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Relevant bibliographies by topics / Cardiac receptors. Opioids

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Journal articles
Dissertations / Theses

Academic literature on the topic 'Cardiac receptors. Opioids'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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Journal articles on the topic "Cardiac receptors. Opioids"

1

Kunecki, Marcin, Wojciech Płazak, Piotr Podolec, and Krzysztof S. Gołba. "Effects of endogenous cardioprotective mechanisms on ischemia-reperfusion injury." Postępy Higieny i Medycyny Doświadczalnej 71 (January 10, 2017): 20–31. http://dx.doi.org/10.5604/01.3001.0010.3786.

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Ischemic heart disease have been remarked as a leading cause of morbidity and mortality in adults. Early restoration of cardiac perfusion is necessary to restore perfusion of ischemic heart muscle. Effective revascularization reduce mortality by limiting myocardial necrosis at the acute phase of the cardiac infarction. However, reperfusion may induce a cascade of pathophysiological reactions causing the increase of the infarct area of the myocardium This phenomenon known as ischemia-reperfusion injury is responsible for up to 50% of the final infarct size. Sequences of brief episodes of nonlet

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Farias, Martin, Keith E. Jackson, Darice Yoshishige та James L. Caffrey. "Cardiac enkephalins interrupt vagal bradycardia via δ2-opioid receptors in sinoatrial node". American Journal of Physiology-Heart and Circulatory Physiology 284, № 5 (2003): H1693—H1701. http://dx.doi.org/10.1152/ajpheart.00730.2002.

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Local cardiac opioids appear to be important in determining the quality of vagal control of heart rate. Introduction of the endogenous opioid methionine-enkephalin-arginine-phenylalanine (MEAP) into the interstitium of the canine sinoatrial node by microdialysis attenuates vagally mediated bradycardia through a δ-opioid receptor mechanism. The following studies were conducted to test the hypothesis that a δ2-opiate receptor subtype mediates the interruption of vagal transmission. Twenty mongrel dogs were anesthetized and instrumented with microdialysis probes inserted into the sinoatrial node.

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Venkatesan, Priya, Sunit Baxi, Cory Evans, Robert Neff, Xin Wang та David Mendelowitz. "Glycinergic Inputs to Cardiac Vagal Neurons in the Nucleus Ambiguus Are Inhibited by Nociceptin and μ-Selective Opioids". Journal of Neurophysiology 90, № 3 (2003): 1581–88. http://dx.doi.org/10.1152/jn.01117.2002.

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Most parasympathetic regulation of heart rate originates from preganglionic cardiac vagal neurons within the nucleus ambiguus. Little is known regarding the modulation of glycinergic transmission to these neurons. However, the presence of μ-opioid receptors and opioid-receptor-like (ORL1) receptors within the ambiguus, together with the presence of endogenous ligands for both receptor types in the same area, suggests opioids may modulate synaptic transmission to cardiac vagal neurons. This study therefore examined the effects of endomorphin-1 and endomorphin-2 (the μ-selective endogenous pepti

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Bolte, Craig, Gilbert Newman та Jo El J. Schultz. "Hypertensive state, independent of hypertrophy, exhibits an attenuated decrease in systolic function on cardiac κ-opioid receptor stimulation". American Journal of Physiology-Heart and Circulatory Physiology 296, № 4 (2009): H967—H975. http://dx.doi.org/10.1152/ajpheart.00909.2008.

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Opioids/opiates are commonly administered to alleviate pain, unload the heart, or decrease breathlessness in patients with advanced heart failure. As such, it is important to evaluate whether the myocardial opioidergic system is altered in cardiac disease. A hamster model of spontaneous hypertension was investigated before the development of hypertension (1 mo of age) and in the hypertensive state (10 mo of age) to evaluate the effect of prolonged hypertension on myocardial opioidergic activity. Plasma β-endorphin was decreased before the development of hypertension and in the hypertensive sta

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Imai, N., M. Kashiki, P. D. Woolf, and C. S. Liang. "Comparison of cardiovascular effects of mu- and delta-opioid receptor antagonists in dogs with congestive heart failure." American Journal of Physiology-Heart and Circulatory Physiology 267, no. 3 (1994): H912—H917. http://dx.doi.org/10.1152/ajpheart.1994.267.3.h912.

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We have shown previously that right heart failure (RHF) in dogs is associated with activated endogenous opiate systems, and that administration of the opioid receptor antagonist, naloxone, increases arterial pressure, cardiac contractile function and organ blood flows. To study whether the cardiovascular effects of naloxone are mediated via the mu- or delta-opioid receptors, we administered ICI-154,129, a delta-receptor antagonist, and naloxonazine, a mu-receptor antagonist, to 10 conscious dogs with RHF on 2 separate days. Like naloxone, ICI-154,129 increased mean aortic pressure, cardiac out

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Kienbaum, Peter, Norbert Thurauf, Martin C. Michel, Norbert Scherbaum, Markus Gastpar, and Jurgen Peters. "Profound Increase in Epinephrine Concentration in Plasma and Cardiovascular Stimulation after [micro sign]-Opioid Receptor Blockade in Opioid-addicted Patients during Barbiturate-induced Anesthesia for Acute Detoxification." Anesthesiology 88, no. 5 (1998): 1154–61. http://dx.doi.org/10.1097/00000542-199805000-00004.

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Background Acute displacement of opioids from their receptors by administration of large doses of opioid antagonists during general anesthesia is a new approach for detoxification of patients addicted to opioids. The authors tested the hypothesis that mu-opioid receptor blockade by naloxone induces cardiovascular stimulation mediated by the sympathoadrenal system. Methods Heart rate, cardiac index, and intravascular pressures were measured in 10 patients addicted to opioids (drug history; mean +/- SD, 71 +/- 51 months) during a program of methadone substitution (96 +/- 57 mg/day). Cardiovascul

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Lishmanov, Yu B., L. N. Maslov, N. Yu Naryzhnaya, et al. "ENDOGENOUS OPIOID SYSTEM AS A MEDIATOR OF ACUTE AND LONG-TERM ADAPTATION TO STRESS. PROSPECTS FOR CLINICAL USE OF OPIOID PEPTIDES." Annals of the Russian academy of medical sciences 67, no. 6 (2012): 73–82. http://dx.doi.org/10.15690/vramn.v67i6.287.

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It has been well established that opioid peptides (OPs) affect various hormonal systems. Opioids exhibit stress-limiting and gastro-protective effects in stressed animals, acting via μ- and δ-opioid receptors (OR). Peripheral μ-OR stimulation by endogenous and exogenous opioids increases cardiac tolerance to pathological consequences of stress. Enhancement of prostacyclin synthesis, decrease of thromboxane production as well as suppression of lipid peroxidation can be directly responsible for cardioprotective effects of OPs in stressed animals. Adaptive responses are accompanied by increased O

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8

Fu, Liang-Wu, and John C. Longhurst. "Functional role of peripheral opioid receptors in the regulation of cardiac spinal afferent nerve activity during myocardial ischemia." American Journal of Physiology-Heart and Circulatory Physiology 305, no. 1 (2013): H76—H85. http://dx.doi.org/10.1152/ajpheart.00091.2013.

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Thinly myelinated Aδ-fiber and unmyelinated C-fiber cardiac sympathetic (spinal) sensory nerve fibers are activated during myocardial ischemia to transmit the sensation of angina pectoris. Although recent observations showed that myocardial ischemia increases the concentrations of opioid peptides and that the stimulation of peripheral opioid receptors inhibits chemically induced visceral and somatic nociception, the role of opioids in cardiac spinal afferent signaling during myocardial ischemia has not been studied. The present study tested the hypothesis that peripheral opioid receptors modul

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9

Periyasamy, S. M. "Inhibition of cardiac sarcolemmal Na+/H+ antiporter by opioids." Canadian Journal of Physiology and Pharmacology 70, no. 7 (1992): 1048–56. http://dx.doi.org/10.1139/y92-144.

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In our routine screening of chemicals that would inhibit cardiac sarcolemmal Na+/H+ antiporter, we discovered that some of the opioids produced inhibition of cardiac sarcolemmal Na+/H+ antiporter in micromolar concentrations. Using U-50,488H, a selective κ-opioid agonist, we characterized the nature of interaction between opioids and the Na+/H+ antiporter. The inhibitory effect of U-50,488H on Na+/H+ antiporter was immediate and reversible, and was not mediated through the interaction with the opioid receptors but due to the direct interaction of U-50.488H with the Na+/H+ antiporter. The kinet

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10

McLaughlin, P. J. "Regulation of DNA synthesis of myocardial and epicardial cells in developing rat heart by [Met5]enkephalin." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 271, no. 1 (1996): R122—R129. http://dx.doi.org/10.1152/ajpregu.1996.271.1.r122.

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Endogenous opioids serve as negative growth factors in neural and nonneural tissues in addition to being neuromodulators. This study investigated the hypothesis that native opioid peptides are inhibitory growth factors in heart development. DNA synthesis of ventricular myocardial and epicardial cells in 1-day-old rats was examined. Administration of a variety of opioids and peptides revealed that [Met5]enkephalin had the greatest inhibitory effect on DNA synthesis; peptides related to mu-, delta-, kappa-, epsilon-, and sigma-receptors had no influence on cell proliferation, even at concentrati

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Dissertations / Theses on the topic "Cardiac receptors. Opioids"

1

Zhang, Weimin, and 張為民. "Cardiac k-opioid receptor: multiplicity, regulation, signal transduction and function." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B3123804X.

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Zhang, Weimin. "Cardiac k-opioid receptor : multiplicity, regulation, signal transduction and function /." Hong Kong : University of Hong Kong, 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19588999.

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卞勁松 and Jin-song Bian. "The role of protein kinase C upon K-opioid receptor stimulation in theheart." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2000. http://hub.hku.hk/bib/B31239900.

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Bian, Jin-song. "The role of protein kinase C upon K-opioid receptor stimulation in the heart /." Hong Kong : University of Hong Kong, 2000. http://sunzi.lib.hku.hk/hkuto/record.jsp?B21790863.

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Gu, Hong. "Opioid/Adrenergic Interaction in Regulating Canine Cardiac Function." Thesis, University of North Texas, 1990. https://digital.library.unt.edu/ark:/67531/metadc331004/.

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Opioid/adrenergic interactions were studied to evaluate two hypotheses: (1) naloxone potentiates the effect of epinephrine on cardiac contractility by increasing circulating epinephrine concentrations; and (2) endogenous and exogenous opioids alter left cardiac nerve stimulationinduced norepinephrine release and cardiac function. A canine isolated heart-lung preparation was used for the first study. Plasma epinephrine was determined and myocardial epinephrine uptake was calculated during intravenous epinephrine infusion. Naloxone (4 mg) was given and the epinephrine infusion was repeated. Nalo

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盛建中 and Jianzhong Sheng. "Phosphoinositol/Ca2+ pathway in the cardiac k-opioid receptor: physiological role and alternations upontolerance." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B31237654.

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Sitsapesan, R. "Opioid receptors and ischaemia-induced cardiac arrhythmias." Thesis, University of Strathclyde, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381536.

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Tai, Kwok-keung, and 戴國強. "A study on the cardiac k-opioid receptors: function, binding properties & signal transduction." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1993. http://hub.hku.hk/bib/B31233211.

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Tai, Kwok-keung. "A study on the cardiac k-opioid receptors : function, binding properties & signal transduction /." [Hong Kong] : University of Hong Kong, 1993. http://sunzi.lib.hku.hk/hkuto/record.jsp?B13441863.

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Sheng, Jianzhong. "Phosphoinositol/Ca2+ pathway in the cardiac k-opioid receptor : physiological role and alternations upon tolerance /." Hong Kong : University of Hong Kong, 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19616193.

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