Log in

Relevant bibliographies by topics / C-Met Kinase

Contents

Journal articles
Dissertations / Theses
Books
Book chapters
Conference papers
Reports

Academic literature on the topic 'C-Met Kinase'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'C-Met Kinase.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "C-Met Kinase"

1

Smotrov, Nadya, Anjili Mathur, Ilona Kariv, Christopher M. Moxham, and Nathan Bays. "Development of a Cell-Based Assay for Measurement of c-Met Phosphorylation Using AlphaScreenTM Technology and High-Content Imaging Analysis." Journal of Biomolecular Screening 14, no. 4 (2009): 404–11. http://dx.doi.org/10.1177/1087057109331803.

Full text

Abstract:

c-Met is a receptor tyrosine kinase (RTK) with a critical role in many fundamental cellular processes, including cell proliferation and differentiation. Deregulated c-Met signaling has been implicated in both the initiation and progression of human cancers and therefore represents an attractive target for anticancer therapy. Monitoring the phosphorylation status of relevant tyrosine residues provides an important method of assessing c-Met kinase activity. This report describes a novel assay to monitor c-Met phosphorylation in cells using Amplified Luminescent Proximity Homogeneous Assay (Alpha

APA, Harvard, Vancouver, ISO, and other styles

2

Xu, Xiangming, and Lei Yao. "Recent Patents on the Development of c-Met Kinase Inhibitors." Recent Patents on Anti-Cancer Drug Discovery 15, no. 3 (2020): 228–38. http://dx.doi.org/10.2174/1574892815666200630102344.

Full text

Abstract:

Background : Receptor Tyrosine Kinases (RTKs) play critical roles in a variety of cellular processes including growth, differentiation and angiogenesis, and in the development and progression of many types of cancer. Mesenchymal-Epithelial Transition Factor (c-Met) kinase is one of the types of RTKs and has become an attractive target for anti-tumor drug designing. c-Met inhibitors have a broad prospect in tumor prevention, chemotherapy, biotherapy, and especially in tumor resistance. Objective: The purpose of this article is to review recent research progress of c-Met inhibitors reported in p

APA, Harvard, Vancouver, ISO, and other styles

3

Chen, Zhengxia, Meibi Dai, Deheng Sun, et al. "Preclinical evaluation of WXSH0011, a selective small molecule dual inhibitor of FGFR/c-Met." Journal of Clinical Oncology 38, no. 15_suppl (2020): e15641-e15641. http://dx.doi.org/10.1200/jco.2020.38.15_suppl.e15641.

Full text

Abstract:

e15641 Background: FGFRs (fibroblast growth factor receptors) are a family of receptor tyrosine kinases. c-Met is a receptor tyrosine kinase with a high-affinity ligand, hepatocyte growth factor/scatter factor (HGF/SF). Aberrations in the FGFR pathway are associated with a variety of human cancers. c-Met and HGF have been implicated in carcinogenesis and metastatic tumor progression because of their ability to enhance angiogenesis, cancer cell proliferation, migration, and invasion. WXSH0011 is an orally bioavailable novel potent and selective FGFR/c-Met dual kinase inhibitor, which displayed

APA, Harvard, Vancouver, ISO, and other styles

4

al., G. "Synthesis of a c-Met Kinase Inhibitor." Synfacts 2010, no. 10 (2010): 1108. http://dx.doi.org/10.1055/s-0030-1258070.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Ma, P. C., S. Jiang, R. Du, S. Dietrich, Z. Tang, and J. A. Kern. "Modeling targeted inhibition of wild type and mutated c-MET receptor." Journal of Clinical Oncology 25, no. 18_suppl (2007): 14010. http://dx.doi.org/10.1200/jco.2007.25.18_suppl.14010.

Full text

Abstract:

14010 Background: c-MET belongs to the semaphorin superfamily of signaling proteins, containing three protein families (semaphorins, plexins, c-MET and RON) that have central roles in cell signaling. The c-MET receptor tyrosine kinase is involved in regulating cell growth/proliferation, survival, angiogenesis, cell scattering, cell motility and migration. Mutations in c-MET have been identified in various human cancers including lung cancer and papillary renal cell carcinomas. c-MET mutations occur within the extracellular seven-blade β-propeller fold sema domain (E168D, L229F, S325G, N375S),

APA, Harvard, Vancouver, ISO, and other styles

6

Otuokere, IE, KK Igwe, JF Amaku, and OV Ikpeazu. "Pharmacophore modelling of 2-chlorobenzimidazole and its specific docking to the active site c-Met Kinase: A search for potent c-Met Kinase inhibitor." Pharmaceutical and Chemical Journal 7, no. 4 (2020): 86–96. https://doi.org/10.5281/zenodo.13955732.

Full text

Abstract:

In this paper, we demonstrate how pharmacophore modelling was used to design the analogues of 2-chlorobenzimidazole and the application of molecular docking studies in the evaluation of the ligand affinity for the target.  The lead molecule (1-benzyl-2-chloro-1<em>H</em>-benzimidazole) had the highest docking score of -8.0 kcal/mol and was observed to interact with 17 amino acids within the pocket of c-Met Kinase (ALA159, VAL39, TYR32, MET144, ASN142, ARG141, ALA154, ASP155, ALA49, LYS51, LEU90, TYR92, MET93, GLY32, ILE, ASP97 and GLY96).  Meanwhile, the ADME characteristics of 1-ben

APA, Harvard, Vancouver, ISO, and other styles

7

Sachs, M., K. M. Weidner, V. Brinkmann, et al. "Motogenic and morphogenic activity of epithelial receptor tyrosine kinases." Journal of Cell Biology 133, no. 5 (1996): 1095–107. http://dx.doi.org/10.1083/jcb.133.5.1095.

Full text

Abstract:

Receptor tyrosine kinases play essential roles in morphogenesis and differentiation of epithelia. Here we examined various tyrosine kinase receptors, which are preferentially expressed in epithelia (c-met, c-ros, c-neu, and the keratin growth factor [KGF] receptor), for their capacity to induce cell motility and branching morphogenesis of epithelial cells. We exchanged the ligand-binding domain of these receptors by the ectodomain of trkA and could thus control signaling by the new ligand, NGF. We demonstrate here that the tyrosine kinases of c-met, c-ros, c-neu, the KGF receptor, and trkA, bu

APA, Harvard, Vancouver, ISO, and other styles

8

Bosse, Tanja, Julia Ehinger, Aleksandra Czuchra, et al. "Cdc42 and Phosphoinositide 3-Kinase Drive Rac-Mediated Actin Polymerization Downstream of c-Met in Distinct and Common Pathways." Molecular and Cellular Biology 27, no. 19 (2007): 6615–28. http://dx.doi.org/10.1128/mcb.00367-07.

Full text

Abstract:

ABSTRACT Activation of c-Met, the hepatocyte growth factor (HGF)/scatter factor receptor induces reorganization of the actin cytoskeleton, which drives epithelial cell scattering and motility and is exploited by pathogenic Listeria monocytogenes to invade nonepithelial cells. However, the precise contributions of distinct Rho-GTPases, the phosphatidylinositol 3-kinases, and actin assembly regulators to c-Met-mediated actin reorganization are still elusive. Here we report that HGF-induced membrane ruffling and Listeria invasion mediated by the bacterial c-Met ligand internalin B (InlB) were sig

APA, Harvard, Vancouver, ISO, and other styles

9

Ye, Qianwen, Chenggong Fu, and Jiazhong Li. "Studying the Binding Modes of Novel 2-Aminopyridine Derivatives as Effective and Selective c-Met Kinase Type 1 Inhibitors Using Molecular Modeling Approaches." Molecules 26, no. 1 (2020): 52. http://dx.doi.org/10.3390/molecules26010052.

Full text

Abstract:

The mesenchymal epithelial cell transforming factor c-Met, encoded by c-Met proto-oncogene and known as a high-affinity receptor for Hepatocyte Growth Factor (HGF), is one of the receptor tyrosine kinases (RTKs) members. The HGF/c-Met signaling pathway has close correlation with tumor growth, invasion and metastasis. Thus, c-Met kinase has emerged as a prominent therapeutic target for cancer drug discovery. Recently a series of novel 2-aminopyridine derivatives targeting c-Met kinase with high biological activity were reported. In this study, 3D quantitative structure-activity relationship (QS

APA, Harvard, Vancouver, ISO, and other styles

10

Tulasne, David, Réjane Paumelle, K. Michael Weidner, Bernard Vandenbunder, and Véronique Fafeur. "The Multisubstrate Docking Site of the MET Receptor Is Dispensable for MET-mediated RAS Signaling and Cell Scattering." Molecular Biology of the Cell 10, no. 3 (1999): 551–65. http://dx.doi.org/10.1091/mbc.10.3.551.

Full text

Abstract:

The scatter factor/hepatocyte growth factor regulates scattering and morphogenesis of epithelial cells through activation of the MET tyrosine kinase receptor. In particular, the noncatalytic C-terminal tail of MET contains two autophosphorylation tyrosine residues, which form a multisubstrate-binding site for several cytoplasmic effectors and are thought to be essential for signal transduction. We show here that a MET receptor mutated on the four C-terminal tyrosine residues, Y1311F, Y1347F, Y1354F, and Y1363F, can induce efficiently a transcriptional response and cell scattering, whereas it c

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "C-Met Kinase"

1

Wagner, Heidi. "Investigating post-translational modifications of c-Met in cancer." Thesis, Queensland University of Technology, 2020. https://eprints.qut.edu.au/203995/1/Heidi_Wagner_Thesis.pdf.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Lefebvre, Jonathan. "Etude des propriétés apoptotiques du récepteur tyrosine kinase Met." Thesis, Lille 1, 2011. http://www.theses.fr/2011LIL10151/document.

Full text

Abstract:

Le récepteur Met et son ligand l’HGF/SF sont essentiels pour le développement embryonnaire tandis que la dérégulation de la signalisation du récepteur Met est associée à la progression tumorale. Activé par son ligand, Met induit un large panel de réponses biologiques telles que la survie cellulaire, la migration ou la prolifération. En revanche, le fragment p40 Met issu du clivage du récepteur par les caspases participe à l’apoptose. Cette dualité fonctionnelle est caractéristique de la famille des récepteurs à dépendance. Bien que la signalisation positive des RTK soit bien décrite, les mécan

APA, Harvard, Vancouver, ISO, and other styles

3

ANGIONI, MARIA MADDALENA. "Response to treatment with chemical and biological inhibitors of c-met mutated form." Doctoral thesis, Università degli Studi di Cagliari, 2012. http://hdl.handle.net/11584/266057.

Full text

Abstract:

c-MET is a receptor tyrosine kinase that, after binding with its ligand, hepatocyte growth factor (HGF), activates many signaling pathways, driving proliferation, motility, migration and invasion. Although c-MET is important in the control of tissue homeostasis under normal physiological conditions, it has also been found to be aberrantly activated in human cancers via mutation, amplification or protein overexpression. Activating point mutations were identified in the kinase domain of MET, either in the germline of patients affected by hereditary papillary renal carcinoma (HPRC) or in spontan

APA, Harvard, Vancouver, ISO, and other styles

4

Durrant, Michael 1982. "Differential regulation of c-Cbl and Cbl-b ubiquitin ligases downstream of the Met receptor tyrosine kinase." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112619.

Full text

Abstract:

The Cbl family of E3 ubiquitin ligases are important negative regulators of multiple receptor and cytoplasmic tyrosine kinases, and participate in a wide variety of cellular processes. Uncoupling of Cbl-mediated negative regulation allows activated receptor tyrosine kinases such as the Met receptor to escape degradation, enhancing their oncogenic potential in vitro and in vivo. Despite the consequences of loss of Cbl-mediated negative regulation for human disease, little is known about the mechanisms regulating Cbl protein levels themselves.<br>In this thesis work, I demonstrate a differential

APA, Harvard, Vancouver, ISO, and other styles

5

Langford, Peter R. "c-Met Initiates Epithelial Scattering through Transient Calcium Influxes and NFAT-Dependent Gene Transcription." BYU ScholarsArchive, 2011. https://scholarsarchive.byu.edu/etd/3186.

Full text

Abstract:

Hepatocyte growth factor (HGF) signaling drives epithelial cells to scatter by breaking cell-cell adhesions and migrating as solitary cells, a process that parallels epithelial-mesenchymal transition. HGF binds and activates the c-Met receptor tyrosine kinase, but downstream signaling required for scattering remains poorly defined. This study addresses this shortcoming in a number of ways.A high-throughput in vitro drug screen was employed to identify proteins necessary in this HGF-induced signaling. Cells were tested for reactivity to HGF stimulation in a Boyden chamber assay. This tactic yie

APA, Harvard, Vancouver, ISO, and other styles

6

Ponzo, Marisa Grace 1980. "Gene expression profiling of Met receptor tyrosine kinase-induced mouse mammary tumors." Thesis, McGill University, 2009. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=115881.

Full text

Abstract:

Breast cancer is a heterogeneous disease comprised of distinct biological entities that correlate with diverse clinical outcomes. Gene expression profiling has divided this heterogeneity into luminal, ERBB2+ and basal molecular subtypes. Basal breast cancers are difficult to treat as they lack expression of candidates suitable for targeted therapies and are associated with poor outcome.<br>Elevated protein level of the hepatocyte growth factor receptor, MET, is observed in 20% of human breast cancers and correlates with poor prognosis. However, the role of MET in mammary tumorigenesis is poorl

APA, Harvard, Vancouver, ISO, and other styles

7

Asses, Yasmine. "Conception par modélisation et criblage in silico d'inhibiteurs du récepteur c-Met." Phd thesis, Université Henri Poincaré - Nancy I, 2011. http://tel.archives-ouvertes.fr/tel-00653609.

Full text

Abstract:

L'enjeu des travaux effectués au cours de cette thèse est l'extraction in silico de molécules potentiellement intéressantes dans le processus d'inhibition du récepteur tyrosine kinase c-Met. La faculté de cette protéine à interagir dans les phénomènes d'embryogenèse et de réparation tissulaires rendent son inhibition cruciale dans les traitements contre les développements tumoraux où c-Met se trouve impliquée. Dans ce but, la stratégie que nous avons employée implique l'utilisation de plusieurs méthodes in silico de conception rationnelle de médicaments. Nous avons utilisé comme support les mu

APA, Harvard, Vancouver, ISO, and other styles

8

Chabot, Thomas. "Modulation de l'activité du Rad51 par le récepteur tyrosine kinase c-Met dans la réparation des cassures double-brin de l'ADN." Thesis, Nantes, 2020. http://archive.bu.univ-nantes.fr/pollux/show.action?id=360755d5-6a18-407f-9af7-fe215a83747f.

Full text

Abstract:

L'instabilité génomique due à la dérégulation des voies de réparation de l'ADN peut être à l’initiation de cancer et entraîner par la suite une résistance à la chimiothérapie et à la radiothérapie. La compréhension de ces mécanismes biologiques est donc essentielle dans la lutte contre le cancer. RAD51 est la protéine centrale de la voie de réparation des cassures double-brin de l'ADN par recombinaison homologue. Cette réparation conduit à une réparation fidèle de l'ADN. L'activité recombinase de la protéine RAD51 est finement régulée par des modifications post- traductionnelles telles que la

APA, Harvard, Vancouver, ISO, and other styles

9

Mekki, Meriem Sarah. "Conséquences de l'hypoxie sur la régulation de la signalisation HGF/SF-MET." Thesis, Lille 2, 2015. http://www.theses.fr/2015LIL2S047/document.

Full text

Abstract:

Le récepteur à activité tyrosine kinase MET et son ligand le facteur de croissance des hepatocytes (Hepatocyte Growth Factor/Scattor Factor (HGF/SF)) sont essentiels pour la migration, la morphogenèse et la survie des cellules épithéliales. En plus de son implication et son importance physiologiques, la dérégulation de la signalisation de MET favorise la progression et l’invasion tumorales dans plusieurs types de cancers. Au sein des tumeurs, l’hypoxie est également un phénomène crucial qui induit une réponse adaptative menant à l’invasion, la métastase cancéreuses et la résistance aux traitem

APA, Harvard, Vancouver, ISO, and other styles

10

Simonneau, Claire. "Mécanismes d'activation du récepteur tyrosine kinase MET par son ligand l'HGF/SF : rôles des domaines N et K1." Thesis, Lille 2, 2015. http://www.theses.fr/2015LIL2S071.

Full text

Abstract:

L’HGF/SF (Hepatocyte Growth Factor/Scatter Factor) est le ligand du Récepteur Tyrosine Kinase (RTK) MET. Ce couple ligand-récepteur joue un rôle essentiel dans de nombreux processus biologiques tels que l’embryogenèse, la régénération tissulaire et l’angiogenèse. Comme pour de nombreux RTK, la dérégulation de l’activité de MET est associée à la progression et l’invasion tumorales. Bien que le récepteur MET ait été intensivement étudié au cours de ces dernières décennies, les processus moléculaires conduisant à son activation par l’HGF/SF restent encore mal connus et controversés.NK1, un varian

APA, Harvard, Vancouver, ISO, and other styles

More sources

Books on the topic "C-Met Kinase"

1

Gentlemen's disagreement: Alfred Kinsey, Lewis Terman, and the sexual politics of smart men. The University of Chicago Press, 2013.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

2

Hegarty, Peter. Gentlemen's Disagreement: Alfred Kinsey, Lewis Terman, and the Sexual Politics of Smart Men. University of Chicago Press, 2013.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

3

Hegarty, Peter. Gentlemen's Disagreement: Alfred Kinsey, Lewis Terman, and the Sexual Politics of Smart Men. University of Chicago Press, 2013.

Find full text

APA, Harvard, Vancouver, ISO, and other styles

4

Scolieri, Paul A. Ted Shawn. Oxford University Press, 2019. http://dx.doi.org/10.1093/oso/9780199331062.001.0001.

Full text

Abstract:

This book is the first critical biography of Ted Shawn (1891–1972), the self-proclaimed “Father of American Dance.” Based on extensive archival research, it offers an in-depth examination of Shawn’s pioneering role in the formation of Denishawn (the first American modern dance company and school), Ted Shawn and His Men Dancers (the first all-male dance company), and Jacob’s Pillow (the internationally renowned dance festival and school located in the Berkshire Hills of Massachusetts). For many years and with great frustration, Shawn attempted to tell the story of his life’s work in terms of it

APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "C-Met Kinase"

1

Tang, Chad, M. Angelica Cortez, David Hong, and James W. Welsh. "Targeting the c-Met Kinase." In Targeted Therapy in Translational Cancer Research. John Wiley & Sons, Inc., 2015. http://dx.doi.org/10.1002/9781118468678.ch34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Lokker, Nathalie A., Melanie R. Mark, and Paul J. Godowski. "Structure-Function Analysis of Hepatocyte Growth Factor and its Tyrosine-Kinase Receptor c-Met." In Tyrosine Phosphorylation/Dephosphorylation and Downstream Signalling. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-78247-3_8.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Birchmeier, Carmen, Friedhelm Bladt, and Tomoichiro Yamaai. "The Functions of HGF/SF and its Receptor, the c-Met Tyrosine Kinase, in Mammalian Development." In Ciba Foundation Symposium 212 - Plasminogen-Related Growth Factors. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470515457.ch11.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Cutinho, Pretisha Flora, C. H. S. Venkataramana, and B. V. Suma. "In Silico Hit Identification, Drug Repurposing, Pharmacokinetic and Toxicity Prediction of c-Met Kinase Inhibitors for Cancer Therapy." In Special Publications. Royal Society of Chemistry, 2019. http://dx.doi.org/10.1039/9781839160783-00054.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Sha’afi, Ramadan I., Mario Volpi, and Paul H. Naccache. "Differences between the Effects of f-Met-Leu-Phe and Leukotriene B4 on Phosphoinositide Turnover and Their Relationship to Calcium Mobilization and Protein Kinase C Activation." In Prostaglandins, Leukotrienes, and Lipoxins. Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4946-4_23.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Comoglio, P. M., and A. Graziani. "Hepatocyte Growth Factor (HGF) Receptor." In Guidebook to Cytokines and Their Receptors. Oxford University PressOxford, 1995. http://dx.doi.org/10.1093/oso/9780198599470.003.0056.

Full text

Abstract:

Abstract The HGF receptor was identified as the product of the proto-oncogene c-Met, the pl 90Met protein (Bottaro et al. 1991; Naldini et al. 1991a,b). p190Met is composed of a 50 kDa (a) chain disulphide linked to a 145 kDa (/J) chain in an apcomplex of 190 kDa (GenBank accession number X54559) (Giordano et al. 1989a; Ponzetto et al. 1991). Both the a chain and the p chain are exposed at the cell surface; the pchain contains a 23-amino-acid membrane-spanning segment and an intracellular region containing the tyrosine kinase domain.

APA, Harvard, Vancouver, ISO, and other styles

7

El Sayed, Khalid A. "S-(−)-Oleocanthal as a c-Met receptor tyrosine kinase inhibitor and its application to synergize targeted therapies and prevent breast cancer recurrence." In Olives and Olive Oil in Health and Disease Prevention. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-12-819528-4.00037-7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Kursunluoglu, Gizem, Duygu Erdogan, Elcin Cagatay, et al. "The Role of Kinase Inhibitors in Cancer Therapies." In Protein Kinases - Promising Targets for Anticancer Drug Research. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99070.

Full text

Abstract:

Protein kinases are enzymes that transfer a phosphate group to the threonine, serine, or tyrosine residues of the target protein, regulating its activity. The activity of these enzymes are very important and strictly regulated in the cell as they promote cell proliferation, survival, and migration. In the case of any dysregulation of these enzymes, they can be associated with cancer initiation and progression. Small-molecule kinase inhibitors approved by the FDA for their improved clinical benefits are currently used in targeted therapy for the treatment of various cancers. So far, there are 6

APA, Harvard, Vancouver, ISO, and other styles

9

Lan, Sheng-Hui, Shan-Ying Wu, Giri Raghavaraju, Nan-Haw Chow, and Hsiao-Sheng Liu. "The Crosstalk of c-MET with Related Receptor Tyrosine Kinases in Urothelial Bladder Cancer." In Advances in the Scientific Evaluation of Bladder Cancer and Molecular Basis for Diagnosis and Treatment. InTech, 2013. http://dx.doi.org/10.5772/53718.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Chen, Chengxin, Hui Lin, and Zixia Li. "Analysis of Two Kinds of Rheological Properties Data of Asphalt." In Advances in Frontier Research on Engineering Structures. IOS Press, 2023. http://dx.doi.org/10.3233/atde230238.

Full text

Abstract:

In order to study the rutting factor G*/sin δ and complex shear modulus G*, two kinds of rheological performance data obtained under asphalt temperature scanning, this paper performs regression analysis on the relevant empirical formulas of these two kinds of data, using the determination coefficient R2 and the comparison of the numerical magnitude of the fitted and true values of the regression against the error value Δ, and selects a reasonable formula for the next analysis and research, and uses a small number of known measurement point data of two different temperature spans for regression

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "C-Met Kinase"

1

Masuyama, Fujimitsu. "R&D Program for A-USC Material Development with Creep Strength/Degradation Assessment Studies." In AM-EPRI 2010, edited by D. Gandy, J. Shingledecker, and R. Viswanathan. ASM International, 2010. http://dx.doi.org/10.31399/asm.cp.am-epri-2010p0011.

Full text

Abstract:

Abstract Recently advanced ultra-super critical (A-USC) pressure power plants with 700°C class steam parameters have been under development worldwide. Japanese material R&D program for A- USC beside the plant R&D program started in 2008, launched in 2007 under the METI/NEDO foundation includes not only alloy design explores and novel ideas for developing new steels and alloys that can fill critical needs in building 700°C class advanced power plants, but also fundamental studies on creep strength and degradation assessment, which are absolutely needed to assure the long-term safe use o

APA, Harvard, Vancouver, ISO, and other styles

2

Yun, Chang-Soo, Sung Yun Cho, Hyoung Rae Kim, et al. "Abstract A287: Discovery of c-Met kinase inhibitors for anticancer therapeutics." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Oct 19-23, 2013; Boston, MA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1535-7163.targ-13-a287.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Vakkalanka, Swaroop, Meyyappan Muthuppalaniappan, Babu Govindarajulu, et al. "Abstract 1789: Preclinical profile of novel and potent c-Met kinase inhibitors." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1789.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Ai, Jing, ZhengSheng Zhan, Ying Wang, et al. "Abstract B285: A novel, potent, highly selective inhibitor of c-Met kinase, Simm559, inhibits c-Met-dependent neoplastic phenotypes in vitro and in vivo." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Oct 19-23, 2013; Boston, MA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1535-7163.targ-13-b285.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Etnyre, Deven, Jason Fong, Ceyda Bertram, and Neelu Puri. "Abstract 1788: Mechanism of resistance to c-Met tyrosine kinase inhibitors in human melanoma." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1788.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Jacobs, Ryan, Jason Fong, Ryan Bomgarden, John Rogers, and Neelu Puri. "Abstract 1890: Novel targets mediating resistance to EGFR/c-Met tyrosine kinase inhibitors in NSCLC." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1890.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Janetka, James W., Manjula Agarwal, and Darin E. Jones. "Abstract LB-197: Hepatocyte Growth Factor Activator (HGFA) Inhibitors of c-MET/RON Kinase Signaling." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-lb-197.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Schadt, Oliver, Friedhelm Bladt, Andree Blaukat, et al. "Abstract 5777: EMD 1214063, an exquisitely selective c-Met kinase inhibitor in clinical phase 1." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5777.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Jacobs, Ryan, Jason Fong, David Moravec, et al. "Abstract 5653: Novel targets mediating resistance to EGFR/c-Met tyrosine kinase inhibitors in NSCLC." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5653.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Kaplan-Lefko, Paula, Karen Rex, Yihong Zhang, et al. "Abstract 3558: In vitro and in vivo profiling of class I and class II ATP-competitive c-Met kinase inhibitors defines potential c-Met-specific sensitivity biomarkers." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-3558.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Reports on the topic "C-Met Kinase"

1

Lunn, Pete, Marek Bohacek, Jason Somerville, Áine Ní Choisdealbha, and Féidhlim McGowan. PRICE Lab: An Investigation of Consumers’ Capabilities with Complex Products. ESRI, 2016. https://doi.org/10.26504/bkmnext306.

Full text

Abstract:

Executive Summary This report describes a series of experiments carried out by PRICE Lab, a research programme at the Economic and Social Research Institute (ESRI) jointly funded by the Central Bank of Ireland, the Commission for Energy Regulation, the Competition and Consumer Protection Commission and the Commission for Communications Regulation. The experiments were conducted with samples of Irish consumers aged 18-70 years and were designed to answer the following general research question: At what point do products become too complex for consumers to choose accurately between the good ones

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!