Academic literature on the topic 'Buruli, Ulcère de'
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Journal articles on the topic "Buruli, Ulcère de"
Mortier, Emmanuel, Anne Grasland, Raluca Sterpu, Comlan Affo, and Isabelle Mahé. "Ulcère creusant et indolore : ulcère de Buruli." La Presse Médicale 41, no. 9 (September 2012): 892–93. http://dx.doi.org/10.1016/j.lpm.2011.10.018.
Full textSouillac, G., F. N. Fitoussi, F. M. Fitoussi, G. F. Penneçot, and A. Bourrillon. "Ulcère de Buruli : une pathologie d’exportation." Archives de Pédiatrie 7, no. 12 (December 2000): 1311–15. http://dx.doi.org/10.1016/s0929-693x(00)00149-4.
Full textYedomon, G. H., F. Guedenon, A. Chauty, H. Adegbidi, F. Atadokpede, and F. Do Ango-Padonou. "C044 - Ulcère de Buruli : huit cas avec rechute." Annales de Dermatologie et de Vénéréologie 134, no. 1 (January 2007): 50–51. http://dx.doi.org/10.1016/s0151-9638(07)89076-4.
Full textKNIPPER, P., R. ZILLIOX, C. JOHNSON, and P. ANTOINE. "Ulcère de Buruli et chirurgie plastique, au dispensaire." Annales de Chirurgie Plastique Esthétique 49, no. 3 (June 2004): 265–72. http://dx.doi.org/10.1016/s0294-1260(04)00057-3.
Full textStoffel, V., B. Barthelmé, and Frédéric Chagué. "Écopathologie tropicale : ulcère de Buruli par monts et par vaux." Santé Publique 17, no. 2 (2005): 191. http://dx.doi.org/10.3917/spub.052.0191.
Full textKouassi, Y. I., K. C. Ahogo, B. Vagamon, M. Kaloga, H. S. kourouma, E. J. Ecra, A. Sangaré, et al. "Ulcère de Buruli : multiples lésions céphaliques après instauration du traitement médical." Annales de Dermatologie et de Vénéréologie 140 (April 2013): S30—S31. http://dx.doi.org/10.1016/j.annder.2013.01.400.
Full textAzanmasso, H., B. Addy Lolla, N. S. Diagne, G. T. Kpadonou, E. Alagnide, F. Lmidmani, and A. El Fatimi. "Intérêt du suivi des enfants opérés pour ulcère de Buruli au Bénin." Annals of Physical and Rehabilitation Medicine 56 (October 2013): e282-e283. http://dx.doi.org/10.1016/j.rehab.2013.07.728.
Full textGnassingbe, W., B. Saka, J. Teclessou, G. Mahamadou, S. Akakpo, A. Mouhari-Toure, E. Belei, Y. Elegbede, K. Kombaté, and P. Pitché. "F18 : Basidiobolomycose simulant un ulcère de Buruli chez un garçon de 5 ans." Annales de Dermatologie et de Vénéréologie 143, no. 4 (April 2016): S19. http://dx.doi.org/10.1016/s0151-9638(16)30125-9.
Full textVagamon, B., K. C. Ahogo, B. R. Aka, A. Diabaté, Y. I. Kouassi, S. H. Kourouma, C. Traoré, et al. "Ulcère de Buruli à début bifocal : multiples lésions céphaliques après instauration du traitement médical." Annales de Dermatologie et de Vénéréologie 140, no. 2 (February 2013): 125–28. http://dx.doi.org/10.1016/j.annder.2012.11.010.
Full textPradinaud, R. "C10 - L’infection à Mycobacterium ulcerans ne doit plus être restreinte à l’impropre appellation « ulcère de Buruli »." Annales de Dermatologie et de Vénéréologie 134, no. 1 (January 2007): 28–29. http://dx.doi.org/10.1016/s0151-9638(07)89031-4.
Full textDissertations / Theses on the topic "Buruli, Ulcère de"
Millogo, Anselme. "Le diagnostic des mycobactérioses cutanées en Afrique de l'Ouest." Thesis, Montpellier, 2020. http://www.theses.fr/2020MONTT030.
Full textCutaneous mycobacterioses remains prevalent in West Africa, despite the existence of treatments due to problems linked to early diagnosis. The aim of this thesis is to contribute to the improvement of their microbiological diagnosis. The results of the work of this thesis allow us to understand the epidemiology of cutaneous mycobacterioses in Burkina Faso, West Africa. This work is a first that focuses on the simultaneous microbiological diagnosis of Buruli ulcer, leprosy and cutaneous tuberculosis in Burkina Faso using innovative approaches. A review of the literature carried out on the microbiology of skin and subcutaneous ulcers in West Africa made it possible to identify the following objectives: i) to study the molecular prevalence of Mycobacterium ulcerans in clinical samples of chronic wounds in patients living in the rural areas of South West and West of Burkina Faso; ii) set up a microscopic technique for hybridization of fluorescent molecular probes for precise detection of Mycobacterium leprae in clinical skin samples; iii) used this technique to search in non-invasive samples (stool and nasal secretions) of Mycobacterium ulcerans in order to propose non-invasive methods of diagnosing leprosy in Burkina Faso, West Africa; iii) to put in place a microscopic staining technique making it possible to distinguish living, dormant or dead mycobacteria in clinical samples for monitoring treatments and to provide researchers with a tool for selecting samples eligible for attempts to culture Mycobacterium leprae. The clinical samples for the research of Mycobacterium ulcerans were made in Bobo-Dioulasso, in Djikologo (Diébougou) and in Bomborokuy (Nouna) that is 64 swabs of chronic skin wounds. As for skin samples (biopsies and skin incisions), they were carried out in the dermatology department of the “Centre Hospitalier Universitaire Souro Sanou de Bobo – Dioulasso”. Molecular tests carried out for Mycobacterium ulcerans targeting the following markers: IS2404, IS2606 and KR-B. A specific oligonucleotide probe designer on the role of Mycobacterium leprae and labeled with fluorochrome Alexa555 was used by Fluorescence microscopy - leprosy. Fluorescein diacetate, red Nile and DAPI were used for the characterization of Mycobacterium leprae according to the three stages: living, dynamic and dead. For Burkina Faso, there is a first molecular proof of the circulation of Mycobacterium ulcerans in chronic wounds of rural patients. A more specific microscopic tool made it possible to make a precise diagnosis which enabled the dermatologists of CHUSS to treat the sick. For clinicians, DDD-leprosy remains a tool for monitoring patients on anti-leprosy drugs. The researchers will find in DDD a tool to relaunch research on the culture of leprosy based on samples they have an overview of the proportion of living mycobacteria. These results confirm on the one hand a geographical extension of Buruli ulcer in West Africa and on the other hand call for a strengthening of efforts in the fight against leprosy in Burkina Faso, West Africa with the end a prospect of strengthening approaches to leprosy culture for scientists.Keywords: Cutaneous mycobacterioses / West Africa / Burkina Faso / Mycobacterium ulcerans / Mycobacterium leprae / FISH / DDD
Debacker, Martine M. J. "L'ulcère de Buruli dans un centre de santé rural au Bénin." Doctoral thesis, Universite Libre de Bruxelles, 2005. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/211042.
Full textUne première publication porte sur 1700 cas consécutifs admis au CSNG entre 1997 et 2001. Ces données nous ont permis d’illustrer l’évolution du nombre des cas d’UB au cours des années, par département et par sous-préfecture. Le nombre de patients UB qui se présentent au centre est fonction de différents facteurs comme par exemple l’ouverture d’autres centres de traitement, l’organisation de campagnes d’information sur l’UB mais aussi de changements dans l’environnement. Au niveau des formes de la maladie, nous avons montré que les formes non ulcérées sont aussi fréquentes que les formes ulcérées. La forme de la maladie est liée au délai à consulter le CSNG. Les atteintes osseuses sont fréquentes et sont présentes chez plus de 13% des cas. Au fil des années, le délai à consulter le CSNG ainsi que la durée de l’hospitalisation ont été fortement réduits, passant de 4 mois à 1 mois dans le premier cas et de 9 mois à 1 mois en ce qui concerne l’hospitalisation. Les personnes ressources à cibler lors des campagnes de sensibilisation sur la maladie ont été identifiées. Il s’agit des anciens malades, qui dans 68% des cas, réfèrent les nouveaux malades au CSNG.
Une deuxième publication, portant sur la même période, nous a permis de mettre en évidence pour la première fois des taux d’UB élevés chez les sujets de 60 ans et plus, probablement du fait d’une diminution de leur immunité. Chez les 60 ans et plus, les hommes sont plus à risque de développer un UB que les femmes. Par contre aucune différence dans le risque d’UB en fonction du sexe n’est trouvée chez les moins de 60 ans. Les lésions d’UB prédominent au niveau des membres inférieurs pour tous les groupes d’âge. Du fait de leur petite stature, les enfants ont une répartition des lésions qui touchent tout le corps. Chez les adultes, les femmes présentent plus de lésions que les hommes au niveau de la tête, du cou et du tronc. Les sujets de moins de 15 ans développent souvent des lésions multifocales, associées à des atteintes osseuses. Ces atteintes osseuses constituent les formes graves de la maladie.
Notre troisième publication porte sur le suivi des malades au niveau des villages, entre mars 2000 et février 2001, afin de déterminer les taux de récurrences de l’UB. Le taux de récurrence de la maladie est faible (6.1%) pour un temps de suivi des malades allant jusqu’à 7 ans. La majorité des malades soignés au CSNG étaient en bonne santé. Nous n’avons néanmoins pu retrouver qu’un nombre limité de malades.
Dans notre quatrième publication, nous avons pu mettre en évidence le développement d’un UB sur le site d’une morsure humaine. La surface de la peau du malade a pu être contaminée par M. ulcerans et la morsure (= traumatisme) a occasionné l’entrée de M. ulcerans dans le derme. Une autre explication plausible est que la morsure ait réactivé un foyer latent de M. ulcerans au niveau du site de la morsure.
Notre base de données nous a également permis de contribuer à d’autres études, notamment celles sur l’influence du BCG et de la schistosomiase sur l’UB. Dans les deux cas, nous trouvons une association significative avec les formes graves de l’UB, l’absence de vaccination BCG et la schistosomiase favorisant le développement de formes osseuses. Aucun lien n’a pu être établi entre la présence ou l’absence de l’infection à Schistosoma haematobium et l’UB.
Dans la dernière partie de notre travail, nous nous sommes attachés à l’étude de quelques facteurs de risque de l’UB. Nous avons montré que l’UB est essentiellement associé à l’âge, le lieu de résidence et le type d’eau utilisé. Pour les sujets de 5 ans et plus, le risque d’UB est plus élevé chez les sujets vaccinés à la naissance avec le BCG. L’analyse par strate d’âge des sujets âgés de 5 ans et plus, nous a permis de voir que dans le groupe des 50 ans et plus l’influence du type d’eau utilisé est moindre que dans les autres groupes d’âge, laissant envisager qu’un autre facteur entre en jeu :une réactivation de la maladie à la faveur d’une baisse d’immunité.
Notre étude démontre que la prise en charge de l’UB doit être pluridisciplinaire et doit pouvoir englober diverses interventions telles que :
- les campagnes d’information à la population,
- la formation du personnel sanitaire ainsi qu’une implication de l’état,
- une bonne prise en charge des malades au niveau des centres de traitement de l’UB en collaboration avec les laboratoires régionaux et internationaux,
- la confirmation microbiologique des cas,
- la mise en place d’un suivi des malades en s’appuyant sur les structures déjà mises en place pour d’autres maladies,
- la mise à disposition des populations de sources d’eau potable (puits, pompes) qui par la même occasion permettront de limiter/diminuer d’autres affections liées à la consommation d’eau non potable,
- la poursuite des activités de recherche sur un éventuel vaccin ou une médication appropriée, ainsi que sur le réservoir de M. ulcerans afin de pouvoir mettre en place des stratégies de prévention de la maladie.
Doctorat en Sciences de la santé publique
info:eu-repo/semantics/nonPublished
Bouam, Amar. "Les sources et les réservoirs de Mycobacterium ulcerans, agent causal de l'ulcère de Buruli." Thesis, Aix-Marseille, 2018. http://www.theses.fr/2018AIXM0270.
Full textBuruli ulcer is a dermis, epidermis and sometimes bone infection leading to deformities and permanent disabilities. It is caused by Mycobacterium ulcerans, a mycobacterium associated to the aquatic ecosystems but its sources and reservoirs are not yet defined. Therefore, no prophylaxis is established. This thesis contribute to unmask the sources of contamination of M. ulcerans. My review has identified potential sources of M. ulcerans in the environment and highlighted the missing pieces for understanding the epidemiology of Buruli ulcer. My study on the role of amoeba in the survival of M. ulcerans in the environment, install M. ulcerans as susceptible to amoeba rendering amoeba an unlikely host of M. ulcerans. Hereafter, i studied carbon substrates metabolized by M. ulcerans strains. Literature survey indicated that the environmental sources of carbon substrates metabolized by M. ulcerans were bacteria, fungi, algae and mollusks. I therefore studied the interactions of M. ulcerans with fungi by testing the effect of mycolactones on fungi. Mycolactones showed an attraction effect on Mucor circinelloides. This observation suggest a novel role for mycolactones as chemoatractants to fungi. The mode of transmission of M. ulcerans to humans remains unknown. I showed that M. ulcerans DNA can be detected on the healthy skin of asymptomatic persons, suggesting an asymptomatic carriage. These data could help promote prophylaxis based on wearing protective clothing in contact with risky environments. In perspective to my thesis work, we set up a collaboration with the Buruli Ulcer Program (PNLUB) and Institut Pasteur Côte d'Ivoire to study the reservoirs of M. ulcerans
Meyin, A. Ebong Solange. "Punaises aquatiques du Cameroun : biodiversité, phylogéographie et rôle dans l’écologie de Mycobacterium ulcerans, agent étiologique de l’ulcère de Buruli." Thesis, Montpellier, 2015. http://www.theses.fr/2015MONTS273/document.
Full textOne of 17 neglected tropical diseases declared by WHO, Buruli ulcer is now a real public health problem in Central and West Africa, where it occurs in nearly 30 countries. This disease mainly affecting people in remote areas remains a myth because we don't know today the exact mode of Mycobacterium ulcerans transmission, the causal agent to humans. A better understanding of the ecology of this bacillus is an important lever to make the information about its transmission as the fight against this scourge. Strongly suspected to playing a role of host or vector of M. ulcerans, some aquatic bugs’ taxa seem to be predisposed by their ecology or behavior to harbor the bacillus or to convey it. The works presented here are intended firstly to identify on anatomical bases complemented by molecular approaches different taxa of aquatic bugs present in Cameroon. Beyond proposing an identification key of taxa and classification of aquatic bugs in Cameroon based on the nucleotide sequences, we discuss the important role of the ecology of some taxa of aquatic bugs in the transmission of M. ulcerans. Different comparative and biostatistics approaches developed in this thesis support the hypothesis that some taxa of water bugs, especially from the Belostomatidae and Naucoridae families acquire the mycobacteria from the environment by sharing habitats (attendance of emerged aquatic vegetation) where the bacilli proliferate, and feeding on prey such as molluscs and small fish themselves contaminated with the bacteria. The flight capacity of aquatic bugs do not appear to facilitate the acquisition of M. ulcerans but seems to be involved in their dissemination by contaminating new the environments. At the end of this thesis, we discuss the ecology of aquatic bugs’ communities and their macroecology in Cameroon, and examine the importance of sampling as an important component of any research work in ecology and evolution of infectious diseases
Vincent, Quentin. "Epidémiologie et génétique humaine de l’ulcère de Buruli." Thesis, Paris 5, 2014. http://www.theses.fr/2014PA05S019/document.
Full textBuruli ulcer (BU), caused by Mycobacterium ulcerans, is the third most frequent mycobacteriosis worldwide. It has been rapidly emerging in sub-Saharan African countries since 1980. Until now, knowledge of BU epidemiology relied on series of non laboratory-confirmed clinical cases. From 2005-2011, we recruited the current largest cohort of laboratory-confirmed cases (more than 1,200 patients) at the Pobe CDTUB, Benin, to describe the clinical epidemiology of the disease and to explore the genetic architecture of human susceptibility to BU. Typically, patients with BU were children (median age at diagnosis 12 years) presenting with a unique (96%) large (≥15 cm, 36%) ulcerative (66%) lesion of the lower limb (60%). Atypical clinical presentation of BU included osteomyelitis with no identifiable present or past BU skin lesions. The sex ratio of BU widely varied with age, with male patients accounting for 57% of patients aged 15 years and younger, but only 33% of those older than 15 years. Clinical presentation of BU was significantly dependent on age and sex. 9% male patients had BU osteomyelitis, whereas only 4% of female patients did. 1 year after treatment, 22% of patients with follow-up information presented with permanent functional sequelae. Presentation with oedema, osteomyelitis, or large (≥15 cm in diameter), or multifocal lesions was significantly associated with occurrence of permanent functional sequelae (OR 7•64, 95% CI 5•29–11•31) and operationally defines severe BU. When coinfected with HIV, patients had a significantly higher risk to develop severe BU (OR 2.77, IC95% [1.32-6.33]). We explored the genetic architecture of susceptibility to BU in both mendelian and complex genetic frameworks. The most severe case of the disease to have been treated at the Pobe CDTUB belonged to a consanguineous family in which the segregation of the phenotype was indicative of a recessive mendelian genetic defect. Genetic linkage analysis by homozygosity mapping suggested the implication of the beta-defensin locus on chromosome 8 in BU pathogenesis and lead to the identification of a homozygous deletion, which co-segregated perfectly with the disease in the family. In a complex genetics approach, we undertook a genome-wide association study, which involved the genotyping of more than 2 million SNPs (Illumina Omni2.5) in a cohort of 400 cases and 400 exposed controls. We identified many signals of interest. The replication study is ongoing. Understanding BU physiopathology is crucial to the development of efficient vaccines and drugs. Dissection of the genetic control of the infection by M. ulcerans by its human host therefore constitutes an indispensable step
Landier, Jordi. "Epidémiologie de l'ulcère de Buruli et de la transmission de Mycobacterium ulcerans au Cameroun : personnes, saisons, espaces et insectes." Thesis, Paris 6, 2014. http://www.theses.fr/2014PA066370/document.
Full textBuruli ulcer (BU) is a tropical disease caused by Mycobacterium ulcerans. It appears as cutaneous lesions causing severe morbidity and permanent incapacities in affected populations. BU occurs mainly in West and Central Africa, where it is found endemic in very localized regions. The transmission mode of M. ulcerans to humans remains mysterious, which, combined with the lack of basic epidemiological knowledge on the disease, hampers considerably the design of effective control strategies. The goals of this thesis were to identify the different risks associated with Buruli ulcer, and to contribute to the improvement of knowledge on the circulation and transmission of M. ulcerans. We studied two Buruli ulcer foci in Cameroon, and analyzed the individual, behavioral, spatial and temporal risk factors for BU. We described populations groups, environments and seasons at higher risk. We also engaged in entomological fieldwork to verify if M. ulcerans was circulating in the domestic environment, an hypothesis never tested in African endemic regions. We identified several taxa carrying M. ulcerans, and possible vector candidates among them.This work proposes an update of the knowledge on Buruli ulcer epidemiology in Cameroon, and several results can be transposed to other endemic regions, especially in Central or West Africa, where endemic areas present similar environments. These results could provide useful elements to design more efficient public health strategies against Buruli ulcer and to target them accurately to populations, places and persons at highest risk. These results finally suggest new leads for future research on the mode of transmission of M. ulcerans
Marion, Estelle. "Ecologie de M. Ulcerans et physiopathologie de l'ulcère de Buruli : rôle des punaises aquatiques et développement d'un modèle murin pour l'étude de la cicatrisation spontanée." Angers, 2012. http://www.theses.fr/2012ANGE0010.
Full textBuruli ulcer or M. Ulcerans infection is the third most common mycobacterial disease in the world. M. Ulcerans produces a toxin, mycolactone, which induces extensive destruction of the skin and soft tissues with the formation of large ulcers. Therapeutic and preventive arsenals are still limited. The mode of transmission of M. Ulcerans is unclear and the role of water bugs was evocated for 10 years without strong evidence in natura. Firstly, we conducted a large study in Cameroon with the aim to assess the role of water bugs as hosts and vectors of M. Ulcerans. Our results could be put to advantage for surveillance and prevention purposes. More precisely, our work suggests that the detection of M. Ulcerans in water bug saliva could be used as an environmental indicator of the risk of M. Ulcerans infection. Secondly, we have studied the infection control in an original mouse model that mimic all clinical disease stages in particular the spontaneous healing. Indeed, FVB/N mice are sensitive as all other mouse strains with respect to M. Ulcerans infection, but present a spontaneous healing response after the ulcerative stage. A fascinating observation was the dynamic of the bacterial load in healing tissues suggesting that mycolactone production is regulated. During the healing process, the loco-regional inflammatory response is important, while adaptive response seems to play a minor role. Altogether, these results represent new perspectives for the development of preventive and therapeutic strategies. Currently, the lack of a powerful tool to detect mycolactone remains an obstacle for bacilli-host interaction understanding. In this context, we realized the critical steps to obtain nucleotidic aptamer against mycolactone
Chany, Anne-Caroline. "Synthèse d'analogues des mycolactones, toxines de l'ulcère de Buruli." Thesis, Mulhouse, 2011. http://www.theses.fr/2011MULH4076.
Full textBuruli ulcer, a severe necrotizing skin disease caused by Mycobacterium ulcerans, is one of the most neglected tropical diseases. The World Health Organization has started in 1998 a Global Initiative against this disease. The pathogen belongs to the same family as the mycobacteria responsible for tuberculosis and leprosy and 6000 new cases are registered each year. Infection leads to extensive destruction of the skin and soft tissues with the formation of large ulcers usually on the legs or the arms and can caver up 15% of the skin surface. These effects are due to the presence of the bacterial toxins mycolactones, secreted by M ulcerans. Mycolactones are the first polyketides isolated from a human pathogen. Deciphering their functional interactions is of fundamental importance for the understanding and ultimately the control of this devastating mycobacterial infection. A Diverted Total Synthesis approach of mycolactones analogues has been developed and provides the first insights into their structure-activity relationship based on cytopathic assays on L929 fibroblasts
Saint-Auret, Sarah. "Synthèse totale de mycolactone A/B et d'analogues ciblés pour l'étude mécanistique de l'ulcère de Buruli." Thesis, Strasbourg, 2017. http://www.theses.fr/2017STRAF049.
Full textBuruli ulcer is a necrotizing skin disease present in more than thirty countries in the world, located mainly in West and Central Africa but also in Australia and in Japan. This infection is caused by Mycobacterium ulcerans (M. ulcerans) that secretes a macrolide toxin called mycolactone, which is the first polyketide isolated from a human pathogen. The disease is characterized by the formation of painless progressive necrotic lesions combined with a lack of acute inflammatory response, and mycolactone is known to be directly involved in the biological mechanism. To date no specific and completely efficient treatment of Buruli ulcer has been developed which correlates with the dramatic lack of understanding of the associated chemical and biological mechanisms. In this context, this research project aims at a better understanding of mycolactone A/B molecular interactions by using total synthesis as main tool. To this end, our research team has developed an efficient synthetic pathway allowing the preparation of the natural toxin and its differents analogues for purposes of their biological evaluation and fine-tuning our mechanical understanding of this infection
Mbondji, Wonje Christelle. "Paramètres épidémiologiques, immunologiques et entomologiques liés au mode de transmission à l'homme de Mycobacterium ulcerans, agent causal de l'ulcère de Buruli, dans une région endémique du Cameroun." Nantes, 2008. http://archive.bu.univ-nantes.fr/pollux/show.action?id=ef545ba1-b58e-48e7-ba57-c79f6748b80f.
Full textThe exact mechanism of transmission of Mycobacterium ulcerans, the causative agent of Buruli ulcer, an emerging tropical disease, remains elusive. An epidemiological and immunological investigation was carried out in an endemic area of Cameroon. A case-control study enabled to identify three protective factors: covering limbs during farming activities, use od bed nets and wound treatment. Given that water bugs can be reservoirs and vectors of M. Ulcerans, we have reported that exposure of laboratory mice to aquatic insect saliva could protect against M. Ulcerans lesion development. Furthermore we have observed in two cohorts of individuals in Benin and Cameroon distinct human immune signatures with a level of antibodies IgG higher for the safe subjects than Buruli patients and the specific recognition of several saliva molecules bound to M. Ulcerans
Books on the topic "Buruli, Ulcère de"
Pluschke, Gerd, and Katharina Röltgen, eds. Buruli Ulcer. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4.
Full textPluschke, Gerd. Buruli Ulcer: Mycobacterium Ulcerans Disease. Cham: Springer Nature, 2019.
Find full textPluschke, Gerd, and Katharina Röltgen. Buruli Ulcer: Mycobacterium Ulcerans Disease. Springer, 2019.
Find full textAgbenorku, P., L. Lehman, P. Saunderson, and V. Simonet. Buruli Ulcer: Prevention of Disability (Pod). World Health Organization, 2006.
Find full textGarchitorena, Andes, Matthew H. Bonds, Jean-Francois Guégan, and Benjamin Roche. Interactions between ecological and socio-economic drivers of Buruli ulcer burden in Sub-Saharan Africa: opportunities for an improved control. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789833.003.0014.
Full textRoche, Benjamin, Hélène Broutin, and Frédéric Simard. Afterword IV Case studies. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198789833.003.0024.
Full textPearson, Amber L. A retrospective, applied case study of environmental and host characteristics of Buruli ulcer patients in the Amansie West District, 1996-1999. 2001.
Find full textBook chapters on the topic "Buruli, Ulcère de"
Röltgen, Katharina, and Gerd Pluschke. "Buruli Ulcer: History and Disease Burden." In Buruli Ulcer, 1–41. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_1.
Full textRöltgen, Katharina, Israel Cruz, Joseph M. Ndung’u, and Gerd Pluschke. "Laboratory Diagnosis of Buruli Ulcer: Challenges and Future Perspectives." In Buruli Ulcer, 183–202. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_10.
Full textOmansen, Till Frederik, Tjip S. van der Werf, and Richard Odame Phillips. "Antimicrobial Treatment of Mycobacterium ulcerans Infection." In Buruli Ulcer, 203–20. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_11.
Full textJunghanss, Thomas. "Thermotherapy of Buruli Ulcer." In Buruli Ulcer, 221–26. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_12.
Full textKpeli, Grace Semabia, and Dorothy Yeboah-Manu. "Secondary Infection of Buruli Ulcer Lesions." In Buruli Ulcer, 227–39. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_13.
Full textO’Brien, Daniel P., Vanessa Christinet, and Nathan Ford. "Management of BU-HIV Co-infection." In Buruli Ulcer, 241–48. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_14.
Full textNichter, Mark. "Social Science Contributions to BU Focused Health Service Research in West-Africa." In Buruli Ulcer, 249–72. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_15.
Full textHausmann-Muela, Susanna, and Ann-Marie Sevcsik. "Transdisciplinary Research and Action to Stop Buruli Ulcer: A case Study from Philanthropy." In Buruli Ulcer, 273–87. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_16.
Full textTabah, Earnest Njih, Christian R. Johnson, Horace Degnonvi, Gerd Pluschke, and Katharina Röltgen. "Buruli Ulcer in Africa." In Buruli Ulcer, 43–60. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_2.
Full textJohnson, Paul D. R. "Buruli Ulcer in Australia." In Buruli Ulcer, 61–76. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-11114-4_3.
Full textConference papers on the topic "Buruli, Ulcère de"
Rui Hu, C. M. Queen, and G. Zouridakis. "Lesion border detection in Buruli ulcer images." In 2012 34th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2012. http://dx.doi.org/10.1109/embc.2012.6347210.
Full textHu, Rui, Courtney M. Queen, and George Zouridakis. "Detection of Buruli ulcer disease: Preliminary results with dermoscopic images on smart handheld devices." In 2013 IEEE Point-of-Care Healthcare Technologies (PHT). IEEE, 2013. http://dx.doi.org/10.1109/pht.2013.6461311.
Full textAnogwih, Joy ANURI. "Ecology of insect species in Buruli ulcer endemic and non-endemic parts of Nigeria, West Africa." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.109445.
Full textHu, Rui, CourtneyM Queen, and George Zouridakis. "A novel tool for detecting Buruli ulcer disease based on multispectral image analysis on handheld devices." In 2014 IEEE-EMBS International Conference on Biomedical and Health Informatics (BHI). IEEE, 2014. http://dx.doi.org/10.1109/bhi.2014.6864298.
Full text"Applying Satellite Imagery and Geospatial Techniques to Explore Patterns of Buruli Ulcer Prevalence in Central Cameroon." In GI_Forum 2014 - Geospatial Innovation for Society. Vienna: Austrian Academy of Sciences Press, 2015. http://dx.doi.org/10.1553/giscience2014s217.
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