Relevant bibliographies by topics / Carcinogenesis; Cancer

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  1. Dissertations / Theses

Academic literature on the topic 'Carcinogenesis; Cancer'

Author: Grafiati
Published: 4 June 2021
Last updated: 5 February 2022

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Dissertations / Theses on the topic "Carcinogenesis; Cancer"

1

Fischer, Heléne. "Gene expression in carcinogenesis /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4961-1/.

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2

陳君怡 and Kwan-yi Queeny Chan. "Molecular studies on endometrial and ovarian carcinogenesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39634474.

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3

Zeki, Sebastian Simon. "Clonal interactions in Barrett's carcinogenesis." Thesis, Queen Mary, University of London, 2013. http://qmro.qmul.ac.uk/xmlui/handle/123456789/9069.

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Introduction: Barrett’s oesophagus (BO) is a metaplastic premalignant disease which can undergo a metaplasia-­‐dysplasia-­‐adenocarcinoma pathway. It represents an example of field cancerization by which an area occupied by BO can undergo molecular and genetic changes associated with carcinogenesis without being phenotypically cancerous. Previous work suggested that non-­‐cancerous BO contains a monoclonal population. More recent work demonstrated that premalignant Barrett’s fields are polyclonal suggesting that clonal interactions may be important in carcinogenesis. It is the aim of this thes
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4

Yonekura, Satoru. "Cancer-Induced Ileopathy and Dysbiosis Contribute to Carcinogenesis." Thesis, université Paris-Saclay, 2020. http://www.theses.fr/2020UPASL033.

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Le microbiote intestinal est important pour l'immunothérapie anticancéreuse en tant que régulateur clé de l'immunité de l'hôte. La dysbiose chez les patients cancéreux peut être délétère pour le métabolisme et le système immunitaire des patients, aggravant alors la croissance tumorale. Cependant, le mécanisme sous-jacent ainsi que la façon dont modifier ce microbiote n'ont pas encore clairement élucidés. Dans plusieurs modèles de tumeurs sous-cutanées murines, nous avons trouvé un rapport de longueur des villosités / crypte diminué, corrélé à une augmentation de la perméabilité intestinale et
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5

Frede, Julia. "Cellular dynamics in Oesophageal Squamous Carcinogenesis." Thesis, University of Cambridge, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708817.

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6

Cheung, Yen-Ling. "The role of CYP1A induction in chemical carcinogenesis." Thesis, University of Surrey, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.239996.

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7

Ho, Chung-man. "Non-small cell lung cancer from bench to bedside /." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B39432592.

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8

Mellick, Albert S. Jr, and n/a. "Tissue Specific Gene Expression Patterning and Carcinogenesis." Griffith University. School of Health Science, 2004. http://www4.gu.edu.au:8080/adt-root/public/adt-QGU20041102.114313.

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Despite significant advances in diagnosis and treatment, breast cancer remains the leading cause of cancer-related deaths in Australian women. Colorectal cancer is the second most common cancer in both males and females; after prostate and breast cancer, respectively, and excluding non-melanocytic skin cancer. Both breast cancer and colorectal cancer follow a common progressive course of illness; presenting (at least initially) with benign symptoms that can be treated by ablation (or removal) of the affected area. Cancer progression is associated with breakdown of tissue barriers (such as base
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9

Efstathiou, Jason Alexander. "The role of adhesion molecules in colorectal carcinogenesis." Thesis, University of Oxford, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670210.

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10

Al-Damouk, Jawdet Dakhel. "Malnutrition and experimental oral carcinogenesis." Thesis, University of Glasgow, 1988. http://theses.gla.ac.uk/2731/.

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The work was undertaken to examine the effects of nutritional deficiencies on cancer induction. Two of the most common and widely distributed nutrients, iron and folic acid, were examined to evaluate the effects of their deficiency on animals. The Syrian golden hamster was the animal model for all experimental work. In the first part of the study an attempt was made to induce iron deficiency in young adult male hamsters by feeding iron deficient diet coupled with repeated venesection of 1.5ml every two weeks. Following twelve weeks on this regime a superficial biopsy was taken, on week 13, fro
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