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Journal articles on the topic 'Cardiac injury'

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1

Laouan Brem, Falmata, Miri Chaymae, Hammam Rasras, et al. "Acute Myocardial Injury Assessed by High-Sensitive Cardiac Troponin Predicting Severe Outcomes and Death in Hospitalized Patients with COVID-19 Infection." Clinical and Applied Thrombosis/Hemostasis 28 (January 2022): 107602962210902. http://dx.doi.org/10.1177/10760296221090227.

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Background Cardiac injury has been linked to a poor prognosis during COVID-19 disease. Nevertheless, the risk factors associated are yet to be thoroughly investigated. Objectives We sought to compare demographical characteristics and in-hospital outcomes in patients infected by the SARS-CoV-2 with and without cardiac injury, to further investigate the prevalence of acute cardiac injury as well as its impact on their outcomes in COVID-19-patients. Methods We included in a retrospective analysis, all COVID-19 patients admitted between October first and December first, 2020, at the University Hos
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2

N, Dublin, H. K. Chow, and Y. C. Lim. "Penetrating Cardiac Injury." Journal of Health and Translational Medicine 4, no. 2 (1999): 117–18. http://dx.doi.org/10.22452/jummec.vol4no2.12.

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3

Hsieh, Hung-Jen, Pan-Fu Kao, Hsuan-Li Huang, and Yu-Hsiang Chou. "Cardiac Stab Injury." Clinical Nuclear Medicine 35, no. 2 (2010): 121–22. http://dx.doi.org/10.1097/rlu.0b013e3181c7be78.

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4

&NA;. "BLUNT CARDIAC INJURY." Journal of Trauma: Injury, Infection, and Critical Care 33, no. 5 (1992): 649–50. http://dx.doi.org/10.1097/00005373-199211000-00001.

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5

Iserson, Kenneth V., and Dan Judkins. "BLUNT CARDIAC INJURY." Journal of Trauma: Injury, Infection, and Critical Care 36, no. 3 (1994): 462. http://dx.doi.org/10.1097/00005373-199403000-00050.

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6

HEALEY, MARK A., REA BROWN, and DAVID FLEISZER. "Blunt Cardiac Injury." Journal of Trauma: Injury, Infection, and Critical Care 30, no. 2 (1990): 137–46. http://dx.doi.org/10.1097/00005373-199002000-00002.

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7

Schultz, Jess M., and Donald D. Trunkey. "Blunt cardiac injury." Critical Care Clinics 20, no. 1 (2004): 57–70. http://dx.doi.org/10.1016/s0749-0704(03)00092-7.

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8

Marcolini, Evie G., and Joshua Keegan. "Blunt Cardiac Injury." Emergency Medicine Clinics of North America 33, no. 3 (2015): 519–27. http://dx.doi.org/10.1016/j.emc.2015.04.003.

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9

O'Connor, J., M. Ditillo, and T. Scalea. "Penetrating Cardiac Injury." Journal of the Royal Army Medical Corps 155, no. 3 (2009): 185–90. http://dx.doi.org/10.1136/jramc-155-03-02.

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10

Giurgius, Magdy, Fahad Al Asfar, Piyaray M. Dhar, and Nema Al Awadi. "Penetrating Cardiac Injury." Medical Principles and Practice 15, no. 1 (2005): 80–82. http://dx.doi.org/10.1159/000089392.

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11

Bock, Jeremy S., and R. Michael Benitez. "Blunt Cardiac Injury." Cardiology Clinics 30, no. 4 (2012): 545–55. http://dx.doi.org/10.1016/j.ccl.2012.07.001.

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12

Banki, Nader M., and Jonathan G. Zaroff. "Neurogenic cardiac injury." Current Treatment Options in Cardiovascular Medicine 5, no. 6 (2003): 451–58. http://dx.doi.org/10.1007/s11936-003-0034-8.

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13

PHILIPPIDES, GEORGE, ERIC AWTRY, and THOMAS J. RYAN. "Blunt Cardiac Injury." Cardiology News 6, no. 10 (2008): 16. https://doi.org/10.1016/s1544-8800(08)70395-6.

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14

Zhang, Jianfeng, Yicheng Wu, Anqing Chen, and Qiang Zhao. "Mesenchymal Stem Cells Promote Cardiac Muscle Repair via Enhanced Neovascularization." Cellular Physiology and Biochemistry 35, no. 3 (2015): 1219–29. http://dx.doi.org/10.1159/000373945.

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Background/Aims: Transplantation of bone-marrow derived stem/progenitor cells has an established therapeutic effect on cardiac muscle repair after injury. However, the exact mechanism that underlies this phenomenon is not completely understood. Methods: Here we transplanted mesenchymal stem cells (MSCs), a major population from the bone-marrow derived stem/progenitor cells, and studied its effects on cardiac muscle repair after injury. Results: MSCs transplantation significantly improved cardiac muscle repair after injury. The grafted MSCs did not significantly differentiate into cardiac muscl
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15

Jaworska-Wilczyńska, Maria, Mariusz Kuśmierczyk, Elżbieta Abramczuk, and Tomasz Hryniewiecki. "Post-cardiac injury syndrome." Polish Journal of Cardio-Thoracic Surgery 1 (2013): 20–26. http://dx.doi.org/10.5114/kitp.2013.34299.

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16

George, Elisabeth L., and Melanie Shatzer. "Troponin targets cardiac injury." Nursing Critical Care 2, no. 3 (2007): 10–12. http://dx.doi.org/10.1097/01.ccn.0000269800.35576.59.

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17

Dowd, M. Denise, and Steven Krug. "Pediatric Blunt Cardiac Injury." Journal of Trauma: Injury, Infection, and Critical Care 40, no. 1 (1996): 61–67. http://dx.doi.org/10.1097/00005373-199601000-00012.

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18

Stahl, Jonathan, and Nitsana A. Spigland. "Cardiac nail gun injury." Journal of Pediatric Surgery Case Reports 50 (November 2019): 101290. http://dx.doi.org/10.1016/j.epsc.2019.101290.

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19

George, Elisabeth L., and Melanie Shatzer. "Troponin targets cardiac injury." Nursing 38, Supplement (2008): 15–17. http://dx.doi.org/10.1097/01.nurse.0000312401.04652.5e.

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20

Kayki-Mutlu, Gizem, Kimberly M. Ferrero, and Walter J. Koch. "Sponging Away Cardiac Injury." JACC: Basic to Translational Science 7, no. 11 (2022): 1117–19. http://dx.doi.org/10.1016/j.jacbts.2022.06.019.

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21

Astrow, Jared, John MacKnight, and Peter Dean. "Cardiac Injury- Cross Country." Medicine & Science in Sports & Exercise 55, no. 9S (2023): 630. http://dx.doi.org/10.1249/01.mss.0000985696.03950.47.

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22

Sergio, Sandoval Tapia, and Sánchez Castrejón Esperanza. "Penetrating cardiac trauma. A case report." American Journal of Medicine and Surgery 7, no. 1 (2022): 17–20. https://doi.org/10.5281/zenodo.8088130.

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Cardiac trauma is the second most common cause of trauma-related death. Penetrating cardiac injury (PCI) is very lethal, with mortality rates of 70 % to 80 %. The common clinical presentation is cardiac tamponade with/ or hemorrhagic shock in most cases, which constitute a surgical emergency and require prompt treatment. Ultrasound and computed tomography (CT) are the main modality to evaluate it. The choice of the surgical approach depends on the experience of the surgeon, the expected injuries according to the probable trajectory of the wounds, and the evidence of associated injuries. We pre
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23

Kino, Tabito, Mohsin Khan, and Sadia Mohsin. "The Regulatory Role of T Cell Responses in Cardiac Remodeling Following Myocardial Infarction." International Journal of Molecular Sciences 21, no. 14 (2020): 5013. http://dx.doi.org/10.3390/ijms21145013.

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Ischemic injury to the heart causes cardiomyocyte and supportive tissue death that result in adverse remodeling and formation of scar tissue at the site of injury. The dying cardiac tissue secretes a variety of cytokines and chemokines that trigger an inflammatory response and elicit the recruitment and activation of cardiac immune cells to the injury site. Cell-based therapies for cardiac repair have enhanced cardiac function in the injured myocardium, but the mechanisms remain debatable. In this review, we will focus on the interactions between the adoptively transferred stem cells and the p
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24

Li, Lingfei, Xingyue Zhang, Qiong Zhang, et al. "Myocardial Adipose Triglyceride Lipase Overexpression Protects against Burn-Induced Cardiac Lipid Accumulation and Injury." Oxidative Medicine and Cellular Longevity 2019 (May 16, 2019): 1–16. http://dx.doi.org/10.1155/2019/6428924.

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Maladaptive cardiac metabolism is a common trigger of cardiac lipid accumulation and cardiac injury under serious burn challenge. Adipose triglyceride lipase (ATGL) is the key enzyme that catalyzes triglyceride hydrolysis; however, its alteration and impact on cardiac function following serious burn injury are still unknown. Here, we found that the cardiac fatty acid (FA) metabolism increased, accompanied by augmented FA accumulation and ATGL expression, after serious burn injury. We generated heterozygous ATGL knockout and heterozygous cardiac-specific ATGL overexpression thermal burn mice. T
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25

Ibrahim, Ahmed, Ahmed Megahed, Ahmed Salem, and Osama Zekry. "Impact of Cardiac Injury on the Clinical Outcome of Children with Convulsive Status Epilepticus." Children 9, no. 2 (2022): 122. http://dx.doi.org/10.3390/children9020122.

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Objectives: the aim of this study was to determine the impact of cardiac injury on clinical profile, cardiac evaluation and outcome in patients hospitalized with convulsive status epilepticus (CSE). Materials and methods: this prospective observational study included 74 children with CSE. Cardiac injury was evaluated and defined using combination of cardiac troponin, electrocardiography (ECG) and echocardiography. Clinical outcome and mortality rates were compared in patients with and without cardiac injury. Results: A total of 74 patients with CSE were included in the study. Thirty-six (48.6%
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26

&NA;. "Cardiac troponin T measurements can identify cardiac injury." Inpharma Weekly &NA;, no. 776 (1991): 18. http://dx.doi.org/10.2165/00128413-199107760-00045.

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27

van Beek, Maria H. C. T., Richard C. Oude Voshaar, Femke M. van Deelen, Anton J. L. M. van Balkom, Gheorghe Pop, and Anne E. M. Speckens. "Inverse Correlation Between Cardiac Injury and Cardiac Anxiety." Journal of Cardiovascular Nursing 29, no. 5 (2014): 448–53. http://dx.doi.org/10.1097/jcn.0b013e3182982550.

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28

Ishida, Kenichiro, Yusuke Katayama, Tetsuhisa Kitamura, et al. "Factors Associated with Cardiac/Pericardial Injury among Blunt Injury Patients: A Nationwide Study in Japan." Journal of Clinical Medicine 11, no. 15 (2022): 4534. http://dx.doi.org/10.3390/jcm11154534.

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The lack of established diagnostic criteria makes diagnosing blunt cardiac injury difficult. We investigated the factors associated with blunt cardiac injury using the Japan Trauma Data Bank (JTDB) in a multicenter observational study of blunt trauma patients conducted between 2004 and 2018. The primary outcome was the incidence of blunt cardiac/pericardial injury. Multivariable logistic regression analysis was used to identify factors independently associated with blunt cardiac injuries. Of the 228,513 patients, 1002 (0.4%) had blunt cardiac injury. Hypotension on hospital arrival (adjusted o
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29

Li, Lin, Qi Zhou, and Jiancheng Xu. "Changes of Laboratory Cardiac Markers and Mechanisms of Cardiac Injury in Coronavirus Disease 2019." BioMed Research International 2020 (May 27, 2020): 1–7. http://dx.doi.org/10.1155/2020/7413673.

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Some patients with coronavirus disease 2019 (COVID-19) show abnormal changes in laboratory myocardial injury markers, suggesting that patients with myocardial injury have a higher mortality rate than those without myocardial injury. This article reviews the possible mechanism of myocardial injury in patients with COVID-19. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) affects the patients with COVID-19 in aspects of direct infection of myocardial injury, specific binding to functional receptors on cardiomyocytes, and immune-mediated myocardial injury. During hospitalization, the
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30

Bektik, Emre, and Ji-dong Fu. "Ameliorating the Fibrotic Remodeling of the Heart through Direct Cardiac Reprogramming." Cells 8, no. 7 (2019): 679. http://dx.doi.org/10.3390/cells8070679.

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Coronary artery disease is the most common form of cardiovascular diseases, resulting in the loss of cardiomyocytes (CM) at the site of ischemic injury. To compensate for the loss of CMs, cardiac fibroblasts quickly respond to injury and initiate cardiac remodeling in an injured heart. In the remodeling process, cardiac fibroblasts proliferate and differentiate into myofibroblasts, which secrete extracellular matrix to support the intact structure of the heart, and eventually differentiate into matrifibrocytes to form chronic scar tissue. Discovery of direct cardiac reprogramming offers a prom
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31

Sara, Ijaz Sohaira Shahid Hina Tahir. "AN ASSESSMENT OF POTENTIAL PENETRATING HEART DISEASES AND LUNG DISEASES AMONG PATIENTS DIAGNOSED WITH BOTH DISEASES." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 05, no. 12 (2018): 16677–81. https://doi.org/10.5281/zenodo.2457157.

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<strong><em>Objective:</em></strong><em> Multiple research and real-life experiences have alarmingly made the case that heart injury (penetrating) usually carries the fatal potential. It mostly happens because of cardiac blockage or haemorrhage (exsanguinous). Therefore, this research aims to assess the patients who are suffering from lungs and penetrating heart diseases. Furthermore, through this study, we also took an account of findings and results of both ailments as a whole. </em> <strong><em>Methodology:</em></strong><em> We included (20) patients who enrolled with lungs and penetrating
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32

Surinkaew, Poomarin, Nattayaporn Apaijai, Passakorn Sawaddiruk, et al. "Mitochondrial Fusion Promoter Alleviates Brain Damage in Rats with Cardiac Ischemia/Reperfusion Injury." Journal of Alzheimer's Disease 77, no. 3 (2020): 993–1003. http://dx.doi.org/10.3233/jad-200495.

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Background: Cardiac ischemia/reperfusion (I/R) injury induces brain damage through increased blood-brain barrier (BBB) breakdown, microglial hyperactivity, pro-inflammatory cytokines, amyloid-β deposition, loss of dendritic spines, brain mitochondrial dysfunction, and imbalanced mitochondrial dynamics. Previous studies demonstrated that mitochondrial fusion promoter reduced cardiac damage from cardiac I/R injury; however, following cardiac I/R injury, the roles of mitochondrial dynamics on the brain have not been investigated. Objective: To investigate the effects of pharmacological modulation
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33

Vatutin, N. T., G. A. Ignatenko, G. G. Taradin, E. V. Eshchenko, M. S. Goncharuk, and S. O. Kulikova. "Cardiac disorders in burn injury." Bulletin of Siberian Medicine 19, no. 4 (2021): 198–206. http://dx.doi.org/10.20538/1682-0363-2020-4-198-206.

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34

HATTORI, Yoshinobu, Tadashi IRIYAMA, Koji WATANABE, et al. "A Review of Cardiac Injury." Nihon Gekakei Rengo Gakkaishi (Journal of Japanese College of Surgeons) 26, no. 1 (2001): 76–79. http://dx.doi.org/10.4030/jjcs1979.26.1_76.

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35

Helms, Julie, Alain Combes, and Nadia Aissaoui. "Cardiac injury in COVID-19." Intensive Care Medicine 48, no. 1 (2021): 111–13. http://dx.doi.org/10.1007/s00134-021-06555-3.

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36

Millichap, J. Gordon. "Cardiac Surgery and Brain Injury." Pediatric Neurology Briefs 7, no. 10 (1993): 73. http://dx.doi.org/10.15844/pedneurbriefs-7-10-1.

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37

El-Menyar, Ayman, Hassan Al Thani, Ahmad Zarour, and Rifat Latifi. "Understanding traumatic blunt cardiac injury." Annals of Cardiac Anaesthesia 15, no. 4 (2012): 287. http://dx.doi.org/10.4103/0971-9784.101875.

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38

Hudson, A. J. "Cardiac air gun pellet injury." Emergency Medicine Journal 18, no. 6 (2001): 519. http://dx.doi.org/10.1136/emj.18.6.519.

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39

Fleisch, Martin, and Bernhard Meier. "Radiation Therapy–Induced Cardiac Injury." Circulation 96, no. 7 (1997): 2462–63. http://dx.doi.org/10.1161/01.cir.96.7.2462.

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40

Preiss, Michael, Kirsten Besler, and Hans-Reinhard Zerkowski. "Suicidal crossbow bolt cardiac injury." Surgery 133, no. 2 (2003): 228–29. http://dx.doi.org/10.1067/msy.2003.35.

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41

Chan, Christopher T., Guo Hua Li, Alan Valaperti, and Peter Liu. "Intensive Hemodialysis Preserved Cardiac injury." ASAIO Journal 61, no. 5 (2015): 613–19. http://dx.doi.org/10.1097/mat.0000000000000255.

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42

ROSATO, RALPH M., MARC J. SHAPIRO, MARY J. KEEGAN, ROBERT H. CONNORS, and CLAUDE B. MINOR. "Cardiac Injury Complicating Traumatic Asphyxia." Journal of Trauma: Injury, Infection, and Critical Care 31, no. 10 (1991): 1387–89. http://dx.doi.org/10.1097/00005373-199110000-00012.

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43

Clancy, Keith, Catherine Velopulos, Jaroslaw W. Bilaniuk, et al. "Screening for blunt cardiac injury." Journal of Trauma and Acute Care Surgery 73 (November 2012): S301—S306. http://dx.doi.org/10.1097/ta.0b013e318270193a.

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44

Engelhardt, W., and E. Hartung. "BRAIN INJURY AFTER CARDIAC SURGERY." Journal of Neurosurgical Anesthesiology 9, no. 1 (1997): 101. http://dx.doi.org/10.1097/00008506-199701000-00089.

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45

Cingoz, Faruk, Hakan Bingol, Erkan Kuralay, and Harun Tatar. "A Concealed Penetrating Cardiac Injury." Journal of Cardiac Surgery 22, no. 4 (2007): 350–52. http://dx.doi.org/10.1111/j.1540-8191.2007.00354.x.

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46

Straus, Jason P., Randy J. Woods, Mary C. McCarthy, Mark P. Anstadt, and Nancy Kwon. "Cardiac pneumatic nail gun injury." Journal of Thoracic and Cardiovascular Surgery 132, no. 3 (2006): 702–3. http://dx.doi.org/10.1016/j.jtcvs.2006.04.030.

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47

MATTOX, KENNETH L., MARIAN C. LIMACHER, DAVID V. FELICIANO, et al. "Cardiac Evaluation following Heart Injury." Journal of Trauma: Injury, Infection, and Critical Care 25, no. 8 (1985): 758–65. http://dx.doi.org/10.1097/00005373-198508000-00005.

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48

Li, Xue-feng, and Xian Wang. "Acupuncture therapy related cardiac injury." Chinese Journal of Integrative Medicine 19, no. 12 (2013): 885–88. http://dx.doi.org/10.1007/s11655-013-1355-4.

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49

El Bekkali, Youssef, Abdellatif Boulahya, Abdelmajid El Kirat, and Hassane El Kabiri. "Trans-sternal penetrating cardiac injury." Indian Journal of Thoracic and Cardiovascular Surgery 24, no. 2 (2008): 164. http://dx.doi.org/10.1007/s12055-008-0029-x.

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50

Wiwanitkit, Viroj. "Troponin determination and cardiac injury." Injury 42, no. 9 (2011): 965–66. http://dx.doi.org/10.1016/j.injury.2010.07.503.

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