Relevant bibliographies by topics / Calcium channel drugs

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Calcium channel drugs'

Author: Grafiati
Published: 10 December 2022
Last updated: 31 July 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Calcium channel drugs.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Calcium channel drugs"

1

Triggle, David J. "Calcium-Channel Drugs." Journal of Cardiovascular Pharmacology 18 (1991): S1—S6. http://dx.doi.org/10.1097/00005344-199106191-00002.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Triggle, David J. "Calcium-Channel Drugs." Journal of Cardiovascular Pharmacology 18 (1991): S1—S6. http://dx.doi.org/10.1097/00005344-199118101-00002.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Triggle, David J. "Calcium, calcium channels, and calcium channel antagonists." Canadian Journal of Physiology and Pharmacology 68, no. 11 (1990): 1474–81. http://dx.doi.org/10.1139/y90-224.

Full text
Abstract:
Voltage-dependent Ca2+ channels are an important pathway for Ca2+ influx in excitable cells. They also represent an important site of action for a therapeutic group of agents, the Ca2+ channel antagonists. These drugs enjoy considerable use in the cardiovascular area including angina, some arrhythmias, hypertension, and peripheral vascular disorders. The voltage-dependent Ca2+ channels exist in a number of subclasses characterized by electrophysiologic, permeation, and pharmacologic criteria. The Ca2+ channel antagonists, including verapamil, nifedipine, and diltiazem, serve to characterize th
APA, Harvard, Vancouver, ISO, and other styles
4

MILLER-HANCE, WANDA C. "Calcium Channel-Blocking Drugs." Archives of Pediatrics & Adolescent Medicine 140, no. 12 (1986): 1216. http://dx.doi.org/10.1001/archpedi.1986.02140260018012.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Chen, Xinzhe. "Research progress on calcium-activated potassium ion channels." Theoretical and Natural Science 62, no. 1 (2024): 160–65. http://dx.doi.org/10.54254/2753-8818/62/20241522.

Full text
Abstract:
Abstract. Potassium ion channels are diverse and widely distributed, playing a key role in a variety of physiological and pathological processes. According to the size of the conductance, they can be divided into large conductive calcium activated potassium channel (BK channel), medium conductive calcium activated potassium channel (IK channel) and small conductive calcium activated potassium channel (SK channel). In recent years, remarkable progress has been made in studying these three ion channels, and a series of therapeutic drugs have emerged. According to the single channel conductance,
APA, Harvard, Vancouver, ISO, and other styles
6

Schoenfeld, N., J. Aelion, Y. Beigel, O. Epstein, and A. Atsmon. "The porphyrogenic effects of calcium channel blocking drugs." Clinical Science 69, no. 5 (1985): 581–86. http://dx.doi.org/10.1042/cs0690581.

Full text
Abstract:
1. Treatment of monolayers of chick embryo hepatocytes with the calcium channel blocking drugs nifedipine and verapamil resulted in a decrease in the activity of uroporphyrinogen decarboxylase, an increase in the activity of δ-aminolaevulinate synthase and accumulation of porphyrins with uroporphyrin and heptacarboxylic porphyrin predominating. 2. Diltiazem, another calcium channel blocking drug, did not affect uroporphyrinogen decarboxylase activity and had a slight effect only on the accumulation of porphyrins. 3. Experiments with nifedipine and verapamil in the presence of various concentra
APA, Harvard, Vancouver, ISO, and other styles
7

Muralinath, E., Devi Pooja, Chbukdhara Prasanta, et al. "Drugs Acting on Arrhythmics." Journal of Advanced Research and Reviews in Virology & Microbiology 1, no. 1 (2024): 10–12. https://doi.org/10.5281/zenodo.10716057.

Full text
Abstract:
<em>Antiarrhythmic drugs play a key role regarding abnormal heart rhythms. Anti-arrhythmic drugs are divided into different classes dependent on their primary mechanism of action. These drugs are regarded as into four main categories such as class I (sodium channel blockers), class II(beta _ blockers), class III (potassium channel blockers) and class IV (calcium channel blockers). Class I anti arrhythmics specifically act on sodium channels, which play a role in the initiation and conduction of action potentials particularly in Cardiac cells. Class II anti arrhythmics act by decreasing the eff
APA, Harvard, Vancouver, ISO, and other styles
8

CHO, CORNELIA. "Calcium Channel-Blocking Drugs-Reply." Archives of Pediatrics & Adolescent Medicine 140, no. 12 (1986): 1216. http://dx.doi.org/10.1001/archpedi.1986.02140260018013.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

White, Pamela. "Calcium Channel Blockers." AACN Advanced Critical Care 3, no. 2 (1992): 437–46. http://dx.doi.org/10.4037/15597768-1992-2015.

Full text
Abstract:
Calcium channel blockers are widely used in the treatment of ischemic heart disease, hypertension, and supraventricular tachycardia. The prototype agents, verapamil, nifedipine, and diltiazem, represent three classes of calcium channel blockers, each of which has different pharmacologic effects. Nifedipine and the other dihydropyridines primarily are vasodilators and have no clinical effects on cardiac conduction or contractility. Diltiazem and verapamil also are vasodilators, but they possess, to varying degrees, negative inotropic, chronotropic, and dromotropic effects. Side effects of these
APA, Harvard, Vancouver, ISO, and other styles
10

Qiu, Yong, Chiyu Ma, Nan Jiang, et al. "A Silicon-Based Field-Effect Biosensor for Drug-Induced Cardiac Extracellular Calcium Ion Change Detection." Biosensors 14, no. 1 (2023): 16. http://dx.doi.org/10.3390/bios14010016.

Full text
Abstract:
Calcium ions participate in the regulation of almost all biological functions of the body, especially in cardiac excitation–contraction coupling, acting as vital signaling through ion channels. Various cardiovascular drugs exert their effects via affecting the ion channels on the cell membrane. The current strategies for calcium ion monitoring are mainly based on fluorescent probes, which are commonly used for intracellular calcium ion detection (calcium imaging) and cannot achieve long-term monitoring. In this work, an all-solid-state silicone–rubber ion-sensitive membrane was fabricated on l
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Calcium channel drugs"

1

Young, Lois-May. "Evaluation of polycyclic amines as modulators of calcium homeostasis in models of neurodegeneration / Young L." Thesis, North-West University, 2012. http://hdl.handle.net/10394/7591.

Full text
Abstract:
Compromised calcium homeostasis in the central nervous system (CNS) is implicated as a major contributor in the pathology of neurodegeneration. Dysregulation of Ca2+ homeostasis initiates downstream Ca2+–dependent events that lead to apoptotic and/or necrotic cell death. Increases in the intracellular free calcium concentration ([Ca2+]i) may be the result of Ca2+ influx from the extracellular environment or Ca2+ release from intracellular Ca2+ stores such as the endoplasmic reticulum (ER). Influx from the extracellular environment is controlled predominantly by voltage gated calcium channels (
APA, Harvard, Vancouver, ISO, and other styles
2

Zhang, Changfeng. "Investigation of the endoplsmic reticulum calcium stores for their potential roles in neuroprotection using the NG115-401L neuronal cell line model." Scholarly Commons, 2014. https://scholarlycommons.pacific.edu/uop_etds/142.

Full text
Abstract:
There is significant interest in the field of neuroscience to gain a better understanding of how neurons die in neurodegenerative diseases such as Alzheimer's and Parkinson's diseases. We have used the neuronal cell line NG115-401L with unique calcium signaling characteristics to test the hypothesis that improving calcium loading into the endoplasmic reticulum (ER) to increase ER calcium levels acts as a possible neuroprotective response. We approached this problem using both pharmacological and genetic approaches targeting the central mediator of calcium uptake in the ER localized sarco/endop
APA, Harvard, Vancouver, ISO, and other styles
3

Kannancheri, Puthooru Dheeraj. "Calcium Channels in Prostate Cancer Drug Resistance." Electronic Thesis or Diss., Université de Lille (2022-....), 2025. https://pepite-depot.univ-lille.fr/ToutIDP/EDBSL/2025/2025ULILS100.pdf.

Full text
Abstract:
Malgré des avancées scientifiques significatives, la guérison du cancer reste une question complexe. Cela est dû non seulement à l'hétérogénéité des tumeurs, mais aussi à l'émergence d'une résistance à la plupart, sinon tous les traitements anticancéreux. Le cancer de la prostate (PCa), qui représente le deuxième cancer le plus fréquent chez les hommes, n'est pas différent: les thérapies conventionnelles, y compris l'hormonothérapie et la chimiothérapie, échouent face à des tumeurs réfractaires. Cette situation rend nécessaire l'élaboration de nouvelles stratégies pour lutter contre ce problèm
APA, Harvard, Vancouver, ISO, and other styles
4

Ruchala, Iwona. "EXPANDING MONOAMINE TRANSPORTERS PHARMACOLOGY USING CALCIUM CHANNELS." VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/5032.

Full text
Abstract:
Research in drug development meets many challenges including lengthy, complex and costly procedures to identify novel pharmacotherapies. In our lab, we developed a method for fast screening of small molecules that interact with monoamine transports – dopamine and serotonin (DAT, SERT). These membrane proteins play important roles in brain neurotransmission responsible for cognition, motion and pleasure. Dysfunction in dopaminergic and serotonergic systems result in neurological disorders such as depression, Attention Deficit Hyperactivity Disorder (ADHD), schizophrenia and addiction. DAT and S
APA, Harvard, Vancouver, ISO, and other styles
5

Dowell, Margaret Anne. "Influence of three-tier cost sharing on patient compliance with and switching of cardiovascular medications." Columbus, Ohio : Ohio State University, 2002. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1030118543.

Full text
Abstract:
Thesis (M.S.--Ohio State University, 2002.<br>Title from first page of PDF file. Document formatted into pages; contains xvi, 173 p.: ill. Includes abstract and vita. Co-advisors: Craig A. Pedersen, Dept. of Pharmacy; Anne Scheck McAlearney, School of Public Health. Includes bibliographical references (p. 169-173).
APA, Harvard, Vancouver, ISO, and other styles
6

Herzinger, Thomas Andreas. "Effects of the Cardioprotective Drugs Dexrazoxane and ADR-925 on Doxorubicin Induced Ca2+ Release from the Sarcoplasmic Reticulum." PDXScholar, 1996. https://pdxscholar.library.pdx.edu/open_access_etds/5069.

Full text
Abstract:
The sarcoplasmic reticulum is the intramuscular organelle responsible for the regulation of cytoplasmic calcium levels in muscle. This thesis investigates the effects of the cardioprotective drug, dexrazoxane, and its metabolite ADR-925 on doxorubicin induced calcium release from skeletal sarcoplasmic reticulum. Doxorubicin is a widely used antineoplastic agent. One of the major side effects of doxorubicin usage is chronic cardiotoxicity. Doxorubicin is a potent activator of the calcium release mechanism from the SR. The interaction between doxorubicin and the calcium release channel has been
APA, Harvard, Vancouver, ISO, and other styles
7

Whittington, Miles A. "The ethanol withdrawal syndrome : a role for dihydropyridine-sensitive calcium channels in neural hyperexcitability states." Thesis, University of Bristol, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.279774.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Brown, Jason Peter. "The novel antiepileptic drug, gabapentin (Neurontin), binds to the α₂δ subunit of a voltage-dependent calcium channel". Thesis, University of Cambridge, 1996. https://www.repository.cam.ac.uk/handle/1810/252157.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Florent, Romane. "Intérêt de la modulation pharmacologique des voies de signalisation calcique pour restaurer le contrôle de l'apoptose dans les cancers ovariens chimiorésistants Inhibition of store-operated channels by carboxyamidotriazole sensitizes ovarian carcinoma cells to anti-BclxL strategies through Mcl-1 down-regulation Drug Repositioning of the α1-Adrenergic Receptor Antagonist Naftopidil: A Potential New Anti-Cancer Drug? Bim, Puma and Noxa upregulation by Naftopidil sensitizes ovarian cancer to the BH3-mimetic ABT-737 and the MEK inhibitor Trametinib". Thesis, Normandie, 2020. http://www.theses.fr/2020NORMC413.

Full text
Abstract:
Le sombre pronostic du cancer de l’ovaire s’explique notamment par une fréquence importante de résistance à la chimiothérapie conventionnelle présentée par les patientes. La mise en place de stratégies thérapeutiques alternatives à la chimiothérapie, mais aussi la découverte de biomarqueurs prédictifs de la réponse à ce traitement, représentent donc un enjeu majeur pour améliorer la prise en charge de cette pathologie. La chimiorésistance des cellules cancéreuses ovariennes s’explique principalement par leur résistance à l’apoptose, résultant d’un déséquilibre entre les membres pro- et anti-ap
APA, Harvard, Vancouver, ISO, and other styles
10

Olah, Mark E. "Effects of calcium channel blockade and intracellular calcium antagonism on endothelium-dependent responses of the isolated rat aorta and influence of the endothelium on drug action /." The Ohio State University, 1988. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487590702989006.

Full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Books on the topic "Calcium channel drugs"

1

Opie, Lionel H., and William A. Coetzee. Clinical Use of Calcium Channel Antagonist Drugs. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0863-8.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Opie, Lionel H. Clinical use of calcium channel antagonist drugs. Kluwer Academic Publishers, 1989.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
3

Opie, Lionel H. Clinical use of calcium channel antagonist drugs. 2nd ed. Kluwer, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
4

Japan-U.S.A. Symposium on Cardiovascular Drugs (1989 Kahuku, Hawaii). Recent advances in calcium channels and calcium antagonists: Proceedings of the Japan-U.S.A. Symposium on Cardiovascular Drugs. Edited by Yamada Kazuo and Shibata Shoji. Pergamon Press, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
5

1937-, Epstein Murray, and Loutzenhiser Rodger, eds. Calcium antagonists and the kidney. Hanley & Belfus, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
6

W, Tsien R., Clozel Jean-Paul, and Nargeot Joël, eds. Low-voltage-activated T-type calcium channels: Proceedings from the International Electrophysiology Meeting, Montpellier, 21-22 October 1996. Adis International, 1998.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
7

Albrecht, Fleckenstein, ed. Cardiovascular effects of dihydropyridine-type calcium antagonists and agonists. Springer-Verlag, 1985.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
8

Godfraind, T. Calcium channel blockers. Birkhauser Verlag, 2003.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
9

S, Meldrum Brian, Williams, Michael, 1947 Jan. 3-, and Princeton Drug Research Symposia (1st : 1989 : Princeton, N.J.), eds. Current and future trends in anticonvulsant, anxiety, and stroke therapy: Proceedings of a symposium held at Princeton, New Jersey, May 21-23, 1989. Wiley-Liss, 1990.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
10

Roberto, Robles Nicolás, ed. Calcium channel blockers and renal disease. Nova Science Publishers, 2009.

Find full text
APA, Harvard, Vancouver, ISO, and other styles
More sources

Book chapters on the topic "Calcium channel drugs"

1

Özkaya, Esen, and Kurtuluş Didem Yazganoğlu. "Calcium Channel Blockers." In Adverse Cutaneous Drug Reactions to Cardiovascular Drugs. Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-6536-1_8.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Tülümen, Erol, and Martin Borggrefe. "Modulation of Calcium Handling: Calcium-Channel Modulators." In Antiarrhythmic Drugs. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-34893-9_5.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Rudic, Boris, Erol Tülümen, and Martin Borggrefe. "Modulation of Calcium Handling—Calcium-Channel Modulators." In Antiarrhythmic Drugs. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-74046-6_6.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Merin, R. "Calcium Channel Blocking Drugs: New Developments." In Anesthesiology and the Heart. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1966-2_13.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Doyle, Austin E. "Calcium Channel Blocking Drugs as Antihypertensive Agents." In International Yearbook of Nephrology 1989. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-1673-2_5.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Coetzee, William A. "Channel-Mediated Calcium Current in the Heart." In Clinical Use of Calcium Channel Antagonist Drugs. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0863-8_1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Herbette, Leo G., Giovanni Gaviraghi, Robert J. Dring, Lawrence J. Briggs, and R. Preston Mason. "Interactions of Lacidipine and other Calcium Channel Drugs with Biological Membranes: A Structural Model for Receptor/Drug Binding Utilizing the Membrane Bilayer." In Calcium Antagonists. Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1725-8_3.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Opie, Lionel H., and William A. Coetzee. "Fundamental Properties: Mechanisms, Classification, Sites of Action." In Clinical Use of Calcium Channel Antagonist Drugs. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0863-8_2.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Opie, Lionel H., and William A. Coetzee. "Use and Comparative Properties of the Three Prototypical Calcium Antagonists in Ischemic Heart Disease, Including Recommendations Based on an Analysis of 41 Trials." In Clinical Use of Calcium Channel Antagonist Drugs. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0863-8_3.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Opie, Lionel H., and William A. Coetzee. "Use and Comparative Efficacy in Hypertension and Supraventricular Arrhythmias. Minor Indications." In Clinical Use of Calcium Channel Antagonist Drugs. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0863-8_4.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Calcium channel drugs"

1

Kakariqi (Pepa), Laerta, and Sonila Vito. "Utilization of vascular calcium channel blocker drugs in Primary Health Care in Albania during 2004-2014." In The 5th Virtual International Conference on Advanced Research in Scientific Areas. Publishing Society, 2016. http://dx.doi.org/10.18638/arsa.2016.5.1.815.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Azaševac, Tijana. "Arterial hypertension and chronic kidney disease - is everything under control?" In 7th International Congress of Cardionephrology KARNEF 2025. Punta Niš, 2025. https://doi.org/10.46793/karnef25.321a.

Full text
Abstract:
Hypertension is one of the most common diseases that affects the heart and blood vessels. The number of people suffering from chronic kidney disease is increasing, but this disease often remains unrecognized until advanced stages. Kidney diseases can be the causes of arterial hypertension, but this connection is bidirectional and hypertension often leads to the development of chronic kidney disease. Of the non-pharmacological measures, dietary Na+ restriction is particularly effective, while pharmacological treatment of arterial hypertension in these patients should be started with renin–angio
APA, Harvard, Vancouver, ISO, and other styles
3

Yalcin, Huseyin C., Mohamed A. Elrayess, Hadeel T. Al-Jighefee, Mahmoud Khatib A. A. Al-Ruweidi, Shamma Almuraikhy, and Hadi M. Yassine. "Soluble ACE2 and Angiotensin II levels Modulated in Hypertensive COVID-19 Patients treated with different Antihypertension Drugs." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0085.

Full text
Abstract:
Hypertension is a major risk factor and common comorbidity among severe Coronavirus Disease 2019 (COVID-19) patients. Prominent antihypertensive drugs, such as angiotensin-converting-enzyme inhibitors (ACEi) and angiotensin receptor blockers (ARB) can modulate the expression of angiotensin-converting enzyme 2 (ACE2) and may influence COVID-19 prognosis. Other classes of antihypertensive drugs, such as beta-blockers (BB) and calcium channel blockers (CCB) are associated with reduced mortality. Still, their effect on the circulating levels of ACE2 and angiotensin II, as well as the severity of C
APA, Harvard, Vancouver, ISO, and other styles
4

Miloradović, Vladimir, and Neda Ćićarić. "Approaches in the Treatment of Uncontrolled Hypertension." In 7th International Congress of Cardionephrology KARNEF 2025. Punta Niš, 2025. https://doi.org/10.46793/karnef25.332m.

Full text
Abstract:
Despite the availability of antihypertensive drugs, many patients worldwide have uncontrolled hypertension (1,2). In this regard, the term „resistant hypertension“ was introduced, with a prevalence of 10%-20% in hypertensive patients (3). According to the current guidelines of the European Association of Cardiology, hypertension is defined as resistant when the treatment strategy, which includes appropriate lifestyle measures and treatment with maximum or maximally tolerated doses of diuretics (thiazides and thiazide-like diuretics), blockers of the renin-angiotension-aldosterone system (RAAS)
APA, Harvard, Vancouver, ISO, and other styles
5

Krasnoshtanova, Alla, and Anastasiya Bezyeva. "DETERMINATION OF THE OPTIMAL CONCENTRATIONS OF PECTIN AND CALCIUM CHLORIDE FOR THE SYNTHESIS OF CHITOSAN-PECTIN MICROPARTICLES." In GEOLINKS Conference Proceedings. Saima Consult Ltd, 2021. http://dx.doi.org/10.32008/geolinks2021/b1/v3/09.

Full text
Abstract:
"The oral route of drug inclusion is the most convenient for the patient. In addition to ease of use, this method of drug inclusion has such advantages as non-invasiveness of inclusion, absence of complications during injection; comparative safety for the organism due to the passage of the active substance and auxiliary compounds through the gastrointestinal tract; the possibility of introducing larger doses of the drug at one time. However, despite the obvious advantages, the oral route of inclusion has a number of significant disadvantages that significantly limit its use for a number of dru
APA, Harvard, Vancouver, ISO, and other styles
6

Vaibavi, S. R., Rahul Vaippully, Basudev Roy, and Saumendra K. Bajpai. "Anti-hypertensive Calcium-Blocking Drugs Induce a Change in Viscoelasticity of Mcf-7 Cancer Cells." In ICBET 2020: 2020 10th International Conference on Biomedical Engineering and Technology. ACM, 2020. http://dx.doi.org/10.1145/3397391.3397420.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Silva, Vitoria Pimentel da, Laura Provenzi, Nicole Becker, et al. "Mesenchymal stem cells modulate the gene expression of T- type Calcium Channel Subunit Alpha 1G (Cav3.1) in acute phase of epilepsy." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.709.

Full text
Abstract:
Introduction: Temporal Lobe Epilepsy (TLE) is a disorder caused by neuronal electrical imbalance, clinically manifested by spontaneous and recurrent seizures1,2. Its pathogenesis involves channelopathies of calcium channels, which contributes to hyperexcitability and hypersynchrony in TLE3 . About 30% of patients do not respond to drug treatment4 , making it necessary to develop new therapeutic alternatives, such as cell therapy. This work aimed to evaluate the modulation of mesenchymal stem cells (MSCs) in the calcium channel CACNA1G (Cav3.1) gene expression. Methods: MSCs were extracted from
APA, Harvard, Vancouver, ISO, and other styles
8

Souza, Isabela Silva, Beatriz Cassarotti, Lucas de Oliveira Pinto Bertoldi, et al. "Fahr syndrome associated with post-thyroidectomy hypoparathyroidism." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.567.

Full text
Abstract:
Context: Fahr’s syndrome is a rare disorder characterized by bilateral and symmetrical abnormal calcifications in basal ganglia and cerebral cortex. Those calcified deposits are due to changes in calcium and phosphorus metabolisms that can be caused by endocrine disorders, mitochondrial myopathies, dermatological and infectious diseases. Clinical manifestations may include a variety of extrapyramidal, cerebelar and neuropsychiatric syndromes. Case report: This study describes a 75-year-old female patient that underwent total thyroidectomy in 1985 due to a multinodular goiter and presented post
APA, Harvard, Vancouver, ISO, and other styles
9

Patil, Vaishali, and Neeraj Masand. "Applications of Structure Based Drug Design Approaches towards Design and Development of Calcium Channel Blockers." In MOL2NET 2017, International Conference on Multidisciplinary Sciences, 3rd edition. MDPI, 2017. http://dx.doi.org/10.3390/mol2net-03-05051.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Wang, C. T., J. Y. Lee, J. C. Chen, Y. J. Shiao, and W. J. Tsai. "EFFECT OF TRIFLUOPERAZINE (TFP) ON HUMAN PLATELET MEMBRANE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644816.

Full text
Abstract:
TFP is a lipophilic antipsychotic drug. The drug will first encounter with cell membrane when adding it into a cell suspension. The effect of TFP on plasma membrane of the gel-filtered human platelet was investigated by : 1) scanning electron microscopy (SEM); 2) measuring theleakiness of marker enzymes and compound; 3) estimating its solubility in membrane. The cells were suspended in the modified Tyrode's buffer containing 0.1% dextrose, 0.2% of bovine serum albumin and without calcium. The SEM study showed that platelet changed shape from disc to ellipsoid in 10 μM TFP.,Increasingthe TFP co
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Calcium channel drugs' for particular source types:
Journal articles Dissertations / Theses Books
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography