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Dissertations / Theses on the topic 'Calcium channels'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Roberts, Dewi. "Calcium-dependent inactivation of Cav1.3 calcium channels." Thesis, University of Bristol, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.446186.

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2

Peterson, Blaise. "Molecular determinants of dihydropyridine binding on L-type calcium channels /." Thesis, Connect to this title online; UW restricted, 1996. http://hdl.handle.net/1773/6269.

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3

Farrington, Jasmine. "Calcium release activated calcium channels in human lung mast cells." Thesis, University of Sheffield, 2014. http://etheses.whiterose.ac.uk/6609/.

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4

Nakayama, Shinsuke. "Calcium channels in detrusor smooth muscle." Thesis, University of Oxford, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.334328.

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5

Xie, Mian. "Calcium Channel Beta Subunits and SCA6-Type Calcium Channel Alpha Subunits C-Termini Regulate Targeting and Function of Presynaptic Calcium Channels in Hippocampal Neurons." Case Western Reserve University School of Graduate Studies / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=case1188326628.

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6

Yasuda, Takahiro. "Modulation of calcium channel function and toxin sensitivity by auxiliary subunits /." [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe18052.pdf.

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7

Doughty, Stephen William. "Molecular modelling of voltage-gated calcium channels." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.362014.

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8

Warburton, Steven Peter Marc. "Calcium ion channels in insect skeletal muscle." Thesis, University of Nottingham, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.363592.

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9

Richardson, C. Mark. "Presynaptic calcium channels in skate electric organ." Thesis, University of Nottingham, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.319614.

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10

Pearson, Hugh Anthony. "Physiology and pharmacology of insect calcium channels." Thesis, University College London (University of London), 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.308295.

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11

Brosnan, James N. "Biochemical characterization of red beet calcium channels." Thesis, University of York, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.304314.

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12

Burgess, Alison J. "Immunological characterization of voltage-sensitive calcium channels." Thesis, University of Leicester, 1988. http://hdl.handle.net/2381/34291.

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A panel of monoclonal antibodies were raised against the 1,4-dihydropyridine sensitive Ca2+ channel of rabbit skeletal muscle. When tested on immunoblot assay of denatured and reduced transverse tubule membranes, four of the antibodies specifically recognized a polypeptide of Mr 140,000. This component co-migrated with the large glycoprotein ?2 subunit of purified Ca2+ channel preparations. On immunoblots of nonreducing gels the antibodies detected a component that migrated more slowly in the gel, with a Mr of 170,000, consistent with the disulphide-linkage of the ?2 subunit to a small compone

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13

Georgiou, Panayiotis Paulou. "Calcium-activated potassium-channels in mammalian eggs." Thesis, University of Edinburgh, 1985. http://hdl.handle.net/1842/29774.

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14

Ruchala, Iwona. "EXPANDING MONOAMINE TRANSPORTERS PHARMACOLOGY USING CALCIUM CHANNELS." VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/5032.

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Research in drug development meets many challenges including lengthy, complex and costly procedures to identify novel pharmacotherapies. In our lab, we developed a method for fast screening of small molecules that interact with monoamine transports – dopamine and serotonin (DAT, SERT). These membrane proteins play important roles in brain neurotransmission responsible for cognition, motion and pleasure. Dysfunction in dopaminergic and serotonergic systems result in neurological disorders such as depression, Attention Deficit Hyperactivity Disorder (ADHD), schizophrenia and addiction. DAT and S

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15

Wang, Shiyi. "Regulation of voltage-gated calcium channels Cav1.2." Diss., University of Iowa, 2017. https://ir.uiowa.edu/etd/6009.

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Voltage-gated Ca2+ (Cav) channels are activated upon depolarization. They specifically allow Ca2+ ions to come into the cell. These Ca2+ ions are bi-functional because they not only control cell excitability but also couple electrical activity to complex downstream signaling events, such as excitation-contraction coupling in muscles and neurotransmitter release in neurons. In the brain, Cav channels are expressed in the pre- or post-synaptic membrane of most excitable cells, neurons. In the past few years, their expression and function have also been characterized in many nonexcitable cells su

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16

Pifferi, Simone. "Calcium Activated Chloride Channels In Olfactory Transduction." Doctoral thesis, SISSA, 2008. http://hdl.handle.net/20.500.11767/4668.

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Ca2+-activated Cl ̄ channels are an important component of olfactory transduction. Odorant binding to odorant receptors in the cilia of olfactory sensory neurons (OSNs) leads to an increase of intraciliary Ca2+ concentration by Ca2+ entry through cyclic nucleotide-gated channels. Ca2+ activates a Cl ̄ channel that leads to an efflux of Cl ̄ from the cilia, contributing to the amplification of the OSN depolarization. The molecular identity of this Cl ̄ channel remains elusive. Recent evidences have indicated that bestrophins are able to form Ca2+-activated Cl ̄ channels channels in heterologous

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17

Kannancheri, Puthooru Dheeraj. "Calcium Channels in Prostate Cancer Drug Resistance." Electronic Thesis or Diss., Université de Lille (2022-....), 2025. https://pepite-depot.univ-lille.fr/ToutIDP/EDBSL/2025/2025ULILS100.pdf.

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Malgré des avancées scientifiques significatives, la guérison du cancer reste une question complexe. Cela est dû non seulement à l'hétérogénéité des tumeurs, mais aussi à l'émergence d'une résistance à la plupart, sinon tous les traitements anticancéreux. Le cancer de la prostate (PCa), qui représente le deuxième cancer le plus fréquent chez les hommes, n'est pas différent: les thérapies conventionnelles, y compris l'hormonothérapie et la chimiothérapie, échouent face à des tumeurs réfractaires. Cette situation rend nécessaire l'élaboration de nouvelles stratégies pour lutter contre ce problèm

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18

Muller, Yunhua Li 1963. "Developmentally regulated expression of the calcium-dependent potassium channel and calcium channels during maturation of the rat cerebellum." Diss., The University of Arizona, 1996. http://hdl.handle.net/10150/282231.

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Potassium channels govern the duration and frequency of excitable membrane events, and thus may regulate voltage-dependent signals that are important in neuronal development. This study assesses the developmental expression of two classes of K⁺ channels in vivo and in vitro in the rat cerebellum. In vivo, the level of mslo-related transcript for the Ca²⁺-dependent K⁺ channel (KCa) was shown by Northern analysis to be upregulated during development, whereas transcripts for delayed rectifier (KD) channels remained fairly constant. The same pattern of in vivo development was demonstrated with fun

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19

Hagen, Brian M. "Regulation of calcium-activated potassium channels by localized calcium transients in murine colon." abstract and full text PDF (free order & download UNR users only), 2005. http://0-gateway.proquest.com.innopac.library.unr.edu/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3209955.

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20

Victory, Jason G. G. "Ion channels and the myocardium : interactions between general anaesthetics and calcium channel blockers." Thesis, University of Oxford, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.253420.

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21

Balasubramanian, Bharathi. "Regulation of cloned cardiac channels." Texas A&M University, 2005. http://hdl.handle.net/1969.1/2525.

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Activation of a5??1 integrin potentiates L-type calcium current in vascular smooth muscle, which is partly mediated by tyrosine phoshorylation of the a1c channel subunit. Expressed rabbit VSM and neuronal isoforms are also potentiated by a5??1 integrin activation and require dual phosphorylation of a1c by PKA and c-Src. To explore common mechanisms of regulation by a5??1 integrin, whole cell patch clamp experiments were used to investigate the effects of a5??1 integrin antibody on expressed cardiac calcium channels. In HEK cells transfected with a1c, ??2a and a2-d1 subunits alone, currents inc

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22

Ketchum, Karen Ann. "A calcium-dependent potassium channel in corn (Zea mays) suspension cells /." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=74658.

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Three distinct K$ sp+$ currents were identified in corn (Zea mays) protoplasts using the whole-cell patch-clamp technique. Inward-rectifying K$ sp+$ currents were evoked at membrane potentials more negative than $-$100 mV. The activation range was sensitive to external K$ sp+$ and shifted in the positive direction as the K$ sp+$ concentration was elevated. The second K$ sp+$ current was voltage-independent and contributed to the resting membrane conductance of the protoplast. Finally, a voltage- and Ca$ sp{2+}$-dependent K$ sp+$ current was observed at potentials positive to $-$60 mV. This cur

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23

Yassine, Maya. "Calcium, Calcium-permeable channels and autophagy modulators in control of autophagy and cancer." Thesis, Lille 1, 2013. http://www.theses.fr/2013LIL10159/document.

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L'autophagie est une voie cellulaire strictement régulée dont le but principal est la dégradation lysosomale et le recyclage ultérieur du matériel cytoplasmique afin de maintenir l'homéostasie cellulaire normale. Des défauts dans l'autophagie sont liés à une variété d'états pathologiques, dont le cancer. Le cancer est une maladie associée aux modifications des processus cellulaires fondamentaux tels que l'apoptose et l'autophagie. Le calcium régule une série de processus physiologiques et pathologiques tels que le vieillissement, la neurodégénérescence et le cancer. Si le rôle du calcium et de

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24

Gelli, Angie Cristina. "The electrophysiological characterization of plant calcium channels and their role in calcium signaling." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape16/PQDD_0007/NQ27928.pdf.

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25

Calcraft, Peter James. "Two-pore channels and NAADP-dependent calcium signalling." Thesis, St Andrews, 2010. http://hdl.handle.net/10023/888.

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26

Conrad, Rachel [Verfasser]. "Trafficking of voltage-activated calcium channels / Rachel Conrad." Düsseldorf : Universitäts- und Landesbibliothek der Heinrich-Heine-Universität Düsseldorf, 2017. http://d-nb.info/1136717919/34.

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27

Millership, Joanne Ella. "Regulation and function of calcium-activated potassium channels." Thesis, University of Manchester, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.503016.

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28

Muir, Shelagh R. "Ligand-gated calcium channels in higher plant membranes." Thesis, University of York, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.319717.

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29

Wajdner, Hannah. "Characterisation of calcium channels in human immune cells." Thesis, University of Sheffield, 2015. http://etheses.whiterose.ac.uk/12414/.

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30

Stevens, Louisa Rebecca. "Structure and function of potassium and calcium channels." Thesis, University of Leeds, 2005. http://etheses.whiterose.ac.uk/382/.

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In this thesis, potassium channels human Kv2.1 and rat Kv2.1, along with calcium channels Ca, 1.2, Ca, 3.1, and chimeras have been studied. These channels were expressed in Xenopuso ocytesf or electrophysiological experiments using two electrode voltage clamp. For protein expression studies,DNA was expressed in BL21 or COS-7 cells and purified using glutathione or an ID4 affinity column. Firstly, the roles of the N- and C- terminal domains in the activation kinetics of rat and human forms of Kv2.1 were investigated. A mutant in the N- terminal domain and chimeras between the rat and human form

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31

Pardo, Pastor Carlos 1989. "Piezo ion channels in cancer cell mechanotransduction." Doctoral thesis, Universitat Pompeu Fabra, 2018. http://hdl.handle.net/10803/664209.

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The mechanical dependence of transformation and metastasis is an emerging field, but the role of mechanosensitive channels has been largely omitted. This thesis focuses on the roles played by the mechanosensitive ion channels Piezo1 and Piezo2 in the transduction of mechanical stimuli (confinement, adhesion, substrate rigidity, adhesive ligand concentration) by cancer cells. In a first chapter, we show that confinement triggers Piezo1-mediated calcium entry. This activates phosphodiesterase 1, reducing cAMP levels and, consequently, PKARac1 activity, relieving Myosin II from its inhibition.

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32

Lao, Yuanzhi. "Calcium signaling in apoptotic mammalian cells /." View abstract or full-text, 2008. http://library.ust.hk/cgi/db/thesis.pl?BIOL%202008%20LAO.

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33

Zhang, Hongling. "Sigma Receptors Modulation of Voltage-gated Ion Channels in Rat Autonomic Neurons." [Tampa, Fla.] : University of South Florida, 2005. http://purl.fcla.edu/fcla/etd/SFE0001183.

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34

Thrower, Edwin C. "A study of the inositol (1,4,5) triphosphate-sensitive Ca'2'+ channel." Thesis, University of East Anglia, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.361749.

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35

Forristal, Ailish. "MRS studies on the role and function of divalent cations in the cerebral cortex." Thesis, University of Nottingham, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299709.

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36

Cifelli, Carlo. "Impairment of force development in K(ATP) channel deficient skeletal muscle involves calcium ion influx through L-type calcium ion channels." Thesis, University of Ottawa (Canada), 2006. http://hdl.handle.net/10393/27342.

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ATP-sensitive potassium (KATP) channels link membrane excitability to metabolism. They are regulated by intracellular nucleotides and other factors, and have been shown to play a role in development of skeletal muscle force, but controversy surrounds their role during fatigue. The aim of this research project was to determine the role of KATP channel under conditions that allow for better assessment of changes in force during fatigue, by virtue of using a smaller whole muscle model less subject to anoxia. Thus, the first objective was to determine the effect of the loss of KATP channel activit

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37

Kouzai, Daisuke. "Chemical biological studies on oxidation status-sensitive calcium channels." 京都大学 (Kyoto University), 2014. http://hdl.handle.net/2433/188546.

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38

Skeer, Jacqueline Mary. "Calcium channels of insect nervous system and skeletal muscle." Thesis, University of Cambridge, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.239592.

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39

Harding, Louise M. "The effects of locusta peptides on mammalian calcium channels." Thesis, Oxford Brookes University, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.389106.

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40

Hendrich, Janek K. "Alpha-2-delta subunits of voltage-gated calcium channels." Thesis, University College London (University of London), 2008. http://discovery.ucl.ac.uk/1444197/.

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The calcium channel alpha-2-delta (0,28) subunit is an auxiliary subunit associated with voltage-dependent calcium channels. It is implicated in the trafficking and functional expression of the calcium channel complex. This study expands the functional role of the VWA domain and the RRR motif of the 0:26 subunit, and the interaction between this subunit and the anti-epileptic drug, gabapentin The VWA domain is normally found in integrins, where it mediates binding to extracellular proteins. A mutation in the 0:28-2 subunit VWA domain (uMIDAS) did not produce the increase in current amplitude e

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41

Wang, Ming Chuan. "Structural studies of L-type voltage-gated calcium channels." Thesis, University of Manchester, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.525174.

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42

Dedman, Alexandra Margaret. "Regulation of expression and function of TRPC calcium channels." Thesis, University of Leeds, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.436392.

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43

Brice, Nicola. "Voltage dependent calcium channels : subcellular subunit localisation and targeting." Thesis, University College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299977.

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44

Gillard, Samantha Ellen. "Voltage-dependent calcium channels of cerebellar neurones in culture." Thesis, Royal Veterinary College (University of London), 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268052.

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45

Zeng, Bo. "Pharmacological regulation and function of store-operated calcium channels." Thesis, University of Hull, 2012. http://hydra.hull.ac.uk/resources/hull:6432.

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Store-operated Ca²⁺ entry (SOCE) is an important Ca²⁺ influx pathway existing in almost all types of mammalian cell. STIM1, ORAI and TRPC have been regarded as the molecular basis of SOCE. Once the endoplasmic reticulum (ER) Ca²⁺ store is depleted, STIM1 proteins move to the plasma membrane and activate ORAI and TRPC channels to allow Ca²⁺ influx. In this thesis, the pharmacological aspects and regulatory mechanisms of SOCE were investigated using HEK293 cells overexpressing STIM1, ORAI or TRPC genes. The expression and function of TRPC channels and their spliced variants in native cells were

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46

Mulgrew, Christopher James. "T-type calcium channels and human mesangial cell proliferation." Thesis, King's College London (University of London), 2012. https://kclpure.kcl.ac.uk/portal/en/theses/ttype-calcium-channels-and-human-mesangial-cell-proliferation(8d963ed0-7b21-4f73-bc94-ec5a76205bdd).html.

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Aberrant proliferation of human mesangial cells (MC) is a critical step in the pathogenesis of mesangioproliferative renal diseases. The T-type calcium channel (T-CaCN) has been proposed to play an important role in the proliferation of a number of non-excitable cell types, but T-CaCN expression and functional significance in MC is not known. The aim of this thesis was to investigate the hypothesis that T-CaCN may play an important role in the proliferation of MC in primary culture. Expression of mRNA encoding the α1H isoform (Cav3.2 clone) (but not the α1G nor α1I T-CaCN isoforms) was demonst

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47

DeRemigio, Hilary. "Markov chain models of instantaneously coupled intracellular calcium channels." W&M ScholarWorks, 2008. https://scholarworks.wm.edu/etd/1539623334.

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Localized calcium elevations known as calcium puffs or sparks are cellular signals arising from cooperative activity of clusters of inositol 1,4,5-trisphosphate receptors (IP3Rs) or ryanodine receptors (RyRs) located at calcium release sites on the endoplasmic or sarcoplasmic reticulum membrane. When Markov chain models of these intracellular calcium-regulated calcium channels are coupled via a mathematical representation of the calcium microdomain, simulated calcium release sites may exhibit the phenomenon of "stochastic calcium excitability" where the IP3Rs or RyRs open and close in a concer

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48

Amjad, Asma. "Calcium-activated chloride channels in mouse vomeronasal sensory neurons." Doctoral thesis, SISSA, 2013. http://hdl.handle.net/20.500.11767/3899.

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In vomeronasal sensory neurons, signal transduction occurs in microvilli that are present at the neuron’s apical surface. The binding of pheromone to vomeronasal receptors causes an increase of the intracellular calcium concentration by calcium entry through TRPC2. An important issue is the impact of Ca2+ entry in pheromonal transduction. In the first part of this thesis we have investigated the functional role played by the increase in intracellular Ca2+ concentration in the apical region of vomeronasal sensory neurons. By taking advantage of flash photolysis of caged calcium restricted to t

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49

Thomas, Balbir. "A model of mitochonrial [sic] calcium induced calcium release." The Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=osu1190053132.

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50

Sharma, Aarushi. "HUMAN CLCA2 MODULATES THE CONDUCTANCE OF CALCIUM-ACTIVATED CHLORIDE CHANNELS BY REGULATION OF INTRACELLULAR CALCIUM." OpenSIUC, 2016. https://opensiuc.lib.siu.edu/dissertations/1252.

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Chloride channels play an essential role in the physiology of the respiratory system, the gastrointestinal tract, and secretory glands. Their dysregulation underlies debilitating pathologies such as cystic fibrosis, asthma, and certain cancers. The CLCA (Chloride Channel Accessory) gene family is thought to determine severity of these diseases by modulating an unidentified Calcium-activated Chloride Channel (CaCC). Recent evidence indicates Ano1 to be the mediator of strong quintessential calcium-activated chloride current in several cell types. Ano1 is highly expressed in airway epithelium an

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