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Books on the topic 'Calcium intake'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

National Institutes of Health (U.S.), ed. Optimal calcium intake. National Institutes of Health, Office of the Director, 1994.

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2

NIH Consensus Development Conference on Optimal Calcium Intake (1994 National Institutes of Health). NIH Consensus Development Conference on Optimal Calcium Intake: [program and abstracts]. National Institutes of Health, 1994.

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3

Ulincy, Loretta D. Optimal calcium intake: January 1990 through April 1994 plus selected earlier citations : 775 citations. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Library of Medicine, Reference Section, 1994.

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Ulincy, Loretta D. Optimal calcium intake: January 1990 through April 1994 plus selected earlier citations : 775 citations. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, National Library of Medicine, Reference Section, 1994.

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5

NIH Consensus Development Conference on Optimal Calcium Intake (1994 National Institutes of Health). NIH Consensus Development Conference on Optimal Calcium Intake: NIH Consensus Development Conference, June 6-8, 1994, Masur Auditorium, Warren Grant Magnuson Clinical Center, National Institutes of Health, Bethesda, Maryland. The Institutes, 1994.

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6

NIH Consensus Development Conference on Optimal Calcium Intake (1994 National Institutes of Health). NIH Consensus Development Conference on Optimal Calcium Intake: NIH Consensus Development Conference, June 6-8, 1994, Masur Auditorium, Warren Grant Magnuson Clinical Center, National Institutes of Health, Bethesda, Maryland. The Institutes, 1994.

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7

NIH Consensus Development Conference on Optimal Calcium Intake (1994 National Institutes of Health). NIH Consensus Development Conference on Optimal Calcium Intake: NIH Consensus Development Conference, June 6-8, 1994, Masur Auditorium, Warren Grant Magnuson Clinical Center, National Institutes of Health, Bethesda, Maryland. The Institutes, 1994.

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8

Institute of Medicine (U. S.). Committee to Review Dietary Reference Intakes for Vitamin D and Calcium, ed. Dietary reference intakes for calcium and vitamin D. National Academies Press, 2011.

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9

Institute of Medicine (U.S.). Standing Committee on the Scientific Evaluation of Dietary Reference Intakes., ed. Dietary reference intakes: For calcium, phosphorus, magnesium, vitamin D, and fluoride. National Academy Press, 1997.

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10

Faraut, Jacques. Calcul inte gral. EDP Sciences, 2006.

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11

Hairer, E. L'analyse au fil de l'histoire. Springer, 2001.

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12

Ikeda, Nobuyuki. Stochastic differential equations and diffusion processes. 2nd ed. North-Holland Pub. Co., 1989.

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13

Calcium Channel Blockers: The Comprehensive Guide to Managing Calcium Intake and Ensuring Sufficient Calcium Intake for Optimal Health and Performance. Independently Published, 2022.

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14

Alder, Robert John. Calcium intake and the risk of colorectal cancer. 1989.

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15

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Calcium in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0018.

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Most calcium in the body is present in the skeleton, where it serves a structural role and also as a reservoir for use in other tissues. During pregnancy, calcium is accumulated in the fetal skeleton, mostly during the third trimester when bone growth is at its peak. Although this increases the demand on maternal bone stores, the calcium transfer to the fetus is balanced by increased intestinal calcium absorption in the mother, mediated by compensatory changes in vitamin D synthesis and endogenous hormone levels. Bone loss is minimized if calcium intake is maintained at 1,000#amp;#x2013;1,200

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16

Turney, Ben, and John Reynard. Prevention of idiopathic calcium stones. Edited by John Reynard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0015.

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The main principles of idiopathic calcium oxalate stone prevention are to maintain dilute urine through increasing fluid intake and to reduce calcium and oxalate excretion. The influence of various urinary factors on the risk of stone formation has been quantified mathematically. Urine volume and urinary oxalate concentration are most influential on the risk of stone formation, while magnesium concentration contributes a small amount to risk. It is estimated that around 50% of stone formers will form another stone within five years. Some stone formers have frequent recurrences. Most stone form

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17

Berg, Lawrence Raymond. Effect of Varying Levels of Calcium Intake on the Calcium Balance, Shell Thickness, and Blood Calcium Level of White Leghorn Pullets. Creative Media Partners, LLC, 2021.

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18

Bilezikian, John P. Optimal Calcium Intake: National Institutes of Health Development Conference: Proceedings, 1994. Diane Pub Co, 1994.

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19

Parent, Marie-Elise *. Effect of caffeine and calcium intake on the bone mass of postmenopausal women. 1988.

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20

Physical activity, calcium intake, body composition and stature as predictors of bone indices in college-aged men. 1988.

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21

Physical activity, calcium intake, body composition and stature as predictors of bone indices in college-aged men. 1990.

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22

Physical activity, calcium intake, body composition and stature as predictors of bone indices in college-aged men. 1990.

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23

Physical activity, calcium intake, body composition and stature as predictors of bone indices in college-aged men. 1990.

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24

Proudfoot, Morwenna. Forearm bone mineral density and its relation to calcium intake and grip strength: Measurements of young normal women. 1996.

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25

Chazminare, Allex Sun. Home Remedies for Kidney Stones: Drink Lots of Water, Lemons, Increase Magnesium Intake, Calcium Intake, Apple Cider Vinegar, Pomegranate Juice, Reduce Vitamin C Intake, Cut Back on Oxalate-Rich Foods, Reduce Animal Protein, Reduce Sodium. Independently Published, 2021.

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26

Shepherd, Angela J., and Juliet M. Mckee. Osteoporosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190466268.003.0015.

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Osteoporotic fractures are major causes of suffering and death. Dual-energy x-ray absorptiometry (DEXA) is the standard of care for diagnosis (T-score ≤ –2.5) of osteoporosis. Prevention of fractures requires addressing bone and muscle strength and balance. Physical exercise, good nutrition (fruits, vegetables, adequate calcium), adequate vitamin intake (C, D, and K), tobacco cessation, and no more than moderate alcohol intake enhance bone health and decrease fracture risk. Long-term treatment with glucocorticoids, certain drugs used in breast or prostate cancer treatment, and proton pump inhi

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27

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Vitamin D in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0015.

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Vitamin D, which is synthesized in skin exposed to UV light, or is consumed in the diet, plays a key role in maintaining bone integrity via the regulation of calcium and phosphorus homeostasis. It also influences a number of extra-skeletal processes, including immune function and blood glucose homeostasis. Maternal vitamin D deficiency in pregnancy leads to poor fetal skeletal mineralization in utero that can manifest as rickets in newborns. In addition to skeletal effects, women with very low vitamin D status face increased risks of other adverse pregnancy outcomes and possible long-term effe

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28

The Effects of Calcium Intake and Physical Activity on the Bone Mineral Content and Bone Mineral Density of the United States Naval Academy Midshipmen. Storming Media, 1997.

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29

Bardin, Thomas, and Tilman Drüeke. Renal osteodystrophy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0149.

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Renal osteodystrophy (ROD) is a term that encompasses the various consequences of chronic kidney disease (CKD) for the bone. It has been divided into several entities based on bone histomorphometry observations. ROD is accompanied by several abnormalities of mineral metabolism: abnormal levels of serum calcium, phosphorus, parathyroid hormone (PTH), vitamin D metabolites, alkaline phosphatases, fibroblast growth factor-23 (FGF-23) and klotho, which all have been identified as cardiovascular risk factors in patients with CKD. ROD can presently be schematically divided into three main types by h

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30

Turney, Ben, and John Reynard. Kidney stones. Edited by John Reynard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0013.

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The composition of kidney stones is variable and the predisposing factors multifactorial. Consequently, a detailed evaluation of the patient’s lifestyle, diet, fluid intake, medical history, drug history, urinary tract anatomy, blood, and urine biochemistry and stone composition is required determine predisposing factors for stone formation in an individual patient. Combinatorial subtle variants in biochemistry may act synergistically to increase risk of stone formation/recurrence. Many medications may alter blood and/or urine biochemistry and predispose to stone formation. Corticosteroids inc

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31

Jadon, Deepak R., Tehseen Ahmed, and Ashok K. Bhalla. Disorders of bone mineralization—osteomalacia. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0146.

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Disorders of bone mineralization cause rickets in children and osteomalacia in adults. Both remain common in developing countries. Incidence in Western countries had declined since the fortification of foodstuffs, but appears to be increasing again. Calcium and inorganic phosphate are the key precursors for bone mineralization and growth. The commonest aetiology of osteomalacia is vitamin D deficiency, primarily due to low dietary intake and inadequate sun exposure. In the last decade gene mutations have been identified that are responsible for inherited rickets and osteomalacia, particularly

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32

Jadon, Deepak R., Tehseen Ahmed, and Ashok K. Bhalla. Disorders of bone mineralization—osteomalacia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0146_update_001.

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Disorders of bone mineralization cause rickets in children and osteomalacia in adults. Both remain common in developing countries. Incidence in Western countries had declined since the fortification of foodstuffs, but appears to be increasing again. Calcium and inorganic phosphate are the key precursors for bone mineralization and growth. The commonest aetiology of osteomalacia is vitamin D deficiency, primarily due to low dietary intake and inadequate sun exposure. In the last decade gene mutations have been identified that are responsible for inherited rickets and osteomalacia, particularly

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33

Taylor, Eric N., and Gary C. Curhan. Epidemiology of nephrolithiasis. Edited by Mark E. De Broe. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0199.

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Nephrolithiasis is common, costly, and painful. The prevalence of nephrolithiasis, defined as a history of stone disease, varies by age, sex, race, and geography while the incidence of nephrolithiasis, defined as the first stone event, varies by age, sex, and race. Epidemiologic studies have quantified the burden of kidney stone disease and expand our understanding of risk factors. A variety of dietary, non-dietary, and urinary risk factors contribute to the risk of stone formation and the importance of these varies by age, sex, and body mass index.Low fluid intake, high urinary oxalate or cal

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34

Dietary Reference Intakes for Calcium and Vitamin D. National Academies Press, 2011. http://dx.doi.org/10.17226/13050.

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35

Committee to Review Dietary Reference Intakes for Vitamin D and Calcium, Heather B. Del Valle, Food and Nutrition Board, Institute of Medicine, and Ann L. Yaktine. Dietary Reference Intakes for Calcium and Vitamin D. National Academies Press, 2011.

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36

Committee to Review Dietary Reference Intakes for Vitamin D and Calcium, Heather B. Del, Ann L. Yaktine, Christine L. Taylor, and A. Catharine Ross. Dietary Reference Intakes for Calcium and Vitamin D. National Academies Press, 2011.

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37

Standing Committee on the Scientific Evaluation of Dietary Reference Intakes, Food and Nutrition Board, and Institute of Medicine. Dietary Reference Intakes for Calcium, Phosphorus, Magnesium, Vitamin D, and Fluoride (Dietary Reference Intakes). National Academies Press, 1999.

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38

Dietary Reference Intakes for Calcium, Phosphorus, Magnesium, Vitamin D, and Fluoride. National Academies Press, 1997. http://dx.doi.org/10.17226/5776.

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39

Standing Committee on the Scientific Evaluation of Dietary Reference Intakes, Food and Nutrition Board, and Institute of Medicine. Dietary Reference Intakes for Calcium, Phosphorus, Magnesium, Vitamin d, and Fluoride. National Academies Press, 1999.

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40

Intakes, Standing Committee on the Scientific Evaluation of Dietary Reference, Food and Nutrition Board, and Institute of Medicine. Dietary Reference Intakes for Calcium, Phosphorus, Magnesium, Vitamin D, and Fluoride (Dietary Reference Series). National Academies Press, 1999.

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41

The influence of glucose on calcium transport by isolated rat enterocytes and intact intestinal preparations. 1987.

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42

Benoist, Joël, and Alain Salinier. Exercices calcul intégral : Avec rappels de cours. Dunod, 2001.

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43

Voinescu, Alexandra, Nadia Wasi Iqbal, and Kevin J. Martin. Management of chronic kidney disease-mineral and bone disorder. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0118_update_001.

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In all patients with chronic kidney disease (CKD) stages 3–5, regular monitoring of serum markers of CKD-mineral and bone disorder, including calcium (Ca), phosphorus (P), parathyroid hormone (PTH), 25-hydroxyvitamin D, and alkaline phosphatase, is recommended. Target ranges for these markers are endorsed by guidelines. The principles of therapy for secondary hyperparathyroidism include control of hyperphosphataemia, correction of hypocalcaemia, use of vitamin D sterols, use of calcimimetics, and parathyroidectomy. of hyperphosphataemia is crucial and may be achieved by means of dietary P rest

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44

Clarke, Andrew. Water. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780199551668.003.0005.

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Liquid water is essential for life, and a metabolically active cell is ~70% water. The physical properties of liquid water, and their temperature dependence, are dictated to a significant extent by the properties of hydrogen bonds. From an ecological perspective, the important properties of liquid water include its high latent heats of fusion and vapourisation, its high specific heat, the ionisation, low dynamic viscosity and high surface tension. The solubility in water of oxygen, carbon dioxide and the calcium carbonate used to build skeletons in many invertebrates groups all increase with d

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45

Stochastic differential equations and diffusion processes. kodansha, 1997.

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46

Hamilton, Scott. An Analog Electronics Companion: Basic Circuit Design for Engineers and Scientists. Cambridge University Press, 2007.

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47

An Analog Electronics Companion. Cambridge University Press, 2003.

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48

Skiba, Grzegorz. Fizjologiczne, żywieniowe i genetyczne uwarunkowania właściwości kości rosnących świń. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_gs_2020.

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Bones are multifunctional passive organs of movement that supports soft tissue and directly attached muscles. They also protect internal organs and are a reserve of calcium, phosphorus and magnesium. Each bone is covered with periosteum, and the adjacent bone surfaces are covered by articular cartilage. Histologically, the bone is an organ composed of many different tissues. The main component is bone tissue (cortical and spongy) composed of a set of bone cells and intercellular substance (mineral and organic), it also contains fat, hematopoietic (bone marrow) and cartilaginous tissue. Bones a

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