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Books on the topic 'Cardiac injury'

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Published: 4 June 2021
Last updated: 26 July 2025

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1

Ošt’ádal, Bohuslav, and František Kolář. Cardiac Ischemia: From Injury to Protection. Springer US, 1999. http://dx.doi.org/10.1007/978-1-4757-3025-8.

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2

Ostadal, Bohuslav. Cardiac ischemia: From injury to protection. Kluwer Academic Publishers, 1999.

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3

A, Jonas Richard, Newburger Jane W, and Volpe Joseph J, eds. Brain injury and pediatric cardiac surgery. Butterworth Heinemann, 1996.

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4

František, Kolář, ed. Cardiac ischemia: From injury to protection. Kluwer Academic Publishers, 1999.

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5

Friedhelm, Beyersdorf, ed. Ischemia-reperfusion injury in cardiac surgery. Landes Bioscience, 2000.

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6

Michel, Piper Hans, and Preusse C. J, eds. Ischemia-reperfusion in cardiac surgery. Kluwer Academic Publishers, 1993.

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7

A, Salerno Tomas, and Ricci Marco, eds. Myocardial protection. Blackwell Pub., 2004.

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8

Initiative, Acute Dialysis Quality, Conference on Biomarkers in AKI (10th : 2011 : Dublin, Ireland), and Conference on CRS (11th : 2012 : Venice, Italy), eds. ADQI consensus on AKI biomarkers and cardiorenal syndromes. Karger, 2013.

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9

Ost'ádal, Bohuslav, and Frantisek Kolár. Cardiac Ischemia: From Injury to Protection. Springer London, Limited, 2013.

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10

Jonas, Richard A., Jane W. Newburger, Joseph J. Volpe, and John W. Kirklin. Brain Injury and Pediatric Cardiac Surgery. CRC Press, 2019. http://dx.doi.org/10.1201/9780367813864.

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11

Volpe, Joseph, Richard Jonas, and Jane Newburger. Brain Injury and Pediatric Cardiac Surgery. Taylor & Francis Group, 2019.

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12

Volpe, Joseph, Richard Jonas, and Jane Newburger. Brain Injury and Pediatric Cardiac Surgery. Taylor & Francis Group, 2019.

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13

Volpe, Joseph, Richard Jonas, and Jane Newburger. Brain Injury and Pediatric Cardiac Surgery. Taylor & Francis Group, 2019.

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14

Volpe, Joseph, Richard Jonas, and Jane Newburger. Brain Injury and Pediatric Cardiac Surgery. Taylor & Francis Group, 2019.

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15

Ostádal, Bohuslav, and Frantisek Kolár. Cardiac Ischemia: From Injury to Protection. Springer, 2010.

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16

Beyersdorf, Friedhelm. Ischemia-Reperfusion Injury in Cardiac Surgery. Taylor & Francis Group, 2000.

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17

Rivera-Lara, Lucia, and Romergryko G. Geocadin. Neurobiology of Brain Injury after Cardiac Arrest. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0189.

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The publication of two sentinel, multicenter, randomized controlled trials in The New England Journal of Medicine in 2002 provided evidence for the beneficial use of therapeutic hypothermia (TTH) to 32° to 34°C in resuscitated patients after cardiac arrest with a shockable rhythm. The number needed to treat to provide a favorable neurological outcome was 6, and TTH is a recommended treatment in the American Heart Association (AHA) Resuscitation Guidelines. This chapter describes the biological basis of disorders of arousal and awareness after cardiac arrest, the mechanisms of ischemic cell dea
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18

McLean, Anthony S., and Stephen J. Huang. Cardiac injury biomarkers in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0301.

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To be clinically relevant, a good cardiac biomarker should have four main characteristics. It should be organ-, disease- and stage-specific to be useful in diagnosis. Its release should be timely and its half-life should be long enough to make measurement possible and meaningful. Its serum or blood concentration should be proportional to disease severity; hence, can be used as a monitoring tool. Finally, their concentrations have implications on long-term outcomes. To date, only a handful of cardiac biomarkers have clinical relevance in the intensive care setting—cardiac troponins (as a marker
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19

Ischemia-Reperfusion in Cardiac Surgery. Island Press, 1993.

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20

Chong, Ji Y., and Michael P. Lerario. Cardiac Arrest. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0028.

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Hypoxic–ischemic brain injury is common following cardiopulmonary arrest and is associated with high rates of mortality and morbidity. Therapeutic hypothermia has been helpful in increasing survival and functional outcomes in these patients. The neurological examination, neuroimaging studies, and ancillary serological and neurophysiological testing can be helpful in prognostication post-arrest.
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21

Pitcher, Joseph H., and David B. Seder. Neuroprotection for Cardiac Arrest. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0009.

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This chapter reviews the pathophysiology of brain injury after resuscitation from cardiac arrest and describes a pragmatic approach to neuroprotection. Common mechanisms of brain injury in the postresuscitation milieu are discussed and strategies for optimizing physiological variables such as blood pressure, oxygen, ventilation, and blood glucose in order to minimize secondary injury are presented. Neuroprotective therapies, such as targeted temperature management and pharmacologic neuroprotective agents, are covered in detail. Finally, the use of raw and processed electroencephalography and o
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22

Gevaert, Sofie A., Eric Hoste, and John A. Kellum. Acute kidney injury. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0068.

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Acute kidney injury is a serious condition, occurring in up to two-thirds of intensive care unit patients, and 8.8-55% of patients with acute cardiac conditions. Renal replacement therapy is used in about 5-10% of intensive care unit patients. The term cardiorenal syndrome refers to combined heart and kidney failure; three types of acute cardiorenal syndrome have been described: acute cardiorenal syndrome or cardiorenal syndrome type 1, acute renocardiac syndrome or cardiorenal syndrome type 3, and acute cardiorenal syndrome type 5 (cardiac and renal injury secondary to a third entity such as
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23

Gevaert, Sofie A., Eric Hoste, and John A. Kellum. Acute kidney injury. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0068_update_001.

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Acute kidney injury is a serious condition, occurring in up to two-thirds of intensive care unit patients, and 8.8-55% of patients with acute cardiac conditions. Renal replacement therapy is used in about 5-10% of intensive care unit patients. The term cardiorenal syndrome refers to combined heart and kidney failure; three types of acute cardiorenal syndrome have been described: acute cardiorenal syndrome or cardiorenal syndrome type 1, acute renocardiac syndrome or cardiorenal syndrome type 3, and acute cardiorenal syndrome type 5 (cardiac and renal injury secondary to a third entity such as
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24

Hori, Masatsugu, Robert S. Reneman, and Yukio Maruyama. Cardiac Adaptation and Failure. Springer, 2013.

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25

Hori, Masatsugu, Robert S. Reneman, and Yukio Maruyama. Cardiac Adaptation and Failure. Springer London, Limited, 2013.

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26

Brain Injury and Cardiac Arrest, An Issue of Neurologic Clinics. Saunders, 2006.

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27

(Editor), Bohuslav Ost'ádal, and Frantisek Kolár (Editor), eds. Cardiac Ischemia: - From Injury to Protection (Basic Science for the Cardiologist). Springer, 1999.

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28

Schultz, Vince L. Commotio Cordis: The Potential Traumatic Injury Behind Damar Hamlin's Cardiac Arrest. Independently Published, 2023.

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29

Brown, Jeremiah R., and Chirag R. Parikh. Cardiovascular surgery and acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0245.

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Over the last decade, cardiac surgery-associated acute kidney injury (AKI) has been recognized as a frequent adverse event following cardiac surgery. In this clinical context and others, AKI has been strongly associated with increased morbidity, mortality, and length of hospitalization. These adverse events that accompany AKI have been shown to be directly proportional to the magnitude of the peak rise in serum creatinine and the duration of AKI making AKI a costly complication and a target for prevention in hospitalized patients around the world. This chapter discusses the subsequent healthca
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30

Demetriades, Demetrios, Leslie Kobayashi, and Lydia Lam. Cardiac complications in trauma. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0062.

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Post-traumatic cardiac complications may occur after penetrating or blunt injuries to the heart or may follow severe extracardiac injuries. The majority of victims with penetrating injuries to the heart die at the scene and do not reach hospital care. For those patients who reach hospital care, an immediate operation, sometimes in the emergency room, cardiac injury repair, and cardiopulmonary resuscitation provide the only possibility of survival. Many patients develop perioperative cardiac complications such as acute cardiac failure, cardiac arrhythmias, coronary air embolism, and myocardial
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31

Demetriades, Demetrios, Leslie Kobayashi, and Lydia Lam. Cardiac complications in trauma. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0062_update_001.

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Post-traumatic cardiac complications may occur after penetrating or blunt injuries to the heart or may follow severe extracardiac injuries. The majority of victims with penetrating injuries to the heart die at the scene and do not reach hospital care. For those patients who reach hospital care, an immediate operation, sometimes in the emergency room, cardiac injury repair, and cardiopulmonary resuscitation provide the only possibility of survival. Many patients develop perioperative cardiac complications such as acute cardiac failure, cardiac arrhythmias, coronary air embolism, and myocardial
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32

Lam, Lydia, Leslie Kobayashi, and Demetrios Demetriades. Cardiac complications in trauma. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0062_update_002.

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Post-traumatic cardiac complications may occur after penetrating or blunt injuries to the heart or may follow severe extracardiac injuries. The majority of victims with penetrating injuries to the heart die at the scene and do not reach hospital care. For those patients who reach hospital care, an immediate operation, sometimes in the emergency room, cardiac injury repair, and cardiopulmonary resuscitation provide the only possibility of survival. Many patients develop perioperative cardiac complications such as acute cardiac failure, cardiac arrhythmias, coronary air embolism, and myocardial
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33

Lam, Lydia, Leslie Kobayashi, and Demetrios Demetriades. Cardiac complications in trauma. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0062_update_003.

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Post-traumatic cardiac complications may occur after penetrating or blunt injuries to the heart or may follow severe extracardiac injuries. The majority of victims with penetrating injuries to the heart die at the scene and do not reach hospital care. For those patients who reach hospital care, an immediate operation, sometimes in the emergency room, cardiac injury repair, and cardiopulmonary resuscitation provide the only possibility of survival. Many patients develop perioperative cardiac complications such as acute cardiac failure, cardiac arrhythmias, coronary air embolism, and myocardial
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34

Lucien, Jamie George. The necrotic and apoptotic injury of cardiac xenotransplants caused by human serum. 2001.

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35

Pepper, John. Cardioprotection During Cardiac Surgery. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199544769.003.0007.

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• Overall early mortality for cardiac surgery is low at 2–3% but in high risk patients it can be high as 10–15%• The demography of cardiac surgical patients is changing to older and sicker patients• Myocardial ischaemia-reperfusion injury and the systemic inflammatory response are closely related• Several pharmacological agents that have been demon-strated to confer cardioprotection in the experimental setting have been applied to the clinical setting of cardiac surgery. However, the transfer of these findings from the bench to the bedside has been largely disappointing• Potential cardioprotec
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36

Jumean, Marwan F., and Mark S. Link. Post-cardiac arrest arrhythmias. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0065.

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Our understanding of arrhythmias following resuscitated cardiac arrest has evolved over the past two decades to entail complex pathophysiological processes including, in part, ischaemia and ischaemia-reperfusion injury. Electrical instability after the return of spontaneous circulation (ROSC) is common, ranging from atrial fibrillation to recurrent ventricular tachycardia and fibrillation. Electrical instability following out-of-hospital cardiac arrest is most commonly due to myocardial ischaemia and post-arrest myocardial dysfunction. However, electrolyte disturbances, elevated catecholamine
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37

Lameire, Norbert. Prevention of acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0226_update_001.

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This chapter summarizes the pharmacological interventions that can be used in the prevention of acute kidney injury (AKI). These following interventions are discussed: the use and selection of vasopressors; the administration of loop diuretics and mannitol; vasodilating drugs including dopamine, atrial natriuretic peptide, nesiritide, fenoldopam, and adenosine antagonists. The role of N-acetylcysteine in the prevention of contrast-induced AKI and cardiac surgery is discussed. The chapter concludes with a summary of the potential role of insulin-like growth factor and erythropoietin in the prev
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38

Lameire, Norbert. Prevention of acute kidney injury. Edited by Norbert Lameire. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0224_update_001.

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The prevention of acute kidney injury (AKI) should start with an assessment of the risk to develop AKI, by identification of co-morbidities, use of potentially nephrotoxic medications, and early recognition of acute reversible risk factors associated with AKI. This chapter discusses first the most relevant general risk factors for AKI and describes the recent introduction of several surveillance systems. In addition, some specific risk factors play a role in the pathogenesis of post-cardiac surgery AKI. Finally risks associated with commonly used drugs such as non-steroidal anti-inflammatory d
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39

Elmer, Jonathan, and Abhishek Freyer. In-Hospital Cardiac Arrest (DRAFT). Edited by Raghavan Murugan and Joseph M. Darby. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190612474.003.0004.

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In-hospital cardiac arrest (IHCA) is a major public health problem. Despite its prevalence, there remains a paucity of high-level evidence to guide patient management during and after resuscitation from IHCA and most guidelines are extrapolated from studies of out-of-hospital cardiac arrest. This chapter reviews the cornerstones of IHCA management: early recognition, provision of high quality compressions, and early defibrillation of shockable rhythms. It also summarizes key actions in early post-resuscitation care, including multiple system organ support to prevent rearrest and restore hemody
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40

Nolan, Jerry P., and Michael J. A. Parr. Management after resuscitation from cardiac arrest. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0066.

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Systemic ischaemia during cardiac arrest and the reperfusion response after return of spontaneous circulation (ROSC) cause the post-cardiac arrest syndrome (PCAS). The severity and duration of this syndrome is determined by the cause and duration of cardiac arrest, quality of resuscitation, and interventions after ROSC. Four key clinical components are recognized—post-cardiac arrest brain injury, myocardial dysfunction, other organ ischaemia/reperfusion (e.g. liver, kidney), and potential persistence of the precipitating pathology causing the cardiac arrest. The interventions applied after ROS
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41

Cruz, Dinna N., Anna Giuliani, and Claudio Ronco. Acute kidney injury in heart failure. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0248.

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Acute kidney injury (AKI) occurring during heart failure (HF) has been labelled cardiorenal syndrome (CRS) type 1. CRS is defined as a group of ‘disorders of the heart and kidneys whereby acute or chronic dysfunction in one organ may induce acute or chronic dysfunction of the other’. This consensus definition was proposed by the Acute Dialysis Quality Initiative, with the aim to standardize those disorders where cardiac and renal diseases coexist. Five subtypes have been proposed, according to which organ is affected first (cardiac vs renal) and whether the dysfunction is acute or chronic. Ano
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42

Wise, Matt, and Paul Frost. ICU treatment of acute kidney injury. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0151.

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Traditionally, the etiology of acute kidney injury (AKI) is considered in terms of prerenal, renal, and obstructive causes. However, this categorization is less useful in the ICU, where the etiology of AKI is usually multifactorial and often occurs in the context of multi-organ failure. Hypotension, nephrotoxic drugs, and severe sepsis or septic shock are the most important identifiable factors. Less frequently encountered causes include pancreatitis, abdominal compartment syndrome, and rhabdomyolysis. Primary intrinsic renal disease such as glomerulonephritis is extremely uncommon. A previous
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43

Fichtner, Alexander, and Franz Schaefer. Acute kidney injury in children. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0239.

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In the past few decades, the overall incidence of acute kidney injury (AKI) in paediatric patients has increased and the aetiological spectrum has shifted from infection-related and intrinsic renal causes towards secondary forms of AKI related to exposure to nephrotoxic drugs and complex surgical, oncological, and intensive care manoeuvres. In addition, neonatal kidney impairment and haemolytic uraemic syndrome continue to be important specific paediatric causes of AKI raising unique challenges regarding prevention, diagnosis, and treatment. The search for new biomarkers is a current focus of
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44

Golper, Thomas A., Andrew A. Udy, and Jeffrey Lipman. Drug dosing in acute kidney injury. Edited by William G. Bennett. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0364.

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Drug dosing in acute kidney injury (AKI) is one of the broadest topics in human medicine. It requires an understanding of markedly altered and constantly changing physiology under many disease situations, the use of the drugs to treat those variety of diseases, and the concept of drug removal during blood cleansing therapies. Early in AKI kidney function may be supraphysiologic, while later in the course there may be no kidney function. As function deteriorates other metabolic pathways are altered in unpredictable ways. Furthermore, the underlying disorders that lead to AKI alter metabolic pat
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45

Nolan, Jerry P. Cardiopulmonary resuscitation and the post-cardiac arrest syndrome. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0006.

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Cardiac arrest is the most extreme of medical emergencies. If the victim is to have any chance of high-quality neurological recovery, cardiac arrest must be diagnosed quickly, followed by summoning for help as basic life support (chest compressions and ventilations) is started. In most cases, the initial rhythm will be shockable, but this will have often deteriorated to a non-shockable rhythm by the time a monitor and/or defibrillator is applied. While basic life support will sustain some oxygen delivery to the heart and brain and will help to slow the rate of deterioration in these vital orga
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46

Nolan, Jerry P. Cardiopulmonary resuscitation and the post-cardiac arrest syndrome. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0006_update_001.

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Cardiac arrest is the most extreme of medical emergencies. If the victim is to have any chance of high-quality neurological recovery, cardiac arrest must be diagnosed quickly, followed by summoning for help as basic life support (chest compressions and ventilations) is started. In most cases, the initial rhythm will be shockable, but this will have often deteriorated to a non-shockable rhythm by the time a monitor and/or defibrillator is applied. While basic life support will sustain some oxygen delivery to the heart and brain and will help to slow the rate of deterioration in these vital orga
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47

Bellomo, Rinaldo, and John R. Prowle. Pathophysiology of oliguria and acute kidney injury. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0211.

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Oliguria and acute kidney injury (AKI) are common in critically-ill patients with studies reporting AKI affecting more than 50% of critically-ill patients. AKI is independently associated with increased mortality and is a potentially modifiable aspect of critical illness. The pathogenesis of AKI is complex and varies according to aetiology. The most common trigger in ICU patients is sepsis—the pathophysiology of septic AKI is poorly understood and probably involves intrarenal haemodynamic and inflammatory processes. In the setting of septic AKI, the classic acute tubular necrosis described in
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48

Benson, Carolyn, and G. Bryan Young. Ethical and end-of-life issues after cardiac arrest. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0067.

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Many survivors of cardiac arrest, especially out-of-hospital cardiac arrest, suffer varying degrees of anoxic-ischaemic brain injury. Accurate neurological prognostication to determine which patients will have poor neurological outcome is important to guide appropriate medical care and advise surrogate decision makers. Accurate prognostication generally requires the presence of two or more negative prognostic indicators, especially following treatment with therapeutic hypothermia. Medical care should be directed at achieving survival that the patient would consider acceptable. Poor quality sur
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49

Salerno, Tomas A., and Marco Ricci. Myocardial Protection. Wiley & Sons, Incorporated, John, 2008.

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50

Salerno, Tomas A., and Marco Ricci. Myocardial Protection. Wiley & Sons, Limited, John, 2007.

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