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Books on the topic 'Calcium channel drugs'

Author: Grafiati

Published: 10 December 2022

Last updated: 31 July 2025

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1

Opie, Lionel H., and William A. Coetzee. Clinical Use of Calcium Channel Antagonist Drugs. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4613-0863-8.

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2

Opie, Lionel H. Clinical use of calcium channel antagonist drugs. Kluwer Academic Publishers, 1989.

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3

Opie, Lionel H. Clinical use of calcium channel antagonist drugs. 2nd ed. Kluwer, 1990.

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4

Japan-U.S.A. Symposium on Cardiovascular Drugs (1989 Kahuku, Hawaii). Recent advances in calcium channels and calcium antagonists: Proceedings of the Japan-U.S.A. Symposium on Cardiovascular Drugs. Edited by Yamada Kazuo and Shibata Shoji. Pergamon Press, 1990.

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5

1937-, Epstein Murray, and Loutzenhiser Rodger, eds. Calcium antagonists and the kidney. Hanley & Belfus, 1990.

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6

W, Tsien R., Clozel Jean-Paul, and Nargeot Joël, eds. Low-voltage-activated T-type calcium channels: Proceedings from the International Electrophysiology Meeting, Montpellier, 21-22 October 1996. Adis International, 1998.

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7

Albrecht, Fleckenstein, ed. Cardiovascular effects of dihydropyridine-type calcium antagonists and agonists. Springer-Verlag, 1985.

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8

Godfraind, T. Calcium channel blockers. Birkhauser Verlag, 2003.

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9

S, Meldrum Brian, Williams, Michael, 1947 Jan. 3-, and Princeton Drug Research Symposia (1st : 1989 : Princeton, N.J.), eds. Current and future trends in anticonvulsant, anxiety, and stroke therapy: Proceedings of a symposium held at Princeton, New Jersey, May 21-23, 1989. Wiley-Liss, 1990.

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10

Roberto, Robles Nicolás, ed. Calcium channel blockers and renal disease. Nova Science Publishers, 2009.

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11

Howl, John. Calcium-mobilizing drugs and pathological changes in Mammalian muscle. University of Birmingham, 1988.

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12

Sklar, Grant. A retrospective drug utilization review of the calcium channel blockers. Ottawa Civic Hospital, 1989.

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13

Campbell, Anthony K. Intracellular calcium. John Wiley & Sons, Ltd, 2014.

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14

1937-, Epstein Murray, ed. Calcium antagonists in clinical medicine. 3rd ed. Hanley & Belfus, 2002.

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15

K-H, Bichler, and Strohmaier W. L. 1957-, eds. Nephrocalcinosis, calcium antagonists, and kidney. Springer-Verlag, 1988.

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16

P, Rubin Ronald, Weiss George B, Putney James W, Federation of American Societies for Experimental Biology. Meeting, and American Society for Pharmacology and Experimental Therapeutics., eds. Calcium in biological systems. Plenum, 1985.

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17

Gary, Fiskum, ed. Cell calcium metabolism: Physiology, biochemistry, pharmcology and clinical implications. Plenum Press, 1989.

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18

Gary, Fiskum, and International Spring Symposium on Health Sciences (7th : 1987 : George Washington University), eds. Cell calcium metabolism: Physiology, biochemistry, pharmacology, and clinical implications. Plenum Press, 1989.

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19

T, Godfraind, and International Symposium on Pharmacological Control of Calcium and Potassium Homeostasis: Biological, Therapeutical, and Clinical Aspects (6th : 1994 : Florence, Italy), eds. Pharmacological control of calcium and potassium homeostasis: Biological, therapeutical, and clinical aspects. Kluwer Academic Publishers, 1995.

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20

Albrecht, Fleckenstein, and Laragh John H. 1924-, eds. Hypertension--the next decade: Verapamil in focus : proceedings of an international symposium, Berlin ICC, 10-11 October 1985. Churchill Livingstone, 1987.

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21

Albrecht, Fleckenstein, Laragh John H. 1924-, and Knoll AG (Ludwigshafen am Rhein, Germany), eds. Hypertension--the next decade: Verapamil in focus : proceedings of an international symposium, Berlin ICC, 10-11 October 1985. Churchill Livingstone, 1987.

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22

L, Tranquilli A., ed. Calcium antagonists in the treatment of hypertension in pregnancy. Parthenon Pub. Group, 1999.

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23

Jonathan, Abrams, Pepine Carl J, and Thadani Udho, eds. Medical therapy of ischemic heart disease: Nitrates, beta blockers, and calcium antagonists. Little, Brown and Co., 1992.

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24

R, Watson Ronald, ed. Alcohol and neurobiology: Receptors, membranes, and channels. CRC Press, 1992.

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25

Felix, Bronner, and Peterlik Meinrad, eds. Extra- and intracellular calcium and phosphate regulation: From basic research to clinical medicine. CRC Press, 1992.

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26

D, Nathan Richard, ed. Cardiac muscle: The regulation of excitation and contraction. Academic Press, 1986.

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27

McDonough, Stefan I. Calcium Channel Pharmacology. Springer London, Limited, 2011.

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28

McDonough, Stefan I. Calcium Channel Pharmacology. Springer, 2012.

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29

H, Opie Lionel. Clinical Use of Calcium Channel Antagonist Drugs. Springer, 2011.

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30

Clinical Use of Calcium Channel Antagonist Drugs. Springer, 2013.

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31

H, Opie Lionel. Clinical Use of Calcium Channel Antagonist Drugs. Springer London, Limited, 2012.

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32

Isbister, Geoffrey, та Colin Page. Management of β‎-blocker and calcium channel blocker poisoning. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0325.

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β‎-blocker and calcium channel-blockers can cause life-threatening toxicity due to cardiogenic shock. Both β‎-blockers and calcium channel-blockers are heterogenous groups of drugs and particular drugs, such as propranolol, diltiazem, and verapamil are far more toxic than the others in their class. The most important investigations in β‎-blocker and calcium channel-blocker overdose are an electrocardiogram, blood glucose measurement, and electrolytes. Like most overdoses, supportive treatment is the most important, with emphasis on the primary pathophysiology. Early decontamination should be c

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33

Kurachi, Y., M. Mishina, and M. Endo. Pharmacology of Ionic Channel Function: Activators and Inhibitors. Springer London, Limited, 2012.

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34

Kurachi, Y., Masayoshi Mishina, and M. Endo. Pharmacology of Ionic Channel Function: Activators and Inhibitors. Springer Berlin / Heidelberg, 2012.

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35

The Emergence of Drugs Which Block Calcium Entry (Progress in Clinical Biochemistry and Medicine). Springer, 1988.

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36

(Editor), M. Endo, Y. Kurachi (Editor), and M. Mishina (Editor), eds. Pharmacology of Ionic Channel Function: Activators and Inhibitors (Handbook of Experimental Pharmacology). Springer, 2000.

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37

Sutter, Johan De, Miguel Mendes, and Oscar H. Franco. Cardioprotective drugs. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656653.003.0019.

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Cardioprotective drugs are important in the treatment of patients at risk for or with documented cardiovascular disease. Beta-blockers are indicated after acute coronary syndromes, stable coronary artery disease, heart failure, and arrhythmias. Angiotensin-converting enzyme inhibitors (ACEi) are important in congestive heart failure, stable angina, post-acute myocardial infarction, and secondary prevention after any event or revascularization. Angiotensin receptor blockers are mainly alternative drugs for the same indications in case of intolerance to ACEi. Calcium channel blockers are first l

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38

Sutter, Johan De, Miguel Mendes, and Oscar H. Franco. Cardioprotective drugs. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199656653.003.0019_update_001.

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Cardioprotective drugs are important in the treatment of patients at risk for or with documented cardiovascular disease. Beta-blockers are indicated after acute coronary syndromes, stable coronary artery disease, heart failure, and arrhythmias. Angiotensin-converting enzyme inhibitors (ACEi) are important in congestive heart failure, stable angina, post-acute myocardial infarction, and secondary prevention after any event or revascularization. Angiotensin receptor blockers are mainly alternative drugs for the same indications in case of intolerance to ACEi. Calcium channel blockers are first l

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39

Drugs looking for diseases: Innovative drug research and the development of the beta blockers and the calcium antagonists. Kluwer Academic Publishers, 1991.

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40

Hoffmeister, F., A. Fleckenstein, C. Van Breemen, and R. Groß. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists. Springer London, Limited, 2013.

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41

Hoffmeister, F., A. Fleckenstein, C. Van Breemen, and R. Groß. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists. Springer, 2011.

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42

Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists. Springer, 2011.

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43

Fleckenstein, A., C. Van Breemen, and R. Grob. Cardiovascular Effects of Dihydropyridine-Type Calcium Antagonists and Agonists (Bayer-Symposium//(Proceedings)). Springer, 1985.

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44

Campbell, Anthony K. Intracellular Calcium. Wiley & Sons, Incorporated, John, 2014.

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45

Campbell, Anthony K. Intracellular Calcium. Wiley & Sons, Incorporated, John, 2014.

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46

Campbell, Anthony K. Intracellular Calcium. Wiley & Sons, Incorporated, John, 2014.

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47

Kazuo, Yamada. Recent Advances in Calcium Channels and Calcium Antagonists: Proceedings of the Japan-U.S.A. Symposium on Cardiovascular Drugs. Pergamon, 1989.

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48

Kazuo, Yamada. Recent Advances in Calcium Channels and Calcium Antagonists: Proceedings of the Japan-U.S.A. Symposium on Cardiovascular Drugs. Pergamon, 1989.

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49

Suri, Ajay, and Jean R. McEwan. Anti-anginal agents in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0037.

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Angina is chest pain resulting from the lack of blood supply to heart muscle most commonly due to obstructive atherosclerotic. Intensive care unit patients are subject to various stresses that will increase the demand on the heart and are in a pro-thrombotic state. Patients in an intensive treatment unit may be sedated and so cardiac ischaemia may be detected by electrocardiogram, haemodynamic monitoring, and echocardiographic imaging of function. These signs may indicate critical coronary perfusion heralding a myocardial infarction and are alleviated by anti-anginal drugs. Beta-blockers and c

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50

Cavanna, Andrea E. Antiepileptic drugs and behaviour: mechanisms of action. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198791577.003.0002.

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Antiepileptic drugs (AEDs) exert their pharmacological properties on both epileptic seizures and behaviour through different mechanisms of action. These include modulation of ion (mainly sodium and calcium) conductance through voltage-gated channels located within the neuronal membrane, as well as facilitation of inhibitory (GABAergic) neurotransmission and inhibition of excitatory (glutamatergic) neurotransmission, resulting in regulation of neuronal excitability.

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