical stability of epithelial cells. By performing DNA sequencing in a female patient with EBS, we found the pathogenic variant c.967G>A (p.Val323Met) in the KRT5 gene. This variant co-segregated with EBS in the family pedigree and was transmitted in an autosomal dominan

APA, Harvard, Vancouver, ISO, and other styles

38

Krasnova, Nadezhda V., Tatyana A. Chernova, Irina V. Alekseeva, Geliya G. Gimalieva, Tatiana Misyakova, and Larisa G. Sinitsyna. "Clinical case of Bloch — Sulzberger syndrome." Vestnik dermatologii i venerologii 96, no. 3 (2020): 63–67. http://dx.doi.org/10.25208/vdv1117.

Full text

Abstract:

Purpose.To present a clinical case of Bloch Sulzberger syndrome. Material and methods. The examinations were performed to diagnose the disease: а visual examination of the skin, cytological analysis of the gallbladder fluid, general and biochemical blood tests, genetic research. Results.During a visual examination of the skin, a differential diagnosis was made with infectious dermatitis, toxic-allergic dermatitis, epidermolysis bullosa and linear IgA-dependent dermatosis in children. Crucial in the diagnosis belonged to a genetic study, after which a deletion of exons 410 of the IK

APA, Harvard, Vancouver, ISO, and other styles

39

Agrawal, P., R. George, M. Thomas, et al. "A childhood subepidermal autoimmune bullous disease resembling mechanobullous epidermolysis bullosa acquisita." British Journal of Dermatology 173, no. 3 (2015): 871–74. http://dx.doi.org/10.1111/bjd.13833.

Full text

APA, Harvard, Vancouver, ISO, and other styles

40

Mooney, Ellen. "Studies on Complement Deposits in Epidermolysis Bullosa Acquisita and Bullous Pemphigoid." Archives of Dermatology 128, no. 1 (1992): 58. http://dx.doi.org/10.1001/archderm.1992.01680110068008.

Full text

APA, Harvard, Vancouver, ISO, and other styles

41

Mooney, E. "Studies on complement deposits in epidermolysis bullosa acquisita and bullous pemphigoid." Archives of Dermatology 128, no. 1 (1992): 58–60. http://dx.doi.org/10.1001/archderm.128.1.58.

Full text

APA, Harvard, Vancouver, ISO, and other styles

42

Premalatha, BR, Roopa S. Rao, and Vijaya Mysorekar. "Immunofluorescence in Oral Pathology—Part II: Pathology and Immunofluorescent Patterns in Subepidermal Immunobullous Disorders." World Journal of Dentistry 3, no. 1 (2012): 68–73. http://dx.doi.org/10.5005/jp-journals-10015-1130.

Full text

Abstract:

ABSTRACT The immunobullous disorders are a group of autoimmune diseases in which components of the epidermis and basement membrane zone are targeted, resulting in the formation of cutaneous and mucosal blisters. Based on the level of blistering, the autoimmune blistering diseases may be subdivided into intraepidermal and subepidermal. An exhaustive list of immunobullous disorders is beyond the scope of this review, but those involving oral mucosa are taken into consideration. One major group namely the subepidermal immunobullous diseases which includes bullous pemphigoid (BP), mucosal pemphigo

APA, Harvard, Vancouver, ISO, and other styles

43

Setyowatie, Lita, Yustian Devika Rahmawati, Arif Widiatmoko, and Diah Prabawati Retnani. "Epidermolysis Bullosa Acquisita Occuring In A Patient With Systemic Lupus Erythematosus." Saintika Medika 15, no. 2 (2019): 100. http://dx.doi.org/10.22219/sm.vol15.smumm2.9884.

Full text

Abstract:

Epidermolysis Bullosa Acquisita (EBA) is a rare, chronic autoimmune subepidermal bullous disease and has been noted to be associated with systemic lupus erythematosus (SLE). The incidence of EBA and SLE in one patient within the period of 1980-1990 found only 7 published case reports. A 23 years old woman with exfoliate skin since 12 years ago. Initially itchy on her buttock then appeared small blister. Blister spread almost the entire body and rupture. This complaint got worsening in a year accompanied with hair loss, weight loss, and oral ulcer. Dermatological examination showed patch eritem

APA, Harvard, Vancouver, ISO, and other styles

44

Aalfs, A. Susanne, and Marcel F. Jonkman. "Tinea corporis bullosa due to Microsporum canis mimicking linear IgA bullous dermatosis." European Journal of Dermatology 22, no. 6 (2012): 805–6. http://dx.doi.org/10.1684/ejd.2012.1837.

Full text

APA, Harvard, Vancouver, ISO, and other styles

45

Neri, I., B. M. Piraccini, E. Guareschi, and A. Patrizi. "Bullous tinea pedis in two children. Bullose Tinea pedis bei zwei Kindern." Mycoses 47, no. 11-12 (2004): 475–78. http://dx.doi.org/10.1111/j.1439-0507.2004.01027.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

46

KAWACHI, Y., M. IKEGAMI, T. HASHIMOTO, K. MATSUMURA, T. TANAKA, and F. OTSUKA. "Autoantibodies to bullous pemphigoid and epidermolysis bullosa acquisita antigens in an infant." British Journal of Dermatology 135, no. 3 (1996): 443–47. http://dx.doi.org/10.1046/j.1365-2133.1996.d01-1018.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

47

KAWACHI, Y., M. IKEGAMI, T. HASHIMOTO, K. MATSUMURA, T. TANAKA, and F. OTSUKA. "Autoantibodies to bullous pemphigoid and epidermolysis bullosa acquisita antigens in an infant." British Journal of Dermatology 135, no. 3 (1996): 443–47. http://dx.doi.org/10.1111/j.1365-2133.1996.tb01511.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

48

MURDOCH, MICHELE E., and IRENE M. LEIGH. "(36) Mixed features of bullous ichthyosiform erythroderma and ichthyosis bullosa of Siemens." British Journal of Dermatology 125, s38 (1991): 64–65. http://dx.doi.org/10.1111/j.1365-2133.1991.tb05505.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

49

Pérez, A., N. Almaani, C. M. Stefanato, et al. "Bullous pemphigoid in a patient with suspected non-Herlitz junctional epidermolysis bullosa." Clinical and Experimental Dermatology 35, no. 8 (2010): 881–84. http://dx.doi.org/10.1111/j.1365-2230.2010.03828.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

50

Prost-Squarcioni, C., F. Caux, E. Schmidt, et al. "International Bullous Diseases Group: consensus on diagnostic criteria for epidermolysis bullosa acquisita." British Journal of Dermatology 179, no. 1 (2018): 30–41. http://dx.doi.org/10.1111/bjd.16138.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!