Relevant bibliographies by topics / Cardiac Resynchronization Therapy Devices

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Cardiac Resynchronization Therapy Devices'

Author: Grafiati
Published: 4 June 2021
Last updated: 26 January 2023

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Journal articles on the topic "Cardiac Resynchronization Therapy Devices"

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Chapa, Deborah W., Hyeon-Joo Lee, Chi-Wen Kao, Erika Friedmann, Sue A. Thomas, Jill Anderson, and Gust H. Bardy. "Reducing Mortality With Device Therapy in Heart Failure Patients Without Ventricular Arrhythmias." American Journal of Critical Care 17, no. 5 (September 1, 2008): 443–52. http://dx.doi.org/10.4037/ajcc2008.17.5.443.

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Use of device therapy to prevent sudden cardiac death in patients with heart failure is expanding on the basis of evidence from recent clinical trials. Three multicenter prospective clinical trials—Sudden Cardiac Death in Heart Failure (SCD-HeFT); Comparison of Medical Therapy, Pacing, and Defibrillation in Heart Failure (COMPANION); and Cardiac Resynchronization-Heart Failure (CARE-HF)—were conducted to determine the effectiveness of devices in reducing mortality in patients with heart failure who did not have a history of ventricular arrhythmias. The 3 trials varied in the devices used, the population of patients included, and the study designs. In SCD-HeFT, implantable cardioverter defibrillators were more effective than pharmacological therapy in preventing mortality among patients with mild to moderate heart failure. In COMPANION, cardiac resynchronization therapy alone and cardiac resynchronization therapy plus an implantable cardioverter defibrillator were more effective than optimal drug treatment in reducing morbidity and all-cause mortality in patients with moderate to severe heart failure. In CARE-HF, cardiac resynchronization therapy alone was more effective than optimal drug treatment in reducing all-cause mortality in patients with moderate to severe heart failure. No direct comparison of the devices used has been done. These 3 clinical trials provide clear evidence that device therapy is beneficial for some patients with heart failure, even patients who do not have a history of ventricular arrhythmia.
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Bitakova, Fatima I., Victoria E. Gumerova, Elizaveta V. Zbyshevskaya, Vera Y. Zimina, Tatiana N. Novikova, Rodion V. Ratmanov, Sergey A. Saiganov, and Vladislava A. Shcherbakova. "Nuances of Cardiac Resynchronization Therapy in Patients with Dilated Cardiomyopathy and Atrial Fibrillation (a Clinical Case)." Cardiac Arrhythmias 1, no. 1 (June 15, 2021): 33–38. http://dx.doi.org/10.17816/cardar75647.

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Dilated cardiomyopathy (DCM) is a steadily developing disease characterized by progressive chronic heart failure (CHF) resistant to drug therapy. Cardiac resynchronization therapy (CRT) significantly improves the prognosis in these patients if they have indications for implantation of resynchronization devices. The article presents a clinical case of successful implantation of a cardioversion-defibrillation cardiac resynchronization device in a patient suffering from DCM in combination with permanent atrial fibrillation (AF). The nuances of ventricular rate control and the role of the catheter procedure for modifying the atrioventricular junction are discussed.
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Bangaar, Dr Ashish. "Octogenarian with Severe Left Ventricular Dysfunction and Cardiac Resynchronization TherapyDefibrillator Device: Successful Anesthesia Management for Hip Arthroplasty." Corpus Journal of Case Reports (CJCR) 3, no. 3 (July 29, 2022): 1–3. http://dx.doi.org/10.54026/cjcr/1024.

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Cardiac resynchronization therapy has emerged as a key component in management of advanced heart failure with reduced ejection fraction in selected subset of geriatric patients. The burgeoning burden of comorbidities, complexity of these devices and aging population presenting for surgeries necessitates peri-operative physicians to be cognizant and confident to handle these challenging cases. Limited literature about anesthesia management of patients implanted with these devices add to the situational challenge. We present a case of elderly gentleman with ischemic cardiomyopathy and cardiac resynchronization therapy-defibrillator device who underwent hip replacement under general anesthesia. Understanding the disease and the device are the crucial and definitive exigencies to manage such cases.
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Gardini, Armando, Pierpaolo Lupo, Emanuela Zanelli, Silvia Bisetti, and Riccardo Cappato. "Diagnostic capabilities of devices for cardiac resynchronization therapy." Journal of Cardiovascular Medicine 11, no. 3 (March 2010): 186–89. http://dx.doi.org/10.2459/jcm.0b013e3283303036.

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Krishnan, Subramaniam C., Laurens F. Tops, and Jeroen J. Bax. "Cardiac Resynchronization Therapy Devices Guided by Imaging Technology." JACC: Cardiovascular Imaging 2, no. 2 (February 2009): 226–30. http://dx.doi.org/10.1016/j.jcmg.2008.11.010.

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Yang, Ying chi, Thein Tun Aung, and Abdul Wase. "Inappropriate Defibrillator Shocks due to Mechanical Inference from an Investigational Device." Case Reports in Cardiology 2019 (January 6, 2019): 1–3. http://dx.doi.org/10.1155/2019/2810396.

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Cardiac contractility modulation (CCM) is an investigational device-based therapy to enhance ventricular contractility in systolic heart failure patients who are not candidates for cardiac resynchronization therapy (CRT) owing to the absence of wide QRS complexes or who have failed to respond on CRT. The principal mechanism is based on the stimulation of cardiac muscles by nonexcitatory electrical signals to augment the influx of calcium ions into the cardiomyocytes. The majority of patients receiving CCM therapy have concurrent implantable cardioverter defibrillators, and the manufacturer declares both devices can be used in parallel without any interactions. Nevertheless, proper lead positioning of both devices are crucial, and it is mandatory to check device-device interactions during each and every cardiac electronic implantable device-related procedure to prevent adverse outcomes.
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Malik, Jahanzeb, and Kashif Khan. "Cardiac implantable electronic device related chest pain: A focused review." Journal of Shifa Tameer-e-Millat University 4, no. 1 (August 1, 2021): 50–54. http://dx.doi.org/10.32593/jstmu/vol4.iss1.130.

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More than 600,000 patients undergo cardiac implantable electronic device (CIED) implantation in a year, which comprise of pacemakers, implantable cardioverter defibrillators, and cardiac resynchronization therapy devices (CRT). The most common symptom experienced after a CIED implantation is chest pain. In this review, we describe CIED implantation and associated complications causing chest pain.
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Imamura, Teruhiko. "Ischemic Etiology and Clinical Outcomes Following Cardiac Resynchronization Therapy." Medicina 56, no. 3 (March 4, 2020): 110. http://dx.doi.org/10.3390/medicina56030110.

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Optimal patient selection for cardiac resynchronization therapy is crucial. There are several concerns that allow to better clarify the association between the ischemic etiology of heart failure and the response to cardiac resynchronization therapy. The type of ischemic coronary disease has an impact on the responses to cardiac resynchronization therapy. The prognostic impact of cardiac resynchronization therapy on cardiac death including heart transplantation and durable ventricular assist device implantation is another concern.
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Bontempi, Luca, Francesca Vassanelli, and Antonio Curnis. "Resynchronization Therapy in the Elderly." European Journal of Arrhythmia & Electrophysiology 02, no. 01 (2016): 18. http://dx.doi.org/10.17925/ejae.2016.02.01.18.

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Implantable cardioverter-defibrillators (ICDs) are increasingly used in patients ≥80 years old, and the proportion of devices incorporating cardiac resynchronization therapy (CRT) increases with patient age. In this era of patient-centered care, important questions arise about the patterns of use, clinical outcomes and cost-effectiveness of CRT for older patients. Currently, few data are available that specifically address the effects of CRT in the elderly.
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Świerżyńska, Ewa, and Maciej Sterliński. "Decreases in biventricular pacing percentage in remote monitoring of patients with cardiac implantable electronic devices." In a good rythm 1, no. 62 (May 31, 2022): 17–20. http://dx.doi.org/10.5604/01.3001.0015.9157.

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Heart failure is a serious disease and is one of the top causes of death in Poland. A proven method of treatment in some heart failure patients is resynchronization therapy using implantable devices such as cardioverter-defibrillators (CRT-D) or pacemakers (CRT-P). One of the conditions for the effectiveness of this therapy is achieving and maintaining a high biventricular pacing percentage. Remote monitoring of cardiac implantable electronic devices allows daily access to information about a device’s operating status. In patients with heart failure, access and treatment options based on remote monitoring data reduces hospitalization, mortality and treatment costs. This article discusses how information received via remote monitoring about the loss of resynchronization stimulation can be used to determine possible abnormalities and the need for treatment.
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Dissertations / Theses on the topic "Cardiac Resynchronization Therapy Devices"

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Sciaraffia, Elena. "Cardiac Resynchronization Therapy Optimization : Comparison and Evaluation of Non-invasive Methods." Doctoral thesis, Uppsala universitet, Kardiologi-arrytmi, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-179785.

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The general purpose of this thesis was to investigate new cardiac resynchronization therapy (CRT) optimization techniques and to assess their reliability when compared to invasive measurements of left ventricular contractility (LV dP/dtmax).We first assessed whether cardiac output (CO) measured by trans-thoracic impedance cardiography could correctly identify the optimal interventricular (VV) pacing interval while using invasive measurements of LV dP/dtmax as reference. We did not find any significant statistical correlation between the two optimizing methods when their corresponding optimal VV intervals were compared. We also tested the hypothesis that measurements of right ventricular contractility (RV dP/dtmax) could be used to guide VV delay optimization in CRT. The comparison of optimal VV intervals obtained from the left and right ventricular dP/dtmax did not show a statistically significant correlation; however, a positive correlation was found when broader VV intervals were evaluated and we concluded that this finding deserves further investigation. An interesting alternative for CRT optimization is the use of device integrated algorithms or sensors capable to adapt the CRT settings to the current needs of the individual patient. In this respect we investigated the use of cardiogenic impedance (CI) measurements obtained through the CRT-D device as a method for CRT optimization with invasive measurements of LV dP/dtmax as a reference. Our results showed that CI could be measured through the device after implantation and that a patient-specific impedance-based prediction model was capable to accurately predict the optimal AV and VV delays. To follow up on these positive results we re-evaluated the patient-specific impedance-based prediction models 24 hours post implantation and investigated the possibility of calibrating them using parameters derived from non-invasive measurements of arterial pressure obtained by finger pelthysmography at implantation.The results showed that the patient-specific impedance-based prediction models did not perform as well on the follow-up data as they did on the data from implantation day and that they correlated poorly with plethysmographic parameters. Our studies suggest that novel methods for CRT optimization should be thoroughly evaluated and compared to established measures of left ventricular function prior to introduction into clinical practice.
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Kyriacou, Andreas. "Haemodynamic optimization of cardiac resynchronization therapy." Thesis, Imperial College London, 2011. http://hdl.handle.net/10044/1/24666.

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Heart failure carries a very poor prognosis, unless treated with the appropriate pharmacological agents which, have been evaluated in large randomized clinical trials and have demonstrated improvements in morbidity and mortality of this cohort of patients. A significant proportion of these patients develop conduction abnormalities involving both the atrioventricular node and also the specialised conduction tissue (bundle of His and Purkinje fibers) of the ventricular myocardium which is most commonly evidenced by the presence of a wide QRS, typically left bundle branch block. The net effect of these conduction abnormalities is inefficient filling and contraction of the left ventricle. The presence of these conduction abnormalities is an additional strong marker of poor prognosis. Over the last 15 years pacing treatments have been developed aimed at mitigating the conduction disease. Large scale randomized multicentre trials have repeatedly demonstrated the effectiveness of cardiac pacing, officially recognized as cardiac resynchronization therapy (CRT). This mode of pacing therapy has undoubtedly had a positive impact on both the morbidity and mortality of these patients. Despite the large advancement in the management of heart failure patients by pacing therapies, a significant proportion of patients (30%) being offered CRT are classed as non-responders. Many explanations have been put forward for the lack of response. The presence of scar at the pacing site with failure to capture or delayed capture of myocardium, too much left ventricular scar therefore minimal contractile response, incorrect pacing site due to often limited anatomical options of lead placement and insufficient programming i.e optimization, of pacemaker settings such as the AV and VV delay are just some of the suggested areas perceived to be responsible for the lack of patients' response to cardiac resynchronization therapy. The effect of optimization of pacemaker settings is a field that has been investigated extensively in the last decade. Disappointingly, current methods of assessing the effect of optimization of pacemaker settings on several haemodynamic parameters, such as cardiac output and blood pressure, are marred with very poor reproducibility, so measurement of any effect of optimization is close to being meaningless. Moreover, detailed understanding of the effects of CRT on coronary physiology and cardiac mechanoenergetics is equally, disappointingly, lacking. In this thesis, I investigated the acute effects of cardiac resynchronization therapy and AV optimization on coronary physiology and cardiac mechanoenergetics. This was accomplished using very detailed and demanding series of invasive catheterization studies. I used novel analytical mathematical techniques, such as wave intensity analysis, which have been developed locally and this provided a unique insight of the important physiological entities defining coronary physiology and cardiac mechanics. I explored in detail the application and reliability of photoplethysmography as a tool for non-invasive optimization of the AV delay. Photoplethysmography has the potential of miniaturization and therefore implantation alongside pacemaker devices. I compared current optimization techniques (Echocardiography and ECG) of VV delay against beat-to-beat blood pressure using the Finometer device and defined the criteria that a technique requires if such a technique can be used meaningfully for the optimization of pacemaker settings both in clinical practice and in clinical trials. Finally, I investigated the impact of atrial pacing and heart rate on the optimal AV delay and attempted to characterize the mechanisms underlying any changes of the optimal AV delay under these varying patient and pacing states. In this thesis I found that optimization of AV delay of cardiac resynchronization therapy not only improved cardiac contraction and external cardiac work, but also cardiac relaxation and coronary blood flow, when compared against LBBB. I found that most of the increase in coronary blood flow occurred during diastole and that the predominant drive for this was ventricular microcirculatory suction as evidenced by the increased intracoronary diastolic backward-travelling decompression wave. I showed that non-invasive haemodynamic optimization using the plethysmograph signal of an inexpensive pulse oximeter is as reliable as using the Finometer. Appropriate processing of the oximetric signal improved the reproducibility of the optimal AV delay. The advantage of this technology is that it might be miniaturized and implanted to provide automated optimization. In this thesis I found that other commonly used modalities of VV optimization such as echocardiography and ECG lack internal validity as opposed to non-invasive haemodynamic optimization using blood pressure. This finding will encourage avoidance of internally invalid modalities, which may cause more harm than good. In this thesis I found that the sensed and paced optimal AV delays have, on average, a bigger difference than the one assumed by the device manufacturers and clinicians. As a significant proportion of patients will be atrially paced, especially during exercise, optimization during this mode of pacing is equally crucial as it is during atrial sensing. Finally, I found that the optimal AV delay decreases with increasing heart rate, and the slope of this is within the range of existing pacemaker algorithms used for rate adaptation of AV delay, strengthening the argument for the rate adaptation to be programmed on.
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Miri, Raz. "Computer assisted optimization of cardiac resynchronization therapy." Karlsruhe Univ.-Verl, 2008. http://d-nb.info/994987250/04.

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Vernooy, Kevin. "Dyssynchronopathy and the role of cardiac resynchronization therapy." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Universiteit Maastricht [host], 2006. http://arno.unimaas.nl/show.cgi?fid=5665.

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Hettwer, Peter Jacob. "Individualized Cardiac Resynchronization Therapy: Next Generation Pacemaker Controls." Thesis, North Dakota State University, 2015. https://hdl.handle.net/10365/27887.

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Cardiac dyssynchrony (CD) causes some heart muscle regions to contract at different times, and current treatments do not help 30 ? 50% of patients. In this thesis, multi-site pacing control schemes are created to quantitatively and automatically reduce the CD of ventricular wall accelerations by adjusting pacing times. Two and four left ventricular region models are investigated containing model variables that represent numerous muscle parameters. Optimization is performed using exhaustive search and genetic algorithm techniques, with particular attention paid to the latter with regard to development, parameter selection, and limitations. Relative to treatments firing all regions simultaneously, timing adjustment improves acceleration CD by up to 56%. Furthermore, simulations also demonstrate improvements to dyssynchronous region power generation and workload by up to 50% and up to 15% decrease in healthy region workload. Thus, the current model indicates it may be possible to improve acceleration CD by adjustments to regional firing times.
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Suever, Jonathan D. "MRI methods for predicting response to cardiac resynchronization therapy." Diss., Georgia Institute of Technology, 2013. http://hdl.handle.net/1853/50224.

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Cardiac Resynchronization Therapy (CRT) is a treatment option for heart failure patients with ventricular dyssynchrony. CRT corrects for dyssynchrony by electrically stimulating the septal and lateral walls of the left ventricle (LV), forcing synchronous con- traction and improving cardiac output. Current selection criteria for CRT rely upon the QRS duration, measured from a surface electrocardiogram, as a marker of electrical dyssynchrony. Unfortunately, 30-40% of patients undergoing CRT fail to benefit from the treatment. A multitude of studies have shown that presence of mechanical dyssynchrony in the LV is an important factor in determining if a patient will benefit from CRT. Furthermore, recent evidence suggests that patient response can be improved by placing the LV pacing lead in the most dyssynchronous or latest contracting segment. The overall goal of this project was to develop methods that allow for accurate assessment and display of regional mechanical dyssynchrony throughout the LV and at the site of the LV pacing lead. To accomplish this goal, we developed a method for quantifying regional dyssynchrony from standard short-axis cine magnetic resonance (MR) images. To assess the effects of LV lead placement, we developed a registration method that allows us to project the LV lead location from dual-plane fluoroscopy onto MR measurements of cardiac function. By applying these techniques in patients undergoing CRT, we were able to investigate the relationship between regional dyssynchrony, LV pacing lead location, and CRT response.
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Yılmaz, Ayten. "Studies on cardiac pacing emphasis on pacemaker sensors and cardiac resynchronization therapy /." [S.l. : Amsterdam : s.n.] ; Universiteit van Amsterdam [Host], 2005. http://dare.uva.nl/document/79548.

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Aloysius, Romila Mariette. "Market Analysis of Cardiac Electrical Mapping Platform in the Cardiac Resynchronization Therapy Market." Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1363099595.

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Lane, Rebecca Elizabeth. "The assessment of ventricular dyssynchrony and optimisation of cardiac resynchronization therapy." Thesis, University of London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.542956.

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Salukhe, Tushar Vilas. "An Investigation into the Mechanisms of Haemodynamic and Clinical Gain From cardiac Resynchronization Therapy." Thesis, Imperial College London, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.498901.

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Books on the topic "Cardiac Resynchronization Therapy Devices"

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Mayo Foundation for Medical Education and Research, ed. Cardiac pacing, defibrillation, and resynchronization: A clinical approach. 3rd ed. Chichester, West Sussex: Wiley-Blackwell, 2013.

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Cheuk-Man, Yu, Hayes David L, and Auricchio Angelo, eds. Cardiac resynchronization therapy. Malden, Mass: Blackwell Pub., 2006.

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Cheuk-Man, Yu, Hayes David L, and Auricchio Angelo, eds. Cardiac resynchronization therapy. 2nd ed. Malden, Mass: Blackwell Pub., 2008.

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Yu, Cheuk-Man, David L. Hayes, and Angelo Auricchio, eds. Cardiac Resynchronization Therapy. Oxford, UK: Blackwell Futura, 2008. http://dx.doi.org/10.1002/9781444300246.

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Barold, S. Serge, and Philippe Ritter, eds. Devices for Cardiac Resynchronization. Boston, MA: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-71167-6.

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Abraham, William T. Cardiac resynchronization therapy in heart failure. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2010.

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Abraham, William T. Cardiac resynchronization therapy in heart failure. Philadelphia: Wolters Kluwer Health/Lippincott Williams & Wilkins, 2010.

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Clinical cardiac pacing, defibrillation, and resynchronization therapy. 4th ed. Philadelphia, PA: Elsevier/Saunders, 2011.

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McAlister, Finlay. Cardiac resynchronization therapy for congestive heart failure. Rockville, MD: U.S. Department of Health and Human Services, Agency for Healthcare Research and Quality, 2004.

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Miri, Raz. Computer assisted optimization of cardiac resynchronization therapy. Karlsruhe: Univ.-Verl., 2008.

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Book chapters on the topic "Cardiac Resynchronization Therapy Devices"

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Korpas, David. "Cardiac Resynchronization Therapy." In Implantable Cardiac Devices Technology, 87–90. Boston, MA: Springer US, 2013. http://dx.doi.org/10.1007/978-1-4614-6907-0_11.

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Lü, Fei. "Cardiac Resynchronization Therapy." In Handbook of Cardiac Anatomy, Physiology, and Devices, 475–97. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-372-5_28.

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Grenz, Nathan A., and Zhongping Yang. "Cardiac Resynchronization Therapy." In Handbook of Cardiac Anatomy, Physiology, and Devices, 577–97. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-19464-6_31.

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Frisch, Daniel, and Peter J. Zimetbaum. "Cardiac Resynchronization Therapy." In Device Therapy in Heart Failure, 185–213. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-424-7_7.

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Ramsdale, David R., and Archana Rao. "Cardiac Resynchronization Therapy." In Cardiac Pacing and Device Therapy, 357–402. London: Springer London, 2012. http://dx.doi.org/10.1007/978-1-4471-2939-4_16.

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Aranda, Juan M. "Recurrent Heart Failure and Appropriate Evaluation After Cardiac Resynchronization Therapy." In Devices for Cardiac Resynchronization, 507–14. Boston, MA: Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-71167-6_26.

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Darden, Douglas, and Jonathan C. Hsu. "Indications for Cardiac Resynchronization Therapy." In Case-Based Device Therapy for Heart Failure, 287–99. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-70038-6_18.

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Shah, Kunal, and Farshad Raissi. "Cardiac Resynchronization Therapy Programming and Troubleshooting." In Case-Based Device Therapy for Heart Failure, 309–22. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-70038-6_20.

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Lin, Andrew, and Gordon Ho. "Cardiac Resynchronization Therapy in Patients with Left Ventricular Assist Devices." In Case-Based Device Therapy for Heart Failure, 337–41. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-70038-6_22.

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Verdino, Ralph J. "Feeling Poorly after an Upgrade to a Cardiac Resynchronization Therapy Device." In Cardiac Electrophysiology, 557–60. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-28533-3_131.

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Conference papers on the topic "Cardiac Resynchronization Therapy Devices"

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Mohindra, R., J. L. Sapp, J. C. Clements, and B. M. Horacek. "Use of body-surface potential mapping and computer model simulations for optimal programming of cardiac resynchronization therapy devices." In 2007 34th Annual Computers in Cardiology Conference. IEEE, 2007. http://dx.doi.org/10.1109/cic.2007.4745423.

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Sun, Qing, Francois Schwartz, Jacques Michel, and Yannick Herve. "A reinforcement learning algorithm used in analog spiking neural network for an adaptive cardiac Resynchronization Therapy device." In 2010 IEEE International Symposium on Circuits and Systems - ISCAS 2010. IEEE, 2010. http://dx.doi.org/10.1109/iscas.2010.5537111.

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Pindor, Jakub, Marek Penhaker, Jindrich Cernohorsky, David Korpas, and Vladimir Vancura. "Evaluation of effect cardiac resynchronization therapy." In 2011 1st Middle East Conference on Biomedical Engineering (MECBME). IEEE, 2011. http://dx.doi.org/10.1109/mecbme.2011.5752120.

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Albatat, Mohammad, D. Ryan King, Laura A. Unger, Hermenegild Arevalo, Samuel Wall, Joakim Sundnes, Jacob Bergsland, and Ilangko Balasingham. "Electromechanical Model to Predict Cardiac Resynchronization Therapy." In 2018 40th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2018. http://dx.doi.org/10.1109/embc.2018.8513539.

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Lee, Angela, Uyen Chau Nguyen, Justin Gould, Baldeep Sidhu, Benjamin Sieniewicz, Caroline Mendonca Costa, Frits Prinzen, et al. "Predicting Activation Patterns in Cardiac Resynchronization Therapy Patients." In 2018 Computing in Cardiology Conference. Computing in Cardiology, 2018. http://dx.doi.org/10.22489/cinc.2018.079.

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Yang, Yang, Ryo Saegusa, Shuji Hashimoto, Naoyoshi Aoyama, and Kenji Hasegawa. "Modeling of Individual Left Ventricle for Cardiac Resynchronization Therapy." In 2006 IEEE International Conference on Robotics and Biomimetics. IEEE, 2006. http://dx.doi.org/10.1109/robio.2006.340139.

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Reiml, Sabrina, Daniel Toth, Maria Panayiotou, Bernhard Fahn, Rashed Karim, Jonathan M. Behar, Christopher A. Rinaldi, et al. "Interactive visualization for scar transmurality in cardiac resynchronization therapy." In SPIE Medical Imaging, edited by Robert J. Webster and Ziv R. Yaniv. SPIE, 2016. http://dx.doi.org/10.1117/12.2214737.

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Lazzari, Claudio De, and Nicola Alessandri. "Energetic Ventricular Balance During Cardiac Resynchronization Therapy: Numerical Simulation." In Biomedical Engineering. Calgary,AB,Canada: ACTAPRESS, 2010. http://dx.doi.org/10.2316/p.2010.723-115.

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Lazzari, Claudio De, and Nicola Alessandri. "Energetic Ventricular Balance during Cardiac Resynchronization Therapy: Numerical Simulation." In Biomedical Engineering. Calgary,AB,Canada: ACTAPRESS, 2011. http://dx.doi.org/10.2316/p.2011.723-115.

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Seger, Michael, Michael Netzer, Bernhard Pfeifer, Friedrich Hanser, Christian Baumgartner, Florian Hintringer, Otmar Pachinger, et al. "ASSESSMENT OF CARDIAC RESYNCHRONIZATION THERAPY BY NON-INVASIVE RECONSTRUCTION OF CARDIAC ACTIVATION TIMES." In Biomedical Engineering. Calgary,AB,Canada: ACTAPRESS, 2010. http://dx.doi.org/10.2316/p.2010.723-154.

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Reports on the topic "Cardiac Resynchronization Therapy Devices"

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Muyassar, YUSUP, Zhen yu DONG, Yan mei LU, Yao dong LI, Xian hui ZHOU, and Bao peng TANG. Fragmented QRS complex and responsiveness to cardiac resynchronization therapy: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2022. http://dx.doi.org/10.37766/inplasy2022.10.0101.

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