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1

Ikeda, Nobuyuki. Stochastic differential equations and diffusion processes. 2nd ed. Amsterdam: North-Holland Pub. Co., 1989.

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2

Erori de calcul: Poeți români în grai aromân. București: Editura Fundației Culturale Aromâne "Dimândarea Părintească", 2000.

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3

Jouad, Hassan. Le calcul inconscient de l'improvisation: Poésie berbère, rythme, nombre et sens. Paris: Peeters, 1995.

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4

Jouad, Hassan. Le calcul inconscient de l'improvisation: Poésie berbère, rythme, nombre et sens. Paris: Peeters, 1995.

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5

Tegethoff, F. Wolfgang. Calcium Carbonate: From the Cretaceous Period into the 21st Century. Basel: Birkhäuser Basel, 2001.

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6

Wilkie, Martin Erskine. Clinical and laboratory based studies into the effects of calcium antagonists on cyclosporin A nephrotoxicity. Manchester: University of Manchester, 1994.

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7

Brown, Judith A. Turn calls into customers: Maximize customer experience with your call center. Marblehead, MA: HCPro, 2008.

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8

Brown, Judith A. Turn calls into customers: Maximize customer experience with your call center. Marblehead, MA: HCPro, 2008.

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9

Turn calls into customers: Maximize customer experience with your call center. Marblehead, MA: HCPro, 2008.

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10

Doug, Wilberscheid, ed. Insight into calculus using Texas Instruments graphics calculators. Upper Saddle River, NJ: Prentice Hall, 1997.

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11

Sgarbi, Romano. Tecnica dei calchi nella versione armena della [grammatikē technē (romanized form)] attribuita a Dionisio Trace. Milano: Istituto lombardo di scienze e lettere, 1990.

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12

Have you faith in Christ?: A bishop's insight into the historic questions asked of those seeking admission into full connection in the United Methodist Church. Nashville: Abingdon Press, 2015.

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13

A noble task: Entry into the clergy in the first five centuries. Brookline, Mass: Holy Cross Orthodox Press, 2007.

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14

Practices, LLC Best. Service-to-sales excellence: Developing service representatives into high-sales achievers. Chapel Hill, NC: Best Practices, LLC, 2004.

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15

Beirne-Lewis, Sean Michael. The introduction of calcium carbonate into the clay brick mixture with a view to lowering the firing temperature, whilst maintaining the mechanical properties of the fires clay product. [London]: Queen Mary and Westfield College, 1994.

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16

Zhukova, Galina, and Margarita Rushaylo. Mathematical analysis in examples and tasks. Part 1. ru: INFRA-M Academic Publishing LLC., 2020. http://dx.doi.org/10.12737/1072156.

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The purpose of the textbook is to help students to master basic concepts and research methods used in mathematical analysis. In part 1 of the proposed cycle of workshops on the following topics: theory of sets, theory of limits, theory of continuous functions; differential calculus of functions of one variable, its application to the study of the properties of functions and graph; integral calculus of functions of one variable: indefinite, definite, improper integrals; hyperbolic functions; applications of integral calculus to the analysis and solution of practical problems. For the development of each topic the necessary theoretical and background material, reviewed a large number of examples with detailed analysis and solutions, the options for independent work. For self-training and quality control of the obtained knowledge provides exercises and problems with answers and guidance. For teachers, students and postgraduate students studying advanced mathematics.
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17

The Christian as minister: An exploration into the meaning of God's call to ministry and the ways The United Methodist Church offers to live out that call. 7th ed. Nashville, TN: General Board of Higher Education and Ministry, The United Methodist Church, 2009.

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18

Stochastic differential equations and diffusion processes. kodansha, 1997.

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19

Unknown. Calculo De Estructuras. UNKNOWN, 1999.

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20

Herrington, William G., Aron Chakera, and Christopher A. O’Callaghan. Renal calculi. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0166.

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Nephrolithiasis is the presence of kidney stones, which are also known as ‘renal calculi’. Renal calculi arise when urine becomes supersaturated with insoluble components. This may occur when there is excessive production of these components, a decrease in factors maintaining their solubility (e.g. citrate), or a reduction in urine volume (leading to increased concentration). Infection may play a significant role in the initiation of renal calculus formation, by creating a nidus for further crystal growth. Renal calculi are usually classified into two categories: those containing calcium (80%), and non-calcareous calculi (20%).
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21

Georgiev, Svetlin. Foundations of Iso-Differential Calculus Vol. 5: Iso-Stochastic Differential Equations. Nova Science Publishers, Incorporated, 2015.

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22

Georgiev, Svetlin. Foundations of Iso-Differential Calculus: Theory of Iso-Functions of a Real Iso-Variable. Nova Science Publishers, Incorporated, 2016.

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23

Georgiev, Svetlin. Foundations of Iso-Differential Calculus: Iso-Dynamic Equations Georgiev. Nova Science Publishers, Incorporated, 2015.

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24

Georgiev, Svetlin. Foundations of Iso-Differential Calculus Vol. 3: Ordinary Iso-Differential Equations. Nova Science Publishers, Incorporated, 2015.

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25

Georgiev, Svetlin. Foundations of Iso-Differential Calculus: Volume 4 -- Iso-Dynamic Equations. Nova Science Publishers, Incorporated, 2015.

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26

Rohleder, J., and E. Kroker. Calcium Carbonate: From the Cretaceous Period into the 21st Century. Birkhauser, 2002.

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27

Wolfgang, Tegethoff F., Rohleder Johannes, and Kroker Evelyn, eds. Calcium carbonate: From the Cretaceous period into the 21st century. Basel: Birkhäuser Verlag, 2001.

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28

Georgiev, Svetlin. Foundations of Iso-Differential Calculus. Nova Science Publishers, Incorporated, 2014.

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29

Georgiev, Svetlin. Foundations of Iso-Differential Calculus. Nova Science Pub Inc, 2014.

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30

Ho, Christopher P. Milk of Calcium. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0035.

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Milk of calcium (MOC) is simply calcium oxalate crystals precipitated within the microcysts (acini) of the breast lobules. MOC has a very distinct mammographic appearance. It is a benign entity, and when seen and properly identified, it requires no further workup or follow-up. It is, however, important to recognize the proper initial evaluation of MOC so as to avoid misdiagnosis or potential unnecessary biopsies.This chapter, appearing in the section on calcifications, reviews the key imaging and clinical features, imaging protocols and pitfalls, differential diagnoses, and management recommendations for milk of calcium. Topics discussed include appropriate use of magnification views, hints to recognize the distinct appearance of milk of calcium, and management.
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31

Smith, Robert Thomas, Roland B. Minton, Robert Smith undifferentiated, and Roland Minton. Insights Into Calculus Using Maple. McGraw-Hill Science/Engineering/Math, 2001.

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32

William A, Schabas. Part 12 Financing: Financement, Art.117 Assessment of contributions/Calcul des contributions. Oxford University Press, 2016. http://dx.doi.org/10.1093/law/9780198739777.003.0122.

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This chapter comments on Article 117 of the Rome Statute of the International Criminal Court. Article 115(a) of the Rome Statute declares that one of the two sources of the funds of the Court shall be assessed contributions made by States Parties. Article 117 recognizes the principle of differential assessment, using the United Nations scale of assessments as the model, but it does not impose a precise formula. The task of specifying the assessed contributions falls, by implication, to the Assembly of States Parties. Each year, the Assembly of States Parties adopts a resolution on the assessment. The most recent ‘[d]ecides that for 2015, the contributions of States Parties shall be assessed in accordance with an agreed scale of assessment, based on the scale adopted by the United Nations for its regular budget applied for 2013–2015, and adjusted in accordance with the principles on which the scale is based’.
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33

Chakera, Aron, William G. Herrington, and Christopher A. O’Callaghant. Disorders of plasma calcium. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0175.

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The extracellular calcium ion concentration is tightly regulated through the actions of parathyroid hormone (PTH) and vitamin D (1,25-dihydroxyvitamin D) on bone, kidney, and intestines. Abnormalities in these homeostatic mechanisms may lead to increased or decreased serum calcium concentrations, resulting in hypercalcaemia or hypocalcaemia, respectively. Hypercalcaemic disorders may be further divided into those associated with a high/high-normal serum PTH level, and those associated with a low serum PTH concentration. Hypocalcaemia occurs when abnormalities in the physiological regulation of PTH and vitamin D results in calcium levels lower than the desired normal range. Failure of release of calcium from bone, and increased binding of calcium in the circulation, are other factors causing hypocalcaemia. This chapter discusses hypercalcaemia and hypocalcaemia, exploring definitions of the diseases, their etiologies, typical and uncommon symptoms, demographics, natural history, complications, diagnostic approaches, other diagnoses that should be considered, prognosis, and treatment.
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34

Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Calcium in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0018.

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Most calcium in the body is present in the skeleton, where it serves a structural role and also as a reservoir for use in other tissues. During pregnancy, calcium is accumulated in the fetal skeleton, mostly during the third trimester when bone growth is at its peak. Although this increases the demand on maternal bone stores, the calcium transfer to the fetus is balanced by increased intestinal calcium absorption in the mother, mediated by compensatory changes in vitamin D synthesis and endogenous hormone levels. Bone loss is minimized if calcium intake is maintained at 1,000#amp;#x2013;1,200 mg/day during pregnancy. This intake level builds up calcium stores in early pregnancy for increased fetal transfer in the third trimester. Additional dietary calcium is usually not required if pre-pregnancy intake is adequate, although pregnant adolescents and women carrying multiple fetuses may require supplementation.
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35

Ahmed, Tanveer. A detailed investigation into the production of calcium alginate fibre from various algal sources. 1994.

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36

Reynard, John, and Ben Turney. Kidney stones. Edited by John Reynard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0019.

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This chapter summarizes the variety of ways in which kidney stones can present, clinical findings in patients with renal stones, and the diagnostic tests used to identify them. Plain radiography remains a good way of identifying renal stones if calcified, will identify cysteine stones which are relatively radiolucent, but cannot ‘see’ non-calcium-containing stones (e.g. uric acid, triamterene, indinavir). The sensitivity of ultrasound for detecting renal calculi is variably reported at between 50–95%. Unenhanced computed tomography (CT) is nowadays regarded as the diagnostic gold standard for identifying renal calculi, for measuring their size and number and, to a lesser degree, determining their location. Where doubt exists over stone location, precise determination requires either CT urography or retrograde ureterorenography. The ‘limitation’ of CT is its radiation dose, but as a single ‘upfront’ diagnostic test, there is no substitute.
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37

Abhishek, Abhishek, and Michael Doherty. Pathophysiology of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0049.

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Calcium pyrophosphate (CPP) dihydrate crystals form extracellularly. Their formation requires sufficient extracellular inorganic pyrophosphate (ePPi), calcium, and pro-nucleating factors. As inorganic pyrophosphate (PPi) cannot cross cell membranes passively due to its large size, ePPi results either from hydrolysis of extracellular ATP by the enzyme ectonucleotide pyrophosphatase/phosphodiesterase 1 (also known as plasma cell membrane glycoprotein 1) or from the transcellular transport of PPi by ANKH. ePPi is hydrolyzed to phosphate (Pi) by tissue non-specific alkaline phosphatase. The level of extracellular PPi and Pi is tightly regulated by several interlinked feedback mechanisms and growth factors. The relative concentration of Pi and PPi determines whether CPP or hydroxyapatite crystal is formed, with low Pi/PPi ratio resulting in CPP crystal formation, while a high Pi/PPi ratio promotes basic calcium phosphate crystal formation. CPP crystals are deposited in the cartilage matrix (preferentially in the middle layer) or in areas of chondroid metaplasia. Hypertrophic chondrocytes and specific cartilage matrix changes (e.g. high levels of dermatan sulfate and S-100 protein) are related to CPP crystal deposition and growth. CPP crystals cause inflammation by engaging with the NALP3 inflammasome, and with other components of the innate immune system, and is marked with a prolonged neutrophilic inflitrate. The pathogenesis of resolution of CPP crystal-induced inflammation is not well understood.
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38

Turney, Ben, and John Reynard. Prevention of idiopathic calcium stones. Edited by John Reynard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0015.

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The main principles of idiopathic calcium oxalate stone prevention are to maintain dilute urine through increasing fluid intake and to reduce calcium and oxalate excretion. The influence of various urinary factors on the risk of stone formation has been quantified mathematically. Urine volume and urinary oxalate concentration are most influential on the risk of stone formation, while magnesium concentration contributes a small amount to risk. It is estimated that around 50% of stone formers will form another stone within five years. Some stone formers have frequent recurrences. Most stone formers ask how they can prevent future episodes. Advice can be generic or personalized, and treatment may include changes to diet, fluid intake, and addition of drugs to alter urine biochemistry.
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39

Abhishek, Abhishek, and Michael Doherty. Investigations of calcium pyrophosphate deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0051.

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Joint aspiration and microscopic examination of the aspirated synovial fluid remains the gold standard for the diagnosis of calcium pyrophosphate crystal deposition (CPPD). If synovial fluid aspiration is not feasible, plain radiography and/or ultrasound scanning may be used to detect chondrocalcinosis (CC) which predominantly occurs due to calcium pyrophosphate (CPP) crystals, and this can be used as a diagnostic surrogate for CPPD as suggested by the EULAR Task Force. Acute CPP crystal arthritis often associates with a brisk acute phase response (elevated C-reactive protein (CRP) and/or erythrocyte sedimentation rate (ESR), plasma viscosity) and neutrophilia. A mildly raised CRP and/or ESR may be present in chronic CPP crystal inflammatory arthritis. On the contrary, asymptomatic CC, or CPPD with osteoarthritis does not cause raised acute phase reactants. As CPPD most commonly occurs due to increasing age and osteoarthritis, investigations to screen for underlying metabolic abnormalities should be carried out in those with early-onset CPPD (under 55 years), or in those with florid polyarticular CC. As hyperparathyroidism gets more common with ageing its presence should be specifically sought in all age groups. Tests for other predisposing metabolic conditions should only be carried out in the presence of specific clinical features. Genotyping for mutations, especially in the ANKH gene, may be warranted in those with a family history of premature CPPD and no evidence of inherited metabolic predisposition, but such testing is unavailable to most clinicians.
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40

Roddy, Edward, and Michael Doherty. Calcium pyrophosphate crystal deposition (CPPD). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0142.

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Calcium pyrophosphate crystal deposition (CPPD) in articular cartilage is a common age-related phenomenon. Recent important advances in our understanding of the pathophysiology of pyrophosphate metabolism include the identification of a mutation within the ANK gene which associates with familial CPPD, and elucidation of the interleukin-1β‎ (IL-1β‎)-dependent mechanisms by which crystals invoke an inflammatory response. Risk factors for CPPD include age, prior joint damage and osteoarthritis, genetic factors, and occasionally metabolic diseases (hyperparathyroidism, haemochromatosis, hypomagnesaemia, and hypophosphatasia). CPPD is commonly asymptomatic or may present as osteoarthritis with CPPD, acute calcium pyrophosphate (CPP) crystal arthritis, or chronic CPP crystal inflammatory arthritis. Although radiographic chondrocalcinosis is often taken to be synonymous with CPPD, other calcium crystals can also have this appearance and definitive diagnosis requires identification of CPP crystals by compensated polarized light microscopy of aspirated synovial fluid. Recently, the ultrasonographic appearances of CPPD have been described. Treatment of CPPD is targeted to the clinical presentation. Acute CPP crystal arthritis is treated by aspiration and injection of glucocorticosteroid, local ice packs, non-steroidal anti-inflammatory drugs (NSAIDS), low-dose colchicine, oral or parenteral glucocorticosteroids, or adrenocorticotrophic hormone (ACTH). Treatment of osteoarthritis with CPPD is very similar to the treatment of osteoarthritis alone. There is no specific therapy for chronic CPP crystal inflammatory arthritis: options include NSAID, low-dose colchicine, low-dose glucocorticosteroid, methotrexate, and hydroxychloroquine. Recommendations for the management of CPPD are derived from a small evidence base and largely based on clinical experience and extrapolation from gout. Further research into diagnosis and management including novel treatment strategies such as IL-1β‎ blockade is much needed.
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41

Abhishek, Abhishek, and Michael Doherty. Clinical features of calcium pyrophosphate crystal deposition. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0050.

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Calcium pyrophosphate deposition (CPPD) occurs in the elderly, and is commonly asymptomatic. However, it can cause acute calcium pyrophosphate (CPP) crystal arthritis, chronic CPP crystal inflammatory arthritis, and is frequently present in joints with osteoarthritis (OA). Acute CPP crystal arthritis presents with rapid onset of acute synovitis, which frequently affects the knees, wrists, shoulders, and elbows. It can mimic sepsis in the elderly, and may require hospital admission. Patients with CPPD plus OA may have more inflammatory signs and symptoms (e.g. joint swelling, stiffness) than those with OA alone. Additionally, patients with CPPD plus OA may also have intermittent attacks of acute CPP crystal arthritis. Some patients with CPPD may have more chronic inflammatory joint involvement and are classified as chronic CPP crystal inflammatory arthritis. This chapter describes the clinical features and differential diagnosis of common clinical manifestations of CPPD and outlines some of its rarer manifestations.
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42

Rosengart, Matthew R. Disorders of calcium in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0253.

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Calcium is vitally important for normal cellular signalling and function. However, its toxicity necessitates that intracellular calcium concentration [Ca2+] be tightly regulated and compartmentalized. Evolutionary pressures have yielded several regulatory mechanisms to maintain intracellular and extracellular ionized calcium concentrations compatible with life. During periods of critical illness these process are commonly overwhelmed, and disorders of calcium homeostasis are highly prevalent among intensive care unit (ICU) patients. Indeed, hypocalcaemia occurs in up to 88% of critically-ill ICU patients suffering from trauma, sepsis, and burns. Contemporary evidence suggests that although hypocalcaemia may be associated with ICU mortality, it is not in the causal pathway. A systematic review concluded there are no data to support the routine parenteral administration of calcium in the management of asymptomatic critical illness-related hypocalcaemia. Asymptomatic hypocalcaemia of critical illness does not necessitate replacement. However, acute, symptomatic hypocalcaemia necessitates parenteral supplementation to prevent tetany, seizures, and cardiac arrhythmias
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43

Ward, William, and Doug Wilberscheid. Insights into Calculus: Projects Using Texas Instruments Graphics Calculators. Prentice Hall, 1996.

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44

Ward, William, and Doug Wilberscheid. Insights into Calculus: Projects Using Texas Instruments Graphics Calculators. Prentice Hall, 1996.

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45

Fye, W. Bruce. Transforming Cardiac Catheters into Treatment Tools. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199982356.003.0016.

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Percutaneous transluminal coronary angioplasty (PTCA) transformed the cardiac catheter from a diagnostic tool into a treatment tool. The technology involved a special catheter fitted with a balloon near its tip that could be blown up to expand a narrowed coronary artery segment. For patients with angina, the procedure was an attractive alternative to coronary bypass surgery. Mayo cardiologists were among the first to adopt angioplasty and to call for controlled clinical trials to compare it to bypass surgery. Initially, cardiologists (who already performed coronary angiography) learned to perform PTCA informally. After attending one or more live demonstration courses, many began to perform angioplasty in their local hospitals. The philosophy in many contexts was “see one, do one.” By the mid-1980s, however, more rigorous training expectations were elaborated. Heart specialists who performed PTCA were described as “interventional cardiologists,” a phrase that acknowledged that this catheter-based treatment had immediate effects.
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46

Solomon, Norman. 7. Out of the ghetto, into the whirlwind. Oxford University Press, 2014. http://dx.doi.org/10.1093/actrade/9780199687350.003.0008.

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What are the different ‘sects’ (or denominations as they prefer to be called) of Judaism? ‘Out of the ghetto, into the whirlwind’ asks why divisions exist and how they came about. It examines Reform and Orthodox Judaism in particular but also considers Conservative and Reconstructionist Judaism. Conservative Judaism is particularly strong in the USA, where it may be the largest single Jewish denomination. Reconstructionists call for a reappraisal of Judaism, including such fundamental concepts as God, Israel, and Torah and institutions such as the Synagogue, in light of contemporary thinking.
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47

Gould, Karen. The Canadian Distinctiveness into the XXIst Century - La distinction canadienne au tournant du XXIe siecle. Edited by Chad Gaffield. University of Ottawa Press, 2003. http://dx.doi.org/10.1353/book.6595.

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In this collection of essays some of Canada's foremost writers and thinkers, including John Ralston Saul and Margaret Atwood, call for equilibrium among economics, culture, and technological change. While promoting the dynamism and change possible in Canadian society, they also call for a re-examination of Canada's past in order to chart its future.
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48

Gallo, Jason. Translating Science into Policy and Legislation. Edited by Kathleen Hall Jamieson, Dan M. Kahan, and Dietram A. Scheufele. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190497620.013.27.

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Evidence-informed policy is a deliberate process that features analysis of evidence as a necessary step to reaching a public policy decision. Risk is inherent in policy decisions, and decision-makers must often balance consideration of costs; social, economic, and environmental impacts; differential outcomes for various stakeholders; and political considerations. Policymakers rely on evidence to help reduce uncertainty and mitigate these risks. This chapter considers the policymaking process as infrastructure and takes a constructivist approach to the development of evidence. It highlights the reflexivity between the demand for, and supply of, evidence and issues of power, authority, expertise, and inclusion. Finally, the chapter addresses the challenges of applying evidence to complex problems where multiple, heterogeneous variables affect outcomes and concludes with a call for further research to examine the decisions, values, and norms embedded in the design and development of the technical architectures and processes used in policy analysis and decision support.
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49

Bakken, Tore. George Spencer-Brown (1923b). Edited by Jenny Helin, Tor Hernes, Daniel Hjorth, and Robin Holt. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199669356.013.0030.

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George Spencer Brown is a British mathematician and logician, whose book Laws of Form (1969) tackles the very foundation of development of knowledge. An abstract and bold ‘calculus of indications’, Laws of Form was even praised by Bertrand Russell. It presents a calculus that seeks to clarify the laws governing the formation of forms. This chapter examines Spencer Brown’s form calculus and its implications for the study of organizations. After providing an overview of mathematics as a cognition theory, it describes some basic main points of Spencer Brown’s form calculus and its implications for process philosophy. It then discusses the two ‘arithmetic axioms’ developed by Spencer Brown in Laws of Form: the law of calling and the law of crossing. Finally, it shows how the calculus can help elucidate self-reference and paradoxes.
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50

Carberry, George, and Orhan Ozkan. Use of a Fogarty Balloon Catheter to Create Backwall Support and Facilitate Intrahepatic Bile Duct Access During Antegrade Stone Extraction. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0086.

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Percutaneous transhepatic stone removal may be indicated in patients with altered upper gastrointestinal anatomy precluding use of endoscopic stone extraction. When biliary calculi are located in a duct adjacent to the duct cannulated percutaneously, obtaining wire and catheter access into the target duct may be difficult due to the acute angles required of the wire and catheter to access the stone-containing duct. One useful method described and illustrated in this chapter to address this issue involves inflating a balloon catheter downstream from the origin of the target duct to deflect a wire into the target duct and to provide backwall support at the apex of the wire for advancement of the stiff balloon catheter. Once the duct containing the biliary calculi is accessed, sweeps of the calculi can be performed.
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